ライフサイエンスコーパス: Gram-negative bacteria

1 kines were quantified in keratitis caused by Gram-negative bacteria.

2 an early predictive marker of BCV rupture by Gram-negative bacteria.

3 cellular compartment that is the hallmark of Gram-negative bacteria.

4 cant differences among the Gram-positive and Gram-negative bacteria.

5 nfections specifically caused by XDR and PDR Gram-negative bacteria.

6 h3-PA0808 pair are widely distributed across Gram-negative bacteria.

7 ATR system appears to be conserved in other Gram-negative bacteria.

8 enzyme that is conserved in the majority of gram-negative bacteria.

9 medical need created by multidrug resistant Gram-negative bacteria.

10 ever, smokers exhibit faster repopulation of Gram-negative bacteria.

11 fically how the inflammasome is activated by Gram-negative bacteria.

12 responds to osmolarity and acidic pH in many Gram-negative bacteria.

13 eous component of the outer membrane (OM) of Gram-negative bacteria.

14 bacterium and an obligate predator of other Gram-negative bacteria.

15 epresentative of enzymes found in pathogenic Gram-negative bacteria.

16 y carbapenem-resistant, colistin-susceptible Gram-negative bacteria.

17 last-line treatment for multidrug-resistant Gram-negative bacteria.

18 y carbapenem-resistant, colistin-susceptible Gram-negative bacteria.

19 nt virulence trait in many gram-positive and gram-negative bacteria.

20 ous infections caused by multidrug-resistant gram-negative bacteria.

21 sensor for discriminating Gram-positive and Gram-negative bacteria.

22 f of peptidoglycan of both Gram-positive and Gram-negative bacteria.

23 mportant mechanism of clinical resistance in Gram-negative bacteria.

24 , nonredundant, surface-exposed processes in gram-negative bacteria.

25 why most antibiotics are ineffective against Gram-negative bacteria.

26 ride (LPS), the major cell-wall component of Gram-negative bacteria.

27 efficacy of antibiotics, especially against Gram-negative bacteria.

28 al properties against both Gram-positive and Gram-negative bacteria.

29 nvelope, thus, leading to drug-resistance in Gram-negative bacteria.

30 r membrane is a key virulence determinant of gram-negative bacteria.

31 cers (AIs) produced and detected by numerous gram-negative bacteria.

32 MAC perturbs the composite cell envelope of Gram-negative bacteria.

33 researched extensively in Gram-positive and Gram-negative bacteria.

34 division (RND) superfamily are ubiquitous in Gram-negative bacteria.

35 man infections caused by multidrug-resistant Gram-negative bacteria.

36 otein profiling (AfBPP) in Gram-positive and Gram-negative bacteria.

37 approaches to identify putative AMR genes in Gram-negative bacteria.

38 other mollicutes, Gram-positive bacteria, or Gram-negative bacteria.

39 uptake across the different compartments of Gram-negative bacteria.

40 substrate recruitment in toxin exporters in Gram-negative bacteria.

41 y of outer membrane and secreted proteins in Gram-negative bacteria.

42 to mount optimal CD8(+) T cell responses to gram-negative bacteria.

43 ising results against both Gram-positive and Gram-negative bacteria.

44 nt for the treatment of infections caused by gram-negative bacteria.

45 domains, are found on the outer membranes of Gram-negative bacteria.

46 acterized mostly for conjugative elements of Gram-negative bacteria.

47 t commensal and pathogenic Gram-positive and Gram-negative bacteria.

48 eta-barrel outer membrane proteins (OMPs) in Gram-negative bacteria.

49 rel proteins into the outer membrane (OM) of Gram-negative bacteria.

50 e role of Type Vd secreted phospholipases in Gram-negative bacteria.

51 of FMN riboswitch binders against wild-type Gram-negative bacteria.

52 ral Vibrio species and a set of monotrichous Gram-negative bacteria.

53 d enzymes that confer colistin resistance in Gram-negative bacteria.

54 st the rising threat of multi-drug-resistant Gram-negative bacteria.

55 stigation of novel substances active against Gram-negative bacteria.

56 ltiprotein system present in the envelope of Gram-negative bacteria.

57 occus aureus, representing Gram-positive and Gram-negative bacteria.

58 fied antimicrobial resistance (AMR) genes in Gram-negative bacteria.

59 nflammasome activation during infection with Gram-negative bacteria.

