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1 l ulcers, particularly among those caused by Fusarium.
2 se(6), displayed enhanced resistance against Fusarium.
3 t has not always paralleled the evolution of Fusarium.
4 an important new tool for genetic studies of Fusarium.
6 = 14) was isolated in highest number whereas Fusarium (13.4%, N = 6) was the most common fungus speci
7 library alone (Aspergillus, 93% versus 69%; Fusarium, 84% versus 42%; and Scedosporium, 94% versus 1
10 lapse and black roots, symptoms similar to a Fusarium-Alternaria disease complex, recently characteri
13 NAT enzymes in crop-compromising species of Fusarium and Aspergillus, identifying three groups of ho
14 t perspectives of what constitutes the genus Fusarium and how many species occur within the genus.
15 unctional divergence of two beta-tubulins in Fusarium and identified type II variations in FgTub2 res
16 Seed-coated M6 swarms towards root-invading Fusarium and is associated with the growth of root hairs
18 olutionary antagonistic interactions between Fusarium and Streptomyces are driven by resource competi
19 found evidence for local adaptation between Fusarium and Streptomyces populations as indicated by si
20 cteria, decreasing the relative abundance of Fusarium, and increasing the fungal network complexity t
22 sporium, Alternaria, Penicillium, Ulocadium, Fusarium, Arthrinium, Epicoccum, Aureobasidium, Curvular
25 amacril also inhibits the ATPase activity of Fusarium avenaceum myosin-1 but has little or no inhibit
26 naturally occurring hemibiotrophic pathogen, Fusarium brachygibbosum Among all AGOs, only transcripts
27 ir influence on two native fungal pathogens, Fusarium brachygibbosum U4 and Alternaria sp. U10, and t
28 tracellular pH promotes infectious growth of Fusarium by stimulating phosphorylation of a conserved m
33 Zn(II) ions, using a mini-column packed with Fusarium-coated multi-walled carbon nanotubes and induct
35 arum s.s. (37%), Fusarium meridionale (46%), Fusarium cortaderiae (13%), and Fusarium austroamericanu
37 found to mediate systemic resistance against Fusarium crown rot and to simultaneously improve tomato
40 led to a significant decrease (p < 0.01) in Fusarium-damaged rachis rate, Fusarium-diseased kernel r
42 xins in wheat three standardized approaches (Fusarium disease severity, PCR assays for Fusarium spp.
43 (p < 0.01) in Fusarium-damaged rachis rate, Fusarium-diseased kernel rate and DON content in harvest
49 ndary metabolites produced by members of the Fusarium fujikuroi species complex (FFSC) and rare strai
52 d by the presence of mycotoxins derived from Fusarium fungus, and, in particular, by deoxynivalenol (
54 tion, redundancy analysis indicated that the Fusarium genera were significantly related to the diseas
58 ha-L-fucosidase from plant pathogenic fungus Fusarium graminearum (FgFCO1) actively releases fucose f
60 Trichoderma atroviride, the plant pathogens Fusarium graminearum and Colletotrichum graminicola, the
61 (1)CFU g(-1) and the species most found were Fusarium graminearum and Fusarium verticillioides with 2
62 the plasma membrane of the ascomycete fungi Fusarium graminearum and Neurospora crassa and induces a
67 recovered via the heterologous expression of Fusarium graminearum GSK3 homolog gene FGK3, whose codin
68 ecific pollination and during infection with Fusarium graminearum In both Arabidopsis species, the la
77 nthesis of mycotoxin deoxynivalenol (DON) in Fusarium graminearum is regulated by two pathway-specifi
78 reticulum (ER) of the phytopathogenic fungus Fusarium graminearum is reorganized both in vitro and in
79 We also verified that the VdAtf1 ortholog in Fusarium graminearum mediates nitrogen metabolism, sugge
83 he primary inoculum in the wheat scab fungus Fusarium graminearum that was recently shown to have sex
87 in the regulation of sterol biosynthesis in Fusarium graminearum, a fungal pathogen of cereal crops
88 d blight (FHB) is a cereal disease caused by Fusarium graminearum, a fungus able to produce type B tr
89 r events associated with host recognition in Fusarium graminearum, an economically important fungal p
90 c fungi B. cinerea, Alternaria brassicicola, Fusarium graminearum, and Sclerotinia sclerotiorum and y
92 we characterized the only RasGEF protein in Fusarium graminearum, FgCdc25, by combining genetic, cyt
94 head blight (FHB), which is mainly caused by Fusarium graminearum, is a destructive wheat disease tha
96 enotype CI9831, followed by inoculation with Fusarium graminearum, led to the down regulation of key
97 pathogen and phenamacril-susceptible species Fusarium graminearum, phenamacril-resistant Fusarium spe
98 ly characterized the FgMCM1 MADS-box gene in Fusarium graminearum, the causal agent of wheat and barl
99 se gene important for sexual reproduction in Fusarium graminearum, we found that two tandem stop codo
100 nase Cel5A, from the plant-pathogenic fungus Fusarium graminearum, whose production was increased by
116 d the efficacy of clove oil nanoemulsions on Fusarium growth and mycotoxin during malting process.
