ライフサイエンスコーパス: Glc

1 Glc and BR can promote LR emergence at lower concentrati

2 Glc could also regulate several genes involved in BR met

3 Glc could regulate the transcript level of 72% of BR-reg

4 Glc may interact with BR via a hexokinase1 (HXK1)-mediat

5 Glc transport by HvSTP13 heterologously expressed in yea

6 Glc, a fundamental signaling and metabolic molecule, pro

7 Glc-mediated thermotolerance involves HSP induction via

8 Glc-primed accumulation of H3K4me3 at thermomemory-assoc

9 Glc-supplemented cells exhibited a marked reduction in l

10 Feeding experiments using (1-13C)Glc followed by analysis of labeling patterns by 13C-NMR

11 Seedling transfer to plates with 2% Glc plus ACC mimics the high-Glc effect in the HKL1 over

12 ctures revealed the disaccharide Rha-(1-->3)-Glc as a minimal epitope.

13 de repeating unit as well as the Rha-(1-->3)-Glc disaccharide are promising novel vaccine candidates

14 A CRM197-Rha-(1-->3)-Glc disaccharide conjugate was able to elicit antibodies

15 ere observed among all five substrates (1-3, Glc 1P, and Man 1P) for either enzyme-catalyzed reaction

16 lactose [Gal-beta(1 --> 6)-Gal-beta(1 --> 4)-Glc].

17 ial dextran) which also contain alpha-(1,3,6)Glc branch points.

18 --> 6)-Gal], allolactose [Gal-beta(1 --> 6)-Glc] and 6'-O-beta-galactosyl-lactose [Gal-beta(1 --> 6)

19 s (C1alpha, C1beta, C2, C3, C4, and C6) of a Glc-Pt.

20 to the severe mutant aba2; unexpectedly, ABA Glc ester was detected in aba2 seeds, suggesting the exi

21 on was introduced into HvSTP13 and abolished Glc uptake, whereas the V387L mutation reduced Glc uptak

22 a significant reduction in p-coumaric acid, Glc, Man, and cellulose contents.

23 ha) regulates stress tolerance by activating Glc-6-phosphate dehydrogenase (G6PD), which is essential

24 In addition, Glc modifies the chromatin landscape at thermomemory-rel

25 regulation of acetyl-CoA synthetase and ADP-Glc pyrophosphorylase, and increased stability of the PS

26 When expressed individually GlgC had ADP-Glc PPase activity, whereas GlgD was inactive.

27 Results indicate that S. mutans ADP-Glc PPase is an allosteric regulatory enzyme exhibiting

28 It is proposed that the accumulation of ADP-Glc in the ss3/ss4 mutant sequesters a large part of the

29 ucomutase (pgm1) or the small subunit of ADP-Glc pyrophosphorylase (aps1), largely restored photosynt

30 ecrease in the redox-activation state of ADP-Glc pyrophosphorylase and soluble starch synthase, which

31 ccompanied by a decrease in the level of ADP-Glc.

32 putative ADP-glucose pyrophosphorylases (ADP-Glc PPase), a key enzyme for glycogen synthesis in most

33 Restricting ADP-Glc synthesis, by introducing mutations in the plastidia

34 r starch deficient in all plant tissues (ADP-Glc-pyrophosphorylase [ADGase]) or retain starch accumul

35 ha-Fuc2(SO3)1-->3-alpha-Glc4(SO3)1-->3-alpha-Glc-->4-li nked to the central alpha-Glc units.

36 3-alpha-Glc-->4-li nked to the central alpha-Glc units.

37 and MCL promastigotes were alpha-Man, alpha-Glc, and alpha-Gal.

38 Using the bespoke substrate alpha-Glc-1,3-alpha-Man fluoride, the enzyme was shown to act

39 Both BR and Glc coregulate a large number of genes involved in abiot

40 terdependence/overlap occurs between BR- and Glc-regulated gene expression as well as physiological r

41 Experiments with (13)C6 labelled Gal and Glc showed that both monosaccharides act as acceptor sub

42 s indicated increased succinate, malate, and Glc-6-P and decreased Fru-1,6-bisphosphate, illustrating

43 ls lacking NR1D1, fails to inhibit PEPCK and Glc-6-Pase gene expression; and stimulates higher hepati

44 oA-IV suppresses the expression of PEPCK and Glc-6-Pase in hepatocytes; decreases hepatic glucose pro

45 te cross talk between the normal antagonists Glc and ethylene.

