ライフサイエンスコーパス: GI

1 GI adverse events occurred in 42 patients (41%) after a

2 GI bleeding can also occur in patients affected by acqui

3 GI bleeding was the leading presentation in immunocompet

4 GI bleeding was the most common presentation.

5 GI cancers account for 26% of the global cancer incidenc

6 GI complications, including bowel distension, diarrhea,

7 GI events, reported in 97 patients (62%), were predomina

8 GI symptom severity (GSRS-IBS), and anxiety and depressi

9 GI symptoms were grouped into four categories, abdominal

10 GI was associated with CHD risk only in the continuous m

11 GI-CMV infection was frequently observed among immunocom

12 GI.1 Lot 001-09NV appears to be similar in virulence to

13 GI.1-specific cellular responses in peripheral blood wer

14 s ago, well before Greenland Interstadial 1 (GI-1).

15 iment core around Greenland Interstadial 10 (GI-10) ~41 ka BP and the Laschamp geomagnetic excursion.

16 year incidence of late RT-related grade >= 3 GI (2.5% v 3.9%) or genitourinary toxicity (2.9% v 2.9%)

17 Ki-67 indices for 50 GI-NETs were quantitated using SKIE and compared with DS

18 Keywords: Abdomen/GI, Cardiac, Infection, Nervous-Peripheral.

19 ts with advanced-stage CRC.Keywords: Abdomen/GI, CT, Comparative Studies, Large BowelSupplemental mat

20 ic protocol dual-energy CT.Keywords: Abdomen/GI, CT, CT-Dual Energy, CT-Quantitative, PancreasSupplem

21 acterized as category LR-M.Keywords: Abdomen/GI, Evidence Based Medicine, Liver, Neoplasms-Primary, U

22 Keywords: Abdomen/GI, Genetic Defects, Oncology, Pancreas Supplemental mat

23 Keywords: Abdomen/GI, Liver, MR-Imaging, Treatment Effects, Tumor Response

24 Hypoglycemia activates GI-ERalpha(vlVMH) neurons via the anoctamin 4 channel, a

25 creening revealed broad-spectrum activities (GI(50): 1.43-2.09 muM) and 17d had a remarkable selectiv

26 In addition, GI pain can be nociceptive, neuropathic and associated w

27 d RIR achieved similar peptide content after GI digestion.

28 cessibility in enriched eggs decreased after GI digestion except in RIR fried sample.

29 VIII concentrates is generally started after GI-bleeding events, but this therapy is not always succe

30 Pairwise interaction testing of all GI/ZTL allelic combinations shows similar affinities for

31 herapeutics without the intrusion of BBB and GI exposure.

32 of RB1 leads to profound nuclear defects and GI generating aberrant cytoplasmic DNA, however simultan

33 nd January 2018, patients seen in the ED and GI clinic of Loghman Hakim Hospital with unexplained abd

34 ts seen in the Laghman Hakim Hospital ED and GI clinic who were lead-intoxicated, with or without opi

35 Since ELF3 and GI are conserved across flowering plants and represent i

36 ircadian clock assays revealed that ELF3 and GI are essential that enable the oscillator to synchroni

37 ELF3 and GI are two important components of the Arabidopsis circa

38 single loss of function mutants of ELF3 and GI have been well studied, their genetic interaction rem

39 We found that ELF3 and GI repress growth differentially during the night and th

40 lacking both genes, suggesting that ELF3 and GI together convey photoperiod sensing to the central os

41 after first use, but no significant IPS and GI improvement after 4-week post-usage.

42 s somatic L1 insertions in paired normal and GI cancerous tissues.

43 lectivity studies, the NCI60 cell-panel, and GI(50) determinations for several cancer cell lines.

44 As GI microbiome activities are known to affect endocrine f

45 The association between GI and GL and offspring congenital heart defects was est

46 Associations between GI symptoms in IBS and individual and combinations of ne

47 degrees C, but very poor interaction between GI (Cvi) and ZTL (Cvi) at higher temperature.

48 hose with GI symptoms should be evaluated by GI endoscopy.

49 comprehensive treatment strategy for chronic GI diseases.

50 to 30 mug/dL (1.45 mumol/L) with concomitant GI symptoms.

51 rsus 1.47 +/- 1.76 mm; test versus control), GI reduction (-1.14 +/- 1.15 versus -1.04 +/- 0.89; test

52 lammatory (IC(50) = 72 ug/mL) and cytotoxic (GI(50) of 30-79 ug/mL) activities.

