ライフサイエンスコーパス: FSS

1 FSS and SHS mutations affect different myosin residues,

2 FSS applied to endothelial cells (ECs) triggers signalin

3 FSS demonstrated a better predictive accuracy for MACE c

4 FSS increased prostaglandin (PG) E2 release in WT cells

5 FSS increases TPOT's efficiency in application on big da

6 FSS is a self-reported questionnaire consisting of nine

7 FSS provides the option to specify subsets of the featur

8 FSS rapidly increases the intracellular calcium concentr

9 FSS scores were collected by trained abstractors to refl

10 FSS was determined by only counting ischemia-producing l

11 FSS, POPC and PCPC after three months were identical bet

12 FSS-stimulated apical endocytosis was initiated between

13 V-1 Frameshift Stimulating Signal", or HIV-1 FSS) controls the frameshift efficiency and has been hyp

14 ounds displaying high affinity for the HIV-1 FSS.

15 We exposed MPT cells to 0.2 dynes/cm(2) FSS for 3 h and performed confocal microscopy and Wester

16 Scale 7 (FSS-7) score >5; low fatigue n=20, FSS-7 score <3) participated in the study.

17 high fatigue n=21, Fatigue Severity Scale 7 (FSS-7) score >5; low fatigue n=20, FSS-7 score <3) parti

18 test for COVID-19, Fatigue Severity Scale-7 (FSS-7) or Brain Fog Severity Scale (BFSS) score of 28 or

19 al-by-trial modulation of the anticipatory a FSS measure during training, concomitant plasticity of s

20 al-by-trial modulation of the anticipatory a FSS signal as well as significant improvement of sustain

21 oop neurofeedback (NF) of the anticipatory a FSS signal over 10 d of training.

22 w that neurofeedback (NF) training of this a FSS signal within the attention task is feasible.

23 junctional buttressing" model explains why a FSS of only 1/10 of that used in the EC study can cause

24 losing yield gaps, these regions can achieve FSS, which also reduces international trade and increase

25 "minor limitations with strenuous activity" (FSS=9); and 15% scored </=8.

26 l significantly reduced 4 but not 24 h after FSS.

27 Correlations were performed among FSS and motor scales, 6-minute walk test (6MWT) and Hamm

28 An FSS of greater than 4.0 was not observed in controls (me

29 Abnormal fatigue, defined as an FSS score >or=4, was present in 67% of the subjects.

30 cores in each POPC or PCPC interval, with an FSS score increase with each worsening POPC/PCPC rating.

31 ficantly better scores on both the SF-36 and FSS.

32 ith a phenotype intermediate between DA1 and FSS.

33 The HRQoL and FSS were comparable in the 36 mo after liver transplanta

34 No significant differences in HrQol and FSS were seen between the 2 groups.

35 tween these estimated doses and the PBLS and FSS thyroid cancer cases.

36 e transform a single-band FSS to a dual-band FSS.

37 netic resonators, we transform a single-band FSS to a dual-band FSS.

38 were moderate to strong correlations between FSS score and patient-reported anchors (vitality questio

39 re [r = 0.70]) and weak correlations between FSS score and physiological measures (FVC [r = -0.24], p

40 a significant (P < .001) difference between FSS scores in each POPC or PCPC interval, with an FSS sc

41 o elucidate the mechanism of synergy between FSS and SMAD signaling in vascular stability and how dis

42 Accordingly, miR-100 inhibition blunts FSS- and TGFB-inducible Wnt signaling.

43 -like cells, we found that Akt activation by FSS occurred through two parallel pathways; one required

44 ional connections were severely disrupted by FSS.

45 tors for the WT and three of the most common FSS mutants: R672H, R672C, and T178I.

46 nship between the FSS neurologic components (FSS-CNS) and the PCPC.

47 son between simulated and measured conformal FSS results is in good agreement.

48 A comparison between the conformal FSS and a finite planar FSS is presented through simulat

49 te the advantages of utilizing the conformal FSS in the near-field.

