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1 d competed with HGF for binding to the c-Met HGF receptor.
2 containing the entire coding sequence of the HGF receptor.
3  32D cells transfected with c-Met, the human HGF receptor (32D/c-Met).
4 elial cells through phosphorylation of c-Met/HGF receptor and activation of the ERK2 signaling pathwa
5                                              HGF receptor and transforming growth factor alpha (TGF-a
6 receptors for fibroblast growth factors, the HGF receptor c-Met binds tightly to HS.
7 ts showing that a selective inhibitor of the HGF receptor c-Met increased lung collagen to WT levels
8 tein because it interacts with both EGFR and HGF receptor c-Met pathways.
9  Pkd1-null mouse cells due to failure of the HGF receptor c-Met to be properly ubiquitinated and subs
10 is of downstream signaling revealed that the HGF receptor c-Met was equally activated in confluent an
11 els of active HGF and phosphorylation of the HGF receptor c-met were increased after muscle injury in
12          All melanocytic cells expressed the HGF receptor c-Met, and autocrine HGF caused constitutiv
13                                              HGF receptor c-met, fibroblast growth factor (FGF)-3, it
14        PHTx also increased activation of the HGF receptor c-Met, which was detected more then 9 hours
15 ion of HGF and endothelial activation of the HGF receptor c-Met.
16 t to determine how hepatocyte growth factor (HGF) receptor c-Met and epidermal growth factor receptor
17     Bcl-xl and the hepatocyte growth factor (HGF) receptor c-Met are both highly expressed in mesothe
18  engagement of the hepatocyte growth factor (HGF) receptor c-Met by heart-produced HGF during priming
19 n of the oncogenic hepatocyte growth factor (HGF) receptor c-MET in PNETs.
20  Activation of the hepatocyte growth factor (HGF) receptor c-met results in the regulation of cell-ma
21  kidney; (2) similar PTH type I receptor and HGF receptor (c-met) expression levels in the proximal t
22 atocyte growth factor (HGF) levels and renal HGF receptor (c-MET) gene expression.
23 tion of PGE2 as well as expression of Cox-2, HGF receptor (c-Met-R), epidermal growth factor receptor
24 terized mice with pancreatic deletion of the HGF receptor, c-Met (PancMet KO mice), in two models of
25                                          The HGF receptor, c-Met, was minimally phosphorylated in con
26 with this downregulation, phosphorylation of HGF receptor, c-Met, which is frequently overexpressed i
27 oid cell line 32D transfected with the human HGF receptor, c-Met.
28 einases, decreased hepatocyte growth factor (HGF) receptor, c-met proto-oncogene, and impaired wound
29                The hepatocyte growth factor (HGF) receptor, c-met, transduces the HGF multiple biolog
30  cells express the hepatocyte growth factor (HGF) receptor, c-Met.
31  except in the headfold state (when only the HGF receptor can be detected) and in the heart at the 14
32 ing mutations in the MET proto-oncogene (the HGF receptor) cause familial RCC, we investigated whethe
33 nto G(Alert) and that signalling through the HGF receptor cMet is also necessary.
34  prostate cancer cells induces activation of HGF-receptor/cMet and stimulates hyaluronan/CD44v9 signa
35                               Phosphorylated HGF receptor coimmunoprecipitated with Shc, Grb2, and So
36 orting of cells engineered to express HGF or HGF receptor compared with control cell lines, enzyme-li
37                    Hepatocyte Growth Factor (HGF) receptor, encoded by the protooncogene c-met, is ov
38 applied to study the distribution of HGF and HGF receptor expression in rabbit lacrimal gland tissue
39 s express Hgf, and selective deletion of the Hgf receptor gene in respiratory epithelium phenocopies
40 hese data provide fundamental information on HGF receptor gene organization, as well as on the genomi
41 th an increase in the phosphorylation of the HGF receptor (HGFR or MET, encoded by the MET gene), as
42 ed in vivo expression of VEGF receptor-2 and HGF receptor in retrolental membranes from 16 patients w
43  Activation of the hepatocyte growth factor (HGF) receptor in epithelial cells results in lamellipodi
44 inked to cMet, the hepatocyte growth factor (HGF) receptor, in tumor cells, and this interaction decr
45                The hepatocyte growth factor (HGF) receptor is a tyrosine kinase transmembrane protein
46 sufficient for binding and activation of the HGF receptor, lacks the ability to super-activate the Ra
47 n reaction method was used to detect HGF and HGF receptor messenger RNA in human lacrimal gland tissu
48 ussman and colleagues show that blocking the HGF receptor MET abrogates HGF's rescue of drug sensitiv
49  that hepatocyte growth factor (HGF) and the HGF receptor MET might support some types of enteric neu
50  by HMGB1/TLR-4-dependent down-regulation of HGF receptor MET on MSC, thereby impairing HGF-driven MS
51   The hepatocyte growth factor (HGF) and the HGF receptor Met pathway are important in the pathogenes
52 h factor (HGF) resulted in activation of the HGF receptor MET, reactivation of the mitogen-activated
53 nase domain of the hepatocyte growth factor (HGF) receptor Met, enhance Met downstream signaling, and
54  Janus kinase 1 (Jak1), Shc, Grb2, Sos1, and HGF receptor (met).
55 asis, and dysregulated signaling through the HGF receptor, Met, contributes to tumorigenesis, tumor p
56  resistance mediated by up-regulation of the HGF receptor, MET.
57                The hepatocyte growth factor (HGF) receptor, Met, is a strong prognostic indicator of
58                                              HGF receptor mRNA is detected in the mesoderm of the hea
59 t the headfold stage, between E7.5 and E8.0, HGF receptor mRNA was detected in myocardial cells befor
60 on products of the expected size for HGF and HGF receptor mRNAs were detected in lacrimal tissue and
61    hrIL-7/HGFalpha cross-linked the IL-7 and HGF receptors on thymocytes, and the in vivo thymocyte-s
62 st that PIPKIgamma, downstream of EGF and/or HGF receptor, participates in breast cancer progression
63 F also triggered a 13-fold increase in c-Met/HGF receptor phosphorylation (P<0.005) and increased ERK
64 ced increase in ERK2 activity, but not c-Met/HGF receptor phosphorylation.
65 ression of Met, the hepatocyte growth factor(HGF) receptor, plays an important role in tumorigenesis.
66 ng directly to the hepatocyte growth factor (HGF) receptor, present on the surface of host cells.
67                          The distribution of HGF receptor protein expression in the lacrimal gland su
68 ession of hepatocyte growth factor (HGF) and HGF receptor proteins in lacrimal gland, tears, and corn
69                     Both VEGF receptor-2 and HGF receptor proteins were detected mainly in posterior
70                       Binding of InlB to the HGF receptor results in mitogen-activated protein (MAP)
71 ice with inducible deletion of MET, we found HGF receptor signaling to regulate intestinal homeostasi
72 hat beta-catenin forms a complex with c-Met (HGF receptor) that undergoes dissociation because of bet
73   Immunohistochemically, HGF, as well as the HGF receptor (the met gene product), localized to signif
74 chial epithelium, and that expression of the HGF receptor, the c-met proto-oncogene protein, is unifo
75 interaction between the signals activated by HGF receptor tyrosine kinase and TGF-beta receptor serin
76            The Met/Hepatocyte Growth Factor (HGF) receptor tyrosine kinase is oncogenically activated
77                                          The HGF receptor was not different between control and IUGR
78 anchial arches, whilst Met, which encodes an HGF receptor, was expressed by subpopulations of cranial
79 F and InlB elicits similar activation of the HGF receptor, we observed striking differences in downst