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1  three restriction enzymes, EcoRI, PstI, and HindIII.
2 esent in overall gene frequencies of .39 for HindIII, .04 for BamHI, and .02 for EcoRI.
3 agments (AFs) generated with the primer pair HindIII+A and HhaI+A were compared.
4    In CAD patients (>60% stenosis; n = 483), HindIII (+) allelic carriage was increased (93.8% of pat
5             Topical application of liposomal HindIII also induced TNF-alpha in the epidermis of mice.
6 anephora were constructed using two enzymes, HindIII and BstYI.
7  digested with restriction enzymes BamHI and HindIII and cloned in bacteriophage M13mp18.
8 ccus faecium (VRE) isolates was cleaved with HindIII and HaeIII and subjected to agarose gel electrop
9  S. pneumoniae were cloned and used to probe HindIII and HindIII-plus-ApaI genomic DNA digests from o
10 wn shoots of J. regia cv. Chandler using the HindIII and MboI cloning sites.
11 ard arrow AAA, 1 inhibited DNA hydrolysis by HindIII and PspOMI at A downward arrow AGCTT and G downw
12 sis (PFGE) and automated ribotyping by using HindIII and PvuII.
13 to specifically label the HindIII termini in HindIII and Sau3AI restriction digests of clones that ar
14 or plasminogen activator inhibitor-1 (PAI-1) HindIII and tissue plasminogen activator (TPA) EcoRI res
15 urpose of this study was to test whether the HindIII (+) and PvuII (-) or (+) restriction enzyme-defi
16        In no-CAD control subjects (n = 168), HindIII (-) and (+) allelic frequencies were 28.6% and 7
17 ng: type I (EcoBI, EcoAI, Eco124I), type II (HindIII) and type III (EcoP1I).
18 e-site enzymes (BamHI, EcoRI, EcoRV, HaeIII, HindIII, and DNaseI).
19 ng of the different UGPase-cDNAs with BamHI, HindIII, and EcoRI revealed that at least two mRNA popul
20                BAC clones were digested with HindIII, and fragments were size separated on analytical
21 ctrophoretically after digestion with BamHI, HindIII, and MfeI.
22 ints, generated from each clone using BamHI, HindIII, and PstI endonucleases, were combined and used
23 p, the entire library was fingerprinted with HindIII, and the fingerprinted clones were assembled int
24         Live RSV vaccine D46/NS2/N/DeltaM2-2-HindIII, attenuated by deletion of the RSV RNA regulator
25 bridization experiments detected polymorphic HindIII, BamHI, and EcoRI sites.
26 fewer than 10% of the rRNA genes at NOR2 are HindIII-bearing variants.
27                           These ORFs, in the HindIII C fragment of MCMV, are colinear with the UL82,
28 CAD risk factors and the other polymorphism, HindIII (+) carriage was associated with an OR = 2.86, C
29                    The liposome-encapsulated HindIII caused double strand breaks in DNA and induced I
30 -bacteriophage with the fragments of its own HindIII digest, a standard DNA ladder, was sized by leng
31 ng this new protocol, accurate histograms of HindIII digested lambda DNA were demonstrated for DNA co
32  Class I SSRs in end-sequences of EcoRI- and HindIII-digested BAC clones was one SSR per 40 Kb, where
33                Southern blot analysis of the HindIII-digested chromosomal DNA demonstrated the presen
34 Restriction fragment length polymorphisms of HindIII-digested DNAs from six OspA-negative isolates di
35                    Southern blot analysis of HindIII-digested genomic DNA indicated that amplificatio
36 ved alterations in the patterns of PstI- and HindIII-digested proviral DNA were found to be due to th
37 enome was end sequenced and fingerprinted by HindIII digestion.
