ライフサイエンスコーパス: Haemophilus

1 association with percent Proteobacteria and Haemophilus.

2 ups differentiated according to dominance of Haemophilus.

3 the highest levels of Streptococcus (18.4%), Haemophilus (12.7%) and Neisseria (6.8%).

4 ccus pneumoniae, Neisseria meningitidis, and Haemophilus accounted for 66% (76/115), 25% (29/115), an

5 4 [P4], protein E [PE], protein F [PF], and Haemophilus adhesion and penetration protein [Hap]), EF-

6 etry (MALDI-TOF MS) in the identification of Haemophilus, Aggregatibacter, Cardiobacterium, Eikenella

7 ynebacterium, Staphylococcus, Streptococcus, Haemophilus and Moraxella species in both healthy and ch

8 and Fusobacterium genera in chimpanzees and Haemophilus and Streptococcus in AMH.

9 pathogens, including Pseudomonas aeruginosa, Haemophilus, Aspergillus fumigatus, and nontuberculous m

10 y COPD and was characterized by dominance of Haemophilus at genus level (n = 20), high gammaP:F ratio

11 creased abundance of Psuedomonas, Mycoplana, Haemophilus, Blautia, and Dorea genera in MS patients, w

12 Subsequently, 44 Haemophilus clinical isolates were collected, and 36 iso

13 to the role of P4 as an important factor for Haemophilus colonization and subsequent respiratory trac

14 , and increased that of Actinomyces, Rothia, Haemophilus, Corynebacterium, and Streptococci spp.

15 ficantly higher relative abundances, whereas Haemophilus, Corynebacterium, Cellulosimicrobium and Cam

16 The Haemophilus cryptic genospecies (HCG) causes genital tra

17 ssociated with Proteobacteria (predominantly Haemophilus) dominance, is associated with neutrophil-as

18 ng and a clustering approach, infants with a Haemophilus-dominant microbiota profile at hospitalizati

19 Among infants with lower CCL5 levels, the Haemophilus-dominant microbiota profile was associated w

20 s with rhinovirus-A were more likely to have Haemophilus-dominant microbiota profile, while those wit

21 licidin levels, and increased proportions of Haemophilus-dominant or Moraxella-dominant microbiota pr

22 Haemophilus ducreyi (HD) and Treponema pallidum subspeci

23 from Escherichia coli, Salmonella enterica, Haemophilus ducreyi and Neisseria gonorrhoeae, together

24 Treponema pallidum subsp pertenue and Haemophilus ducreyi are causative agents of cutaneous ul

25 Haemophilus ducreyi causes chancroid and is a major caus

26 Haemophilus ducreyi causes chancroid, a genital ulcer di

27 The finding that Haemophilus ducreyi causes lesions similar to yaws is pa

28 Haemophilus ducreyi causes the sexually transmitted dise

29 Haemophilus ducreyi causes the sexually transmitted dise

30 t (p)ppGpp was required for the virulence of Haemophilus ducreyi in humans.

31 Haemophilus ducreyi, the causative agent of chancroid, h

32 Haemophilus ducreyi, the causative agent of chancroid, h

33 In humans inoculated with Haemophilus ducreyi, there are host effects on the possi

34 cause skin infections in the Pacific islands-Haemophilus ducreyi-as causes of skin ulceration in a ya

35 ecies of bacteria: Neisseria gonorrhoeae and Haemophilus ducreyi.

36 r no changes in the prevalence of Neisseria, Haemophilus, Gemella, Leptotrichia, Solobacterium, Parvi

37 Haemophilus haemolyticus and nontypeable Haemophilus inf

38 Haemophilus haemolyticus has been recently discovered to

39 e also investigated their prevalences in 148 Haemophilus haemolyticus strains, a closely related spec

40 We previously demonstrated that Haemophilus haemolyticus, a closely related human commen

41 ilus, Campylobacter rectus, Catonella morbi, Haemophilus haemolyticus, and Parvimonas micra.

42 ersus Haemophilus-low subgroups, whether the Haemophilus-high group might benefit from treatment stra

43 e gammaP:F ratio was able to distinguish the Haemophilus-high versus Haemophilus-low subgroups, wheth

44 Chlamydia and Haemophilus infections increase NLRP3, caspase-1, IL-1be

45 ekly challenged with a lysate of nontypeable Haemophilus influenza (NTHi), which induces COPD-type in

46 (1% rabies-vaccine recipients), one case of Haemophilus influenza meningitis (1% rabies-vaccine reci

47 s, Rubella, Mumps, trivalent MMR vaccine and Haemophilus influenza type B (HiB) vaccine.

