ライフサイエンスコーパス: Forkhead transcription factor

1 hosphorylate and inactivate the proapoptotic forkhead transcription factor.

2 gatively regulates the activity of DAF-16, a Forkhead transcription factor.

3 at are efficiently and specifically bound by Forkhead transcription factors.

4 ypertrophic signaling through its effects on Forkhead transcription factors.

5 that involves regulation of the proapoptotic forkhead transcription factors.

6 y gonadotrope cells is the FOXO subfamily of forkhead transcription factors.

7 e binding competition between POU5F1 and the forkhead transcription factors.

8 udies show FGF15 signaling decreases hepatic forkhead transcription factor 1 (FoxO1) activity through

9 Forkhead transcription factor 3 (FOXO3) was identified a

10 Forkhead transcription factor 3 (FoxP3) expression and T

11 ls induced by CpG ODN-activated PDCs express forkhead transcription factor 3 and produce IL-10, TGF-b

12 We suggest that insulin signaling and FOXO (forkhead transcription factor), a downstream molecule in

13 t KM-Brk, blocked the phosphorylation of the forkhead transcription factor, a downstream Akt target.

14 Here, we study the function of the Tc-foxQ2 forkhead transcription factor, a key regulator of the an

15 e to stress by regulating the FOXO family of Forkhead transcription factors, a family of proteins tha

16 Foxo forkhead transcription factors act as downstream targets

17 These two YF mutants fail to block Forkhead transcription factor activity in 293 cells and

18 ctors glycogen synthase kinase-3beta and the Forkhead transcription factors AFX and FKHR.

19 eduction-of-function mutations in the daf-16/forkhead transcription factor, an effector of the DAF-2/

20 al changes in lipid absorption by regulating forkhead transcription factors and MTP and that inhibiti

21 ce of conserved Akt phosphorylation sites in Forkhead transcription factors and PI3K activity.

22 Akt/protein kinase B phosphorylates forkhead transcription factors and prevents their nuclea

23 tion and inhibition of FKHR and related FOXO forkhead transcription factors and that androgens provid

24 FOX:ETS motif, a composite binding site for Forkhead transcription factors and the Ets transcription

25 rates glycogen synthase kinase (GSK)-3 beta, forkhead transcription factor, and BAD.

26 of Sir2 (SirT1) regulates p53, NF-kappaB and Forkhead transcription factors, and is implicated in str

27 g the pro-apoptotic Bc1-2 family member Bad, Forkhead transcription factors, and the cyclic AMP respo

28 Forkhead transcription factors are critical regulators o

29 Forkhead transcription factors are essential for diverse

30 Forkhead transcription factors are evolutionarily conser

31 Thus mammalian forkhead transcription factors are involved in Epo and S

32 From yeasts to humans, forkhead transcription factors are involved in mitotic g

33 Forkhead transcription factors are key participants in d

34 FoxO Forkhead transcription factors are shown here to act as

35 Our work highlights Forkhead transcription factors as hubs that integrate mu

36 Forkhead transcription factors belonging to the FoxO sub

37 ave identified three consensus sequences for forkhead transcription factor binding in transforming gr

38 e Isl1 SHF enhancer contains three consensus Forkhead transcription factor binding sites that are eff

39 Forkhead transcription factor box A2 (FOXA2) is uniquely

40 3 partner molecule, Foxo3a, a subtype of the forkhead transcription factors, by inhibiting its associ

41 ations in Whn (Hfh11, Foxn1), a winged-helix/forkhead transcription factor, cause the nude phenotype,

42 Haploinsufficiency of FOXL2, a new forkhead transcription factor, causes blepharophimosis/p

43 e an evolutionary conserved subfamily of the forkhead transcription factors, characterized by the for

44 The expression of the forkhead transcription factor checkpoint suppressor 1 (C

45 Forkhead transcription factor class O (FOXO)3a, which pl

46 A), independent of p53, but dependent on the forkhead transcription factor class-O family member, FOX

47 fibroblast growth factor signaling and four Forkhead transcription factors consist the central part

48 The NAD-dependent deacetylase SIR2 and the forkhead transcription factor DAF-16 regulate lifespan i

49 in/IGF-1 signaling pathway, dependent on the forkhead transcription factor DAF-16, but independent of

50 ian homologues of the Caenorhabditis elegans forkhead transcription factor DAF-16, function in the in

51 lization and transcriptional activity of the forkhead transcription factor DAF-16.

