ライフサイエンスコーパス: FRA

1 FRA can be universally performed for heterogeneous, mult

2 FRA discovered 314 hits, 56 of which were determined sta

3 FRA was demonstrated to be important to pathogenesis, wh

4 FRA-1 expression was localized in the differentiating ut

5 FRA-1 forms activator protein-1 complexes in association

6 FRA-1 greatly enhanced the rate of proliferation, motili

7 FRA-1 has been implicated in the development of airway s

8 FRA-1, a member of the FOS family of transcription facto

9 transcription factor FOS-related antigen 1 (FRA-1) as a central node in tumour cell plasticity netwo

10 uding the proinvasive Fos-related antigen 1 (FRA-1) oncoprotein.

11 study we report that Fos-related antigen 1 (FRA-1), a member of the Fos family of transcription fact

12 and that vimentin and fos-related antigen-1 (FRA-1) were consistently overexpressed in the more highl

13 A total of 100 FRA were enrolled in a cross-sectional study that assess

14 One env mutant, which was detected in a FRA-induced thymic lymphosarcoma, had a large internal d

15 ual differences in face recognition ability (FRA), during face learning (Experiment 1) and face recog

16 sociated with strong firing rate adaptation (FRA) and occurred preferentially in large multipolar neu

17 xpressing FRAs at 7:00 PM but did not affect FRAs expression at 7:00 AM when compared with animals re

18 Antibodies against FRAs and tyrosine hydroxylase (TH) identified activated

19 lt intake among females of reproductive age (FRA) in the Oromia region of Ethiopia.

20 and nonpregnant females of reproductive age (FRA; 15-49 y).

21 n downstream effector of PI3K, did not alter FRA-1 expression.

22 discovery tool tile-based F-ratio analysis (FRA), which reduced the large data set to only the chrom

23 ive form of Fisher ratio (F-ratio) analysis (FRA) is developed for use with comprehensive two-dimensi

24 hed for the Fisher ratio (F-ratio) analysis (FRA) with the estimation of tile hit importance using th

25 y to introduce fractional response analysis (FRA), which quantifies changes in fractions of cells wit

26 Frequency response analyzer (FRA) algorithm was used to obtain impedance spectra so a

27 nduced by the AAR, whereas JUN-D, FRA-1, and FRA-2 were not.

28 Vietnam) conducted among PSC (n = 9880) and FRA (n = 14,749) from the Biomarkers Reflecting Inflamma

29 r the pathologic differences between F6A and FRA is the lower propensity for F6A to undergo de novo r

30 pershift analysis with antisera to c-Fos and FRA proteins demonstrated that 4-Fos and the 35 kDa FRA

31 Surprisingly, GUKH and FRA modulate the DL and DPP gradient levels and this reg

32 rather than individual) activity of MPII and FRA is required for the overall B. fragilis virulence in

33 and proteolytic characteristics of MPII and FRA.

34 t substrate cleavage preferences of MPII and FRA.

35 ogens activate both the PI3K-Akt pathway and FRA-1 expression in human bronchial epithelial (HBE) cel

36 th respect to neurons lacking strong PRF and FRA.

37 Among both PSC and FRA, correlations between inflammation and vitamin D bio

38 ly localized the genes for PAPA syndrome and FRA to chromosome 15q and suggested that they are the sa

39 trials with fibrinogen receptor antagonists (FRAs) have been associated with thrombocytopenia.

40 and two-dimensional Western blots with anti-FRA and anti-FosB antibodies.

41 atum of c-Fos, a 35 kDa Fos-related antigen (FRA) and the phosphorylated form of cyclic AMP response

42 gene products Fos and its related antigens (FRA) in SS-immunoreactive (IR) neurons in the PeVN.

43 expression of Fos and its related antigens (FRA) in tuberoinfundibular dopamine (TIDA) neurons locat

44 ling and expression of Fos related antigens (FRAs) as an indicator of neuronal activation.

45 atic nucleus (SCN) and Fos-related antigens (FRAs) expression in neuroendocrine dopaminergic neurons

46 oteins, termed chronic Fos-related antigens (FRAs), that are induced in brain in a region-specific ma

47 ix classes based on frequency response area (FRA) shapes and sideband inhibition depended both on FRA

48 constructing tonal frequency response areas (FRAs) in primary auditory cortex for different SNRs, ton

49 The areas of nonV frequency response areas (FRAs) were modulated, but the areas of almost all V-shap

50 AI frequency response areas (FRAs), derived from tone bursts presented to the poorer

51 M #604416) and familial recurrent arthritis (FRA) are rare inherited disorders of early onset, primar

52 ure Organization Forest Resource Assessment (FRA) in all tropical regions, especially for the 1980s.

53 sing data from Forest Resources Assessments (FRAs) of the United Nations Food and Agriculture Organiz

54 ly caused by folate receptor autoantibodies (FRAs) that interfere with folate transport across the bl

55 t not during learning, was related to better FRAs.

56 face recognition was associated with better FRAs.