48 cellular pertussis-inactivated poliomyelitis-Haemophilus influenza type b-hepatitis B combination vac

49 rway inflammation in response to nontypeable Haemophilus influenza, which was associated with elevate

50 n is performed using the bacterial strain of Haemophilus influenza.

51 (53.4%), Neisseria meningitidis (13.7%), and Haemophilus influenzae (12.3%) were the predominant isol

52 were Streptococcus pneumoniae (93 [73.8%]), Haemophilus influenzae (18 [14.3%]), and Neisseria menin

53 treptococcus pneumoniae (93 of 143, 65%) and Haemophilus influenzae (19 of 143, 13%).

54 Haemophilus influenzae (232/1670 [13.9%]) was the least

55 representing a reduced relative abundance of Haemophilus influenzae (35.3% [5.5-91.6] vs 6.7% [0.8-74

56 %), Streptococcus pneumoniae ([Sp] 13%), and Haemophilus influenzae ([Hi] 2%).

57 on compositional lasso analysis, we selected Haemophilus influenzae (HI) and Mycoplasma penetrans (MP

58 Haemophilus influenzae (Hi) causes respiratory tract inf

59 y to inhibit catalytic activity of DapE from Haemophilus influenzae (HiDapE) and ArgE from Escherichi

60 pathogens were detected frequently, notably Haemophilus influenzae (mostly nontypeable) together wit

61 It is closely related to nontypeable Haemophilus influenzae (NT H. influenzae).

62 Hia is a major adhesin of nontypeable Haemophilus influenzae (NTHi) and has long been investig

63 Biofilms formed by nontypeable Haemophilus influenzae (NTHI) are central to the chronic

64 Haemophilus haemolyticus and nontypeable Haemophilus influenzae (NTHi) are closely related upper

65 by Streptococcus pneumoniae and nontypeable Haemophilus influenzae (NTHi) are frequently implicated

66 Nontypeable Haemophilus influenzae (NTHI) are Gram-negative bacteria

67 ram-negative commensal bacterium nontypeable Haemophilus influenzae (NTHI) can cause respiratory trac

68 entrations, and pneumococcal and nontypeable Haemophilus influenzae (NTHi) carriage were assessed pre

69 le interactions is important for nontypeable Haemophilus influenzae (NTHi) colonization in the airway

70 Non-typeable Haemophilus influenzae (NTHi) contains the phase-variabl

71 Nontypeable Haemophilus influenzae (NTHi) efficiently colonizes the

72 Nontypeable Haemophilus influenzae (NTHI) forms biofilms in the midd

73 Nontypeable Haemophilus influenzae (NTHi) frequently causes noninvas

74 reptococcus pneumoniae (Spn) and nontypeable Haemophilus influenzae (NTHi) in stringently defined oti

75 Invasive nontypeable Haemophilus influenzae (NTHi) infection among adults is

76 xed Streptococcus pneumoniae and nontypeable Haemophilus influenzae (NTHi) infections (M-OM) and thos

77 Nontypeable Haemophilus influenzae (NTHi) initiates infection by col

78 Nontypeable Haemophilus influenzae (NTHi) is a bacterium that reside

79 Nontypeable Haemophilus influenzae (NTHI) is a commensal bacterial s

80 Nontypeable Haemophilus influenzae (NTHI) is a commensal inhabitant

81 Nontypeable Haemophilus influenzae (NTHI) is a common commensal and

82 Nontypeable Haemophilus influenzae (NTHi) is a Gram-negative, opport

83 Nontypeable Haemophilus influenzae (NTHI) is a leading cause of oppo

84 Nontypeable Haemophilus influenzae (NTHi) is a major bacterial patho

85 Non-typeable Haemophilus influenzae (NTHi) is a major cause of mucosa

86 Nontypeable Haemophilus influenzae (NTHi) is a major pathogen causin

87 Nontypeable Haemophilus influenzae (NTHi) is a pathogen known for be

88 Nasopharyngeal colonization with nontypeable Haemophilus influenzae (NTHi) is a prerequisite for deve

89 am-negative pathogenic bacterium nontypeable Haemophilus influenzae (NTHi) is surface exposed and a l