46 ontrolling R(N) Many amino acids, as well as Glc analogs, were found to potently inhibit the R(N) sti

47 Enzymatic cleavage of authentic Glc(1/3)Man(9)GlcNAc(2) yields Glc(1/3)-Man.

48 -Glc-(1->, with branches of short -> 3)-beta-Glc-(1 -> chains or single beta-Glc residues.

49 kali extracts consisted of acidic -> 6)-beta-Glc-(1->, with branches of short -> 3)-beta-Glc-(1 -> ch

50 rminal beta-glucosylserine residue, Ser(beta-Glc)NH2, a modification that has previously been shown t

51 t -> 3)-beta-Glc-(1 -> chains or single beta-Glc residues.

52 insulin-glucagon system for regulating blood Glc levels in animals.

53 e Fru, while under nitrogen starvation, both Glc and Fru, but not Suc, were less abundant.

54 BTA-Glc and BTA-Man are shown to assemble into micrometers l

55 thesized with mono- (BTA-beta-d-glucose; BTA-Glc and BTA-alpha-d-mannose; BTA-Man) or disaccharides (

56 used in a copolymerization approach with BTA-Glc, BTA-Man, or ethylene glycol BTA (BTA-OEG(4)) to giv

57 , C-GlcNAc Ser, has been prepared from the C-Glc Ser by a double inversion strategy using azide to in

58 The C-Glc Ser was available by a ring-closing metathesis and h

59 The alpha- and beta-anomers of the C1-Glc-Pt also differ significantly in their cellular uptak

60 ently compared to the others, whereas the C3-Glc-Pt (3) is taken up least efficiently.

61 lucose-1-phosphate cytidylyltransferase, CDP-Glc 4,6-dehydratase, NADH-dependent SAM:C-methyltransfer

62 For uptake into cells, Glc-Pt 1 exploits both glucose and organic cation transp

63 e phenotypes occur independently of cellular Glc signaling activities, we have tested whether HKL1 mi

64 onally designed glucose-platinum conjugates (Glc-Pts) were synthesized and their biological activitie

65 cal activity of glucose-platinum conjugates (Glc-Pts).

66 We previously reported that CSF114(Glc) detects diagnostic autoantibodies in multiple scler

67 4)-alpha-Neu5Ac-(2 -->)n and (--> 6)-alpha-D-Glc-(1 --> 4)-alpha-Neu5Ac-(2 -->)n, respectively.

68 pha-Neu5Ac-(2-->)n in NmW and (-->6)-alpha-d-Glc-(1-->4)-alpha-Neu5Ac-(2-->)n in NmY.

69 ccharide pathways instead require an alpha-d-Glc-beta-1,4-l-rhamnosyltransferase.

70 eu5Ac-(2 --> 3)]-beta-D-Gal-(1 --> 4)-beta-D-Glc-ceramide (GM1), and between a recombinant fragment o

71 hibits hepatic gluconeogenesis by decreasing Glc-6-Pase and PEPCK gene expression through NR1D1.

72 f the 8-O-methylated benzoxazinoids, DIM2BOA-Glc and HDM2BOA-Glc.

73 e start codon of Bx13 and lacks both DIM2BOA-Glc and HDM2BOA-Glc, and Il14H, which has an inactive Bx

74 thyltransferase (BX14) that converts DIM2BOA-Glc to HDM2BOA-Glc.

75 nd Oh43 revealed that the absence of DIM2BOA-Glc and HDM2BOA-Glc does not alter the constitutive accu

76 rformance increased, suggesting that DIM2BOA-Glc and/or HDM2BOA-Glc provide specific protection again

77 TRIMBOA-Glc is then converted to DIM2BOA-Glc by a previously described O-methyltransferase BX7.

78 yltransferases (Bx10a-c) that convert DIMBOA-Glc to HDMBOA-Glc.

79 thoxy-1,4-benzoxazin-3-one glucoside (DIMBOA-Glc).

80 on those with high HDMBOA-Glc and low DIMBOA-Glc.

81 X13) that catalyzes the conversion of DIMBOA-Glc into a new benzoxazinoid intermediate (TRIMBOA-Glc)

82 activity and the resulting decline of DIMBOA-Glc upon methylation to HDMBOA-Glc were associated with

83 -Glc was more toxic to R. maidis than DIMBOA-Glc in vitro, BX10c activity and the resulting decline o

84 The disaccharides Glc-(1-->3)-Hep4P Hep-(1-->3)-Hep4P and Hep-(1-->7)-Hep4

85 ected frame-shifted thiodisaccharide donors, Glc-GlcA and GlcA-Glc, were compared.

