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1 ure (single monomer vs. tandemly repeated in Felis).
2 similar (97 to 99%) to "H. heilmannii" or H. felis.
3 eukin-10-deficient (IL-10(-/-)) mice with H. felis.
4 and the mice were orally inoculated with H. felis.
5 type (WT) C57BL/6 mice were infected with H. felis.
6 ion with Helicobacter pylori or Helicobacter felis.
7 nisms and evaluate the immune response to H. felis.
8 ainly CD4(+) for H. pylori and CD8(+) for H. felis.
9 cts mice against challenge with Helicobacter felis.
10 e to be designated a new species, Rickettsia felis.
11 nd congenic mice with a single isolate of H. felis.
12 h either Helicobacter pylori or Helicobacter felis.
13 a after infection of mice with H pylori or H felis.
14 n and IL-8Tg mice infected with Helicobacter felis.
15 urease B (ureB) gene of H. heilmannii and H. felis.
16 re positive for H. heilmannii and two for H. felis.
17 is of the sialome of cat flea Ctenocephaides felis.
19 re gavaged with H pylori SS1 or Helicobacter felis; 3 months later, stomachs, spleens, and sera were
20 s, 20 were qPCR positive for FHV-1, 7 for M. felis, 5 for FCV, 1 for C. felis, and 0 for B. bronchise
21 ridis (17 persons), C. canis (6 persons), C. felis (6 persons), and C. suis (1 person) infection.
23 ted to that of the Ohio-Florida strain of H. felis (89%) than to that of the California strain of H.
26 solates of A. fumigatus, A. viridinutans, A. felis, A. pseudoviridinutans, and A. wyomingensis but wa
27 vitro studies demonstrated that Helicobacter felis activates complement in normal mouse serum but not
28 erum from Rag2(-/-) mice, indicating that H. felis activates complement through the classical pathway
30 fection in a host infected with Helicobacter felis alters the natural outcome of T. gondii infection,
31 acteria were closely related to Helicobacter felis, although there was heterogeneity among the sequen
35 C57BL/6 (-/-)] were orally infected with H. felis and examined longitudinally using routine histolog
36 L/6-T-bet knockout (KO) litter mates with H. felis and examined the bacterial colonization, immune re
38 NS-GAS mice) were infected with Helicobacter felis and given the CCK2/gastrin receptor antagonist YF4
39 ori and non-Helicobacter pylori organisms H. felis and H. heilmannii and analyzed the association bet
42 that the major protein bands of Helicobacter felis and Helicobacter bizzozeronii, two Helicobacter sp
43 PRIL Tg mice were infected with Helicobacter felis and Helicobacter pylori and compared with noninfec
45 C57BL/6 mice were infected with Helicobacter felis and received bacterial eradication therapy after 2
46 currence of high relative copy numbers of C. felis and severe clinical signs in all cats was seen.
48 ently (e.g., Bartonella henselae, Rickettsia felis), and their mechanisms of transmission and impact
51 e orally infected with a single strain of H. felis, and 2 and 11 weeks after infection, the mice were
52 argeted mice were infected with Helicobacter felis, and a series of adoptive transfer experiments was
54 diarrhea, only infections with C. canis, C. felis, and subtype family Id of C. hominis were associat
55 of the H. muris strains, four strains of H. felis, and two strains of Eperythrozoon suis were sequen
56 atusRickettsia amblyommii,"R. montanensis,R. felis, andR. belliiwere frequently identified species, a
60 th C. hominis and C. parvum; C. canis and C. felis are responsible for only a small number of cases.
61 of recent reports have implicated Rickettsia felis as a human pathogen, paralleling the increasing de
63 after challenge with either H. pylori or H. felis, as confirmed by the complete absence of any bacte
64 ygous mice were inoculated with Helicobacter felis at 6 weeks of age and compared at various time poi
65 e-linked immunosorbent assay (ELISA) with H. felis ATCC 49179 antigen were performed with 101 serum s
71 L/6 x 129SvEv)F1] mice were infected with H. felis by oral gavage and were assessed histologically an
74 obacter pylori (H. pylori) in domestic cats (Felis cattus) less than 2 years of age has been well des
76 mental and theoretical analysis reveals that Felis catus exploits fluid inertia to defeat gravity and
77 present predominantly in one felid species: Felis catus GHV 1 (FcaGHV1) in domestic cats, Lynx rufus
78 sent the earliest evidence for domestic cat (Felis catus L., 1758) from Kazakhstan, found as a well p
79 ary fibroblasts isolated from domestic cats (Felis catus) and pumas (Puma concolor) with FeLV and qua
80 e disease and mortality in the domestic cat (Felis catus) and serves as a natural model for HIV infec
82 direction and retinal disparity in the cat (Felis catus) are all strongly related to the organizatio
84 nce spanning 758,291 bp of the domestic cat (Felis catus) extended and classical class II region.
