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1 e present at very low levels in cells at the G0 phase.
2 f normal HBE cells, which accumulated in the G0 phase.
3 cess, required for both cell-cycle arrest in G0 phase and high-level expression of late muscle-differ
4 lock the self-renewal, cell cycle entry into G0 phase and invasiveness of CSCs, and improve the sensi
5       However, fermenting cells arrest in G1/G0 phase and suffer massive vacuolization and eventual c
6 eated with 1.0 microM tamoxifen underwent G1-G0-phase arrest but did not undergo apoptosis.
7 populating cells were predominant within the G0 phase, because transplantation of CD34(+) cells resid
8 r investigations revealed an increase in the G0 phase cell population depicting that majority of cell
9  study, the effect of caffeine in quiescent (G0 phase) cells was investigated.
10 able to leave the division cycle and enter a G0 phase; cells past this point are unaffected by a shor
11 A replication in S phase cytosol, but not in G0 phase cytosol.
12 ed an incomplete arrest in the cell cycle at G0 phase, increased phagocytic ability, and enhanced exp
13 nparenchymal cells from the normal quiescent G0 phase into the cell cycle during liver regeneration a
14 dy on Ku70 deletion, DNA-PKcs deletion in G1/G0-phase mouse progenitor B cell lines, significantly im
15 Bone marrow (BM) CD34+ cells residing in the G0 phase of cell cycle may be the most suited candidates
16 ngle human marrow CD34+CD38-/lo cells in the G0 phase of cell cycle were cultured under 7 different c
17 les, whereas the fraction of cells in the G1/G0 phase of the CDC increases monotonically from 90 to 9
18  show that Bcl11b induces cells to enter the G0 phase of the cell cycle and become quiescent.
19                  Human dermal fibroblasts in G0 phase of the cell cycle and Chinese hamster ovary cel
20  each instance, most of the HSCs were in the G0 phase of the cell cycle and exhibited reduced oxidati
21                    Cells arrest in the G1 or G0 phase of the cell cycle in response to a variety of n
22 t-RA) arrests the growth of HBE cells in the G0 phase of the cell cycle through activation of retinoi
23 ntiation to cells that have entered into the G0 phase of the cell cycle under mitogen withdrawal.
24 RNAi) as a major requirement for quiescence (G0 phase of the cell cycle) in Schizosaccharomyces pombe
25                              They are in the G0 phase of the cell cycle, and their transcriptome is t
26 ppropriate co-stimulation triggers exit from G0 phase of the cell cycle, increased size and metabolis
27 arge number of the Flt3low cells were in the G0 phase of the cell cycle, whereas Flt3high cells were
28  for the initial steps of ciliogenesis in G1/G0 phase of the cell cycle.
29 f AML cells, but not of normal cells, in the G0 phase of the cell cycle.
30 AT1, a gene known to regulate entry into the G0 phase of the cell cycle.
31 elial cells in culture to be arrested in the G0 phase of the cell cycle.
32 critical for cell cycle withdrawal in the G1/G0 phase of the cell cycle.
33 protein that controls progression out of the G0 phase of the cell cycle.
34 w cytometry revealed growth arrest in the G1/G0 phase of the cell cycle.
35  p21/WAF1 and arrested cell growth in the G1/G0 phase of the cell cycle.
36 ower in the G1 phase than those in the S and G0 phases of the cell cycle.
37 as indicated by accumulation of cells in the G0-phase of the cell cycle and growth recovery when cell
38 ling glial cells recruit quiescent cells (in G0 phase) onto the cell cycle, via a calcium signalling
39 ival factor, failing to advance B cells from G0 phase or stimulate thymidine incorporation.
40 tiwell-based variant of the chemical-induced G0-phase Premature Chromosome Condensation Assay that de
41  Subconfluent cells were synchronized in the G0 phase (quiescence) by serum starvation for 24 hours.
42                In vivo cell cycle quiescent (G0 phase) stem cell keratinocytes in primary culture seq
43 o need to postulate a restriction point or a G0 phase to explain the serum starvation results.
44 sitively controls p27(Kip1) stability in the G0 phase via p27(Kip1) Ser(10) phosphorylation, which is
45 feration and induced cell cycle arrest at G1/G0 phase via PPARgamma signalling pathway.
46                    Quiescent cells reside in G0 phase, which is characterized by the absence of cell