ライフサイエンスコーパス: Fusobacterium

1 l lineages, such as Aquifex, Thermotoga, and Fusobacterium.

2 s, Roseburia, and Subdoligranulum but higher Fusobacterium.

3 ies, e.g., Selenomonas noxia, F. alocis, and Fusobacterium.

4 s (39), Campylobacter (8), Clostridium (96), Fusobacterium (12), Leptotrichia (8), Mobiluncus (8), Pe

5 were B. forsythus (59%), spirochetes (54%), Fusobacterium (41%), P. micros (39%), and P. gingivalis

6 ed organisms for AP were B. forsythus (83%), Fusobacterium (80%), spirochetes (79%), P. gingivalis (5

7 ponding data for RP were B. forsythus (85%), Fusobacterium (83%), P. gingivalis (60%), spirochetes (5

8 c regression was used to compare tertiles of Fusobacterium abundance.

9 n between local cytokine gene expression and Fusobacterium abundance.

10 Various taxa increased, including Fusobacterium, Actinomyces, and Corynebacterium, while o

11 In this study, a novel adhesin, FadA (Fusobacterium adhesin A), was demonstrated to bind to th

12 s, B. forsythus, and Capnocytophaga species, Fusobacterium alocis, Actinomyces odontolyticus, Actinom

13 We also confirmed previous reports on Fusobacterium and CRC in 10 CRC tumor tissues and 9 matc

14 33, p = 0.06 while it was 0.44, p = 0.01 for Fusobacterium and IL-10.

15 adenocarcinomas were found to retain viable Fusobacterium and its associated microbiome through succ

16 olonization of human colorectal cancers with Fusobacterium and its associated microbiome-including Ba

17 Two main additional taxa, Fusobacterium and Parvimonas, correlated with the clinic

18 by blocking or mutating the adhesin RadD on Fusobacterium and removal of flagella on C difficile.

19 tococcus, Eikenella, and Leptotrichia, while Fusobacterium and Veillonella dominated the 5% DMAHDM-20

20 An overabundance of Porphyromonas, Fusobacterium, and Bacteroides taxa was also identified

21 Firmicutes, Proteobacteria, Actinobacteria, Fusobacterium, and Bacteroidetes, with the first 3 phyla

22 reviously (Prevotella, Sneathia, Aerococcus, Fusobacterium, and Gemella) as well as several novel tax

23 mica, Streptococcus, Prevotella nanceiensis, Fusobacterium, and Janthinobacterium lividum, in the nas

24 abundance of Bacteroides, Faecalibacterium, Fusobacterium, and Lachnospiraceae was noted in fecal sa

25 ed in members of the Haemophilus, Neisseria, Fusobacterium, and Porphyromonas species and the Sphingo

26 on of lung commensals, including Prevotella, Fusobacterium, and Porphyromonas with gut Candida.

27 be enriched on HNSCC mucosa, Capnocytophaga, Fusobacterium, and Porphyromonas, were at low levels in

28 s, Clostridium, Desulfovibrio, Enterococcus, Fusobacterium, and several new genera described in this

29 s as a potential treatment for patients with Fusobacterium-associated colorectal cancer.

30 elial cells (HOECs) and designated it FAD-I (Fusobacterium-associated defensin inducer).

31 taxa of lower abundance including the genera Fusobacterium, Atopobium, Gluconacetobacter, Hydrogenoph

32 Common anaerobes included fusobacterium, bacteroides, porphyromonas, and prevotell

33 s associated with lower Faecalibacterium and Fusobacterium but higher Bacteroides.

34 emonstrated greater abundance of Parvimonas, Fusobacterium, Campylobacter, Bacteroides, and Treponema

35 The prevalences of Fusobacterium, Campylobacter, Prevotella, Capnocytophaga

36 eriodontal pathogens belonging to the genera Fusobacterium, Cardiobacterium, Synergistes, and Selenom

37 Dysbiosis, abundances of Fusobacterium, Cl. hiranonis and fecal concentrations of

38 Lastly, we use the model to co-culture 28 Fusobacterium clinical isolates and demonstrate that the

39 ra including the Bacteroides fragilis group, Fusobacterium, Clostridium, and Peptostreptococcus.

40 ell Host & Microbe provide insights into the Fusobacterium-CRC relationship.

