ライフサイエンスコーパス: HMW

1 HMW concentration increases the fluid elasticity, thereb

2 HMW fraction had the highest total phenolics, condensed

3 HMW multimers and the closure time with adenosine diphos

4 HMW peaks contained PDE3A1 and PDE3A2, whereas LMW peaks

5 HMW tau derived from CSF of AD patients was seed compete

6 HMW, highly aromatic, alkylated compounds decreased in r

7 HMW-Abeta(1-42) disturbed membrane integrity by inducing

8 HMW-HA triggers hypersensitivity of naked mole rat cells

9 HMW-MAA-specific CARs containing an antigen recognition

10 00, Siluron 5000, 7% HMW + Siluron 1000, 10% HMW + Siluron 1000, and 15% HMW + Siluron 1000; Fluoron

11 Fluid elasticity was optimum using 10% HMW.

12 iluron 1000, 10% HMW + Siluron 1000, and 15% HMW + Siluron 1000; Fluoron GmbH, Ulm, Germany) were mea

13 ation was at a minimum when using 10% or 15% HMW blends.

14 Siluron 1000, Siluron 2000, Siluron 5000, 7% HMW + Siluron 1000, 10% HMW + Siluron 1000, and 15% HMW

15 , 95% CI -0.38 to -0.30, P = 2.0 x 10(-70)), HMW adiponectin (beta = -0.40, 95% CI -0.43 to -0.36, P

16 Body composition (by absorptiometry), HMW adiponectin, and IGF-I were assessed at birth and 4

17 analyzed the reassembly of the most abundant HMW adiponectin species, the octadecamer, following its

18 ntly, under certain conditions, the abundant HMW oAbeta can dissociate into low molecular weight spec

19 plasma HYAL-1 concentration and accelerated HMW-HA degradation.

20 erobic microbial metabolism for accelerating HMW-PAHs removal occurred within sediments after combini

21 In addition, HMW adiponectin could be separated into three distinct o

22 uated the relationship of total adiponectin, HMW adiponectin, and the HMW-to-total adiponectin ratio

23 ovel insight into the mechanisms that affect HMW adiponectin homeostasis.

24 onstituted <2% of the sum of the 15 analyzed HMW PAHs.

25 vely, inhibited the effects of LMW-FGF-2 and HMW-FGF-23 to stimulate FGF-23 promoter activity.

26 se TGFbeta1 and TGFbeta2 as well as FGF2 and HMW-HA.

27 lators of epicardial cell behavior, FGF2 and HMW-HA.

28 invasion in response to TGFbeta2, FGF2, and HMW-HA.

29 ry demonstrated co-localization of FGF23 and HMW FGF2 protein in osteoblasts and osteocytes from Hyp

30 IAIP and HMW-HA colocalized with histones in necrotic tissues and

31 plasmon resonance showed that both IAIP and HMW-HA directly bind to recombinant histone H4.

32 ivo neutralization of histones with IAIP and HMW-HA prevented histone-induced thrombocytopenia, bleed

33 lammatory response after exposure to LMW and HMW agents by specific inhalation challenge test (SIC).

34 Antioxidant capacities of the LMW and HMW fractions were determined using in vitro assays.

35 into low- and high-molecular-weight (LMW and HMW) fractions by Sephadex LH-20 column chromatography.

36 suggest that IAIP, chondroitin sulfate, and HMW-HA are potential therapeutic agents to protect again

37 Total and HMW adiponectin are inversely associated with incident P

38 variants strongly influence plasma total and HMW adiponectin levels in East Asian populations but app

39 Total and HMW adiponectin plasma concentrations were measured befo

40 Both total and HMW-adiponectin decreased, and IL-6 increased with incre

41 t number affects both hmwA transcription and HMW-adhesin production such that as the number of repeat

42 links were discovered in monoclonal antibody HMW species.

