ライフサイエンスコーパス: HeLa cell

1 scope on a coverslip, or uptaken in a single HeLa cell.

2 CRs) that inhibit ZIKV infection in Vero and HeLa cells.

3 luorescence recovery after photobleaching in HeLa cells.

4 the characterization of protein C termini of HeLa cells.

5 nes that controlled MHC-I Ag presentation in HeLa cells.

6 r C (Tat-C) or their position 57 variants in HeLa cells.

7 ion in both patient cells and CLN5 knockdown HeLa cells.

8 the apoptotic phenotype induced by PaTox in HeLa cells.

9 nucleus of human cells, with an emphasis on HeLa cells.

10 Xenopus laevis oocytes, HEK-293T cells, and HeLa cells.

11 rotein involved in invasion of nonphagocytic HeLa cells.

12 y and function in GRASP65/55-double depleted HeLa cells.

13 s the 10-23 DNAzyme for imaging of Mg(2+) in HeLa cells.

14 ich exhibited high selectivity toward LDs in HeLa cells.

15 nd an EPAC activator mimicked this effect in HeLa cells.

16 otoxic activity (GI(50) = 116 ug/mL) against HeLa cells.

17 or HL6/QD complexes are cytotoxic to A549 or HeLa cells.

18 sease patient fibroblasts and CLN5-deficient HeLa cells.

19 opy in neurons, human fibroblasts, U2OS, and HeLa cells.

20 pproximately 120 nm x approximately 80 nm in HeLa cells.

21 e that OA induces mature miR-7 production in HeLa cells.

22 pol II pausing index on a subset of genes in HeLa cells.

23 autophagosome recruitment to mitochondria in HeLa cells.

24 s recovered from the cytoplasm of individual HeLa cells.

25 the newly formed virions are able to infect HeLa cells.

26 by selective labeling of lipid and hCAII in Hela cells.

27 emonstrated improved apoptosis efficiency in HeLa cells.

28 teracting with TCTP in human cervical cancer HeLa cells.

29 f-organization of the actin cortex in living HeLa cells.

30 n is induced upon siRNA knock-down of SMN in HeLa cells.

31 aurosporine-induced (intrinsic) apoptosis in HeLa cells.

32 cing of untreated, apoptotic, and recovering HeLa cells.

33 y tryptic digestion of protein extracts from HeLa cells.

34 viability-based genome-wide siRNA screen in HeLa cells.

35 EGF-mediated activations of both kinases in HeLa cells.

36 ed to HAE cells but also includes HEK293 and HeLa cells.

37 en protein-coding and lincRNA genes in human HeLa cells.

38 unoglobulin M (micros), is well-tolerated in HeLa cells.

39 them to have generally low cytotoxicities in Hela cells.

40 ective variant reduces cell proliferation in HeLa cells.

41 red blood cell lysis or cytotoxicity against HeLa cells.

42 pective G1/S and G2/M phases in synchronized HeLa cells.

43 ts (pH 5.0-5.5) after 5 h of incubation with HeLa cells.

44 to human glioblastoma A172 and synchronized HeLa cells.

45 macrophages and reduced plaque formation in HeLa cells.

46 during and reactivation from persistence in HeLa cells.

47 abling the formation of gel fibers on living HeLa cells.

48 miRNAs in total RNA extracted from 293T and HeLa cells.

49 nretrovirus Gag localizing to the nucleus of HeLa cells.

50 hat DF proteins do not induce translation in HeLa cells.

51 Pi-mediated cellular response in HEK293 and HeLa cells.

52 the trafficking of H2B into mitochondria of HeLa cells.

53 ring of endogenous formaldehyde secretion in HeLa cells.

54 bunit of PP2A (PPP2R1A) (to inhibit PP2A) in HeLa cells.

55 n filament disassembly in the CAP1 knockdown HeLa cells.

56 as microRNA inhibitors of hsa-miR-15b-5p in HeLa cells.