52 he sir-2.1 gene in a manner dependent on the forkhead transcription factor DAF-16.

53 way negatively regulates the activity of the forkhead transcription factor DAF-16.

54 The Forkhead transcription factor DAF-16/FOXO is the major d

55 diated by intrinsic neuronal activity of the forkhead transcription factor DAF-16/FOXO.

56 dylinositol 3-OH kinase (age-1) regulating a forkhead transcription factor (daf-16) [6, 7], as well a

57 Downregulation of this pathway activates a forkhead transcription factor (daf-16), which may regula

58 The forkhead transcription factor, DAF-16, a downstream targ

59 overexpressing the phosphatase dPTEN or the forkhead transcription factor dFOXO, prevents the declin

60 FoxJ1 is a forkhead transcription factor expressed in multiple tiss

61 Foxp3, an X chromosome-encoded forkhead transcription factor family member, is indispen

62 FOXA1 and FOXA2, members of the forkhead transcription factor family, are critical for e

63 Mei4p, a member of the forkhead transcription factor family, in turn regulates

64 FOXO1, a member of forkhead transcription factor family, was phosphorylated

65 Foxc1 belongs to the forkhead transcription factors family, which plays a cri

66 We find that the forkhead transcription factor FKH-6 promotes male gonada

67 The activity of the forkhead transcription factor FKH-9 in neurons is requir

68 t early origins through its interaction with forkhead transcription factors Fkh1 and Fkh2.

69 Here we show that the yeast Forkhead transcription factors, Fkh1 and Fkh2, are globa

70 d Hi-C experiments on cells depleted for the Forkhead transcription factors, Fkh1 and Fkh2, previousl

71 cycle regulation in a mutant that lacks two forkhead transcription factors, Fkh1 and Fkh2.

72 Here we show that the yeast forkhead transcription factors, Fkh1p and Fkh2p, associa

73 aromyces cerevisiae, Mcm1 interacts with the forkhead transcription factor Fkh2.

74 Akt activation, is a potent inhibitor of the forkhead transcription factor FKHR (FOXO1), identifying

75 Here we demonstrated that forkhead transcription factor FKHR (FOXO1)-induced death

76 gen synthase kinase 3beta (GSK3beta) and the Forkhead transcription factor FKHR were phosphorylated a

77 d PDGF-BB-induced phosphorylation of BAD and forkhead transcription factor FKHR-L1, and these events

78 action between ER alpha and the proapoptotic forkhead transcription factor FKHR.

79 apoptosis by phosphorylating the substrates forkhead transcription factor (FKHR) and glycogen syntha

80 ylation of glycogen synthase kinase (GSK)-3, forkhead transcription factor (FKHR), p70(S6K), and ERK,

81 activity, and prevents the translocation of forkhead transcription factors (FKHR).

82 DAF-16, the closely-related FOXO subclass of forkhead transcription factors (FKHR/FOXO1, FKHRL1/FOXO3

83 K), -mammalian target of rapamycin (-mTOR), -forkhead transcription factor (-FKHR), -caspase-9, and -

84 synthase kinase--3 beta (GSK-3 beta), and a forkhead transcription factor, FKHR, in a human T-cell l

85 Akt exhibits a more inhibitory effect on the forkhead transcription factor, FKHR.

86 eins such as the BCL2 family member Bad, the forkhead transcription factor FKHRL-1, and caspase 9.

87 essed the phosphorylation and degradation of forkhead transcription factor FKHRL1 (FOXO3a), resulting

88 Galpha12QL stimulates the phosphorylation of forkhead transcription factor FKHRL1 via AKT in a PDGFRa

89 arly, induction of FasL by the Akt-regulated forkhead transcription factor FKHRL1 was dependent upon

90 The Forkhead transcription factor FKHRL1 was observed to pro

91 hat Akt-mediated signals, acting through the forkhead transcription factor FKHRL1, can regulate colla

92 rylate and negatively regulate pro-apoptotic forkhead transcription factor FKHRL1.

93 ltant loss of activity of the FOXO family of forkhead transcription factors FKHRL1 and FKHR.

94 or proteins, glycogen synthase kinase-3, and forkhead transcription factors, FKHRL1/FOXO3 and FKHR/FO

95 We used this assay to screen Forkhead transcription factors for control of periodic g

96 revealed an inverse correlation between the Forkhead transcription factor Forkhead box class O (FOXO

97 ia complementation group D2 (FANCD2) and the forkhead transcription factor forkhead box O 3a (FOXO3a)

98 ance was mediated directly by inhibiting the Forkhead transcription factor Forkhead box O3a (FOXO3a).

