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1 dy weight loss and death upon infection with H3N2 influenza virus.
2 mperature very similar to that of a seasonal H3N2 influenza virus.
3 ith similar efficiency to that of a seasonal H3N2 influenza virus.
4 y between genetic and antigenic evolution of H3N2 influenza viruses.
5 ontact and airborne transmission of H1N1 and H3N2 influenza viruses.
6 er titers compared with five common seasonal H3N2 influenza viruses.
7 oad cross-reactivity against H1N1 as well as H3N2 influenza viruses.
8 ssion between the pandemic H1N1 and seasonal H3N2 influenza viruses.
9 additional glycosylation on the virulence of H3N2 influenza viruses.
10 bodies did neutralize antigenically distinct H3N2 influenza viruses.
11 s that were previously exposed to historical H3N2 influenza viruses.
12 lded 21 novel (H1N1) viruses and 2 seasonal (H3N2) influenza viruses.
13               The first pandemic season of A/H3N2 influenza virus (1968/1969) resulted in significant
14 , but it was the second pandemic season of A/H3N2 influenza virus (1969/1970) that caused the majorit
15  (influenza virus A-1), 40 influenza virus A H3N2 (influenza virus A-3), 37 influenza virus A "equivo
16 monocytes could be readily infected with the H3N2 influenza virus A/Udorn/72 (Udorn), irrespective of
17 ted receptor specificities of human H1N1 and H3N2 influenza viruses and animal H5N1 viruses that pose
18 es had high HAIs against a panel of H1N1 and H3N2 influenza viruses and were protected against both m
19                                        Human H3N2 influenza viruses are subject to rapid antigenic ev
20 ent was unique because the causative agents, H3N2 influenza viruses, are infrequently isolated from s
21  (SK06, H5N1) and A/Tokyo/Ut-Sk-1/07 (Tok07, H3N2) influenza viruses by reverse genetics.
22 y in elderly subjects, suggesting that older H3N2 influenza viruses confer some cross-reactive antibo
23                                Circulating A/H3N2 influenza viruses drifted significantly after strai
24                           Evolution of human H3N2 influenza viruses driven by immune selection has na
25 ve impaired protection against circulating A(H3N2) influenza viruses during the 2016-2017 and 2017-20
26 nactivated A/Aichi/68 (H3N2) or A/Sydney/97 (H3N2) influenza virus elicited complete protection again
27 o escape antibody recognition for decades, A/H3N2 influenza viruses emerged with altered receptor spe
28 ted that the X-ORFs of equine H3N8 and avian H3N2 influenza viruses encoded 61 amino acids but were t
29 ral activity against diverse H1N1, H5N1, and H3N2 influenza viruses encoding HA proteins from both gr
30      The RAMM model calibrated to historical H3N2 influenza virus evolution in humans fit well to the
31 tive antibodies against recently circulating H3N2 influenza viruses from 2019.
32                                        Novel H3N2 influenza viruses (H3N2v) containing seven genome s
33                 The SA binding properties of H3N2 influenza viruses have been observed to change duri
34                    Due to immune pressure, A/H3N2 influenza viruses have emerged with altered recepto
35 stances) caused an antigenic drift event for H3N2 influenza viruses historically.
36 on to TMPRSS2, is able to activate the HA of H3N2 influenza virus in vivo IMPORTANCE: Influenza epide
37 emics of equine-origin H3N8 and avian-origin H3N2 influenza viruses in canine populations are example
38                   Isolation of human subtype H3N2 influenza viruses in embryonated chicken eggs yield
39 ency of severe infections seen with seasonal H3N2 influenza viruses in recent decades compared to the
40 r that is involved in cleavage activation of H3N2 influenza viruses in vivo.
41 al antibody for the therapeutic treatment of H3N2 influenza virus infection.
42 ls of antibodies in mice and protect against H3N2 influenza virus infection.
43 the mice were comparably susceptible to X31 (H3N2) influenza virus infection.
44  that the aerosol transmission of a seasonal H3N2 influenza virus is most efficient under cold, dry c
45 ch has become immunodominant in recent human H3N2 influenza viruses, is also evolutionarily constrain
46 his report we describe a multidrug-resistant H3N2 influenza virus isolated from an immunocompromised
47  Here, NK lysis of cells infected with human H3N2 influenza viruses isolated between 1969 and 2003 wa
48 ve antibodies that neutralized cocirculating H3N2 influenza viruses isolated over a 20-year period.
49 nst a panel of historical and co-circulating H3N2 influenza viruses isolated over the last 15 years,
50                  hCK cells supported human A/H3N2 influenza virus isolation and growth much more effe
51 ected antigenic variants from human H1N1 and H3N2 influenza virus libraries possessing random mutatio
52 r infection of Tmprss2 knockout mice with an H3N2 influenza virus, only a slight increase in survival
53 d then determined the role of that domain in H3N2 influenza virus pathogenicity.
54 recovery from infection caused by a shifted (H3N2) influenza virus, probably through the induction of
55 n induced protein 35 (IFI35) inhibits swine (H3N2) influenza virus replication by directly interactin
56 e PB1-F2 reading frame in a recent, seasonal H3N2 influenza virus strain did not affect these paramet
57          Animals previously infected with an H3N2 influenza virus succumbed to systemic disease and e
58 component of recent antigenic drift in human H3N2 influenza viruses, supporting the idea that influen
59 acts of ferrets infected with H5N1, H1N1, or H3N2 influenza viruses that exhibit diverse virulence an
60 e/INFIMH-16-0019/2016-like and circulating A(H3N2) influenza viruses using HA pseudoviruses.
61 e/INFIMH-16-0019/2016-like and circulating A(H3N2) influenza viruses using HA-pseudoviruses.
62 mized broadly reactive antigens (COBRAs) for H3N2 influenza viruses utilize current viral surveillanc
63    Here, we evaluated whether prior seasonal H3N2 influenza virus vaccination or infection affects vi
64 ") were inoculated with A/Wisconsin/67/2005 (H3N2) influenza virus via intranasal drops.
65 ever, we recently found that a human-lineage H3N2 influenza virus was highly restricted in its abilit
66 currently cocirculating avian H5N1 and human H3N2 influenza viruses, we generated all the 254 combina
67 demic risk assessment for currently-dominant H3N2 influenza viruses, we investigated HA stability of
68  that the PA-X genes of equine H3N8 or avian H3N2 influenza viruses were full-length, with X-ORFs enc
69 providing long-term cross-protection against H3N2 influenza virus when compared to other vaccination
70  10 different genotypes of novel reassortant H3N2 influenza viruses with 2009 pandemic H1N1 [A(H1N1)p
71                   We showed that reassortant H3N2 influenza viruses with 3 or 5 genes from A(H1N1)pdm