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1 0) in cutaneous and pulmonary infection with Francisella tularensis.
2 by infection by the gram-negative bacterium, Francisella tularensis.
3 infections using Rift Valley fever virus and Francisella tularensis.
4 ress and virulence by the bacterial pathogen Francisella tularensis.
5 ent of tularemia and category A select agent Francisella tularensis.
6 a category A intracellular mucosal pathogen, Francisella tularensis.
7 el organism for the study of highly virulent Francisella tularensis.
8 ellular pathogens Listeria monocytogenes and Francisella tularensis.
9 he highly infectious intracellular bacterium Francisella tularensis.
10 ss caused by the category A biodefense agent Francisella tularensis.
11 the highly infectious and zoonotic pathogen Francisella tularensis.
12 (EHEC and UPEC), Salmonella typhimurium, and Francisella tularensis.
13 mulation would protect the host from inhaled Francisella tularensis.
14 arance of the lethal intracellular bacterium Francisella tularensis.
15 TLR2-activating human intracellular pathogen Francisella tularensis.
16 cated in the O-antigen biosynthetic locus of Francisella tularensis.
17 is caused by the category A biodefense agent Francisella tularensis.
18 on of T helper 1 (Th1) cell immunity against Francisella tularensis.
19 in the protective innate immune response to Francisella tularensis.
20 in Bacillus anthracis, Yersinia pestis, and Francisella tularensis.
21 r the intramacrophage growth and survival of Francisella tularensis.
22 llenge with the live vaccine strain (LVS) of Francisella tularensis.
23 _1680/FTT_0166c as a new virulence factor in Francisella tularensis.
24 ed by a Gram-negative coccoid rod bacterium, Francisella tularensis.
25 been reported with Dichelobacter nodosus and Francisella tularensis.
26 uivalent but highly divergent sequences from Francisella tularensis.
27 on by using the live vaccine strain (LVS) of Francisella tularensis.
28 from individuals infected with the bacteria Francisella tularensis, a category A biodefense pathogen
29 th the Gram-negative intracellular bacterium Francisella tularensis, a category A biological threat a
30 rticularly good antibacterial effect against Francisella tularensis, a Category A biowarfare pathogen
33 e attenuated vaccine is needed for combating Francisella tularensis, a highly infectious bacterial pa
34 macrophages the intracellular life cycle of Francisella tularensis, a highly infectious bacterium th
35 of genes that contribute to the virulence of Francisella tularensis, a highly infectious pathogen and
40 chanisms by which virulent type A strains of Francisella tularensis accomplish this evasion are not u
41 ion of the lipid A 1-phosphatase, LpxE, from Francisella tularensis allowed us to construct recombina
42 However, a comparative genome analysis of Francisella tularensis allowed us to predict the existen
47 aureus (MRSA) and priority pathogens such as Francisella tularensis and Burkholderia pseudomallei.
49 cted in membranes of Helicobacter pylori and Francisella tularensis and may be responsible for the re
50 and Prevention Category A bioterrorism agent Francisella tularensis and prototype of a superfamily of
53 agilis, Bacillus anthracis, Yersinia pestis, Francisella tularensis, and Brucella abortus), the last
54 spp., Toxoplasma gondii, Coxiella burnetii, Francisella tularensis, and Neospora caninum, estimate c
56 dentified the two major lipid A species from Francisella tularensis as asymmetric tetraacylated struc
57 hances murine susceptibility to infection by Francisella tularensis as indicated by accelerated morta
58 lian pathogens such as Coxiella burnetii and Francisella tularensis, as well as Coxiella-like and Fra
60 r nodosus and FtPilE from the human pathogen Francisella tularensis at 2.3 and 1 A resolution, respec
61 ae, Pseudomonas aeruginosa, Yersinia pestis, Francisella tularensis, Bacillus anthracis and Vibrio ch
64 O-antigen of the lipopolysaccharide (LPS) of Francisella tularensis bacteria, a Tier 1 Select Agent o
66 racis, Yersinia pestis, Burkholderia mallei, Francisella tularensis, Brucella abortus, and ricin.
67 ned for Bacillus anthracis, Yersinia pestis, Francisella tularensis, Brucella melitensis, Clostridium
68 anisms (Bacillus anthracis, Yersinia pestis, Francisella tularensis, Brucella spp., Burkholderia spp.
69 ains of Bacillus anthracis, Yersinia pestis, Francisella tularensis, Burkholderia mallei, Burkholderi
70 m four pathogenic bacteria: Yersinia pestis, Francisella tularensis, Burkholderia pseudomallei and Ac
71 protection against infection with attenuated Francisella tularensis, but their role in infection medi
72 Schu S4 strain of the intracellular pathogen Francisella tularensis by host macrophages involves CR3/
73 urface exposed and required for virulence of Francisella tularensis by subverting the host innate imm
77 egative, facultative intracellular bacterium Francisella tularensis causes acute, lethal pneumonic di
85 R recruitment, we evaluated Escherichia coli-Francisella tularensis chimeric variants of tmRNA and Sm
88 pectrum efficacy against Bacillus anthracis; Francisella tularensis; Coxiella burnetii; and Ebola, Ma
93 chanisms by which the intracellular pathogen Francisella tularensis evades innate immunity are not we
94 VS and Schu S4 strains of the human pathogen Francisella tularensis express a siderophore when grown
95 (VOCs) emitted in culture by bacterial taxa Francisella tularensis (F. tularensis) subspecies novici
96 es of the Gram-negative facultative anaerobe Francisella tularensis: F. tularensis subsp. tularensis
98 se appears to contribute to the virulence of Francisella tularensis following pulmonary infection.
