ライフサイエンスコーパス: HRV

1 HRV accumulation and replication inside the B lymphocyte

2 HRV also impairs cytokine responses to bacteria via the

3 HRV also significantly reduced phagocytosis of S. pneumo

4 HRV and HEV showed higher population-attributable risk p

5 HRV changes are associated with cardiac diseases.

6 HRV infection induces the phosphorylation of PKD, and in

7 HRV infection of LAD2 MCs induced expression of IFN-beta

8 HRV metrics didn't differ by delivery mode.

9 HRV reduced (small effect size) and P3 wave latency incr

10 HRV significantly reduced cytokine responses to H. influ

11 HRV SNPs tag non-synonymous SNPs (in NDUFA11 and KIAA175

12 HRV was assessed through time domain measures [natural l

13 HRV was assessed under two circumstances: spontaneous sw

14 HRV was measured using a Polar RS800CX heart rate monito

15 HRV-bacterial coinfections synergistically induced IL-17

16 HRV-positive samples were sequenced for phylogenetic ana

17 HRV-specific CD8 T cell epitopes describe here are expec

18 x known HRV types (13 HRV-A, 3 HRV-B, and 10 HRV-C) were identified (A75, C1, and C35 being most freq

19 Twenty-six known HRV types (13 HRV-A, 3 HRV-B, and 10 HRV-C) were identified (A75, C1,

20 nt survival of airborne human rhinovirus-16 (HRV-16).

21 Twenty-six known HRV types (13 HRV-A, 3 HRV-B, and 10 HRV-C) were identified (A75, C1, and C35 b

22 into 3 species: 104 (40.6%) HRV-A; 14 (5.5%) HRV-B, and 138 (53.9%) HRV-C.

23 were classified into 3 species: 104 (40.6%) HRV-A; 14 (5.5%) HRV-B, and 138 (53.9%) HRV-C.

24 .6%) HRV-A; 14 (5.5%) HRV-B, and 138 (53.9%) HRV-C.

25 specific CD8 T cell epitopes from species A (HRV A) and C (HRV C), the most frequent subtypes in the

26 a, but not IFN-lambda, protected MCs against HRV infection.

27 ignalling offered limited protection against HRV.

28 ever, among children 13 to 59 months of age, HRV detection, in particular, HRV-C detection, was assoc

29 All HRV measurements and BRS were found to be negatively cor

30 Among all HRV parameters, MSE scale 5 had the greatest power to di

31 ficant correlations (P = < 0.05) between all HRV(DO) measures to each HRR measure and are presented i

32 s explained most of the correlations for all HRV indices with BP.

33 found consistent but weak correlations among HRV, HR, BRS and ambulatory/office beat-to-beat BP.

34 opulation and could be key for developing an HRV vaccine.

35 an arterial telemetry transmitter to analyze HRV indices, including SD (SD of all pulse-to-pulse inte

36 .04; 95% confidence interval, 1.06-3.91) and HRV (1.54; 1.04-2.30).

37 sis revealed a consistent pattern of DBT and HRV ultradian rhythm (2-5 h) power that uniquely enabled

38 HR and HRV were similar in patients across groups.

39 Those with pneumonia and HRV-C were older (12.1 versus 9.4 months, P = 0.033) and

40 ency in mice increased the M. pneumoniae and HRV loads in cell supernatants and BAL fluid.

41 hether ST2 contributes to M. pneumoniae- and HRV-mediated airway inflammation is poorly understood.

42 argely temperature insensitive, while PV and HRV-C RdRps replication kinetics are activated by temper

43 k In Communities) cohort with heart rate and HRV measures obtained from 2-min electrocardiogram recor

44 interference, we evaluated cases of RSV and HRV codetection by polymerase chain reaction in 2 prospe

45 s, 24.5% (798) and 37.3% (1216) were RSV and HRV positive, respectively.

46 HEVs and HRVs may contribute to the development of AOM in a relat

47 tify a novel host target as a potential anti-HRV therapy.

48 ontrolled trial (CRT) in Matlab, Bangladesh, HRV was included in Matlab's routine immunization progra

49 The association between HRV and CAEP are anticipated to be involved in this mech

50 tudy sought to study the association between HRV and risk of AF.

51 inhibitors of this kinase effectively block HRV replication at an early stage of the viral life cycl

52 = 0.009), with the results mainly driven by HRV-C (12% versus 7%; P = 0.001).

