ライフサイエンスコーパス: Golgi apparatus

1 trograde transport of endosomes to the trans-Golgi apparatus.

2 mutation, Kit localizes predominantly on the Golgi apparatus.

3 ifications that are expected to occur in the Golgi apparatus.

4 eavage of the gp160 precursor protein in the Golgi apparatus.

5 thway regulating precise localization of the Golgi apparatus.

6 subunit, which binds ganglioside GM1, to the Golgi apparatus.

7 ction in vesicle movement from the ER to the Golgi apparatus.

8 suggestive of the endoplasmic reticulum and Golgi apparatus.

9 eins that regulate CSC assembly in the plant Golgi apparatus.

10 rt between the endoplasmic reticulum and the Golgi apparatus.

11 s selective defects both in mitochondria and Golgi apparatus.

12 ains, while the LamG domain localized to the Golgi apparatus.

13 ibuted to a bypass or a hypo-function of the Golgi apparatus.

14 ls, coincident with the flagellar pocket and Golgi apparatus.

15 the endoplasmic reticulum and matures in the Golgi apparatus.

16 E1 interacts and colocalizes with APP in the Golgi apparatus.

17 ce of these connections linking WPBs and the Golgi apparatus.

18 x phase polysaccharides are assembled in the Golgi apparatus.

19 lycoproteins by sequestering them within the Golgi apparatus.

20 (G-CK), which is exclusively resident in the Golgi apparatus.

21 t (ERQC), and, surprisingly, recently to the Golgi apparatus.

22 VPS13B gene, which encodes a protein of the Golgi apparatus.

23 s from the endoplasmic reticulum (ER) to the Golgi apparatus.

24 sponse that in mammalian cells relies on the Golgi apparatus.

25 dantly in pectin methylesterification in the Golgi apparatus.

26 arked by Rab5, Rab7 and Rab9, as well as the Golgi apparatus.

27 GE synthase-1 (mPGES-1) but not COX-1 in the Golgi apparatus.

28 eases AAV2 transduction and transport to the Golgi apparatus.

29 IP probes, it triggered fragmentation of the Golgi apparatus.

30 ticipate in membrane-tethering events at the Golgi apparatus.

31 olved in heparan sulfate substitution in the Golgi apparatus.

32 downstream docking and fusion events at the Golgi apparatus.

33 autophagic response that required an intact Golgi apparatus.

34 he activity of glycosyltransferases from the Golgi apparatus.

35 nized interphase distribution of Mad1 at the Golgi apparatus.

36 tributions with varying association with the Golgi apparatus.

37 two distinct populations of vesicles at the Golgi apparatus.

38 rane--xylan is synthesized by enzymes in the Golgi apparatus.

39 e secretory pathways and was retained in the Golgi apparatus.

40 the organization of membrane traffic at the Golgi apparatus.

41 oteins from the endoplasmic reticulum to the Golgi apparatus.

42 nsferases and glycosyl hydrolases within the Golgi apparatus.

43 inds phosphatidylinositol 4-phosphate in the Golgi apparatus.

44 nsport from the endoplasmic reticulum to the Golgi apparatus.

45 needed for glycosylation in the lumen of the Golgi apparatus.

46 newly synthesized proteins and lipids to the Golgi apparatus.

47 s shuttled away from phagosomes and into the Golgi apparatus.

48 ed Ca(2+) transport pathway localized in the Golgi apparatus.

49 e core in the endoplasmic reticulum (ER) and Golgi apparatus.

50 is demonstrated here to be localized at the Golgi apparatus.

51 bidirectional traffic between the ER and the Golgi apparatus.

52 raffic from the endoplasmic reticulum to the Golgi apparatus.

53 etrograde route, from early endosomes to the Golgi apparatus.

54 s retention in the endoplasmic reticulum and Golgi apparatus.

55 ive uptake into the Endoplasmic Reticulum or Golgi apparatus.

