戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ose naltrexone is an effective treatment for Hailey-Hailey disease.
2 role of low-dose naltrexone for treatment of Hailey-Hailey disease.
3 ure of the mutation and clinical features of Hailey-Hailey disease.
4 ansport ATPase, were recently found to cause Hailey-Hailey disease.
5  (SPCA1a) is implicated in breast cancer and Hailey-Hailey disease.
6 s low-dose naltrexone as a novel therapy for Hailey-Hailey disease.
7 a(2+) transporter ATP2C1 have been linked to Hailey-Hailey disease.
8 ory pathway Ca(2+)-ATPase isoform 1), causes Hailey-Hailey disease, a skin disorder.
9 the apparently normal skin of a patient with Hailey-Hailey disease and studied a biopsy of this lesio
10                           Darier disease and Hailey-Hailey disease arise from mutations in the endopl
11 f the human gene, hSPCA1, has been linked to Hailey-Hailey disease, characterized by skin ulceration
12 eplicated in vivo, because clinically normal Hailey-Hailey disease epidermis contained lower Ca2+ sto
13 reen all 28 translated exons of ATP2C1 in 24 Hailey-Hailey disease families and three sporadic cases
14                                              Hailey-Hailey disease (HHD) (MIM 16960) is an autosomal-
15                                              Hailey-Hailey disease (HHD) is a chronic genodermatosis
16                                              Hailey-Hailey disease (HHD) is blistering skin disease,
17 recently identified as the defective gene in Hailey-Hailey disease (HHD), an autosomal dominant skin
18                Familial benign pemphigus, or Hailey-Hailey disease (HHD), is a rare and debilitating
19                                              Hailey-Hailey disease (HHD, MIM 16960) is inherited in a
20                               Characteristic Hailey-Hailey disease histopathology was observed by ele
21 appeared normal and exhibited no evidence of Hailey-Hailey disease; however, aged heterozygotes had a
22  acid identity with the protein defective in Hailey Hailey disease, hSPCA1.
23                                              Hailey-Hailey disease is a severe genetic blistering dis
24                                              Hailey-Hailey disease is an autosomal dominant skin diso
25 levels comparable to other epithelial cells, Hailey-Hailey disease keratinocyte Golgi Ca2+ refill is
26                                        Since Hailey-Hailey disease keratinocytes can assemble normal-
27 und that the abnormal Ca2+ signaling seen in Hailey-Hailey disease keratinocytes correlates with decr
28 mbly of intercellular junctions, we cultured Hailey-Hailey disease keratinocytes in medium containing
29      The blisters in the inherited disorder, Hailey-Hailey disease, may be caused by defective epider
30                                              Hailey-Hailey disease (MIM16960) is a blistering skin di
31  outpatient clinic of 3 patients with severe Hailey-Hailey disease recalcitrant to at least 4 therapi
32                                              Hailey-Hailey disease severely affects patient quality o
33 esent herein 3 cases of patients with severe Hailey-Hailey disease treated with low-dose naltrexone w
34 y in yeast and provides new perspectives for Hailey-Hailey disease treatment.