ライフサイエンスコーパス: GABA receptor

1 e genes such as acetylcholinesterase and the GABA receptor.

2 e action of inhibitory steroids on the rho 1 GABA receptor.

3 mily that depend on cholesterol, such as the GABA receptor.

4 re noncompetitive antagonists (NCAs) for the GABA receptor.

5 nit, can impart picrotoxin resistance to the GABA receptor.

6 ibitory GABA receptor and a novel excitatory GABA receptor.

7 s-talk between these two distinct classes of GABA receptor.

8 tions between these two different classes of GABA receptor.

9 (i.e. fiprole), high-affinity probe for the GABA receptor.

10 release is necessary for maintaining axonal GABA receptors.

11 rophysiological activity of the transplanted GABA receptors.

12 oth small and large cholangiocytes expressed GABA receptors.

13 response to voltage pulses in cells lacking GABA receptors.

14 e VMH can be reversed by local modulation of GABA receptors.

15 otoxin and thus not mediated by conventional GABA receptors.

16 or rho subunit-containing GABA(C) over other GABA receptors.

17 gy of native, bicuculline-insensitive insect GABA receptors.

18 ases of resistance to insecticides acting on GABA receptors.

19 gs inhibit neural activity through targeting GABA receptors.

20 tor (CB1R) but not metabotropic glutamate or GABA receptors.

21 receptors, and bicuculline, an antagonist of GABA receptors.

22 the resistant to dieldrin (RDL) category of GABA receptors.

23 screen, the proteins of which interact with GABA receptors.

24 and by blockers of ionotropic glutamate and GABA receptors.

25 xin-sensitive nAChRs or picrotoxin-sensitive GABA receptors.

26 by picrotoxin-sensitive chloride conducting GABA receptors.

27 tes expressing recombinant alpha1beta2gamma2 GABA receptors.

28 inals where they are apposed by postsynaptic GABA receptors.

29 picrotoxin, indicating spontaneously opening GABA receptors.

30 M GABA-induced currents in alpha1beta2gamma2 GABA receptors.

31 gher concentrations, the steroid also blocks GABA receptors.

32 impairing the inhibitory action of neuronal GABA receptors.

33 in and hot plate tests that are dependent on GABA receptors.

34 ion of previously uncharacterized ionotropic GABA receptors.

35 marily mediated by chloride-permeable Type A GABA receptors.

36 e during simultaneous activation of NMDA and GABA receptors.

37 t-term plasticity and the configuration with GABA receptors.

38 e propensity of caged GABAs to interact with GABA receptors.

39 ration by LS/MS and its bioactivity on human GABA receptors.

40 drin), gene encoding a subunit of inhibitory GABA receptors.

41 due to clathrin-dependent internalization of GABA receptors.

42 ty of granule cells by acting on presynaptic GABA receptors.

43 nists of ionotropic gamma-aminobutyric acid (GABA) receptors.

44 ticide targeting on gamma-aminobytyric acid (GABA) receptors.

45 Aergic neurons or disruption of metabotropic GABA receptor 1 and 2 (GABA(B)R1/2) signaling in astrocy

46 feedback pathway is mediated by metabotropic GABA receptors.(1,2,5,6-Tetrahydropyridine-4-yl)-methylp

47 t microinjection of gamma-aminobutyric acid (GABA) receptor A antagonist substance, bicuculline, into

48 identified M1 residues to inhibit the UNC-49 GABA receptor, a homomeric GABA receptor from Caenorhabd

49 ucing intrastriatal gamma-aminobutyric acid (GABA) receptor-A inhibition synchronizes striatal dynami

50 We saw no evidence for changes in GABA receptor abundance or the overall number of glutama

51 oversy surrounding the role of Loop F during GABA receptor activation.

52 ation with propofol and other compounds with GABA receptor activity are frequently used in patients w

53 ic synaptic transmission but does not affect GABA receptor activity in cultured neurons.

54 ntrast, gaboxadol, a selective extrasynaptic GABA receptor agonist and late-stage investigational tre

55 nal nicotinic antagonist mecamylamine or the GABA receptor agonist muscimol, agents that reduce activ

56 isting of approximately 150-200 muscimols (a GABA receptor agonist) covalently joined to the qdot via

57 duration of sleep when the animals receive a GABA receptor agonist.