56 as mixture of monomers and dimers within the Golgi apparatus.

57 membrane and proteins, bypassing the distant Golgi apparatus.

58 ism which alters its localization within the Golgi apparatus.

59 e from the endoplasmic reticulum (ER) to the Golgi apparatus.

60 roteins trafficking from the ER to the early Golgi apparatus.

61 asm, some of which contained remnants of the Golgi apparatus.

62 ression both glycoproteins were found in the Golgi apparatus.

63 inked glycans from further processing in the Golgi apparatus.

64 polarity as demonstrated by a non-polarized Golgi apparatus.

65 n accumulation of phosphorylated HER3 in the Golgi apparatus.

66 trafficking of ATF6alpha from the ER to the Golgi apparatus.

67 y to recycling endosomes, independent of the Golgi apparatus.

68 n the endoplasmic reticulum and COX-2 in the Golgi apparatus.

69 ates with the endoplasmic reticulum (ER) and Golgi apparatus.

70 assembly compartments near the disorganized Golgi apparatus.

71 l for MMP2 and MT1-MMP trafficking along the Golgi apparatus.

72 rect role in GPI anchor glycosylation in the Golgi apparatus.

73 n within the highly dynamic membranes of the Golgi apparatus.

74 d abnormal swelling and fragmentation of the Golgi apparatus.

75 g protein located at both the centrosome and Golgi apparatus.

76 -biological ingredients needed to generate a Golgi apparatus?

77 particles and disrupts the structure of the Golgi apparatus, a key cellular organelle involved in se

78 These activities are carried out by the Golgi apparatus, a membrane organelle in the center of t

79 Interestingly, the Golgi apparatus, an essential organelle for neurite grow

80 was due to their pronounced retention in the Golgi apparatus and also to their rapid internalization

81 smembrane proteins that are expressed in the Golgi apparatus and are able to homo-oligomerize.

82 ) to simultaneously localize proteins to the Golgi apparatus and assess their relative quantity.

83 ated vesicles mediate trafficking within the Golgi apparatus and between the Golgi and the endoplasmi

84 protein that resides on the cis face of the Golgi apparatus and binds alpha-SNAP-like proteins, but

85 pressed in HeLa cells, can both fragment the Golgi apparatus and block secretion, whereas viral infec

86 by STING required its relocalization to the Golgi apparatus and caused mitotic errors.

87 n to monitor the effects on structure of the Golgi apparatus and cell migration.

88 s of the cell extract, Endoplasmic Reticulum/Golgi apparatus and conditioned medium of T24 vs. its me

89 ng of proteins are critical functions of the Golgi apparatus and depend on its highly complex and com

90 that unlike oBST2A, oBST2B is limited to the Golgi apparatus and disrupts JSRV envelope (Env) traffic

91 ail to undergo proteolytic processing in the Golgi apparatus and drive IFN-gamma-induced gene express

92 lar locations including the plasma membrane, Golgi apparatus and endoplasmic reticulum.

93 vacuole, and Stv1p, which is targeted to the Golgi apparatus and endosomes.

94 the primary cell wall, is synthesized in the Golgi apparatus and exported to the cell wall in a highl

95 phosphatidylinositol 4-phosphate within the Golgi apparatus and failure to maintain residence of a m

96 ated vesicles mediate trafficking within the Golgi apparatus and from the Golgi to the endoplasmic re

97 causes an ERK-dependent fragmentation of the Golgi apparatus and inhibits Golgi polarization and dire

98 sferase (herein T1-T20) that localize to the Golgi apparatus and initiate O-glycosylation.

99 tially sorted into different vesicles in the Golgi apparatus and inserted into distinct domains of th

100 interacting (GORAB) protein localizes to the Golgi apparatus and interacts with the small GTPase RAB6

101 YIPF5 resides in the Golgi apparatus and is thought to play a critical role i

102 tibody, we localized RAB43 expression to the Golgi apparatus and LAMP1(-) cytoplasmic vesicles.

