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1  with the gastrointestinal helminth parasite Heligmosomoides polygyrus.
2 onically infected with the helminth parasite Heligmosomoides polygyrus.
3 culated with Nippostrongylus brasiliensis or Heligmosomoides polygyrus.
4 n mice infected with the intestinal helminth Heligmosomoides polygyrus.
5  2 responses required for protection against Heligmosomoides polygyrus.
6 s during infection with the enteric nematode Heligmosomoides polygyrus.
7 those observed in a secondary infection with Heligmosomoides polygyrus.
8 response to the intestinal nematode parasite Heligmosomoides polygyrus.
9 ction with the intestinal nematode parasite, Heligmosomoides polygyrus.
10 onse by infection with the nematode parasite Heligmosomoides polygyrus.
11 nfection of mice with the nematode parasite, Heligmosomoides polygyrus.
12 oral inoculation with the nematode parasite, Heligmosomoides polygyrus.
13 culation of mice with the nematode parasite, Heligmosomoides polygyrus.
14 erium Citrobacter rodentium and the helminth Heligmosomoides polygyrus.
15 ing produced by the murine helminth parasite Heligmosomoides polygyrus.
16  bakeri, a model parasite of house mice, and Heligmosomoides polygyrus, a closely related parasite of
17  was evaluated in mice infected earlier with Heligmosomoides polygyrus, a gastrointestinal worm known
18 e 2 immune response following infection with Heligmosomoides polygyrus, a natural murine parasitic ne
19                       We colonized mice with Heligmosomoides polygyrus, an intestinal helminth, and a
20 IL-10(-/-) T cell transfer model of colitis, Heligmosomoides polygyrus, an intestinal helminth, preve
21 ost defense to the gastrointestinal helminth Heligmosomoides polygyrus and blunted muscularis immune
22 omologs belonging to the parasitic nematodes Heligmosomoides polygyrus and Heterodera glycines.
23  IL-4/IL-13 during challenge infections with Heligmosomoides polygyrus and Nippostrongylus brasiliens
24  to intestinal nematode parasites, including Heligmosomoides polygyrus and Nippostrongylus brasiliens
25 established by using the intestinal nematode Heligmosomoides polygyrus and S. Typhimurium.
26 oinfection with two natural mouse pathogens, Heligmosomoides polygyrus and Toxoplasma gondii, to inve
27 rugia pahangi, Teladorsagia circumcincta and Heligmosomoides polygyrus, as well as a multidrug resist
28 t chronic infection with the murine helminth Heligmosomoides polygyrus bakeri (Hpb) altered the intes
29 in mice coinfected with the enteric helminth Heligmosomoides polygyrus bakeri (Hpb) and West Nile vir
30 ate that infection of mice with the parasite Heligmosomoides polygyrus bakeri (Hpb) reprograms the in
31      We evaluated the impact of infection by Heligmosomoides polygyrus bakeri (Hpb), a murine intesti
32 l infection with the natural murine helminth Heligmosomoides polygyrus bakeri alters Ag-specific Ab a
33 g infection with gastrointestinal helminths (Heligmosomoides polygyrus bakeri and Trichuris muris).
34                               Helminths like Heligmosomoides polygyrus bakeri can induce regulatory T
35  response in mice infected with the helminth Heligmosomoides polygyrus bakeri H. polygyrus elicited c
36                                              Heligmosomoides polygyrus bakeri infection also promotes
37 leukin-25 (IL-25) in host protection against Heligmosomoides polygyrus bakeri infection in mice.
38 igated the effect of the intestinal nematode Heligmosomoides polygyrus bakeri on Th1 responses to Myc
39  production following infection of mice with Heligmosomoides polygyrus bakeri or Nippostrongylus bras
40      In a murine colitis model, the helminth Heligmosomoides polygyrus bakeri prevents colitis via in
41           In IBD murine models, the helminth Heligmosomoides polygyrus bakeri prevents colitis.
42          The model gastrointestinal nematode Heligmosomoides polygyrus bakeri secretes the Alarmin Re
43                       The parasitic nematode Heligmosomoides polygyrus bakeri secretes the HpARI fami
44           During infection with the helminth Heligmosomoides polygyrus bakeri, Bhlhe40 was required f
45 l nematodes Nippostrongylus brasiliensis and Heligmosomoides polygyrus bakeri.
46 ssue-migrating larvae of the murine parasite Heligmosomoides polygyrus bakeri.
47 trointestinal nematodes is commonly known as Heligmosomoides polygyrus bakeri.