90 Nontypeable Haemophilus influenzae (NTHI) is the causative agent of

91 Nontypeable Haemophilus influenzae (NTHi) is the leading bacterial p

92 Nontypeable Haemophilus influenzae (NTHi) is the primary cause of ba

93 vaccine against nonencapsulated isolates of Haemophilus influenzae (NTHi) lies in the genetic divers

94 The type IV pilus (Tfp) of nontypeable Haemophilus influenzae (NTHI) mediates adherence, coloni

95 ere we examine the impact of the nontypeable Haemophilus influenzae (NTHI) ModA2 phasevarion on patho

96 Pneumococci and nontypeable Haemophilus influenzae (NTHi) often cocolonize children.

97 Nontypeable Haemophilus influenzae (NTHi) persists in the airways in

98 Nontypeable Haemophilus influenzae (NTHi) was selected as a model pa

99 were calculated for pneumococcal, nontypable Haemophilus influenzae (NTHi), Moraxella catarrhalis, St

100 In nontypeable Haemophilus influenzae (NTHi), the oligopeptide-binding

101 l collapse of biofilms formed by nontypeable Haemophilus influenzae (NTHI), those directed against a

102 ctions with pathogens, including nontypeable Haemophilus influenzae (NTHI), yet the reasons for this

103 diseases are commonly caused by nontypeable Haemophilus influenzae (NTHi).

104 asion by lung microbiota such as nontypeable Haemophilus influenzae (NTHi).

105 E) responses of ccl3(-/-)mice to nontypeable Haemophilus influenzae (NTHi).

106 by respiratory pathogens such as nontypeable Haemophilus influenzae (NTHi).

107 onization strategies employed by nontypeable Haemophilus influenzae (NTHi).

108 evelopment and colonization with nontypeable Haemophilus influenzae (NTHi).

109 l impairment of phagocytosis for nontypeable Haemophilus influenzae (NTHI).

110 ce exposed to cigarette smoke or nontypeable Haemophilus influenzae (NTHi).

111 us (PilA), two major antigens of nontypeable Haemophilus influenzae (NTHi).

112 tly colonized with bacteria [eg, nontypeable Haemophilus influenzae (NTHi)] that cause pulmonary infl

113 s simplex virus [n = 5], adenovirus [n = 5], Haemophilus influenzae [n = 5], and Streptococcus pneumo

114 Haemophilus influenzae also uses an enzyme, GlpQ, to hyd

115 colonization by Streptococcus pneumoniae and Haemophilus influenzae among children has been noted in

116 s pneumoniae and the Gram-negative pathogens Haemophilus influenzae and Moraxella catarrhalis .

117 nization with Gram-negative bacteria such as Haemophilus influenzae and Moraxella catarrhalis was fou

118 animal models of the Gram-negative pathogens Haemophilus influenzae and Neisseria meningitidis We hyp

119 otent antibody responses against nontypeable Haemophilus influenzae and S. pneumoniae, engendering pr

120 vaccination with conjugate vaccines against Haemophilus influenzae and Streptococcus pneumoniae has

121 Haemophilus influenzae and Streptococcus pneumoniae were

122 Haemophilus influenzae and Streptococcus pneumoniae were

123 Haemophilus influenzae and Streptococcus pneumoniae were

124 piratory syncytial virus; RSV) and bacteria (Haemophilus influenzae and Streptococcus pneumoniae) in

125 In the Gram-negative bacteria Haemophilus influenzae and Vibrio cholerae, the master r

126 Immunoglobulin (Ig)A proteases of Haemophilus influenzae are highly specific endopeptidase

127 Neisseria meningitidis (meningococcus), and Haemophilus influenzae are major causes of this invasive

128 ococcus aureus, Streptococcus pneumoniae and Haemophilus influenzae are the major causes of conjuncti

129 Here, using non-typeable Haemophilus influenzae as a model organism, we report th

130 pneumoniae ATCC 49619 (disk and broth), and Haemophilus influenzae ATCC 49247 (disk and broth).