57 eplication of H1N1, but not H5N1, viruses in HeLa cells.

58 droxycoumarin dye was demonstrated in living HeLa cells.

59 (PM) cholesterol and PI(4,5)P(2) content in HeLa cells.

60 ndrial depolarization-mediated cell death in HeLa cells.

61 cterized the Golgi structure and function in HeLa cells after exposure to hydrogen peroxide (H(2)O(2)

62 on was less potent in causing vacuolation of HeLa cells, AGS gastric cells, and AZ-521 duodenal cells

63 In HeLa cells, all three mutations also localized to region

64 tem, and the assessment of cell viability in HeLa cells allowed us to classify eleven variants as pro

65 Here, using HEK293T and HeLa cells, along with immunoprecipitation and immunoblo

66 conserved, as depletion of Snx1 and Snx2 in HeLa cells also led to greater overlap of Rme-8 and Hrs

67 comet) Now we have isolated from extracts of HeLa cells an ATP-dependent factor that releases Cdc20 f

68 ces an increase in Golgi cisternal number in HeLa cells and delays the cell surface appearance of the

69 ty through the regulation of TSC dynamics in HeLa cells and Drosophila.

70 Green tag into the endogenous MBNL1 locus in HeLa cells and established a flow cytometry-based screen

71 NM2A associates with the plasma membrane of HeLa cells and fibrosarcoma cells independently of F-act

72 oteins in WT and CRISPR-mediated GNPTAB(-/-) HeLa cells and identified changes in numerous glycoprote

73 red using RPAD from human cervical carcinoma HeLa cells and mouse C2C12 myoblasts led to two surprisi

74 rans-Golgi network (TGN) of both transfected HeLa cells and mouse primary neurons.

75 on two parental cell lines (e.g., wild-type HeLa cells and MRP1-overexpressing HeLa-R cells).

76 We used autoPOTS to analyze 1-500 HeLa cells and observed only a moderate reduction in pep

77 lease, we knocked down ATP6V0C expression in HeLa cells and observed that ATP6V0C depletion impairs V

78 devices supported physiological cultures of HeLa cells and ovarian tissues in vitro, with superior o

79 major site for alpha-secretase processing in HeLa cells and primary neurons and indicate that both AP

80 we demonstrate that BACE1 exits the Golgi in HeLa cells and primary neurons by a pathway distinct fro

81 spatial resolution region is demonstrated on HeLa cells and rat brain tissue, monitoring molecules th

82 We used HeLa cells and screened 231 FDA-approved oncology and na

83 ior differs, when fully informed by data for HeLa cells and show that model predictions remain consis

84 toxicity of synthesized persulfide donors on HeLa cells and the cytoprotective ability in the highly

85 s of cell-surface Gb3, and both AHR knockout HeLa cells and tissues from Ahr knockout mice displayed

86 ty and LD sizes during glucose starvation of HeLa cells and transforming growth factor beta-induced e

87 ctivity, the UBE2T genes were knocked out in HeLa cells and U2OS cells.

88 ic accumulation of ubiquitinated proteins in HeLa cells and wild-type mouse cortical neuron cultures.

89 In HeLa cells and Xenopus oocytes, we show that Cx43-G8V, C

90 tandem mass spectrometry (MS/MS) from single HeLa cells, and 874 protein groups were identified using

91 199 localized with ERGIC and COPI markers in HeLa cells, and electron microscopy of a liver biopsy sh

92 sensors in the cytosol compared to the ER of HeLa cells, and identify the formation of oxidative olig

93 ophages, human monocyte-derived macrophages, HeLa cells, and mouse embryonic fibroblasts.

94 ated independently of PERK in both LNCaP and HeLa cells, and our further examination revealed that AT

95 ll culture medium as well as noncytotoxic in HeLa cells, and their spectroscopic and photophysical pr

96 , exhibited substantial cytotoxicity towards HeLa cells, and was a highly sensitive substrate of the

97 er, the two techniques did not agree for the HeLa cells, and we postulate potential reasons for this.