53 modulate sympathovagal balance signified by HRV at either 30 min or 60 min following exercise cessat

54 attern recognition receptor, is triggered by HRV, driving inflammation that can worsen asthma.

55 cell epitopes from species A (HRV A) and C (HRV C), the most frequent subtypes in the clinics of var

56 RNA poliovirus (PV) and human rhinovirus C (HRV-C).

57 emonstrate that similar to epithelial cells, HRVs induce the production of pro-inflammatory cytokines

58 ihood of future PD not captured by classical HRV metrics.

59 RV replication because replication-competent HRV antagonizes the type III IFN response at pre- and po

60 Conclusions: HRV impairs phagocytosis of bacteria in COPD, which may

61 a-ELISPOT positive responses to 23 conserved HRV-specific peptides on peripheral blood mononuclear ce

62 discovery and characterization of conserved HRV-specific CD8 T cell epitopes from species A (HRV A)

63 The carrier fluid containing HRV-16 was nebulized into the sub-hysteresis zone (RH(<E

64 volume and inflammation prospectively dampen HRV in hypercholesterolemic pigs.

65 ificantly (P<0.05 versus baseline) depressed HRV (SD of all pulse-to-pulse intervals over a single 5-

66 Time and frequency domain HRV indices, BRS, office beat-to-beat BP, and heart rate

67 The two-dose HRV rotavirus vaccination program significantly reduced

68 odeled by sines embedded in white noise; (e) HRV changes associated with cardiac diseases and aging c

69 imations of HRV are highly correlated to ECG HRV for both time and frequency domain parameters and pr

70 in the magnetosphere can affect and enhance HRV indices in space, involving an anti-aging or longevi

71 ate measurements, have been used to estimate HRV peripherally but decline in accuracy during increase

72 For HRV(AV3,) lnRMSSD(AV3) and HRR1 were positively correlat

73 ontrols (for HRV-A, 45%; for HRV-B, 10%; for HRV-C, 45% [P = 0.496]).

74 RV-C, 45%) and controls (for HRV-A, 45%; for HRV-B, 10%; for HRV-C, 45% [P = 0.496]).

75 prevalence among cases (for HRV-A, 48%; for HRV-B, 7%; for HRV-C, 45%) and controls (for HRV-A, 45%;

76 ng cases (for HRV-A, 48%; for HRV-B, 7%; for HRV-C, 45%) and controls (for HRV-A, 45%; for HRV-B, 10%

77 Limits of agreement for HRV parameters were higher for the inter-researcher proc

78 PG measurements are a viable alternative for HRV estimation when ECG measurements are impractical.

79 ples were collected monthly and analyzed for HRV, HEV, and HPeV.

80 ty of 31.9% and 20.6%, respectively, and for HRV indices heritability ranged from 11.1% to 20.5%.

81 the HRV species prevalence among cases (for HRV-A, 48%; for HRV-B, 7%; for HRV-C, 45%) and controls

82 HRV-B, 7%; for HRV-C, 45%) and controls (for HRV-A, 45%; for HRV-B, 10%; for HRV-C, 45% [P = 0.496]).

83 licit some clinical relevant differences for HRV parameters.

84 Significant genetic correlation is found for HRV with heart rate (-0.74<rg<-0.55) and blood pressure

85 tematically characterize candidate genes for HRV in live zebrafish embryos.

86 to determine whether CDHR3 is necessary for HRV-C infection of primary airway epithelial cells (AECs

87 However, LAD2 MCs were permissive for HRV replication and release of infectious HRV particles.

88 , 12, 18, 24, and 36 months and screened for HRV and HEV using real-time reverse-transcription quanti

89 6-month period, with molecular screening for HRV and typing by sequencing VP4/VP2 junction.

90 Furthermore, HRV was associated with activity patterns in the ventrom

91 y be the optimal molecular method for future HRV quantification studies and for quantitating other vi

92 leotide polymorphism (SNP) exhibited greater HRV-C infection compared with cells homozygous for the n

93 mains by testing the hypothesis that greater HRV would be associated with better dietary self-control

94 rophylaxis might increase the risk of having HRV infection.

95 HF-HRV was assessed at rest and during a cognitive task pro

96 between ADSCT and both HF-HRV at rest and HF-HRV reactivity.

97 ated relationships between ADSCT and both HF-HRV at rest and HF-HRV reactivity.

98 gnitive task protocol designed to capture HF-HRV reactivity.

