ライフサイエンスコーパス: Hg

1 Hg methylation occurs in the cytosol of certain obligate

2 Hg(0) adsorption and regeneration efficiencies of raw an

3 d by MIC were 0.034 (As), 0.015 (Cd), 0.021 (Hg) and 0.105 (Pb) mug g(-1), which are suitable to atta

4 202)Hg values of Hg(0), but not in Delta(199)Hg or Delta(200)Hg values.

5 variation in both delta(202)Hg and Delta(199)Hg, but not Delta(200)Hg.

6 trends of Hg isotope (particularly Delta(199)Hg: 0.96-1.13 per mille) and carbon isotope (delta(13)C:

7 Hg(0), but not in Delta(199)Hg or Delta(200)Hg values.

8 n-mass independent isotope values (Delta(200)Hg) in amphipods that average 0.03 per mille (+/-0.02, n

9 a(202)Hg and Delta(199)Hg, but not Delta(200)Hg.

10 hort-lived Hg spike, and nadirs in delta(202)Hg and delta(13)C values at the marine PTME are best exp

11 s displayed diel variation in both delta(202)Hg and Delta(199)Hg, but not Delta(200)Hg.

12 displayed strong diel variation in delta(202)Hg values of Hg(0), but not in Delta(199)Hg or Delta(200

13 nic mercury (Hg(IN)) and methylmercury (CH(3)Hg) was developed.

14 in Group 1 (20.16 +/- 3.3 to 15.05 +/- 2.4mm Hg; P = .001), compared to Group 2 (21.2 +/- 5.6 to 20.0

15 d to Group 2 (21.2 +/- 5.6 to 20.0 +/- 5.8mm Hg; P = .38).

16 A Hg adsorption/regeneration mechanism was proposed to exp

17 drologic cycle, which will likely accelerate Hg cycling in tandem with changing inputs from thawing p

18 Wetlands are common sites of active Hg methylation by anaerobic microbes; however, the amoun

19 In primary analyses, Hg and Ni ranked highest as predictors of BW for GA.

20 We find that the photochemistry of Hg(I) and Hg(II) leads to insufficient Hg oxidation globally.

21 otochemistry of gas-phase oxidized Hg(I) and Hg(II) species postulate their photodissociation back to

22 gh 2030, the annual mobilization of soil and Hg may increase by an additional 20-25% relative to 2014

23 eration atomic fluorescence spectrometry and Hg by cold vapor atomic fluorescence spectrometry after

24 Anthropogenic Hg added to the surface ocean is, therefore, expected to

25 lizing naturally occurring and anthropogenic Hg from terrestrial landscapes to aquatic environments i

26 g and analysis methods for measuring aqueous Hg(II) concentrations down to the nanomolar level in fre

27 of methylmercury produced varies greatly, as Hg methylation is dependent upon both the availability o

28 OQ) of 0.72, 0.12, and 1.5 mug L(-1) for As, Hg, and Se, respectively.

29 tify a significant amount of cell-associated Hg-S(3)/S(4) species, as studied by high energy-resoluti

30 photodissociation mechanisms on atmospheric Hg chemistry, lifetime, and surface deposition remains u

31 o Hg(0) as a crucial step in the atmospheric Hg redox cycle.

32 est as a predictor of BW for GA, followed by Hg and Ni.

33 terferences in the quantification of As, Cd, Hg, and Pb.

34 ely enough to be involved in a 2-coordinated Hg(Mem-RS)(2) structure in Geobacter.

35 s of the potentially toxic elements (As, Cr, Hg, Ni and Pb) varied from 0.9 to 1.4, pointing to a sim

36 th low molecular mass (LMM) thiols like Cys, Hg(Cys)(Mem-RS), or with neighboring O/N membrane functi

37 ons where greater denudation likely enhances Hg mobilization.

38 An east-to-west increase of head feather Hg concentrations (1.74-3.48 mug.g(-1)) was accompanied

39 Fish Hg concentrations do not exceed United States Environmen

40 For Hg, all analyzed samples were below the limit of quantif

41 The diffusion coefficient for Hg(2+) complexed with common inorganic ligands was an or

42 s an order of magnitude higher than that for Hg(2+) complexed with natural dissolved organic matter:

43 ant, is emitted mainly in its elemental form Hg(0) to the atmosphere where it is oxidized to reactive

44 th neighboring O/N membrane functionalities, Hg(Mem-RSRO).