60 re key antibiotic resistance determinants in Gram-negative bacteria.

61 SS) is a pivotal virulence mechanism of many Gram-negative bacteria.

62 mes that are secreted by almost all forms of Gram-negative bacteria.

63 ell wall component of both Gram-positive and Gram-negative bacteria.

64 ars ago and have been extensively studied in Gram-negative bacteria.

65 positive bacteria and lipopolysaccharides of Gram-negative bacteria.

66 cell wall is widely conserved across diverse Gram-negative bacteria.

67 c leukemia (ALL) to decrease infections with gram-negative bacteria.

68 effectors through the outer membrane of many Gram-negative bacteria(1-3).

69 lity of most drug leads to accumulate inside Gram-negative bacteria(1-7).

70 not included): Gram-positive bacteria, 58%; Gram-negative bacteria, 78%; and Candida species, 83%.

71 The cell envelope of gram-negative bacteria, a structure comprising an outer

72 zed filters quickly killed Gram-positive and Gram-negative bacteria aerosols in vitro, with CFU reduc

73 tivity that includes gram-positive bacteria, gram-negative bacteria, anaerobes, atypicals, and other

74 urpose, we chose the pilus protein FimG from Gram-negative bacteria and a disulfide-bonded variant of

75 le to rapidly traverse the outer membrane of Gram-negative bacteria and accumulate inside these cells

76 is one of the largest dynamic assemblies in gram-negative bacteria and allows for delivery of toxins

77 Based on mouse studies, roles of gram-negative bacteria and altered intestinal homeostasi

78 used higher dose regimens of amoxicillin on gram-negative bacteria and antibiotic resistance genes a

79 ificities, moderately high sensitivities for Gram-negative bacteria and Candida species, and elevated

80 (P < 0.0001)-with pronounced differences for Gram-negative bacteria and Candida species.

81 s toward understanding betaOMP biogenesis in Gram-negative bacteria and in mitochondria.

82 eta-barrel assembly machine (Bam) complex in Gram-negative bacteria and its counterparts in mitochond

83 rkers such as lipopolysaccharides (LPS) from Gram-negative bacteria and lipoteichoic acids (LTA) from

84 cal regions with high resistance rates among Gram-negative bacteria and M tuberculosis.

85 reptococcus spp. are the most common causes, gram-negative bacteria and mixed infections can occur in

86 of the endotoxin lipopolysaccharide (LPS) of gram-negative bacteria and on the assembly of the bacter

87 S) are essential envelope components in many Gram-negative bacteria and provide intrinsic resistance

88 udy was to demonstrate this vicious cycle in gram-negative bacteria and show the utility of vector au

89 a lipoprotein functionally conserved amongst Gram-negative bacteria and that loss of DolP increases m

90 Gram-negative bacteria and their complex cell envelope,

91 eptation is critical to faithful division in Gram-negative bacteria and vital to the barrier function

92 the beta-barrel assembly machinery (BAM) in Gram-negative bacteria, and by the sorting and assembly

93 OMP folding is an essential process in all Gram-negative bacteria, and considering the looming cris

94 In the case of Gram-negative bacteria, and in particular of Helicobacte

95 orescence intensity levels for cultures with Gram-negative bacteria, and only ~ 3.5-fold increased fl

96 m-positive bacteria, intrinsic resistance in Gram-negative bacteria, and other forms of noninherited

97 Lipopolysaccharides (LPS) of Gram-negative bacteria are critical for the defence agai

98 Mitochondria, chloroplasts and Gram-negative bacteria are encased in a double layer of

99 Although a number of Gram-negative bacteria are known to catabolize quinate a

100 Gram-negative bacteria are surrounded by an inner cytopl

101 Gram-negative bacteria are surrounded by an outer membra

102 rimeric porins in the outer membrane (OM) of Gram-negative bacteria are the conduits by which nutrien

103 A key group of quorum sensing molecules in Gram-negative bacteria are the N-acylhomoserine lactones

104 investigated the binding of CTRP6 to various Gram-negative bacteria as well as PRMs and enzymes of th

105 A range of Gram-negative bacteria associated with high morbidity an

106 d cannot disrupt the cytoplasmic membrane of gram-negative bacteria because it cannot penetrate the O

107 In Gram-negative bacteria, beta-lactamase enzymes that hydr

108 ant differences in the relative abundance of Gram-negative bacteria between BIA-ALCL and control spec