58 thalamus reproduce limbic cortex injury, and GABA-receptor agonist injections into anterior thalamus

59 b) reversible inactivation of the dlCPu with GABA receptor agonists (baclofen and muscimol) immediate

60 To test this hypothesis, GABA receptor agonists (baclofen/muscimol) were microinj

61 ext, we reversibly inactivated the vSub with GABA receptor agonists (muscimol+baclofen) before the co

62 In contrast to GABA receptor agonists which induce sleep by generalized

63 gents that influenced (lidocaine) or spared (GABA receptor agonists) fibers of passage blocked cocain

64 rescued by suppressing GABA reuptake and by GABA receptor agonists.

65 ex in adult rodents that can be prevented by GABA-receptor agonists and antipsychotics such as halope

66 arrangement of glutamate, acetylcholine, and GABA receptors along the dendrite that matched the previ

67 Activation of ionotropic glutamate and/or GABA receptors along the GnRH neuron projection is capab

68 acetylcholine, serotonin Type 3, glycine and GABA receptors) along with the crystal structure of the

69 Candidate gene GABRA2, which encodes the GABA receptor alpha2 subunit, was evaluated independentl

70 e that blockade of the alpha5 subtype of the GABA receptor (alpha5-GABA(A)Rs) leads to behavioral phe

71 s GABA neuron identity, a classic inhibitory GABA receptor and a novel excitatory GABA receptor.

72 ain receptors involved in chemotaxis, namely GABA receptor and EGFR.

73 acting as allosteric inhibitors of the rho 1 GABA receptor and support the hypothesis that divergent

74 However, ChIs express GABA receptors and are therefore candidates for potentia

75 n-presenting cells (APCs) express functional GABA receptors and respond electrophysiologically to GAB

76 pport for direct inhibition of DA release by GABA receptors and reveal that striatal GABA operates a

77 Combination of the blockade of mNTS GABA receptors and spinal iGLURs also abolished PVN-indu

78 Also, transcription of mRNA for both GABA receptors and the GABA reuptake transporter was aff

79 underlying mechanisms, including the role of GABA receptors and their link to synaptic adhesion molec

80 tion, suggesting potential links between the GABA receptors and these conditions.

81 ors, transporters, and interacting proteins, GABA receptors and transporters, synaptic-related marker

82 ogeneity within the gamma-aminobutyric acid (GABA) receptor and transporter families, a detailed insi

83 did not lead to the expression of functional GABA receptors, and injected oocytes failed to generate

84 can be attributed to their effects at insect GABA receptors, and the presence of a Val at the 2' posi

85 Activation of these GABA receptor anion channels can depolarize horizontal c

86 y to VIP(-/-) SCN cultures during continuous GABA receptor antagonism but not during G(i/o) blockade.

87 In contrast to the relative inability of GABA receptor antagonism in both sites to alter 2-deoxy-

88 A site-specific effect of GABA receptor antagonism was observed for deprivation-in

89 e further analyzed the response of NS to the GABA receptor antagonist bicuculline.

90 the behavior of larvae either treated with a GABA receptor antagonist or carrying a mutation that eli

91 OB interneurons or locally introducing DA or GABA receptor antagonists alters kinship preference.

92 ppressed transient output from bipolar cells.GABA receptor antagonists blocked the effect of ATPA.

93 diated inhibitory synaptic transmission, and GABA receptor antagonists did not block leptin-mediated

94 Glutamate and GABA receptor antagonists did not block this effect.

95 nses and reduces the disinhibitory effect of GABA receptor antagonists on PNs.

96 In addition, focal application of GABA receptor antagonists to sensory ganglia triggered o

97 e presence of either gabazine or picrotoxin (GABA receptor antagonists), many action potentials appea

98 in the presence of ionotropic glutamate and GABA receptor antagonists.

99 ceptors, with effects prevented by selective GABA receptor antagonists.

100 g persisted in the presence of glutamate and GABA receptor antagonists.

101 nce of muscarinic, ionotropic glutamate, and GABA receptors antagonists were also reduced by NRG 1bet

102 GABA receptor are involved in a number of complex disord

103 Ionotropic GABA receptors are abundant in both vertebrate and inver

104 ficance and mechanism(s) of the UV effect on GABA receptors are discussed.

105 M1) and pre-M1 of alpha and beta subunits in GABA receptors are essential for positive modulation of

106 These results suggest that GABA receptors are present and functional in the periphe

107 nts, indicating that a subset of presynaptic GABA receptors are tonically active.

