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1 the basis for the immunologic attack seen in Graves ophthalmopathy.
2 leading to the clinical findings typical of Graves ophthalmopathy.
3 ent of lower lid retraction in patients with Graves ophthalmopathy.
4 to the intense inflammation associated with Graves' ophthalmopathy.
5 y a role in the etiology and pathobiology of Graves' ophthalmopathy and/or other ocular myopathies.
9 r orbital fat decompression in patients with Graves' ophthalmopathy (GO) by measuring their midpupil
10 angiography (OCTA) in patients with inactive Graves' ophthalmopathy (GO) compared with healthy contro
11 n 85 years, the efficacy of radiotherapy for Graves' ophthalmopathy (GO) has not been established con
14 isease (GD) is a common thyroid disease, and Graves ophthalmopathy(GO) is the most common extra-thyro
16 for those undergoing the orbital changes of Graves' ophthalmopathy is often challenging, even as our
17 fic situations, for example, Duane syndrome, Graves' ophthalmopathy, may be particularly problematic.
19 excision (n = 6), orbital decompression for Graves ophthalmopathy (n = 3), dacryocystorhinostomy (n
21 ponder rate, and a larger improvement in the Graves' Ophthalmopathy Quality of Life overall score.
22 ommendations only extending to patients with Graves ophthalmopathy, selenium supplementation is widel
23 and the mean change in overall score on the Graves' ophthalmopathy-specific quality-of-life (GO-QOL)
24 Clinical Activity Score, and results on the Graves' ophthalmopathy-specific quality-of-life question
25 ve involvement of the extraocular muscles in Graves' ophthalmopathy, the absence of N-CAM expression
26 lasts or fat accumulation, such as occurs in Graves' ophthalmopathy, tissue fibrosis, abnormal wound
27 was a retrospective review of patients with Graves ophthalmopathy undergoing lower eyelid retraction