41 < 0.05), including Prevotella, Mitsuokella, Fusobacterium, Desulfovibrio, and bacteria belonging to

42 Bacteriodes and Fusobacterium dominated the microbiome and were positive

43 In decreasing order of prevalence, Fusobacterium, enteric rods, Prevotella intermedia/Prevo

44 We found that Fusobacterium (F.) nucleatum was abundant in colorectal

45 r Subdoligranulum but higher Escherichia and Fusobacterium (false discovery rate-adjusted P values <0

46 ytophaga, Cytophaga, Dialister, Eubacterium, Fusobacterium, Gemella, Mogibacterium, Peptostreptococcu

47 ion period, dog exposure was associated with Fusobacterium genera enrichment, as well as enrichment o

48 ese clusters are driven by Porphyromonas and Fusobacterium genera in chimpanzees and Haemophilus and

49 is absent in the nonoral species, including Fusobacterium gonidiaformans, Fusobacterium mortiferum,

50 did not achieve remission had enrichment of Fusobacterium gonidiaformans, Sutterella wadsworthensis,

51 icle proposes additional strategies to block Fusobacterium-host interactions, as well as treatment of

52 fied an abundance of bacteria from the genus Fusobacterium in appendixes from patients with AA.

53 ent studies have implicated overabundance of Fusobacterium in association with colorectal adenomas an

54 ults support a link between the abundance of Fusobacterium in colonic mucosa and adenomas and suggest

55 nt studies have reported a high abundance of Fusobacterium in CRC subjects compared to normal subject

56 Most notably, the relative abundance of Fusobacterium increased whereas Streptococcus decreased

57 In situ hybridization analysis revealed that Fusobacterium is predominantly associated with cancer ce

58 ft with the antibiotic metronidazole reduced Fusobacterium load, cancer cell proliferation, and overa

59 from the oral cavity and stomach, including Fusobacterium, Megasphaera, Campylobacter, Capnocytophag

60 11-year-old boy taking oral antibiotics for Fusobacterium meningitis diagnosed 3 months earlier pres

61 11-year-old boy taking oral antibiotics for Fusobacterium meningitis diagnosed 3 months earlier pres

62 The Fusobacterium mortiferum malH gene, encoding 6-phospho-a

63 tibody to maltose 6-phosphate hydrolase from Fusobacterium mortiferum, and the two proteins exhibit a

64 ies, including Fusobacterium gonidiaformans, Fusobacterium mortiferum, Fusobacterium naviforme, Fusob

65 ibody against phospho-alpha-glucosidase from Fusobacterium mortiferum.

66 osidase, EC 3.2.1.86) has been purified from Fusobacterium mortiferum.

67 um gonidiaformans, Fusobacterium mortiferum, Fusobacterium naviforme, Fusobacterium russii, and Fusob

68 uggest that in patients aged 15 to 30 years, Fusobacterium necrophorum causes at least 10% of cases o

69 young adults (persons aged 15 to 24 years), Fusobacterium necrophorum causes endemic pharyngitis at

70 After Fusobacterium necrophorum grew in blood cultures, anaero

71 Fusobacterium necrophorum is a gram-negative, rod-shaped

72 Fusobacterium necrophorum was detected in 20.5% of patie

73 a patient with Lemierre's syndrome caused by Fusobacterium necrophorum who developed a right frontal

74 The most commonly implicated pathogen is Fusobacterium necrophorum, a gram-negative anaerobe that

75 Fusobacterium necrophorum, a gram-negative, rod-shaped,

76 pparently highly specialised for feet, while Fusobacterium necrophorum, a secondary pathogen in footr

77 Fusobacterium necrophorum, an obligate anaerobic bacteri

78 Fusobacterium necrophorum-positive pharyngitis occurs mo

79 sociated bacteria, Dichelobacter nodosus and Fusobacterium necrophorum.

80 For Fusobacterium nucleatum (100%/100%), there was no differ

81 7.4%), Porphyromonas gingivalis (15.1%), and Fusobacterium nucleatum (14.2%).

82 Further specificity was seen when Fusobacterium nucleatum (a middle colonizer), Aggregatib

83 Effects of Fusobacterium nucleatum (F. nucleatum) biofilm on epithe

84 pic dento-epithelial (OD-E) model exposed to Fusobacterium nucleatum (Fn) biofilm.