45 eeps, lake/ponds, and a wetland) to identify Hg methylation hotspots and seasonal differences in MeHg

46 able to distinguish between Cu(II), Cd(II), Hg(II), and Pb(II) at a concentration of 100 muM.

47 However, the processes involved in Hg(II) biouptake and methylation are not well understood

48 Sulfate addition failed to increase Hg methylation rates in the peatlands, suggesting that S

49 on in aquatic food webs; and (3) Se inhibits Hg bioavailability to, and/or methylmercury production b

50 n experiments were conducted with an initial Hg(0) concentration of 260-300 mug/m(3) at room temperat

51 lly mediated process that converts inorganic Hg into bioaccumulative, neurotoxic methylmercury (MeHg)

52 ry of Hg(I) and Hg(II) leads to insufficient Hg oxidation globally.

53 95% of Mem-RSH is involved in mixed-ligation Hg(II)-complexes, combining either with low molecular ma

54 We show that the large short-lived Hg spike, and nadirs in delta(202)Hg and delta(13)C valu

55 We measured Hg concentrations and stable isotope ratios of Hg, carbo

56 rmodynamics and kinetics of divalent mercury Hg(II) chemical speciation need to be understood.

57 Mercury (Hg) methylation is a microbially mediated process that c

58 Mercury (Hg), a global contaminant, is emitted mainly in its elem

59 Compared with lead (Pb)- and mercury (Hg)-based liquid metal electrodes, the nontoxic Ga alloy

60 te the limited direct anthropogenic mercury (Hg) inputs in the circumpolar Arctic, elevated concentra

61 Cadmium (Cd), cobalt (Co), mercury (Hg), nickel (Ni), molybdenum (Mo), lead (Pb), antimony (

62 Here we develop mercury (Hg) stable isotopes as a proxy for paleoatmospheric chem

63 ic understanding of bacteria-driven mercury (Hg) transformation processes in natural environments, th

64 combination transition of elemental mercury (Hg(0)).

65 re research is required to evaluate mercury (Hg) speciation in DBS and to validate the agreement betw

66 a significant global reservoir for mercury (Hg) and its isotopic characterization is important to un

67 speciation procedure for inorganic mercury (Hg(IN)) and methylmercury (CH(3)Hg) was developed.

68 cted a national-scale assessment of mercury (Hg) bioaccumulation in aquatic ecosystems, using dragonf

69 ironmental and dietary exposures to mercury (Hg), a highly toxic metal traditionally regarded as a ne

70 mean systolic blood pressure fell by 9.0 mm Hg in the intervention group and by 3.9 mm Hg in the con

71 P in the intervention than UC group (-5.0 mm Hg, 95% CI -7.1 to -3.0; P < 0.001) and a greater declin

72 lic pressure (11.3+/-2.5 versus 5.7+/-2.0 mm Hg; P<0.0001) and diminished fractional area change (24.

73 (-5.97, -1.17), and -3.28 (-5.55, -1.00) mm Hg, respectively]; insulin sensitivity increased at 3 an

74 in-converting enzyme inhibitors and -3.07 mm Hg (95% CI, -4.99 to -1.44) for angiotensin receptor blo

75 essure (from 23.4 +/- 4.9 to 10.5 +/- 3.1 mm Hg), pulmonary artery systolic pressure (from 60.6 +/- 1

76 had PASP of <50 mm Hg (mean: 36.3 +/- 8.1 mm Hg).

77 d a greater decline in diastolic BP (-2.1 mm Hg, 95% CI -3.6 to -0.6; P < 0.006), but no detectable d

78 3.3), 6.9 (3.7), 8.8 (5.0), and 9.5 (4.1) mm Hg, respectively (P < .001, Kruskal-Wallis test).

79 -weighted average of hypotension was 0.10 mm Hg (IQR, 0.01-0.43 mm Hg) in the intervention group vs 0

80 adjustment including OBP and 24-h ABP, 10 mm Hg higher systolic and diastolic HBP were associated wit

81 higher MAPs (86 +/- 9 mm Hg vs. 72 +/- 10 mm Hg, p < 0.001).

82 reoperative systolic BP (aOR, 1.16 per 10-mm Hg increase; 95% CI, 1.05 to 1.28), and left ventricular

83 al perfusion pressure on the order of 100 mm Hg at the cranial end of the carotid arteries.