99 Higher HF-HRV at rest was significantly related to both more sever

100 re also related to greater suppression of HF-HRV reactivity.

101 S (high frequency heart rate variability; HF-HRV).

102 for 24 h resulted in a dose-dependent higher HRV and lower heart rate at 5 days post-fertilization.

103 self-control, with individuals having higher HRV being better able to downregulate their cravings in

104 Specifically, higher HRV was associated with more effective downregulation of

105 We found that higher HRV was associated with better self-control and improved

106 Specifically, individuals with higher HRV showed both higher overall vmPFC blood-oxygen-level-

107 However, SNA, resting HR, HRV, and atrial (p = 0.03) and ventricular (p = 0.03) pr

108 In HRV villages, 4,808 (73.7%) infants received at least on

109 cumulation is associated with alterations in HRV and the autonomic nervous system.

110 We attained no changes in HRV between the two swallowing events [HR: spontaneous s

111 ck-coupling can be estimated from changes in HRV.

112 first evidence of a ventilatory component in HRV similar to mammalian respiratory sinus arrhythmia in

113 In study 2 declines in HRV were found to mediate the effect of hypo-hydration o

114 epithelial resistance and an 80% decrease in HRV-C infection of the mucociliary epithelium.

115 d inflammation are associated with a fall in HRV in Ossabaw mini-pigs, implying aggravated autonomic

116 hetic response as measured by an increase in HRV, versus the resting-state.

117 ighlighted a novel gene that plays a role in HRV (KIAA1755).

118 ages compared to 2.8 per 100 person-years in HRV villages, indicating an overall effectiveness of 29.

119 lterations in heart rate dynamics, including HRV and complexity.

120 tested for respiratory pathogens, including HRV, using quantitative real-time PCR assays.

121 ivabradine reduces heart rate and increases HRV in zebrafish embryos, as it does in humans; and high

122 hma exacerbations, likely through increasing HRV-C infection levels and protein surface localization.

123 or HRV replication and release of infectious HRV particles.

124 rtilization highlighted genes that influence HRV (hcn4 and si:dkey-65j6.2 [KIAA1755]); heart rate (rg

125 MCs) were infected with HRV or UV-irradiated HRV at increasing multiplicities of infection (MOI) with

126 Twenty-six known HRV types (13 HRV-A, 3 HRV-B, and 10 HRV-C) were identif

127 Last, HRV(AV4) showed positive relationships (P = < 0.05) betw

128 DFA) and multiscale entropy (MSE) and linear HRV parameters were analyzed.

129 Traditional linear HRV parameters and heart rhythm complexity including det

130 The traditional linear HRV, MSE parameters and DFAalpha1 were significantly low

131 term newborns at <72 hours of age to measure HRV, the asymmetry index, and EEG power.

132 ompleted four visits (V1-V4), where a 10-min HRV was recorded.

133 D was 4.10 cases per 100 person-years in non-HRV villages compared to 2.8 per 100 person-years in HRV

134 Twelve nonlinear HRV indices covering the irregularity, complexity, asymm

135 Most (89.0%) of HRV infection episodes were limited to <14 days.

136 examination of longitudinal associations of HRV nonlinear features with cancer prognosis/survival ar

137 we demonstrate a strong and quick binding of HRV types 16 and 1B to monocytes, and slower interaction

138 ntinuous invasion and temporal clustering of HRV types in households (range 5-13 over 6 months).

139 ely play an important role in the control of HRV infection but, surprisingly, HRV-specific CD8 T cell

140 73.7%) infants received at least one dose of HRV.

141 Surveillance System to include two doses of HRV with the standard infant vaccines at 6 and 10 wk of

142 The effect of HRV on macrophage function is unknown.

143 Objectives: To investigate the effect of HRV on phagocytosis and cytokine response to bacteria by

144 was ineffective in attenuating the effect of HRV-16 challenge on lung function, asthma control, and s

145 acilities, we evaluated the effectiveness of HRV against acute diarrhea associated with enterotoxigen

146 The total effectiveness of HRV against ARD among vaccinees was 41.4% (95% CI, 23.2%

147 compare SPG and PPG to ECG for estimation of HRV during an orthostatic challenge performed by 17 subj

148 We find that SPG estimations of HRV are highly correlated to ECG HRV for both time and f

149 e increased accuracy over PPG estimations of HRV.

150 e series were used to estimate the impact of HRV introduction.

