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1 Goldman shows that such feedback is not necessary for me
2 Goldman-Rakic discovered the circuit basis for working m
5 ion, we distinguished area 6Ds of Preuss and Goldman-Rakic between PMV and PMDc, and a more posterior
6 In particular, we answer a question posed by Goldman and place the recent work of Bourgain, Gamburd,
9 a2+ channels and can be described by classic Goldman flux analysis with a Ca2+/Cs+ selectivity of the
10 etter estimate these localized ion currents, Goldman-Hodgkin-Katz (GHK) theory was used to model ion
11 global postmarketing surveillance databases, Goldman and colleagues report that progressive multifoca
12 tance (reversal, -95+/-4 mV) which displayed Goldman-Hodgkin-Katz outward rectification, supportive o
13 eurons and symmetrical chloride to eliminate Goldman rectification, which results in chloride flow in
14 ntial to extreme negative values, and follow Goldman-Hodgkin-Katz constant field equation at extreme
16 than the wild-type KASH5, based on the GES (Goldman-Engelman-Steitz) amino acid hydrophobicity scale
18 t al., 2009), which are reciprocally linked (Goldman-Rakic and Porrino, 1985; Xiao et al., 2009).
19 for channel selectivity by using a modified Goldman-Hodgkin-Katz (GHK) configuration that employs vo
20 is perspective, we summarize the findings of Goldman et al. and discuss the prospect that nanoRNA-med
21 hermore, this correlation was independent of Goldman rectification and persisted under altered chlori
22 ences (> 600 nucleotides), and the method of Goldman et al. for shorter sequences (< 600 nucleotides)
23 U and emergency department patients, and one Goldman's Class I unexpected finding in the ward patient
24 ies in nanopores often employs the Nernst or Goldman-Hodgkin-Katz (GHK) equation to interpret transme
26 tients were identified based on a pathologic Goldman-Witmer coefficient indicative of an underlying R
27 diac complications, according to the revised Goldman index, who were treated for at least 36 hrs in t
30 occurring model of enhanced S-cone syndrome, Goldman-Favre syndrome and clumped pigmentary retinopath
31 retina, a model of enhanced S-cone syndrome, Goldman-Favre syndrome, and clumped pigmentary retinopat
32 ng SPQ fluorescence quenching by I-) and the Goldman flux equation (for modeling the electrodiffusive
33 r clinical diagnoses were categorized by the Goldman method and compared with post mortem findings to
40 REV) followed closely the predictions of the Goldman-Hodgkin-Katz constant field equation at low conc
41 abilities were estimated on the basis of the Goldman-Hodgkin-Katz equation as follows: PH:PK:PNa = 40
42 or polarized epithelia the equivalent of the Goldman-Hodgkin-Katz equation for single isolated cells.
43 inations of Em by previous work based on the Goldman-Hodgkin-Katz equation and its derivatives and th
45 cardial infarction < or =7% according to the Goldman algorithm, no biochemical evidence of infarction
47 e model the device characteristics using the Goldman-Hodgkin-Katz (GHK) solution to the Nernst-Planck