ライフサイエンスコーパス: Golgi

1 Golgi mannosidase II (GMII) catalyzes the sequential hyd

2 Golgi outposts can nucleate new microtubules in speciali

3 Golgi outposts have also been implicated in the patholog

4 Golgi studies have been used to parse this region into t

5 s widely accepted that FAM20C functions as a Golgi casein kinase and has large numbers of kinase subs

6 ain protein Neurobeachin-like (NBEAL) 1 as a Golgi- associated protein required for regulation of cho

7 dipoQ receptor family member IV (PAQR4) as a Golgi-localized ceramidase.

8 This U21-containing carrier also carries a Golgi membrane protein engineered to form inducible olig

9 conserved oligomeric Golgi (COG) complex, a Golgi apparatus vesicular tether.

10 hosphatidylinositol 4-kinase beta (PI4KB), a Golgi-associated lipid kinase.

11 t a fraction of hnRNPA2B1 colocalized with a Golgi marker.

12 mary visual cortices and investigated with a Golgi stain and computer-assisted morphometry to provide

13 By co-staining PC-3 cells with a Golgi staining reagent, NBD C(6) -ceramide, NIR imaging

14 Rab6, the most abundant Golgi associated small GTPase, consists of 2 equally com

15 to form trans-oligomers that tether adjacent Golgi membranes into stacks and ribbons in mammalian cel

16 tional link between proteostasis control and Golgi architecture, which may be critical in various sec

17 ing dynamin, clathrin, sorting endosomes and Golgi trafficking where the cargo is released intracellu

18 dified in the endoplasmic reticulum (ER) and Golgi apparatus by glycoside hydrolases and glycosyltran

19 Both the endoplasmic reticulum (ER) and Golgi complex participated in delivering (D)CDX-modified

20 sma membrane, endoplasmic reticulum (ER) and Golgi.

21 nosomes, the endoplasmic reticulum (ER), and Golgi complex.

22 that the transport machinery between LE and Golgi facilitates PS-ASO release.

23 el role for CDR1 in promoting metastasis and Golgi trafficking.

24 tes lung squamous migration, metastasis, and Golgi trafficking through its complimentary transcript,

25 athways: anterograde traffic, recycling, and Golgi integrity.

26 s retention in the endoplasmic reticulum and Golgi apparatus.

27 m (associated with endoplasmic reticulum and Golgi proteins).

28 osomes, lysosomes, endoplasmic reticulum and Golgi.

29 The transport and Golgi organization 1 (TANGO1) proteins play pivotal role

30 Dendritic arborization (Golgi Sholl analyses), spine morphology, and MSN excitat

31 -acetylglucosaminyltransferases (B3GNTs) are Golgi-resident glycosyltransferases involved in the bios

32 ity 20, member C (Fam20C), formerly known as Golgi casein kinase (G-CK), which is exclusively residen

33 These findings establish PAQR4 as Golgi-localized ceramidase required for cellular growth

34 at are important for viral assembly, such as Golgi- and recycling endosome-derived membranes.

35 o trafficking by serving as a bridge between Golgi-bound golgin-97/245 and the WASH/FAM21 complex on

36 not only reinforce the existing link between Golgi fragmentation and neurodegeneration but also demon

37 Both Golgi and nuclear envelope exhibit MTOC activity utilizi

38 ucin domains that are diversely decorated by Golgi glycosyltransferases to become extended rodlike st

39 In infected DeltahnRNPA2B1 cells, Golgi-dependent transport of virus from the apical surfa

40 etained in the endoplasmic reticulum and cis-Golgi, but not in early endosomes, suggesting the nonsen

41 lia as a model and show that a conserved cis-Golgi membrane protein eas-1/GOLT1B negatively regulates

42 peptide precursors are processed in the cis-Golgi by the subtilase SBT6.1.