99 oinsufficiency of the Foxo1 gene, encoding a forkhead transcription factor (forkhead box transcriptio

100 regeneration phenotypes and that RNAi of the forkhead transcription factor FoxA, which is expressed i

101 We have previously shown that a forkhead transcription factor Foxa1 interacts with andro

102 We find that the forkhead transcription factor, FoxA1, is required to gen

103 in normal mice, plasma insulin inhibits the forkhead transcription factor Foxa2 by nuclear exclusion

104 or Tbx1 failed to activate expression of the forkhead transcription factor Foxa2 in the pharyngeal me

105 We found that the forkhead transcription factor Foxa2, acting in progenito

106 sis and defined a CSVD locus adjacent to the forkhead transcription factor FOXC1.

107 ng, nonsense and frameshift mutations in the forkhead transcription factor FOXC2 in affected members

108 Forkhead transcription factor Foxc2 is an essential regu

109 Forkhead transcription factor FOXC2 is an important regu

110 ctional and positional candidate gene, helix/forkhead transcription factor (FOXC2), by sequencing and

111 en reported to be caused by mutations in the forkhead transcription factor, FOXC2.

112 ere dominated by less-known factors, such as forkhead transcription factor FOXD1.

113 ies described in this report reveal that the forkhead transcription factor, Foxd1, is not expressed i

114 Genetic deletion of the forkhead transcription factor, Foxd1, results in strikin

115 nic stem to epiblast cells and uncovered the forkhead transcription factor FOXD3 as a major regulator

116 sing a neural crest-specific deletion of the Forkhead transcription factor Foxd3, we ablated neural c

117 ment of RCs is additionally dependent on the forkhead transcription factor Foxd3, which is more broad

118 Here, we describe mutations in the forkhead transcription factor FOXE3 that predispose muta

119 t fibroblast growth factor signaling and the forkhead transcription factor FoxF directly upregulate t

120 mesodermal precursors expressing or not the Forkhead transcription factor FOXF1.

121 we show that the closely linked mesenchymal forkhead transcription factors Foxf1 and Foxl1 are part

122 vestigated how in intestinal fibroblasts the forkhead transcription factor Foxf2 controls Wnt signali

123 ory that is defined by the expression of the forkhead transcription factor, foxg.

124 We observe that expression of the forkhead transcription factor FoxG1 is dynamically regul

125 Here, the authors identify the forkhead transcription factor Foxh1 as regulating FLK1+

126 We identify the forkhead transcription factor Foxh1, in part through Eom

127 membrane transport proteins regulated by the forkhead transcription factor FOXI1.

128 Here, we show that expression of the forkhead transcription factor FoxJ1 in mice is required

129 which is dependent on the expression of the forkhead transcription factor Foxj1 in mice.

130 The forkhead transcription factor Foxj1 inhibits spontaneous

131 Previous studies suggest that the forkhead transcription factor Foxj1 inhibits spontaneous

132 lling-regulated transcriptome identified the forkhead transcription factor Foxj1.

133 tify a novel interaction between SRF and the Forkhead transcription factor FOXK1 in human cells.

134 Here we show that the Forkhead transcription factors FoxK1 and FoxK2 are also

135 e closely related fasting/starvation-induced forkhead transcription factors FOXK1 and FOXK2 induce ae

136 ns as well as novel E1A targets, such as the forkhead transcription factors, FOXK1/K2.

137 While mutations of the forkhead transcription factor FOXL2 are associated with

138 We find that mouse XX gonads lacking the forkhead transcription factor Foxl2 form meiotic prophas

139 c analysis identified a central role for the forkhead transcription factor FOXM1 and its targets, and

140 Expression of the forkhead transcription factor FoxM1 correlates with prol

141 The forkhead transcription factor FOXM1 has a key role in DN

142 The forkhead transcription factor FoxM1 has been reported to

143 Here we define a critical role for the Forkhead transcription factor FoxM1 in modulating the de

144 (PA) in mice increased the expression of the forkhead transcription factor FoxM1 in type II cells coi

145 The forkhead transcription factor FoxM1 is essential for end

146 nstrate that the expression of the oncogenic forkhead transcription factor FOXM1 is upregulated by GO

147 rongly correlated with the expression of the forkhead transcription factor FoxM1, which binds to the

148 ide chromatin binding mechanisms used by the forkhead transcription factor FOXM1.