48 hat excretory/secretory products (HpES) from Heligmosomoides polygyrus blocked the effects of IL-4/13
49                           The mouse parasite Heligmosomoides polygyrus can expand the host Treg popul
50 road activation of an antimicrobial program; Heligmosomoides polygyrus caused an increase in the abun
51                                              Heligmosomoides polygyrus coinfection is reported to hav
52                                              Heligmosomoides polygyrus colonization inhibits Th1 and
53  gut, we studied the influence of intestinal Heligmosomoides polygyrus colonization on LPS-induced la
54                                              Heligmosomoides polygyrus did not change the normal micr
55              Mice infected with the helminth Heligmosomoides polygyrus displayed increased levels of
56              The murine intestinal helminth, Heligmosomoides polygyrus, down-modulates the host immun
57                                              Heligmosomoides polygyrus elevated Th2 cytokine expressi
58 ts from the model mouse intestinal parasite, Heligmosomoides polygyrus (equivalent to 7 mug of protei
59 ermined if exposure to the duodenal helminth Heligmosomoides polygyrus establishes cytokine pathways
60 and shown that coinfection with the helminth Heligmosomoides polygyrus exacerbates colitis induced by
61 infection model with the intestinal helminth Heligmosomoides polygyrus followed by infection with Tox
62 hallenge with the strictly enteric helminth, Heligmosomoides polygyrus, GFP-positive innate and adapt
63    Oral infection with the nematode parasite Heligmosomoides polygyrus H. polygyrus is entirely restr
64 ostrongylus brasiliensis (N brasiliensis) or Heligmosomoides polygyrus (H polygyrus) or injected intr
65 TGF-beta1, secreted by the helminth parasite Heligmosomoides polygyrus, had an almost identical prote
66 ion of intestinal epithelial function during Heligmosomoides polygyrus (Hp) infection was investigate
67  C57BL/6 mice to infection with the helminth Heligmosomoides polygyrus, including TGF-beta signaling,
68 vided into four groups: uninfected; helminth-Heligmosomoides polygyrus infected; Pseudomonas aerugino
69          Acute GVHD was induced in helminth (Heligmosomoides polygyrus)-infected or uninfected BALB/c
70 f Th2 cells derive from Foxp3(+) cells after Heligmosomoides polygyrus infection and airway allergy.
71 rasiliensis or were drug-cured of an initial Heligmosomoides polygyrus infection and later reinfected
72 tode infection, we compared the responses to Heligmosomoides polygyrus infection between 2 mouse stra
73 te T helper 2 (T(H)2) cell responses against Heligmosomoides polygyrus infection, consistent with cDC
74                    Using influenza virus and Heligmosomoides polygyrus infections, we show that these
75 a normally chronic intestinal infection with Heligmosomoides polygyrus into an infection that was rap
76                                              Heligmosomoides polygyrus is a strictly murine enteric n
77 ng infection with the gut-dwelling roundworm Heligmosomoides polygyrus is critical for protective imm
78   We demonstrate that infection of mice with Heligmosomoides polygyrus leads to enteric gliosis and t
79                The mouse intestinal helminth Heligmosomoides polygyrus modulates host immune response
80  rodents infected with Trichinella spiralis, Heligmosomoides polygyrus, Nippostronglyus brasiliensis,
81  of strictly enteric helminth infection with Heligmosomoides polygyrus on respiratory syncytial virus
82 fects of infection with a helminth parasite, Heligmosomoides polygyrus, on type 1 diabetes (T1D) in n
83 ice failed to expel the intestinal helminths Heligmosomoides polygyrus or Nippostrongylus brasiliensi
84 fected with the intestinal nematode parasite Heligmosomoides polygyrus prior to infection with S. man
85 that ongoing infection with the gut helminth Heligmosomoides polygyrus protects against RSV infection
86 tization to OVA or intestinal infection with Heligmosomoides polygyrus Specific Igs and plasmablasts
87      Products secreted by the mouse parasite Heligmosomoides polygyrus suppress type 2 (allergic) imm
88    BALB/c mice received an oral infection of Heligmosomoides polygyrus third-stage larvae, were given
89  Th2 cell-inducing gastrointestinal nematode Heligmosomoides polygyrus to influence experimentally in
90 small intestine-restricted helminth pathogen Heligmosomoides polygyrus to test the hypothesis that pa
91 tion with three distinct helminth parasites, Heligmosomoides polygyrus, Trichuris muris, and Schistos
92 in secreted by the model intestinal nematode Heligmosomoides polygyrus, which binds and blocks IL-33.
93 xpulsion of Nippostrongylus brasiliensis and Heligmosomoides polygyrus, which both live in the intest
94 monstrate that the gastrointestinal nematode Heligmosomoides polygyrus, which infects mice, secretes
95  infection with the murine nematode parasite Heligmosomoides polygyrus, which resides in the duodenum