103 to an increase in cytoplasmic, cytoskeletal, Golgi apparatus and lipid metabolism proteins associated

104 This protein was shown to locate on the Golgi apparatus and mainly catalyze the Nt-acetylation o

105 with incorrectly positioned centrosomes and Golgi apparatus and mislocalized molecules of the slit d

106 clumping of mitochondria, disruption of the Golgi apparatus and missorting of synaptic proteins.

107 e findings suggest communication between the Golgi apparatus and mitochondria through homeostatically

108 ellular matrix, and for the formation of the Golgi apparatus and organization of F-actin bundles in m

109 dition, monensin rapidly induced swelling of Golgi apparatus and perturbed mitochondrial function.

110 a golgin that localizes predominantly at the Golgi apparatus and physically interacts with small guan

111 The same complexes were detected both in Golgi apparatus and plasma membrane by using FRET micros

112 Trypsin depleted PAR2-Kaede from the Golgi apparatus and repleted PAR2-Kaede at the plasma me

113 this organelle is strictly derived from the Golgi apparatus and revealing a novel function of mitoch

114 atrix polysaccharides are synthesized in the Golgi apparatus and secreted.

115 on of viral particles are transported to the Golgi apparatus and that Golgi apparatus disruption caus

116 (YFP) fusion of BdCSLF6 is localized to the Golgi apparatus and that the Golgi localization of BdCSL

117 ce images that resolve the highly convoluted Golgi apparatus and the close contacts between the endop

118 ed a higher level of colocalization with the Golgi apparatus and the endoplasmic reticulum (ER).

119 vesicles that transport contents between the Golgi apparatus and the endoplasmic reticulum, and they

120 retrograde trafficking of cargo between the Golgi apparatus and the endoplasmic reticulum, as well a

121 eukaryotes, occurs in the lumen of both the Golgi apparatus and the endoplasmic reticulum.

122 mes into retrieval pathways destined for the Golgi apparatus and the plasma membrane [1].

123 veal that the sorting of ion channels at the Golgi apparatus and their subsequent trafficking by uniq

124 ) complexes (CSCs) that are assembled in the Golgi apparatus and then delivered to the plasma membran

125 , which promptly accumulated in the cellular Golgi apparatus and then translocated to the extracellul

126 Sorting within the Golgi apparatus and trafficking along microtubules and t

127 predicts the size of experimentally measured Golgi apparatus and vacuole abundance fluctuations, sugg

128 Both Irgm1 isoforms localize to the Golgi apparatus and weakly to mitochondria; however, onl

129 g is found in the endoplasmic reticulum, the Golgi apparatus, and at the plasma membrane but not the

130 are localized in the endoplasmic reticulum, Golgi apparatus, and endosome.

131 t localizes to organelles, in particular the Golgi apparatus, and has a preference for acetylating N

132 somes, well-developed endoplasmic reticulum, Golgi apparatus, and indented heterochromatin.

133 se involved in acidic amino acid metabolism, Golgi apparatus, and ion and phospholipid transport.

134 I) vesicles, maintenance and function of the Golgi apparatus, and mitochondria migration and position

135 GH-C53S was exclusively stained in the Golgi apparatus, and no secretory granules formed for th

136 icking from the endoplasmic reticulum to the Golgi apparatus, and the nuclear pore complex (NPC), whi

137 ergo also prevents export from the ER to the Golgi apparatus, and this traffic block results in break

138 surface of mitochondria, peroxisomes, or the Golgi apparatus, and thus prevention of plasma membrane

139 ulum (ER)-to-Golgi intermediate compartment, Golgi apparatus, and trans-Golgi network form a ring tha

140 All three transporters localize to the Golgi apparatus, and UXT1 also localizes to the endoplas

141 heir disruption of endoplasmic reticulum and Golgi apparatus architecture.