86 complete absence of structures for any EIIA(Glc)-transporter complexes.

87 ains lacking EI, Hpr, or the associated EIIA(Glc) protein produced less cholera toxin (CT) and had a

88 A crystal structure of Escherichia coli EIIA(Glc) in complex with the maltose transporter, an ATP-bin

89 mbrane anchor to increase the effective EIIA(Glc) concentration at the membrane.

90 How EIIA(Glc) is targeted to the membrane, how it interacts with

91 phosphotransferase system, enzyme IIA (EIIA(Glc)).

92 itory concentrations of the full-length EIIA(Glc) and an amino-terminal truncation mutant differ by 6

93 ds together with the N-terminal tail of EIIA(Glc) are essential for the high affinity binding of the

94 Only the unphosphorylated form of EIIA(Glc) bound to CsrD in vitro and was capable of activatin

95 ferred carbon source via the binding of EIIA(Glc) of the glucose transport system to the GGDEF-EAL do

96 l of how the central regulatory protein EIIA(Glc) allosterically inhibits maltose uptake in E. coli.

97 The signal-transducing protein EIIA(Glc) belongs to the phosphoenolpyruvate carbohydrate pho

98 In its dephosphorylated state, EIIA(Glc) is a negative regulator for several permeases, incl

99 ical cross-linking, we demonstrate that EIIA(Glc) binds to the MalK dimer, interacting with both the

100 ection of the ATPase cycle reveals that EIIA(Glc) does not affect the binding of ATP but rather inhib

101 Here, we show that EIIA(Glc) of the glucose-specific PTS system is also required

102 sponsible for cAMP generation, that the EIIA(Glc) component of glucose transport could enhance cAMP p

103 The structure shows that two EIIA(Glc) molecules bind to the cytoplasmic ATPase subunits,

104 It is known that unphosphorylated EIIA(Glc) binds to and inhibits a variety of transporters whe

105 main, and an important new way in which EIIA(Glc) shapes global regulatory circuitry in response to n

106 and the glucose-specific enzyme IIBC (EIIBC(Glc)) in defined media that lack PTS substrates.

107 present, the unphosphorylated form of EIIBC(Glc) sequesters Mlc to the cell membrane, preventing its

108 Our measurements indicated likely elevated Glc levels in hyphal tips during infection.

109 Finally, Glc-bleached SSB01 cells appeared unable to efficiently

110 Following Glc addition in the light, C. zofingiensis shuts off pho

111 in signaling mutants that were defective for Glc-induced LR production.

112 te pathway (OPPP) metabolism is required for Glc-mediated NITRATE TRANSPORTER2.1 (NRT2.1) expression.

113 are formed from acceptor reactions with free Glc and not by rearrangement of Glc in the active site.

114 Consequently, melanoidins formed from Glc/Ala contain more sugar degradation products with low

115 on of the biosynthetic pathways leading from Glc to anthocyanins.

116 re amino acid as compared to melanoidin from Glc/Ala and exhibit higher absorption in the UV/Vis.

117 arrangement in contrast to the reaction from Glc/Ala.

118 ucing 2.1 mm (640 mg L(-1)) salidroside from Glc in shake flasks, whereas an engineered C glutamicum

119 N-linked glycans are synthesized from Glc(3)Man(9)GlcNAc(2) precursors that are trimmed and mo

120 epI, whereas a mutant that expressed Gal-Gal-Glc-HepI fully resisted killing (>100% survival).

121 enotypes of the lactose-HepI and the Gal-Gal-Glc-HepI LOS structures were recapitulated with phase va

122 Despite lack of killing of the Gal-Gal-Glc-HepI mutants, mAb 2C7 deposited sufficient C3 on the

123 le lacto-N-neotetraose (LNnT; Gal-GlcNAc-Gal-Glc) moiety from heptose I (HepI) of the lipooligosaccha

124 Mutants that elaborated 4- (Gal-GlcNAc-Gal-Glc-HepI) and 5-glycan (GalNAc-Gal-GlcNAc-Gal-Glc-HepI)

125 lc-HepI) and 5-glycan (GalNAc-Gal-GlcNAc-Gal-Glc-HepI) structures displayed intermediate phenotypes (