131 and ATCC 25923, Escherichia coli ATCC 25922, Haemophilus influenzae ATCC 49247, and Streptococcus pne

132 umoniae ATCC 49619, and 0.12 to 1 mug/ml for Haemophilus influenzae ATCC 49247.

133 in Southern England immunized with DTaP5/IPV/Haemophilus influenzae b (Hib-TT) vaccine at 2-3-4 month

134 syncytial virus, parainfluenza viruses, and Haemophilus influenzae being the most common.

135 We present the Arg160His mutation of Haemophilus influenzae carbonic anhydrase (HICA), which

136 a meningitidis, Streptococcus pneumoniae, or Haemophilus influenzae cases were confirmed and N. menin

137 Pneumococcus, meningococcus, and Haemophilus influenzae cause a similar spectrum of infec

138 Non-typeable Haemophilus influenzae contains an N(6)-adenine DNA-meth

139 Using Haemophilus influenzae Eagan strains expressing well-cha

140 Haemophilus influenzae exclusively colonizes the human n

141 son of derivatives of a laboratory strain of Haemophilus influenzae expressing either surface-associa

142 Unencapsulated Haemophilus influenzae frequently causes noninvasive upp

143 Staphylococcus aureus in 22% of samples and Haemophilus influenzae in 14%, and both a viral and bact

144 ficiency further affected internalization of Haemophilus influenzae in bronchial epithelial cells.

145 d for TolR is of the periplasmic domain from Haemophilus influenzae in which N- and C-terminal residu

146 ry clearance of Streptococcus pneumoniae and Haemophilus influenzae in wild-type mice but not CD68.hM

147 In this study, we found that nontypeable Haemophilus influenzae induces the association of Itch w

148 he survival rate after a lethal non-typeable Haemophilus influenzae infection in wild-type mice, but

149 Haemophilus influenzae is a Gram-negative human pathogen

150 Haemophilus influenzae is a rare cause of soft tissue in

151 Haemophilus influenzae is a significant causative agent

152 Non-typeable Haemophilus influenzae is an opportunistic pathogen of t

153 lmonary inflammation induced by non-typeable Haemophilus influenzae is significantly attenuated in IR

154 configurations were predicted in nontypeable Haemophilus influenzae isolates based on the presence of

155 e operon was significantly more prevalent in Haemophilus influenzae isolates causing otitis media and

156 -resolution X-ray structure determination of Haemophilus influenzae KDO8PP bound to KDO/VO3(-) and Ba

157 ith's phylogenetic diversity (P = 0.026) and Haemophilus influenzae load (P < 0.0001).

158 we have shown that the C-terminal domain of Haemophilus influenzae LpoA (HiLpoA) has a highly conser

159 , and detailed enzymatic characterization of Haemophilus influenzae LpxH (HiLpxH).

160 Haemophilus influenzae meningitis fluctuated over the su

161 ed as having meningococcal, pneumococcal, or Haemophilus influenzae meningitis in the period 1977-200

162 neumoniae (Spn), Neisseria meningitidis, and Haemophilus influenzae meningitis within the WHO African

163 Phagocytosis of fluorescently labeled Haemophilus influenzae or Streptococcus pneumoniae was a

164 A 10-valent pneumococcal non-typeable Haemophilus influenzae protein D conjugate vaccine (PCV1

165 ts of the 10-valent pneumococcal nontypeable Haemophilus influenzae protein D-conjugate vaccine (PHiD

166 Haemophilus influenzae protein F (PF) is an important vi

167 lls) to the respiratory pathogen nontypeable Haemophilus influenzae resulted in a marked increase in

168 us, while challenge of Trim29(-/-) mice with Haemophilus influenzae resulted in lethal lung inflammat

169 The introduction of the Haemophilus influenzae serotype b (Hib) conjugate vaccin

170 tis in a healthy adult patient, secondary to Haemophilus influenzae serotype f infection, and we revi

171 e show that glucocorticoids and non-typeable Haemophilus influenzae synergistically upregulate IRAK-M

172 n the first detection of 2 cases of invasive Haemophilus influenzae type a (Hia) disease in Italy.

173 e than 95% of all preterm groups, except for Haemophilus influenzae type b (88.1%).