84 Hypercapnia (PCO2 90-100 mm Hg) was established in mechanically ventilated swine by

85 oxygen therapy (target Pao(2), 90 to 105 mm Hg; Spo(2), >=96%) for 7 days.

86 Predelivery mean gradient was 11 mm Hg, and systolic pulmonary artery pressure was 32 mm Hg.

87 m Hg) compared to 800 IU vitamin D3 (0.11 mm Hg; difference: -0.48 mm Hg; 95% CI: -0.94, -0.01; P = 0

88 difference, -13 mm Hg; 95% CI, -15 to -11 mm Hg; P < .001).

89 the mean systolic blood pressure was 115 mm Hg.

90 dard systolic BP lowering (targeting <120 mm Hg versus <140 mm Hg, respectively).

91 24-hour systolic BP (SBP; from 138 to 124 mm Hg) compared with sodium restriction (from 134 to 129 mm

92 eline and mean achieved SBP of 120 to 129 mm Hg identified the lowest risk patients with HFpEF.

93 with sodium restriction (from 134 to 129 mm Hg), as well as a significantly greater effect on extrac

94 tion group (between-group difference, -13 mm Hg; 95% CI, -15 to -11 mm Hg; P < .001).

95 tegories (<95, 95-109, 110-129, and >=130 mm Hg).

96 IDH, by 2017 ACC/AHA (systolic BP <130 mm Hg, diastolic BP >=80 mm Hg) and by JNC7 (systolic BP <1

97 During follow-up, average SBP was 135 mm Hg (125-145).

98 tion-only group and from 150 mm Hg to 135 mm Hg in the renal denervation group (between-group differe

99 comes included upper IOP thresholds of 14 mm Hg and 21 mm Hg with and without a 20% IOP reduction fro

100 was 75.6% for an upper IOP cut-off of 14 mm Hg and 76.9% for 21 mm Hg, and qualified success was 91.

101 ssure (systolic blood pressure level >140 mm Hg or diastolic blood pressure level >90 mm Hg), uncontr

102 iles (<120, 120 to 129, 130 to 139, >=140 mm Hg) to the primary outcome (cardiovascular death and tot

103 >=80 mm Hg) and by JNC7 (systolic BP <140 mm Hg, diastolic BP >=90 mm Hg) definitions.

104 owering (targeting <120 mm Hg versus <140 mm Hg, respectively).

105 12 months decreased from 151 mm Hg to 147 mm Hg in the isolation-only group and from 150 mm Hg to 135

106 and pulmonary artery wedge pressure <=15 mm Hg without another cause of pulmonary hypertension.

107 as systolic blood pressure lower than 150 mm Hg at 12-week follow-up.

108 in the isolation-only group and from 150 mm Hg to 135 mm Hg in the renal denervation group (between-

109 ad systolic blood pressure lower than 150 mm Hg, and were receiving at least 2 antihypertensive medic

110 baseline to 12 months decreased from 151 mm Hg to 147 mm Hg in the isolation-only group and from 150

111 scites/HE, those with baseline HVPG <= 16 mm Hg (n = 16) had a low rebleeding risk (13%).

112 tension-brain oxygen tension gradient (16 mm Hg [sd, 6] vs 39 mm Hg SD, 11]; p < 0.001) and in the re

113 if the patient's baseline HVPG is over 16 mm Hg improves detection of high-risk patients while marked

114 ts with ascites/HE and baseline HVPG > 16 mm Hg, only the HVPG responders (n = 32) had a good prognos

115 ith (1) no 2 consecutive IOP readings >17 mm Hg or clinical hypotony without (complete) or with glauc

116 rom those of the controls (13.46 +/- 2.17 mm Hg; P = .006).

117 9 mm Hg); median postoperative IOP was 18 mm Hg (range 5-40 mm Hg, mean 20.3 mm Hg].

118 defined as intraocular pressure (IOP) >18 mm Hg or not reduced by 30% below baseline on 2 consecutive

119 ry artery occlusion pressure less than 18 mm Hg.

120 sion pressure greater than or equal to 18 mm Hg.