126 2C7 of a mutant that expressed lactose (Gal-Glc) from HepI, whereas a mutant that expressed Gal-Gal-

127 Lactose (Gal-Glc) from HepII, although phase variable, is commonly ex

128 m sativum agglutinin, and the bacterial Gal-/Glc-binding protein from Escherichia coli, it became pos

129 flours were, on average, Rha:Ara:Xyl:Man:Gal:Glc:GalA in a 3:32:2:13:11:20:19 M ratio, with varying G

130 Our analyses indicate the GalNAc/Glc polymer and glycine are exported by the ExoA-I syste

131 ion, catalyzing the transfer of GlcNAc, Glc, Glc and GlcNAc residues to the protein backbone sequenti

132 ion, catalyzing the transfer of GlcNAc, Glc, Glc, and GlcNAc residues to the protein backbone sequent

133 d thiodisaccharide donors, Glc-GlcA and GlcA-Glc, were compared.

134 sylation, catalyzing the transfer of GlcNAc, Glc, Glc and GlcNAc residues to the protein backbone seq

135 sylation, catalyzing the transfer of GlcNAc, Glc, Glc, and GlcNAc residues to the protein backbone se

136 Glucose (Glc) plays a fundamental role in regulating lateral root

137 accumulates over 170 times more ADP-glucose (Glc) than wild-type plants.

138 ied the interactions of nitrate and glucose (Glc) on gene expression, nitrate transport, and growth u

139 Brassinosteroid (BR) and glucose (Glc) regulate many common responses in plants.

140 recorded by expressing a cytosolic glucose (Glc) Forster resonance energy transfer sensor.

141 idin formed at 160 degrees C from d-glucose (Glc) and l-alanine (Ala) as well as from fructosylalanin

142 standing performance for enzymeless glucose (Glc) sensing in alkaline media with high sensitivity (31

143 Using a library of fluorinated glucose (Glc), mannose (Man), and galactose (Gal) derived by syst

144 iana) Hexokinase-Like1 (HKL1) lacks glucose (Glc) phosphorylation activity and has been shown to act

145 We show that 1-4-linked glucose (Glc) is likely a minor component of the spore coat with

146 1 transported sucrose (Suc) but not glucose (Glc).

147 g of beta-(1-4)-linked backbones of glucose (Glc) and mannose (Man) units.

148 re hypersensitive to high levels of glucose (Glc) but responded normally to high salinity and osmotic

149 N, WbdO and WbdP) and they transfer glucose (Glc), L-fucose (L-Fuc) and N-acetylperosamine (PerNAc) o

150 cuole-located carrier, transporting glucose (Glc), fructose (Fru), and sucrose (Suc) after heterologo

151 nin, ethylene) and nutrient status (glucose [Glc], sucrose).

152 that the absence of DIM2BOA-Glc and HDM2BOA-Glc does not alter the constitutive accumulation or degl

153 Bx13 and lacks both DIM2BOA-Glc and HDM2BOA-Glc, and Il14H, which has an inactive Bx14 allele and la

154 ated benzoxazinoids, DIM2BOA-Glc and HDM2BOA-Glc.

155 as an inactive Bx14 allele and lacks HDM2BOA-Glc in leaves.

156 , suggesting that DIM2BOA-Glc and/or HDM2BOA-Glc provide specific protection against phloem feeding i

157 (BX14) that converts DIM2BOA-Glc to HDM2BOA-Glc.

158 Although HDMBOA-Glc was more toxic to R. maidis than DIMBOA-Glc in vitro

159 thoxy-1,4-benzoxazin-3-one glucoside (HDMBOA-Glc) and low levels of 2,4-dihydroxy-7-methoxy-1,4-benzo

160 root-derived benzoxazinoid glucosides HDMBOA-Glc and MBOA-Glc.

161 duced more progeny on those with high HDMBOA-Glc and low DIMBOA-Glc.

162 The larvae can hydrolyze HDMBOA-Glc, but not MBOA-Glc, to produce toxic MBOA upon predat

163 Variation in HDMBOA-Glc production was attributed to a natural CACTA family

164 tivates Bx10c in maize lines with low HDMBOA-Glc accumulation.

165 ine of DIMBOA-Glc upon methylation to HDMBOA-Glc were associated with reduced callose deposition as a

166 (Bx10a-c) that convert DIMBOA-Glc to HDMBOA-Glc.