43 phology and disappearance of OSBP at the cis-Golgi compartment, together with a complete loss of GOLP

44 rly developmental stages, rnf-145 in the cis-Golgi network inhibits sbp-1 activation to promote the g

45 Here we report that the cis-Golgi protein nucleobindin-1 (NUCB1) is critical for MMP

46 on of fluorescently tagged proANP in the cis-Golgi region.

47 sicle-mediated recycling of PAM from the cis-Golgi to the endoplasmic reticulum plays an essential ro

48 oteins with a KDEL-like peptide from the cis-Golgi to the ER through COPI retrograde transport.

49 -dependent shuttling of rnf-145 from the cis-Golgi to the trans-Golgi network to stop glial growth.

50 However, classical Golgi staining studies did not find interneurons with lo

51 ce images that resolve the highly convoluted Golgi apparatus and the close contacts between the endop

52 SCs of mutant mice showed a distorted Golgi morphology and disappearance of OSBP at the cis-Go

53 First, at what point during Golgi maturation does a biosynthetic vacuolar cargo depa

54 roteins trafficking from the ER to the early Golgi apparatus.

55 h necessary and sufficient for the efficient Golgi export of Tac chimeras.

56 etastasis and demonstrated that they enhance Golgi-mediated function and secretion.

57 th a complete loss of GOLPH3 from the entire Golgi.

58 ER lumen and recruits membranes from the ER Golgi intermediate compartment to create an exit route f

59 ng/sorting in the Endoplasmic Reticulum (ER)-Golgi in a temporal order consistent with the progressiv

60 in plant tissues and is localized to the ER, Golgi apparatus, prevacuolar compartment, and plasma mem

61 g showed that orexin is localized in the ER, Golgi, and in the lysosomes in LMH cells.

62 ER lumen to COPII machinery, tethers, and ER-Golgi intermediate compartment (ERGIC) in the cytoplasm

63 interactions with components required for ER-Golgi trafficking, coincident with reduced levels of the

64 hese data highlight the importance of the ER-Golgi axis in the control of autoinflammation and inform

65 mic reticulum (ER) membrane away from the ER-Golgi interface.

66 te bidirectional transport portals at the ER-Golgi interface.

67 teins can directly engage with DR5 in the ER-Golgi intermediate compartment, where DR5 assembles pro-

68 d KDELR2 are known to be localized to the ER-Golgi pathway, we studied their common mechanism of driv

69 le contacts, and to reconstitute OSBP-VAP ER-Golgi tethering implicated in phosphatidylinositol-4-pho

70 served in vitro), while competing for the ER/Golgi/PM pathway.

71 of the endoplasmic reticulum (ER) and ERGIC/Golgi (Raote et al., 2018).

72 ky secretory cargos from the ER to the ERGIC/Golgi via a tunnel.

73 traffics following stimulation to the ERGIC/Golgi, where signaling occurs.

74 lic surface of Golgi membranes to facilitate Golgi Apparatus-Related Degradation (GARD) and degradati

75 97/VCP and 26S proteasomes, and required for Golgi dispersal.

76 tor that retrieves ER resident proteins from Golgi compartments in a pH-dependent manner.

77 entification of pathogenic variants in GBF1 (Golgi brefeldin A-resistant guanine nucleotide exchange

78 O- and N-glycans might function as a generic Golgi export signal at the trans-Golgi to promote the co

79 through vesicular carriers that recruit GGA (Golgi-localized, gamma-ear-containing, ARF-binding prote

80 n leads to an accumulation of Arm with GM130 Golgi marker in Klp64D knockdown.

81 een and the mechanistic validation highlight Golgi function as one of the key cellular features alter

82 we demonstrated a critical role for the host Golgi compartment-resident ATP-powered calcium pump (sec

83 mutations within the gene encoding the human Golgi TMEM165 (transmembrane protein 165), belonging to

84 ced ACh release is to strongly hyperpolarize Golgi cells.