98 NF764 on the glucocorticoid receptor (GR) in HeLa cells as a model system.

99 Using cell extracts from HeLa cells, as well as transfection of luciferase replic

100 Incubating HeLa cells at 39.5 degrees C stimulated modest p38 activ

101 n E, as lipid peroxidation was suppressed in HeLa cells both under basal conditions and in the presen

102 tein does not affect apoptotic cell death in HeLa cells but enhances necroptosis in L929 cells.

103 expected, SRSF2 and SRSF5 shuttle poorly in HeLa cells but surprisingly display considerable shuttli

104 d for the productive infection of HEK293 and HeLa cells by AAV2, whereas NS4 is sufficient for viral

105 f the mitochondrial H2O2 reaction network in HeLa cells by creating a kinetic model of this system an

106 on the microtubule network was visualized in HeLa cells by immunofluorescence microscopy using Bimole

107 emonstrated that capsule impeded invasion of HeLa cells by masking the bacterial cell wall-anchored p

108 f transient modulation of TFEB expression in HeLa cells by measuring the cytosolic Ca(2+) response af

109 mechanistically the ICWs elicited in single HeLa cells by the tandem bubble-induced jetting flow in

110 that abortive replication of H1N1 viruses in HeLa cells can be circumvented upon the introduction of

111 The Mg(2+) or Zn(2+) in HeLa cells can be detected using both confocal imaging a

112 vity, or knockout of HDAC4 from HEK-293T and HeLa cells, caused a defective response to IFN-alpha.

113 Furthermore, knockdown of IQGAP1 in THP1 and HeLa cells causes significantly more IFN-beta production

114 In the late stage, HeLa cells change from proliferating to migratory.

115 mammalian (rat) brain, cultured human cells (HeLa cells), chicken brain, chicken erythrocytes, and th

116 We found that HeLa cell chromatin-associated nascent pre-mRNA (CA-RNA)

117 ty-purified MRP ribonucleoprotein (RNP) from HeLa cells cleaves the human pre-rRNA in vitro at at lea

118 By using time-lapse confocal microscopy of HeLa cells co-expressing GFP-tagged GW182, we demonstrat

119 Studies in HeLa cells confirmed that global H3K4me3 levels are ROS-

120 an altered expression in RALY-down-regulated HeLa cells, consequently causing impairments in transcri

121 ed ~170 to ~620 proteoforms from ~70 to ~770 HeLa cells containing ~10 to ~115 ng of total protein.

122 ospitable to implanted cells and showed that HeLa cells could survive for up to a week using this met

123 fluorescent glucose analogue (NBDG) between HeLa cells coupled by Cx26 gap junctions.

124 eurolastin also localizes to mitochondria in HeLa cells, cultured neurons, and brain tissue.

125 phthalate) substrate is proposed for in-situ HeLa cell culturing and real-time detection of the relea

126 duced the secretion of effector proteins and HeLa cell cytotoxicity.

127 Knockout of UBQLN2 expression in HeLa cells decreased turnover of ATP6v1g1, while overexp

128 ase-based DENV-2 protease reporter system in HeLa cells (DENV2proHeLa) was employed to determine the

129 e anchorage-independent growth capability of HeLa cells depleted of Cat-1 expression.

130 In HeLa cells depleted of clathrin by siRNA, activated PAR4

131 cells and induced syncytia at neutral pH in HeLa cells despite the expression of A56/K2.

132 models on multiple proteomics datasets and a HeLa cell digest case study consisting of more than a mi

133 the proteomics analysis of 10 ng of tryptic HeLa cell digest.

134 ssion of BK(Ca) with the BK-beta1 subunit in HeLa cells doubled the density of BK(Ca) in mitochondria

135 de of action, peroxide treatment of parental HeLa cells elevated phospho-Met levels whereas antioxida

136 description of LDs in their native state in HeLa cells enabled by cellular cryoelectron microscopy.