85 Fusobacterium nucleatum (Fn) has been associated with co

86 Fusobacterium nucleatum (Fn) is one of the most frequent

87 Fusobacterium nucleatum (Fn), a bacterium present in the

88 Fusobacterium nucleatum (Fn), a commensal in the human o

89 ction was used for detecting and quantifying Fusobacterium nucleatum (Fn), Aggregatibacter actinomyce

90 Aggregatibacter actinomycetemcomitans (Aa), Fusobacterium nucleatum (Fn), and Prevotella intermedia

91 ivalis (Pg), Eubacterium saburreum (Es), and Fusobacterium nucleatum (Fn).

92 ermedia (Pi), Tannerella forsythia (Tf), and Fusobacterium nucleatum (Fn).

93 factor in coaggregated mixed infection with Fusobacterium nucleatum (Fn).

94 (Sg)/S. oralis (So)/S. sanguinis (Ss) and Sg/Fusobacterium nucleatum (Fn)/Porphyromonas gingivalis (P

95 l as with Veillonella sp. (early colonizer), Fusobacterium nucleatum (middle colonizer), and Aggregat

96 Endothelial cells were infected with Fusobacterium nucleatum (strain 25586) for periods of 4,

97 alis (Pg); 4) group G-PgFn: oral gavage with Fusobacterium nucleatum + Pg; 5) group I-Pg: heat-killed

98 monas gingivalis, Prevotella intermedia, and Fusobacterium nucleatum activated both TLRs, but TLR4 pl

99 eration sequencing implicated coinfection of Fusobacterium nucleatum and Actinomyces israelii, resolv

100 also significantly associated with pathogens Fusobacterium nucleatum and Aggregatibacter actinomycete

101 A higher abundance of Fusobacterium nucleatum and an increased LPS pathway wer

102 contribute to carcinogenesis, in particular, Fusobacterium nucleatum and Porphyromonas gingivalis, ba

103 mixed infection with the periodontopathogens Fusobacterium nucleatum and Porphyromonas gingivalis.

104 The most frequently found bacteria were Fusobacterium nucleatum and Prevotella denticola.

105 , Tannerella forsythia, Treponema denticola, Fusobacterium nucleatum and Prevotella intermedia) in sa

106 acterial pathogens Enterococcus faecalis and Fusobacterium nucleatum and remains catalytically active

107 ycetemcomitans, Streptococcus parasanguinis, Fusobacterium nucleatum and several species belonging to

108 he opportunistic pathogens Escherichia coli, Fusobacterium nucleatum and Streptococcus agalactiae to

109 omonas gingivalis and its consortium members Fusobacterium nucleatum and Streptococcus gordonii confi

110 Fusobacterium nucleatum appears to play a role in colore

111 Candida albicans and Fusobacterium nucleatum are well-studied oral commensal

112 these findings by identifying the bacterium Fusobacterium nucleatum as a previously unrecognized che

113 gregatibacter actinomycetemcomitans JP2, and Fusobacterium nucleatum ATCC 10953 were unable to grow a

114 s biofilm (Streptococcus sanguinis DSM20068, Fusobacterium nucleatum ATCC10953, and Porphyromonas gin

115 Attachment of Fusobacterium nucleatum ATCC25586 and Prevotella interme

116 h extraction followed by oral infection with Fusobacterium nucleatum caused BONJ-like lesions and del

117 vivo experiments, the combination of Pam and Fusobacterium nucleatum caused the death of gingival fib

118 ous studies showed that hBD-2 was induced by Fusobacterium nucleatum cell wall extract without the in

119 Gnotobiotic studies revealed that while Fusobacterium nucleatum clinical isolates with FadA and

120 Obligate anaerobes other than Fusobacterium nucleatum coaggregated only poorly with ox

121 ve enhanced proliferation in the presence of Fusobacterium nucleatum compared to HPV+ cells, suggesti

122 Co-culture studies of HNSCC cell lines with Fusobacterium nucleatum demonstrated that HPV-negative c

123 for the presence and amount of EBV, CMV, and Fusobacterium nucleatum DNA using real-time polymerase c

124 F together with either Escherichia coli DNA, Fusobacterium nucleatum DNA, or Porphyromonas gingivalis

125 eviously demonstrated that sonic extracts of Fusobacterium nucleatum FDC 364 were capable of inhibiti

126 employed by the Gram-negative oral pathogen Fusobacterium nucleatum for cell death induction of huma

127 s were significantly higher for P. micra and Fusobacterium nucleatum for the screw-retained group.