142 Taking the Golgi apparatus as a biological example, three different

143 at in plants MNS3 exclusively resides in the Golgi apparatus at steady-state.

144 intained by two factors that localize to the Golgi apparatus, ATP7 and the conserved oligomeric Golgi

145 ligases localize to the nucleus, centrosome, Golgi apparatus, axon and dendrite cytoskeleton, and syn

146 plasmic tail that confers retrieval from the Golgi apparatus back to the ER.

147 GfsA was found to be localized at the Golgi apparatus based on cellular fractionation experime

148 Despite the Golgi apparatus being the major protein processing and s

149 interactions as well as repositioning of the Golgi apparatus, both of which can be controlled by the

150 important AGP glycans are synthesized in the Golgi apparatus, but the relationships among their glyco

151 t enables prolonged live-cell imaging of the Golgi apparatus by 3D confocal and STED microscopy.

152 abidopsis thaliana, is biosynthesized in the Golgi apparatus by a series of glycan synthases and glyc

153 rmore, FAM21 prevents cargo transport to the Golgi apparatus by controlling levels of phosphatidylino

154 dified in the endoplasmic reticulum (ER) and Golgi apparatus by glycoside hydrolases and glycosyltran

155 alactose, as UDP-Gal, are delivered into the Golgi apparatus by SLC35A3 and SLC35A2 transporters, res

156 on of proteins and lipids takes place in the Golgi apparatus by the consecutive actions of functional

157 The Golgi apparatus consists of stacked cisternae filled wit

158 toward the trans-Golgi network (TGN) and the Golgi apparatus correlates with transduction efficiency

159 PR:MHC class I complex trafficks through the Golgi apparatus, demonstrating a function of TAPBPR beyo

160 nzymes within successive compartments of the Golgi apparatus determine where new monomer building blo

161 ation of Gbetagamma, a PKD activator, to the Golgi apparatus, determined by bioluminescence resonance

162 2014) show that the size and topology of the Golgi apparatus determines the size and functionality of

163 both cells whereas the disintegration of the Golgi apparatus did not affect the process.

164 of cells with HRV16 also caused significant Golgi apparatus dispersal; however, this did not result

165 transported to the Golgi apparatus and that Golgi apparatus disruption caused by the drug brefeldin

166 udies; consequently, to address the issue of Golgi apparatus disruption for HRV16, we have systematic

167 3A protein from HRV14 or HRV2 did not cause Golgi apparatus disruption or a block in secretion, wher

168 that infection of cells with HRV1A did cause Golgi apparatus disruption which was replicated by the e

169 ture, cellular location, and function of the Golgi apparatus during male germ cell differentiation is

170 The role of Golgi apparatus during phagocytic uptake by macrophages

171 on, we examined mitochondria, actin, and the Golgi apparatus dynamics in three dimensions.

172 , as exemplified by the fragmentation of the Golgi apparatus (effector function).

173 ding localizers for mitochondria, lysosomes, Golgi apparatus, endoplasmic reticulum, and plasma membr

174 membrane to the endoplasmic reticulum (ER), Golgi apparatus, endosomes, and cytosol.

175 the functional relevance of transport to the Golgi apparatus for AAV transduction remains to be estab

176 identified as a perinuclear substrate in the Golgi apparatus for mAKAP-scaffolded PLCepsilon.

177 atively examined the effect of HRV16 on both Golgi apparatus fragmentation and protein secretion in H

178 ion of diverse molecular pathways, including Golgi apparatus fragmentation, as well as extensive post

179 ll polysaccharides are biosynthesized in the Golgi apparatus from cytosolic-derived nucleotide sugars

180 ng proteins to the endoplasmic reticulum and Golgi apparatus from membrane-bound ribosomes were not t

181 ell wall polysaccharides are produced in the Golgi apparatus from nucleotide sugars that are predomin

182 racellular Ca(2+) and Mn(2+) influx into the Golgi apparatus from the cytosol.