85 After SREBP2 is cleaved in Golgi, its CTD remains bound to Scap and returns to the

86 onsistent with the described role of GBF1 in Golgi function and maintenance, we observed marked incre

87 de Golgi trafficking, induces an increase in Golgi cisternal number in HeLa cells and delays the cell

88 maintenance, we observed marked increase in Golgi fragmentation in primary fibroblasts derived from

89 onent of the Syntaxin 18 complex involved in Golgi-to-ER trafficking, as a novel NMD factor.

90 Perturbations in Golgi structure are associated with numerous disorders f

91 t MPP5a interacts with small GTPase Rab11 in Golgi to transport cadherin and Crumbs components synerg

92 AGUK Scaffold Protein 3 (DLG3), resulting in Golgi complex fragmentation, and reduced protein glycosy

93 iew, we summarize their established roles in Golgi structure formation and function under physiologic

94 ts topics of functional relevance, including Golgi outpost heterogeneity, formation and transport, as

95 C is independent of endocytosis or an intact Golgi, but depends on Ltc1, a LAM/StARkin-family protein

96 impaired, although UDP-GlcNAc transport into Golgi vesicles was not decreased.

97 the endoplasmic reticulum, as well as intra-Golgi transport.

98 Similar intra-Golgi transport delays were seen at 37 degrees C with RU

99 ding localizers for mitochondria, lysosomes, Golgi apparatus, endoplasmic reticulum, and plasma membr

100 etastasis by activating ISGs and maintaining Golgi complex integrity.

101 COPI vesicles mediate Golgi-to-ER recycling, but COPI vesicle arrival sites at

102 ceptor resulting in impaired KDELR2-mediated Golgi-ER transport.

103 se involved in acidic amino acid metabolism, Golgi apparatus, and ion and phospholipid transport.

104 itioning promiscuous enzymes across multiple Golgi compartments.

105 Remarkably, there have been no Golgi studies of these neurons in nonhuman primates.

106 residence times, we found that the observed Golgi localization of O-glycan-deficient cargos is due t

107 Genetic or pharmacologic activation of Golgi complex fragmentation blocks PDAC growth and metas

108 ures with drugs that block the activation of Golgi-localized Arf proteins and COPI vesicle formation,

109 o longer promoted migration upon blockade of Golgi trafficking.

110 ulated under stress, and the consequences of Golgi dispersal, remain unknown.

111 cellular functions, ranging from control of Golgi structure, fly fertility, and Akt signaling.

112 EM165 expression in a yeast strain devoid of Golgi Ca(2+) and Mn(2+) transporters abrogates Ca(2+)- a

113 ced type I IFN signaling due to a failure of Golgi-to-ER STING retrieval.

114 We conclude that the integrity of Golgi-dependent copper homeostasis mechanisms, requiring

115 n together, this work reveals a mechanism of Golgi-localized proteasomal degradation, providing a fun

116 have employed the old but powerful method of Golgi-impregnation to rabbit retina, studying the range

117 s best with a cisternal progression model of Golgi function.

118 Finally, we show that perturbation of Golgi homeostasis induces cell death of multiple myeloma

119 s correlate strongly with a proliferation of Golgi cisternae observed in earlier electron microscopy.

120 Here, we studied the role of Golgi in myelination of peripheral nerves in mice throug

121 Here, we examined the role of Golgi-endosome transport, specifically M6PR shuttling me

122 rotein of 25 kDa) is mediated by a subset of Golgi zinc finger DHHC-type palmitoyltransferase (zDHHC)

123 are associated with the cytosolic surface of Golgi membranes to facilitate Golgi Apparatus-Related De

124 e COPI complex, which regulates transport of Golgi apparatus-resident cargos.

125 vidence for the existence of a third type of Golgi-derived carrier that is vesicular, yet clathrin in

126 ATP7 paralogs with the conserved oligomeric Golgi (COG) complex, a Golgi apparatus vesicular tether.

127 apparatus, ATP7 and the conserved oligomeric Golgi (COG) complex.