53 ng the follow-up, 962 participants developed GI cancers.

54 rtificial intelligence-based medical device (GI-Genius, Medtronic) trained to process colonoscopy ima

55 tions, including bowel distension, diarrhea, GI bleeding and ileus, were evaluated during the first 3

56 this study was to determine whether dietary GI, GL, and available carbohydrates are associated with

57 sttraumatic stress disorder, mood disorders, GI problems and chronic fatigue.

58 Proteolysis was highly affected by elderly GI alterations (p < 0.05) (50% of reduction compared to

59 ract, and many cancers, including epithelial GI tumors.

60 mmalian paralog gene pairs reveals extensive GIs and uncovers phenotypes normally masked by functiona

61 cking SHP2 in Osx(+) cells developed a fatal GI pathology with dramatic villus hypoplasia.

62 f OSTERIX with OLFM4 and LGR5, two bona fide GI stem cell markers, at the crypt cells.

63 nchanged after the introduction of FilmArray GI panel, but length of hospital stay was shorter (3 vs.

64 ibiotics after introduction of the FilmArray GI panel (14.0% vs. 4.5%; p < 0.001).

65 earlier after introduction of the FilmArray GI panel (hospital day 1 vs. 2, p < 0.0001).

66 t common pathogens detected by the FilmArray GI panel were Clostridium difficile (55.0%), Campylobact

67 presents an effective screening approach for GI mapping and the functional analysis of sizable genomi

68 hat perform functions that are important for GI health, nutrient harvest and growth hormone signaling

69 culation, and fecal shedding as a marker for GI colonization, we showed that K. pneumoniae can asympt

70 Stool specimens were tested by RT-qPCR for GI and GII noroviruses and subsequently genotyped by seq

71 Gastrointestinal (GI) bleeding is distinctive of severe von Willebrand dis

72 Gastrointestinal (GI) colonization with group B Streptococcus (GBS) is an

73 Gastrointestinal (GI) pain - a form of visceral pain - is common in some d

74 Gastrointestinal (GI) tract involvement is the major cause of morbidity an

75 howed reduced fitness in a gastrointestinal (GI) colonization model, yet induced greater damage to ep

76 687 patients with advanced gastrointestinal (GI) cancer in SCRUM-Japan GOZILA (no.

77 s' bio-accessibility after gastrointestinal (GI) digestion.

78 fering substantially among gastrointestinal (GI) locations.

79 een household fuel use and gastrointestinal (GI) cancers.

80 nd no associations between gastrointestinal (GI) symptoms and combined low disaccharidase activity.

81 ess is severely limited by gastrointestinal (GI) tract toxicity caused by gut bacterial beta-glucuron

82 characteristics of the CF gastrointestinal (GI) tract, including inflammation, maldigestion and mala

83 in multiple human chronic gastrointestinal (GI) disorders.

84 udy was to identify common gastrointestinal (GI) symptom groups using the Patient-Reported Outcomes M

85 fecal SCFA concentration, gastrointestinal (GI) symptoms, dietary intake, and quality of life were m

86 teroceptive cortex, during gastrointestinal (GI) malaise, a state akin to the emotion of disgust in h

87 lyze the effect of elderly gastrointestinal (GI) conditions on proteolysis, lipolysis and calcium and

88 l mucosa regulate feeding, gastrointestinal (GI) digestive, and immune functions.

89 re particularly robust for gastrointestinal (GI) cancers, such as those affecting the GI tract, liver

90 ion of NIR-II emitters for gastrointestinal (GI) tract imaging remains challenging due to fluorescenc

91 er-metabolism in the human gastrointestinal (GI) tract and respiratory system are largely unknown and

92 that fundamentally impact gastrointestinal (GI) biology.

93 SHP2 is involved in gastrointestinal (GI) epithelium development and homeostasis, but the unde

94 eports of taste changes in gastrointestinal (GI) inflammatory conditions.

95 Multiple gastrointestinal (GI) symptoms, including diarrhea, nausea/vomiting, and a

96 d 4.8 million new cases of gastrointestinal (GI) cancers and 3.4 million related deaths, worldwide, i

97 Development of gastrointestinal (GI) complications is adversely associated with prognosis

98 inked to the disruption of gastrointestinal (GI) tract epithelial barrier integrity and subsequent mi

99 ted in the pathogenesis of gastrointestinal (GI)-related cancers, including gastric carcinoma.