137 al tubular epithelial cells, 293T cells, and HeLa cells enabled the infection of these cells; exposur

138 In addition, LIMCH1-depleted HeLa cells exhibited a decrease in the number of actin s

139 AHR knockout HeLa cells exhibited significantly reduced levels of cel

140 f the DNA-protein cross-link in vitro and in HeLa cells exposed to alkylating agent methylmethanesulf

141 emonstrated that when gap junction-deficient HeLa cells expressed the N14K and D50N mutants, they und

142 HeLa cells expressing Cx43 or Cx50 were loaded with Fluo

143 illustrate the approach using the example of HeLa cells expressing paxillin-EGFP to visualize focal a

144 Using membranes from HeLa cells expressing SERT and intact rat basophilic leu

145 We subsequently purified an activity from HeLa cell extracts and identify this as the E3 ubiquitin

146 OH in vitro; depletion of endogenous RtcB in HeLa cell extracts reduces U6/L1 RNA ligation efficiency

147 ing electrophoretic mobility shift assays in HeLa cell extracts, we show that OA treatment disrupts p

148 spin-labeled protein variants in E. coli and HeLa cell extracts.

149 beled Bax variants were reasonably stable in HeLa cell extracts.

150 the pH of the intracellular compartments of HeLa cell from the pH dependent ratiometric calibration.

151 To assay HeLa cells from an attached starting state, we culture a

152 Although knocking down Cat-1 prevents HeLa cells from forming colonies in soft agar, when paxi

153 fferent time points following the release of HeLa cells from G1/S phase.

154 the ability to label differentially necrotic HeLa cells from living cells.

155 ty, as well as lower rates of cell growth in HeLa cells, further supporting a role for cell prolifera

156 HeLa cells growth in the very close vicinity of the work

157 r 1 (N-CoR) that accumulated in synchronized HeLa cells in late G2 phase and mitosis.

158 dtii (ChRe) and Euglena gracilis (EuGr), and HeLa cells in their native growth media.

159 HeLa cells in which both variants were knocked out (ORP1

160 l of Hutchinson-Gilford progeria syndrome in HeLa cells in which increased progerin expression leads

161 n cargoes in permeabilized mouse neurons and HeLa cells, in a manner that can be rescued by RNA.

162 verexpression of ATP6v1g1 in UBQLN2 knockout HeLa cells increased autophagosome acidification, sugges

163 The SILAC analyses of HeLa cells indicated that assembly of RCV, comprising F(

164 ant in WI-38 diploid fibroblasts and weak in HeLa cells, indicating profound differences in the regul

165 (IC50) values that were less than 20 muM in HeLa cells, indicating that these compounds represent a

166 previously identified abortive infections in HeLa cells infected with herpes simplex virus 1 (HSV-1)

167 L-type prostaglandin D synthase (L-PGDS) in HeLa cells inhibited recycling of the prostaglandin D(2)

168 In vitro tests show that HeLa cells irradiated with muLights undergo a 70% decrea

169 g this construct, detecting Zn(2+) in living HeLa cells is demonstrated.

170 w that the ICP0-independent loss of IFI16 in HeLa cells is dependent in part on the activity of the v

171 ytotoxic compound monomethyl-auristatin-F to HeLa cells is increased several fold in presence of Ca(2

172 demonstrate that DOCK5 recruitment to FAs in Hela cells is restricted by GIT2, an established regulat

173 Using a chemically-fixed HeLa cell labeled with fluorescent core-shell nanopartic

174 show that knocking down Cat-1 expression in HeLa cells leads to a reduction in Akt activation, which

175 Depletion of YTHDF2 in HeLa cells leads to the delay of mitotic entry due to ov

176 Expression of this mutant motor in HeLa cells led to a dramatic reorganization of cortical

177 ation of 1 000 protein groups for a standard HeLa cell line digest.

