ライフサイエンスコーパス: HPC

1 HPC expression and GC-affected DNAm profiles were enrich

2 HPC-PFC interactions have rarely been studied in monkeys

3 HPC-treated wastewater showed higher copy numbers of int

4 HPCs are bipotent liver stem cells that can self-replica

5 HPCs are known to be bipotential cells, capable of formi

6 HPCs are thought to play an important role in liver rege

7 rs of HSCs, hematopoietic progenitor cell-1 (HPC-1), HPC-2, and Lin(-)Sca-1(+)c-Kit(+) subpopulations

8 Cs, hematopoietic progenitor cell-1 (HPC-1), HPC-2, and Lin(-)Sca-1(+)c-Kit(+) subpopulations.

9 We attribute the revived activity with 3-HPC to the alpha-effect, where tandem electronegative at

10 omparison with 2D monolayer culture and a 3D HPC-only model, our 3D triculture model shows both pheno

11 or Prostate Cancer Genetics (combined: 2,836 HPC cases, 2,206 controls of European ancestry).

12 on a single workstation or in parallel on a HPC cluster.

13 Thus, dysfunctional ACC/HPC signaling to the BLA may be a predominant underlying

14 ypropyl cellulose-graft-poly (acrylic acid) (HPC-g-PAA) as a template and was coated with PDA to cons

15 conspicuously show the presence of activated HPC response and proliferation.

16 rent review highlights the role of activated HPCs in both hepatic regeneration and fibrosis during li

17 After HPC transplantation, new red cell antibodies were seen i

18 of context fear conditioning was found after HPC damage.

19 tion of context fear memory was normal after HPC damage up to 30 d after learning.

20 All HPC-7 data sets are freely available both through standa

21 However, not all HPCs in the aorta, vitelline and umbilical arteries, and

22 ed the incidence of such complications among HPC transplant recipients with sickle cell disease.

23 results, either on a single computer, in an HPC cluster or on Google Cloud Compute resources.

24 Here we present an HPC-oriented version of PESCADOR's native text mining to

25 Our findings show that the ACC and HPC projections to the BLA regulate generalized fear in

26 SpaRC can run on both cloud computing and HPC environments without modification while delivering s

27 tions of RBPJ during biliary development and HPC-associated biliary regeneration after hepatectomy.

28 EGA and HPC results were highly correlated for endospore recover

29 n peroxide treatments, measured with EGA and HPC.

30 As platelet-activating factor and HPC share structural semblances and both induce killing

31 on of the portal inflammatory infiltrate and HPC/DR niche in NAFLD will shape future functional studi

32 e coordination and stabilization of mPFC and HPC neuronal sequences during slow oscillations, possibl

33 These findings suggest that the OFC and HPC represent only partially overlapping information and

34 ic brain profiling analysis revealed PFC and HPC changes in various molecular pathways associated wit

35 We recorded neural activity in the PFC and HPC of the trisomic Ts65Dn mouse model of DS during quie

36 and cross-frequency coupling in the PFC and HPC while prefrontal-hippocampal synchronization was str

37 ater (negative control), chlorinated WW, and HPC WW.

38 ed the proliferation of MF CD34(+) cells and HPCs in a dose-dependent fashion.

39 (BM) and the retention of activated HSCs and HPCs but not of quiescent HSCs in their BM niches.

40 rt-term GC pretreatment of human CB HSCs and HPCs promoted SDF-1-CXCR4-axis-mediated chemotaxis, homi

41 opoietic stem and progenitor cells (HSCs and HPCs) remains elusive.

42 population of MF-HPCs, while sparing another HPC subpopulation as well as MF-SCs.

43 nct memories for individual events, anterior HPC and posterior MPFC integrate across memories.

44 that neural representations in left anterior HPC correspond with model predictions of concept organiz

45 quences at the onset of UP states as well as HPC sequences present during sharp-wave ripples.

46 al feeding and metabolic function as well as HPC-dependent memory.

47 (+)Lin(-) cells and all classes of assayable HPCs (colony-forming unit-megakaryocyte [CFU-MK], CFU-gr

48 Theta-band HPC-PFC synchrony was stronger after errors, was driven

49 Before HPC transplantation, three patients had antibodies incom

50 Phase-amplitude coupling between HPC theta and gamma oscillations strongly and specifical

51 and frequency-specific interactions between HPC and PFC of monkeys learning object pair associations

52 normal heart development requires bilateral HPCs to undergo a critical behavioral and phenotypic tra

53 n recordings of neural activity from the BLA-HPC-mPFC circuit during fear conditioning, extinction, a

54 ger after correct trials, was driven more by HPC and increased with learning.