167 While HDMBOA-Glc and MBOA reduce the growth and infectivity of both t

168 abi4, scr became much more tolerant of high Glc.

169 plates with 2% Glc plus ACC mimics the high-Glc effect in the HKL1 overexpression line but not in gi

170 IIA(Glc) binding to MelB(St) in the absence or presence of m

171 their thermodynamic response to binding IIA(Glc).

172 ity of LacY are subject to regulation by IIA(Glc) (inducer exclusion).

173 duced-fit mechanism that is inhibited by IIA(Glc) binding.

174 e mechanism of the inhibition of LacY by IIA(Glc) elucidated by ITC differs from the inhibition of me

175 By suppressing conformational dynamics, IIA(Glc) blocks inducer entry into cells and favors constitu

176 ; in addition, thermodynamic features of IIA(Glc) binding to other proteins are also unknown.

177 The phosphotransfer protein IIA(Glc) of the bacterial phosphoenolpyruvate:carbohydrate p

178 bacteria, the phosphotransferase protein IIA(Glc) plays a key regulatory role in catabolite repressio

179 The studies show that IIA(Glc) binds to LacY with a Kd of about 5 muM and a stoich

180 ine-specific cross-linking, we show that IIA(Glc) directly binds to MelB of Salmonella typhimurium (M

181 consistent with the interpretation that IIA(Glc) inhibits the induced fit process and restricts the

182 We further found that the IIA(Glc)-bound MelB(St) exhibits a decreased binding affinit

183 report the thermodynamic features of the IIA(Glc)-LacY interaction as measured by isothermal titratio

184 s one among several permeases subject to IIA(Glc) regulation.

185 Upon IIA(Glc) binding, the conformational entropy of LacY is rest

186 ts show that csdAB and csdCD are involved in Glc side-chain addition on the CWPS components rhamnan a

187 However, the altered carbon metabolism in Glc-supplemented cells was correlated with modest altera

188 hat HKL1 and HXK1 have differential roles in Glc-dependent repression of some ethylene biosynthesis g

189 f the models for some metabolites, including Glc-6-P, Fru-6-P, malate, fumarate, Xyl, and ribose.

190 sed exclusively of Man or also incorporating Glc.

191 Starch is a water-insoluble, Glc-based biopolymer that is used for energy storage and

192 een the glucose derivative-modified insulin (Glc-Insulin) and glucose transporters on erythrocytes (o

193 ferences in sugar accumulation, such as less Glc, Fru, and Suc at the end of the night.

194 ssigned as Man8 glycan, was found to be Man7+Glc glycan as its 1,3 branch containing three mannoses a

195 found that the newly identified "Man8" (Man7+Glc) was also present in different batches and in some c

196 MBOA-Glc is produced by D. virgifera through stabilization of

197 benzoxazinoid glucosides HDMBOA-Glc and MBOA-Glc.

198 of both the nematodes and the bacteria, MBOA-Glc repels infective juvenile nematodes.

199 arvae can hydrolyze HDMBOA-Glc, but not MBOA-Glc, to produce toxic MBOA upon predator attack.

200 pair the Muller cell's ability to metabolize Glc.

201 1brassinosteroid insensitive1 double mutant, Glc-induced LR production/emergence was severely reduced

202 antibodies specific to a gluco-asparagine (N-Glc) glycopeptide, CSF114(N-Glc), were identified in ser

203 co-asparagine (N-Glc) glycopeptide, CSF114(N-Glc), were identified in sera of an MS patient subpopula

204 expressing cell-surface adhesins including N-Glc, to establish a connection between H. influenzae inf

205 y editing 17 glycogenes, we discovered novel Glc(0-2)-Man(6)-GlcNAc(2)-type N-glycans, a novel HexNAc

206 cing 12-O-glucopyranosyl-jasmonic acid (12-O-Glc-JA) is still elusive.

207 paired growth, a decrease in the activity of Glc-6-P dehydrogenase, a decrease of the transcript abun

208 Analysis of Glc and BR sensitivity in mutants defective in auxin res

209 Application of Glc, Fru, or Suc, as well as cold, osmotic stress, or lo

210 STANT2,3 and SOLITARY ROOT act downstream of Glc and BR.

211 nosteroid (BR) signaling works downstream of Glc in controlling LR production/emergence in Arabidopsi

212 BR signaling works downstream of Glc signaling in regulating LR production, as in the glu

213 gar levels, points to a superior function of Glc and Suc for frost tolerance.

