ライフサイエンスコーパス: ICA

1 ICA and heat map suggest four components best describe t

2 ICA confirmed 81% of calcific stenoses, 91% of mixed, an

3 ICA decomposed the S. aureus transcriptome into 29 indep

4 ICA duration and GCS 24 h after the event had the best p

5 ICA identified three DMNs.

6 ICA information-based methods identify both weakly activ

7 ICA is a mixed agonist of mutant EAG and EAG/ERG chimera

8 ICA of group resting-state functional MR imaging data re

9 ICA was formed <25 million years ago and, consequently,

10 ICA z images of DMN components were compared between the

11 ICA-069673 exhibits a functional signature that depends

12 ICA-105574 (ICA, or 3-nitro-N-[4-phenoxyphenyl]-benzamid

13 ents with dissection, right CCA (p = 0.000), ICA (p = 0.001), ECA (p = 0.004) diameters, total CCA (p

14 p = 0.004) diameters, total CCA (p = 0.001), ICA (p = 0.009), and ECA (p = 0.003) diameters were also

15 00-fold selectivity for human Nav1.3/Nav1.1 (ICA-121431, IC50, 19 nM) or Nav1.7 (PF-04856264, IC50, 2

16 which 33 arterial segments were treated (10 ICA, 15 MCA, and 8 ACA).

17 o-n-(4-phenoxyphenyl) benzamide [ICA-105574 (ICA)] has been discovered to activate hERG1 by strong at

18 ICA-105574 (ICA, or 3-nitro-N-[4-phenoxyphenyl]-benzamide) has oppos

19 Complete data were available for 1064 ICAs and 3348 CCAs.

20 zed property of the KCNQ2-specific activator ICA-069673 to identify assembly of heteromeric channels.

21 In acute ICA occlusion, the absence of AChA infarction or restric

22 hors studied consecutive patients with acute ICA occlusion admitted to an academic medical centre bet

23 Obstructive coronary artery disease after ICA was found in 61% of patients in the routine clinical

24 aboratory data before and 24 hours (h) after ICA were analysed as predictors for no return of spontan

25 kull), air-filled balloon [intracranial air (ICA)], water-filled balloon (cerebrospinal fluid and blo

26 We found empirical evidence of alternative ICA-based metrics of connectivity when comparing subject

27 Although ICA greatly attenuates ERG inactivation by shifting its

28 Isthmian Central America (ICA) is one of the most biodiverse regions in the world,

29 f sub-clinical isolated cardiac amyloidosis (ICA) at autopsy and the odds of AF in these patients.

30 S without the verbal component 24 h after an ICA had the highest AUC (0.616 [0.792-0.956]), with a se

31 outcome and their prognostic value after an ICA.

32 ast one significant stenosis (>= 50%) and an ICA within three months.

33 and qualitative analyses of the results, an ICA volume of 20 ml and initial ICP of 15 mmHg were reco

34 Then, isotope cluster analysis (ICA) was applied to the LC-ToF-MS data allowing specific

35 ims that two independent component analysis (ICA) algorithms, Infomax and FastICA, which are widely u

36 stment using independent component analysis (ICA) and feature selection using DecompPipeline, (ii) de

37 alyzed using independent component analysis (ICA) and network analysis.

38 iltering and independent component analysis (ICA) and require no prior assumptions about the spatial

39 e selection, independent component analysis (ICA) and SE to create orthogonal representations that de

40 onents using independent component analysis (ICA) and seed-based correlations.

41 is following independent component analysis (ICA) and voxel-level, brain-wide univariate correlation

42 d by spatial independent component analysis (ICA) and working-memory-load-dependent connectivity betw

43 Independent component analysis (ICA) for neuroimaging data allows the identification of

44 Independent component analysis (ICA) further identified between-group differences in eng

45 nalysis used independent component analysis (ICA) in 324 healthy controls, 296 SZ probands, 300 PBP p

46 ielded by an independent component analysis (ICA) of EEG data and measured event-related responses by

47 Local independent component analysis (ICA) revealed that this activity contained three distinc

48 Independent Component Analysis (ICA) showed that higher empathy scores were associated w

49 We use independent component analysis (ICA) to extract synergies from electromyography (EMG).

50 s the use of Independent Component Analysis (ICA) to identify variation likely caused by broad impact

51 onnectivity, independent component analysis (ICA) to reveal functional networks with a data-driven ap

52 We applied independent component analysis (ICA) to task-free fMRI data within a local region around

53 Independent component analysis (ICA) was applied to 20 variables indexing obesity and BP

54 Independent component analysis (ICA) was performed to derive resting-state functional MR

55 In addition, independent component analysis (ICA) was used to assess group differences in the express

56 Group independent component analysis (ICA) was used to decompose fMRI scans into 75 brain netw

57 with spatial independent component analysis (ICA), called ME-ICA, which distinguishes BOLD (neuronal)

58 By applying independent component analysis (ICA), we find that the spatial pattern of correlation is

59 Independent component analysis (ICA)-based mapping has shown advantage, as no a priori i

60 clination of independent component analysis (ICA).