55 e cognitive map or state space, supported by HPC when memory demands are high.

56 oduction of IL-5 and IL-13, but not IL-4, by HPCs.

57 contribution to hereditary prostate cancer (HPC) in independent study populations of the Nashville F

58 stem and in primary ex vivo-cultured CD34(+) HPCs.

59 eration and differentiation of human CD34(+) HPCs.

60 us replication in fibroblasts and in CD34(+) HPCs for latency.

61 ding to its entry receptor, EGFR, in CD34(+) HPCs initiates early events necessary for successful lat

62 required for successful infection of CD34(+) HPCs by HCMV.IMPORTANCE HCMV establishes lifelong persis

63 GFR signaling following infection of CD34(+) HPCs may also contribute to changes in hematopoietic pot

64 gene transfer in thalassemia patient CD34(+) HPCs, which we will implement in the first US trial in p

65 or efficient reactivation in primary CD34(+) HPCs.

66 virus from latently infected primary CD34(+) HPCs.

67 conventional hematopoietic progenitor cell (HPC) compartment.

68 In a human hippocampal progenitor cell (HPC) line, we assessed the short- and long-term effects

69 Haemopoietic progenitor cell (HPC) transplantation can cure sickle cell disease.

70 differentiation of hepatic progenitor cells (HPC) has not been investigated, and little is known abou

71 signaling in hematopoietic progenitor cells (HPC), myeloid-derived suppressor cells (MDSC), and dendr

72 aling affects hepatic progenitor/oval cells (HPCs) and beta-adrenoceptor agonism will expand HPCs to

73 d numbers of hematopoietic progenitor cells (HPCs) at the expense of repopulating hematopoietic stem

74 for the growth of hepatic progenitor cells (HPCs) at the periportal area and subsequent development

75 Foxl1(+) hepatic progenitor cells (HPCs) differentiate into cholangiocytes and hepatocytes

76 lethality in hematopoietic progenitor cells (HPCs) expressing germline mtRUNX1 from patients with AML

77 zing CD34(+) hematopoietic progenitor cells (HPCs) in adults with beta-thalassemia major.

78 In CD34(+) hematopoietic progenitor cells (HPCs) infected in vitro, disruption of the 23- and 19-kD

79 as assayable hematopoietic progenitor cells (HPCs) irrespective of the JAK2 and calreticulin mutation

80 ields of mesenchymal heart progenitor cells (HPCs) move from the anterior lateral plate mesoderm to t

81 Human progenitor cells (HPCs) support human cytomegalovirus (HCMV) latency, and

82 roliferative human hepatic progenitor cells (HPCs) through a cocktail of growth factors and small mol

83 cted CD34(+) hematopoietic progenitor cells (HPCs) through an increase in TGF-beta production.

84 nd committed hematopoietic progenitor cells (HPCs) undergo a gradual, rather than precipitous, transi

85 c homing of haematopoietic progenitor cells (HPCs) via the blood is critical for normal T-cell develo

86 function of hematopoietic progenitor cells (HPCs), and myeloid and erythroid hyperplasia.

87 rent loss of hematopoietic progenitor cells (HPCs), leading to fatal BM aplasia.

88 the fate of the DR hepatic progenitor cells (HPCs), regulating the balance between liver repair and f

89 l entry into CD34(+) human progenitor cells (HPCs), resulting in distinct cellular trafficking and nu

90 ived CD34(+) hematopoietic progenitor cells (HPCs), which include eosinophil lineage-committed progen

91 hly dividing hematopoietic progenitor cells (HPCs), which produce all blood cell lineages.

92 the activation of hepatic progenitor cells (HPCs), which then participate in the restoration of the

93 atopoietic stem (HSCs) and progenitor cells (HPCs).

94 in hematopoietic stem and progenitor cells (HPCs).

95 CB HSCs and hematopoietic progenitor cells (HPCs).

96 s (HSCs) and hematopoietic progenitor cells (HPCs).

97 s (HSCs) and hematopoietic progenitor cells (HPCs).

98 latently in hematopoietic progenitor cells (HPCs).

99 cells (HSCs)/hematopoietic progenitor cells (HPCs).

100 g latency in hematopoietic progenitor cells (HPCs).

