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1                                              HHV-6A and HHV-6B have recently been classified as two d
2                                              HHV-6A established a productive infection with CPE, visi
3                                              HHV-6A U51 has been reported to bind to CC chemokines in
4                                              HHV-6A-induced apoptosis was associated with activation
5 ntly infected with Epstein-Barr virus/HHV-4, HHV-6A, HHV-6B, HHV-7, and KSHV.
6     Here, we describe a human herpesvirus 6 (HHV-6A) chemokine, U83A, which binds CCR5 with higher af
7         The genomes of human herpesvirus 6A (HHV-6A) and HHV-6B have the capacity to integrate into t
8 e been correlated with human herpesvirus 6A (HHV-6A) and HHV-6B, the lack of animal models has preven
9                        Human herpesvirus 6A (HHV-6A) is associated with an increased risk of developi
10                        Human herpesvirus 6A (HHV-6A) strain U1102 was previously shown to contain a 1
11 ation is infected with human herpesvirus 6A (HHV-6A), a betaherpesvirus family member.
12                        Human herpesvirus 6A (HHV-6A), a member of the betaherpesvirus family, is asso
13                        Human herpesvirus 6A (HHV-6A), HHV-6B, and HHV-7 are classified as roseoloviru
14                        Human herpesvirus 6A (HHV-6A), HHV-6B, and HHV-7 are classified as roseoloviru
15 e human roseoloviruses human herpesvirus 6A (HHV-6A), HHV-6B, and HHV-7 comprise the Roseolovirus gen
16  T cells infected with human herpesvirus 6A (HHV-6A), the E2F1 protein and its cofactor DP1 increased
17        Roseoloviruses (human herpesvirus 6A [HHV-6A], -6B, and -7) infect >90% of the human populatio
18 tein-Barr virus [EBV], human herpesvirus 6A [HHV-6A], HHV-6B, herpes simplex virus 1 [HSV-1], HSV-2,
19                 Human herpesvirus 6A and 6B (HHV-6A and HHV-6B) are closely related viruses that infe
20               Human herpesviruses 6A and 6B (HHV-6A and HHV-6B) are human viruses capable of chromoso
21               Human herpesviruses 6A and 6B (HHV-6A/6B) are ubiquitous pathogens that persist lifelon
22               Human herpesviruses 6A and 6B (HHV-6A/B) can integrate into the germline, resulting in
23 ry a copy of the human herpesvirus 6A or 6B (HHV-6A/B) in every cell of their body.
24 irus, BK polyomavirus, human herpesvirus 6B, HHV-6A, adenovirus, and Epstein-Barr virus between days
25 oloviruses, human herpesvirus-6A -6B and -7 (HHV-6A, HHV-6B and HHV-7) cause acute infection, establi
26                                 Accordingly, HHV-6A infection resulted in the depletion of both CD4(+
27                                 In addition, HHV-6A induced secretion of interleukin-6 (IL-6), tumor
28 , using phosphonoacetic acid, did not affect HHV-6A/B integration.
29            Increasing seroreactivity against HHV-6A was detectable before the rise of sNfL (significa
30 -6A(GS) B701 ORF (U16) was used to screen an HHV-6A(GS) cDNA library, and two different but overlappi
31 sociation between the rs73185306 C/T SNP and HHV-6A/B chromosomal integration (odds ratio, 0.90 [95%
32                    Human herpesviruses 6A/B (HHV-6A/B) can integrate their viral genomes in the telom
33  As do human herpesvirus 6 variants A and B (HHV-6A and -6B), HHV-7 encodes a homolog of the alphaher
34        Human herpesvirus 6 variants A and B (HHV-6A and HHV-6B) are closely related viruses that can
35 s study, we describe the association between HHV-6A antibodies and the degree of axonal injury in the
36                      The association between HHV-6A serology and sNfL was assessed by stratified t-te
37 overall nucleotide sequence identity between HHV-6A and HHV-6B is 90%.
38 e response was highly cross-reactive between HHV-6A and HHV-6B variants.
39  To assess the temporal relationship between HHV-6A antibodies and sNfL, these ratios were plotted ag
40 ent an analysis of the relationships between HHV-6A and HHV-6B.
41                   Our studies show that both HHV-6A (GS) and HHV-6B (Z-29) can infect highly purified
42 oded miRNAs, although an miRNA(s) encoded by HHV-6A has yet to be reported.