61 troscopy and Independent Component Analysis (ICA).

62 followed by Independent Components Analysis (ICA) and by classical methods such as free radical scave

63 dependent and Principal Components Analysis (ICA, PCA) as well as Orthogonal Partial Least Squares Di

64 gies, including KCa3.1 blockers (TRAM-34 and ICA-17043) or KCa3.1-specific small hairpin RNA delivere

65 compared with SR coronary CT angiography and ICA (83% vs 53%, P < .001).

66 ovided with both coronary CT angiography and ICA has poor discriminatory power for ischemia-inducing

67 al networks with a data-driven approach, and ICA-based interhemispheric correlation analysis.

68 separate day, however, exercise capacity and ICA, MCA Vmean and CCA dynamics were preserved.

69 sitybicyclo[2.2.1]heptane-2-carboxamide) and ICA-069673 N-(6-chloropyridin-3-yl)-3,4-difluorobenzamid

70 obstructive CAD (>50% stenosis) on CCTA and ICA.

71 re read by 3 experts blinded to clinical and ICA data.

72 ned by a stenosis of 50% or larger on CT and ICA.

73 ent both quantitative H(2)(15)O PET/CTCA and ICA.

74 t the combination of UV-Vis measurements and ICA makes possible the EVOO evaluation, and can contribu

75 Based on these results, ML213 and ICA-069673 likely bind to different sites and are differ

76 We compared the effects of ML213 and ICA-069673 on homomeric human Kv7.4, Kv7.5, and heterome

77 Folin-Ciocalteu, ORAC, DPPH, ABTS, PFRAP and ICA), inhibitory property against beta-amyloid and alpha

78 r 2-day rest-stress Tc-99m-labeled SPECT and ICA.

79 of follow-up invasive coronary angiography (ICA) and revascularization (REV) after coronary computed

80 (MBIR) using invasive coronary angiography (ICA) as a standard of reference.

81 (AF) by using invasive coronary angiography (ICA) as the reference method and to compare the results

82 referred to immediate coronary angiography (ICA) irrespective of their first postresuscitation ECG a

83 Invasive coronary angiography (ICA) with measurement of fractional flow reserve (FFR) b

84 inely undergo invasive coronary angiography (ICA), but many do not have obstructive coronary artery d

85 underwent CT, invasive coronary angiography (ICA), FFR, and FFR(CT) between October 2010 and October

86 UC underwent invasive coronary angiography (ICA), whereas 4.6% of patients undergoing CCTA versus 2.

87 lso underwent invasive coronary angiography (ICA).

88 (48 to 72 h) invasive coronary angiography (ICA).

89 PET/CTCA and invasive coronary angiography (ICA).

90 stenosis) on invasive coronary angiography (ICA).

91 ts undergoing invasive coronary angiography (ICA).

92 y referral to invasive coronary angiography (ICA).

93 comparison to invasive coronary angiography (ICA).

94 quantitative invasive coronary angiography (ICA).

95 Isolated congenital anosmia (ICA) is a rare condition that is associated with life-lo

96 antibodies, including islet cell antibodies (ICAs), reflect adaptive immunity, while increased serum

97 Applying ICA to a model problem, construction of custom transcrip

98 predictors of intraoperative cardiac arrest (ICA) outcomes are scarce in the literature.

99 the internal and external carotid arteries (ICA and ECA, respectively) and vertebral artery (VA) (Du

100 s (CCAs) and in one internal carotid artery (ICA) 2 mm above the flow divider.

101 was measured at the internal carotid artery (ICA) and vertebral artery (VA) and CBF velocity at the m

102 avernous segment of internal carotid artery (ICA) is a relatively rare entity.

103 guish true cervical internal carotid artery (ICA) occlusion from pseudo-occlusion (defined as an isol

104 s) and extracranial internal carotid artery (ICA) stenosis as risk factors for silent cerebral infarc

105 cervical segment of internal carotid artery (ICA) together with a dissecting aneurysm in the cavernou

106 asospasm>50% of the internal carotid artery (ICA), anterior cerebral artery (ACA), and / or middle ce

107 bral blood vessels [internal carotid artery (ICA), basilar artery (BA), middle cerebral artery (MCA)]

108 of the intracranial internal carotid artery (ICA; 26 patients: median National Institutes of Health S

109 rtery (CCA) and the internal carotid artery (ICAs) and with incident or progressive plaque in the ICA

110 occlusion (terminal internal carotid artery [ICA] with M1 middle cerebral artery and/or A1 anterior c

111 Isolated congenital asplenia (ICA) is characterized by the absence of a spleen at birt

112 he etiology of isolated congenital asplenia (ICA), a life-threatening human condition, are unknown.