183 Abnormalities in the Golgi apparatus function are important to the developmen

184 her inhibitors of endoplasmic reticulum (ER)/Golgi apparatus function: brefeldin A and monensin.

185 The Golgi apparatus (GA) is an important site of insulin pro

186 ndritic translation, most dendrites lack the Golgi apparatus (GA), an essential organelle for convent

187 n, where it co-localized with markers of the Golgi apparatus (GA).

188 is in the endoplasmic reticulum (ER) and the Golgi apparatus (GA).

189 The unique stacked morphology of the Golgi apparatus had been a topic of intense investigatio

190 gent, NBD C(6) -ceramide, NIR imaging in the Golgi apparatus has been demonstrated using these NIR em

191 s-respectively involving proteins related to Golgi apparatus, hemoglobin complex, and endoplasmic ret

192 interference with endosome formation or the Golgi apparatus impairs migration to a similar extent as

193 PAT4 is predominantly associated with the Golgi apparatus in a range of cell types, and in situ pr

194 ate that Gc is significantly enriched in the Golgi apparatus in absence of other viral components, wh

195 , we provide evidence for a dual role of the Golgi apparatus in controlling the size of these secreto

196 ns between the endoplasmic reticulum and the Golgi apparatus in eukaryotic cells.

197 s produced by the enzymes encountered in the Golgi Apparatus in human cell expression systems.

198 This ensures proper functioning of the Golgi apparatus in protein trafficking and processing.

199 nerate diacylglycerol (DAG) from PI4P in the Golgi apparatus, in close proximity to the nuclear envel

200 es of posttranslational modifications in the Golgi apparatus, including attachment of carbohydrate mo

201 m (without endoplasmic reticulum stress) and Golgi apparatus, increased vesicular trafficking, and a

202 However, invasive pathogens can disrupt the Golgi apparatus, interfering with secretion and compromi

203 In mammalian cells, the inheritance of the Golgi apparatus into the daughter cells during each cycl

204 taxin 5-mediated retrograde transport to the Golgi apparatus is a broadly conserved feature of AAV tr

205 The Golgi apparatus is a membrane-bound organelle that serve

206 The Golgi apparatus is a multicompartment central sorting st

207 However, whether and how dispersal of the Golgi apparatus is actively regulated under stress, and

208 on of the Arabidopsis (Arabidopsis thaliana) Golgi apparatus is currently reasonably well documented;

209 The Golgi apparatus is increasingly recognized as a major hu

210 s between the endoplasmic reticulum (ER) and Golgi apparatus is indispensable for eukaryotic cell fun

211 of these proteins and their products in the Golgi apparatus is largely unknown.

212 been well documented, its consequence on the Golgi apparatus is less well understood.

213 at in SCYL1-deficient human fibroblasts, the Golgi apparatus is massively enlarged, which is in line

214 Synthesis of CS and DS in the Golgi apparatus is mediated by sulfotransferases that mo

215 The Golgi apparatus is optimized separately in different tis

216 The Golgi apparatus is the central hub of intracellular traf

217 The plant endoplasmic reticulum-Golgi apparatus is the site of synthesis, assembly, and

218 ss through the endoplasmic reticulum and the Golgi apparatus - is one of the most frequent protein mo

219 erized regulator of vesicle formation at the Golgi apparatus, is also a component of the synaptic rib

220 involves fragmentation and dispersal of the Golgi apparatus, known as the Golgi-dispersal response (

221 mbrane trafficking between endosomes and the Golgi apparatus lead to neurodegenerative diseases.

222 oteins from the endoplasmic reticulum to the Golgi apparatus, leading to the lysosomal degradation of

223 unique view on the organization of the plant Golgi apparatus, leading toward unique hypotheses center

224 P and cathepsin B was observed in a distinct Golgi apparatus-like pattern, which required a 1-h OA tr

225 At the Golgi apparatus, lipid-synthesizing, lipid-flippase, and

226 s attached to MTs is observed, including the Golgi apparatus, lytic granules, and mitochondria.