89 nal referentiality in a common domestic cat (Felis catus) vocalization, the authors conducted 2 exper
90 and DRB gene homologues of the domestic cat (Felis catus) were cloned and sequenced to compare the pa
91 stic felids, vocalizations by domestic cats (Felis catus) were compared with cries by their closest w
92 entify felid GHVs, we screened domestic cat (Felis catus), bobcat (Lynx rufus), and puma (Puma concol
93 GHVs present in the blood of domestic cats (Felis catus), bobcats (Lynx rufus), and pumas (Puma conc
94 uenced the red and green opsin cDNAs of cat (Felis catus), horse (Equus caballus), gray squirrel (Sci
95 of FIV have been described for domestic cat (Felis catus), puma (Puma concolor), lion (Panthera leo),
98 roduced species, particularly the feral cat, Felis catus, and European red fox, Vulpes vulpes, and ch
99 bp mitochondrial genome of the domestic cat, Felis catus, has been sequenced and conforms largely to
100 comparative gene maps, the feline gene map (Felis catus, Order Carnivora, 2N = 38) displays the high
102 FHV-1), feline calicivirus (FCV), Mycoplasma felis, Chlamydophila felis, and Bordetella bronchiseptic
104 atment did not alter the overall level of H. felis colonization but did result in significant down-re
106 onounced gastric atrophy after short-term H. felis colonization with a similar extent of preneoplasia
108 A high percentage of free-ranging pumas (Felis concolor) are infected with feline lentiviruses (p
109 rresponding transmembrane segments of the H. felis CopA pump were identified by hydrophobicity analys
112 69 cats with cytauxzoonosis for which the C. felis cytb genotypes had been characterized previously.
115 tovaquone, a ubiquinone analogue, targets C. felis cytochrome b (cytb), of which 30 unique genotypes
118 felis were monitored for the presence of C. felis DNA on ocular swabs by using real-time PCR and for
121 um pratense 1 (Phl p 1) and the cat allergen Felis domesticus 1 (Fel d 1) greater than 3.5 Immuno Sol
122 st mite (Dermatophagoides farinae), and cat (Felis domesticus) allergens in home dust samples, and sp
128 vitro, induction of oxidative defense by H. felis failed to prevent a direct bacteriostatic effect a
129 tion to its annoyance to pets and humans, C. felis felis is responsible for flea bite allergy dermati
131 by experimental infection with Helicobacter felis, followed by antibiotic eradication therapy and su
132 mice chronically infected with Helicobacter felis for 2 mo with the SOM analogue octreotide resolved
139 6-bp DNA fragment of the 16S rRNA gene of H. felis from each of four experimentally infected cats at
140 rs from the numt previously described in the Felis genus in: (1) chromosomal location (F2-telomeric r
144 ther helicobacters (H. canis, H. cineadi, H. felis, H. mustelae, H. nemestrinae, H. pullorum, H. pylo
147 , or both, produced a specific fecal anti-H. felis IgA response, with the highest IgA levels occurrin
148 ection with T. gondii alters the specific H. felis immune response, converting a previously resistant
151 , paralleling the increasing detection of R. felis in arthropod hosts across the globe, primarily in
152 whole-cell sonicate vaccine of Helicobacter felis in conjunction with cholera toxin as a mucosal adj
153 Cellular stress was induced with IR and H felis in Ikkbeta(Deltastom), Ikkbeta(F/F), and cis-NF-ka
155 ed with a Florida isolate of Haemobartonella felis in order to collect organisms and evaluate the imm
160 out mice have increased susceptibility to H. felis-induced gastritis, with enhanced gastric inflammat
163 n of noggin in mice increased H pylori- or H felis-induced inflammation and epithelial cell prolifera
164 ative of an impaired Th1 component of the H. felis-induced inflammatory response when the influence o
165 of SPEM to the neoplastic process in the H. felis -infected C57BL/6 mouse, we have now studied the a
167 lesterol levels were observed between the H. felis-infected and -uninfected iNOS-/- mice in this stud
173 cter-associated gastric carcinogenesis in H. felis-infected mice on a uniform C57BL/6 background hous
176 us metaplasia in the fundic mucosa, while H. felis-infected wild-type mice had severe atrophic and me
177 cant reduction of gastric inflammation in H. felis-infected, as well as immunized/challenged, mice.