178 e molecular function of A6, we established a HeLa cell line that inducibly expressed VACV-A6, which a

179 this possibility, we utilized a GNPTAB(-/-) HeLa cell line that lacks lysosomal hydrolases in endocy

180 , whereas leaves were effective only against HeLa cell line.

181 which led to estimates of low-mM ribose in a HeLa cell line.

182 combination (HR) in FA cells, we constructed HeLa cell lines containing the UBE2T AluYa5 elements and

183 PR-associated protein 9 was used to generate HeLa cell lines in which ENT expression was limited to E

184 igate the effects of biological variation in HeLa cell lines using a systems-wide approach.

185 To test this idea, transformed HeLa cell lines were created with fluorescent cargos (mC

186 - 0.12 and 82.38 +/- 0.00% against HT-29 and HeLa cell lines, respectively, at 25 ug/mL.

187 ability and adhesion capacity in NIH-3T3 and HeLa cell lines, revealing that Pvfp-5beta has no cytoto

188 in hamster (CHO), monkey (COS7), and human (HeLa) cell lines.

189 aging and quantification are demonstrated in HeLa cells loaded with nanosensors and their responsiven

190 SLC45A2 expressed ectopically in HeLa cells localizes to lysosomes and raises lysosomal p

191 Conversely, MARCH6-deficient HEK293 and HeLa cells lost their ability to degrade squalene monoox

192 The Ub-tetrazole probe was also assessed in HeLa cell lysate and showed robust labeling only upon ph

193 e) and TSLP receptor-knockout mice with sham HeLa cell lysate or RV.

194 unoprecipitation of rat liver or transfected HeLa cell lysates with rOATP1A1 antibody specifically co

195 ome-wide protein interactions in E. coli and HeLa cell lysates, respectively, identifying 1,158 and 3

196 In HeLa cells, MAP7, MAP7D1, and MAP7D3 act redundantly to

197 membranes, Candida albicans mitochondria, or HeLa cell mitochondria.

198 The HeLa cell model, including organelles and sub-compartmen

199 l human fibroblast and nitrous oxide-treated HeLa cell models.

200 and export in poly-GR or poly-PR expressing Hela cells, neuronal-like SH-SY5Y cells and iPSC-derived

201 s studies, we reported that fractionation of HeLa cell nuclear extracts on glycerol gradients reveale

202 We found that the expression in HeLa cells of a Merlin variant that is phosphorylation-d

203 on visualization of the intracellular LDs in HeLa cells, offering us the quantitative results of the

204 In HeLa cells, our SirReal-based PROTAC induced isotype-sel

205 DiI-SiR, we visualized filopodia dynamics in HeLa cells over 25 min at 0.5 s temporal resolution, and

206 h-resolution 3D images showing MOF uptake by HeLa cells over a 24 h period.

207 croscopy through the visualization of living HeLa cells overexpressing the somatostatin subtype-2 rec

208 Transcriptomic analyses of HeLa cells overexpressing wild type or a nuclear-targete

209 HRV replication was significantly reduced in HeLa cells overexpressing wild-type and mutant forms of

210 chip) analysis showed that DHTS treatment of HeLa cells paradoxically enriched HuR binding to mRNAs w

211 ow that knocking down paxillin expression in HeLa cells promotes their ability to form colonies in so

212 Applied to HeLa cells, RBDmap uncovered 1,174 RNA-binding sites in

213 We identified 1,174 binding sites within 529 HeLa cell RBPs, discovering numerous RNA-binding domains

214 Previously we showed that HeLa cells readjust homeostasis upon proteostatically dr

215 for biological investigation by imaging live HeLa cells, red blood cells, and neurons.

216 Inactivation of UBQLN2 expression in HeLa cells reduced autophagic flux and autophagosome aci

217 ited during Chlamydia trachomatis infection, HeLa cells regained susceptibility to apoptosis inductio

218 s eliminated while cell uptake in HEK293 and HeLa cells remained high, which improved the overall cha

219 ters during mitosis in C. elegans zygotes or HeLa cells, respectively.