109 In Gram-negative bacteria, biodegradation depends on facili

110 that distinguished between Gram-positive and Gram-negative bacteria, both in vitro and after in vivo

111 arides are anchored to the outer membrane of Gram-negative bacteria by a hydrophobic moiety known as

112 nel is an essential step during infection of Gram-negative bacteria by all short-tailed phages, becau

113 in permeability across the outer membrane of Gram-negative bacteria by electrophysiology experiments

114 Mouse and human RELMbeta selectively killed Gram-negative bacteria by forming size-selective pores t

115 that neutrophils recognize Gram-positive and Gram-negative bacteria by means of multiple phagosomal T

116 represents one prevalent mechanism by which Gram-negative bacteria can become resistant to key antib

117 acterial assays with wild-type and resistant Gram negative bacteria carrying either single or multipl

118 Endotoxin (LPS) released from gram-negative bacteria causes strong immunologic and inf

119 In case of all gram-negative bacteria, cephalosporin consumption led to

120 membrane or evade the host immune response, Gram-negative bacteria chemically modify LPS in a wide v

121 S to recycle fatty acids may help pathogenic gram-negative bacteria circumvent FAS inhibition.

122 positive bacteria and increased abundance of gram-negative bacteria, compared with mice given only wa

123 Lipopolysaccharides (LPSs) of Gram-negative bacteria comprise lipid A, core, and O-pol

124 The outer membrane (OM) of gram-negative bacteria confers innate resistance to toxi

125 Carbapenem-resistant Gram-negative bacteria (CRGNB) continue to present a glo

126 Outer membrane proteins (OMPs) in Gram-negative bacteria dictate permeability of metabolit

127 (LPS), a component of the outer membrane of gram-negative bacteria, disrupts the alveolar-capillary

128 In the gram-negative bacteria, DNA is first pulled toward the o

129 We find that infection by Gram-negative bacteria drives an increase in mitochondri

130 his development is especially problematic in gram-negative bacteria due to the outer membrane (OM) pe

131 yotic E3s, are utilized by a wide variety of Gram-negative bacteria during pathogenesis.

132 The Gram-negative bacteria E. coli and P. aeruginosa were pa

133 ylococcus aureus, Enterococcus faecalis) and Gram negative bacteria (e.g., Escherichia coli, Klebsiel

134 In the biotechnology industry, gram-negative bacteria (e.g., Escherichia coli) are wide

135 HFM and showed that HFM increases rat fecal Gram-negative bacteria, elevates lipopolysaccharides (LP

136 per 100 bed-days and incidence densities of gram-negative bacteria (Escherichia coli, Klebsiella spp

137 of wild-type and ampicillin (amp)-resistant Gram-negative bacteria, Escherichia coli (E. coli).

138 Among Gram-negative bacteria, Escherichia coli were predominan

139 evealed it to be an antibiotic selective for Gram-negative bacteria, especially against Vibrio specie

140 The outer membrane (OM) of Gram-negative bacteria exhibits unique lipid asymmetry,

141 olipids, found in the outer membranes of all Gram-negative bacteria, exhibits considerable structural

142 Gram-negative bacteria expressing class A beta-lactamase

143 polycationic peptides that efficiently kill gram-negative bacteria: facile penetration of the outer

144 It is, however, inactive toward Gram-negative bacteria, for which we are in dire need of

145 The asymmetric outer membrane (OM) of Gram-negative bacteria functions as a selective permeabi

146 The highly asymmetric outer membrane of Gram-negative bacteria functions in the defense against

147 Carbapenem-resistant Gram-negative bacteria (GNB) are heading the list of pat

148 However, some gram-negative bacteria harbor an enzyme known as the acy

149 Carbapenem resistance in gram-negative bacteria has caused a global epidemic that

150 espread antibiotic resistance, especially of Gram-negative bacteria, has become a severe concern for

151 tein commonly found in the outer membrane of Gram-negative bacteria, has served as a paradigm for the

152 with efficacy against both gram-positive and gram-negative bacteria, has the potential to enhance tre