121 ic blood pressure ranging from 140 to 190 mm Hg), anemia requiring blood transfusions, thrombocytopen

122 control group; the mean reduction was 5.2 mm Hg greater with the intervention (95% confidence interva

123 +/- standard deviation) was -0.6 +/- 6.2 mm Hg in the placebo group, -1.2 +/- 6.8 mm Hg in the potas

124 all the cases and mean gradient was 7+/-2 mm Hg.

125 load-independent ones (mean change = 49.2 mm Hg.s; p = 0.114).

126 Large reductions (> 20 mm Hg) in arterial carbon dioxide tension over 24 hours wer

127 evidenced-based, is inclusive of mPAP >20 mm Hg, and emphasizes early diagnosis.

128 Sixty POAG patients with IOP >=21 mm Hg taking maximal topical medication and scheduled for t

129 d upper IOP thresholds of 14 mm Hg and 21 mm Hg with and without a 20% IOP reduction from baseline, m

130 IOP cut-off of 14 mm Hg and 76.9% for 21 mm Hg, and qualified success was 91.9% and 92.5%.

131 t estimate, -12.36 mm Hg [-23.52 to -1.21 mm Hg]; p = 0.032) and lower rates of return of spontaneous

132 ect intracranial pressure greater than 22 mm Hg was 0.84 (0.76-0.90).

133 (SD 15.17), and mean diastolic BP by 4.23 mm Hg (SD 8.68).

134 r pressure (IOP) less than or equal to 24 mm Hg with or without medications and no additional surgery

135 o had mean pulmonary artery pressure >=25 mm Hg or pulmonary vascular resistance (PVR) > 400 dyn s cm

136 ed by mPAP of greater than or equal to 25 mm Hg, and 51 participants had PH defined by PVR of greater

137 ts had mean pulmonary artery pressure >25 mm Hg, pulmonary vascular resistance >240 dyn-sec/cm(-5) ,

138 lysis showed an average reduction of 1.29 mm Hg (95% confidence interval (95% CI) (-2.17, -0.41)) in

139 , and cerebral perfusion pressure were 29 mm Hg (SD, 9), 45 mm Hg (SD, 9), and 80 mm Hg (SD, 7), resp

140 4 mm Hg (95% CI: 2.8 to 5.5 mm Hg) and 3 mm Hg (95% CI: 1.6 to 3.5 mm Hg).

141 lative to cardiac output with exercise >3 mm Hg/l/min.

142 +/- 0.3 mm Hg; p = 0.04).

143 was 18 mm Hg (range 5-40 mm Hg, mean 20.3 mm Hg].

144 tensibility by 2.3, 1.9, and 3.1 x 10(-3) mm Hg(-1), respectively (p < 0.05 for all).

145 Median preoperative IOP was 30 mm Hg (range 18-49 mm Hg, mean 30.9 mm Hg); median postoper

146 een solution at a constant pressure of 30 mm Hg and 10 degrees C.

147 had an intraocular pressure (IOP) over 30 mm Hg at POD1.

148 ed IOP, and 4 of these 5 eyes had IOP >30 mm Hg.

149 A mean pressure gradient >=31 mm Hg had a 7x higher risk of the primary end point in the

150 systolic pulmonary artery pressure was 32 mm Hg.

151 ic blood pressure (point estimate, -12.36 mm Hg [-23.52 to -1.21 mm Hg]; p = 0.032) and lower rates o

152 2000 IU (average real variability: -0.37 mm Hg) compared to 800 IU vitamin D3 (0.11 mm Hg; differenc

153 Pooled reductions of SBP were -4.38 mm Hg (95% CI, -7.27 to -2.16) for angiotensin-converting e

154 ol group, for a median difference of 0.38 mm Hg (95% CI, 0.14-0.43 mm Hg; P = .001).

155 er for Blacks, with median declines of 38 mm Hg (95% CI, 32-40 mm Hg) at 45 to 54 years of age and 50

156 inal mean, 294 [264-323] vs 365 [346-385] mm Hg in uninfected patients; p = 0.0005) as in potential l

157 tension gradient (16 mm Hg [sd, 6] vs 39 mm Hg SD, 11]; p < 0.001) and in the relationship of jugula

158 change in systolic blood pressure was 3.4 mm Hg (95% CI, 1.1 to 5.8 mm Hg) higher in the intervention

159 olic central (aortic) blood pressure by 4 mm Hg (95% CI: 2.8 to 5.5 mm Hg) and 3 mm Hg (95% CI: 1.6 t

160 HFpEF vs. control subjects: calf 16 +/- 4 mm Hg vs. 22 +/- 4 mm Hg; p < 0.005; forearm 17 +/- 4 mm Hg

161 +/- 4 mm Hg; p < 0.005; forearm 17 +/- 4 mm Hg vs. 25 +/- 5 mm Hg; p < 0.001).

