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1 cid), salt (NaCl) and wilt causing pathogen (Fusarium oxysporum).
2 i species complex (FFSC) and rare strains of Fusarium oxysporum.
3 to the root-infecting vascular wilt pathogen Fusarium oxysporum.
4 is and virulence in the vascular wilt fungus Fusarium oxysporum.
5 against several filamentous fungi, including Fusarium oxysporum.
6 ain molecule secreted by the vascular fungus Fusarium oxysporum.
7 t members of the family Sclerotiniaceae, and Fusarium oxysporum.
8 or resistance to the important root pathogen Fusarium oxysporum.
9 tants, as shown for Pseudomonas syringae and Fusarium oxysporum.
10 ated significant antifungal activity against Fusarium oxysporum.
12 across plant species, confers resistance to Fusarium oxysporum, a devastating soil-borne fungal path
13 haracterized a PR-1-like protein, Fpr1, from Fusarium oxysporum, an ubiquitous fungal pathogen that c
14 f the most widespread phytopathogenic fungi, Fusarium oxysporum and Botrytis cinerea, were chosen to
15 ainst Rhizopus stolonifer, Botrytis cinerea, Fusarium oxysporum and Colletotrichum gloeosporioides.
18 also exhibited excellent antifungal (against Fusarium oxysporum) and antibacterial (versus Staphyloco
19 Formae speciales (ff.spp.) of the fungus Fusarium oxysporum are often polyphyletic within the spe
20 capsulatum, Coccidioides immitis/posadasii, Fusarium oxysporum, Aspergillus spp., and Bipolaris spp.
21 required for host-specific pathogenicity in Fusarium oxysporum can be acquired through horizontal ch
23 flavoprotein nitroalkane oxidase (NAO) from Fusarium oxysporum catalyzes the oxidation of nitroalkan
24 The flavoprotein nitroalkane oxidase from Fusarium oxysporum catalyzes the oxidation of nitroalkan
26 flavoprotein nitroalkane oxidase (NAO) from Fusarium oxysporum catalyzes the oxidation of primary an
27 The fungal pathogens Fusarium solani and Fusarium oxysporum cause severe corneal disease in the U
28 nse can overgrow another hypocrealean fungus Fusarium oxysporum, cause sporadic cell death and arrest
44 genotypes with respect to resistance against Fusarium oxysporum f. sp. ciceri Race1 (Foc1), we demons
45 n the FSA biosynthetic gene (FUB) cluster in Fusarium oxysporum f. sp. cubense tropical race 4 (Foc T
46 nama disease of banana, caused by the fungus Fusarium oxysporum f. sp. cubense, is a serious constrai
47 omologue of the yeast SNARE protein Vam7p in Fusarium oxysporum f. sp. lycopersici (Fol), a fungal pa
49 mosomes within strains pathogenic to tomato (Fusarium oxysporum f. sp. lycopersici) and pea (Fusarium
52 of a set of strains of the melon wilt fungus Fusarium oxysporum f. sp. melonis (Fom), bioinformatics-
56 was horizontally transferred from the fungus Fusarium oxysporum f. sp. vasinfectum to V. dahliae and
57 yses suggested that G-LSR2 was acquired from Fusarium oxysporum f. vasinfectum through horizontal gen
59 ight generations of cucumber inoculated with Fusarium oxysporum f.sp. cucumerinum in a split-root sys
60 ase-resistance pathway, the response against Fusarium oxysporum f.sp. lycopersici race 2, mediated by
62 of a set of strains of the melon wilt fungus Fusarium oxysporum f.sp. melonis (Fom), bioinformatics-b
64 ed with ARD, as well as endophytes including Fusarium oxysporum, F. solani, Nectria ramulariae, Ilyon
67 Ecotype Taynuilt-0 (Ty-0) is susceptible to Fusarium oxysporum forma specialis (f.) matthioli wherea
68 the infiltrative and degradative ability of Fusarium oxysporum fungal strain on polyethylene terepht
69 ces dermatitidis, Cladophialophora bantiana, Fusarium oxysporum, Fusarium solani, Histoplasma capsula
70 avus, Aspergillus fumigatus, Bipolaris spp., Fusarium oxysporum, Fusarium solani, Pseudallescheria bo
71 orium prolificans, Scedosporium apiospermum, Fusarium oxysporum/Fusarium solani, Rhizopus arrhizus, R
73 s- and ethylene-inducing peptide (Nep1) from Fusarium oxysporum inhibited both root and cotyledon gro
74 usarium wilt caused by the ascomycete fungus Fusarium oxysporum is a devastating disease of many econ
78 ronment, and for soilborne pathogens such as Fusarium oxysporum, microbial competition in the rhizosp
82 eromone from the plant pathogenic ascomycete Fusarium oxysporum revealed the presence of a central be
83 nd Col-0 revealed six dominant RESISTANCE TO FUSARIUM OXYSPORUM (RFO) loci that significantly contrib
84 ofaciens) and fungi (Phytophthora cinnamomi, Fusarium oxysporum, Rhizoctonia solani, and Nectria hema
85 t Aspergillus flavus, Aspergillus ochraceus, Fusarium oxysporum, Saccharomyces cerevisiae and Candida
86 s terreus, Rhizopus oryzae, Fusarium solani, Fusarium oxysporum, Scedosporium prolificans, and Scedos
88 ins (Staphyloccucs aureus, Escherichia coli, Fusarium oxysporum), showing their efficacy as an antimi
89 several fungi, including Pythium irregulare, Fusarium oxysporum solani, Alternaria solani, Trichoderm
91 e Fusarium solani species complex (FSSC) and Fusarium oxysporum species complex (FOSC), the most comm
92 and one each within the FIESC (1-a) and the Fusarium oxysporum species complex (ST-33) were widespre
94 ssaging of the cross-kingdom fungal pathogen Fusarium oxysporum through tomato plants or axenic media
95 ive fungal system such as the plant pathogen Fusarium oxysporum, to control hyphal chemotropism and r
96 Here, we show that the root-infecting fungus Fusarium oxysporum uses a functional homologue of the pl
98 ngal potency of PP-AgNPs (150 ug/mL) against Fusarium oxysporum was found to be 80.9% (Colony diamete
99 (Botrytis cinerea, Pseudomonas syringae, and Fusarium oxysporum) were used to demonstrate potential t
100 ting the pathogenesis of the fungal pathogen Fusarium oxysporum, which has multiple genes homologous