43 olecular weight melanoma-associated antigen (HMW-MAA), which is highly expressed on more than 90% of

44 olecular variant of rat DSP, referred to as "HMW-DSP", has been speculated to be a proteoglycan form

45 Unphosphorylated DUE-B HMW complex formation is decreased by PP2A inhibition or

46 se inhibition [% decrease from the baseline (HMW vs. LMW) was 36.9 vs. 74.1% (Abeta40, P<0.05) and 25

47 Because HMW is absent from mature flagella, we propose that HMW

48 m an AD brain contains potentially bioactive HMW tau species, giving new insights into the role of CS

49 ignificant associations between OA caused by HMW agents and work-related rhinitis (OR [95% CI]: 4.79

50 Furthermore, OA caused by HMW agents showed a higher risk of airflow limitation (1

51 utum inflammatory profiles, but OA caused by HMW agents showed higher baseline blood eosinophilia and

52 distinct phenotypic profiles in OA caused by HMW and LMW agents.

53 nd the transient memory impairment caused by HMW oligomers, but did not prevent the persistent cognit

54 On the other hand, memory deficit induced by HMW AbetaOs (10 pmol) was found to be reversible.

55 study was to determine whether OA induced by HMW and LMW agents shows distinct phenotypic profiles.

56 and gelatin was effectively precipitated by HMW fraction.

57 attachment site(s), we further characterized HMW-DSP.

58 Conversely, in children, HMW phthalate metabolites were inversely associated with

59 correlation was detected between circulating HMW-HA and apnoea-hypopnoea-index (r = - 0.195, p = 0.04

60 Our aim was to evaluate circulating HMW-HA and HYAL-1 in OSA.

61 Low levels of circulating HMW adiponectin appear to increase the risk for insulin

62 may be involved in regulation of circulating HMW adiponectin levels and provide novel insight into th

63 ular weight RNA-induced silencing complexes (HMW-RISC) associated with target mRNA.

64 a- and omega-types whereas glutenins contain HMW- and LMW-types.

65 ugmented secretion of mature WPBs containing HMW forms of VWF.

66 In contrast, HMW-FGF-2 stimulated FGF-23 promoter activity in osteobl

67 The PA appear to preferentially crosslink HMW-GS via hydrophobic interactions and hydrogen bonding

68 enic properties of cell culture-derived D123-HMW in guinea pigs.

69 uently, mechanisms responsible for decreased HMW adiponectin in insulin resistance are not well under

70 anbaalenii PYR-1 mutant (6G11) that degrades HMW PAHs but not LMW PAHs.

71 ain lacking three variable regions, Delta123-HMW, elicits broad neutralizing activity against the sev

72 We conclude that conditions that destabilize HMW oAbeta or retard the sequestration of smaller, more

73 We conclude that conditions that destabilize HMW oligomers or retard the sequestration of their small

74 components might be involved in determining HMW and total adiponectin levels.

75 omain inhibits the ability of Tax to disrupt HMW P-TEFb complexes.

76 Western blotting showed that endogenous HMW PDE3A1 was the principal PKA-phosphorylated isoform.

77 ributes to pioglitazone's ability to enhance HMW adiponectin levels, but additional factors likely af

78 tumors, which were not engineered to express HMW-MAA.

79 , 0.66 (0.39-1.13), and 0.40 (0.22-0.74) for HMW and 1.0, 0.74 (0.43-1.25), and 0.35 (0.18-0.65) for

80 data with R(2) = 0.988 for LMW and 0.998 for HMW PAHs.

81 After adjustment for HMW adiponectin levels, the minor allele was associated

82 ith insulin resistance before adjustment for HMW adiponectin levels.

83 d odds ratio and 95% confidence interval for HMW: 1.0, 0.62 [0.29-1.34], 0.30 [0.12-0.74]; total: 1.0

84 nders, associations were not significant for HMW or DEHP metabolites, and results did not change subs

85 ortance of bacterial community structure for HMW-OM degradation.

86 ght sleep study blood samples were taken for HMW-HA and HYAL-1 measurements.

87 0.28) and specificity higher (0.89) than for HMW tests.