43 ein-Barr virus (EBV), cytomegalovirus (CMV), HHV-6A, HHV-6B, and HHV-8, using quantitative polymerase
44                                 In contrast, HHV-6A induced apoptosis primarily in T(N) and T(CM) CD4
45 sons and the complete viral genome of either HHV-6A or HHV-6B is present in every nucleated cell in t
46          The findings indicate that elevated HHV-6A antibodies both precede and are associated with a
47 al DNA-now proposed to be called "endogenous HHV-6A/B (eHHV-6A/B)." Present in 0.2-3% of humans, this
48  suggest that viral miRNAs are important for HHV-6A and that they may serve as an important therapeut
49 new models have been established, mainly for HHV-6A, which reproduce some pathological features seen
50 cifically on developing an in vivo model for HHV-6A.
51                Organs and cells positive for HHV-6A DNA were the plasma and cellular blood fractions,
52 ne expression in the brain (specifically for HHV-6A), testis, esophagus, and adrenal gland.
53     Surprisingly, although U20 proteins from HHV-6A and -6B share 92% identity, recent studies ascrib
54 B701, found within a 22-kb HHV-6A strain GS [HHV-6A(GS)] genomic fragment and a 3.8-kb SalI subfragme
55 whereas in viral integration, 4 patients had HHV-6A and 17 patients HHV-6B.
56 ll RNA species isolated from cells harboring HHV-6A to identify five novel small noncoding RNA specie
57                      Like all herpesviruses, HHV-6A establishes a lifelong, latent infection in its h
58  objective of this study was to determine if HHV-6A serostatus is associated with the level of sNfL i
59 V-6A/B-specific antibody response.IMPORTANCE HHV-6A and -6B are human herpesviruses that have the uni
60 llular and viral factors that play a role in HHV-6A/B integration.
61 ession, as well as the release of infectious HHV-6A/B from the integrated state.IMPORTANCE The analys
62 of the world's population carries integrated HHV-6A/B genome in every cell of their body.
63 ission of inherited chromosomally integrated HHV-6A/6B (iciHHV-6A/6B) in telomeres.
64 ng and analyzed for chromosomally integrated HHV-6A/B (ciHHV-6A/B).
65 red to as inherited chromosomally integrated HHV-6A/B (iciHHV-6A/B).
66  spontaneous gene expression from integrated HHV-6A/B leads to an increase in antigenic burden that t
67                   Reactivation of integrated HHV-6A virus from individuals' PBMCs as well as cell lin
68  viral genome identified the same integrated HHV-6A strain within members of families, confirming ver
69 re employed genome imaging of the integrated HHV-6A and HHV-6B genomes using whole-genome optical sit
70  (ORF) designated B701, found within a 22-kb HHV-6A strain GS [HHV-6A(GS)] genomic fragment and a 3.8
71                Two viral variants are known: HHV-6A and HHV-6B.
72 ed analysis using novel, fluorescent-labeled HHV-6A or HHV-6B reagents demonstrated strong G1/S phase
73 ression signatures were analyzed, low levels HHV-6A/B gene expression was found across multiple tissu
74 produce human T cells that express the major HHV-6A receptor, CD46.
75 ction, establish latency, and in the case of HHV-6A and HHV-6B, whole virus can integrate into the ho
76  of the human population carries one copy of HHV-6A/B integrated into every cell in their body, refer
77 and provided a model to study the effects of HHV-6A on AIDS progression.
78 ntribute to the less-defined epidemiology of HHV-6A infection.
79 event may have deregulated the expression of HHV-6A or 19q genes or else disrupted telomere function.
80 e sequence of the 3.8-kb genomic fragment of HHV-6A(GS) is nearly identical to the published sequence
81 6-positive individuals inherit the genome of HHV-6A or HHV-6B in the germline, and viral genomes are
82 ongly suggest that MRV is a mouse homolog of HHV-6A, HHV-6B, and HHV-7.IMPORTANCE Herein we describe
83  full reactivation.IMPORTANCE Inheritance of HHV-6A or HHV-6B integrated into a telomere occurs at a
84                        Within-pair ratios of HHV-6A seroreactivity and sNfL were calculated for each
85                          The reactivation of HHV-6A did not affect disease severity and outcome.
86                              Reactivation of HHV-6A is frequent within the immunosuppressed and immun
87                                   Reports of HHV-6A or HHV-6B encephalitis in immunocompetent older c
88 pectives for studying the pathogenic role of HHV-6A in humans.
89 ome, which would support a causative role of HHV-6A.
90 early identical to the published sequence of HHV-6A strain U1102, with minor differences.