100 ed in the modelling of the gastrointestinal (GI) absorption of drugs and in oral drug development.

101 intain the function of the gastrointestinal (GI) epithelium are critical for health and survival of m

102 The gastrointestinal (GI) tract (GIT) holds significant potential as a target

103 ion and progression in the gastrointestinal (GI) tract and associated organs.

104 phil infiltration into the gastrointestinal (GI) tract and is characterized by various digestive symp

105 many of which involve the gastrointestinal (GI) tract and liver.

106 rial composition along the gastrointestinal (GI) tract and to assess SIBO in IBS.

107 hways mutation rate in the Gastrointestinal (GI) tract cancers in a systematic review and meta-analys

108 zed by inflammation of the gastrointestinal (GI) tract, and it is associated with different neurologi

109 yos, adult brains, and the gastrointestinal (GI) tract, and many cancers, including epithelial GI tum

110 imarily synthesized in the gastrointestinal (GI) tract, where it is secreted into the lumen and subse

111 crobial communities in the gastrointestinal (GI) tract.

112 reatment of lesions in the gastrointestinal (GI) tract.

113 ed for 24 hours, and then, gastrointestinal (GI) tissues were collected for histological examination.

114 ures and their relation to gastrointestinal (GI) symptoms are poorly understood.

115 otential is limited due to gastrointestinal (GI) syndrome, elucidation of the regenerative response f

116 acquisition, we assessed the kinetics of GBS GI colonization, progression to invasive disease, and th

117 ty of different disaccharidases with general GI symptom presentations in a large cohort of pediatric

118 f the Arabidopsis circadian clock, GIGANTEA (GI) and ZEITLUPE (ZTL), which likely act as compensatory

119 nous with the sharp stadial/interstadial (GS/GI) transitions within dating uncertainties.

120 es not support an association between a high GI and GL in midpregnancy and increased offspring risk o

121 Substitution of high-GI foods with minimally processed, whole, fiber-rich car

122 The results suggest that high-GI diets could be a risk factor for insomnia in postmeno

123 up continued to exhibit statistically higher GI scores than the control group (P = 0.029).

124 His GI symptoms and anemia improved on a multiple-food-elimi

125 n immunocompetent mice and modifies the host GI microbiota.

126 Further, few have addressed how GIs can be imputed when data are scarce.

127 edictability of drug absorption in the human GI tract than a Caco-2 Transwell system (Spearman's corr

128 st groups resulted in significant changes in GI and BOP at Day 42 compared to control and un-flossed

129 e found no evidence of global differences in GI between domestic and wild canids, certain regional di

130 for the first time found to be expressed in GI crypt cells, and SHP2 expression in the crypt Osx+ ce

131 strated significantly greater improvement in GI and IPS from baseline (p < 0.01) and greater efficacy

132 ing titers (BT50) and a 161-fold increase in GI.1-specific immunoglobulin (Ig)G titers when compared

133 y variable, 2) most somatic L1 insertions in GI cancer tissues are absent from normal tissues, and 3)

134 the accuracy of Ki-67 index quantitation in GI-NETs: (1) Synaptophysin-KI-Estimator (SKIE), a pipeli

135 lity, showing >50-fold higher sensitivity in GI tract, and migrated homogenously during gastrointesti

136 pectedly revealed a critical role of SHP2 in GI biology.

137 obustness of TC through natural variation in GI and ZTL alleles found on the Cape Verde Islands.

138 robing depth (PD) >=4 mm and gingival index (GI) >=1 at >=4 sites distributed over >=2 anterior teeth

139 Gingival index (GI) and bleeding on probing (BOP) were evaluated in addi

140 attachment level (CAL), and gingival index (GI) were performed by calibrated masked examiners for up

141 ental plaque score (IPS) and gingival index (GI) were recorded at baseline and 4-week post-usage.

142 lifestyle intervention (low glycaemic index (GI) diet plus physical activity) in pregnant women with

143 s indicate that high dietary glycemic index (GI) and glycemic load (GL) are associated with increased

144 between midpregnancy dietary glycemic index (GI), glycemic load (GL), and sugar-sweetened beverages a

145 luate the nutritional value, glycemic index (GI), total phenol content (TPC), and total flavonoid con

146 surface, we compared the gyrification index (GI) in 19 carnivore species, including six wild canid an

147 vel (CAL), gingival recession, plaque index, GI, and bleeding on probing (BOP) were recorded at basel

148 wn to partially alleviate irinotecan-induced GI tract damage and resultant diarrhea in mice.