149 Increased hepatic expression of the Forkhead transcription factor FoxM1B in adult mice accel

150 the key nodes in the metabolic network, the forkhead transcription factor FOXO has been shown to int

151 Forkhead transcription factor FOXO-1, activated by SIRT-

152 evelopment by regulating the activity of the forkhead transcription factor Foxo.

153 ressed in adult worms, and this requires the forkhead transcription factor (FOXO) encoded by daf-16.

154 Second, we deduced the forkhead transcription factor (FOXO) family to be a down

155 ans extends lifespan by activating DAF-16, a forkhead transcription factor (FOXO) repressed by insuli

156 to do so by converging on the regulation of forkhead transcription factor (FOXO).

157 factor 1 (HIF-1) through the inactivation of Forkhead transcription factors (FOXO) in PTEN-null cells

158 physiological roles in this process are the forkhead transcription factor FOXO1 (also known as FKHR)

159 Forkhead transcription factor FoxO1 also is an important

160 Forkhead transcription factor FoxO1 and the NAD(+)-depen

161 ce, but Akt-dependent phosphorylation of the forkhead transcription factor FoxO1 at serine 256, which

162 The forkhead transcription factor Foxo1 has previously been

163 ng evidence documents a key function for the forkhead transcription factor FoxO1 in cellular metaboli

164 The Forkhead transcription factor FoxO1 inhibits through its

165 The forkhead transcription factor FoxO1 is a critical regula

166 The forkhead transcription factor Foxo1 is regulated by insu

167 We report that haploinsufficiency for the forkhead transcription factor Foxo1 reverses beta cell f

168 The forkhead transcription factor FOXO1, a downstream target

169 determined the molecular mechanisms whereby forkhead transcription factor Foxo1, a key downstream si

170 is associated with reduced expression of the forkhead transcription factor Foxo1, a substrate of the

171 Disinhibition of the forkhead transcription factor FoxO1, increases expressio

172 se AKT and subsequent phosphorylation of the forkhead transcription factor FOXO1.

173 Forkhead transcription factors FOXO1 (FKHR), FOXO3a (FKH

174 The forkhead transcription factor (FoxO1) binds the PDK4 gen

175 an target of rapamycin (mTOR), the family of forkhead transcription factors (FOXO1, FOXO3a, and FOXO4

176 glycogen synthase kinase-3alpha/beta and the forkhead transcription factor, FoxO1, in protection of F

177 rmone deficiencies, we are investigating the forkhead transcription factor, FOXO1, which has been imp

178 Forkhead transcription factor FOXO3 plays a critical rol

179 D31 signals prevents the localization of the forkhead transcription factor FoxO3 to the nucleus, thus

180 demonstrated that Sirt6 can be recruited by forkhead transcription factor FoxO3 to the proximal prom

181 s primordial follicle activation through the forkhead transcription factor Foxo3.

182 In this context, the Forkhead transcription factor, Foxo3, amplified GATA-1-m

183 acetylates and represses the activity of the forkhead transcription factor Foxo3a and other mammalian

184 Here we identify the forkhead transcription factor FOXO3a as a key target of

185 The phosphorylation of forkhead transcription factor FOXO3a by Akt is critical

186 Nuclear exclusion of the forkhead transcription factor FOXO3a by protein kinase A

187 The role of the Forkhead transcription factor FOXO3a in processes that p

188 Here we examined the role of the forkhead transcription factor FOXO3a, a downstream targe

189 e the first linkage by the identification of Forkhead transcription factor FOXO3a, the PI3K/Akt downs

190 n of the SIRT1 deacetylase and activates the Forkhead transcription factor Foxo3a.