214 These findings reveal the novel function of Glc-regulated HLP1 in mediating thermotolerance/thermome

215 A unifying and hierarchical model of Glc and hormone signaling interplay is proposed.

216 Presence of Glc along with BR in medium could affect BR induction/re

217 HLP1 binds to the promoters of Glc-regulated HS-responsive genes and promotes chromatin

218 by both C60 and C70 columns in the range of Glc-1 to Glc-20 and high blood glucose level being retai

219 ns with free Glc and not by rearrangement of Glc in the active site.

220 The transcriptional regulation of Glc-6-Pase and phosphoenolpyruvate carboxykinase (PEPCK)

221 iporter GPT1 as the putative translocator of Glc-6-phosphate for starch biosynthesis in reproductive

222 with lactose plus the monosaccharides Gal or Glc resulted in altered GOS profiles.

223 sosteric alpha-d-glucopyranosyl 1-phosphate (Glc 1P) analogues have been synthesized.

224 sterically activated by glucose 6-phosphate (Glc-6-P) and adenosine monophosphate (AMP).

225 Physiologically, Glc and BR interact to regulate hypocotyl elongation gro

226 udies included incorporation of radiolabeled Glc, linkage analysis, and imaging of cellulose microfib

227 observations made in yeast, Lr67res reduced Glc uptake in planta These results confirm that the path

228 c uptake, whereas the V387L mutation reduced Glc uptake by ~ 50%.

229 Here, we describe reversible Glc-dependent repression/activation of oxygenic photosyn

230 o a rhodamine fluorophore, which affords RhB-Glc-Ent, it can selectively label Gram-negative bacteria

231 Our results suggest that the RhB-Glc-Ent probe is sensitive not only to the bacterial str

232 idespread occurrence of the glycan structure Glc(alpha1-2)Gal linked to hydroxylysine in animals, the

233 The results reveal that C2-substituted Glc-Pt 2 has the highest GLUT1-specific internalization,

234 The C1alpha- and C2-substituted Glc-Pts (1alpha and 2) accumulate in cancer cells most e

235 ng novel nuclear O-Fuc-type and cell surface Glc-Fuc-type O-glycans; and showed that they are importa

236 homologs with a >15-fold preference for TDP-Glc over UDP-Glc.

237 We also demonstrate that Glc along with heat could induce proliferation activity

238 In this study, we show that Glc has a prominent role in providing thermotolerance.

239 Functional enrichment analyses show that Glc represses photosynthetic pathways while ketocaroteno

240 We also show that Glc stimulates NRT2.1 protein levels and transport activ

241 Here we show that Glc supplementation of SSB01 cultures causes a loss of p

242 on is not influenced by gin2-1, showing that Glc does not influence NRT2.1 expression through nitrate

243 in response/signaling further suggested that Glc and BR signals may converge at S-phase kinase-associ

244 The Glc-Pts, 1-3, exhibit high levels of cytotoxicity toward

245 ly reduced numbers of emerged LRs at all the Glc concentrations tested.

246 nsitive1 (BRI1) is epistatic to HXK1, as the Glc insensitive2bri1-6 double mutant displayed severe de

247 bolism and translocation, and identified the Glc-6-phosphate/phosphate antiporter GPT1 as the putativ

248 Increasing light flux could also mimic the Glc effect on LR production/emergence.

249 transcription, suggesting convergence of the Glc and HS signaling pathways.

250 t of the spore coat with the majority of the Glc arising from contamination with extracellular polysa

251 discoveries revealing the importance of the Glc(3)Man(9)GlcNAc(2) C-branch in generating an ERAD sig

252 ocyclopropane-1-carboxylic acid (ACC) of the Glc-dependent developmental arrest of wild-type Arabidop

253 The GLUT1 specificity of the Glc-Pts was evaluated by determining the cellular uptake

254 target and cellular uptake mechanism of the Glc-Pts were elucidated.

255 No significant differences in uptake of the Glc-Pts were observed in non-cancerous RWPE2 cells.

256 RAD signal, the ebs3-1 mutation prevents the Glc(3)Man(9)GlcNAc(2) assembly and inhibits the ERAD of

257 d incorporating the SPE sensor for real-time Glc detection in human urine samples; the results obtain

258 lant sugar levels and loss of the fungal tip Glc gradient, supporting a tight link between fungal sug

259 C60 and C70 columns in the range of Glc-1 to Glc-20 and high blood glucose level being retained in gr

260 ruitment of NR1D1 and activity by apoA-IV to Glc-6-Pase promoter was verified with ChIP and a lucifer

261 transferase, transferring GlcNAc residues to Glc-Glc-GlcNAc-modified Fap1.