88 OC size classes was +/-0.25 per thousand for HMW fraction, +/- 0.54 per thousand for LMW fraction, an

89 variable analyses showed that the values for HMW multimers and CT-ADP at the end of TAVR were each as

90 trations, vesicular Abeta aggregates to form HMW species which are capable of seeding amyloid fibril

91 ative) form and lower molecular weight form (HMW- and LMW-HA, respectively).

92 omatography, high-molecular weight fraction (HMW) using high-performance size-exclusion chromatograph

93 ping PAD than in those remaining event free (HMW: 3.3 versus 3.8 mug/mL, P=0.0005; total: 5.6 versus

94 the mechanisms of neuronal dysfunction from HMW-Abeta(1-42) exposure by measuring membrane integrity

95 two independent IgE-reactive fragments from HMW Bx7 contained repetitive IgE epitopes.

96 <0.05) with temperature and time from LMW>WM>HMW.

97 High-molecular weight HA (HMW-HA) is an important component of the endothelial wal

98 On the other hand, HMW oligomers, but not LMW oligomers, induced oxidative

99 The HfaD HMW form is dependent on HfaA but not on holdfast polysa

100 o PU which could be attributed to its higher HMW PAH concentration.

101 ulfate and high-molecular-weight hyaluronan (HMW-HA) associated with IAIP.

102 lar weight-polycyclic aromatic hydrocarbons (HMW-PAHs) in sediments.

103 Under inflammation or hypoxia, HMW-HA is degraded by hyaluronidases, such as HYAL-1 res

104 This identifies HMW as a novel, evolutionarily conserved component neces

105 To determine if HMW-DSP is the proteoglycan form of DSP and to identify

106 After the initial implantation, HMW multimers normalized in patients without aortic regu

107 sible for low-molecular-weight (LMW) PAHs in HMW PAH-metabolic networks remain poorly understood.

108 way increased the assembly of microRNAs into HMW-RISC, enhanced expression of the glycine-tryptophan

109 ized version (D123A7), into disulfide-linked HMW-like species (Delta123r and Delta123A7r).

110 2/K(b) transgenic mice immunized with Lm-LLO-HMW-MAA-C.

111 phy into low- and high-molecular-weight (LMW/HMW) fractions.

112 erature and time from FL<FG<RL<RG and LMW<WM<HMW for all pHs.

113 The mAb HMW fractions were collected using preparative size-excl

114 n a dose-dependent manner, while maintaining HMW adiponectin.

115 dian total adiponectin and 32% higher median HMW adiponectin concentrations, as well as 16% lower res

116 PFA-CADP profiles mimicked HMW multimers recovery both in transcatheter aortic valv

117 s in vitro reconstituted (rcHC*HA) by mixing HMW HA, serum IalphaI, and recombinant TSG-6.

118 uently form low MW (LMW) oligomers, high MW (HMW) aggregates such as protofibrils, and ultimately fib

119 n of low MW molecules (LMW) to form high MW (HMW) molecules.

120 x was 0.63 for CRP, IL-6, C-peptide, and non-HMW adiponectin and 0.46 for GLDH, indicating good predi

121 centrations of CRP, IL-6, C-peptide, and non-HMW adiponectin were associated with higher risk of HCC

122 AGA-BRF infants, SGA-BRF infants had normal HMW adiponectin and IGF-I levels at 4 months, whereas SG

123 Notably, HMW Abeta decreased more slowly than other forms of Abet

124 morphism explained 4.1% of total and 6.5% of HMW adiponectin levels.

125 ciated form L166P resulted in the absence of HMW DJ-1 complexes.

126 The amount of HMW (32%) and MMW (42%) were more abundant than that of

127 can play a role in regulating the amount of HMW complex present in the cell by decreasing the bindin

128 Cells expressing large amounts of HMW adhesins may be critical for the establishment and m

129 e found that the major IgE-reactive areas of HMW glutenins are located in the repetitive regions of t

130 vascular smooth muscle cells; the binding of HMW-HA to CD44 inhibits cell cycle progression, whereas

131 rotein of 182 kDa, an essential component of HMW-RISC, and improved the ability of microRNAs to repre

132 Plasma concentrations of HMW adiponectin did not change significantly during the

133 striking example is the opposing effects of HMW- and LMW-HA on the proliferation of vascular smooth

134 The damaging effects of HMW-Abeta(1-42) were significantly greater than those of

135 ittle is known about the combined effects of HMW-GS and PINs on dough functional properties.