117 ould be valuable efficient targets to reduce Fusarium growth and therefore to prevent food contaminat
118 reat diversity of fungal species (Cordyceps, Fusarium, Harpochytrium, Emericellopsis, Volutella, Clad
119 Fusarium graminearum is a causal agent of Fusarium head blight (FHB) and a deoxynivalenol (DON) pr
120 by barley resistant genotype CI89831 against Fusarium head blight (FHB) based on metabolo-transcripto
121 by barley resistant genotype CI89831 against Fusarium head blight (FHB) based on metabolo-transcripto
134 leaf volatile Z-3-HAC protects wheat against Fusarium head blight by priming for enhanced JA-dependen
137 sarium graminearum and related species cause Fusarium head blight on cultivated grasses, such as whea
138 ium graminearum causes the important disease Fusarium head blight on various species of cereals, lead
140 cide residues in wheat grains susceptible to fusarium head blight treated with fungicides, and to eva
146 transferase (NAT) and has been identified in Fusarium infecting cereal plants as responsible for deto
147 0.01) in WFhb1-1 accumulation, 12 hours post Fusarium inoculation in non-Qfhb1-carrier wheat but not
150 tivity of a fungus from the ubiquitous genus Fusarium is related to the presence of nanometre-scale p
152 n elongation factor (TEF)-1alpha analysis of Fusarium isolates included FSSC 1-a, FOSC 33, and FDSC E
155 d not find a benefit for all corneal ulcers, Fusarium keratitis may benefit from the addition of oral
156 006 outbreak of contact lens (CL)-associated Fusarium keratitis, there may have been a rise in CL-ass
158 ee of the FSSC species (Fusarium falciforme, Fusarium keratoplasticum, and Fusarium sp. FSSC 12) acco
159 ex were isolated: F. graminearum s.s. (37%), Fusarium meridionale (46%), Fusarium cortaderiae (13%),
160 escribe 2 patients with post-CAR-T-cell IMI (Fusarium, Mucorales) and review the published literature
161 cribe two patients with post-CAR-T-cell IMI (Fusarium, Mucorales) and review the published literature
164 espectively, but concentrations of the three Fusarium mycotoxins assessed were ~10 times lower than t
166 s study was concerned with the fate of these Fusarium mycotoxins within malting, brewing, milling and
167 urance systems are effective for maintaining Fusarium mycotoxins, but not OTA concentrations, below t
172 f the most widespread phytopathogenic fungi, Fusarium oxysporum and Botrytis cinerea, were chosen to
173 ainst Rhizopus stolonifer, Botrytis cinerea, Fusarium oxysporum and Colletotrichum gloeosporioides.
174 Formae speciales (ff.spp.) of the fungus Fusarium oxysporum are often polyphyletic within the spe
176 genotypes with respect to resistance against Fusarium oxysporum f. sp. ciceri Race1 (Foc1), we demons
177 n the FSA biosynthetic gene (FUB) cluster in Fusarium oxysporum f. sp. cubense tropical race 4 (Foc T
178 omologue of the yeast SNARE protein Vam7p in Fusarium oxysporum f. sp. lycopersici (Fol), a fungal pa
179 mosomes within strains pathogenic to tomato (Fusarium oxysporum f. sp. lycopersici) and pea (Fusarium
180 of a set of strains of the melon wilt fungus Fusarium oxysporum f. sp. melonis (Fom), bioinformatics-
183 was horizontally transferred from the fungus Fusarium oxysporum f. sp. vasinfectum to V. dahliae and
184 yses suggested that G-LSR2 was acquired from Fusarium oxysporum f. vasinfectum through horizontal gen
187 of a set of strains of the melon wilt fungus Fusarium oxysporum f.sp. melonis (Fom), bioinformatics-b
190 eromone from the plant pathogenic ascomycete Fusarium oxysporum revealed the presence of a central be
191 When infecting a host plant, the fungus Fusarium oxysporum secretes several effector proteins in
193 e Fusarium solani species complex (FSSC) and Fusarium oxysporum species complex (FOSC), the most comm
194 and one each within the FIESC (1-a) and the Fusarium oxysporum species complex (ST-33) were widespre
195 Here, we show that the root-infecting fungus Fusarium oxysporum uses a functional homologue of the pl
196 (Botrytis cinerea, Pseudomonas syringae, and Fusarium oxysporum) were used to demonstrate potential t
197 capsulatum, Coccidioides immitis/posadasii, Fusarium oxysporum, Aspergillus spp., and Bipolaris spp.