174 t diphtheria, tetanus, pertussis, polio, and Haemophilus influenzae type b (DTaP-IPV-Hib) administere

175 tetanus, pertussis, hepatitis B, polio, and Haemophilus influenzae type b (DTaP-IPV-Hib) and pneumoc

176 A conjugate vaccine containing Haemophilus influenzae type b (Hib) and group C meningoc

177 treptococcus pneumoniae (S. pneumoniae), and Haemophilus influenzae type b (Hib) are three most commo

178 attributable to Streptococcus pneumoniae and Haemophilus influenzae type b (Hib) between 2000 and 201

179 ed (DTaP), inactivated poliovirus (IPV), and Haemophilus influenzae type b (Hib) conjugate vaccine (D

180 Haemophilus influenzae type b (Hib) conjugate vaccine, d

181 The incidence of invasive Haemophilus influenzae type b (Hib) disease has signific

182 ng hospital in Malawi during introduction of Haemophilus influenzae type b (Hib) vaccination and the

183 Haemophilus influenzae type b (Hib) vaccine and the 13-v

184 Protection against Haemophilus influenzae type b (Hib), a rapidly invading

185 ribitol (PRP) polysaccharides extracted from Haemophilus influenzae type b (Hib), and the correspondi

186 om RSV, 12,600 from influenza, and 7200 from Haemophilus influenzae type b and 24,700 diarrheal death

187 become the predominant invasive pathogen as Haemophilus influenzae type b and pneumococcal vaccine u

188 ca, the widespread use of vaccines targeting Haemophilus influenzae type b and Streptococcus pneumoni

189 cal serotypes varied between 83.0% and 100%, Haemophilus influenzae type b between 34.7% and 46.2% (4

190 -tetanus-acellular pertussis-inactived polio-Haemophilus influenzae type b combined vaccine (DTaP-IPV

191 C, W, Y polysaccharide vaccine (PsACWY); or Haemophilus influenzae type b conjugate vaccine (Hib-TT)

192 clinical significance and characteristics of Haemophilus influenzae type b genogroup strains isolated

193 a, tetanus, pertussis, measles, rubella, and Haemophilus influenzae type b vaccine antigens were comp

194 vaccine in 2, meningococcal serogroup A and Haemophilus influenzae type b vaccine each in 1 patient)

195 s-acellular pertussis-inactivated poliovirus/Haemophilus influenzae type b vaccine; age 6/10/ 14 week

196 , and whole-cell pertussis; hepatitis B; and Haemophilus influenzae type b) and pneumococcal vaccine.

197 , tetanus, pertussis, hepatitis B virus, and Haemophilus influenzae type b), yellow fever, measles, a

198 ent vaccine (diphtheria, tetanus, pertussis, Haemophilus influenzae type b, and hepatitis B) at 6, 10

199 include pneumococcus, group B Streptococcus, Haemophilus influenzae type b, and meningococcus vaccine

200 to characterise disease syndromes caused by Haemophilus influenzae type b, pneumococcus, rotavirus,

201 diphtheria, tetanus, pertussis, hepatitis B, Haemophilus influenzae type b, Streptococcus pneumoniae,

202 r the primary series and booster, except for Haemophilus influenzae type b.

203 The bacterium Haemophilus influenzae typically colonizes the human upp

204 The pathogens Vibrio cholerae and Haemophilus influenzae use tripartite ATP-independent pe

205 ribution of solvent to ligand binding in the Haemophilus influenzae virulence protein SiaP.

206 the sialic acid-specific SBP, SiaP, from the Haemophilus influenzae virulence-related SiaPQM TRAP tra

207 ctivity against an efflux-negative strain of Haemophilus influenzae was 4- to 8-fold higher, the comb

208 Neisseria meningitidis (meningococcus), and Haemophilus influenzae was performed by microbiological

209 Haemophilus influenzae was unencapsulated in all 10 epis

210 phtericum and Corynebacterium propinquum and Haemophilus influenzae were significantly more abundant

211 enes, Staphylococcus aureus, and potentially Haemophilus influenzae).

