ライフサイエンスコーパス: Gilbert

1 Gilbert damping increases approximately linearly with in

2 Gilbert et al. conclude that evidence from the Open Scie

3 Gilbert et al. presented DNA analysis of coprolites reco

4 Gilbert et al. report in a recent issue of Cell on the a

5 Gilbert Stork, professor emeritus at Columbia University

6 Gilbert syndrome (GS) is an autosomal recessive inherite

7 Gilbert syndrome (GS) is characterized by intermittent u

8 Gilbert syndrome is a common genetic disorder associated

9 Gilbert syndrome is a frequent inherited disorder in Egy

10 Gilbert syndrome, a hyperbilirubinemic syndrome, has a p

11 Gilbert's syndrome is a common inherited disorder of bil

12 Gilbert-Diamond D, Emond JA, Baker ER, Korrick SA, Karag

13 In 1968, Gilbert and Pollak conjectured that for any P, Ls(P) >/=

14 syndrome, from 6 members of a family with 4 Gilbert members, and from 77 non-smoking, alcohol-free,

15 The linewidth is a Gilbert type at the saturated state, with damping of 0.0

16 More than 40 years ago, Gilbert Gottlieb and like-minded scholars argued for the

17 elopment of the chemical method of Maxam and Gilbert and the dideoxy method of Sanger, Nicklen and Co

18 he explanation too difficult to succeed, and Gilbert's makes it too easy.

19 e north arm (Gunnison Bay) to the south arm (Gilbert Bay) previously drove the perennial stratificati

20 on velocity gradients have been predicted by Gilbert and Jenkins.

21 rase 1 (UGT1A1) gene has been shown to cause Gilbert syndrome, a benign form of unconjugated bilirubi

22 curonosyltransferase-1A1 deficiency, causing Gilbert's syndrome, has been attributed to two extra (TA

23 curonosyltransferase-1A1 deficiency, causing Gilbert's syndrome, may be carried by the donor liver an

24 l Nino-influenced coral reefs in the central Gilbert Islands of the Republic of Kiribati, which exper

25 One confirmed Gilbert's patient, two recurrent jaundice patients (with

26 experimental study of temperature-dependent Gilbert damping in permalloy (Py) thin films of varying

27 Analysis of the effective Gilbert damping and phase shift indicates a clear signat

28 vel controls on gene expression."In essence, Gilbert's conjecture, that DNA methylation is not one of

29 bin; the effects can be benign and frequent (Gilbert syndrome) or rare but severe, increasing the ris

30 Fan H, Gilbert R, O'Callaghan F, Li L.

31 211 (6%) patients had Gilbert's syndrome and 3168 (94%) did not have this cond

32 rwent a 24 h fasting test to see if they had Gilbert's syndrome, and all four positives had the 7/7 g

33 efore relapse was recorded (38 [18%] who had Gilbert's syndrome vs 461 [15%] who did not have Gilbert

34 00 after transplant (47 [22%] of 211 who had Gilbert's syndrome vs 617 [19%] of 3168 who did not have

35 ndrome vs 617 [19%] of 3168 who did not have Gilbert's syndrome), and for non-relapse mortality 499 (

36 ert's syndrome vs 461 [15%] who did not have Gilbert's syndrome).

37 Upon reactive pathway progression, as in Gilbert's Syndrome (GS; UGT1A1*28 polymorphism), aggrava

38 dition associated with jaundice in adults is Gilbert's syndrome, which is characterized by an allelic

39 ffusion model coupled to the Landau-Lifshitz-Gilbert equation numerically.

40 By solving stochastic Landau-Lifshitz-Gilbert equation of magnetization dynamics in the presen

41 s a broad consensus that the Landau-Lifshitz-Gilbert equation reliably describes the magnetization dy

42 ations based on a stochastic Landau-Lifshitz-Gilbert equation suggest that this rotation is driven so

43 s of iron oxide clusters and Landau-Lifshitz-Gilbert simulations confirmed our hypothesis, indicating

44 mployed atomistic stochastic Landau-Lifshitz-Gilbert simulations to investigate skyrmions in amorphou

45 Using atomistic Landau-Lifshitz-Gilbert simulations we quantify the polarization changes

46 ferromagnet) governed by the Landau-Lifshitz-Gilbert-Slonczewski equation in the absence of magnetic

47 ow FMR linewidths as low as 17.5 G and a low Gilbert damping coefficient of 1.1 x 10(-3), values that

48 anisotropy energy of 0.95 MJ/m(3) and a low Gilbert damping of 0.01.

49 omagnetic resonance (FMR) linewidths and low Gilbert damping coefficients-crucial prerequisites for t

50 -suited for quantum magnonics due to its low Gilbert damping and substrate versatility.

51 nAl-ferrite thin films with an unusually low Gilbert damping parameter (<3 x 10(-3) ), as well as str

52 roximately 10% of the population manifesting Gilbert's syndrome.

53 rom Michael Bratman, the other from Margaret Gilbert.

54 e of the difference between Sanger and Maxam-Gilbert indexing is examined for a number of duplexes of

55 1) promoter, as well as providing good Maxam-Gilbert sequence information.

56 fication and cleavage according to the Maxam-Gilbert sequencing protocol.

57 ive conditioning regimens, compared with non-Gilbert's syndrome patients.