136 Details are included for the expression of HMW TpsA glycoproteins as polyhistidine-tagged molecules

137 rostructure rather than through formation of HMW aggregates with resultant light scattering.

138 hology was not detected in the hippocampi of HMW oligomer-injected mice.

139 However, incubation of HMW oAbeta in mildly alkaline buffer led to their quanti

140 ereas SGA-FOF infants had elevated levels of HMW adiponectin (particularly SGA-FOF1) and IGF-I (parti

141 Baseline median levels of HMW and total adiponectin were significantly lower in wo

142 dealkylation trends and the overall loss of HMW species observed by FT-ICR MS has not previously bee

143 The results suggest that overexpression of HMW FGF2 increases FGF23/FGFR/KLOTHO signaling to down-r

144 The presence of HMW-multimer defects and a high value for a point-of-car

145 We further investigated the recovery of HMW multimers and monitored these changes with PFA-CADP

146 the time course of the induction/recovery of HMW multimers defects under instantaneous changes in she

147 ents demonstrated that induction/recovery of HMW multimers occurs within 5 minutes.

148 esidual aortic regurgitation, no recovery of HMW multimers was observed.

149 e decrease in shear stress and a recovery of HMW multimers within minutes of implantation which was s

150 Therapeutic reduction of HMW-Abeta(1-42) may prevent AD progression by ameliorati

151 -, thrombin-, or collagen-induced release of HMW-EGF.

152 all population of adherent cells in spite of HMW adhesin specific antibody-mediated immunity.

153 length-dependent epitope better expressed on HMW and VHMW antigens, which bind with higher affinity t

154 take do not significantly increase plasma or HMW adiponectin concentrations in overweight-to-moderate

155 ns high-molecular-weight polyethylene oxide (HMW PEO).

156 ve highly toxic, high-molecular weight PAHs (HMW-PAHs) in coal-tar samples.

157 irreversibly shuffle HCs from pathological, HMW HC-HA to HA oligosaccharides, thereby restoring HC-H

158 hat a rare species of soluble phosphorylated HMW tau is the endogenous form of tau involved in propag

159 iesel exhaust, the model accurately predicts HMW PAH concentrations with R(2) = 0.976 and overestimat

160 ins a high-molecular-weight S-layer protein (HMW SLP) and its low-molecular-weight partner protein (L

161 icular CSF from AD patients contained a rare HMW tau species that exerted a higher seeding activity.

162 duced the migration rate for portions of rat HMW-DSP to the level of DSP.

163 Disaccharide analysis showed that rat HMW-DSP contains glycosaminoglycan chains made of chondr

164 Recombinant HMW Bx7 may be included into the panel of allergens for

165 There was a trend for reduced HMW-HA concentrations in OSA (31.63/18.11-59.25/ng/mL vs

166 adenine DI-phosphate [PFA-CADP]), reflecting HMW multimers changes, could be used to monitor in real-

167 xyl phthalate metabolites (both representing HMW phthalate exposures) were positively associated with

168 lysis of a nationally representative sample, HMW phthalate metabolites, particularly MBzP, were posit

169 y alpha-crystallin and form light-scattering HMW aggregates.

170 ere found in the potentially metal-sensitive HMW (Ag and Ni) and LMW (Tl) pools, whereas the MMW pool

171 ilar magnitude as TE, while increasing serum HMW adiponectin above SHAM and GX animals (p<0.05).

172 enome-wide association study (GWAS) of serum HMW adiponectin levels in individuals of European ancest

173 ng ADIPOQ locus (3q27) were related to serum HMW adiponectin levels.

174 equence reads have barcodes linked to single HMW DNA molecules.

175 aracts use chemical chaperones to solubilize HMW aggregates, while attempts are being made to regener

176 ble amyloid plaques likely sequester soluble HMW oligomers, limiting their potential to dissociate.