101 al human HSC/hematopoietic progenitor cells [HPCs], revealing that several chemokine ligands (CXCL1-4

102 Hydroxypropyl cellulose (HPC) is a biocompatible cellulose derivative capable of

103 transition to affect hematopoietic cluster, HPC, and HSC formation.

104 ories in rats with prior partial or complete HPC damage.

105 st 30 d after training in rats with complete HPC damage.

106 ation of the halogenated phenolic compounds (HPCs).

107 By taking advantage of parallel computing HPC infrastructure, the full collection of MEDLINE abstr

108 ational power of high performance computing (HPC) and cloud resources, we demonstrated that the metho

109 llelization on a high performance computing (HPC) cluster.

110 igently adapt to high performance computing (HPC) cluster.

111 High performance computing (HPC) clusters play a pivotal role in large-scale bioinfo

112 mputing power of high performance computing (HPC) clusters, enabling massive inference on huge datase

113 migrated to any high-performance computing (HPC) environment.

114 dent of existing high-performance computing (HPC) infrastructures.

115 ssively parallel high-performance computing (HPC) platforms is reviewed.

116 nfigure links to high-performance computing (HPC) resources, view and manage output, apply analysis a

117 ing and utilizes high performance computing (HPC) techniques.

118 ironment (XSEDE) high-performance computing (HPC) virtual system, iPlant cloud data storage resources

119 work, including high performance computing (HPC), bioinformatics support, multistep workflows, updat

120 Conclusion: HPC induction holds promise for a variety of application

121 when concept updating is most consequential, HPC is functionally coupled with prefrontal regions.

122 EGA and heterotrophic plate counting (HPC) were used to evaluate the swab/rinse recovery effic

123 Gata factors, compared with Gata2-dependent HPCs.

124 ctivity of LC-CA1 fibers in the mouse dorsal HPC.

125 but striking accumulation of HSCs and early HPCs that are defective in multilineage reconstitution,

126 Significant increases in mature eosinophil, HPC, and eosinophil lineage-committed progenitor cell co

127 SNS catecholamines expand HPCs, which are both targets and sources of Wnt ligands.

128 s) and beta-adrenoceptor agonism will expand HPCs to reduce AILI.

129 ssociated weak inducer of apoptosis expanded HPCs and protected against AILI.

130 e beta-catenin pathway in both the expanding HPCs and the liver tumors but spared its orthodox pathwa

131 h we could trace and delete Foxl1-expressing HPCs and their descendants (Foxl1-Cre;Rosa(YFP/iDTR)-ind

132 n, data management, and scaling analyses for HPC-acknowledging that data science skills will be requi

133 y that governs thymic EC differentiation for HPC homing.

134 We investigated the requirement for Foxl1(+) HPCs in recovery from liver injury in mice.

135 s of Foxl1-Cre;Rosa(YFP/iDTR) mice, Foxl1(+) HPCs and/or their descendants are required for the devel

136 y a discussion of the modern and near-future HPC landscape and the relevant computational traits of t

137 ortal EC population with features that guide HPC homing.

138 betaR) directly controls thymic ECs to guide HPC homing.

139 ted a cohort of 52 SFTs/hemangiopericytomas (HPCs) by whole-exome sequencing (one case) and multiplex

140 Miltefosine [hexadecylphosphocholine (HPC)] is the only orally bioavailable drug for the disea

141 on prefrontal cortex (PFC) and hippocampal (HPC) tissue from Df(16)A(+/-) mice, a model of the 22q11

142 re, our aim is to determine how hippocampal (HPC) object representations are organized and updated to

143 During spatial learning, hippocampal (HPC) place maps reorganize to represent new goal locatio

144 (aWK) similarly in the RSC and hippocampus (HPC) but not in ACA.

145 orbitofrontal cortex (OFC) and hippocampus (HPC), but there is little evidence regarding the relativ

146 tive motivational behaviors and hippocampus (HPC)-dependent memory [2-5].

147 Both hippocampus (HPC) and orbitofrontal cortex (OFC) have been shown to b

148 ed stress effects on the dorsal hippocampus (HPC), basolateral amygdala (BLA), and somatosensory cort

149 in the absence of a functional hippocampus (HPC) context and visual memories are formed rapidly and

150 lation of the amygdala, ACC, or hippocampus (HPC) has been hypothesized to contribute to increased fe

151 Prefrontal cortex (PFC), hippocampus (HPC), and spleen were then collected and analyzed for me

152 end on interactions between the hippocampus (HPC) and other memory storage networks.

