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1 Infectious illnesses usually precede Miller Fisher syndrome.
2 nt to the clinical pathophysiology of Miller Fisher syndrome.
3 neuromyotonia, Morvan's syndrome and Miller-Fisher syndrome.
4 0 was isolated from a patient who had Miller-Fisher syndrome.
5 potential to ameliorate the course of Miller Fisher syndrome.
6 ies in the diagnosis and causation of Miller-Fisher syndrome?
7 BS (18 demyelinating, 8 axonal, and 1 Miller Fisher syndrome), 5 had encephalitis (3 with concomitant
9 ater understanding of the spectrum of Miller Fisher syndromes and to refine clinical diagnoses in pat
12 n-Barre syndrome and its variant, the Miller Fisher syndrome, during TNFalpha antagonist therapy.
13 arre syndrome, the motor variant, and Miller Fisher syndrome from controls with high sensitivity and
14 hed motor Guillain-Barre syndrome and Miller Fisher syndrome from controls, with antibodies to glycol
16 f clinical features is reminiscent of Miller Fisher syndrome, in which acute-phase anti-disialylated
19 o facilitates differentiation between Miller-Fisher Syndrome (MFS) and BBE in cases of diagnostic dou
21 s are the serological hallmark of the Miller Fisher syndrome (MFS) variant of the paralytic neuropath
22 triggering of the Guillain-Barre and Miller-Fisher syndrome neuropathies after C. jejuni infection.
23 he Guillain-Barre syndrome, including Miller-Fisher syndrome, subsets of immunoglobulin M paraprotein
24 with rheumatoid arthritis in whom the Miller Fisher syndrome variant of the Guillain-Barre syndrome d
27 f the role of anti-GQ1b antibodies in Miller Fisher syndrome with particular respect to the motor ner
28 persons had Brighton level 1, 2, or 3 GBS or Fisher Syndrome, with symptom onset 1-119 days after vac