ライフサイエンスコーパス: GST

1 GST activity can be further controlled by adding specifi

2 GST aims to establish an association relationship betwee

3 GST is widely studied in the metabolism of xenobiotics a

4 GST omega 1 (GSTO1) is an atypical GST isoform that is o

5 GST pull-down indicated that nesprin-1/lamin/SUN interac

6 GST pulldown and coimmunoprecipitation studies reveal th

7 GST pulldown and indirect enzyme-linked immunosorbent as

8 GST pulldown assays demonstrate that the dimerization do

9 GST pulldown assays demonstrated that vIRF1 interacts wi

10 GST-AE1 pull-down assays using human kidney membrane pro

11 GST-B1B2 inhibited CLL cell migration as effectively as

12 GST-based pulldowns and coimmunoprecipitation experiment

13 GST-PEX9 inhibited MMP-9-driven gelatin proteolysis, mea

14 GST-PX is purely a tether, as it supports fusion without

15 GST-PX tethering supports the assembly of new, active SN

16 sessed for cross-reactivity with the other 3 GSTs using antibody binding assays.

17 ents from tropical Colombia had IgE to all 4 GSTs.

18 significant IgE cross-reactivity among the 4 GSTs is in agreement with the low shared amino acid iden

19 ructured phase-change alloy Ge(2)Sb(2)Te(5) (GST) can support a diverse set of multipolar Mie resonan

20 Sam35 and Sam37 each adopt a GST-like fold, with no functional, structural, or sequen

21 sion of the SMPL-engineered Mtd variant as a GST-bound fusion protein demonstrated specific binding t

22 n when IRE1alpha is forced to dimerise, by a GST-tag, phospho-enhancement of activity is still observ

23 (ii) A GST fusion protein comprising the C terminus of SERT pul

24 e inhibiting the function of KDM1A maps to a GST-tag.

25 assay, the His-tagged Myb1 interacted with a GST-TvCyP1 fusion protein, which had an enzymatic profic

26 mouse hearts, Gstp1/2 was the most abundant GST transcript followed by Gsta4 and Gstm4.1, and GSTP a

27 Accordingly, GST-PEX9 also abolished the degradation of gelatin by MM

28 The ppITAM peptide strongly activated GST-SYK but was less effective in activating upSYK untag

29 , the question of whether GDAP1 is an active GST has not been clearly resolved.

30 based probes (ABPs) that characterize active GSTs in mammalian tissues.

31 Arabidopsis lines overexpressing TNT-active GSTs AtGST-U24 and AtGST-U25 were compromised in biomass

32 at increased levels of endogenous TNT-active GSTs catalyse excessive glutathionylation of endogenous

33 he hepatic antioxidant enzymatic activities (GST and GPx), clearly showing that this combination incr

34 We hypothesized that altered enzyme activity GST genotypes influence the susceptibility for esophagea

35 ein mixture containing bovine serum albumin, GST, and ubiquitin could be specifically probed in paral

36 lly flexible building units of the amorphous GST network, intimately linked to the presence of distin

37 aining with phospho-specific antibodies, and GST pull-down assays showed that Nck determines spatiote

38 P60, HSP70, alphaTUB, UBC, RPS18, ATPase and GST, were analyzed using a panel of analytical tools, in

39 rdingly, ELISAs demonstrated that GST-B4 and GST-B1 specifically bound to gelatin.

40 capacities and flexibilities of the EROD and GST activities in Antarctic fish were significantly lowe

41 As both, fission and GST activities of GDAP1, are critically dependent on HD1

42 y increase in glutathione reductase (GR) and GST activities.

43 Furthermore, Immunoprecipitation and GST pull-down assays demonstrated that TRIM28 interacts

44 chain (KLC-1/2) and immunoprecipitation and GST pull-down showed that these proteins interacted via

45 Mutation analysis, immunoprecipitation, and GST pulldown assays based on the theoretical predictions

46 s (GSTs) [microsomal GST (mGST)2, mGST3, and GST-mu (GSTM)4] from their epoxide precursors [16S,17S-e

47 s from the combination of ROS production and GST-pi inhibition.