153 mple, face-name), for which the hippocampus (HPC) and prefrontal cortex (PFC) are critical.

154 The hippocampus (HPC) has recently emerged as a brain region that integra

155 hough theories suggest that the hippocampus (HPC) is dedicated to represent specific episodes while t

156 ry or neurotoxic lesions of the hippocampus (HPC), medial dorsal striatum (DSM), or lateral dorsal st

157 prefrontal cortex (PFC) and the hippocampus (HPC)-regions critical for sensory associations-of monkey

158 refrontal cortex (mPFC) and the hippocampus (HPC).

159 prefrontal cortex (PFC) and the hippocampus (HPC).

160 en by theta oscillations in the hippocampus (HPC).

161 iPSC-derived hepatic progenitor cells (hiPSC-HPCs) in a 3D environment that depicts the physiological

162 gel-based triculture model that embeds hiPSC-HPCs with human umbilical vein endothelial cells and adi

163 pic and functional enhancements in the hiPSC-HPCs over weeks of in vitro culture.

164 nsion and differentiation of CB CD133(+) HSC/HPC after 8-day culture on a 3D scaffold.

165 tion and differentiation of the CD133(+) HSC/HPC subset from human umbilical CB.

166 tion and differentiation of the CD133(+) HSC/HPC.

167 hematopoietic stem and progenitor cells (HSC/HPC) in ex vivo culture with cytokines and small molecul

168 regulatory programs that guide neonatal HSC/HPC ontogeny, but it creates heterogeneity within these

169 play a role in the maintenance of normal HSC/HPC cell fates, including survival and self-renewal.

170 ne or small molecule on the expansion of HSC/HPC is a laborious and expensive process.

171 These effects of LCP4 on MF HSCs/HPCs were associated with inhibition of JAK-STAT activit

172 Human HPCs from healthy donors and pediatric patients prolifer

173 ut sample treatment, hydroxypropylcellulose (HPC) coated capillary was used to prevent analyte adsorp

174 onally, VAN-specific GHSR knockdown impaired HPC-dependent contextual episodic memory and reduced HPC

175 ) in Gfi-1(-/-) mice did not rescue impaired HPC function or erythropoiesis.

176 hanical stress within the HFR and changes in HPC migratory behaviors.

177 resolution promoter-enhancer interactomes in HPC-7 cells.

178 task goals change, object representations in HPC can be organized in new ways, resulting in updated c

179 Because the MIEP has poor activity in HPCs, it is unclear how viral transactivators are expres

180 cell populations phenotypically enriched in HPCs and HSCs show expression of Gata2, there has been n

181 fies with mature virions and is expressed in HPCs upon virus entry although its expression at the tim

182 h a significant allergen-induced increase in HPCs expressing TSLPR, CD127, and ST2.

183 Interestingly, while DPY30 knockdown in HPCs impaired their differentiation into the myelomonocy

184 the underlying endoderm, we find that MET in HPCs can be accelerated in response to microenvironmenta

185 To test whether MET in HPCs was responsive to purely physical mechanical cues,

186 Stable knockdown of ACTN4 by shRNA in HPCs significantly reduces dexamethasone-mediated induct

187 outcomes in the OFC, as well as by increased HPC-OFC connectivity.

188 These Gata2-independent HPCs exhibit a different functional output and genetic p

189 tories and traction generation of individual HPCs, we find that the onset of MET correlates with a pe

190 uring HCMV reactivation in latently infected HPCs is reexpression of viral major immediate early (MIE

191 xon 4 mRNA is expressed in latently infected HPCs.

192 retion of TGF-beta while protecting infected HPCs from TGF-beta-mediated effects on viral latency and

193 In contrast, we show that infected HPCs are refractory to TGF-beta signaling as another HCM

194 genome-wide data sets, this study integrates HPC-7 data into a genomic resource on par with ENCODE ti

195 an iPS-derived hepatic progenitor cells (iPS-HPCs) and human iPS-derived hepatic stellate cell-like c

196 Hepatocytic maturation in iPS-HPCs was significantly increased in direct co-culture wi

197 antly improved hepatocytic maturation in iPS-HPCs, such as their capacity for albumin production.

198 -HSCs promotes hepatocytic maturation of iPS-HPCs, and indicates that genetically modified iPS-HSCs w

199 Direct co-culture of iPS-HSCs with iPS-HPCs significantly improved hepatocytic maturation in iP

200 r-dependent multipotent progenitor cell line HPC-7 represents a well-recognized cell line model for H

201 he recovery phase to delete Foxl1-Cre-marked HPCs and their descendants.