162 reater than or equal to 5 cm H2O (i.e., 4 mm Hg) during passive leg raising can predict preload unres

163 ean LVOT gradient at discharge: 5.4+/-1.4 mm Hg).

164 n in PaCO2 (104.0 +/- 8.1 vs 74.2 +/- 8.4 mm Hg; p < 0.001).

165 ubjects: calf 16 +/- 4 mm Hg vs. 22 +/- 4 mm Hg; p < 0.005; forearm 17 +/- 4 mm Hg vs. 25 +/- 5 mm Hg

166 ts in low-gradient (LG, mean gradient <40 mm Hg) AS are conflicting.

167 edian declines of 38 mm Hg (95% CI, 32-40 mm Hg) at 45 to 54 years of age and 50 mm Hg (95% CI, 33-60

168 ht ventricular (RV) systolic pressure <40 mm Hg, and normal RV function by echocardiography.

169 ostoperative IOP was 18 mm Hg (range 5-40 mm Hg, mean 20.3 mm Hg].

170 gitation or maximum Doppler gradients >40 mm Hg.

171 ds, PCWP was significantly reduced (-2.40 mm Hg; 95% confidence interval: -3.96 to -0.84 mm Hg; p = 0

172 ng donors (299 [248-350] vs 379 [350-408] mm Hg; p = 0.04; linear mixed models).

173 ypotension was 0.10 mm Hg (IQR, 0.01-0.43 mm Hg) in the intervention group vs 0.44 mm Hg (IQR, 0.23-0

174 fference of 0.38 mm Hg (95% CI, 0.14-0.43 mm Hg; P = .001).

175 mm Hg) in the intervention group vs 0.44 mm Hg (IQR, 0.23-0.72 mm Hg) in the control group, for a me

176 nt estimate, -6.68 mm Hg [-10.92 to -2.44 mm Hg]; p = 0.003) and systolic blood pressure (point estim

177 fusion pressure were 29 mm Hg (SD, 9), 45 mm Hg (SD, 9), and 80 mm Hg (SD, 7), respectively.

178 /min when normalized to venous PCO2 of 45 mm Hg), corresponding to a 29% reduction in PaCO2 (104.0 +/

179 vitamin D3 (0.11 mm Hg; difference: -0.48 mm Hg; 95% CI: -0.94, -0.01; P = 0.045).

180 reoperative IOP was 30 mm Hg (range 18-49 mm Hg, mean 30.9 mm Hg); median postoperative IOP was 18 mm

181 ic blood pressure the difference was -6.5 mm Hg (-9.6 to -3.5).

182 t least 0.5 percentage points in HbA1c, 5 mm Hg in SBP, or 10 mg/dL in LDL cholesterol.

183 vities compared with patients with Zva <5 mm Hg mL(-1) m(-2).

184 years (adjusted hazard ratio: 0.91 per -5 mm Hg PASP; 95% confidence interval: 0.86 to 0.96; p = 0.00

185 d pressure by 4 mm Hg (95% CI: 2.8 to 5.5 mm Hg) and 3 mm Hg (95% CI: 1.6 to 3.5 mm Hg).

186 .5 mm Hg) and 3 mm Hg (95% CI: 1.6 to 3.5 mm Hg).

187 echocardiographic gradient was 15.7+/-5.5 mm Hg.

188 o having postprocedural mean gradient >=5 mm Hg.

189 ed mean systemic blood pressure (28 +/- 5 mm Hg; p < 0.0001) and systemic vascular resistance (1,320

190 .005; forearm 17 +/- 4 mm Hg vs. 25 +/- 5 mm Hg; p < 0.001).

191 40 mm Hg) at 45 to 54 years of age and 50 mm Hg (95% CI, 33-60 mm Hg) for ages >=75 years.

192 right ventricular systolic pressure >=50 mm Hg (HR: 2.27; 95% CI: 1.50 to 3.43; p < 0.01) were indep

193 344 (171 TMVr, 173 GDMT) had PASP of <50 mm Hg (mean: 36.3 +/- 8.1 mm Hg).