198 nse can overgrow another hypocrealean fungus Fusarium oxysporum, cause sporadic cell death and arrest
199 t Aspergillus flavus, Aspergillus ochraceus, Fusarium oxysporum, Saccharomyces cerevisiae and Candida
204 larity to the closely related partitiviruses Fusarium poae virus 1 and Penicillium stoloniferum virus
205 reciprocal inhibition among Streptomyces and Fusarium populations from prairie soil, and explore anti
207 predominantly caused by the fungal pathogen Fusarium pseudograminearum, is a disease of economic sig
209 ization of an orphan gene (Triticum aestivum Fusarium Resistance Orphan Gene [TaFROG]) as a component
210 portant clade of veterinary relevance within Fusarium Six of the multilocus STs within the FSSC (3+4-
212 polytic activity of three microbial lipases: Fusarium solani cutinase, Rv0183, and LipY from Mycobact
214 reported Taxol-producing endophytic fungus, Fusarium solani from the standpoint of spores as seed in
216 o inhibitory effect on the motor activity of Fusarium solani myosin-1, human myosin-1c, and D. discoi
217 st (named as SDFsC) by expressing the enzyme Fusarium solani pisi cutinase (FsC) on the cell surface
218 e sources of human pathogenic strains in the Fusarium solani species complex (FSSC) and Fusarium oxys
219 lates (47/67 = 70.1%) were nested within the Fusarium solani species complex (FSSC), and these includ
222 ota showed a predominance of Alternaria sp., Fusarium sp. and Epicoccum sp. Microdochium nivale (23%)
223 e inhibitor of class I myosin in susceptible Fusarium sp. Chemerin is a leukocyte attractant, adipoki
224 ; however, strains of F. keratoplasticum and Fusarium sp. FSSC 12 were mostly (25/27) isolated from m
225 um falciforme, Fusarium keratoplasticum, and Fusarium sp. FSSC 12) accounted for four-fifths of the v
226 , for the first time, the filamentous fungus Fusarium sp. was utilized for devising a novel method fo
227 ipants, 72 (30.4%) were culture positive for Fusarium species (41 [56.9%] male and 31 [43.1%] female;
228 tification of Aspergillus, Scedosporium, and Fusarium species (n = 28) by matrix-assisted laser desor
229 [5], Aspergillus species [2], Mucorales [2], Fusarium species [2], and Candida glabrata [1]) occurred
230 ubgroup analyses looking at ulcers caused by Fusarium species and adjusting for baseline best spectac
231 mptomatic native grasses for the presence of Fusarium species and confirmed infected grasses as hosts
232 sses for the presence of mycotoxin-producing Fusarium species and evaluated the ability of these fung
233 id (FSA) is a phytotoxin produced by several Fusarium species and has been associated with plant dise
235 d for patients with Aspergillus or other non-Fusarium species as the causative organism (1.5 points [
237 ferase (GST) and confers broad resistance to Fusarium species by detoxifying trichothecenes through d
238 exerts its antifungal effect on susceptible Fusarium species by inhibiting the ATPase activity of th
243 sm subgroups, there was some suggestion that Fusarium species might have a decreased rate of perforat
245 ead blight (FHB), a fungal disease caused by Fusarium species that produce food toxins, currently dev
247 Fusarium graminearum, phenamacril-resistant Fusarium species, and the mycetozoan model organism Dict
248 h American grasses are commonly inhabited by Fusarium species, but appear to accommodate these toxige
249 hows highly inhibitory activity towards some Fusarium species, but not to other fungi, indicating tha
257 t frequent microbe (10/17; 59%), followed by Fusarium spp. (4/17; 24%), Pseudomonas aeruginosa (2/17;
259 s (Fusarium disease severity, PCR assays for Fusarium spp. identification and mycotoxin quantificatio
261 ypes: order Mucorales, Aspergillus spp., and Fusarium spp.) significantly prolonged the time to wound
263 neoformans and C. gattii, 98.6% and 100% for Fusarium spp., and 96.2% and 99.9% for Rhodotorula spp.,
264 versity and evolutionary relationships of 67 Fusarium strains from veterinary sources, most of which
265 n-19-ol against Fusarium verticillioides and Fusarium subglutinans may indicate pathogen defensive fu
267 An approach to defining and identifying Fusarium that places the needs of the community of users
268 evidence that horizontal gene transfer from Fusarium to Vd991 contributed significantly to its adapt
271 otoxin method was developed for 12 different Fusarium toxins including modified mycotoxins in beer (d
274 assay was developed and validated for three Fusarium toxins, deoxynivalenol (DON), zearalenone (ZEA)
278 ngal species from the genera Aspergillus and Fusarium using solid-state voltammetry is described.
282 19-ol and syn-pimara-7,15-dien-19-ol against Fusarium verticillioides and Fusarium subglutinans may i
286 ies most found were Fusarium graminearum and Fusarium verticillioides with 26% and 12% of incidence,
291 ophthora sojae and the fungal plant pathogen Fusarium virguliforme that are pathogenic to soybean (Gl
292 dying growth, germination and sporulation in Fusarium virguliforme that causes sudden death syndrome
296 t induces resistance response in chickpea to Fusarium wilt disease by modulating the transcription of
299 ful to select melon cultivars to avoid melon Fusarium wilt, but also to monitor how quickly a Fom pop