202 Foxl1-Cre-marked HPCs were required for the development of cholangiocytes

203 t cholangiocytes arose from Foxl1-Cre-marked HPCs.

204 esulted in the depletion of the number of MF HPCs that were JAK2V617F(+) Moreover, the degree of huma

205 itor treatment affects a subpopulation of MF-HPCs, while sparing another HPC subpopulation as well as

206 e three clinical trials of non-myeloablative HPC transplantation at the National Institutes of Health

207 hese finding score against the idea that non HPC memory storage requires a period of interaction with

208 l field potential and unit recordings in NR, HPC, and mPFC in male rats during slow oscillations unde

209 F receptor in the leishmanicidal activity of HPC.

210 ) reduced binding of a fluorescent analog of HPC, 2) decreased TNF-alpha production, and 3) lower mil

211 From this integrated analysis of HPC polarity and mechanics, we propose that normal heart

212 eration by coordinating the fate decision of HPC and clarifies the molecular mechanisms involved.

213 An FH of HPC conveyed the greatest relative risk for all PC subty

214 However, with the move of HPC facilities towards accelerator based architectures,

215 rs to induce goal-directed reorganization of HPC representations and provide a better understanding o

216 supported by molecular dynamic simulation of HPC docking into PAF receptor and by comparison of its l

217 esting intricate links between activation of HPCs and fibrogenesis.

218 We found that the development of HPCs and erythropoiesis, but not HSC function, was rescu

219 anced cell-cycle entry of HSCs, expansion of HPCs, and defects in long-term engraftment, mimicking th

220 CGD, increased cycling of HSCs, expansion of HPCs, and impaired long-term engraftment capacity were f

221 asthmatic responses increased lung homing of HPCs may be orchestrated by TSLP and IL-33 through an IL

222 Infection of HPCs with an HCMVDeltamiR-US5-2 mutant resulted in decre

223 re to TSLP and IL-33 primed the migration of HPCs to a potent progenitor cell chemoattractant, stroma

224 lly, GR and ACTN4 interact in the nucleus of HPCs.

225 ches, and become activated when the pools of HPCs decrease.

226 dition to regeneration, the proliferation of HPCs also determines the appearance of a ductular reacti

227 ng of the role of neuromodulatory actions on HPC place map plasticity.

228 mputing for high throughput Nanopore data on HPC cluster as well as multiple cloud platforms, such as

229 f premalignant mutations and their impact on HPC self-renewal and differentiation remain poorly under

230 e a user to submit their analyses as jobs on HPC schedulers.

231 the functional effects of these cytokines on HPCs.

232 ctures (local desktop, cloud environments or HPC clusters).

233 exposure clustered into 4 trajectories over HPC differentiation, with transient as well as long-last

234 h man was evaluated for FH of hereditary PC (HPC), HBOC, and Lynch syndrome (LS) and for his own PC s

235 interferon signaling that promotes perinatal HPC expansion and sensitizes progenitors to the leukemog

236 microenvironment that stimulates periportal HPC expansion but arrests differentiation, which predisp

237 vated the beta-catenin pathway in periportal HPCs and was responsible for their expansion and de-diff

238 vation of beta-catenin pathway in periportal HPCs is a previously unrecognized mechanism for replenis

239 ST) and/or hydrogenated phosphatidylcholine (HPC) for the encapsulation lactoferrin (LF) was studied;

240 ation process (heterogeneous photocatalysis (HPC)) were used to disinfect urban WW to the same target

241 ptor (GR) in the nucleus of human podocytes (HPCs), a key cell type in the glomerulus critical for ki

242 We show that while posterior HPC and anterior MPFC maintain distinct memories for ind

243 rthermore, we demonstrate that RBPJ promotes HPC differentiation toward cholangiocytes in vitro and b

244 e TP53 pathway and selective depletion of PV HPCs/HSCs.

245 ndent contextual episodic memory and reduced HPC brain-derived neurotrophic factor expression, but di

246 ytokine signaling in mice with X-CGD reduced HPC numbers but had only minor effects on the repopulati

247 nd/or protein inputs (locally or on a remote HPC cluster), predict gene structures and gene structure

248 ata analysis workflows by integrating remote HPC resources and efficient data management with ease of

249 cellular outcomes of these Notch-responsive HPCs in hepatocyte regeneration.