128 omonas gingivalis, Tannerella forsythia, and Fusobacterium nucleatum from subgingival biofilm were de

129 t substrate, full-length Vibrio cholerae and Fusobacterium nucleatum glycine riboswitch aptamers with

130 nfection was community acquired in 48 (84%); Fusobacterium nucleatum group and/or Streptococcus inter

131 ontal pathogens Porphyromonas gingivalis and Fusobacterium nucleatum growth and attachment to human g

132 In previous studies Fusobacterium nucleatum has been shown to induce either

133 Fusobacterium nucleatum has long been found to cause opp

134 report a case of Lemierre's syndrome due to Fusobacterium nucleatum in a previously healthy 19-year-

135 2019) observe detrimental overpopulation of Fusobacterium nucleatum in mice and patients, suppressin

136 infection with Porphyromonas gingivalis and Fusobacterium nucleatum in mice.

137 inoculation of Porphyromonas gingivalis and Fusobacterium nucleatum in young (4 to 5 mo) and aged (1

138 /+) mouse model of intestinal tumorigenesis, Fusobacterium nucleatum increases tumor multiplicity and

139 ed replication plan of key experiments from 'Fusobacterium nucleatum infection is prevalent in human

140 thelial cells reached its peak 2 h following Fusobacterium nucleatum infection whereas it rapidly dec

141 Fusobacterium nucleatum is a common oral anaerobe involv

142 Fusobacterium nucleatum is a Gram-negative anaerobe asso

143 Fusobacterium nucleatum is a gram-negative anaerobe that

144 Fusobacterium nucleatum is a gram-negative anaerobe ubiq

145 Fusobacterium nucleatum is a Gram-negative oral anaerobe

146 Fusobacterium nucleatum is a gram-negative oral anaerobe

147 Fusobacterium nucleatum is among the most prevalent bact

148 Fusobacterium nucleatum is an opportunistic oral pathoge

149 Fusobacterium nucleatum is an oral bacterium associated

150 Fusobacterium nucleatum is an oral commensal bacterium t

151 Fusobacterium nucleatum is an oral pathogen that is link

152 Fusobacterium nucleatum is associated with colorectal ca

153 Fusobacterium nucleatum is capable of binding to and inv

154 Fusobacterium nucleatum is implicated in accelerating co

155 ristic of the suspected periodontal pathogen Fusobacterium nucleatum is its ability to adhere to a pl

156 The oral bacterium Fusobacterium nucleatum is often found in colorectal can

157 Fusobacterium nucleatum is the most commonly observed sp

158 agonize TLR4-specific activation by agonist, Fusobacterium nucleatum LPS.

159 Fusobacterium nucleatum might be the cause or consequenc

160 ly significant, Porphyromonas gingivalis and Fusobacterium nucleatum occur in higher concentrations m

161 thogens such as Porphyromonas gingivalis and Fusobacterium nucleatum produce five different short-cha

162 ere we present the crystal structures of the Fusobacterium nucleatum riboswitch bound to FMN, ribofla

163 Chemical mapping on the Fusobacterium nucleatum riboswitch with N-methylisatoic

164 e draft genome sequence and its analysis for Fusobacterium nucleatum sub spp. vincentii (FNV), and co

165 Fusobacterium nucleatum subsp. polymorphum was the most

166 ing infection with Porphyromonas gingivalis, Fusobacterium nucleatum subspecies (ssp) nucleatum, ssp

167 rometry (MALDI-TOF MS) for identification of Fusobacterium nucleatum subspecies.

168 eponema denticola, Tannerella forsythia, and Fusobacterium nucleatum to colonize the periodontium and

169 omonas gingivalis, Tannerella forsythia, and Fusobacterium nucleatum using real time polymerase chain

170 givalis, whereas phagocytosis of heat-killed Fusobacterium nucleatum was augmented compared with that

171 es naeslundii, Porphyromonas gingivalis, and Fusobacterium nucleatum was grown on sandblasted and aci

172 Fusobacterium nucleatum was present in 17 patients prior

173 Fusobacterium nucleatum was prevalent in the subgingival

174 omonas gingivalis, Tannerella forsythia, and Fusobacterium nucleatum were analyzed for prediction of

175 , Aggregatibacter actinomycetemcomitans, and Fusobacterium nucleatum were assessed in anaerobic condi

176 Three native plasmids of Fusobacterium nucleatum were characterized, including DN

177 votella intermedia, Eikenella corrodens, and Fusobacterium nucleatum were determined by real-time pol