128 Quantification of spine-density on Golgi-stained BLA principal neurons showed that the same

129 1, we demonstrated that O-glycan's effect on Golgi export is probably additive.

130 isingly, despite these consistent effects on Golgi cells and mossy fibers, ACh can either increase or

131 Fluorescence dialysis or Golgi-impregnation labeling showed reduced dendritic spi

132 educed the number of cisternal membranes per Golgi stack, suggesting a loss of trans-Golgi cisternae.

133 All enzymes were localized in the plant Golgi apparatus, which allowed us to identify the SmFucT

134 nophosphorylated PPIn species within the PM, Golgi complex, and endosomal compartments.

135 in the ER to proceed through Golgi and post-Golgi trafficking.

136 Perturbation of BACE1 post-Golgi trafficking results in an increase in BACE1 cleava

137 As a consequence, post-Golgi trafficking, membrane cholesterol levels, and PI(4

138 s at the TGN, as well as Rab8-dependent post-Golgi trafficking of different classes of apical membran

139 ation by subtilases including SBT3.8 in post-Golgi compartments depends on the N-terminal aspartate o

140 Our results reveal that maturation of post-Golgi-derived SGs requires trafficking via the endosomal

141 Tissue was stained with a modified rapid Golgi technique.

142 We performed rapid Golgi staining on the layer IV and V pyramidal neurons o

143 fect localization or function of the related Golgi-associated retrograde protein (GARP) complex.

144 ently of the classical endoplasmic reticulum-Golgi exocytic route.

145 ilayers that mimic the endoplasmic reticulum-Golgi intermediate compartment (ERGIC) membrane, ETM for

146 Rab6 loss-of-function inhibits retrograde Golgi trafficking, induces an increase in Golgi cisterna

147 Since Golgi outposts are relatively understudied, many outstan

148 glycan-deficient cargos is due to their slow Golgi export.

149 ulin localises asymmetrically to the somatic Golgi within Drosophila neurons.

150 ACh application can modestly depolarize some Golgi cells, the net effect of longer, optogenetically i

151 Here, we identify the germ cell specific Golgi glycoprotein MGAT4D as a protector of male germ ce

152 SP55 (GORASP2) proteins function in stacking Golgi cisternae.

153 ation 8a, unexpectedly displayed substantial Golgi localization when their O-glycosylation was compro

154 is recruited only to PM PtdIns4P rather than Golgi PtdIns4P remains unclear.

155 d a mouse lacking both GORASPs and find that Golgi cisternae remained stacked.

156 We infer that Golgi compartments form at ERES and then produce COPI ve

157 These findings reveal that Golgi exit of BACE1 and APP in primary neurons is tightl

158 Our analysis revealed that Golgi functions supported by PI4KB are critically import

159 The Golgi apparatus (GA) is an important site of insulin pro

160 The Golgi complex is recognized as being a central transport

161 The Golgi is a dynamic organelle whose correct assembly is c

162 th and disease, their trafficking across the Golgi apparatus remains poorly understood.

163 l for MMP2 and MT1-MMP trafficking along the Golgi apparatus.

164 mbrane trafficking between endosomes and the Golgi apparatus lead to neurodegenerative diseases.

165 tween the endoplasmic reticulum (ER) and the Golgi.

166 ics of membrane-bound organelles such as the Golgi apparatus, remain elusive.

167 R in vesicular/tubular structures and at the Golgi compartment.

168 an accumulation of ER-resident STING at the Golgi.

169 e findings suggest communication between the Golgi apparatus and mitochondria through homeostatically

170 retrograde trafficking of cargo between the Golgi apparatus and the endoplasmic reticulum, as well a

171 we show that Cab45 is phosphorylated by the Golgi-specific protein kinase Fam20C.