177 DAMDEC1, which hydrolyzes pro-EGF to soluble HMW-EGF, that HMW-EGF is active, that proteolytic cleava

178 ctions available: high MW glutenin subunits (HMW-GS) over low MW-GS, and omega-gliadins over alpha- a

179 ns, high molecular weight glutenin subunits (HMW-GS), plays an important role in dough functional pro

180 These observations confirmed that HMW-DSP is the proteoglycan form of DSP (renamed "DSP-PG

181 hydrolyzes pro-EGF to soluble HMW-EGF, that HMW-EGF is active, that proteolytic cleavage of pro-EGF

182 absent from mature flagella, we propose that HMW is not a structural component of the motile axoneme

183 se digestion or NaOH treatment revealed that HMW HA was covalently linked with the heavy chains (HCs)

184 We show that HMW is conserved in species with motile cilia and that,

185 s. 88.0% (Abeta42, P<0.01)], suggesting that HMW Abeta oligomers clear more slowly than other forms f

186 The present study suggests that HMW fraction could be utilised as a source of polyphenol

187 The HMW fractions were IdeS digested, reduced, and analyzed

188 The HMW phthalate metabolite monobenzyl phthalate (MBzP) was

189 nanthrene, anthracene, and fluorene, and the HMW PAHs pyrene, fluoranthene, and benzo[a]pyrene, with

190 -0.43 to -0.36, P = 1.1 x 10(-117)), and the HMW-to-total adiponectin ratio (beta = -0.44, 95% CI -0.

191 total adiponectin, HMW adiponectin, and the HMW-to-total adiponectin ratio with incident symptomatic

192 he Abeta42/40 peptide ratio generated by the HMW beta/gamma-secretase complex indistinguishably from

193 hmwA, which encodes the HMW adhesin, undergoes phase variation mediated by 7-bas

194 terized using recombinant fragments from the HMW Bx7 and synthetic peptides thereof for testing of al

195 HPSEC showed differences in the HMW fraction for different degrees of ripeness and both

196 evealed that among the proteins found in the HMW fraction is VPS35, a latent cytosolic component of t

197 ric reducing antioxidant power (FRAP) in the HMW fractions of 3.5-100 kDa and/or >100 kDa from the co

198 as many as seven covalent cross-links in the HMW fractions, where oxidized histidines react with inta

199 Cwp13 cleaves SlpA in the HMW SLP domain, which we suggest may reflect a role in c

200 yosin complex compared to 11 residues in the HMW striated muscle overlap complex.

201 ses to a known HLA-A2 epitope present in the HMW-MAA(2160-2258) fragment was detected in the HLA-A2/K

202 face hydrophobic residues of CD1 mediate the HMW complex assembly and affect the catalytic activity,

203 Notably, the HMW tau species was also detected in lumbar CSF from AD

204 were identified by mass spectrometry of the HMW complexes.

205 hat it is the carbohydrate components of the HMW exudates that have soil-binding properties.

206 fective in BIR contain reduced levels of the HMW form.

207 Following optimization of the HMW-MAA-specific CAR for expression and function in huma

208 ecognition domain based on variations of the HMW-MAA-specific monoclonal antibody 225.28S and a T-cel

209 bioactive on synapses and microglia than the HMW species from which they are derived.

210 Cs and antioxidant assays indicated that the HMW fractions of peach extracts were major contributors

211 bserved in the LMW fractions relative to the HMW fractions were substantially enhanced following a re

212 emia, a glycoconjugate vaccine made with the HMW polysaccharide coupled to tetanus toxoid (HMW-TT) co

213 Furthermore, within the HMW-GS, PA bound more of the larger x-type than the smal

214 Therefore, HMW HA can serve as both an HC acceptor and an HC donor.

215 tly encountered with the expression of these HMW proteins, namely plasmid instability and protein deg

216 To evaluate this HMW-MAA-specific CAR in patients with metastatic melanom

217 nts lacking IgE to omega(5) -gliadin, and to HMW glutenin in 59%.