91                  In cases, seropositivity of HHV-6A was significantly associated with the level of sN
92                                Serostatus of HHV-6A and Epstein-Barr virus was analysed with a bead-b
93 sociated and cell-free viral transmission of HHV-6A into the peritoneal cavity resulted in detectable
94  this virus in which differential tropism of HHV-6A and HHV-6B may be associated with different disea
95 IMPORTANCE The analysis and understanding of HHV-6A/B genome integration into host DNA is currently l
96 dy responses against EBV and FLU antigens or HHV-6A/B gene products either not expressed or expressed
97 r virus [EBV], and influenza virus [FLU]) or HHV-6A/B antigens.
98 nteroviruses (including human parechovirus), HHV-6A, HPIV-1, and adenoviruses were not detected.
99 ic burden that translates into a more robust HHV-6A/B-specific antibody response.IMPORTANCE HHV-6A an
100  more closely related to the roseoloviruses, HHV-6A, HHV-6B, and HHV-7, than to another murine betahe
101 ate that humanized mice can be used to study HHV-6A in vivo infection and replication as well as aspe
102 nd reproducible cell culture models to study HHV-6A/B integration.
103       While the assay detects both subtypes, HHV-6A and HHV-6B, it is specific and does not cross-rea
104                 Our results demonstrate that HHV-6A and HHV-6B have differential tropisms and pattern
105 itu hybridization, we could demonstrate that HHV-6A/B integrated in most human cell lines tested, inc
106                           We discovered that HHV-6A infection results in a reduction of CD45 on the s
107 se findings provide additional evidence that HHV-6A may play a role in human immunodeficiencies.
108 HHV-6) encephalitis recognizing firstly that HHV-6A and HHV-6B are separate species with differing pr
109 xonal injury, supporting the hypothesis that HHV-6A infection may contribute to multiple sclerosis de
110                         We hypothesized that HHV-6A, like other members of the human herpesvirus fami
111 dds support to previous data indicating that HHV-6A and HHV-6B are distinct herpesvirus species.
112                          Here we report that HHV-6A encodes at least one miRNA, which we named miR-U8
113                                 We show that HHV-6A induces cell division, as measured by 5,6-carboxy
114                                          The HHV-6A(GS) B701 ORF (U16) was used to screen an HHV-6A(G
115          In this study, we characterized the HHV-6A/B integration efficiencies in several human cell
116 a shed light on the genetic structure of the HHV-6A and HHV-6B integration locus, demonstrating the u
117                       Whether release of the HHV-6A genome from the telomere contributed to lymphomag
118 cell-free compartments was predominantly the HHV-6A variant, which has been reported to be neurotropi
119  viral miRNA candidate (miR-U86) targets the HHV-6A IE gene U86, thereby regulating lytic replication
120 -6B lysate, and fewer (33%) responded to the HHV-6A lysate.
121 that the lymphoproliferative response to the HHV-6A variant, which was recently reported to have grea
122 tion and biological characterization of this HHV-6A-specific miRNA is the first step to defining how
123 f viral and cellular factors contributing to HHV-6A/B integration and the screening of drugs influenc
124 e viral and cellular factors contributing to HHV-6A/B integration remain largely unknown, mostly due
125 s a favorable predisposing factor leading to HHV-6A/B integration.
126                No disease has been linked to HHV-6A, whereas HHV-6B may cause encephalitis.
127 NA samples were probed for PCR reactivity to HHV-6A and HHV-6B.
128  CD4(+) and CD8(+) T cells were resistant to HHV-6A-induced apoptosis.
129 tients had a lymphoproliferative response to HHV-6A, which is a significant increase in comparison wi
130 ve compared lymphoproliferative responses to HHV-6A (U1102)-, HHV-6B (Z29)-, and HHV-7 (H7SB)-infecte
131 may be due to differential susceptibility to HHV-6A-induced apoptosis.
132 C)-derived virus in Jjhan cells or wild-type HHV-6A strain U1102 virus in HSB2 cells and are associat
133                 HHV-6B DNAemia was uncommon, HHV-6A DNAemia was not observed, and HHV-7 DNAemia was p
134 tterns of infection for HPDA in vitro, where HHV-6A results in a productive lytic infection.
135                        We next asked whether HHV-6A infection of naive cell lines could lead to integ
136  Here we describe a novel mechanism by which HHV-6A, a member of the human herpesvirus family, may co
137 eal shedding by contacts was associated with HHV-6A or HHV-6B transmission.
138             Second, cells were infected with HHV-6A/B and allowed to grow in bulk for 4 weeks or long
139  T cells were efficiently infected only with HHV-6A.

 
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