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1 ABA but can be closed by some noncompetitive GABA antagonists.
2 bination of pharmacological experiments with GABA antagonists, agonists, and uptake inhibitors, we fo
3                           Application of the GABA antagonists alone had little or no effect on basal
4                                              GABA antagonists also differentially alter opioid-induce
5                           Application of the GABA antagonists also increased the size of on-centre re
6                        Microinjection of the GABA antagonist bicuculline (BIC; 1 nmol) into the same
7                            Functionally, the GABA antagonist bicuculline had a greater influence on b
8 ing the MLR, by unilateral injections of the GABA antagonist bicuculline induced leg movements and su
9 on is greatly reduced in the presence of the GABA antagonist bicuculline.
10 d 17betaE2 antagonist ICI182,780, but not by GABA antagonist, bicuculline and phaclofen.
11 is rescued by intrastriatal injection of the GABA antagonist, bicuculline.
12 d-aspartate and sigma 1 antagonists, but not GABA antagonists, could completely eliminate the effects
13 further, whether general opioid or selective GABA antagonists decreased feeding elicited by GABA(A) o
14                                         Both GABA antagonists failed to significantly affect orexin A
15   Thus, opioid antagonist-opioid agonist and GABA antagonist-GABA agonist feeding interactions have b
16                                          The GABA antagonists had no effect on the responses of horiz
17 elicited by homeostatic challenges following GABA antagonists in these sites.
18 found that microinjections of bicuculline, a GABA antagonist, into the superior colliculus of both al
19                        Moreover, exposure to GABA antagonists or ACh agonists has a similar effect, w
20 ulation of GABAA receptors by bicuculline, a GABA antagonist, or by diazepam, a GABAergic agonist, di
21                                          The GABA antagonist pentylenetetrazol restored learning with
22 xperiment, excitation of the mPFC alone with GABA antagonist picrotoxin was insufficient to elicit th
23 cs similar to that in flies treated with the GABA antagonist picrotoxin.
24 local injection of BMI or the noncompetitive GABA antagonist picrotoxin.
25                                 However, the GABA antagonists picrotoxin and bicuculline enhanced the
26 uration was increased in the presence of the GABA antagonists picrotoxin and CGP55845.
27                           Application of the GABA antagonists, picrotoxin, bicuculline and 1,2,5,6-te
28 bited by 5-HT, concurrent application of the GABA antagonists significantly reversed this effect.
29                              The competitive GABA antagonist SR-95531 and a low, non-activating conce
30 is stimulation was completely blocked by the GABA antagonist SR95531.
31 BAergic, which is demonstrated by the use of GABA antagonists, uptake inhibitors, and double-labeling
32    Moreover, the effect of gabazine (GBZ), a GABA antagonist, was also studied on the neural activity
33                                              GABA antagonists were then iontophoresed with both gluta