149 the rate of change in gingival inflammation (GI) was dramatically different between the APE test grou

150 ctivated by hypoglycemia (glucose-inhibited [GI] neurons) have been assumed to play a critical althou

151 Genomic instability (GI) predisposes cells to malignant transformation, howev

152 staining both proteome and genome integrity (GI) requires the integration of a wide range of mechanis

153 Systematic mapping of genetic interactions (GIs) and interrogation of the functions of sizable genom

154 Mapping genetic interactions (GIs) can reveal important insights into cellular functio

155 a foundation for new avenues of inquiry into GI stem cell biology and of OSTERIX's therapeutic and di

156 ro and in vivo stability of genomic islands (GIs), integrative conjugative elements (ICEs), and proph

157 Isolated GI symptoms were reported rarely.

158 plored correlations between global and local GI with brain mass, cortical thickness, white and gray m

159 nalysis was stratified by upper GI and lower GI lesions.

160 Upper and lower GI tract endoscopic examinations found no bleeding.

161 elayed bleeding after EMR of upper and lower GI tract lesions.

162 Lesions in upper and lower GI tracts that underwent EMR between January 2012 and De

163 D included pre-existing lung diseases, lower GI endoscopy and antibiotics.

164 conventional HMT in the development of lower GI, higher value-added functional starch foodstuffs.

165 section procedures of the upper and/or lower GI tract in 6 patients in 2 endoscopy centers.

166 In the lower GI subgroup, clip application did not alter the effect o

167 at dysplastic mucosal lesions of the luminal GI tract can be challenging.

168 estigated the global burden from the 5 major GI cancers, as well as geographic and temporal trends in

169 e, attack rate, and duration of illness make GI.1 Lot 001-09NV a useful challenge strain for future v

170 is a prognostic factor for survival in many GI cancers, there is limited knowledge regarding its imp

171 et of experiments on these models in matched GI datasets from baker's and fission yeast.

172 t breads may serve as products with a medium GI, as a source of TPC, and as products with a high AC.

173 Both the Mild and Moderate GI groups demonstrated a similar configuration of sympto

174 ignificantly higher in the Mild and Moderate GI symptom groups, and they indicated significantly lowe

175 Mild to moderate GI symptoms appear to emerge at much earlier ages and ar

176 sequencing in the same centers and network (GI-SCREEN, 5,621 patients).

177 uctuations, being glucose-inhibited neurons (GI-ERalpha(vlVMH)) or glucose-excited neurons (GE-ERalph

178 No GI adverse event-related deaths were recorded.

179 -negative bacteria, is present in the normal GI tract and levels increase during high-fat feeding and

180 Here, we utilize a NoV GI.1 Jun-Fos-assisted phage display library constructed

181 A stock of HuNoV strain Norwalk virus ([NV] GI.1) was prepared.

182 In physiological studies, absence of GI neuron activity after AMPK suppression in the VMN had

183 n of the Cape Verde Islands (Cvi) alleles of GI and ZTL into the Landsberg erecta (Ler) background, w

184 useful interventions to reduce the burden of GI cancers worldwide.

185 However, identifying the cause of GI pain frequently represents a diagnostic challenge as

186 s and polyps or cancer as possible causes of GI bleeding.

187 Additionally, molecular characterization of GI (Cvi) reveals a novel functional motif that can modul

188 NG) to propagate cells with a high degree of GI.

189 is a promising target for the development of GI tract therapies, yet it has been under-exploited due,

190 oit cross-species information in the form of GI data across multiple species, and arbitrary side info

191 bdominal pain and have a unique frequency of GI symptoms due to low frequency of diarrhea and weight

192 We outline a vision for the future of GI electroceuticals, building on advances in mechanistic

193 ased from 25% to 37% after implementation of GI PCR (P = .01).

194 dy, we aimed to investigate the incidence of GI complications and their influence on prognosis of hos

195 Is for a CHD event, in relation to intake of GI, GL, and carbohydrates, were estimated using covariat

196 p 1 and 2 continued having reduced levels of GI, BOP, and PI at Day 60 (P<0.05).