227 mechanisms of UDP-GlcNAc transport into the Golgi apparatus may exist.

228 the mode of cytosolic Naa60 anchoring to the Golgi apparatus, most likely occurring post-translationa

229 nuclear envelope, endoplasmic reticulum and Golgi apparatus, must be disassembled or remodelled, dis

230 Microtubules and the Golgi apparatus of entotic cells provided membrane expan

231 b) induction of morphological changes of the Golgi apparatus of plant and mammalian cells, and (c) in

232 affics through the endoplasmic reticulum and Golgi apparatus on its way to the cell membrane and is h

233 uires mobilization of intact PAR(2) from the Golgi apparatus or de novo synthesis of new receptors by

234 structures colocalize with Rab5 but not with Golgi apparatus or endoplasmic reticulum markers.

235 embers of the Arabidopsis GT14 family to the Golgi apparatus or the endoplasmic reticulum (ER).

236 The Golgi apparatus performs crucial functions in the sortin

237 r, localize to endoplasmic reticulum and the Golgi apparatus, presumably through the recycling endoso

238 in plant tissues and is localized to the ER, Golgi apparatus, prevacuolar compartment, and plasma mem

239 is traffic block results in breakdown of the Golgi apparatus, primarily due to maintenance of the con

240 ortholog of the gene encoding human STK16, a Golgi apparatus protein kinase with undefined function.

241 In filamentous fungi, organization of the Golgi apparatus reflects the unique challenges brought a

242 ve shown that binding of UL20 to GODZ in the Golgi apparatus regulates trafficking of UL20 and its su

243 lic surface of Golgi membranes to facilitate Golgi Apparatus-Related Degradation (GARD) and degradati

244 red Golgi morphology and fewer cisternae per Golgi apparatus relative to wild-type cells, indicative

245 ics of membrane-bound organelles such as the Golgi apparatus, remain elusive.

246 th and disease, their trafficking across the Golgi apparatus remains poorly understood.

247 point that a spatially organized and dynamic Golgi apparatus represents an adaptation that is as impo

248 se genes, TbGT11, and show that it encodes a Golgi apparatus resident UDP-GlcNAc:alpha3-D-mannoside b

249 e COPI complex, which regulates transport of Golgi apparatus-resident cargos.

250 tion of the spatial separation of the ER and Golgi apparatus restored cleavage of ATF6alpha in the pr

251 These results suggest that the Golgi apparatus serves as a platform for oncogenic Kit s

252 However, electron micrographs of WPBs at the Golgi apparatus show that these forming WPBs contain ver

253 in RAB7L1 or LRRK2 led to endolysosomal and Golgi apparatus sorting defects and deficiency of the VP

254 UL20 binds to GODZ (also known as DHHC3), a Golgi apparatus-specific Asp-His-His-Cys (DHHC) zinc fin

255 ly to GODZ (also known as DHHC3), a cellular Golgi apparatus-specific Asp-His-His-Cys (DHHC) zinc fin

256 trafficking route differs from that of known Golgi apparatus-targeted cargos, and we raise the possib

257 been described to cause fragmentation of the Golgi apparatus that blocks the secretory pathway.

258 phosphatase of the endoplasmic reticulum and Golgi apparatus that controls organelle membrane composi

259 er variable delay times, are captured by the Golgi apparatus that is reached either by random diffusi

260 of onset and evolution of PNG components in Golgi apparatus that shaped diversity of eukaryotic prot

261 anslocation of the SCAP-SREBP complex to the Golgi apparatus, the activation of SREBP proteins (SREBP

262 organelles (early endosomes, lysosomes, the Golgi apparatus, the endoplasmic reticulum or the nucleu

263 , from the endoplasmic reticulum (ER) to the Golgi apparatus, thereby triggering their secretion by e

264 me of the GmExo70J proteins are localized to Golgi apparatus through a novel N-terminal transmembrane

265 ng WPBs maintain multiple connections to the Golgi apparatus throughout their biogenesis.