50 The conformal FSS is 3D printed and copper electroplated, which leads

51 Consequently, FSS and Template help reduce TPOT computation time and m

52 PT cells exposed to continuous FSS also acquired an extensive brush border and basolate

53 we show that cells cultured under continuous FSS develop an expanded apical endocytic pathway and inc

54 In contrast, FSS-induced V-ATPase redistribution and expression are l

55 R FSS-supplemented media compared to control FSS-supplemented media.

56 C scores and the baseline and PICU discharge FSS scores, the dispersion of FSS scores within each of

57 In this paper, a spherical dome FSS is presented, aiming to provide improved angular sta

58 Primarily in CC-genotype ECs, FSS elicited a marked rise in COX (cyclooxygenase)-2 and

59 gned to identify miRNAs involved in elevated FSS-induced collateral vessel growth in rat hind limbs.

60 We previously showed that elevated FSS increases PLGF in a reactive oxygen species (ROS)-de

61 i-inflammatory phenotype through an enhanced FSS-dependent release of 15d-PGJ(2).

62 action of suicides completed with a firearm (FSS).

63 t Akt phosphorylation and were necessary for FSS to induce nuclear translocation of beta-catenin, c-f

64 eover, addition of ATP bypassed the need for FSS in enhancing endocytic capacity.

65 The wines made from FSS were methanol free and contained higher levels of te

66 dentify factors associated with abnormal FS (FSS</=8) post-HT.

67 ompared with the more objective and granular FSS.

68 HFR, FSS, and their correlations and differences.

69 % of patients in the low-, medium-, and high-FSS groups, respectively (p < 0.001).

70 Children with delirium (n = 12) had a higher FSS (10.0 [9.8, 12.0] vs. 7.5 [7.0, 9.0], p = 0.009) at

71 Therefore, our results identify FSS-responsive miRNAs in osteocytes, including one that

72 ning of the endocytic response by changes in FSS may contribute to glomerulotubular balance in vivo.

73 ted cells is rapidly modulated by changes in FSS.

74 ponsible for skeletal muscle contractures in FSS patients.

75 an intact microtubule network is critical in FSS-induced modulation of V-ATPase in proximal tubule ce

76 Sustained increases or decreases in FSS induce vessel remodeling to maintain proper perfusio

77 Increases in FSS scores occur with each higher POPC and PCPC rating a

78 showed 94 differentially expressed miRNAs in FSS-stressed collaterals including miRNA-352 which was d

79 ctin cytoskeleton plays an important role in FSS-induced NHE3 and Na/K-ATPase trafficking, and an int

80 sion models explained 62% of the variance in FSS and 78% of the variance in ProF-S scores.

81 Variations in FSS may also contribute to the maturation of PT cells du

82 How endothelial cells (ECs) integrate FSS and BMP signals in vascular development and homeosta

83 arting at the 5' resolution, we investigated FSS from the lowest administrative levels to continents.

84 t types replicated less (P < 0.05) with IUGR FSS-supplemented media compared to control FSS-supplemen

85 ent, 64% were "fully active/no limitations" (FSS=10), 21% had "minor limitations with strenuous activ

86 Mean FSS score was 6.6.

87 Higher mean FSS scores, indicating greater fatigue, were observed am

88 After 10 months mean FSS score had not differed from baseline, 3.84 +/- 1.25.

89 After 6 months mean FSS score significantly reduced to 3.92 +/- 1.35.

90 The mean FSS score for all 28 patients at baseline was 4.61 +/- 1

91 Methods: FSS scores and several anchors were measured in 1,881 pa

92 Moreover, FSS provides an essential stimulus in the differentiatio

93 2.5-11.7 years]), 74 (17.3%) of whom had new FSS domain morbidity at discharge.

94 The proportion of new FSS domain morbidity was highest among those with multip

95 cy for MACE compared with SS (Harrell's C of FSS, 0.677 vs. SS, 0.630, p = 0.02; integrated discrimin

96 satility, and directional characteristics of FSS are constantly sensed by the endothelium.

97 The chemical composition of FSS generated from the premium quality Vostitsa currants

98 te negative correlation of the difference of FSS and 6MWT after 6 months compared to baseline conditi

99 PICU discharge FSS scores, the dispersion of FSS scores within each of the POPC/PCPC ratings, and the

100 The dispersion of FSS scores within each POPC and PCPC rating was substant

101 The effect of FSS on NOR performance was further examined in mice lack

102 n and HO-1 knockdown abolished the effect of FSS on PLGF.