212 Pathogenic bacteria such as Haemophilus influenzae, a major cause of lower respirato

213 ccus aureus, 10 Streptococcus pneumoniae, 10 Haemophilus influenzae, and 5 Escherichia coli isolates

214 antitative PCR for Streptococcus pneumoniae, Haemophilus influenzae, and Moraxella catarrhalis were p

215 c airway bacteria (Streptococcus pneumoniae, Haemophilus influenzae, and Moraxella catarrhalis) were

216 BM: Streptococcus pneumoniae (pneumococcus), Haemophilus influenzae, and Neisseria meningitidis (meni

217 of Streptococcus pneumoniae (pneumococcus), Haemophilus influenzae, and Neisseria meningitidis (meni

218 identification of Streptococcus pneumoniae, Haemophilus influenzae, and Neisseria meningitidis, the

219 as Escherichia coli, Neisseria meningitidis, Haemophilus influenzae, and Pasteurella multocida.

220 phocholine, phosphocholine-modified LPS from Haemophilus influenzae, and phosphocholine-modified prot

221 phylococci (CoNS), Streptococcus pneumoniae, Haemophilus influenzae, and Pseudomonas aeruginosa.

222 eumoniae, Staphylococcus aureus, Nontypeable Haemophilus influenzae, and Pseudomonas aeruginosa.

223 thogens: Pseudomonas aeruginosa, nontypeable Haemophilus influenzae, and Salmonella enterica serovar

224 ptococcus pneumoniae, Moraxella catarrhalis, Haemophilus influenzae, and Staphylococcus aureus.

225 athogens, uropathogenic E. coli, nontypeable Haemophilus influenzae, and Staphylococcus epidermidis I

226 d Moraxella, Corynebacterium, Streptococcus, Haemophilus influenzae, and Staphylococcus.

227 atory tract pathogens Moraxella catarrhalis, Haemophilus influenzae, and Streptococcus pneumoniae, bu

228 is caused mainly by Neisseria meningitidis, Haemophilus influenzae, and Streptococcus pneumoniae, in

229 itis cases caused by Neisseria meningitidis, Haemophilus influenzae, and Streptococcus pneumoniae.

230 eudomonas aeruginosa, Staphylococcus aureus, Haemophilus influenzae, Aspergillus species, Streptococc

231 Neisseria meningitidis (meningococcus), and Haemophilus influenzae, at a sentinel hospital within th

232 tive, Moraxella catarrhalis and non-typeable Haemophilus influenzae, bacterial colonizers and pathoge

233 protection against PC-expressing nontypeable Haemophilus influenzae, but not PC-negative nontypeable

234 , we turned our attention to bacteria, i.e., Haemophilus influenzae, expressing cell-surface adhesins

235 Novel mouse models of Chlamydia, Haemophilus influenzae, influenza, and respiratory syncy

236 a catarrhalis, Streptococcus pneumoniae, and Haemophilus influenzae, is associated with later develop

237 tly from CSF specimens: Escherichia coli K1, Haemophilus influenzae, Listeria monocytogenes, Neisseri

238 ccines with/without protein D of nontypeable Haemophilus influenzae, M. catarrhalis has become a high

239 irways with the pathogenic bacterial strains Haemophilus influenzae, Moraxella catarrhalis, and Strep

240 y associated with bacterial coinfection with Haemophilus influenzae, Moraxella catarrhalis, or Strept

241 We assessed this association for Haemophilus influenzae, Moraxella catarrhalis, Staphyloc

242 Infection with Haemophilus influenzae, Neisseria meningitidis, and Stre

243 cterial pathogens (Streptococcus pneumoniae, Haemophilus influenzae, Neisseria meningitidis, Mycoplas

244 evarion systems in the major human pathogens Haemophilus influenzae, Neisseria meningitidis, Neisseri

245 influenzae, but not PC-negative nontypeable Haemophilus influenzae, relative to wild-type mice.

246 morphism in humans evades TbpA variants from Haemophilus influenzae, revealing a functional basis for

247 tococcus pneumoniae, Neisseria meningitidis, Haemophilus influenzae, S suis) and O tsutsugamushi, Ric

248 Streptococcus (GBS), Listeria monocytogenes, Haemophilus influenzae, S. aureus, Klebsiella spp. and n

249 exacerbations.Measurements and Main Results: Haemophilus influenzae, Staphylococcus aureus, Pseudomon

250 usters characterized by enrichment of either Haemophilus influenzae, Streptococcus, Corynebacterium,

251 zed with the P6 lipoprotein from nontypeable Haemophilus influenzae, using 17-HDHA and aspirin-trigge

252 Working with the bacterium Haemophilus influenzae, we found that MolBC-A functions

253 een H. parainfluenzae and its close relative Haemophilus influenzae, which is also commonly carried w

254 sis initiates with Staphylococcus aureus and Haemophilus influenzae, with later emergence of Pseudomo

255 protective vaccine responses to tetanus and Haemophilus influenzae.