191 we show that such inflammation requires the forkhead transcription factor Foxo3a: Foxo3a-deficient m

192 vation of a ubiquitously expressed mammalian forkhead transcription factor, Foxo3a, induces cerebral

193 In addition, the forkhead transcription factor, FoxO3a, interacts with ea

194 The mammalian forkhead transcription factors, FOXO3a (FKHRL1), FOXO1a

195 the in vitro interactions among 14-3-3, the Forkhead transcription factor FOXO4, and its target DNA,

196 The forkhead transcription factor FoxO6 is prominently expre

197 Forkhead transcription factors (FOXOs) alter a diverse a

198 pproaches were used to evaluate the roles of forkhead transcription factors (FoxOs) in endoplasmic re

199 Down-regulation of the forkhead transcription factor Foxp1 by integrin engageme

200 We found that the forkhead transcription factor Foxp1 is crucial for maint

201 Expression of the forkhead transcription factor FOXP1 is essential for ear

202 The forkhead transcription factor FOXP1 is involved in B-cel

203 strate that absence of the gene encoding the forkhead transcription factor Foxp1 resulted in a profou

204 Heterozygous disruptions of the Forkhead transcription factor FoxP2 impair acquisition o

205 The forkhead transcription factors Foxp2 and Foxp1 are expre

206 Here, we demonstrate that two Forkhead transcription factors, Foxp2 and Foxp4, are pro

207 The forkhead transcription factor Foxp3 (forkhead box P3) is

208 Here, we present data arguing that the forkhead transcription factor Foxp3 acts as the regulato

209 Expression of forkhead transcription factor Foxp3 defines a distinct l

210 The forkhead transcription factor FOXP3 is necessary for ind

211 Here we report that the forkhead transcription factor Foxp3 is specifically expr

212 alogous cooperative complex of NFAT with the forkhead transcription factor FOXP3, a lineage specifica

213 lls, both mRNA and protein expression of the forkhead transcription factor Foxp3, a specific marker o

214 , they efficiently induced expression of the forkhead transcription factor FoxP3, the master regulato

215 al circumstances, male mice deficient in the forkhead transcription factor FoxP3, which lack regulato

216 and function with particular emphasis on the forkhead transcription factor Foxp3.

217 and function are critically dependent on the forkhead transcription factor Foxp3.

218 The forkhead transcription factor, FoxP3, is a key molecule

219 The forkhead transcription factor, Foxp3, is thought to act

220 ntified stomach-restricted expression of the forkhead transcription factor FOXQ1.

221 nd -gamma), members of the FoxA subfamily of forkhead transcription factors, function as initial chro

222 esenchymal markers, including members of the forkhead transcription factor gene family, have been det

223 The recently described forkhead transcription factor gene Foxe3 was shown to be

224 Mutations in the forkhead transcription-factor gene (FOXC1), have been sh

225 The forkhead transcription factor genes Foxc1 (formerly Mf1

226 ed members of the Fox family of winged-helix/forkhead transcription factor genes.

227 Forkhead transcription factors have been found to play a

228 The forkhead transcription factor hepatocyte nuclear factor

229 FOXP2 is a forkhead transcription factor implicated in developmenta

230 J1, which encodes a well-known member of the forkhead transcription factors important for ciliogenesi

231 onstrating a critical role for at least this forkhead transcription factor in the regulation of B lym

232 he phosphorylation and hence inactivation of forkhead transcription factors in a p66Shc-dependent man

233 There are numerous forkhead transcription factors in mammalian cells but we

234 he Forkhead box, class O (FOXO) subfamily of Forkhead transcription factors in the regulation of BIM

235 t Isl1 is a direct transcriptional target of Forkhead transcription factors in the SHF and establish

236 f genetic programs induced by a subfamily of forkhead transcription factors including AFX.

237 FOXO3a, a member of the family of Forkhead transcription factors, interacted with the prom

238 as a primary downstream target for Foxn4, a forkhead transcription factor involved in the genesis of

239 We show here that the Foxn4 winged helix/forkhead transcription factor is coexpressed with the bH

240 Here, we show that the Foxn4 winged helix/forkhead transcription factor is expressed in a subset o

241 Finally, a putative binding element for forkhead transcription factors is necessary for promoter

242 Forkhead box gene, group O (FoxO) family of Forkhead transcription factors is phopsphorylated and in

243 DAF-16, a forkhead transcription factor, is a key regulator of lon

244 Sep1, another forkhead transcription factor, is an activator for a sma

245 Foxn4, a forkhead transcription factor, is expressed in the commo

246 malian FOXO3, a member of the FOXO family of Forkhead transcription factors known to promote resistan

247 osphorylation of FOXO3a, a tumor suppressive forkhead transcription factor, leading to its cytoplasmi

248 ted a physical interaction of SMA-3 with the forkhead transcription factor LIN-31, which is enhanced

249 nase promoter and that increased activity of forkhead transcription factors may underlie the increase