38 gulator gene (fur) as a probe, a 2.6-kb SalI/HindIII DNA fragment from an A. hydrophila chromosome wa
39 s, made using different restriction enzymes (HindIII, EcoRI, and MboI, respectively), were evaluated
40 tion efficiencies severalfold, while Asp718, HindIII, EcoRI, SalI, SmaI, HpaI, MscI, and SnaBI do not
41                                     A 3.1-kb HindIII-EcoRI fragment found in both cosmids was shown t
42 obe (395-bp G217B yps 21:E-9 probe or 512-bp HindIII-EcoRI-labelled Downs yps21:E-9).
43 llowed by a multiple restriction digest with HindIII, EcoRV, and AspI to generate restriction fragmen
44 second outbreak with eight patient isolates, HindIII found six were the same type and two were unique
45          The strong effect of a 1.9-kilobase HindIII fragment containing HS3 after, but not prior to,
46                   If for a given isolate the HindIII fragment detected by the probe was reduced in si
47  intronless genes contained on a common 8-kb HindIII fragment in a "head-to-head" orientation.
48                                     A 3.0-kb HindIII fragment of the 24.7-kb linear plasmid was used
49 kb of its promoter were isolated on a 5.4-kb HindIII fragment of this cosmid.
50 ous probe, revealed the presence of a 6.4 kb HindIII fragment that was detectable at only low stringe
51                           However, a 1.18-kb HindIII fragment was common among 18 of the 20 biotype 1
52 The GSTM1-0 deletion produces a novel 7.4-kb HindIII fragment with the loss of 10.3- and 11.4-kb Hind
53                                A 572-bp SacI-HindIII fragment, BmScH, that was previously shown to ex
54                                     A 4.4-kb HindIII fragment, encoding an unusual rubredoxin (denote
55 6.2-kb SacI fragment, P </= 0.05 and a 19-kb HindIII fragment, P </= 0.03).
56 range, 24 to 36 kb) that hybridized with the HindIII fragment.
57 wo CR1 alleles that differ in having genomic HindIII fragments of either 7.4 or 6.9 kb and that deter
58  (Zn2+ and Cu2+) interacting with lambda-DNA-HindIII fragments ranging from 2,027 to 23,130 bp in Tri
59  of Mg2+, Ca2+, and Co(NH3)63+ to lambda-DNA-HindIII fragments ranging from 2.0 to 23.1 kbp was used
60  fragment with the loss of 10.3- and 11.4-kb HindIII fragments.
61 termined the nucleotide sequence of a 5.2-kb HindIII genomic DNA fragment which contains the complete
62                          D46/NS2/N/DeltaM2-2-HindIII had excellent infectivity and immunogenicity and
63 ed with HaeIII (cna and fnbA-fnbB probes) or HindIII (hlb probe).
64 B radiation, however, treatment of mice with HindIII in liposomes before contact sensitization did no
65            Unlike UV-B irradiation, however, HindIII in liposomes failed to induce suppressor T cell
66                       Topical application of HindIII in liposomes to murine skin in vivo impaired the
67            Another chimeric enzyme, mBE I-II HindIII, in which the amino-terminal end of mBE II was r
68                    Automated ribotyping with HindIII is an accurate method for genetic fingerprinting
69           The common LPL polymorphic allele, HindIII (+), is moderately associated with CAD, and the
70 es demonstrate that deletion of sequences in HindIII-J and -I confer altered cell and tissue tropism.
71 deletion of 7.7 kb spanning portions of MCMV HindIII-J and -I.
72 ifference was observed between the BstYI and HindIII libraries (14.9 vs. 8.8 %).
73 re markers were hybridized to filters of the HindIII library, representing 6x coverage of the genome,
74                                            A HindIII M. pneumoniae fragment containing the lambda MP
75 -forming units [PFUs] of D46/NS2/N/DeltaM2-2-HindIII [n = 21] or placebo [n = 11]) and were monitored
76 om those of ESRVs upon digestion with EcoRI, HindIII, NdeI, KpnI, and ScaI.