194 184 (82 TMVr, 102 GDMT) had PASP of >=50 mm Hg (mean: 59.1 +/- 8.8 mm Hg) and 344 (171 TMVr, 173 GDM

195 IQR 6-10]), mean systolic BP fell by 6.55 mm Hg (SD 15.17), and mean diastolic BP by 4.23 mm Hg (SD 8

196 BP starting at 70 mm Hg and peaking at 55 mm Hg and in direct proportion to RHR starting at 60 bpm.

197 h-hour spent below 75, 70, 65, 60, and 55 mm Hg, respectively.

198 Candesartan reduced SBP by -6.56 mm Hg (P < .001; n = 240).

199 potassium nitrate group, and -0.5 +/- 6.6 mm Hg in the leafy green vegetable group.

200 tudy comprised 90 patients with HVPG >= 6 mm Hg who underwent paired HVPG, TE, and VITRO assessments

201 observed with IF (-4.9 mm Hg; -7.2, -2.6 mm Hg) and Mediterranean (-5.9 mm Hg; -9.0, -2.7 mm Hg) die

202 essure gradient (41.2+/-18.7 to 5.6+/-9.6 mm Hg) and the invasive peak systolic pressure gradient (34

203 load-dependent cases (mean change = 510.6 mm Hg.s; p = 0.005) and remained stable in preload-independ

204 07; 95% CI, 1.00-1.14; p = 0.05) and < 60 mm Hg (odds ratio, 1.10; 95% CI, 1.01-1.18; p = 0.04).

205 Initial examination revealed BP 90/60 mm Hg and tachycardia.

206 The 95th percentile of SBP decreased 60 mm Hg for Whites and 70 mm Hg for Blacks.

207 years of age and 50 mm Hg (95% CI, 33-60 mm Hg) for ages >=75 years.

208 arterial partial pressure of CO2 of >=60 mm Hg.

209 d for mean arterial pressure less than 65 mm Hg (odds ratio, 1.07; 95% CI, 1.00-1.14; p = 0.05) and <

210 rial pressure greater than or equal to 65 mm Hg and signs of altered tissue perfusion.

211 s a mean arterial pressure (MAP) below 65 mm Hg for at least 1 minute.

212 mean arterial pressure of greater than 65 mm Hg may be a reasonable target in patients with cirrhosis

213 .25 +/- 1.69 mm Hg) and PK (12.0 +/- 2.67 mm Hg) groups (P = .95); however, the IOP values for both o

214 lic blood pressure (point estimate, -6.68 mm Hg [-10.92 to -2.44 mm Hg]; p = 0.003) and systolic bloo

215 ot differ between the MCD (11.25 +/- 1.69 mm Hg) and PK (12.0 +/- 2.67 mm Hg) groups (P = .95); howev

216 and Mediterranean (-5.9 mm Hg; -9.0, -2.7 mm Hg) diets, and reduced glycated hemoglobin with the Medi

217 sure (from 60.6 +/- 14.2 to 33.8 +/- 10.7 mm Hg), RV/left ventricular ratio (from 1.19 +/- 0.33 to 0.

218 , median mean mitral valve gradient was 7 mm Hg, most patients (96.7%) had mitral regurgitation grade

219 VH, 13.4+/-2.7 versus control, 11.7+/-1.7 mm Hg, P<0.0001).

220 astance index (3.3+/-0.9 versus 2.9+/-0.7 mm Hg/mL.m(2); P<0.001) and lower total arterial compliance

221 nlinearity = 0.002) to DBP starting at 70 mm Hg and peaking at 55 mm Hg and in direct proportion to R

222 strategy with a Pao(2) between 55 and 70 mm Hg did not increase survival at 28 days.

223 SBP decreased 60 mm Hg for Whites and 70 mm Hg for Blacks.

224 e oxygen therapy (target Pao(2), 55 to 70 mm Hg; oxygen saturation as measured by pulse oximetry [Spo

225 ntion group vs 0.44 mm Hg (IQR, 0.23-0.72 mm Hg) in the control group, for a median difference of 0.3

226 ressure threshold (55, 60, 65, 70, and 75 mm Hg) for ICU-mortality.