108 Evidence that GABA receptors are transported to peripheral terminals a

109 Ionotropic GABA receptors are widely distributed throughout the ver

110 ctrophysiological properties of human native GABA receptors as a consequence of AD.

111 signaling by using a mimetic peptide of the GABA receptor associated protein-interacting domain of G

112 proteins N-ethylmaleimide-sensitive factor, GABA receptor-associated protein (GABARAP), and glutamat

113 ric binder of GABA receptors, GABARAPL1, the GABA receptor-associated protein, and SLC6A11, a postsyn

114 ated protein-1 light chain 3) family and the GABA receptor-associated proteins.

115 Lower levels of gamma-aminobutyric acid (GABA) receptor-associated protein (GABARAP) gene express

116 It is well established that GABA receptors at the central terminals of primary affer

117 , widely diverse NCA structures fit the same GABA receptor beta subunit site with important implicati

118 procedures yield a good model of the insect GABA receptor binding site and the location of agonists

119 ns of 3 alpha 5 beta PC, we characterize the GABA receptor block in some detail.

120 tiation and NMDA receptor block and diminish GABA receptor block may lead to a clinically useful neur

121 ated the increase in 5-HT efflux produced by GABA receptor blockade in the DRN.

122 VLM can be activated in the presence of RVLM GABA receptor blockade, but sympathoinhibitory influence

123 ke propagation becomes stereotyped following GABA receptor blockade.

124 Depression was prevented by MOR- but not GABA-receptor blockade.

125 rainstem slice with ionotropic glutamate and GABA receptors blocked, whole-cell patch-clamp recording

126 Here we show that the application of the GABA receptor blocker picrotoxin unmasks a robust excita

127 uropil or of interneurons in the presence of GABA receptor blockers caused no alteration in granule c

128 Indeed, we found that application of GABA receptor blockers increased the sensitivity of most

129 In the presence of GABA receptor blockers, residual taurine currents averag

130 und effects on the EPSCs were insensitive to GABA receptor blockers.

131 dback is mediated by at least three types of GABA receptor, both metabotropic and ionotropic.In conju

132 icotinic receptors (nAChRs) and also express GABA receptors, but they have not been eliminated as pot

133 we investigate the inhibition of the UNC-49 GABA receptor by another sulfated neurosteroid, dehydroe

134 sponse augmentation and the direct gating of GABA receptors by 5alpha-reduced potentiating steroids.

135 bunit is capable of forming fully functional GABA receptors by itself in Xenopus oocytes and suggest

136 e, we find that co-activation of presynaptic GABA receptors by photolytic uncaging of RuBi-GABA has a

137 obes is evident, the mechanisms by which the GABA receptors can be photocontrolled have not been full

138 Finally, blocking NMDA, but not GABA, receptors causes ATM levels to rise while ATR leve

139 GABA receptor/channel rho 1 subunit expression in the rh

140 We studied the expression localization of GABA receptor/channel rho 1 subunit in mouse spinal cord

141 To understand the functions of the GABA receptor/channel rho 1 subunit in these crucial sit

142 f sensory transmission in vivo, we generated GABA receptor/channel rho 1 subunit mutant mice (rho 1-/

143 These findings indicate that the GABA receptor/channel rho 1 subunit plays an important r

144 immunohistochemistry results indicated that GABA receptor/channel rho 1 subunits were expressed in m

145 gamma-Aminobutyric acid (GABA) receptor/channel rho 1 subunits are important comp

146 he discrete insertion and removal of AMPA or GABA receptor channels.

147 Therefore, the functional role of GABA receptor/channels in the brain, retina, and spinal

148 etaDB double mutants, the size and number of GABA receptor clusters are decreased at cerebellar inhib

149 Ionotropic gamma-amino butyric acid (GABA) receptors composed of heterogeneous molecular subu

150 Insect GABA receptors contain Ala at their 2' position in the p

151 Since the functional pentameric GABA receptor contains two alpha subunits, two beta subu

152 By recording endogenous GABA receptor currents directly from BC terminals in gol

153 Whole-cell GABA receptor currents were moderately sensitive to GABA

154 also show a significant reduction in DGC and GABA receptor distribution as well as abnormal neuronal

155 eceptor-like family member GrlB as the major GABA receptor during early development, and either disru

156 GrlE but not GrlB is the GABA receptor during late development.

157 e beta8-sheet of the adjacent subunit in the GABA receptor (E175-K46 being the structurally equivalen

158 0-F227) in the N-terminal domain of the rho1 GABA receptor expressed in Xenopus oocytes using a site-

159 Thus, the GABA receptors expressed by the yeast mRNA retained all

160 nts underlying the ionic selectivity of rho1 GABA receptors expressed in Xenopus oocytes and human em

161 that proton alters the apparent affinity of GABA receptors for agonist.