266 al stages of membrane trafficking, including Golgi apparatus to endosome and vacuole, peroxisomal fis

267 o form a multiprotein complex that links the Golgi apparatus to F-actin, which regulates muscle integ

268 Notch1 is first cleaved by furin in the Golgi apparatus to produce the biologically active heter

269 secretory vesicles are transported from the Golgi apparatus to the cell periphery by kinesin-1 KIF5B

270 is essential in vesicle trafficking from the Golgi apparatus to the endoplasmic reticulum (ER) throug

271 e subcellular localization of VAMP4 from the Golgi apparatus to the ER.

272 f VEGFR2 due to trafficking defects from the Golgi apparatus to the plasma membrane.

273 s to examine receptor translocation from the Golgi apparatus to the plasma membrane.

274 holera toxin from the plasma membrane to the Golgi apparatus, Tpcn2(-/-) MEFs show slower kinetics of

275 at label the nucleus, endoplasmic reticulum, Golgi apparatus, trans-Golgi network, plasma membrane, a

276 ilomycin A1 emphasizes the importance of the Golgi apparatus/trans-Golgi network as a platform in the

277 The Golgi apparatus undergoes a ubiquitin-dependent disassem

278 vior and the spatio-temporal distribution of Golgi apparatus upon release.

279 lear space, and in the cytoplasm adjacent to Golgi apparatus vesicles.

280 conserved oligomeric Golgi (COG) complex, a Golgi apparatus vesicular tether.

281 cytoplasmic domain targets receptors to the Golgi apparatus, vesicular structures, and the cell surf

282 tween the endoplasmic reticulum (ER) and the Golgi apparatus via Coat Protein I (COPI)- and COPII-coa

283 fficked between the endosomal system and the Golgi apparatus via multiple pathways and provides evide

284 ganelle fractionation approach targeting the Golgi apparatus was combined with a label-free quantitat

285 egrees C, permissive conditions, through the Golgi apparatus was locally delayed, almost tenfold, bet

286 e neurogenic zone were eliminated, a compact Golgi apparatus was positioned exclusively at the base o

287 ated with the endoplasmic reticulum (ER) and Golgi apparatus, we report that MxA formed membraneless

288 ugars from the cytosol into the lumen of the Golgi apparatus where glycosyltransferases use them for

289 sol, it is transported into the lumen of the Golgi apparatus, where it is converted to UDP-galacturon

290 localized to the soma, neurites, and to the Golgi apparatus, where it is prominently expressed.

291 on of this polytopic membrane protein is the Golgi apparatus, where its accumulation is strictly regu

292 and 2-furoyl-LIGRLO-NH2 activated PKD in the Golgi apparatus, where PKD regulates protein trafficking

293 LR and PDGFRbeta are then transported to the Golgi apparatus, where those complexes trigger Galphai3-

294 tUXS1, AtUXS2, and AtUXS4 are located in the Golgi apparatus whereas AtUXS3, AtUXS5, and AtUXS6 are l

295 in cell lines, mutant Kit accumulates on the Golgi apparatus, whereas normal Kit localizes to the pla

296 onsistent with virion assembly in the medial Golgi apparatus, whereas oligomannose-type glycans requi

297 All enzymes were localized in the plant Golgi apparatus, which allowed us to identify the SmFucT

298 Isoforms Tm1J and Tm2A colocalize around the Golgi apparatus with the formin-family protein Diaphanou

299 Within cells, SLC9A7 resides in the Golgi apparatus, with prominent enrichment in the trans-

300 etion, whereas viral infection fragments the Golgi apparatus without blocking secretion.