227 CI) (-2.17, -0.41)) in systolic and 0.76 mm Hg (95% CI (-1.39, -0.13)) in diastolic blood pressure.

228 95% CI: 1.04 to 2.26; p < 0.01), TMG >=8 mm Hg (HR: 1.68; 95% CI: 1.12 to 2.51; p = 0.012), and righ

229 a peak IOP (mean +/- SD) of 29.6 +/- 7.8 mm Hg and peak incidence at 2-3 months after surgery.

230 mm Hg in the placebo group, -1.2 +/- 6.8 mm Hg in the potassium nitrate group, and -0.5 +/- 6.6 mm H

231 d pressure was lower by approximately 5.8 mm Hg with the polypill and with combination therapy than w

232 ad PASP of >=50 mm Hg (mean: 59.1 +/- 8.8 mm Hg) and 344 (171 TMVr, 173 GDMT) had PASP of <50 mm Hg (

233 ressure was 3.4 mm Hg (95% CI, 1.1 to 5.8 mm Hg) higher in the intervention group compared with the c

234 pulmonary arterial pressure (-6.5 +/- 1.8 mm Hg; p = 0.005) and tended to decrease pulmonary vascular

235 9 mm Hg (SD, 9), 45 mm Hg (SD, 9), and 80 mm Hg (SD, 7), respectively.

236 ss than 140/90 mm Hg and less than 130/80 mm Hg without medications, respectively.

237 systolic BP <130 mm Hg, diastolic BP >=80 mm Hg) and by JNC7 (systolic BP <140 mm Hg, diastolic BP >=

238 according to current guidelines (<130/80 mm Hg).

239 ; 95% confidence interval: -3.96 to -0.84 mm Hg; p = 0.003), but not CI (-0.09 l/min/m(2); 95% confid

240 t flow were maintained at median of 30.88 mm Hg, 9.77 degrees C, and 31.13 mL/min, respectively.

241 for 24-h systolic blood pressure was -3.9 mm Hg (Bayesian 95% credible interval -6.2 to -1.6) and for

242 m Hg in the intervention group and by 3.9 mm Hg in the control group; the mean reduction was 5.2 mm H

243 = 0.01) and reached higher MAPs (86 +/- 9 mm Hg vs. 72 +/- 10 mm Hg, p < 0.001).

244 olic pressure gradient (34+/-12 to 11+/-9 mm Hg).

245 as 30 mm Hg (range 18-49 mm Hg, mean 30.9 mm Hg); median postoperative IOP was 18 mm Hg (range 5-40 m

246 AVR at 1 year (3.7+/-0.8 versus 3.9+/-0.9 mm Hg/mL/m(2); P<0.001).

247 blood pressure was observed with IF (-4.9 mm Hg; -7.2, -2.6 mm Hg) and Mediterranean (-5.9 mm Hg; -9.

248 -7.2, -2.6 mm Hg) and Mediterranean (-5.9 mm Hg; -9.0, -2.7 mm Hg) diets, and reduced glycated hemogl

249 djusted least squares mean: -4.0 vs. -0.9 mm Hg; p = 0.006), a change that was associated with reduce

250 the MT group achieved BP less than 140/90 mm Hg and less than 130/80 mm Hg without medications, respe

251 systolic BP <140 mm Hg, diastolic BP >=90 mm Hg) definitions.

252 Blood-pressure control (<140/90 mm Hg) was achieved in 53.2% of the participants in the int

253 ry outcome was control of BP (BP < 140/90 mm Hg), analysed using mixed effects regression, clustered

254 otension (systolic arterial pressure <=90 mm Hg), and pneumonia.

255 Hg or diastolic blood pressure level >90 mm Hg), uncontrolled diabetes (hemoglobin A1c level >8%), o

256 om for improvement in BP control (<140/90 mm Hg), which was 58% overall, and in the clinical processe

257 ctors of incident CKD included BP >140/90 mm Hg, higher glycated hemoglobin, lower baseline eGFR, and

258 ons while maintaining BP less than 140/90 mm Hg.

259 ance index (0.8+/-0.3 versus 1.2+/-0.5 mL/mm Hg.m(2); P<0.001).

260 luding MI (MI hazard ratio, 1.07 per unit mm Hg increase in DBP; P<0.001).