151 Significant impacts of HRV on RV+ diarrhea incidences in GSA villages were not

152 imed to examine whether nonlinear indices of HRV can be biomarkers of GC severity.

153 (HRV), and the averaged baseline measures of HRV to heart rate recovery (HRR) following maximal exerc

154 s arrhythmia (RSAc)-an established metric of HRV that reflects cardiac vagal tone.

155 The odds of HRV infection were significantly lower in RSV-infected i

156 urbations in nonlinear dynamical patterns of HRV predict increased GC severity.

157 unding the interpretation of the presence of HRV in nasopharyngeal samples for attribution of a causa

158 ere were no differences in the prevalence of HRV detection among cases and controls (21% versus 20%,

159 n different moments on the quantification of HRV parameters, respectively) derived from short-term re

160 primers and probes for the quantification of HRV, RT-dPCR outperformed RT-qPCR by consistently and ac

161 Thus, the rate of HRV detection was high, with similar degrees of genetic

162 e events were identified among recipients of HRV, but none were considered related to receipt of stud

163 obe set targeting the 5' noncoding region of HRV.

164 ermissive for the replication and release of HRV, which is prevented by exogenous IFN-beta treatment.

165 mutant MDA5, showed increased replication of HRV but not influenza or RSV.

166 all molecules can inhibit the replication of HRV, PV, and FMDV, and therefore, PKD may represent a no

167 ter- and intra-researcher reproducibility of HRV parameters (i.e., the influence of R-R interval sele

168 bronchial epithelium being the major site of HRV infection and replication.

169 onocytes and lymphocytes, and the ability of HRVs to induce the activation of in vitro-cultured human

170 however, among children 13 to 59 months old, HRV detection was more often case associated (21% versus

171 This induction was dependent on HRV replication and required NF-kappaB-mediated signalin

172 little is known about respiratory effects on HRV in lower vertebrates.

173 CDHR3 function and influence on HRV-C infection were investigated by using single-cell t

174 ctions of ST2 during airway M. pneumoniae or HRV infection.

175 .5% during ISS01, P = 0.0110), as were other HRV indices during ISS02 (SDANN, 12.5%, P = 0.0243; Tria

176 Lower overall HRV as well as increased sympathetic/parasympathetic ton

177 months of age, HRV detection, in particular, HRV-C detection, was associated with case status, especi

178 discern the SAN's contribution, we performed HRV analysis on canine electrocardiograms containing bas

179 HR, systolic blood pressure, HRV and skin conductance recovered faster when subjects

180 Blood pressure, HRV, and glucose homeostasis were unaffected.

181 ildren, but had no effect on blood pressure, HRV, or glucose homeostasis.

182 R by consistently and accurately quantifying HRV RNAs across more genotype groups, despite the presen

183 iption-digital PCR (RT-dPCR) for quantifying HRV RNA using genotype-specific primers and probes and a

184 lic blood pressure (p's < 0.01), and reduced HRV (p's < 0.05) when compared to controls.

185 subjects with MetS had significantly reduced HRV, including SDNN and pNN20 in time domain, VLF, LF an

186 e deactivations were associated with reduced HRV, greater perceived effort, and more anxiety.

187 exposure substantially increases HR, reduces HRV, and increases WBC.

188 relations to HRR therefore, averaged resting HRV measures do not strengthen the prediction of cardiov

189 proactive coping styles, while high resting HRV typifies reactive individuals.

190 consistent individual differences in resting HRV across years.

191 Area 25 inactivation also increased resting HRV.

192 ng heart-rate variability (HRV); low resting HRV indicating proactive coping styles, while high resti

193 those who began the trial with lower resting HRV also engaged more in the intervention, possibly as t

194 cessive breeding seasons we measured resting HRV of 57 lactating grey seals.

195 , this endogenous response does not restrict HRV replication because replication-competent HRV antago

196 contrast, exogenous IFN treatment restricts HRV replication, with type I IFN being more potent than

197 Measurements and Main Results: HRV significantly impaired phagocytosis of H. influenzae

198 syncytial virus (RSV) and human rhinovirus (HRV) are the most common viruses associated with acute r

199 The pathogenesis of human rhinovirus (HRV) during severe respiratory disease remains undefined

200 fection of HeLa cells with human rhinovirus (HRV) induced the phosphorylation of PKD.