58 at manifests a colossal anisotropic nonlocal Gilbert damping with a maximum-to-minimum ratio of up to

59 We assessed the association of Gilbert's syndrome with overall mortality and non-relaps

60 The genetic basis of Gilbert's syndrome is ill-defined.

61 he (TA)7 allele was considered diagnostic of Gilbert's syndrome.

62 However, the effect of Gilbert's syndrome on the disposition of some drugs can

63 The effect of Gilbert's syndrome on the risk of overall mortality and

64 ether or not they had laboratory evidence of Gilbert's syndrome.

65 xistence of a mild and a more severe form of Gilbert's syndrome, depending on whether the gene defect

66 udy was conducted to assess the frequency of Gilbert syndrome among Egyptian adults.

67 applied to a cohort from PGP, predictions of Gilbert syndrome, Graves' disease, non-Hodgkin lymphoma,

68 ) peaks can be modeled by isotherms based on Gilbert-Jenkins theory, providing a robust approach to e

69 xperimental observation of giant oscillatory Gilbert damping in the superconducting niobium/nickel-ir

70 ues acting against the equilibrium-restoring Gilbert damping of the magnetization dynamics.

71 nucleophilic addition-elimination, Seyferth-Gilbert homologation, transphosphorylation, and a 1,3-di

72 gh material throughput, and the key Seyferth-Gilbert homologation is optimized to avoid racemization.

73 reagents for the tandem processes (Seyferth-Gilbert homologation and Sonogashira coupling) to occur.

74 n the 40 years since Harvard medical student Gilbert Omenn first described a rare, inherited disorder

75 from 12 patients with confirmed or suspected Gilbert's syndrome, from 6 members of a family with 4 Gi

76 recurrent jaundice patients (with suspected Gilbert's syndrome), and nine clinically diagnosed cases

77 s part of the "psychological immune system" (Gilbert 2006; Mandelbaum 2019) and functions to protect

78 east milk jaundice, and the realization that Gilbert's syndrome may play a greater role in neonatal j

79 The Gilbert damping is found to be almost independent of the

80 The Gilbert damping is very low but increases dramatically w

81 The Gilbert damping of ferromagnetic materials is arguably t

82 The Gilbert Ridge and Tokelau Seamounts are the only seamoun

83 relation between the noise amplitude and the Gilbert damping enhancement associated with these metals

84 umanized UGT1 mice that expressed either the Gilbert's UGT1A1*28 allele [Tg(UGT1(A1*28))Ugt1(-/-) mic

85 a single-mode damped sinusoid to extract the Gilbert damping parameter.

86 The pathway of ATP hydrolysis follows the Gilbert-Johnson pathway determined previously for a simi

87 GT flakes and extract an upper limit for the Gilbert damping parameter.

88 0Ar/39Ar ages indicate that the bends in the Gilbert Ridge and Tokelau seamount trail were formed muc

89 mically homogeneous products obtained in the Gilbert-Seyferth homologation for C=C bond formation.

90 n indinavir-treated HIV patients lacking the Gilbert's polymorphism versus 1.45 mg/dl in those who we

91 d, key parameters of spin dynamics, like the Gilbert damping, crucial for designing ultra-fast spintr

92 ortant insight to the physical origin of the Gilbert damping in ultrathin magnetic films.

93 Understanding the physical origin of the Gilbert damping is highly relevant to developing future

94 We first show how the increase of the Gilbert damping, caused by the inclusion rare-earth dopa

95 opment, a finding that is independent of the Gilbert's UGT1A1*28 promoter polymorphism.

96 agnetic resonance measurements show that the Gilbert damping is mostly insensitive to temperature ove

97 nside the poly(A) tail, at the same time the Gilbert group at Harvard was sequencing the 5' end.

98 ndence, two independent contributions to the Gilbert damping are identified, namely bulk damping and

99 While the Gilbert damping constant can reach 10(-4) to 10(-5) in s

100 Segregation of the 7/7 genotype with the Gilbert phenotype was also demonstrated in the family wi

101 n HIV-infected patients with and without the Gilbert's polymorphism.

102 ia in liver transplant recipients was due to Gilbert's syndrome acquired through the liver allograft.

103 e diffusion-free limit previously subject to Gilbert-Jenkins theory.

104 In 1985 Walter Gilbert challenged members of the DNA methylation commun

105 in my career were to stumble upon the Watson-Gilbert laboratory at Harvard when I entered graduate sc

106 Since the work of William Gilbert in 1600, it has been widely believed that the Ea

107 homozygous for the mutation associated with Gilbert syndrome and heterozygous for a second mutation

108 A1 promoter variant sequence associated with Gilbert's syndrome, and the fourth was a heterozygote.

109 Patients with Gilbert syndrome have low levels of a normal form of uri

110 Patients with Gilbert's syndrome should receive busulfan-containing my

111 Patients with Gilbert's syndrome were defined as having laboratory val

112 nt were significantly worse in patients with Gilbert's syndrome who received busulfan-containing myel

113 that contained busulfan (n=1131), those with Gilbert's syndrome (n=60) were at a significantly increa

114 melphalan conditioning regimens, those with Gilbert's syndrome had similar outcomes to those without

115 with low UGT1A1 activity, such as those with Gilbert's syndrome, may be at an increased risk for irin

116 ality by day 200 compared with those without Gilbert's syndrome (n=1071; hazard ratio [HR] 2.30, 95%

117 ndrome had similar outcomes to those without Gilbert's syndrome (overall mortality at day 200 HR 0.90