178 treptococcus mitis, Veillonella parvula, and Fusobacterium nucleatum) and the same biofilm plus the p

179 i, Actinobacillus actinomycetemcomitans, and Fusobacterium nucleatum) biofilm formation under anaerob

180 inations that resulted in tailing endpoints (Fusobacterium nucleatum, 86% agreement) or in cases of l

181 be colonized by potential pathogens such as Fusobacterium nucleatum, a bacterium linked with intraut

182 production of CCL20 and hBDs in response to Fusobacterium nucleatum, a commensal bacterium of the or

183 Using Fusobacterium nucleatum, a Gram-negative anaerobe freque

184 entified a cell wall-associated protein from Fusobacterium nucleatum, a Gram-negative bacterium of th

185 t evidence for the immunosuppressive role of Fusobacterium nucleatum, a gram-negative oral bacterium

186 We previously reported that Fusobacterium nucleatum, a ubiquitous gram-negative bact

187 Key quorum-sensing plaque bacteria, such as Fusobacterium nucleatum, act as bridging species between

188 microbial species (Porphyromonas gingivalis, Fusobacterium nucleatum, Actinomyces naeslundii, Tannere

189 helial cell response to the common bacterium Fusobacterium nucleatum, an important bridging species t

190 It can be coisolated with Fusobacterium nucleatum, an opportunistic bacterial path

191 Fusobacterium nucleatum, an opportunistic pathogen, is t

192 D-2 mRNA was induced by cell wall extract of Fusobacterium nucleatum, an oral commensal microorganism

193 ophilus aphrophilus, Actinomyces naeslundii, Fusobacterium nucleatum, and A. actinomycetemcomitans, a

194 as Prevotella intermedia, Selenomonas noxia, Fusobacterium nucleatum, and Actinobacillus actinomycete

195 hyromonas gingivalis, Prevotella intermedia, Fusobacterium nucleatum, and Aggregatibacter actinomycet

196 lls stimulated with a periodontal bacterium, Fusobacterium nucleatum, and other stimulants.

197 inae, Mobiluncus mulieris, Prevotella bivia, Fusobacterium nucleatum, and Peptoniphilus species.

198 tella intermedia, Streptococcus intermedius, Fusobacterium nucleatum, and Peptostreptococcus micros,

199 ates were Prevotella sp., Porphyromonas sp., Fusobacterium nucleatum, and Peptostreptococcus sp.

200 eptococcus mutans, Porphyromonas gingivalis, Fusobacterium nucleatum, and Pseudomonas aeruginosa.

201 Of those, Bergeyella, Fusobacterium nucleatum, and Sneathia sanguinegens had n

202 Aggregatibacter aphrophilus, Fusobacterium nucleatum, and Streptococcus intermedius o

203 pecies biofilms of Porphyromonas gingivalis, Fusobacterium nucleatum, and Streptococcus oralis were f

204 gar Candida medium, coaggregation assay with Fusobacterium nucleatum, and sugar assimilation profiles

205 hyromonas gingivalis, Prevotella intermedia, Fusobacterium nucleatum, and Tannerella forsythia.

206 Prevotella intermedia, Tannerella forsythia, Fusobacterium nucleatum, and total bacteria count.

207 pathogens, such as Porphyromonas gingivalis, Fusobacterium nucleatum, and Treponema denticola, are am

208 Streptococcus mutans, Enterococcus faecalis, Fusobacterium nucleatum, and Veillonella dispar was used

209 Bacteria, such as Fusobacterium nucleatum, are present in the tumor microe

210 cus anginosus, Porphyromonas gingivalis, and Fusobacterium nucleatum, as well as Campylobacter rectus

211 hyromonas gingivalis, Prevotella intermedia, Fusobacterium nucleatum, Campylobacter rectus, and Trepo

212 hyromonas gingivalis, Prevotella intermedia, Fusobacterium nucleatum, Campylobacter rectus, Eikenella

213 dance was positively correlated with that of Fusobacterium nucleatum, consistent with hypothesized pr

214 omonas gingivalis, Campylobacter rectus, and Fusobacterium nucleatum, could cause localized bone reso

215 Fusobacterium nucleatum, Eikenella corrodens, Actinobaci

216 s as potentiators of tumorigenesis-including Fusobacterium nucleatum, enterotoxigenic Bacteroides fra