102 The macrophage proinflammatory response to Francisella tularensis (Ft) live vaccine strain (LVS) wa
107 cid phosphatase from the category A pathogen Francisella tularensis (FtHAP) has been implicated in in
108 ymbionts (FLEs) with significant homology to Francisella tularensis (gamma-proteobacteria) have been
111 accinations with the intracellular bacterium Francisella tularensis have been studied using the live
113 onstrated that targeting fixed (inactivated) Francisella tularensis (iFT) organisms to FcR in mice i.
114 w-derived macrophages (BMDMs) to inactivated Francisella tularensis (iFt)-containing immune complexes
116 thetic knee joint infections (PJI) caused by Francisella tularensis in Europe (one in Switzerland and
118 ion of the lipid A 1-phosphatase, LpxE, from Francisella tularensis in Y. pestis yields predominantly
121 used by the Gram-negative bacterial pathogen Francisella tularensis Infection of macrophages and thei
123 ocked Th17 cell generation in the lung after Francisella tularensis infection, and inhibited the incr
176 A fundamental step in the life cycle of Francisella tularensis is bacterial entry into host cell
181 the highly infectious intracellular pathogen Francisella tularensis is directly related with the abil
196 The highly infectious and zoonotic pathogen Francisella tularensis is the etiologic agent of tularem
200 any other gram-negative bacteria, the LPS of Francisella tularensis isolated from in vitro cultures i
201 tularemia, a zoonose caused by the bacterium Francisella tularensis, largely refer to Parinaud's ocul
202 .) and intradermal (i.d.) inoculation of the Francisella tularensis live vaccine strain (Ft-LVS).
203 IKKs in myeloid cells in vivo in response to Francisella tularensis Live Vaccine Strain (Ft. LVS) inf
204 subset in the lungs of mice during pulmonary Francisella tularensis live vaccine strain (LVS) infecti
209 ammation, priming with glycolipid (FtL) from Francisella tularensis live vaccine strain induces splen
210 hown that priming with glycolipid (FtL) from Francisella tularensis live-vaccine strain (i) induces F
211 and public health pathogens-Influenza virus, Francisella tularensis, Mycobacterium tuberculosis, Micr
212 eumonic tularemia is caused by inhalation of Francisella tularensis, one of the most infectious micro
216 s, successful protective immune responses to Francisella tularensis require rapid and efficient induc
217 to various assemblies of the loblolly pine, Francisella tularensis, rice and budgerigar genomes.
220 study, we describe novel inhibitors against Francisella tularensis SchuS4 FabI identified from struc
225 gions of two slightly more distantly related Francisella tularensis strains were also compared agains
226 shown to be associated with highly virulent Francisella tularensis strains, including Schu S4, while
228 nd illustrate its use on three datasets from Francisella tularensis, Streptococcus pyogenes, and Esch
230 ble to the live attenuated vaccine strain of Francisella tularensis subsp. holarctica in their abilit
231 -flight (MALDI-TOF) mass spectrometry on the Francisella tularensis subsp. holarctica LVS defined thr
232 eral highly pathogenic subspecies, including Francisella tularensis subsp. holarctica, whose distribu
238 species, including the opportunistic microbe Francisella tularensis subsp. novicida, there are consid
240 b O-PS gene cluster from the highly virulent Francisella tularensis subsp. tularensis (type A) strain
241 Comparative genome hybridization of the Francisella tularensis subsp. tularensis and F. tularens
242 n Francisella philomiragia ATCC 25015 and on Francisella tularensis subsp. tularensis CCUG 2112, the
247 Clinical isolates of highly virulent type A Francisella tularensis subsp. tularensis organisms were
248 n of the virulent prototypical type A strain Francisella tularensis subsp. tularensis Schu S4 affects
249 Inactivation of both copies of iglE rendered Francisella tularensis subsp. tularensis Schu S4 avirule
251 scribe the isolation and characterization of Francisella tularensis subsp. tularensis strain Schu S4
252 dings concerning the four subspecies and two Francisella tularensis subsp. tularensis subpopulations
254 AIM2 inflammasome during infection with the Francisella tularensis subspecies novicida (F. novicida)
256 onal exposure to lethal doses of aerosolized Francisella tularensis subspecies tularensis, strain SCH
262 the virulent SCHU S4 strain of the bacterium Francisella tularensis, that infects alveolar macrophage
273 istinct lack of activation of these cells by Francisella tularensis, the causative agent of tularemia
277 ased, whole-genome resequencing platform for Francisella tularensis, the causative agent of tularemia
280 he current study, we developed a model using Francisella tularensis, the causative agent of tularemia
288 including virulent strains of the bacterium Francisella tularensis, to enable colonization and infec
289 ogens, such as the highly virulent bacterium Francisella tularensis, to ensure their replication and
291 ts ortholog FTT_0166c in the highly virulent Francisella tularensis type A strain SchuS4 are required
292 elates of vaccine-induced protection against Francisella tularensis using murine splenocytes and furt
294 l for inflammasome activation in response to Francisella tularensis, vaccinia virus and mouse cytomeg
296 of animals as early as 4 hrs post-exposure, Francisella tularensis was associated with an almost com
299 ctions with the live vaccine strain (LVS) of Francisella tularensis within the mouse liver are the fo
300 nd Prevention (CDC) and include the bacteria Francisella tularensis, Yersinia pestis, Burkholderia ma