103 S exposure on gene expression and effects of FSS at differing rates on gene expression in hPTCs has n

104 hypothesis that HO-1 mediates the effects of FSS on PLGF.

105 To investigate the effects of FSS on TGFB signaling in osteocytes, we stimulated osteo

106 tubule cells (PTCs) were subjected to 5 h of FSS (1 dyn/cm(2)) to investigate the dynamic responses o

107 The impact of FSS exposure on gene expression and effects of FSS at di

108 ly typed GP and enables the incorporation of FSS at the beginning of each pipeline.

109 lines to physiologically relevant levels of FSS led to dramatically increased internalization of the

110 Culture under suboptimal levels of FSS led to intermediate phenotypes, suggesting a thresho

111 After 30 min of FSS, P2X7R-mediated pore formation was observed in wild

112 iated between 15 and 30 min postinduction of FSS, occurred via a clathrin- and dynamin-dependent path

113 There was no reduction of FSS 10 months after the beginning of treatment when comp

114 lectromagnetic waves, we morph the shapes of FSS designs based on origami patterns to attain new degr

115 so identifies potential biases in the use of FSS as a proxy for firearm ownership rates.

116 Only FSS and procedure time were independent predictors of 1-

117 Also, our origami FSS can fold/unfold thereby tuning (i.e., reconfiguring)

118 to examine longitudinal patterns in patient FSS categories and identify trajectory classes, with cla

119 atients with STS in whom carefully performed FSS may serve as definitive therapy and in whom adjuvant

120 Physiological FSS promotes and maintains vascular stability via synerg

121 At a physiological FSS level, we observed cell morphology, enhanced polariz

122 etween the conformal FSS and a finite planar FSS is presented through simulations at the frequency ra

123 ndition, and 110 (76%) had normal preillness FSS scores.

124 Therefore, the proposed FSS is a dynamic reconfigurable electromagnetic structur

125 ng in the murine tibia and by cellular scale FSS in OCY454 cells.

126 was used to compare Fatigue Severity Scale (FSS) and SF-36 scores during treatment by treatment grou

127 d reliability of the Fatigue Severity Scale (FSS) for measuring fatigue in a national cohort of patie

128 mpact Profile (SIP), Fatigue Severity Scale (FSS), and neurological examination.

129 al analog scale, the Fatigue Severity Scale (FSS), and the Profile of Fatigue (ProF).

130 as self-rated by the Fatigue Severity Scale (FSS).

131 measured within the Fatigue Severity Scale (FSS).

132 istent fatigue using Fatigue Severity Scale (FSS-7), a self-report questionnaire that has been widely

133 Finnish translation of the Flow Short Scale (FSS).

134 n skin colors on the Fitzpatrick Skin Scale (FSS).

135 measured using the Functional Status Scale (FSS) (scores range from 1 [normal] to 5 [very severe dys

136 al status using the Functional Status Scale (FSS) reflecting preillness baseline, PICU and hospital d

137 assessed using the Functional Status Scale (FSS), the pediatric overall performance category (POPC)

138 measured using the Functional Status Scale (FSS).

139 problematic fatigue (Fatigue Severity Scale [FSS] score >=4) were randomly assigned (1:1:1), using a

140 elation across states (r = 0.84), but scaled FSS differed from HFR by as many as 20 percentage points

141 Fatigue severity score (FSS) was 5.0 +/- 1.4 in patients with SLE, with 14 of th

142 atigue questionnaire Fatigue Severity Score (FSS).

143 2005 to 2014 with a functional status score (FSS) available at 3 time points (listing, transplant, >/

144 core (SS), termed "functional SYNTAX score" (FSS), would predict clinical outcome better than the cla

145 system's scalability: Feature Set Selector (FSS) and Template.

146 ts by applying Functional Source Separation (FSS) to their EEG recordings.

147 ining 10% control or IUGR fetal sheep serum (FSS).

148 gether called frameshift stimulating signal (FSS).

149 erates a considerable amount of side-stream (FSS) with great potential for biotechnological exploitat

150 er the influence of both fluid shear stress (FSS) and soluble factors.

151 Fluid shear stress (FSS) from blood flow acting on the endothelium criticall

152 Fluid shear stress (FSS) from blood flow sensed by vascular endothelial cell

153 Increased fluid shear stress (FSS) is a key initiating stimulus for arteriogenesis, th

154 is directly regulated by fluid shear stress (FSS) is unknown, despite work suggesting that fluid flow

155 GFR and the accompanying fluid shear stress (FSS) modulate acute changes in PT ion transport thought

156 l fluid flow, leading to fluid shear stress (FSS) of osteoblasts.