48 enosine triphosphate), HOPS is required, and GST-PX does not suffice.

49 und this gene organization of a split RO and GST gene cluster to occur more broadly, implying a large

50 , Asc s 1 (ABA-1), Asc l 3 (tropomyosin) and GST (glutathione transferase).

51 rabidopsis plants overexpressing GST-U24 and GST-U25 exhibit significantly enhanced ability to withst

52 physiological concentrations of GST-U24 and GST-U25 result in only a limited innate ability to cope

53 glutathione transferases (GSTs), GST-U24 and GST-U25, from Arabidopsis (Arabidopsis thaliana) that ar

54 To assess the role of GST-U24 and GST-U25, we purified and characterized recombinant forms

55 NT-active Arabidopsis thaliana (Arabidopsis) GSTs.

56 hibited CLL cell migration as effectively as GST-B3B4.

57 otyrosine-recognition sequences expressed as GST-fusion proteins.

58 boundary resistance of 28 +/- 8 m(2)K/GW at GST-SiO2 interfaces and an effective thermopower 350 +/-

59 effective thermopower 350 +/- 50 microV/K at GST-Pt interfaces.

60 S-transferase omega 1 (GSTO1) is an atypical GST isoform that is overexpressed in several cancers and

61 GST omega 1 (GSTO1) is an atypical GST isoform that is overexpressed in several cancers and

62 transferase Omega 1 (GSTO1-1) is an atypical GST reported to play a pro-inflammatory role in response

63 ere, we identify novel features of bacterial GSTs that cleave beta-aryl ether bonds typically found i

64 eep's milk Tulum cheeses in goat's skin bag (GST) or plastic barrel (PBT) were investigated.

65 H is a derivative of a known mechanism-based GST inhibitor that binds within the active site and inhi

66 vity of bacteria that express only Cys-based GSTs could be related to the low or null affinity of the

67 hat only the more recently evolved Tyr-based GSTs display enough affinity for DNDGIC (KD < 10(-9) M)

68 ne-S-transferase-fused recombinant biglycan (GST-BGN) on craniofacial bone regeneration.

69 Furthermore, our results from both GST pulldown assays and analytical ultracentrifugation s

70 Vav3-Cdc37 interaction was confirmed by GST pulldown and, for native proteins, by co-immunopreci

71 tion and to assess potential interactions by GST polymorphisms.

72 direct interaction between Med19 and Med1 by GST pulldown experiments indicating privileged contacts

73 rminal 44 amino acids of PDZD11, as shown by GST-pulldown assays.

74 f these proteoliposomes is also supported by GST-PX or GST-FYVE, recombinant dimeric proteins which t

75 phobic domain that is not found in canonical GSTs.

76 Quantitative proteomic analysis of Cav1-GST (amino acids 1-101) pull downs showed sixfold-increa

77 cCPE.GST or GST (GST is glutathione S-transferase) was conjug

78 plastic lesions and accumulated (111)In-cCPE.GST (3.17 +/- 0.51 %ID/g) but not (111)In-GST (0.99 +/-

79 The uptake of (111)In-cCPE.GST in claudin-4-positive MDA-MB-468 xenograft tumors in

80 Taken together, (111)In-cCPE.GST targets claudin-4 expression in frank tumors and pre

81 rs were claudin-4-positive, and (111)In-cCPE.GST uptake was 3.2 +/- 0.70 %ID/g, significantly higher

82 The affinity of radiolabeled cCPE.GST for claudin-4 was confirmed using claudin-4-expressi

83 GSTT1-active genotypes.Conclusions: Certain GST genotypes may have protective effects against the lo

84 mparative sequence analysis of characterized GSTs of the PHYA1, PHYB, and HY5 genes of G. hirsutum an

85 Sigma class GST (Prostaglandin D synthase), FhGST-S1, is present in

86 h structural similarity to other Sigma class GSTs.

