ライフサイエンスコーパス: Giemsa

1 Giemsa stain revealed that BI were aggregates of mesench

2 they were younger than 180 months and had a Giemsa-stained thick blood smear that was positive for P

3 itivity of 54% and a specificity of 87%; and Giemsa stain (> 2% ICO) had a sensitivity of 46% and a s

4 tide composition, of different isochores and Giemsa light and Giemsa dark bands.

5 of different isochores and Giemsa light and Giemsa dark bands.

6 immunofluorescence, electron microscopy, and Giemsa staining.

7 is correlated with subtelomeric regions and Giemsa-light bands (R bands).

8 high-resolution molecular karyotype arrays, Giemsa banding (G-banding) and fluorescent in situ hybri

9 ity (CIN) induced by CHEK1 were confirmed by Giemsa staining, exon sequencing, immunofluorescence and

10 at a dosage of 10 mg/kg/day as determined by Giemsa-stained organ impression smears.

11 the cultures were monitored for infection by Giemsa staining and PCR.

12 osinophils, they were purified (>95% pure by Giemsa-stained cytospin preparations) from liver granulo

13 the Apis mellifera karyotype as revealed by Giemsa stain.

14 S technique is approximately 400 chromosomal Giemsa bands, the data presented here provide the first

15 For Giemsa stained sections, the decision support algorithm

16 PCR-HRM analysis of DNA extracts from Giemsa-stained thick blood smears or corresponding blood

17 Gram, Giemsa, calcofluor white, and acridine orange stains and

18 by flow cytometric analysis and May-Grunwald Giemsa staining.

19 films stained with Wright's or May-Grunwald-Giemsa, determination of blood counts, platelet size and

20 Biopsy-based tests (i.e., culture, histology Giemsa stain and rapid urease test) and non-invasive tes

21 s with atrophy, the sensitivity of histology Giemsa stain was 100%, 100%, 88%, and 66%, respectively,

22 e frequency of induced chromatid breakage in Giemsa-stained preparations was determined.

23 Morphology of the parasite was confirmed in Giemsa-reagent stained blood smears for the Type I sampl

24 aplasma-like inclusions were demonstrated in Giemsa-stained culture samples.

25 hin 2 weeks, infected cells were detected in Giemsa-stained culture samples, and the organisms subseq

26 icroscopic identification of the organism in Giemsa-stained thin blood smears, detection of babesial

27 ted 4 to 5 hours earlier than it was seen in Giemsa-stained preparations and 8 hours earlier than it

28 ed by six different procedures that included Giemsa, trichrome, chromotrope, Gram-chromotrope, acid-f

29 Standard malaria diagnosis is Giemsa stained peripheral blood smear but false negative

30 in regions of high GC, and in R and lightest Giemsa bands.

31 H&E staining for histology and modified Giemsa staining for the detection of H. pylori was condu

32 tiation was based on microscopic analysis of Giemsa-stained slides.

33 ted PCR assay and microscopic examination of Giemsa-stained blood films for detection and identificat

34 d objective supplement to the examination of Giemsa-stained blood smears and may replace microscopy f

35 nal method, i.e., microscopic examination of Giemsa-stained lesion scraping (46.7%), biopsy culture (

36 iosis was made by microscopic examination of Giemsa-stained thin blood smears or a real-time polymera

37 ts for duplicate microscopic examinations of Giemsa-stained blood smears as the reference diagnostic

38 detection remains basic light microscopy of Giemsa-stained patient blood smears to first enable dete

39 roperties of cells, morphological studies of Giemsa-stained cells, annexin V binding, and DNA fragmen

40 was as sensitive and specific as the use of Giemsa-stained blood smears and inoculation of hamsters.

41 ency of infected red blood cells assessed on Giemsa-stained blood smears.

42 In detail, we can detect H. pylori both on Giemsa- and regular H&E stained whole slide images.

43 Parasitemia was monitored on Giemsa-stained blood smears or by flow cytometry.

44 now been coupled with the fluorescence-plus-Giemsa method of Perry and Wolff to produce harlequin en

45 se bands showed this more than the gene-poor Giemsa dark bands, and morphometric analyses demonstrate

46 ith clinical symptoms of malaria, a positive Giemsa-stained blood film for P falciparum, and no signs

47 ng superior performance over manually-scored Giemsa-stained smears, and a limit of detection below 0.

48 sex-mismatched transplants using a two-step Giemsa/fluorescence in situ hybridization assay on isola

49 We have sequenced 1949 kb from the terminal Giemsa light band of human chromosome 16p, enabling us t

50 s of blood from infected mice stained by the Giemsa or the indirect immunofluorescence method, numero

51 1 lie distally, near the lower border of the Giemsa band adjacent to the distal one-third of CFA9.

52 s the parasitemia detection rate compared to Giemsa staining (90%), it offers a significant advantage

53 btained by reading 100 fields of traditional Giemsa-stained thick-smear blood films.

54 men who underwent karyotyping analysis using Giemsa-Trypsin-Leishman (GTL) banding prior to the ICSI

55 Microscopic diagnosis of malaria using Giemsa-stained blood smears is the standard of care in r

56 and the presence of gastric H. pylori using Giemsa stain.

57 ure was monitored by staining the cells with Giemsa and quantifying the wound area with SigmaS can co

58 mated blood culture, malaria microscopy with Giemsa-stained blood films, and human immunodeficiency v

59 from three slides, two of them stained with Giemsa (on which Plasmodium parasites could still be see

60 id and in the pouch wall after staining with Giemsa or after enzymatic digestion followed by fluoresc

61 raphs of hematologically stained (eg, Wright-Giemsa) examples of mouse basophils exist in the literat

62 We observed, using Wright-Giemsa and 4',6-diamidine-2-phenylindole-dihydrochloride

63 cted STAT1(-/-) mice and stained with Wright-Giemsa had basophil characteristics.

64 ir-dried smears that are stained with Wright-Giemsa or other Romanowsky-type stains.