153 Gram-negative bacteria have a cell envelope that compris

154 Gram-negative bacteria have been found to be a major pop

155 The LPS of Gram-negative bacteria have been shown to activate a nov

156 Outer membrane vesicles produced by Gram-negative bacteria have been studied for half a cent

157 Pathogenic Gram-negative bacteria have developed several strategies

158 Not surprisingly, Gram-negative bacteria have evolved diverse posttranslat

159 Gram-negative bacteria have evolved numerous pathways to

160 Outer membrane vesicles (OMVs) produced by Gram-negative bacteria have roles in cell-to-cell signal

161 trum of activity against multidrug-resistant Gram-negative bacteria; however, breakpoints have been e

162 prevalent cause of antibiotic resistance in Gram-negative bacteria, i.e., the deactivation of the mo

163 Artificial sterilization of Gram-negative bacteria in aerobic bottles containing cli

164 ogically efficacious therapy for fermenting, gram-negative bacteria in blood culture(s) if they were

165 m-resistant and/or multidrug-resistant (MDR) Gram-negative bacteria in clinical settings.

166 ntibacterial role against this bacterium and gram-negative bacteria in general.

167 ority pathogens listed by the WHO, including Gram-negative bacteria in the critical priority category

168 design drugs with increased activity against Gram-negative bacteria in the face of the antibiotic res

169 avelength) against multidrug-resistant (MDR) Gram-negative bacteria in vitro and in vivo.

170 In gram-negative bacteria including Escherichia coli, the p

171 identify tripartite alpha-PFTs in pathogenic Gram negative bacteria, including Aeromonas hydrophila (

172 bactericidal activity on novobiocin against Gram-negative bacteria, including carbapenem-resistant a

173 Many Gram-negative bacteria, including causative agents of dy

174 in vitro to predate on a wide range of other Gram-negative bacteria, including CDC category A/B patho

175 Many Gram-negative bacteria, including context-dependent huma

176 tent activity against gram-positive and many gram-negative bacteria, including methicillin-resistant

177 range of antibiotics and antiseptics in many gram-negative bacteria, including pathogens.

178 S in vitro and inhibit the growth of diverse Gram-negative bacteria, including polymyxin-resistant st

179 In many Gram-negative bacteria, including Rhodobacter capsulatus

180 ular nanomachine utilized by many pathogenic Gram-negative bacteria, including the causative agents o

181 tibacterial agents with activity against MDR Gram-negative bacteria, including WHO priority pathogens

182 The zauPzapA operon is present in diverse Gram-negative bacteria, indicating a common mechanism fo

183 Stimulation beyond a TLR4 threshold and Gram-negative bacteria-induced responses were characteri

184 Ubiquitously present LPS in Gram-negative bacteria induces NLRP3 inflammasome activa

185 g proteins (GBPs) assemble on the surface of Gram-negative bacteria into polyvalent signaling platfor

186 Cell-surface signaling (CSS) in Gram-negative bacteria involves highly conserved regulat

187 Pneumonia induced by Gram-negative bacteria is a common and serious disease a

188 The cell envelope of Gram-negative bacteria is a multilayered structure essen

189 Lipopolysaccharide derived from Gram-negative bacteria is a potent activator of circulat

190 Carbapenem resistance in Gram-negative bacteria is a public health concern.

191 The outer membrane (OM) of Gram-negative bacteria is a selective permeability barri

192 The double-membrane cell envelope of Gram-negative bacteria is a sophisticated barrier that f

193 f the outer leaflet of the outer membrane of Gram-negative bacteria is already neutralized by peptide

194 The outer membrane (OM) of Gram-negative bacteria is an asymmetric bilayer having p

195 The outer membrane (OM) of Gram-negative bacteria is an asymmetric lipid bilayer th

196 Pneumonia due to Gram-negative bacteria is associated with high mortality

197 Resistance to beta-lactam antibiotics in Gram-negative bacteria is commonly associated with produ

198 The outer-membrane of Gram-negative bacteria is critical for surface adhesion,

199 The outer membrane of Gram-negative bacteria is essential for their survival i

200 mall molecule permeation and accumulation in Gram-negative bacteria is important for drug development

201 The outer membrane of Gram-negative bacteria is of great scientific interest b

202 resulting from acute pneumonic infections by Gram-negative bacteria is often characterized by dysfunc

203 Antimicrobial resistance in pathogenic gram-negative bacteria is one of the most pressing chall

204 The cell envelope of Gram-negative bacteria is synthesized and maintained via

205 A major resistance mechanism in Gram-negative bacteria is the production of beta-lactama

206 (LPS), an inflammatory stimulus derived from gram-negative bacteria, is present in the normal GI trac