250 iogenesis, transcriptionally suppresses AFX1 forkhead transcription factor-mediated apoptosis and inc

251 e Xenopus and zebrafish homologs of Foxj1, a forkhead transcription factor necessary for ciliogenesis

252 A homeodomain and a forkhead transcription factor, NKX2.1 and HNF-3, respect

253 Recent studies have indicated that forkhead transcription factor O1 (Foxo1) and silent info

254 atic nuclear receptor 4alpha (HNF4alpha) and forkhead transcription factor O1 (FOXO1), two critical t

255 ating both AKT and MAPK signaling to inhibit forkhead transcription factor O3a (FOXO3a) function and

256 lycogen synthase kinase-3beta (GSK3beta) and forkhead transcription factor of the O class (FOXO)3A, k

257 Forkhead transcription factors of the forkhead box gene,

258 Forkhead transcription factors of the FoxO subfamily are

259 Forkhead transcription factors of the O class (FOXOs) ar

260 Forkhead transcription factors often function in concert

261 es through the inhibitory phosphorylation of forkhead transcription factors, our results describe for

262 Despite elevated expression of forkhead transcription factor P3 (foxP3), they exerted o

263 sure and was mediated through an AKT-1/PDK-1/forkhead transcription factor pathway overlapping with b

264 2 is a let-7 target in seam cells, while the forkhead transcription factor pha-4 is a target in the i

265 Forkhead transcription factors play critical roles in th

266 Forkhead transcription factors play crucial and diverse

267 Forkhead transcription factors play key roles in the reg

268 The FoxO family of Forkhead transcription factors plays an important role i

269 FoxD4/5, a forkhead transcription factor, plays a critical role in

270 escribe a mechanistic model whereby Foxd3, a forkhead transcription factor, prevents neural crest-der

271 mitotic genes in close proximity to Fkh2, a forkhead transcription factor previously implicated in r

272 Forkhead transcription factors regulate many aspects of

273 The FOXO family of Forkhead transcription factors, regulated by the phospho

274 Moreover, transcripts dependent on the forkhead transcription factor Sep1, which are expressed

275 e antioxidant defenses and inhibition of the forkhead transcription factor signaling pathways.

276 In addition to inactivation of forkhead transcription factor signaling through enhanced

277 erior domain by activating expression of the forkhead transcription factors sloppy paired 1 and slopp

278 Akt phosphorylates Forkhead transcription factors such as FKHRL1, leading t

279 f FOXO4 (also known as AFX), a member of the forkhead transcription factor superfamily that is negati

280 ential display PCR, we have cloned an 85-kDa forkhead transcription factor (termed Mac-1-regulated fo

281 FoxD4L1 is a forkhead transcription factor that expands the neural ec

282 Foxa is a forkhead transcription factor that is expressed in the e

283 Foxe3 encodes a winged helix-forkhead transcription factor that is initially expresse

284 the nuclear accumulation of DAF-16/FOXO, the forkhead transcription factor that is the major downstre

285 ic protein consisting of MLL fused to AFX, a forkhead transcription factor that normally regulates ge

286 FOXO3a is a forkhead transcription factor that regulates a multitude

287 Klotho protein activates the FoxO forkhead transcription factors that are negatively regul

288 AFX belongs to a subfamily of Forkhead transcription factors that are phosphorylated b

289 ar location of FoxO1 and FoxO3a, two Class O Forkhead transcription factors that mediate lymphocyte q

290 FOXC1 and FOXC2 are forkhead transcription factors that play essential roles

291 cyte nuclear factors-3 (Foxa-1-3) are winged forkhead transcription factors that regulate gene expres

292 homologue (CHES-1-like), two genes encoding forkhead transcription factors that we discovered utiliz

293 DAF-16/forkhead transcription factor, the downstream target of

294 on and represses the expression of FOXP2, a forkhead transcription factor tightly associated with sp

295 Our results link Forkhead transcription factors to a previously unexplore

296 Akt; induces phosphorylation of the FKHRL-1 Forkhead transcription factor; upregulates a series of i

297 Furthermore, Foxa2, a forkhead transcription factor, was induced by active Wnt

298 FKHR is a member of the FOXO subfamily of Forkhead transcription factors, which are important targ

299 n by up-regulating the expression of Class-O Forkhead transcription factors, which play essential rol

300 t of insulin signaling is the FoxO family of Forkhead transcription factors, which translocate from t