172 GALNT2 encodes the Golgi-localized polypeptide N-acetyl-d-galactosamine-tra

173 ingly, the vacuolar cargo begins to exit the Golgi near the midpoint of maturation, significantly bef

174 Here we demonstrate that BACE1 exits the Golgi in HeLa cells and primary neurons by a pathway dis

175 secretory vesicles are transported from the Golgi apparatus to the cell periphery by kinesin-1 KIF5B

176 dent transport of unstimulated EGFR from the Golgi compartment to the cell surface that we describe i

177 icantly delayed entry into and exit from the Golgi in fibroblasts derived from one of the affected su

178 protein I (COPI) mediated transport from the Golgi to the endoplasmic reticulum (ER).

179 enhanced NIS vesicular trafficking from the Golgi to the plasma membrane, whereas VCP-a principal co

180 Microtubules originate from the Golgi with an initial growth preference towards the axon

181 d proteolytic maturation after exit from the Golgi.

182 AP1-dependent lysosomal trafficking from the Golgi.

183 However, the Golgi is also an important site of key autophagy regulat

184 ing occurring under acidic conditions in the Golgi and release under conditions of higher pH in the E

185 YIPF5 resides in the Golgi apparatus and is thought to play a critical role i

186 gent, NBD C(6) -ceramide, NIR imaging in the Golgi apparatus has been demonstrated using these NIR em

187 GH-C53S was exclusively stained in the Golgi apparatus, and no secretory granules formed for th

188 important AGP glycans are synthesized in the Golgi apparatus, but the relationships among their glyco

189 (G-CK), which is exclusively resident in the Golgi apparatus.

190 assembly protein domain was retained in the Golgi of fibroblasts from the three patients, whereas co

191 ins (prior to enzymatic modifications in the Golgi), as well as those that are commonly observed on a

192 for understanding N-glycan maturation in the Golgi; however, the structural basis of these substrate

193 s attached to MTs is observed, including the Golgi apparatus, lytic granules, and mitochondria.

194 alactose, as UDP-Gal, are delivered into the Golgi apparatus by SLC35A3 and SLC35A2 transporters, res

195 mechanisms of UDP-GlcNAc transport into the Golgi apparatus may exist.

196 s is characterized by down-regulation of the Golgi alpha-mannosidase I coding gene MAN1A1, leading to

197 nzymes within successive compartments of the Golgi apparatus determine where new monomer building blo

198 This ensures proper functioning of the Golgi apparatus in protein trafficking and processing.

199 However, whether and how dispersal of the Golgi apparatus is actively regulated under stress, and

200 I) vesicles, maintenance and function of the Golgi apparatus, and mitochondria migration and position

201 ichment within the cytosolic leaflets of the Golgi complex, peroxisomes, and outer mitochondrial memb

202 iverse and essential roles in and out of the Golgi in different organisms.

203 Atg16L1 is an effector of the Golgi resident GTPase Rab33B.

204 d Parkinson's diseases, and mutations of the Golgi-associated retrograde protein (GARP) complex cause

205 e disorders associated with mutations of the Golgi-resident GlcNAc-1-phosphotransferase, which genera

206 been well documented, its consequence on the Golgi apparatus is less well understood.

207 interference with endosome formation or the Golgi apparatus impairs migration to a similar extent as

208 ly synthesized Cx43 that already reached the Golgi was not affected in Abeta(25-35)-exposed astrocyte

209 N-glycosylated GFP substantially reduces the Golgi residence time of a Tac chimera suggests that N-gl

210 Little is known about the role the Golgi compartment plays in Schwann cell (SC) functions.

211 AKAP9 form a protein platform tethering the Golgi to the nucleus.

212 egrees C, permissive conditions, through the Golgi apparatus was locally delayed, almost tenfold, bet

213 s as whether vesicular transport through the Golgi occurs in an anterograde (from entry to exit) or r

214 effect on anterograde transport through the Golgi suggesting that Rab6A and Rab6A' act coordinately.