261 .88] l x 100 ml of tissue(-1) x min(-1) x mm Hg(-1) vs. 4.66 [IQR: 3.70 to 6.15] mul x 100 ml of tiss

262 .43] l x 100 ml of tissue(-1) x min(-1) x mm Hg(-1) vs. 5.66 [IQR: 4.69 to 8.38] mul x 100 ml of tiss

263 8] mul x 100 ml of tissue(-1) x min(-1) x mm Hg(-1); p > 0.05), in keeping with blood vascular rarefa

264 5] mul x 100 ml of tissue(-1) x min(-1) x mm Hg(-1); p < 0.01; forearm: 5.16 [IQR: 3.86 to 5.43] l x

265 ges, and cascaded organic matter, nutrients, Hg and other organically-bound species into the marine s

266 n is dependent upon both the availability of Hg and the composition and activity of the microbial com

267 and sources, distribution, and deposition of Hg to the Earth's surface.

268 differences in the vertical distribution of Hg in the atmosphere as Mount Bachelor received free tro

269 to develop an integrated impairment index of Hg risk to aquatic ecosytems and found that 12% of site-

270 (26-72%), whereas the reduction in levels of Hg, Cd, and Pb was much lower.

271 While, the estimated total mass of Hg transported by rivers is substantially less than the

272 of community dynamics on the methylation of Hg in wetlands with different trophic status.

273 We find that the photochemistry of Hg(I) and Hg(II) leads to insufficient Hg oxidation glob

274 n, with Hg(0) being the main photoproduct of Hg(II) photolysis in the atmosphere, which significantly

275 concentrations and stable isotope ratios of Hg, carbon, and nitrogen in the feathers and blood of ge

276 ubstantially less than the estimated tons of Hg used with ASGM in Peru, this research shows that defo

277 hange and soil erosion on river transport of Hg in a heavily ASGM-impacted watershed.

278 accompanied by significant spatial trends of Hg isotope (particularly Delta(199)Hg: 0.96-1.13 per mil

279 ong diel variation in delta(202)Hg values of Hg(0), but not in Delta(199)Hg or Delta(200)Hg values.

280 surface thiols have no significant effect on Hg(II) methylation, regardless of Cys addition.

281 in the photochemistry of gas-phase oxidized Hg(I) and Hg(II) species postulate their photodissociati

282 atmosphere where it is oxidized to reactive Hg(II) compounds, which efficiently deposit to surface e

283 Zn(II) and Cd(II) and with HNO(3) to recover Hg(II) after several consecutive adsorption/desorption c

284 y in consumers; (2) environmental Se reduces Hg bioaccumulation and biomagnification in aquatic food

285 s or for applying Se amendments to remediate Hg-contaminated ecosystems.

286 ntemporary (2012-2017) benchmark of riverine Hg export.

287 sh consumption advisories on the basis of Se:Hg ratios or for applying Se amendments to remediate Hg-

288 By 2100, simulated Hg concentrations in the Yukon River increase by 14% for

289 te-related re-emissions of previously stored Hg from thawing sea-ice, glaciers, and permafrost.

290 The Hg EXAFS data suggest that 5% of the total number of mem

291 This work highlights the distribution of the Hg-methylation genes across microbial metabolic guilds a

292 es postulate their photodissociation back to Hg(0) as a crucial step in the atmospheric Hg redox cycl

293 Altering the metal from Pb(II) to Hg(II) or shifting the relative position of the sterical

294 Our results indicate minimal impacts to Hg concentrations in water and fish for the low emission

295 er) ), located within merR, is responsive to Hg(II).

296 Total Hg concentrations were positively related to both delta(

297 The total Hg concentrations ranged from 0.49 to 1.60 mg/kg w.w. an

298 The total Hg exceeded the maximum level defined by the European Co

299 proxy for paleoatmospheric chemistry and use Hg isotope data from 2.5 billion-year-old sedimentary ro

300 peroxide at a constant voltage of - 0.6 V vs Hg/HgSO(4) in which the rate of degradation was correlat

301 transformations of atmospheric mercury with Hg stable isotopes depends on the ability to collect amo

302 learly competes with thermal oxidation, with Hg(0) being the main photoproduct of Hg(II) photolysis i

303 nd capillary sources from 49 volunteers with Hg exposures similar to background populations (i.e., Me

304 tions of 16 elements (K, Na, Mg, Ca, Fe, Zn, Hg, Se, As, Cu, Cd, Mn, Ni, Cr, Pb and Co) were determin