87 Immunohistochemical colocalization, GST pulldown, and surface plasmon resonance studies reve

88 report here experimental results confirming GST-like activity for 82 of them, along with 37 new 3D s

89 Specifically, conventional GST is no longer appropriate due to the gene-set selecti

90 We propose bcGST, a bias-corrected GST by introducing bias-correction terms in the continge

91 on the lens plane by assigning the designed GST crystallization levels to the corresponding slits, a

92 literature along with the lowest detectable GST concentration (200pmolL(-1)) for GST label-free sens

93 blocking JAK2 function increases detoxifying GSTs in hepatocytes and protects against oxidative liver

94 To investigate, we studied 16 different GSTs, revealing that negative cooperativity is present o

95 By examining 42 different GSTs we discovered that only the more recently evolved T

96 s are homodimers that fall into two distinct GST subclasses: LigE homologues, which cleave the beta(R

97 rality of this behavior across the divergent GST family and its evolutionary significance were unclea

98 sensitizer (BPS) and an ethacrynic acid (EA) GST-pi inhibitor.

99 Each GST is needed for accurate molecular diagnosis in differ

100 It revealed that elevated GST and heat loss from basements are dominant factors in

101 hanced dephosphorylation of recombinant ERK2-GST in an in vitro phosphatase assay.

102 ity is present only in more recently evolved GSTs, indicating evolutionary drift in this direction.

103 ylation catalyzed by a bacterially expressed GST-tagged human PRMT7 fusion protein with a broad range

104 Moreover, GST-B4 and, to a lesser extent, GST-B1 also inhibited gelatin degradation by MMP-9, indi

105 eins and could particularly detect extrinsic GST proteins added in crude Escherichia coli or 293T cel

106 s between subunits may be proposed for a few GSTs.

107 Fhb7 GST homologs are absent in plants, and our evidence supp

108 First, GST-DMAP pulled down several acid hydrolases, but not no

109 -2) and detection limit of 1.3 ng mL(-1) for GST-p16 protein which is equivalent to 0.49 ng mL(-1) fo

110 ectable GST concentration (200pmolL(-1)) for GST label-free sensors.

111 Participants were genotyped for GST polymorphisms (GSTM1/T1/P1) (n = 429 [69.2%]).

112 cis-acting regulatory elements required for GST-specific expression.

113 Furthermore, GST and SOD activities of trout exposed to both Se-Met a

114 Purified PP1c bound to recombinant Gbeta1-GST protein, and PP1c co-immunoprecipitated with Gbeta1

115 t Raman spectra of HfO2, TiO2 and Ge2Sb2Te5 (GST) films, and demonstrate direct measurements of tempe

116 e phases of the PCM section (here Ge2Sb2Te5 (GST)) allows for designing a tunable plasmonic switch fo

117 filled with phase-change material Ge2Sb2Te5 (GST).

118 cular basis of this inhibition, we generated GST fusion proteins containing PEX9 or truncated forms c

119 cifically for a presumed sensitive genotype (GST-theta-1+/+), and PBPK modeling accounting for human

120 , GTL; glucobrassicin, GBC; gluconasturtiin, GST; glucoalyssin, ALY; 4-hydroxyglucobrassicin, 4-OH; 4

121 cCPE.GST or GST (GST is glutathione S-transferase) was conjugated to the

122 ance of two glutathione transferases (GSTs), GST-U24 and GST-U25, from Arabidopsis (Arabidopsis thali

123 sotropy (kappaa/kappac~2) of kappa in bulk h-GST, with the dominant contribution to kappa from optic

124 nductivity (kappa) of hexagonal Ge2Sb2Te5 (h-GST) is studied via direct first-principles calculations

125 s to the thermal conductivity in low-kappa h-GST is unusual, and development of fundamental physical

126 The phonon spectrum of h-GST has very dispersive optic branches with higher group

127 dels for lower GST and validate under higher GST, with three calibration experiments.