157 del to study the role of fluid shear stress (FSS) on apical NHE3 and V-ATPase and basolateral Na/K-AT

158 Kopathies is affected by fluid shear stress (FSS), a physiological stimulus caused by blood or lymph

159 y between filtrate flow, fluid shear stress (FSS), and functionality essential for understanding rena

160 erts a frictional force, fluid shear stress (FSS), on the endothelial cells that line the blood and l

161 tly enhanced by elevated fluid shear stress (FSS), the underlying regulatory mechanism of this proces

162 udy, we demonstrate that fluid shear stress (FSS)-induced actin cytoskeletal reorganization and junct

163 within 1 min of onset of fluid shear stress (FSS).

164 in osteocytes exposed to fluid shear stress (FSS).

165 as well as two levels of fluid shear stress (FSS).

166 onse to acute changes in fluid shear stress (FSS); however, it is not known whether GFR modulates PT

167 ter exposure to repeated forced swim stress (FSS).

168 cal food supply using food self-sufficiency (FSS) as an indicator.

169 dipole and two frequency selective surface (FSS) designs featuring isotropic and anisotropic respons

170 ntional planar frequency selective surfaces (FSSs) are characterized in the far-field region and they

171 Frequency selective surfaces (FSSs) have been used to control and shape electromagneti

172 ine survey (PBLS) and the full-scale survey (FSS).

173 habitual consumers of full-sugar sweetened (FSS-CSD) or low-calorie sweetened carbonated soft drinks

174 e first show that frontal-sensory synchrony (FSS) of a oscillations during attentive preparation sign

175 l sensory cortex [frontal-sensory synchrony (FSS)] significantly correlates with greater task perform

176 called the freshwater salinization syndrome (FSS).

177 s type 1 (DA1) and Freeman-Sheldon syndrome (FSS) are the two most common known causes of inherited m

178 Freeman-Sheldon syndrome (FSS) is a disease associated with missense mutations in

179 s for myosin-based Freeman-Sheldon syndrome (FSS), a dominant form of distal arthrogryposis defined b

180 (MYH3) gene cause Freeman-Sheldon syndrome (FSS), one of the most severe multiple congenital contrac

181 nherited disorder, Freeman-Sheldon syndrome (FSS).

182 Indeed, relative to treatment with TGFB, FSS induced a larger increase in levels of pSmad2/3 and

183 This study demonstrates that FSS acts through iron to induce pro-arteriogenic PLGF, s

184 We find that FSS rapidly upregulates Smad2/3 phosphorylation and TGFB

185 We found that FSS also affects apical endocytosis in PT cells.

186 remain unclear, these results implicate that FSS activates both pathways to generate a downstream res

187 ila models mimic the human condition in that FSS mutations are dominant and display varied degrees of

188 Our hypothesis is that FSS stimulates both apical and basolateral transporter e

189 We observed that FSS changes the expression of PTC-specific genes and imp

190 Immunofluorescence analysis revealed that FSS caused basal stress fiber disruption, more densely d

191 Our findings show that FSS leads to an increment in the amount of protein expre

192 Our results show that FSS stimulation rapidly induces phosphorylation of multi

193 Analysis showed that FSS has a rich volatilome (including Maillard reaction/l

194 The FSS and POPC/PCPC system are closely associated.

195 The FSS mobilizes organic carbon, nutrients, heavy metals, a

196 The FSS possesses acceptable performance characteristics for

197 The FSS scores for the good and mild disability POPC/PCPC ra

198 We examined the HRQoL and the FSS over the course of the study by fitting generalized

199 The relationship between the PCPC and the FSS-CNS paralleled the relationship between the FSS and

200 reference of the USA LCS-CSD cohort, and the FSS-CSD and LCS-CSD cohorts in Mexico.

201 -CNS paralleled the relationship between the FSS and POPC/PCPC system.