218 howed ADAMDEC1 hydrolyzed surface pro-EGF to HMW-EGF that stimulated HeLa EGF receptor (EGFR) reporte

219 samples from 20 subjects who were exposed to HMW (n = 10, Group I) and LMW (n = 10, Group II) at thei

220 Also, subjects exposed to HMW agents showed a significant increase in NL levels of

221 Exposure to HMW and LMW agents by SIC induced a differential nasal a

222 broader spectrum of enzymes upon exposure to HMW-OM, indicating a greater potential to degrade these

223 , correlated with shift of PDE3A from LMW to HMW peaks, and increased co-immunoprecipitation of SERCA

224 Viscosity increased proportionally to HMW concentrations.

225 a positive inhalation challenge response to HMW (n = 544) and LMW (n = 635) agents.

226 e cytolytic, and proliferated in response to HMW-MAA-expressing cell lines.

227 ented data include diagnostic use of sIgE to HMW allergens.

228 MW polysaccharide coupled to tetanus toxoid (HMW-TT) conferred better protection against intranasal c

229 ee high molecular weight glutenin sub units (HMW-GS) were decreased at e[CO2].

230 ease in intracellular high molecular weight (HMW) (>200 kDa) aggregates, which were absent in cells g

231 atient contained both high molecular weight (HMW) (11%) and middle molecular weight (MMW) (28%) adipo

232 MW) (<3.5 kDa) and of high molecular weight (HMW) (3.5-100 kDa and >100 kDa) from cold water, hot wat

233 hesins, including the high molecular weight (HMW) adhesins that mediate attachment to the respiratory

234 High molecular weight (HMW) adiponectin is a predominant isoform of circulating

235 No differences in high molecular weight (HMW) adiponectin were observed between sexes or treatmen

236 tection test for most high molecular weight (HMW) allergens with a pooled sensitivity of 0.74 and spe

237 recovered in distinct high molecular weight (HMW) and low molecular weight (LMW) peaks.

238 racts of AD brain are high molecular weight (HMW) and relatively inactive.

239 An endogenous high molecular weight (HMW) complex (~5 MD) containing beta- and gamma-secretas

240 is associated with a high molecular weight (HMW) complex.

241 1 predominantly forms high molecular weight (HMW) complexes that included RNA metabolism proteins hnR

242 DUE-B forms multiple high molecular weight (HMW) complexes.

243 causative agent was a high molecular weight (HMW) compound and in four cases it was a low molecular w

244 he human genome using high molecular weight (HMW) DNA.

245 MW, 18 kDa) FGF-2 and high molecular weight (HMW) FGF-2 isoforms, which, respectively, activate cell

246 and assembles into a high molecular weight (HMW) form requiring HfaD, but not holdfast polysaccharid

247 othelial cells lacked high-molecular weight (HMW) forms of VWF, demonstrating the importance of BLOC-

248 nd characterized from high-molecular weight (HMW) fractions of an IgG1 monoclonal antibody (mAb).

249 ed into low (LMW) and high molecular weight (HMW) fractions.

250 ular weight (LMW) and high-molecular weight (HMW) fractions.

251 oding for portions of high molecular weight (HMW) glutenin subunits were identified by sequence analy

252 ophoresis showed that high molecular weight (HMW) HA (an average of approximately 3000 kDa) was predo

253 he transfer of HCs to high molecular weight (HMW) HA is a reversible event whereby TSG-6 can shuffle

254 rowth factor 2 (FGF2) high molecular weight (HMW) isoforms in osteoblastic lineage cells in mice resu

255 induction/recovery of high molecular weight (HMW) multimers of von Willebrand factor defect could be

256 confirmed AD elute at high molecular weight (HMW) on nondenaturing size-exclusion chromatography.

257 ugh the conversion of high molecular weight (HMW) physiological alpha-syn conformers into compact, as

258 were analyzed for 15 high molecular weight (HMW) polycyclic aromatic hydrocarbons (PAH), using press

259 mers, hexamers, and higher molecular weight (HMW) species, which are not fully characterized.

260 mers, hexamers, and higher molecular weight (HMW) species.