217 wall extracts of Porphyromonas gingivalis or Fusobacterium nucleatum, Escherichia coli lipopolysaccha

218 tella intermedia, Peptostreptococcus micros, Fusobacterium nucleatum, Fusobacterium polymorphum, Eike

219 monas gingivalis, Prevotella intermedia, and Fusobacterium nucleatum, have recently been shown to sec

220 dentify an anaerobic Gram-negative bacillus, Fusobacterium nucleatum, in a patient with "culture-nega

221 otella intermedia, Campylobacter rectus, and Fusobacterium nucleatum, in subgingival dental plaque of

222 and difficult-to-cultivate species, such as Fusobacterium nucleatum, Leptotrichia (Sneathia) spp., a

223 Fusobacterium nucleatum, long known as a common oral mic

224 ella forsythia [previously T. forsythensis], Fusobacterium nucleatum, Parvimonas micra [previously Pe

225 elevated in AgP in comparison with CP, while Fusobacterium nucleatum, Parvimonas micra, and Campyloba

226 pathogens, including Prevotella intermedia, Fusobacterium nucleatum, Peptostreptococcus micros, and

227 ection protocol using Prevotella intermedia, Fusobacterium nucleatum, Peptostreptococcus micros, and

228 their ability to coaggregate with strains of Fusobacterium nucleatum, Peptostreptococcus micros, Pept

229 , Campylobacter curvus, Eikenella corrodens, Fusobacterium nucleatum, Porphyromonas gingivalis, and P

230 nisms (Actinobacillus actinomycetemcomitans, Fusobacterium nucleatum, Porphyromonas gingivalis, Pepto

231 ctinomycetemcomitans), Campylobacter rectus, Fusobacterium nucleatum, Porphyromonas gingivalis, Prevo

232 ntified virulence mechanisms of oral species Fusobacterium nucleatum, Porphyromonas gingivalis, Strep

233 imens yielded pathogenic bacteria, including Fusobacterium nucleatum, Prevotella heparinolytica, Prev

234 phyromonas gingivalis, Tannerella forsythia, Fusobacterium nucleatum, Prevotella intermedia, and Camp

235 three orange-complex periodontal pathogens (Fusobacterium nucleatum, Prevotella intermedia, and Camp

236 phyromonas gingivalis, Tannerella forsythia, Fusobacterium nucleatum, Prevotella intermedia, and tota

237 same double-labeling techniques to identify Fusobacterium nucleatum, Prevotella intermedia, oral Cam

238 anaerobic pathogens, Prevotella intermedia, Fusobacterium nucleatum, Streptococcus intermedius, and

239 nfections (endodontic pathogens [EP]), i.e., Fusobacterium nucleatum, Streptococcus intermedius, Parv

240 e in corncob formation between S. crista and Fusobacterium nucleatum, this property was examined.

241 ms enumerated were Porphyromonas gingivalis, Fusobacterium nucleatum, Veillonella sp., and total anae

242 the bacterium Helicobacter pylori, and later Fusobacterium nucleatum, were implicated in the developm

243 nd humans increases the amount of intestinal Fusobacterium nucleatum, which releases SFCAs.

244 lla, Salmonella, Haemophilus influenzae, and Fusobacterium nucleatum, which share structural and func

245 xed infection with the otherwise stimulatory Fusobacterium nucleatum.

246 activation by another periodontal bacterium, Fusobacterium nucleatum.

247 bserved for Bacteroides thetaiotaomicron and Fusobacterium nucleatum.

248 llowing stimulation of epithelial cells with Fusobacterium nucleatum.

249 Actinomyces israelii with the coisolation of Fusobacterium nucleatum.

250 adA adhesin from the Gram-negative bacterium Fusobacterium nucleatum.

251 olymicrobial oral infections with or without Fusobacterium nucleatum.

252 cus gordonii and the opportunistic commensal Fusobacterium nucleatum.

253 symporter (NSS) family has been cloned from Fusobacterium nucleatum.

254 olic analysis of the dominant oral bacterium Fusobacterium nucleatum.

255 ggregation with the anaerobic oral bacterium Fusobacterium nucleatum.

256 mediated adherence to a peridontal pathogen, Fusobacterium nucleatum.

257 ella parvula, Peptostreptococcus micros, and Fusobacterium nucleatum.

258 , Aggregatibacter actinomycetemcomitans, and Fusobacterium nucleatum.