201 Mycoplasma pneumoniae and human rhinovirus (HRV) infections are linked to neutrophilic inflammation

202 Rationale: Human rhinovirus (HRV) is a common cause of chronic obstructive pulmonary

203 Human Rhinovirus (HRV) is a major cause of common cold, bronchiolitis, and

204 aused by viruses including human rhinovirus (HRV), influenza virus, and respiratory syncytial virus (

205 n serves as a receptor for human rhinovirus (HRV)-C.

206 erity and the frequency of human rhinovirus (HRV)-initiated exacerbations.

207 exposed to bacteria and/or human rhinovirus (HRV).

208 ing the natural history of human rhinovirus (HRV).

209 Human rhinoviruses (HRV) comprise 3 species representing more than 150 genot

210 Human rhinoviruses (HRVs) are a major trigger of asthma exacerbations, with

211 Human rhinoviruses (HRVs) are one of the main causes of virus-induced asthma

212 Human rhinoviruses (HRVs) commonly precipitate asthma exacerbations.

213 Human rhinoviruses (HRVs), human enteroviruses (HEVs) and human parechovirus

214 m multiple individuals with human rotavirus (HRV) and assessed the host epithelial response by using

215 the ISA villages during 3.5 years of routine HRV use, though only Model 2 was statistically significa

216 isplayed slower return of SBP, rMSSD and SD1 HRV indices during recovery from exercise compared to G1

217 ly, we used a mathematical model to simulate HRV under decreased "coupled-clock" regulation.

218 to the relevant HLA I were confirmed for six HRV A-specific and three HRV C-specific CD8 T cell epito

219 trocardiography (ECG) provides gold standard HRV measurements but is inconvenient for continuous acqu

220 Within the current study HRV(DO) displayed the strongest correlations to HRR ther

221 control of HRV infection but, surprisingly, HRV-specific CD8 T cell epitopes remain yet to be identi

222 Short-term HRV recordings (e.g., 10 min long) produce data that usu

223 NS contribute mainly to long- and short-term HRV, respectively; (b) there is evidence suggesting a si

224 We found that HRV explained a significant portion of the individual va

225 Our results suggest that HRV may serve as an easily acquired, noninvasive, and lo

226 across experimental conditions suggests that HRV may serve as both a readily obtainable and robust bi

227 We prove with ISH that HRVs can enter B cells, form their viral replication cen

228 Importantly, we show that HRVs induce the proliferation of B cells, while the addi

229 All the HRV indices lost significance after adjustment for age a

230 iPSC derived cardiomyocytes in vitro and the HRV in vivo.

231 imed to explore the relationship between the HRV molecular subtyping results obtained during severe a

232 d prior to surgical treatments to enable the HRV analysis.

233 A well-marked peak in the HRV signal matching lung inflation cycle was verified in

234 knockdown strongly reduced expression of the HRV receptor ICAM1.

235 in the results of molecular subtyping of the HRV species prevalence among cases (for HRV-A, 48%; for

236 Overall effectiveness of the HRV vaccination program (primary objective) was measured

237 c risk scores account for 0.9 to 2.6% of the HRV variance.

238 ost coral communities today fall outside the HRV, identifying them as novel ecosystems and corroborat

239 Based on our findings, we recommend the HRV data signal processing to be performed always by the

240 ose community composition remains within the HRV showing that it has not transitioned to a novel stat

241 o monocytes, and slower interaction of these HRVs with CD4+ T cells, CD8+ T cells, and CD19+ B cells.

242 Third, HRV replication was significantly reduced in HeLa cells

243 e confirmed for six HRV A-specific and three HRV C-specific CD8 T cell epitopes.

244 Thus, HRV represents a substantial health care and economic bu

245 ovel mechanism whereby MCs may contribute to HRV-induced asthma exacerbations.

246 n primary AECs indicate CDHR3 is critical to HRV-C infection of ciliated cells.

247 ly reduced induction of CXCL1 in response to HRV-bacterial coinfection as well as neutrophil chemotax

248 relative contributions of the ANS and SAN to HRV are not clear, impeding effective treatment.

249 sm for the locus to confer susceptibility to HRV-induced asthma.

250 Specifically, we examined whether total HRV at sedentary rest (measured as the SD of normal-to-n

251 xity parameters were better than traditional HRV parameters to predict pulmonary hypertension.