57 n 16 children, the concentration of blocking FRA significantly correlated with cerebrospinal fluid 5-

58 Both FRA-1 and PBX1 are required for the mesenchymal changes

59 Interestingly, the -283 to +32 bp FRA-1 promoter fragment containing an SRE and an ATF sit

60 We show in this study that the chronic FRAs are isoforms of deltaFosB, a truncated splice varia

61 nd 37 kDa proteins correspond to the chronic FRAs that are induced in brain by chronic treatments, wh

62 ue to antibody cross-reactivity with chronic FRAs, and that behavioral sensitization to amphetamine i

63 closely related subgroup A molecular clone, FRA, we demonstrated high pathogenicity and a strong pro

64 rats, 10-15% of all SS-IR neurons contained FRA-IR.

65 1.5 h) in the percentage of TH-IR containing FRA-IR nuclei.

66 d the percentage of TH-IR neurons containing FRA-IR nuclei in the DM-ARC, but this effect was of long

67 (TH)-immunoreactive (IR) neurons containing FRA-IR nuclei in the DM-ARC.

68 eased the number of SS-IR neurons containing FRA-IR, without affecting the total number of SS-IR neur

69 ns or the number of SS-IR neurons containing FRA-IR.

70 In contrast, FRA-1 failed to promote tumor formation by BEAS-2B.

71 FOS-B was induced by the AAR, whereas JUN-D, FRA-1, and FRA-2 were not.

72 Deletion of either FRA gene has the same effect on transcription as deletio

73 ated in high iron medium, deletion of either FRA gene results in the translocation of the low iron-se

74 d the percentage of TH-IR neurons expressing FRA in the DM-ARC, but had no effect on haloperidol-indu

75 rease in the number of SS neurons expressing FRA-IR.

76 uroendocrine dopaminergic neurons expressing FRAs at 7:00 PM but did not affect FRAs expression at 7:

77 uroendocrine dopaminergic neurons expressing FRAs was significantly lower than all other time points.

78 ergic neurons in the ARN and PeVN expressing FRAs was greatest and equivalent at 7:00 AM, 9:00 AM, 12

79 NF), Food and Agricultural Organization (FAO FRA), and University of Maryland (Landsat forests), but

80 ion coefficients between CRP and 25(OH)D for FRA ranged from -0.11 to 0.14, and correlations between

81 expanded bandwidths, as is usually seen for FRAs obtained without background noise.

82 ese results suggest that alterations in Fos, FRA and CREB transcription factors are common neuronal r

83 ranscription factor family genes (FOS, FOSB, FRA-1) were particularly upregulated in chemotherapy-res

84 imum threshold parameters are extracted from FRAs, and they are used to quantify interaural response

85 of the newly identified IR-responsive genes, FRA-1 and ATF3, showed a p53-associated component to the

86 be a reasonable and non-invasive approach in FRA-positive children with ASD.

87 Prolactin causes a short-lived increase in FRA expression in TIDA neurons in the VL-ARC which is fo

88 ce of FRAs and the response to leucovorin in FRA-positive children has not been systematically invest

89 ed animals had decreased c-Fos and increased FRA proteins in striatum.

90 oxicants and carcinogens persistently induce FRA-1 expression in vitro and in vivo.

91 Akt had no significant effect on CS-induced FRA-1 expression.

92 K-Akt pathway, completely blocked CS-induced FRA-1 expression.

93 Interestingly, FRA-1 controls the expression of matrix metalloproteinas

94 teins demonstrated that 4-Fos and the 35 kDa FRA are components of the striatal AP-1 binding complex

95 ed epidermal growth factor receptor-mediated FRA-1 induction in non-oncogenic HBE cells.

96 In athymic nude mice, FRA-1, but not the control vector, rapidly enhanced tumo

97 ise levels reduced firing rates and narrowed FRA bandwidths; at higher SNRs, however, increasing the

98 h included 8/24 unique to control-normalized FRA and 16/24 in common with the standard FRA.

99 ce in the patient class, "control-normalized FRA" has been developed to provide complementary informa

100 llowing both standard and control-normalized FRA, the concentration ratio for the top 30 "hits" in ea

101 passed the t-test for the control-normalized FRA, which included 8/24 unique to control-normalized FR

102 with 5/21 undiscovered by control-normalized FRA.

103 s followed by a more prolonged activation of FRA expression in TIDA neurons in the DM-ARC.

104 C axis through transcriptional activation of FRA-1, which binds and activates an evolutionary conserv

105 erstood, it is poorly defined in the case of FRA-1.

106 ining neurons in the PeVN, but expression of FRA in SS neurons is not tonically inhibited by dopamine

107 hown a high level of persistent induction of FRA-1 by lung carcinogens, such as cigarette smoke and a

108 owever, is insufficient for the induction of FRA-1 promoter.

109 effect on haloperidol-induced inhibition of FRA expression in TH-IR neurons in the VL-ARC.