162 nhibit the UNC-49 GABA receptor, a homomeric GABA receptor from Caenorhabditis elegans that is homolo

163 Furthermore, GABA receptors from AD brains were slightly, but signifi

164 onto SACs - removing either GABA release or GABA receptors from SACs.

165 We measured presynaptic GABA receptor function at parallel fibre synapses onto s

166 LRRTM4 knockout could originate from reduced GABA receptor function.

167 s and receptors [glutamate, aspartate, GABA, GABA receptor (GABA-R), NMDA-R, AMPA-R, and kainate-R] a

168 r loop (ICL) of the gamma2 subunit of type A GABA receptors (GABA(A)R), which is required to anchor G

169 A-type and rho-subunit-containing (C-type) GABA receptors (GABA(A)Rs and GABA(C)Rs) mediate both fo

170 otoneurons and is required to recruit type A GABA receptors (GABA(A)Rs) at inhibitory neuromuscular j

171 We identified type-A GABA receptors (GABA(A)Rs) in inhibitory synapses and de

172 xpression of type A gamma-aminobutyric acid (GABA) receptor (GABA(A)R) subunit genes plays a critical

173 o acid GABA via activation of two ionotropic GABA receptors, GABA(A) and GABA(C).

174 ynaptic levels after 48 h blockade of type A GABA receptor (GABAA R)-mediated inhibition with bicucul

175 GABA receptors (GABAARs) mediate inhibition in the adult

176 The GABA receptor (GABABR) is a class C G protein-coupled re

177 iched synapses, virtually devoid of the main GABA receptor (GABAR) subunits alpha1 and gamma2.

178 GABA receptor (GABAR) types C (GABACR) and A (GABAAR) ar

179 are sufficiently hyperpolarized by increased GABA receptor (GABAR)-mediated HCO3- efflux, alkalinizin

180 schizophrenia often involve potentiation of GABA receptors (GABAR) to augment antipsychotic therapy

181 These include DBI, an allosteric binder of GABA receptors, GABARAPL1, the GABA receptor-associated

182 link between PMCA2 and glutamate receptors, GABA receptors (GABARs), and glutamate transporters that

183 ively spliced in TS compared to HC including GABA receptors GABRA4 and GABRG1, the nicotinic ACh rece

184 There is a strong history of mutations in GABA receptor genes being involved in neurologic disease

185 ric acid (GABA), in this putative imbalance: GABA receptor genes have been associated with autism in

186 of variants and mutations have been found in GABA receptor genes in patients with autism, schizophren

187 No variants of the commonly studied GABA receptor genes that have been associated with subst

188 thways strongly dominated in HIVE, including GABA receptors, glutamate signaling, synaptic potentiati

189 BA(A) receptors, study of steroid actions at GABA receptors has been hampered by a lack of pharmacolo

190 of recombinant rho1 gamma-aminobutyric acid (GABA) receptors has previously identified five residues

191 Striatal GABA is thought to modulate DA, but GABA receptors have not been documented conclusively on

192 Therefore, alpha4/6beta3delta GABA receptors have two distinct alcohol modulation site

193 Here, we describe a novel ionotropic GABA receptor in mouse cerebellar Purkinje cells (PCs) u

194 Here, we identify a GABA receptor in the nematode Caenorhabditis elegans tha

195 ping with the distinct expression pattern of GABA receptors in presynaptic cells, we detected no GABA

196 Activation of GABA receptors in rRPa does not mediate the cholinergic

197 ChR in rRPa does not depend on activation of GABA receptors in rRPa.

198 he increase in BAT SNA evoked by blockade of GABA receptors in rRPa.

199 The role of GABA receptors in synaptic transmission to neonatal rat

200 Little is known about the properties of GABA receptors in the amphibian brain.

201 nt NRHypo neurotoxicity mainly by activating GABA receptors in the anterior thalamus, diagonal band o

202 er, these results support the involvement of GABA receptors in the APC in feeding in general and the

203 Blockade of GABA receptors in the medial nucleus tractus solitarius

204 ion of agonists and antagonists, we compared GABA receptors in the medial vestibular nucleus of brain

205 ast inhibitory neurotransmission mediated by GABA receptors in the nervous system.