207 l envelope stability(4); however, most other Gram-negative bacteria lack Lpp so it has been assumed t

208 the major component of the outer membrane of gram-negative bacteria, lipopolysaccharide (LPS), binds

209 In Gram-negative bacteria, lipopolysaccharide is essential

210 The Burkholderia genus encompasses many Gram-negative bacteria living in the rhizosphere.

211 Essential in the growth of Gram-negative bacteria, LpxA is a logical target for ant

212 Adhesive type 1 pili from enteroinvasive, Gram-negative bacteria mediate attachment to host cells.

213 uring LPS transfection; however, its role in Gram-negative bacteria-mediated NLRP3 inflammasome activ

214 In the outer membrane of Gram-negative bacteria, membrane proteins are thought to

215 erial activity against 3 Gram-positive and 3 Gram-negative bacteria (MICs = 0.78-3.12 mg/mL).

216 CLs did not suspect that slow-growing, small Gram-negative bacteria might be harmful.

217 Gram-negative bacteria, mitochondria, and chloroplasts a

218 O-antigens of Gram-negative bacteria modulate the interactions of bact

219 In Gram-negative bacteria, multicomponent protein complexes

220 ay of isoprenoid synthesis, is essential for Gram-negative bacteria, mycobacteria and apicomplexans(2

221 IEps by Gram-negative bacteria (n = 210) outnumbered those by Gr

222 ugs, new antibiotics for multidrug-resistant Gram-negative bacteria (namely, ceftazidime-avibactam, p

223 of different rainbow trout Gram-positive and Gram-negative bacteria, namely Lactococcus garvieae, Aer

224 mediated by lipid A aminoarabinosylation in Gram-negative bacteria, namely, ArnT (undecaprenyl phosp

225 e scored as bacteria/fungi/none (BAC One) or gram-negative bacteria/none (BAC Two) and compared to Gr

226 In the outer membrane of gram-negative bacteria, O-antigen segments of lipopolysa

227 st antibiotics through the outer membrane of Gram-negative bacteria occurs through porin channels.

228 biotic enhancer properties against resistant Gram-negative bacteria of four antibiotics belonging to

229 drug target, whose inhibition could deplete gram-negative bacteria of numerous proteins that reside

230 his breaks the dogma that beta-lactams enter Gram-negative bacteria only by passive diffusion through

231 Gram stain for delineating gram-positive or gram-negative bacteria or fungi within corneal scrapes.

232 II secretion system (T3SS) rod proteins from Gram-negative bacteria or noncanonical inflammasome acti

233 gram-positive bacteria), TLR4 (receptor for gram-negative bacteria), or distilled water (control) an

234 enterococci (P = .008), multidrug-resistant gram-negative bacteria (P = .016), or quinolone-resistan

235 In Gram-negative bacteria, PG is assembled in the cytoplasm

236 Gram-negative bacteria produce outer-membrane vesicles (

237 exposure and recent prior susceptibility of Gram-negative bacteria) provided a modest predictive dis

238 bacteria (P = .016), or quinolone-resistant gram-negative bacteria (QR-GNB) (P = .015).

239 OMPs in Gram-negative bacteria rarely have reactive cysteines.

240 Gram-negative bacteria release outer membrane vesicles i

241 y; however, there is little knowledge on how Gram-negative bacteria release their OMs into their envi

242 Gram-negative bacteria remodel their surfaces to interac

243 Gram-negative bacteria repopulated in the smokers faster

244 Increased Gram-negative bacteria resistance to antibiotics is beco

245 ATCC25922, as examples of Gram-positive and Gram-negative bacteria, respectively, by DUV LED of 280

246 tive staphylococci, other gram-positive, and gram-negative bacteria, respectively.