220 ding deficient mutant (D171A) of Golgin45 in HeLa cells resulted in frequent intercisternal fusion am

221 Wild-type C. trachomatis entry into HeLa cells resulted in host cell shape changes, whereas

222 ve found that the overexpression of GDAP1 in HeLa cells results in a mitochondrial phenotype which is

223 e absence of one or more cellular factors in HeLa cells results in abortive replication of H1N1, H3N2

224 Functional studies of Cx26 in HeLa cells revealed co-expression of Cx26-Asp50Asn and w

225 ination of C. burnetii growth in GNPTAB(-/-) HeLa cells revealed replication and viability are not im

226 PDS/PG-associated PMEL variants expressed in HeLa cells revealed structural changes to pseudomelanoso

227 method for expressing uptake transporters in HeLa cells revealed that OATP1A2, but not OATP1B1 or OAT

228 escence microscopy of LC3-GFP-overexpressing HeLa cells, revealed lower autophagic activity in cells

229 microgram of lysate protein and 2000 sorted HeLa cells (roughly half microgram lysate protein) in ea

230 In HeLa cells, S1P-dependent activation of aPKC suppressed

231 In apoptotically stimulated HeLa cells, short-term exposure to polysulfides triggere

232 N-terminal fragment of NleE (NleE(34-52)) in HeLa cells showed competitive inhibition of wild type Nl

233 Treatment of HeLa cells stably expressing the mutant VPS33A with a pr

234 ction of the hydrogen peroxide released from HeLa cells stimulated with N-Formyl-L-methionyl-L-leucyl

235 In HeLa cells, stress-induced Gle1 hyperphosphorylation was

236 ng and was unable to interact with CHO-K1 or HeLa cells, suggesting a potential change in the conform

237 NER assays in cell-free extracts from human HeLa cells, suggesting that another mechanism is require

238 We also report that KU-32 promotes HeLa cell survival and enhances the refolding of an Hsp9

239 The results show that HeLa cells take up twice more [(18)F]FDG in S, G2 or M p

240 We demonstrate in HeLa cells that carnitine palmitoyltransferase 1C (CPT1C

241 Studies in stably transfected HeLa cells that constitutively expressed PDZK1 showed th

242 Here we show in HeLa cells that large cytoplasmic components are displac

243 o be readily reversible, they demonstrate in HeLa cells that once RNA is released from chromatin, the

244 alidated the effects of identified agents in HeLa cells that stably express TTC7A with point mutation

245 When expressed in HEK293 or stable HeLa cells, the 3 mutated netrin-1 proteins were almost

246 induced apoptosis upon ectopic expression in HeLa cells, the percentage of infected macrophages under

247 al data, we constructed a spatial model of a HeLa cell to capture intracellular crowding effects.

248 AURKB, NEK1, TTK, and WEE1 causes simulated HeLa cells to accumulate in the M phase.

249 Exposure of HeLa cells to Cu(PyBD).SO4 (IC50 = 10 muM) results in a

250 fluorescence lifetime imaging (FLIM)-FRET of HeLa cells to identify protein interactions within the s

251 Simultaneous live cell imaging using HeLa cells to investigate the intracellular concentratio

252 (HPAI) H5N1 virus successfully propagated in HeLa cells to levels comparable to those in a human lung

253 nction, the mutant P-gp also hypersensitizes HeLa cells to Rh123 by 2- to 2.5-fold.

254 EGFP- and mCherry-tagged receptor chains in HeLa cells to study their assembly along the secretory p

255 and measured the [(18)F]FDG uptake by single HeLa cells together with their dry mass and cell cycle p

256 s supported by experimental data obtained in HeLa cells transfected with connexin45, which is among c

257 HeLa cell transfection with c-Jun and c-Fos increased CD

258 In HeLa cells treated with a peptide that disrupts Gle1 nuc

259 yperthermia efforts to kill cancer cells and hela cells under 800 nm laser irradiation.