215 Intracellular trafficking through the Golgi using the marker protein VSVG-GFP-ts045 demonstrat

216 Thus, the Golgi glycoprotein MGAT4D is a novel, intrinsic protecto

217 The ATP7A and ATP7B proteins localize to the Golgi and regulate copper homeostasis.

218 intained by two factors that localize to the Golgi apparatus, ATP7 and the conserved oligomeric Golgi

219 anslocation of the SCAP-SREBP complex to the Golgi apparatus, the activation of SREBP proteins (SREBP

220 raffic from the endoplasmic reticulum to the Golgi apparatus.

221 nsport from the endoplasmic reticulum to the Golgi apparatus.

222 etrograde route, from early endosomes to the Golgi apparatus.

223 ding sphingolipid catabolism enzymes, to the Golgi compartment, which may contribute to the sphingoli

224 lasmic reticulum (ER) and transferred to the Golgi complex by interaction with the Batten disease pro

225 g from the endoplasmic reticulum (ER) to the Golgi complex.

226 on and that it specifically localizes to the Golgi in Min6-K8 cells, a pancreatic beta-cell-like cell

227 low, Scap escorts SREBPs from the ER to the Golgi, where the actions of two proteases release the am

228 the ER focus is constructed proximal to the Golgi-associated BICD2 and BICDR1 dynein motor adaptors;

229 ic v-SNARE from the endocytic pathway to the Golgi.

230 ER, they are translocated from the ER to the Golgi.

231 PM) and is subsequently recycled back to the Golgi.

232 lock of their trafficking from the ER to the Golgi.

233 n the BNC of the baboon and monkey using the Golgi technique.

234 ng also abrogate Rab33B association with the Golgi stacks.

235 the calcium ATPase and processing within the Golgi compartment are essential for priming the capsid t

236 N-glycan trimming and is located within the Golgi, where it allows ER-escaped glycoproteins to bypas

237 In recent years, the Golgi outpost has emerged as a satellite organelle that

238 By quantitatively measuring their Golgi residence times, we found that the observed Golgi

239 lysosomal enzymes at the ER to promote their Golgi transfer.

240 ed beta subunit in the ER to proceed through Golgi and post-Golgi trafficking.

241 the oligomeric U21/class I MHC complexes to Golgi--derived quality control carriers destined for lys

242 ARD) and degradation of GM130 in response to Golgi stress.

243 family, TBC1D23 is critical for endosome-to-Golgi cargo trafficking by serving as a bridge between G

244 transporter and localize CHC22 to the ER-to-Golgi intermediate compartment (ERGIC).

245 complex (herein referred to as EGRESS: ER-to-Golgi relaying of enzymes of the lysosomal system), whic

246 ormation, thus inducing COPII-mediated ER-to-Golgi trafficking of STING.

247 rst report of mutations disrupting the ER-to-Golgi trafficking, resulting in diabetes.

248 s WASp as a novel effector of the nucleus-to-Golgi cell-survival pathway triggered by IR-induced DNA

249 role for FAM83H in endoplasmic reticulum-to-Golgi vesicle trafficking and protein secretion (dbGaP p

250 ed, almost tenfold, between medial and trans Golgi cisterna.

251 rent marker proteins downstream of the trans Golgi.

252 iffer in 3 amino acids and localize to trans Golgi cisternae.

253 terograde transport from the medial to trans Golgi.

254 ial transport was nearly normal as was trans Golgi to TGN transport.

255 eting it for sequestration within the trans- Golgi network (TGN).

256 s to control GLUT4 mobilization from a trans-Golgi network (TGN) storage compartment, establishing th

257 we mapped subapical F-actin fringe and trans-Golgi network positioning relative to sites of bulk secr

258 clear periphery, suggesting a role for trans-Golgi network (TGN) functions and retrograde transport i

259 per Golgi stack, suggesting a loss of trans-Golgi cisternae.