252 the discriminatory power of the traditional HRV parameters in both net reclassification improvement

253 the conclusion of a human rotavirus vaccine (HRV) cluster-randomized, controlled trial (CRT) in Matla

254 ose schedule of the human rotavirus vaccine (HRV; Rotarix) given early at 6 and 10 wk of age.

255 p < 0.04) increased, whereas HR variability (HRV) decreased (p = 0.009).

256 such as heart rate (HR) and HR variability (HRV), and cardiac cycle phase shifts triggered by the pr

257 parameters and heart rate (HR) variability (HRV) (rMSSD, SD1, HF [ms(2)]) were evaluated before and

258 econd, participant's heart-rate variability (HRV) - a marker of parasympathetic nervous system respon

259 Heart rate (HR), heart rate variability (HRV) [rMSSD, SD1, HF (ms(2))] and skin conductance were

260 ived from non-linear heart rate variability (HRV) analysis and has shown excellent performance in pre

261 ived from non-linear heart rate variability (HRV) analysis, has been proposed as a non-invasive metho

262 ntal correlations of heart rate variability (HRV) and baroreceptor reflex sensitivity (BRS) with ambu

263 Heart rate variability (HRV) and CAEP were evaluated before and after the tests.

264 Heart rate variability (HRV) and pulse rate variability are indices of autonomic

265 Heart rate and heart rate variability (HRV) are mainly determined by the autonomic nervous syst

266 dulation assessed by heart rate variability (HRV) during 14-month expeditions at the German Neumayer

267 ol and resting-state heart rate variability (HRV) in children with Attention-Deficit/Hyperactivity Di

268 Thus, we examined heart rate variability (HRV) in healthy (n = 34), dysmenorrheic (n = 103), and B

269 ng maneuver (ESM) on heart rate variability (HRV) in subjects with neurogenic oropharyngeal dysphagia

270 ro equivalent of the heart rate variability (HRV) in vivo.

271 was evaluated using heart rate variability (HRV) indices, cardiovascular autonomic reflex tests (CAR

272 Heart rate variability (HRV) is a valid and non-invasive indicator of cardiac au

273 rol reflected in low heart rate variability (HRV) is associated with greater risks for cardiac morbid

274 inically assessed by heart rate variability (HRV) is involved in tumorigenesis.

275 CGs including simple heart rate variability (HRV) metrics, commonly used signal processing methods, a

276 Heart rate variability (HRV) provides insight into cardiovascular health and aut

277 ure, heart rate, and heart rate variability (HRV) via 3-h continuous electrocardiograms and collected

278 Participant's heart rate variability (HRV) was measured, and they rated their thirst and mood.

279 ration (SpO(2)), and heart rate variability (HRV) were measured at rest, midpoint of the session, imm

280 as been reported for heart-rate variability (HRV), a marker of physiological flexibility.

281 ociated with reduced heart rate variability (HRV), an indicator of cardiac autonomic control.

282 rate (HR), sleeping heart rate variability (HRV), and sleep timing, could be used to anticipate the

283 ingle day measure of heart rate variability (HRV), and the averaged baseline measures of HRV to heart

284 are associated with heart rate variability (HRV), but candidate genes in these loci remain uncharact

285 tion, as measured by heart rate variability (HRV), or cortical electroencephalogram (EEG) activity.

286 nalyzing astronauts' heart rate variability (HRV).

287 tality and decreased heart-rate variability (HRV).

288 parameter of resting heart-rate variability (HRV); low resting HRV indicating proactive coping styles

289 es define the Historical Range of Variation (HRV) in community structure of the reefs.

290 the pre-vaccine time period in all villages (HRV- and control-only) and Model 2 combined the pre-vacc

291 Overall, 311/3468 (8.9%) collections were HRV positive: 256 were classified into 3 species: 104 (4

292 e-eligible for vaccination in villages where HRV was introduced to that among such children in villag

293 o that among such children in villages where HRV was not introduced.

294 However, the mechanisms through which HRVs modulate the immune responses of monocytes and lymp

295 sure to fine particulate matter (PM2.5) with HRV as an indicator of cardiac autonomic control during

296 blood-derived MCs (CBMCs) were infected with HRV or UV-irradiated HRV at increasing multiplicities of

297 respectively, and were then inoculated with HRV-16 within 72 hours.

298 rol concentrations correlated inversely with HRV and directly with sympathovagal balance, while sympa

299 (35% versus 25%, P = 0.031) than those with HRV-A cases.

300 to provide standard infant vaccines without HRV.