128 However, GST-PEX9 did not prevent the degradation of other MMP-9

129 Using yeast two-hybrid, GST pull-down, co-immunoprecipitation and bimolecular fl

130 United States, Bla g 5 is the most important GST allergen and lack of coexposure to GSTs from certain

131 Importantly, GST tests for over-representation of genes in an annotat

132 Importantly, GST-U25 catalyzed the denitration of TNT to form 2-gluta

133 els of total FFA and total OA were higher in GST-cheese in comparison to PBT-cheese but total FAAs we

134 tive affinity for matrix compared with p6 in GST-pulldown experiments was higher for ALIX than for TS

135 uld play a role in negative cooperativity in GSTs.

136 r bond arose independently at least twice in GSTs and that BaeAB homologues may be important for clea

137 PE.GST (3.17 +/- 0.51 %ID/g) but not (111)In-GST (0.99 +/- 0.39 %ID/g; P < 0.001).

138 .70 %ID/g, significantly higher than (111)In-GST (1.00 +/- 0.60 %ID/g; P < 0.05).

139 ice was significantly higher than in (111)In-GST or claudin-4-negative HT1080 tumors (6.72 +/- 0.18 v

140 r amounts of antioxidant proteins, including GSTs and novel carbonyl detoxifying enzymes, such as ald

141 at binds within the active site and inhibits GST activity.

142 Instead, GST P1-1 sequesters and inactivates cisplatin with the a

143 zed immobilization for Schistosoma japonicum GST.

144 se new data, along with experimentally known GST reactions and structures reported in the literature,

145 Furthermore, full-length GST-mu1A and the GST-mu1A C-terminal domain, but not the

146 However, full-length GST-mu1A did not bind to the phospho-L-selectin tail or

147 All three Lig GSTs produced the ketone product (beta-S-glutathionyl-al

148 onstrate for the first time theta-class-like GST activity for GDAP1, and it's activity being regulate

149 terminal secretion of reporter proteins like GST and YopE lacking its native secretion signal.

150 allowed us to calibrate the models for lower GST and validate under higher GST, with three calibratio

151 ompletely blocked upon formation of the MDM2/GST-p53TAD complex owing to charge conversion.

152 indicating that Nutlin-3 disrupted the MDM2/GST-p53TAD complex, thereby releasing MDM2.

153 lutathione S-transferases (GSTs) [microsomal GST (mGST)2, mGST3, and GST-mu (GSTM)4] from their epoxi

154 Moreover, GST-B4 and, to a lesser extent, GST-B1 also inhibited ge

155 This gave the sensitivity of 1.78 muA mL(ng GST-p16)(-1) cm(-2) and detection limit of 1.3 ng mL(-1)

156 maintain a residual conjugating activity of GST against toxins even in the presence of high DNDGIC c

157 he glutathione (GSH) transferase activity of GST P1-1.

158 tivity were both enhanced by the addition of GST-BGN.

159 Analysis of GST structures identified elements that could play a rol

160 One implicit assumption of GST is that the gene expression platform captures the co

161 y in plants, physiological concentrations of GST-U24 and GST-U25 result in only a limited innate abil

162 harnessing the dramatic optical contrast of GST, we realize broadband (Deltalambda/lambda ~ 15%) mod

163 asily regenerated, allowing the detection of GST with the very same device.

164 Here, we investigated the effects of GST-BGN lacking any posttranslational modifications on B

165 By varying the crystallization level of GST from 0% to 90%, the Fabry-Perot resonance supported

166 optimal BMP-2 combined with 4 or 8 microg of GST-BGN.

167 BMP-2 combined with 0, 2, 4, or 8 microg of GST-BGN.

168 ty of cyclin D1/Cdk4 upon phosphorylation of GST-Rb.

169 Both the enzymatic proficiency of GST-TvCyP1 and its binding to His-Myb1 were eliminated b

170 However, a proof of GST activity and its possible impact on membrane dynamic

171 ic signaling, further confirming the role of GST P1-1 as an antiapoptotic protein.