260 atidylinositol 4-phosphate [PI(4)P] on trans-Golgi network (TGN) vesicles were recruited to mitochond

261 The distribution of the M6PR pathway (trans-Golgi network to late endosomes) was constrained in infe

262 ficking distinct from the perinuclear, trans-Golgi pattern observed in normal cells.

263 The plant trans-Golgi network (TGN) is a central trafficking hub where s

264 sidue that accumulates APP in the TGN (Trans-Golgi Network) and diminishes its amyloidogenic cleavage

265 study revealed membrane tethers at the trans-Golgi as novel specific targets of ROS in cells.

266 imeras localize at the interior of the trans-Golgi cisternae.

267 th the loss of membrane tethers on the trans-Golgi cisternae.

268 d in the trafficking of ATP7A from the trans-Golgi complex in a Cu-dependent manner, suggesting that

269 mic reticulum, KOR1 cycles between the trans-Golgi network (TGN) and the plasma membrane (PM).

270 I-do not affect ATP7B targeting to the trans-Golgi network (TGN) but reduce its Cu-transport activity

271 of integral membrane proteins from the trans-Golgi network (TGN) has been shown to occur through tubu

272 ct vesicle classes at the level of the trans-Golgi network (TGN) has remained elusive.

273 Formation of SGs occurs at the trans-Golgi network (TGN) where their soluble cargo aggregates

274 including retrograde transport to the trans-Golgi network (TGN), is involved in the presentation of

275 athrin-coated vesicle formation at the trans-Golgi network (TGN), likely aiding the transport of carg

276 some-derived vesicles destined for the trans-Golgi network (TGN).

277 , such as one that intersects with the trans-Golgi network (TGN).

278 of a protein coat associated with the trans-Golgi network (TGN).

279 ypeptidase E (CPE) accumulate near the trans-Golgi network and are not retained in mature DCVs in the

280 rions due to a wrapping process at the trans-Golgi network and are required for cell-to-cell spread a

281 phagy protein ATG9A accumulated in the trans-Golgi network and was depleted from peripheral compartme

282 and show that its incorporation at the trans-Golgi network depends on cellular abundance of OSBP.

283 lular envelopment of poxviruses at the trans-Golgi network to release infectious extracellular virus

284 g of rnf-145 from the cis-Golgi to the trans-Golgi network to stop glial growth.

285 how that BACE1 is transported from the trans-Golgi network to the plasma membrane in an AP-1- and Arf

286 gle enveloped viral particles from the trans-Golgi network within small vacuoles to the plasma membra

287 at specific basic residues within the trans-Golgi network, granules, or at the cell surface/endosome

288 reduced while ATG9A accumulated in the trans-Golgi network.

289 e they regulate vesicle budding at the trans-Golgi network.

290 UPS5L variant was also detected in the trans-Golgi network/early endosome and at the plasma membrane.

291 CERBERUS colocalized with the trans-Golgi network/early endosome markers.

292 tabilizes LjVPY1 and LjVPY2 within the trans-Golgi network/early endosome, where they might function

293 s a generic Golgi export signal at the trans-Golgi to promote the constitutive exocytic trafficking.

294 eins Golgin-97 and Golgin-245 from the trans-Golgi.

295 sviruses, as is their association with trans-Golgi membranes.

296 ositol 4-phosphate (PI4P) in the yeast trans-Golgi network (TGN) is dependent on intracellular pH, in

297 C1-XPF DNA repair defect (Er1(F/-)) triggers Golgi dispersal, dilation of endoplasmic reticulum, auto

298 ation and functional characterization of two Golgi-localized exo-beta-1,3-galactosidases from the gly

299 It has been demonstrated that two Golgi stacking proteins, GRASP55 and GRASP65, self-inter

300 Grasp55 is a ubiquitous Golgi stacking protein involved in autophagy, protein tr