262 syltransferase, transferring Glc residues to Glc-GlcNAc-modified Fap1.

263 in controlling LR production in response to Glc and BR.

264 ed plants from detrimental HS in response to Glc is largely mediated by HSPs, but the mechanistic bas

265 those of scr, it was much less sensitive to Glc.

266 GT-D-type glycosyltransferase, transferring Glc residues to Glc-GlcNAc-modified Fap1.

267 TRIMBOA-Glc is then converted to DIM2BOA-Glc by a previously des

268 to a new benzoxazinoid intermediate (TRIMBOA-Glc) by an uncommon reaction involving a hydroxylation a

269 (1-->7)-Hep4P and the branched trisaccharide Glc-(1-->3)-[Hep-(1-->7)]-Hep4P, respectively, have been

270 Starch granules contain two Glc polymers, amylopectin and amylose.

271 UDP-Glc also stimulated keratinocyte migration, proliferatio

272 Specificity of binding to UDP and UDP-Glc indicates a properly folded protein, and binding kin

273 we observed a strong correlation between UDP-Glc concentration and the development of AKI in cardiac

274 cked the induction of HAS2 expression by UDP-Glc, the latter inhibitor suggesting that the signaling

275 ascent proteins and the glucose added by UDP-Glc:glycoprotein glucosyltransferase.

276 7GpppG-and 5 'metabolite' caps-NAD, FAD, UDP-Glc, UDP-GlcNAc, and dpCoA.

277 cap structures in humans and mice (FAD, UDP-Glc, UDP-GlcNAc, and m7Gpppm6A), cell- and tissue-specif

278 d (UDP-GlcA) and display specificity for UDP-Glc.

279 ate (GCP) was proposed to be formed from UDP-Glc breakdown and subsequently transferred, thus providi

280 in complex with natural donors UDP-Gal, UDP-Glc and, in an attempt to overcome one of the common pro

281 from keratinocytes and that UDP-glucose (UDP-Glc) added into keratinocyte cultures induced a specific

282 e the conversion of UDP-alpha-d-glucose (UDP-Glc) to the key metabolic precursor UDP-alpha-d-glucuron

283 f the P2Y14 receptor ligand UDP-glucose (UDP-Glc) was higher in urine samples from intensive care uni

284 can use both UDP-GlcUA and UDP-glucose (UDP-Glc), leading to the formation of glucuronide and glucos

285 samine (UDP-GlcNAc) but not UDP-glucose (UDP-Glc).

286 epimerization reactions of UDP-Gal into UDP-Glc and UDP-GalNAc into UDP-GlcNAc with the same level o

287 AD(+)/UDP, NAD(+)/UDP-GlcNAc, and NAD(+)/UDP-Glc.

288 c but allows a more optimal alignment of UDP-Glc for sugar donation.

289 NMR studies of UDP-Glc hydrolysis by yeast glycogen synthase were used to v

290 nity of UDP-GlcUA is higher than that of UDP-Glc.

291 h a >15-fold preference for TDP-Glc over UDP-Glc.

292 L-8 expression, supporting a notion that UDP-Glc signals for epidermal inflammation, enhanced hyaluro

293 Our study identifies the UDP-Glc/P2Y14 receptor axis as a potential target for the pr

294 aliana) lines carrying insertions in the UDP-Glc:sterol glucosyltransferase genes, UGT80A2 and UGT80B

295 ormations in the complex structures with UDP-Glc and UDP-GlcNAc.

296 n a 3:32:2:13:11:20:19 M ratio, with varying Glc:GalA ratios.

297 glucosylation of C(13)-apocarotenols, where Glc is bound either to the cyclohexene ring or the butan

298 ers coated with RBC membrane and loaded with Glc-Insulin.

299 ally or heterotrophically when supplied with Glc, a metabolite normally transferred from the alga to

300 ived from OPPP metabolism can, together with Glc, directly stimulate high levels of NRT2.1 expression

301 of authentic Glc(1/3)Man(9)GlcNAc(2) yields Glc(1/3)-Man.