206 rmed yeast strains produced fully functional GABA receptors in Xenopus oocytes.

207 kade of ionotropric gamma-aminobutyric acid (GABA) receptors in the LH elicits eating in satiated rat

208 ied in principal cells, but INs also express GABA receptors, in particular the GABAA type (GABAARs),

209 nfirm that desensitization reversibly shifts GABA receptors into a high-affinity state.

210 The rho 1 GABA receptor is inhibited by a number of neuroactive st

211 One of the most widely studied insect GABA receptors is constructed from RDL (resistance to di

212 The rho1 subunit of the ionotropic GABA receptors is thought to contribute to the formation

213 domain (TM2) of the gamma-aminobutyric acid (GABA) receptor lines the integral ion pore.

214 These data suggest that in the PVN GABA receptors may be important regulators of cardiopulm

215 Peripheral GABA receptors may serve as a viable target for the trea

216 terations in spinal gamma-aminobutyric acid (GABA) receptors may contribute to persistent pain states

217 We directly recorded dendritic GABA receptor-mediated inhibitory synaptic events in adu

218 abapentin increased the amplitudes of evoked GABA receptor-mediated IPSCs (GABA-IPSCs) in CeA neurons

219 tence of time-locked, glutamate receptor and GABA receptor-mediated mono synaptic responses evoked by

220 Also, we observed a decrease in NMDA and GABA receptor-mediated synaptic transmission in the pyra

221 pocampus, without similarly changing NMDA or GABA receptor-mediated synaptic transmission, and withou

222 psychoses, as well as a marked reduction in GABA-receptor-mediated currents in pyramidal neurons of

223 es insulin-like peptide transcription in the GABA receptor neurons downstream of short neuropeptide F

224 the effects were predominantly directly upon GABA receptors of the neurons being recorded from.

225 he relative expression of the two classes of GABA receptor on each bipolar cell type correlates with

226 Because we could not localize ionotropic GABA receptors on cone axon terminals using electron mic

227 ne (DA) release, but definitive evidence for GABA receptors on DA axons is lacking.

228 The absence of ionotropic glutamate or GABA receptors on DA terminals indicates that modulatory

229 While the presence of GABA receptors on primary afferents has been well descri

230 r, evidence of positive immunoreactivity for GABA receptors on the neurite, as well as evidence for g

231 pic GABA receptor pathways, the metabotropic GABA receptor pathways act to enhance bipolar cell trans

232 n, axon guidance, and signaling of Notch and GABA receptor pathways, as well as factors important for

233 However, unlike the ionotropic GABA receptor pathways, the metabotropic GABA receptor p

234 lend new insights into the structure of the GABA receptor pore.

235 ferent compositions of insect and vertebrate GABA receptor pores are responsible for the differing to

236 We further tested if spinal blockade of GABA receptors prevents the antihyperalgesia produced by

237 Prior studies have shown that the GABA receptor RDL inhibits aversive olfactory learning v

238 ified a single point mutation (A302S) in the GABA receptor RDL that has been identified previously in

239 atures during adolescence, with frontal lobe GABA receptors reaching adult levels late in adolescence

240 TP in mitral cells by enhancing postsynaptic GABA receptor responses.

241 Activation of presynaptic GABA receptors results in a transient ( approximately 10

242 membrane potential ( approximately 8 mV) to GABA receptor reversal potential ( approximately -81 mV)

243 CRF increased postsynaptic GABA receptor sensitivity selectively in 5-HT neurons, a

244 ely modulate GAT expression via metabotropic GABA receptor signaling and highlight the importance of

245 r photoactivation depended on glutamate- and GABA-receptor signaling, and not on dopamine-receptor si

246 e results suggest that increasing peripheral GABA receptor signalling could be used to treat visceral

247 in the spinal cord dorsal horn and activates GABA receptors spinally.

248 ptor subtype antagonist-induced mediation of GABA receptor subtype agonist-induced feeding elicited f

249 nt with previously demonstrated differential GABA receptor subtype antagonist-induced mediation of op

250 ions of agonists or antagonists to the major GABA receptor subtypes.

251 d the role of RVLM gamma-amino butyric acid (GABA) receptor subtypes and glycine receptors in mediati

252 beta1 subunits (SCN1A, SCN2A, SCN1B) and one GABA receptor subunit gene (GABRG2) have been identified

253 nd showed similar effects of sex steroids on GABA receptor subunit gene expression in the AMD and HPC

254 GABA receptor subunit gene expression was generally high

255 These results support the hypothesis that GABA receptor subunit genes are involved in autism, most

256 Fourteen known autosomal GABA receptor subunit genes were studied to look for the

257 ng, adds to the functional diversity of this GABA receptor subunit.