172 To assess the role of GST-U24 and GST-U25, we purified and characterized recom

173 As DNDGIC is also a strong inhibitor of GSTs, we suggest negative cooperativity might have evolv

174 acks a signal sequence but contains an omega GST fold.

175 cCPE.GST or GST (GST is glutathione S-transferase) was conjugated to

176 oteoliposomes is also supported by GST-PX or GST-FYVE, recombinant dimeric proteins which tether by b

177 d not covalently react with UbcH7, USP08, or GST-tagged human rhinovirus 3C protease.

178 strategy coupled with TAP-TAG or orthogonal GST- purification followed by mass spectrometry, we iden

179 trate that Arabidopsis plants overexpressing GST-U24 and GST-U25 exhibit significantly enhanced abili

180 Glutathione-pi (GST-pi) is a key enzyme that militates against ROS-media

181 g GAM, the cutoff value of 14.7 mug/L for pi-GST showed the best performance to predict composite out

182 The addition of pi-GST to the SOFA score improved risk stratification (tota

183 Urinary pi-GST differentiated patients with/without advanced AKI or

184 Thus, urinary pi-GST levels predict advanced AKI or hospital mortality af

185 Urinary pi-GST predicted advanced AKI at 3 hrs post-surgery (p = 0.

186 yzed for urinary alpha-(alpha-) and pi-(pi-) GSTs.

187 ype and pfhrp2-deleted parasite populations (GST = .046, p </= .00001).

188 y potential therapeutic targets, we prepared GST-PEX9 forms containing structural blades B1B2 or B3B4

189 lB, YidC, and LepI) and one soluble protein (GST), each fused to GFP, was examined.

190 g RNAi, in situ epitope tagging of proteins, GST pulldown, and coimmunoprecipitation assays, and immu

191 Pulldown assays with purified GST-l-PGDS and His(6)-Rab4 indicated that both proteins

192 Recombinant GST-theta with monochlorobimane substrate had an optimum

193 Furthermore recombinant GST-theta activity was abolished by the denaturants trit

194 The bonafide of 40.8 kDa recombinant GST+Alr1105 fusion protein was confirmed by immunoblotti

195 Purified recombinant GST-TIMAP interacted directly with purified recombinant

196 Products of recombinant GSTs (LigE, LigP, and LigF) are beta-S-glutathionyl-alph

197 e broadly, implying a larger function for RO-GST protein partners.

198 suggest a potential role for M. rosenbergii GST-theta in detoxification and possibly conferring immu

199 m to discriminate among structurally similar GST isozymes.

200 SNAP-GST pull-down assays demonstrate SCRIB binds multiple al

201 ies with an elevation in levels of GSH, SOD, GST and t-GPx activities.

202 rization of enzyme- and active site-specific GST activity in mammalian model systems.

203 also investigated, which make the structured GST serve as a multispectral optical switch with high ex

204 Current O-PCMs, such as Ge-Sb-Te (GST), exhibit large contrast of both refractive index (D

205 pairs, of the prototypical PCM, Ge2 Sb2 Te5 (GST).

206 he difference in growing season temperature (GST) varied between 2.2 and 8.2 degrees C in different t

207 e Ca(2+)-dependent interaction of N-terminal GST-tagged RcCDPK1 with BTPC.

208 enomes are important tools in Gene-Set Test (GST) that describe gene biological functions and associa

209 Accordingly, ELISAs demonstrated that GST-B4 and GST-B1 specifically bound to gelatin.

210 Furthermore, it is well known that GST P1-1 binding to the c-Jun N-terminal kinase (JNK) in

211 Our demonstration that GSTs are key in vivo regulators of axonal mitochondrial

212 Our study suggests that GSTs play a proinflammatory role in priming astrocytes a

213 The GST active site is composed of a GSH binding "G site" an

214 Furthermore, full-length GST-mu1A and the GST-mu1A C-terminal domain, but not the GST-mu1A N-termi

215 etic background, a Th1 polarization, and the GST-tag.