259 inomycetemcomitans, Eikenella corrodens, and Fusobacterium nucleatum/periodonticum were statistically

260 ecrophorum, and F. varium are the species of Fusobacterium observed in AA samples.

261 The mean log abundance of Fusobacterium or cytokine gene expression between cases

262 It is highly conserved among F. nucleatum, Fusobacterium periodonticum, and Fusobacterium simiae, t

263 CC 25586, gene FN1704) that we hereby rename Fusobacterium phospholipase autotransporter (FplA).

264 reptococcus micros, Fusobacterium nucleatum, Fusobacterium polymorphum, Eikenella corrodens, Eubacter

265 isease associated anaerobes, several genera (Fusobacterium, Prevotella, Tanenerella, Treponema) remai

266 identified from the 39 defects, belonging to Fusobacterium, Pseudomonas, Streptococcus, Filifactor, a

267 cterium mortiferum, Fusobacterium naviforme, Fusobacterium russii, and Fusobacterium ulcerans.

268 Fusobacterium sequences were enriched in carcinomas, con

269 nucleatum, Fusobacterium periodonticum, and Fusobacterium simiae, the three most closely related ora

270 vimonas micra, Peptostreptococcus sp. OT113, Fusobacterium sp. OT203, Pseudoramibacter alactolyticus,

271 E. corrodens, P. micros, Capnocytophaga and Fusobacterium sp., enteric Gram-negative rods, Enterococ

272 inomycetemcomitans (1.1% of total isolates), FusobActerium species (7.9%), Campylobacter species (2.2

273 eroPack system was most efficient in growing Fusobacterium species (P = 0.0001).

274 monas gingivalis, Bacteroides forsythus, and Fusobacterium species as well as beta-hemolytic streptoc

275 ects had a significantly higher abundance of Fusobacterium species compared to controls (p = 0.01).

276 We assessed the abundance of Fusobacterium species in the normal rectal mucosa of sub

277 Eubacterium species, Campylobacter species, Fusobacterium species, and Peptostreptococcus micros.

278 Furthermore, due to its uniqueness to oral Fusobacterium species, fadA may be used as a marker to d

279 thogens, including Porphyromonas gingivalis, Fusobacterium species, Peptostreptococcus micros, Bacter

280 profile with substantial cocolonization with Fusobacterium species.

281 difficile infection harbored high levels of Fusobacterium species.

282 ly encountered anaerobes were Prevotella and Fusobacterium species.

283 taxonomic reassignment of each as a distinct Fusobacterium species.

284 72 isolates), Prevotella spp. (71 isolates), Fusobacterium spp. (21 isolates), Porphyromonas spp. (20

285 cteroides spp. (30%), Clostridium (11%), and Fusobacterium spp. (8%).

286 ostridium tertium (3.6 %; 13.9 [1.96-99.4]), Fusobacterium spp. (excluding F. necrophorum) (3.0 %; 8.

287 Metagenomic analyses indicate that symbiotic Fusobacterium spp. are associated with human colorectal

288 We find that Fusobacterium spp. are enriched in human colonic adenoma

289 ., Cardiobacterium sp., Eikenella corrodens, Fusobacterium spp., Gemella haemoylsans, and Neisseria s

290 e pathogens of noma, such as spirochetes and Fusobacterium spp., were detected in at least one subjec

291 Biochemical analysis of multiple Fusobacterium strains revealed that FplA is expressed as

292 represented 19 novel species of Prevotella, Fusobacterium, Streptococcus, Actinomyces, Capnocytophag

293 pically dominated by the genera Veillonella, Fusobacterium, Streptococcus, Eikenella, and Leptotrichi

294 r GI tract (Gemella, Veillonella, Neisseria, Fusobacterium, Streptococcus, Prevotella, Pseudomonas, a

295 m that in humans, with some bacteria such as Fusobacterium switching roles between the two species (a

296 te the function of the folate stress-sensing Fusobacterium ulcerans ZTP riboswitch, we apply a single

297 cterium naviforme, Fusobacterium russii, and Fusobacterium ulcerans.

298 hort and found that the overall abundance of Fusobacterium was 415 times greater in CRC versus adjace

299 ses, the correlation for local TNF-alpha and Fusobacterium was r = 0.33, p = 0.06 while it was 0.44,

300 est tertile, subjects with high abundance of Fusobacterium were significantly more likely to have ade