256 ccus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae.

257 ng Streptococcus pneumoniae and non-typeable Haemophilus influenzae.

258 nd in some regions, for all pathogens except Haemophilus influenzae.

259 ssue, which included known pathogens such as Haemophilus influenzae.

260 hylococcus and Gram-negative bacteria and to Haemophilus influenzae.

261 t line of defense against the human pathogen Haemophilus influenzae.

262 ococcus pneumoniae, and 54% for non-typeable Haemophilus influenzae.

263 abundance was associated with qPCR levels of Haemophilus influenzae.

264 Here we identified Haemophilus lipoprotein e (P4) as a receptor for ECM pro

265 The genera Haemophilus (log2 fold change -2.15, P=.003), Dialister

266 matory mediators when compared to the larger Haemophilus-low cluster (n = 121), in which Streptococcu

267 e to distinguish the Haemophilus-high versus Haemophilus-low subgroups, whether the Haemophilus-high

268 t incursions of Streptococcus, Moraxella, or Haemophilus marked virus-associated ARIs.

269 nts whose sputum microbiota was dominated by Haemophilus, Moraxella or Neisseria (HMN) were at 1.5 ti

270 t sputum microbiota, with those dominated by Haemophilus, Moraxella or Neisseria associated with enha

271 cts were uniquely enriched in members of the Haemophilus, Neisseria, Fusobacterium, and Porphyromonas

272 ift from an inoculum in which Streptococcus, Haemophilus, Neisseria, Veillonella and Prevotella speci

273 teobacteria or Firmicutes (phylum level) and Haemophilus or Streptococcus (genus level) dominant.

274 le profiles were marked by high abundance of Haemophilus or Streptococcus.

275 For two exemplar taxa, Haemophilus parainfluenzae and the genus Rothia, metapan

276 Haemophilus parainfluenzae is a nutritionally fastidious

277 Streptococcus mitis, Rothia mucilaginosa and Haemophilus parainfluenzae were the most significantly a

278 ubation of asthmatic airway macrophages with Haemophilus parainfluenzae, a uniquely expanded potentia

279 Haemophilus parasuis causes Glasser's disease and pneumo

280 Haemophilus parasuis is a diverse bacterial species that

281 Haemophilus parasuis is an opportunistic pathogen that c

282 Glaesserella (Haemophilus) parasuis is a commensal bacterium of the up

283 y 56% identical amino acids, both FH-binding Haemophilus proteins similarly interacted with the compl

284 We developed mouse models of Chlamydia and Haemophilus respiratory infection-mediated, ovalbumin-in

285 s, we used CS from three different bacteria (Haemophilus, Salmonella, and Mycobacterium) as our model

286 The Haemophilus sp. was more likely to be dominant in patien

287 patients and found a Streptococcus sp. and a Haemophilus sp. were the most relatively abundant pathog

288 obiota diversity (P = .009) and dominance of Haemophilus species operational taxonomic units (P = .01

289 nebacterium, Streptococcus, Alloiococcus, or Haemophilus species, were observed.

290 alidated using a panel of well-characterized Haemophilus spp.

291 en inflammation pattern in which presence of Haemophilus spp. and Corynebacterium propinquum in MEE w

292 atifying empirical CAP antibiotics to target Haemophilus spp. in addition to Streptococcus spp. in th

293 The occurrence and significance of Haemophilus spp. isolated from the genitourinary tract a

294 ross the study group, Rothia, Neisseria, and Haemophilus spp. were associated with good dental health

295 In particular, Haemophilus spp. were depleted in individuals with RA at

296 Streptococcus spp. and Haemophilus spp. were the most common genera (55% and 12

297 results suggest that Streptococcus spp. and Haemophilus spp., may play an important role in early CF

298 We identified Haemophilus, Streptococcus, Neisseria, and Veillonella s

299 d)T2(low); endotype B, virus(RV-A)microbiome(Haemophilus)T2(low); endotype C, virus(RSV/RV)microbiome

300 lls reacting with antibody against Rothia or Haemophilus were prominent in the early biofilm.