216 Changing the GST phase to crystalline via optical heating allows for

217 -dinitrobenzene (CDNB) and the drugs for the GST enzyme in the electrochemical potential at 0.1V vs.

218 5, 6, 7, 11, 12, and 13 participated in the GST-catalyzed conjugation, indicating the substrate tole

219 tion with vWFA2 fused to intein (lacking the GST-tag).

220 the GST-mu1A C-terminal domain, but not the GST-mu1A N-terminal domain, bind to L-selectin tail pept

221 cal properties of the amorphous phase of the GST section we adjusted the extinction peak of the dipol

222 The sensor can quantify the GST enzyme concentration through its biospecific interac

223 overall capacitance values according to the GST concentration.

224 P1 contains primary sequence homology to the GST superfamily; however, the question of whether GDAP1

225 fication of postprandial xenobiotics via the GST action mediated hepatic GSH conjugation.

226 related to the low or null affinity of their GSTs for DNDGIC.

227 the absence of TNT, overexpression of these GSTs reduces root and shoot biomass, and although glutat

228 inal features of the reaction cycle of these GSTs, including stereospecific substrate recognition and

229 mite were tested for IgE reactivity to these GSTs.

230 rization of glutathione s-transferase-theta (GST-theta) from freshwater prawn Macrobrachium rosenberg

231 dative regulation of the active site of this GST protein.

232 CLL cells bound to GST-B1B2 and CD44 was the primary receptor.

233 y replaced by fusing the catalytic domain to GST to promote dimerization.

234 a short GW protein-derived peptide fused to GST and demonstrate that it binds to Ago proteins with h

235 ve moiety for UV-induced covalent binding to GSTs and GSH-binding enzymes.

236 rtant GST allergen and lack of coexposure to GSTs from certain species allows a better assessment of

237 in flower buds was specifically localized to GSTs.

238 mGstp1/2 deletion reduced total GST activity, but no compensatory increase in GSTA and G

239 vity was a significant fraction of the total GST activity.

240 ity occurs in human glutathione transferase (GST) GSTP1-1 when it binds and neutralizes a toxic nitri

241 itrosation of human glutathione transferase (GST) P1-1, a major detoxification enzyme and key regulat

242 probed the role of glutathione transferase (GST) P1-1, an antiapoptotic protein often overexpressed

243 Glutathione S-transferase (GST) affinity pulldowns also suggest that p18 and Gag in

244 Fhb7 encodes a glutathione S-transferase (GST) and confers broad resistance to Fusarium species by

245 , catalase (CAT), glutathione-s-transferase (GST) and non-enzymatic antioxidant - reduced glutathione

246 he stimulation of glutathione S-transferase (GST) and superoxide dismutase (SOD) activities.

247 rsible label-free glutathione-S-transferase (GST) biosensor is demonstrated.

248 were subjected to glutathione S-transferase (GST) E-cadherin pulldown and immunoblot analysis to asse

249 nt members of the glutathione-S-transferase (GST) enzyme family.

250 ties of bacterial glutathione-S-transferase (GST) enzymes that cleave beta-aryl ether bonds in lignin

251 e electrodes with glutathione-s-transferase (GST) enzymes.

252 enzymes from the glutathione S-transferase (GST) family may protect against these lung function defi

253 interactions with glutathione S-transferase (GST) genes due to reduced antioxidant function in partic

254 at the Drosophila glutathione S-transferase (GST) Gfzf prevents mitochondrial hyperfusion in axons.

255 Considering that glutathione S-transferase (GST) is a broadly employed enzymatic fusion tag, we repo

256 ity of functional glutathione S-transferase (GST) metabolic activity, the key activation pathway for

257 ified recombinant glutathione-S-transferase (GST) proteins and could particularly detect extrinsic GS

258 Glutathione S-transferase (GST) pull-down experiments found novel binding partners

259 313 to 549, by a glutathione S-transferase (GST) pulldown assay.