258 mmunopositive for a gamma-aminobutyric acid (GABA) receptor subunit (GABAA Ralpha1 ), and that a syna

259 data demonstrate differential regulation of GABA receptor subunits and GABAergic system components i

260 In addition to expected components including GABA receptor subunits and gephyrin, several novel prote

261 transmission in cortical layer 4, including GABA receptor subunits and KCC2, and thus prevents the s

262 d copy number variation studies [3-7], fewer GABA receptor subunits have been observed in the post-mo

263 The EXP-1 protein resembles ionotropic GABA receptor subunits in almost all domains.

264 are the expression profiles of glutamate and GABA receptor subunits in three immortalized GnRH cell l

265 uctions of mRNA and protein of the principal GABA receptor subunits normally present in the temporal

266 AD groups and analyses of the proportion of GABA receptor subunits revealed down-regulation of alpha

267 s in gene expression levels of glutamate and GABA receptor subunits were compared between sedentary a

268 e evaluated the expression of genes encoding GABA receptor subunits, glutamic acid decarboxylase (GAD

269 Since no change is detected in the level of GABA receptors, such a reduction in tonic inhibition is

270 udies of alpha and beta subunits of type "A" GABA receptors suggest that these linkers couple extrace

271 dings reveal a novel "crosstalk" between the GABA receptor systems, which can be recruited under cond

272 sed in Xenopus laevis oocytes, EXP-1 forms a GABA receptor that is permeable to cations and not anion

273 , prevent the down-regulation of efficacy of GABA receptors that also occurs in these neurons after U

274 dulate postsynaptic excitability at NMDA and GABA receptors, the findings establish zinc as a cotrans

275 In addition to the synaptic GABA receptors, there is a group of GABAA receptors (GAB

276 y dynamics, and the contribution of specific GABA receptors to these dynamics.

277 non-NMDA receptor transmission and depressed GABA receptor transmission in the wild-type mice, both e

278 holine receptors, two glycine receptors, one GABA receptor, two AMPA-type glutamate receptors and one

279 Similar to the widely used GABA receptor type A (GABA(A)) antagonist picrotoxinin,

280 g in the pore of the alpha(2)beta(3)gamma(2) GABA receptor type A receptor at the so-called T6' ring,

281 The GABA receptor type C (GABA(C)) is a ligand-gated ion cha

282 nd that drugs that modulate the two dominant GABA receptor types in the brain, GABA(A) (clobazam) and

283 f D-MNs is mediated by a specific ionotropic GABA receptor (UNC-49) in AVA, and depends on electrical

284 We test this hypothesis in the GABA receptor using simultaneous electrophysiology and s

285 s and antagonists of mammalian glutamate and GABA receptors, using a specially developed 96-well micr

286 The wild-type GABA receptor was chloride selective, with a small but s

287 gative purinergic modulation of postsynaptic GABA receptors was accompanied by small presynaptic enha

288 n training, the sensitivity of glutamate and GABA receptors was unchanged.

289 ion pore and selectivity filter of the rho1 GABA receptor, we used the substituted cysteine accessib

290 Following evaluation at clinically relevant GABA receptors, we have identified a difluoromethyl keto

291 s laevis oocytes expressing recombinant rho1 GABA receptors, we identified agonist-mediated molecular

292 s action of the antagonist was occluded when GABA receptors were blocked, indicating that the reducti

293 c acetylcholine (nACh) and beta2alpha1gamma2 GABA receptors were constructed based on the torpedo neu

294 ergic currents and glia-driven modulation of GABA receptors were significantly reduced in the P2X4 KO

295 ed currents in individual oocytes expressing GABA receptors were tested by two-electrode voltage clam

296 e into Xenopus oocytes, expressed functional GABA receptors whose properties were investigated by usi

297 Thus, we describe a population of ionotropic GABA receptors with a mixed GABA(A)/GABA(C) pharmacology

298 Inhibiting GABA receptors with bicuculline increased NMDA receptor-

299 Antagonism of gamma-aminobutyric acid (GABA) receptors with bicuculline (BIC) phenocopied the d

300 GABA receptors within the mesolimbic circuitry have been