250 mpers competition for CPU time in the shared HPC environment, and jobs submitted during quiet periods

251 ikely initiating molecular events driving SN-HPC tumorigenesis, which places SN-HPC among the growing

252 Sinonasal hemangiopericytoma (SN-HPC) is an uncommon, site-specific, low-grade mesenchyma

253 myoid cell neoplasms were also absent in SN-HPC; thus, the molecular pathogenesis of SN-HPCs remaine

254 riving SN-HPC tumorigenesis, which places SN-HPC among the growing family of beta-catenin-driven mese

255 -HPC; thus, the molecular pathogenesis of SN-HPCs remained unknown.

256 f soft tissue and sinonasal tract origin, SN-HPCs were recently shown to lack recurrent NAB2-STAT6 fu

257 an index case, we analyzed a total of six SN-HPCs for mutations within the amino-terminal region of t

258 ar staining of the tumor cells in all six SN-HPCs.

259 When rats learned one task, HPC and DSL selectively supported spatial navigation and

260 However, when rats learned both tasks, HPC and DSL additionally supported the behavior incongru

261 prevent dissemination of AR markers and that HPC does not offer a clear advantage over chlorination.

262 We have determined that HPC-II exists in an iron(IV) hydroxide state up to pH 11

263 Contrary to expectations that HPC ensembles would represent detailed, even incidental,

264 defining the full task space, we found that HPC ensembles-like those in OFC-failed to distinguish st

265 MPFC) generalizes, other accounts posit that HPC can also integrate related memories.

266 Finally, transcriptome analysis showed that HPCs were more closely related to mature hepatocytes tha

267 ill depend on the available resources at the HPC facility.

268 aging memories and interactions between the HPC and cortical regions.

269 hallmark of information exchange between the HPC and mPFC for memory transfer/consolidation, is not k

270 By contrast, the HPC showed little evidence of learning-related changes i

271 suggest that, in familiar environments, the HPC and OFC may play complementary roles, with the OFC m

272 Exposure to donor antigens from the HPC graft and new red cell antibodies induced by transfu

273 effect on neuroimmune gene expression in the HPC.

274 astically alter the visual appearance of the HPC mesophase in terms of the reflected color, the scatt

275 ordings from single units in area CA1 of the HPC of rats performing the same task.

276 ght of a specific color as a function of the HPC weight fraction.

277 s of memories acquired in the absence of the HPC.

278 ignaling to the amygdala from the ACC or the HPC could underlie overgeneralized fear responses that a

279 The findings suggest that the HPC is not obligatory for these features of long-term me

280 One general idea is that the HPC provides the cognitive map, which is then transforme

281 tions, that underlie the challenges in their HPC realization.

282 taR signalling results in a defect in thymic HPC homing, suggesting the feedback regulation of thymic

283 e the contribution of portal inflammation to HPC differentiation and NAFLD pathogenesis.

284 after errors, was driven primarily by PFC to HPC directional influences and decreased with learning.

285 tion via locus coeruleus (LC) projections to HPC area CA1 (LC-CA1) regulates the overrepresentation o

286 fast and complementary method to traditional HPC for quantitative sterility assurance testing of surf

287 edian age, 47 years) and represented typical HPCs from deep soft tissue with a more aggressive phenot

288 eceptor-deficient macrophages and mice under HPC treatment.

289 n, which inhibits myelopoiesis in uninfected HPCs.

290 elet disorder (FPD), or normal untransformed HPCs.

291 lable algorithms and is adaptable to various HPC environments (QSUB, BSUB, SLURM and others).

292 rgic neurons in either the dorsal or ventral HPC.

293 -derived hormone ghrelin acts in the ventral HPC (vHPC) to increase meal size through interactions wi

294 arallel with behavioral performance, whereas HPC neurons reflected feedback about whether trial-and-e

295 sociative learning may occur in PFC, whereas HPC may guide neocortical plasticity by signaling succes

296 generalization independent of time, whereas HPC projections play a strictly time-dependent role in r

297 rt 433 variants concordantly associated with HPC in both study populations, accounting for 9% of heri

298 ng toward HSC fate but not in all cells with HPC fate, have implications for current reprogramming st

299 torage requires a period of interaction with HPC to establish a stable, precise memory.SIGNIFICANCE S

300 roles in the behavior of HSCs compared with HPCs and is essential for the maintenance of adult hemat

301 mtRUNX1 from patients with AML compared with HPCs from patients with familial platelet disorder (FPD)