260 spectrometry and glutathione S-transferase (GST) pulldown assays identified integrin alpha5 as a nov

261 precipitation and glutathione S-transferase (GST) pulldown assays revealed that GBP1 interacted with

262 tion and in vitro glutathione S-transferase (GST) pulldown assays.

263 r, enzymes in the glutathione S-transferase (GST) superfamily function in stress responses, defense s

264 out an N-terminal glutathione S-transferase (GST) tag, resulting in monomeric or obligatory dimeric S

265 re the use of the glutathione s-transferase (GST) to anchor the bactericidal peptide, melittin, to th

266 hylase (EROD) and glutathione-S-transferase (GST), and (ii) the metabolic clearance of benzo(a)pyrene

267 us, activities of glutathione-S-transferase (GST), general esterases (ESTs) and phenol oxidase (PO) d

268 modifier (Sumo), glutathione S-transferase (GST), maltose-binding protein (MBP), N-utilisation subst

269 oxidative stress (glutathione-S-transferase (GST), superoxide dismutase (SOD)), and fish health (cond

270 lytic activity of glutathione-s-transferase (GST), which is not its natural enzyme partner.

271 n host cells with glutathione S-transferase (GST)-Asp14 significantly inhibited infection of host cel

272 bound directly to glutathione S-transferase (GST)-fused M3R intracellular loops 2 and 3 (M3Ri2 and M3

273 P particles, and glutathione S-transferase (GST)-P domain fusion proteins to sialic acid-containing

274 d the activity of glutathione S-transferase (GST).

275 and NdmE, a novel glutathione-S-transferase (GST).

276 ted function as a glutathione S-transferase (GST).

277 ic translation of glutathione S-transferase (GST-3) from constitutive mRNA levels in vivo is dependen

278 Glutathione S-transferases (GST) were evaluated as biomarkers of AKI.

279 e omega class of Glutathione S-transferases (GST), yet differ from them in their ability to form ion

280 Glutathione transferases (GSTs) are protection enzymes capable of conjugating glut

281 importance of two glutathione transferases (GSTs), GST-U24 and GST-U25, from Arabidopsis (Arabidopsi

282 se (LTC(4)S) and glutathione S-transferases (GSTs) [microsomal GST (mGST)2, mGST3, and GST-mu (GSTM)4

283 e stereospecific glutathione S-transferases (GSTs) called beta-etherases to cleave the beta-aryl ethe

284 Glutathione S-transferases (GSTs) comprise a diverse family of phase II drug metabol

285 sensitization to glutathione S-transferases (GSTs) occurs in tropical and subtropical environments.

286 , we report that glutathione S-transferases (GSTs), particularly GSTM1, promote proinflammatory signa

287 and activity of glutathione S-transferases (GSTs).

288 ties of GDAP1 to glutathione S-transferases (GSTs).

289 localized to glandular secretory trichomes (GSTs) of leaf primordia and top expanded leaves.

290 Thus, in turn, GST P1-1 overexpression and Pt-induced subunit cross-lin

291 s parallels with similar roles for unrelated GSTs in MDR in humans and shows their potential as targe

292 Using GST-CTD fusion protein substrates we find that CDK12/Cyc

293 In addition, using GST affinity purification combined with mass spectrometr

294 By utilizing GST pull-down and immunoprecipitation assays, we validat

295 forms a stable complex with DAT in vivo via GST pulldown and co-immunoprecipitation assays.

296 x and PAT1 was further confirmed by in vitro GST pulldown and overlay assays and in intact neutrophil

297 binding with an in vitro pulldown assay with GST-Rab5(GTP) Of the 35 p110beta helical domain mutants

298 Moreover, preincubating host cells with GST-tagged forms of both Asp14 and outer membrane protei

299 xperimental EBA induced by immunization with GST-COL7, disease manifestation depended on the genetic

300 ell invasion, but increased interaction with GST-B-Raf as compared with wild-type-FLAG-MLK3 in H2O2-t

301 effective in activating upSYK untagged with GST.