260 e of production of lipid peroxyl radicals in HeLa cells under basal conditions is 33 nM/h within the

261 ouse embryonic fibroblasts (MEFs), and human HeLa cells upon IFN stimulation.

262 o monitor the in situ response of individual HeLa cells using a single cell gated transistor (SCGT).

263 coverage up to more than 8000 proteins from HeLa cells using empirically corrected libraries and mor

264 using sucrose gradient sedimentation and in HeLa cells using fluorescence correlation spectroscopy.

265 muM, and photoactivation was demonstrated in HeLa cells using red light.

266 -wide CRISPR/Cas9 loss-of-function screen in HeLa cells using selection for resistance to Shiga toxin

267 ther carried out protein profiling in intact HeLa cells using the new photocaged cell-permeable ubiqu

268 erous cell lines), complex 1 was taken up by HeLa cells very efficiently by a passive transportation

269 rk, further research using H9e hydrogel with HeLa cells was carried out considering H9e hydrogel's in

270 Acetylation of ectopically expressed NAT1 in HeLa cells was decreased by C646, an inhibitor of the pr

271 mplexes in extracts from nocodazole-arrested HeLa cells was inhibited by Polo-like kinase 1 (Plk1), a

272 Using HeLa cells, we could successfully demonstrate markedly d

273 Using siRNA in HeLa cells, we found that reducing endogenous mOGT expre

274 Using this method in HeLa cells, we have observed polyethylenimine/siRNA poly

275 Using single cell analysis in HeLa cells, we show that the CDC20-MAD2 complex is cell

276 In a previous study with HeLa cells, we showed that ER-to-mitochondria Ca(2+) tra

277 By exogenous expression in HeLa cells, we tested the function of a series of mutati

278 The proliferations of Caco-2, MCF-7 and HELA cells were more inhibited when treated with the WSE

279 HeLa cells were refractory to the growth of human H1N1 a

280 se of wild-type (WT) PV, but viral yields in HeLa cells were similar.

281 In the cell culture experiment, HeLa cells were simultaneously embedded in the H9e hydro

282 PLD3 as the principal acid 5' exonuclease in HeLa cells, where it showed a markedly higher specific a

283 sponse to stress caused by dithiothreitol in HeLa cells, where we identified and quantified 6935 and

284 for sister chromatid cohesion in HCT116 and HeLa cells, whereas it was required for the dissociation

285 ed and almost exclusively cytoplasmic in the HeLa cells; whereas the WT-MIP was stable dispersed thro

286 in G(s) is usually higher for HaCaT than for HeLa cells, which agrees with the common usage of DMSO i

287 and triggers caspase-dependent apoptosis in HeLa cells, which are more sensitive to inhibition by 1

288 When expressed in HeLa cells, which do not normally produce adhesion compl

289 cell dry mass have a positive correlation in HeLa cells, which suggests that high [(18)F]FDG uptake i

290 tory light chain (MRLC) diphosphorylation in HeLa cells, which was restored by reexpression of small

291 frame, with 71.5 s dark recovery) in living HeLa cells with a resolution of 35.2 nm.

292 However, following infection of HEK293 and HeLa cells with AAV2 virions, HBoV1 NS2 (but not NS4), N

293 In HeLa cells with cBIN1 overexpression, knockdown of CHMP4

294 Treatment of HeLa cells with fluorescently labeled PMO chimeras demon

295 First, infection of HeLa cells with human rhinovirus (HRV) induced the phosp

296 ChIP and reporter assays in HeLa cells with monoallelic CD177 expression showed that

297 an Epac-based fluorescent biosensor in live HeLa cells with nanometer spatial and picosecond tempora

298 Following challenge of HeLa cells with the dsRNA-analog poly(I:C), PGAM5 oligom

299 wer flake sizes GO rapidly internalizes into HeLa cells with the following 70% fluorescence based cle

300 h arrest of cancer cells in culture and in a HeLa cell xenografted mouse model.