77 two whole-genome restriction maps (EcoRI and HindIII) of R. sphaeroides strain 2.4.1 were constructed
78        Liposomes containing heat-inactivated HindIII or an endonuclease specific for pyrimidine dimer
79 somes, but not liposomes containing inactive HindIII or an irrelevant endonuclease, to the skin of C3
80 ae were cloned and used to probe HindIII and HindIII-plus-ApaI genomic DNA digests from other isolate
81 ily demonstrated that the segregation of the HindIII polymorphic site is consistent with Mendelian in
82                     We also report EcoRI and HindIII polymorphisms that may prove to be useful in dis
83  DNA, standard electrophoresis of BamHI- and HindIII-restricted DNA, Southern hybridization, restrict
84 al DNA damage in immune suppression, we used HindIII restriction endonuclease encapsulated in liposom
85 nd breaks in the DNA of epidermal cells with HindIII restriction endonuclease encapsulated in liposom
86 y primed polymerase chain reaction (AP-PCR), HindIII restriction enzyme analysis (REA), and pulsed-fi
87 ar to that of Co53, and the ApaI, BamHI, and HindIII restriction fingerprints of the total cellular D
88          This probe hybridized with a 4.0-kb HindIII restriction fragment from all 13 strains of S. g
89                         We also identified a HindIII restriction fragment length polymorphism of F8A
90 hich were physically mapped to five distinct HindIII restriction fragments.
91               One aphid clone bore a variant HindIII restriction site in the Buchnera leucine plasmid
92 be due to the appearance of PstI and loss of HindIII restriction sites in the pol region as a result
93                                          The HindIII RFLP and 3 of the latter 6 sites were analyzed i
94                                              HindIII ribotypes correlated well with PFGE.
95 ysis, single-copy landmark content analysis, HindIII-Sau3AI fingerprinting, and sequencing of PCR pro
96                     From Dam methylation and HindIII sensitivity tests, the methylation loci were pre
97 identified in the 5'-flanking region (from a HindIII site at position -1641).
98 es at NOR4 are homogeneous with respect to a HindIII site occurring once per gene.
99 on approximately 1.5 kbp downstream from the HindIII site of 5'Sgamma2 on chromosome 14q32 and identi
100 hloramphenicol resistance cassettes into the HindIII site of the kpsF coding sequence.
101              The presence of the polymorphic HindIII site results in a 7.0-kilobase (kb) band, and th
102 onstruct is extended by 600 bp beyond the 3' HindIII site to include a previously identified regulato
103 ' sequence information beyond the A gamma 3' HindIII site.
104                                    A 10.0 kb HindIII subclone (Hind10) of this insert yields a high f
105 agged dideoxy ATPs to specifically label the HindIII termini in HindIII and Sau3AI restriction digest
106                        In contrast, mBE I-II HindIII transferred more long chains (with a degree of p
107 ining only the RNA coding portion of the OAX HindIII unit both produce OAX RNA transcripts.
108 into Xenopus oocyte nuclei of either the OAX HindIII unit or a subclone containing only the RNA codin
109 NA transcript has been mapped within the OAX HindIII unit using S1 nuclease.
110 he complete DNA sequence of one of these OAX HindIII units is reported here.
111      OAX DNA is present in tandemly repeated HindIII units of 752 bp.
112 cribed from the bovine herpesvirus 4 (BHV-4) HindIII W fragment, in a region of the genome not conser
113  HaeIII was partly inhibited and cleavage by HindIII was enhanced, whereas BamHI, EcoRI, and DNaseI w
114                                         When HindIII-X and EcoRI-D primer sets were used, CMV DNA fro
115  148 specimens, primer pairs directed to the HindIII-X fragment region of CMV detected target DNA wit
116       In conclusion, primers directed to the HindIII-X fragment region were the most optimal for the
117 with symptomatic infection ranged from 100% (HindIII-X) to 20% (MIE gene); however, specificity was i
118  however, specificity was inversely related (HindIII-X, 45%; MIE gene, 91%) to primers directed to th

 
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