197 ping and subsequent individual management of GI pain might be options in the future.

198 Mechanisms of GI pain are complex and include both peripheral and cent

199 onse in the Black Sea region at the onset of GI-10.

200 Rates of GI bleeding (IRR, 1.80 [CI, 1.75 to 1.84]), sepsis (IRR,

201 The highest rates of GI bleeding, sepsis, and heart failure occurred within t

202 tional Study, investigating the relations of GI, glycemic load, other carbohydrate measures (added su

203 regression were conducted to assess risk of GI adverse events.

204 t chimney was associated with higher risk of GI cancers combined (10-y HR: 1.05; 95% CI: 1.00, 1.11),

205 and natural gas with the subsequent risk of GI cancers.

206 The risks of GI cancers combined, esophageal cancer, and gastric canc

207 Treatment of GI pain depends on the precise type of pain and the prim

208 n incidence and mortality for all 5 types of GI cancers.

209 on advances in mechanistic understanding of GI physiology coupled with novel ingestible technologies

210 e providers evaluate individual patterns of "GI health" when young adults present with anxiety and de

211 Three groups were identified based on GI symptom elevations: Normal (n = 649), Mild (n = 257),

212 w and meta-analysis of international data on GI and liver manifestations of COVID-19.

213 rom early-onset disease, yet their impact on GI colonization and LO disease is unexplored.

214 Individual-level information on GI and GL, offspring congenital heart defects, and healt

215 114 (11%) patients experienced at least one GI complication and diarrhea (5.2%) was the most common.

216 ults surveyed were experiencing at least one GI symptom of a severity greater than normative levels.

217 network measures and IBS symptom severity or GI-specific anxiety but we found a significant differenc

218 afferent morphology, interactions with other GI elements, plasticity, and function.

219 s with the FilmArray Gastrointestinal Panel (GI PCR) in June 2016.

220 groups showed significant reductions in PD, GI, and CAL gain overtime.

221 raditional model of care following pediatric GI surgery.

222 ing the safety of early feeding in pediatric GI surgery, with or without a fast-track program.

223 cally handled system that uses large porcine GI tissue explants that are functionally maintained for

224 , n = 163) and from June 2016-May 2017 (post-GI PCR, n = 182) and followed them for 1 year after tran

225 fficile versus 49 patients (27%) in the post-GI PCR cohort (P < .001).

226 C. difficile diarrheal pathogens in the post-GI PCR cohort were enteropathogenic Escherichia coli (n

227 received an HCT from June 2014-May 2015 (pre-GI PCR, n = 163) and from June 2016-May 2017 (post-GI PC

228 Eight patients (5%) in the pre-GI PCR cohort tested positive for a pathogen other than

229 he maximal inhibition of cell proliferation (GI(50)) in the low micromolar range in the PC3 cell line

230 formation System - GI symptom scales (PROMIS-GI) within a large sample of young adults.

231 e ages of 18 and 25 who completed the PROMIS-GI as part of ongoing research monitoring physical and p

232 The PROMIS-GI is a freely available, adaptable, normatively referen

233 It is clear that BMP signaling regulates GI function and disease progression that involve stem/pr

234 immune checkpoint inhibitor therapy-related GI adverse events in patients with cancer and inflammato

235 reexpression of Txn2 in Sf1 neurons restored GI neuron activity.

236 e the first such experiments on genome-scale GI datasets in multiple species in the same study.

237 delta(17)O = -2.92 +/- 0.79 per mille (SD)), GI fluid (GF; deltaD = -35.91 +/- 7.30 per mille (SD), d

238 Compared to a critical care setting, GI complications are not commonly observed in a non-crit

239 Preexisting IBD increases the risk of severe GI adverse events in patients treated with immune checkp

240 ollected, cooked, and subjected to simulated GI digestion.

241 Although the incidence of some GI cancer types has decreased, this group of malignancie

242 f1 neurons of the VMN selectively suppressed GI neuron activity.