247 hronic inflammatory disease characterized by Gram-negative bacteria responsible for the degradation o

248 NA from various, unrelated gram-positive and gram-negative bacteria results in a more pronounced tau

249 ctivity against a panel of Gram-positive and Gram-negative bacteria revealed structure-activity relat

250 On Gram-negative bacteria, S-layers are anchored to cells v

251 The outer membrane (OM) of Gram-negative bacteria serves as a selective permeabilit

252 ed by Gram-positive cocci (GPC), susceptible Gram-negative bacteria (sGNB), resistant GNB (rGNB) and

253 Recent recurrent outbreaks of Gram-negative bacteria show the critical need to target

254 In Gram-negative bacteria, some of these pumps form multi-p

255 ow-derived macrophages (BMDMs) infected with Gram-negative bacteria such as Citrobacter rodentium, Es

256 impact of carbapenem-resistant nonfermenting gram-negative bacteria, such as Acinetobacter baumannii,

257 y the extract against both Gram-positive and Gram-negative bacteria suggests the presence of a broad

258 The broad conservation of the Mla system in Gram-negative bacteria suggests the system may play a co

259 our method detects lipid A modifications in Gram-negative bacteria that are associated with antimicr

260 ls and in water bodies obligate predators of gram-negative bacteria that can affect bacterial communi

261 T6SS) is a dynamic organelle encoded by many gram-negative bacteria that can be used to kill competin

262 Brucella spp. are facultative intracellular Gram-negative bacteria that cause the zoonotic disease b

263 outer membrane (OM) is a defining feature of Gram-negative bacteria that serves as a permeability bar

264 From the three serotypes of Gram-negative bacteria that were tested, there were dist

265 In Gram-negative bacteria, the beta-barrel assembly machine

266 fundamentally different relative to those of Gram-negative bacteria, the evolution and ecology as wel

267 In Gram-negative bacteria, the folding and insertion of bet

268 ment-dependent pleiotropic gene regulator in Gram-negative bacteria, the H-NS protein is crucial for

269 In Gram-negative bacteria, the O antigen polysaccharide rep

270 In Gram-negative bacteria, these ferric-siderophore complex

271 In Gram-negative bacteria, these pumps form tripartite asse

272 unds, which are less likely to accumulate in Gram-negative bacteria; thus there is trade-off between

273 T6SS) is a proteinaceous weapon used by many Gram-negative bacteria to deliver toxins into adjacent t

274 complexes constitute a primary mechanism for Gram-negative bacteria to expel toxic molecules for surv

275 f sodium dodecyl sulfate or PMA enhancer for Gram-negative bacteria to improve the effect of PMA.

276 and utilization of enterobactin permits many Gram-negative bacteria to thrive in environments where l

277 Many pathogenic Gram-negative bacteria use the type III secretion system

278 eview will focus on representative SLPs that gram-negative bacteria use to overcome host innate immun

279 Many Gram-negative bacteria use type III secretion systems (T

280 t thought to be a phenomenon associated with Gram-negative bacteria, vesicle production in Staphyloco

281 ded RNA bacteriophages (ssRNA phages) infect Gram-negative bacteria via a single maturation protein (

282 OmpU, one of the porins of Gram-negative bacteria Vibrio cholerae, induces TLR1/2-M

283 last class of antibiotics that acted against Gram-negative bacteria was developed in the 1960s(2).

284 Colonization by resistant gram-negative bacteria was significantly associated with

285 nal domination (relative abundance >=30%) by gram-negative bacteria was used as predictor of gram-neg

286 The proportion of Gram-positive and Gram-negative bacteria were 135(68.2%) and 63(31.8%) res

287 diversity metrics and relative abundance of Gram-negative bacteria were calculated, and phylogenetic

288 Gram-negative bacteria were more abundant in the smokers

289 aride (LPS) resides in the outer membrane of Gram-negative bacteria where it is responsible for barri

290 re important cell surface polysaccharides in gram-negative bacteria where they extend core lipopolysa

291 process for novel vaccine candidates against gram-negative bacteria, which could be employed to other

292 moting the spread of pathogenic traits among Gram-negative bacteria while protecting them from lytic

293 nhances combinatorial antigenic diversity in Gram-negative bacteria, while reducing associated fitnes

294 is an uncharacterized protein ubiquitous in Gram-negative bacteria whose gene frequently occurs in c

295 el, we can predict AMR protein sequences for Gram-negative bacteria with an accuracy ranging from 93%

296 ns is the decoration of the outer surface of gram-negative bacteria with proteins tethered to the out

297 ctivity against the tested Gram-positive and Gram-negative bacteria, with a large zone of inhibition

298 ipopolysaccharide (LPS), a major compound of Gram-negative bacteria, with stimulatory activities in s

299 olysaccharides, present on the outer wall of Gram-negative bacteria, with the Toll-like Receptor 4.

300 sceptibility profiles of clinically relevant Gram-negative bacteria within two hours of antibiotic in