261 tion of a recombinant high molecular weight (HMW) two-partner secretion exoprotein (generically refer

262 transformed cells, or high molecular weight (HMW), found in muscle and nonmuscle cells.

263 Baseline total and high molecular weight (HMW)-adiponectin and interleukin (IL)-6 levels were meas

264 lecular size [80 kDa, high molecular weight (HMW)], and in a mouse model of tularemia, a glycoconjuga

265 via interacting with high molecular weight (HMW)HA can enhance the anti-inflammatory properties of H

266 126 < MW < 202) and higher molecular weight (HMW, 226 < MW < 302), i.e., smaller and larger than Pyre

267 importantly also the high molecular weight (HMW, 4-6 ring) PAHs that are considerably more toxic tha

268 nt hyaluronan of very high molecular weight (HMW-HA).

269 same pool of complex high molecular weight (HMW-OM).

270 detecting polar and higher molecular weight (HMW; > 400 Da) components abundant in crude and heavy fu

271 DOC into 3 fractions: high molecular weight (HMW; 0.4-10 kDa), low molecular weight (LMW; 50-400 Da),

272 ular-weight (LMW) and high-molecular-weight (HMW) Abeta oligomers differentially impact synapses and

273 ntrations of total or high-molecular-weight (HMW) adiponectin in healthy overweight-to-moderately obe

274 relation to total and high-molecular-weight (HMW) adiponectin using data from a genome-wide associati

275 diponectin, including high-molecular-weight (HMW) adiponectin, and incident peripheral artery disease

276 -6), C-peptide, total high-molecular-weight (HMW) adiponectin, leptin, fetuin-a, and glutamatdehydrog

277 ular-weight (LMW) and high-molecular-weight (HMW) agents have been recognized as causes of occupation

278 We could detect high-molecular-weight (HMW) and low-molecular-weight (LMW) Abeta oligomers in t

279 exists in hypoactive high-molecular-weight (HMW) complexes, which can be activated without apparent

280 mation of an aberrant high-molecular-weight (HMW) form of 2 microm.

281 ved, disulfide-linked high-molecular-weight (HMW) form of the E2 receptor-binding domain lacking thre

282 form or an inactive, high-molecular-weight (HMW) form.

283 a/beta/gamma-gliadin, high-molecular-weight (HMW) glutenin, alpha-amylase inhibitor (AAI) dimer, and

284 A high-molecular-weight (HMW) isoform (110 kDa) that contains an additional large

285 essment of defects in high-molecular-weight (HMW) multimers of von Willebrand factor or point-of-care

286 halates, particularly high-molecular-weight (HMW) phthalates, are suspected to contribute to allergy.

287 nowledge of bacterial high-molecular-weight (HMW) polycyclic aromatic hydrocarbon (PAH) metabolism, t

288 ed by the presence of high-molecular-weight (HMW) protein aggregates or disruption of the lens microa

289 High-molecular-weight (HMW) proteins and low-molecular-weight (LMW) chemicals c

290 Analysis of the high-molecular-weight (HMW) root exudates of both wheat and maize plants indica

291 tly described soluble high-molecular-weight (HMW) species that is found in the postmortem AD brain an

292 oluble phosphorylated high-molecular-weight (HMW) tau, though very low in abundance, is taken up, axo

293 lecular-weight (LMW), high-molecular-weight (HMW), and di-2-ethylhexylphthalate (DEHP) metabolites, c

294 ed platelets released high-molecular-weight (HMW)-EGF, produced by a single cleavage between the EGF

295 e state (as judged by high-molecular-weight [HMW] adiponectin and IGF-I) of infants born small for ge

296 Zn among pools of various molecular weights (HMW: high molecular weight, >670-40 kDa; MMW: medium mol

297 We assessed whether HMW FGF2 expression was altered in the Hyp mouse, a mous

298 While HMW agents act mainly through IgE-mediated mechanisms, L

299 outcomes were observed following dosing with HMW PEO alone.

300 Majority of varieties with HMW-GS combinations of 91kDa+80kDa+78kDa+74kDa PPs showe