110 n convertases, whereas the cleavage motif of FRA (Pro-X-X-Leu-(Arg/Ala/Leu) downward arrow) resembles

111 cessary and sufficient for the regulation of FRA-1 in response to mitogens.

112 However, the exact roles of FRA-1 in regulating lung epithelial cell growth and inva

113 short interfering RNA-mediated silencing of FRA-1 enhances TNF-alpha-induced IL-8 expression, wherea

114 n amino acids of the N-terminal mature SU of FRA env gene, followed by eight amino acids from the fra

115 wn to have an infectivity similar to that of FRA when delivered as a provirus.

116 en with ASD and a high prevalence (75.3%) of FRAs was found.

117 euronal activity, indicated by expression of FRAs, was observed in the same neuron populations after

118 determine the time course of the presence of FRAs and STAT5 in the nuclei of hypothalamic dopaminergi

119 In children with ASD, the prevalence of FRAs and the response to leucovorin in FRA-positive chil

120 stimates of 1.9 (0.6-2.5) Pg.yr(-1) based on FRA national statistics of changes in forest area, this

121 pes and sideband inhibition depended both on FRA shapes and cell types.

122 of JNK activity had no significant effect on FRA-1 induction.

123 prevalence of vitamin D deficiency in PSC or FRA.

124 s (FeLV), which we named FeLV-A (Rickard) or FRA, was characterized with respect to viral interferenc

125 More than 20 other FRAs, including those that induced thrombocytopenia in c

126 ne this aspect, we have stably overexpressed FRA-1 in human type-II-like alveolar malignant cell line

127 y Test-Chinese" which measured participants' FRAs, and (2) an "old/new recognition memory test" encom

128 enous administration of two different potent FRAs, L-738, 167 and L-739,758.

129 rhesus monkey upon administration of potent FRAs.

130 ression of the immediate early gene products FRA in SS-containing neurons in the PeVN, but expression

131 ression of an ERK1 mutant strikingly reduced FRA-1 transcription.

132 I3K through p21-activated kinase 1 regulates FRA-1 proto-oncogene induction by CS and the subsequent

133 he env gene in the ecotropic FeLV-A Rickard (FRA) provirus.

134 In this study, serum FRA concentrations were measured in 93 children with ASD

135 ulated, but the areas of almost all V-shaped FRAs were not.

136 tegrate-and-fire-type model, which simulated FRA and PRF, reproduced the phase lead observed in large

137 imilarity to known chromosomal fragile site (FRA) sequences, this polymorphic 1p21.2 sequence may rep

138 Specifically, FRA provides a supervised discovery of metabolites that

139 Because standard FRA is adversely impacted by metabolites expressed with

140 Likewise, standard FRA provided 21/30 metabolites passing the t-test, with

141 ed FRA and 16/24 in common with the standard FRA.

142 ression significantly reduced TPA-stimulated FRA-1 promoter activity.

143 new molecular clone, FeLV-A, Rickard strain (FRA).

144 in primary uterine stromal cells suppressed FRA-1 expression.

145 nd showed slower progression to disease than FRA-infected cats.

146 consistently carried higher virus loads than FRA-infected cats.

147 s that FRAs may be important in ASD and that FRA-positive children with ASD may benefit from leucovor

148 We propose that FRA-1-overexpressing clones featuring high plasticity un

149 We suggest that FRA-1 can promote motility, invasion, and anchorage-inde

150 Collectively, these results suggest that FRA-1, induced in response to estrogen signaling via ESR

151 This study suggests that FRAs may be important in ASD and that FRA-positive child

152 The FRA-1 proto-oncogene is overexpressed in a variety of hu

153 contrast to an 11% reduction reported by the FRA.

154 , and an ATF site, is also necessary for the FRA-1 induction by TPA and EGF.

155 We also discuss evidence from the FRA-1 perspective supporting the notion that clonal sele

156 omparison with FRA3, a representative of the FRA isoforms.

157 ted by direct intradermal inoculation of the FRA plasmid DNA so as to eliminate the possibility of co

158 Our recent analysis of the FRA-1 promoter has shown a critical role for a TRE locat

159 EB proteins bind to SRE and ATF sites of the FRA-1 promoter, constitutively.

160 t TNF-alpha stimulates the expression of the FRA-1 protooncogene in human pulmonary epithelial cells

161 These observations suggest that the FRA proteins are in the same signal transduction pathway

162 d the subsequent recruitment of c-Jun to the FRA-1 promoter in response to TNF-alpha in pulmonary epi

163 , and ATF1, which constitutively bind to the FRA-1 promoter, but also suppressed the recruitment of c

164 However, while the FRA discovered a more comprehensive hit list (65 hits),

165 Scientists who use FRA data should check how the accuracy of their findings

166 tassium currents contributed to PRF, whereas FRA was predominantly mediated by slow potassium current

167 ease in the percentage of TH-IR neurons with FRA-IR nuclei in the VL-ARC.

168 Children with FRAs were treated with oral leucovorin calcium (2 mg kg(