179 jective of this study was to determine if H. felis infection alters gastric histopathology, proinflam
181 adjuvant and examined for the presence of H. felis infection and leukocyte infiltration into the gast
182 R is a sensitive technique for monitoring C. felis infection and the response to antibiotic treatment
183 Ure produces a long-lasting inhibition of H. felis infection but that residual bacteria may produce a
186 s indicate that, in the absence of MyD88, H. felis infection enhances the activation of non-canonical
191 n the recent report of high prevalence of R. felis infection in patients with "fever of unknown origi
192 y regulate the epithelial consequences of H. felis infection in the stomach, while c-Rel-mediated sig
194 ogenesis, and novel experiments involving H. felis infection of bone marrow transplanted irradiated m
197 s of the antrum and pyloric junction, but H. felis infection of the Apc mutant mouse does not lead to
198 on between hypergastrinemia and Helicobacter felis infection on gastric corpus carcinogenesis in FVB/
202 as upregulated in myeloid cells with acute H felis infection, but in GECs by IR or long-term H felis
203 ted development of gastric pathology upon H. felis infection, but the mechanisms leading to this phen
205 emia, and possible synergy with Helicobacter felis infection, were investigated in insulin-gastrin (I
206 was observed in BALB/c and C3H/HeJ after H. felis infection, whereas sPLA2 expression was absent in
207 -deficient Stat3(SA/SA) mice with chronic H. felis infection, which mimics human H. pylori infection
224 characterized the cat flea (Ctenocephalides felis) innate immune response to R. typhi Initially, we
226 elopment of gastric atrophy and cancer in H. felis/INS-GAS mice, while the proton pump inhibitor show
228 t was concluded that gastritis induced by H. felis is associated with increased HbA(1c) levels in the
229 d Helicobacter that is closely related to H. felis, is associated with little or no gastritis, and sh
231 We have utilized the C57BL/6 Helicobacter felis mouse model to critically analyze the role of the
235 , and four gastric ulcer samples), 10 for H. felis (one gastritis, three duodenal ulcer, and six gast
237 own that infection of mice with Helicobacter felis or induction of acute parietal cell loss with the
238 fed with either blood meal infected with R. felis or infected cellular media administered in membran
239 could be specifically attributed to FCV, M. felis, or C. felis were seen, although interpretation in
240 ples from 8 children with C. meleagridis, C. felis, or C. parvum dog genotype were tested for anti-hu
241 R assay, the minimum detectable number of H. felis organisms was determined to be between 50 and 704.
242 tly up-regulated in WT mice infected with H. felis (P < 0.05) but were slightly elevated or were at b
243 h1-associated IgG2c antibody responses to H. felis (P <0.0003); the Th2-associated IgG1 responses wer
245 l role throughout the evolution of the genus Felis, predating cat exposure to feline retroviruses.
248 th 7 and 14 days of doxycycline decreased C. felis relative copy numbers and clinical signs rapidly.
251 cell-deficient TCRbetadelta-/- mice with H. felis resulted in high levels of colonization, but no de
252 on in the body of the stomach, lower anti-H. felis serum IgG antibody responses (although both the wi
256 st-virus evolution.IMPORTANCE Domestic cats (Felis silvestris catus) were domesticated from wildcats
258 youngest feline ERV groups in domestic cats (Felis silvestris catus); some members are replication co
261 979 domestic cats and their wild progenitors-Felis silvestris silvestris (European wildcat), F. s. ly
262 Here, we detected several ERV-DC loci in Felis silvestris silvestris Notably, a species-specific
263 we investigated ERV-DC in European wildcats (Felis silvestris silvestris) and detected four loci: ERV
264 The survival of indigenous European wildcat (Felis silvestris silvestris) populations can be locally
266 ed by 16S rRNA gene sequencing as Mycoplasma felis (six cases) and Mycoplasma gateae (one case).
269 e proportions, but oral immunization with H. felis sonicate plus cholera toxin (CT) or with CT alone
272 ession of the H2O2-inducible katA gene in H. felis that colonized Duoxa(-/-) mice, compared with that
274 critical regulator of R. typhi burden in C. felis These data suggest that targeting the IMD pathway
275 The untreated cats remained positive for C. felis throughout the trial; clinical signs were most sev
276 control mice were infected with Helicobacter felis to create a model of Helicobacter pylori infection
278 While bioinformatics analysis of the C. felis transcriptome identified homologs to all of the Dr
285 y cytokine expression, or colonization of H. felis was evaluated in CD73-deficient (CD73-/-) mice or
289 er bacteria (H. pylori, H. hepaticus, and H. felis) was mediated not by TLR4 but rather by TLR2.
290 SvEv (B6129) mice infected with Helicobacter felis, we conducted a study to characterize H. pylori in
294 Fifteen cats infected with Chlamydophila felis were monitored for the presence of C. felis DNA on
295 cifically attributed to FCV, M. felis, or C. felis were seen, although interpretation in this cohort
296 rgastrinemic mice infected with Helicobacter felis were studied at multiple stages of gastric dysplas