202 PC ratings, and the relationship between the FSS neurologic components (FSS-CNS) and the PCPC.

203 ignificant (P < .001) difference between the FSS-CNS scores between each of the PCPC ratings with inc

204 isrupting actin by cytochalasin D blocks the FSS-induced changes in NHE3 and Na/K-ATPase, but not V-A

205 usinersen reduces fatigue as measured by the FSS in adult patients with 5q-SMA transiently after init

206 After determining the FSS for each patient, 32% moved to a lower-risk group as

207 ch of the PCPC ratings with increases in the FSS-CNS score for each higher PCPC rating.

208 e sensitivity of ECs to flow by lowering the FSS set point, with resulting AVMs exhibiting features o

209 l actors may work collectively to manage the FSS in the absence of top-down regulatory controls.

210 sults suggest that the nine questions of the FSS reflect one dimension of fatigue.

211 However, the dispersion of the FSS scores indicated a lack of precision in the POPC/PCP

212 es were observed for EX or NEX groups on the FSS.

213 In contrast, rapamycin did not prevent the FSS-induced increase in Na(+)/K(+)-ATPase levels.

214 , and was rapidly reversed upon removing the FSS.

215 Measurements and Main Results: The FSS demonstrated high internal consistency (Cronbach's a

216 In this feature, we propose that the FSS can be understood as a common pool resource problem

217 ngest predictors of fatigue according to the FSS and the somatic fatigue domain of the ProF (ProF-S),

218 Using the FSS, fatigue was measured in 28 adult patients, subdivid

219 We then evaluated how correlated was the FSS with the Flow State Scale and Flow Core Scale.

220 Finally, we evaluated how well the FSS distinguished Flow-inducing experiences from boring

221 compared to its planar counterpart when the FSS is placed in the near-field region of an antenna sou

222 shows that the dose-response curve with the FSS data clearly differs from that with the PBLS data.

223 ned by mechanically stretching its arms, the FSSs exhibit distinct transmittance and reflectance spec

224 Though FSS activates both TGFB and Wnt signaling in osteocytes,

225 All three FSS mutations show dramatic changes in kinetic propertie

226 fermentation-derived volatiles, compared to FSS.

227 Exposure of human ECs to FSS effectively reduced monocyte transmigration particul

228 Dyn gene-disrupted mice exposed to FSS did not show the subsequent learning and memory defi

229 Cells exposed to FSS expressed higher levels of key proteins necessary fo

230 Exposure to FSS also caused a rapid elevation in intracellular Ca(2+

231 However, 1 h after exposure to FSS, vehicle-pretreated mice displayed a significant def

232 ZO-1, E-cadherin, vinculin, and paxillin to FSS.

233 present a dynamic view of hPTCs response to FSS with increasing fluidic shear stress conditions and

234 +)]i and purinergic signaling in response to FSS-dependent ciliary bending triggers a rapid and rever

235 n PIK3CA's role in mediating EC responses to FSS and discusses current understanding of PIK3CA dysreg

236 EC responses to FSS are mediated in part by a junctional mechanosensory

237 K in mediating physiological EC responses to FSS conditions characteristic of lymphatic and capillary

238 n humans, renders the gene less sensitive to FSS, resulting in a reduced endothelial cell (EC) capaci

239 ters in control cells and cells subjected to FSS.

240 We apply TPOT-FSS to real RNA-Seq data from a study of major depressiv

241 ith depression severity of two modules, TPOT-FSS corroborates that one of the modules is largely pred

242 Our simulations show TPOT-FSS significantly outperforms a tuned XGBoost model and

243 lectromagnetic structure whereas traditional FSSs are static and cannot change their performance.

244 were similarly effective in helping the USA FSS-CSD cohort maintain their preference for reduced-swe

245 Using FSS as a proof-of-concept, we show that candidate genes

246 his multi-institutional database study using FSS scores to describe a contemporary, heterogeneous pat

247 participants were FSS types 1-3, and 28 were FSS types 4-6.

248 Forty-six participants were FSS types 1-3, and 28 were FSS types 4-6.

249 ctional buttressing" model for PTCs in which FSS enables the DPABs, TJs, and AJs to become more tight

250 Comparing HFR estimates with FSS revealed the expected high overall correlation acros

251 cultured within a microfluidic platform with FSS.

252 The 7% LR rate after treatment of STS with FSS without RT reported herein is comparable to publishe

253 alized disease, 74 of whom were treated with FSS without RT (31%).