243 Animals exhibit sustained GI colonization following both perinatal and postnatal e

244 ed Outcomes Measurement Information System - GI symptom scales (PROMIS-GI) within a large sample of y

245 We also found that GI in canids is positively correlated with cortical surf

246 Our results indicated that GI values for domestic dogs are largely consistent with

247 Further, we show that GI-ERalpha(vlVMH) neurons preferentially project to the

248 The GI mucosa showed eosinophil infiltration of more than 40

249 The GI score (P = 0.039), BOP% (P = 0.023), serum CRP level

250 the time of maximum plaque accumulation, the GI of the APE test group was a 109% greater than the GI

251 al (GI) cancers, such as those affecting the GI tract, liver, and pancreas, on which this review is f

252 ads in lumenal and mucosal samples along the GI tract.

253 pneumoniae can asymptomatically colonize the GI tract in immunocompetent mice and modifies the host G

254 complex, large mucosal-based lesions in the GI tract using standard endoscopic equipment and a novel

255 cretion and visceral hypersensitivity in the GI tract, and has been implicated in diseases such as ir

256 a cytokine that mediates inflammation in the GI tract, but the transcriptional program that governs G

257 s a nonredundant inflammatory pathway in the GI tract.

258 novel functional motif that can modulate the GI/ZTL interaction as well as nucleocytoplasmic partitio

259 emonstrate that during the disruption of the GI tract and increased microbial translocation, we find

260 Endoscopic examination of the GI tract is necessary to exclude ulcers and polyps or ca

261 mon cause of foreign body perforation of the GI tract.

262 c target for preneoplastic conditions of the GI tract.

263 ualization of the detailed structures of the GI tract.

264 nd samples per day) of whole segments of the GI tract.

265 primary pacemaker conductance in ICs of the GI tract.

266 The lowest values of the GI were determined for oat bread, whereas breads with th

267 rrelate with a difference in strength of the GI/ZTL interaction.

268 intestinal stem cells (ISCs) regenerate the GI epithelium.

269 enteric adipose tissue (MAT) surrounding the GI tract.

270 e APE test group was a 109% greater than the GI of the test group (P <= 0.001) despite similar plaque

271 Thus, the GI microbiota represent a potential therapeutic target f

272 Colonization density within the GI tract and levels of shedding in the feces differed am

273 sentation and pathological damage within the GI tract, positioning GM-CSF as a key regulator of GVHD

274 to growing microbial communities within the GI tract.

275 e genome of V. cholerae to remove all of the GIs, ICEs, and prophages and revealed important interact

276 These GI symptom groups were then compared on key psychosocial

277 Finally, these GI disorders did not negatively influence the bioabsorba

278 and treatment-related factors in addition to GI and genitourinary baseline function, with higher scor

279 sulted in high-magnitude fire peaks close to GI/GS boundaries, even exacerbated by local peatland con

280 ubjects developed strong immune responses to GI.1 with a 30-fold (geometric mean titer) increase in b

281 lic alterations, increased susceptibility to GI disease, and a system-wide recalibration of immune co

282 r in gastrointestinal neuroendocrine tumors (GI-NETs) and defines tumor grade.

283 Analysis was stratified by upper GI and lower GI lesions.

284 Netherlands, using data from the Dutch Upper GI Cancer Audit (DUCA) for transthoracic esophagectomy.

285 All patients registered in the Dutch Upper GI Cancer Audit who underwent potentially curative esoph

286 count < 50 x 10(9) /L) and nonvariceal upper GI bleed (diagnosed after doing upper gastrointestinal e

287 osis with coagulopathy and nonvariceal upper GI bleeding, TEG-guided transfusion strategy leads to a

288 international multicenter study by the Upper GI International Robotic Association (UGIRA) aimed to ga

289 inis obtained from patients undergoing upper GI cancer surgery compared with controls (N = 30; n = 1,

290 abinoids as anecdotal treatments for various GI disorders inspired the search for mechanisms by which

291 drug screen and showed potent activity with GI(50) values reaching the nanomolar level, with mean gr

292 IX expression is found to be associated with GI malignancies.

293 haridase combinations may be associated with GI symptoms in subjects; a prospective study may be need

294 likely after ~15,000 years ago, coeval with GI-1.

295 and SH but showed positive correlation with GI, PI, and BOP in both groups.

296 ctive study included patients diagnosed with GI-CMV infection at Siriraj Hospital (Bangkok, Thailand)

297 cular-weight heparin (LMWH) in patients with GI and potentially genitourinary cancers.

298 nxiety and depression when they present with GI complaints.

299 followed up for a long time, and those with GI symptoms should be evaluated by GI endoscopy.

300 etecting functional features associated with GIs.