113 novel flow cytometry-based Ig capture assay (ICA) for the identification and sorting of individual Ab

114 tenuated in vivo by infectious center assay (ICA).

115 veloped a novel immunochromatographic assay (ICA) with enhanced sensitivity for the visual and quanti

116 d, NY), a rapid immunochromatographic assay (ICA), and the InBios ZIKV Detect 2.0 IgM antibody captur

117 ch we refer to as iterative capped assembly (ICA), that adds DNA repeat monomers individually to a gr

118 of patients were found to have asymptomatic ICA.

119 A >/=50% stenosis at ICA or a fractional flow reserve </= 0.80 was considered

120 3-Nitro-n-(4-phenoxyphenyl) benzamide [ICA-105574 (ICA)] has been discovered to activate hERG1

121 the first direct comparison of RSNs between ICAs and graph-based network analyses at a comparable re

122 0% was found by coronary CTA in 68.9% and by ICA in 67.4% of the patients.

123 CCTA data were confirmed by ICA in 89% of subjects, and in 73% and 94% of patients w

124 37 (54.4%) had an abnormal FFR determined by ICA.

125 able to the corresponding RSNs identified by ICA.

126 hERG1 channel (G628C/S631C) was inhibited by ICA and that the addition of the F557L mutation rendered

127 idaz PET (for detection of >=50% stenosis by ICA) was 71.9% (95% confidence interval [CI]: 67.0% to 7

128 lex ultrasonography at the internal carotid (ICA) and vertebral arteries (VA).

129 In each case, ICA-069673 induced a negative shift of the activation cu

130 Results Cervical ICA pseudo-occlusion occurred in 12 of 37 patients (32.4

131 strate a lack of attenuation in the cervical ICA on the symptomatic side (24 men and 13 women; mean a

132 (32.4%) with nonattenuation of the cervical ICA on the symptomatic side.

133 es to assess whether there was true cervical ICA occlusion.

134 In contrast, ICA-121431, a small-molecule Nav1.1 inhibitor, accelerat

135 Based on these data, ICA could be used to determine the contribution of chemi

136 ) without known CAD who underwent diagnostic ICA within 6 mo of HS-SPECT and 86 consecutive patients

137 arotid artery (cICA) can be caused by distal ICA occlusion.

138 Patients who underwent MT to treat distal ICA occlusions between July 2012 and March 2018 were rev

139 missense mutations-cause autosomal dominant ICA by haploinsufficiency.

140 For patients with <50% stenosis, early ICA rates were elevated; over the entirety of follow-up,

141 ble logistic regression was used to estimate ICA use.

142 For ICA pseudoaneurysms treated with aspirin and observation

143 ive hazard for death was 2.2 (p = 0.011) for ICA versus no ICA.

144 , the relative hazard for death was 0.61 for ICA versus no ICA (p = 0.047).

145 ing to the postresuscitation ECG blinded for ICA results: (1) ST elevation or presumably new left bun

146 ocardial infarction is a safe gatekeeper for ICA.

147 phy (CTA) may serve as a safe gatekeeper for ICA.

148 in detecting patients with an indication for ICA after experiencing a cardiac arrest.

149 ds that form the putative binding pocket for ICA in ERG are conserved in EAG.

150 isk for CAD (n = 184) who were scheduled for ICA were randomly assigned for study with SR (n = 91) or

151 For the testing group, ICA and seed-based correlation were separately computed

152 Elevated levels of CML in ICA(+) children were a persistent, independent predictor

153 nd ERG channels is related to differences in ICA binding site or to intrinsic mechanisms of inactivat

154 rivers of isolation on genetic divergence in ICA.

155 The absolute increase in ICA after CCTA was 21 per 1,000 CCTA patients (95% CI: 1

156 CML was increased in ICA(+) and prediabetic schoolchildren and in diabetic an

157 In all conditions, reductions in ICA and MCA Vmean were associated with declining cerebra

158 tic divergences and ultimately speciation in ICA remain poorly studied.

159 sed ED cost and length of stay but increased ICA and revascularization.

160 We assessed internal (ICA) and common carotid artery (CCA) haemodynamics (indi

161 nt) or tandem (extracranial or intracranial) ICA and M1 occlusion subgroup also showed a trend favori

162 xt of acute ischemic stroke with ipsilateral ICA nonattenuation at single-phase CT angiography, even

163 ismal patients were compared, right and left ICA diameters (p = 0.000, p = 0.002, respectively), tota

164 Subtype-specific channel activators like ICA-069673 are a reliable tool to identify heteromeric a

165 y (269 +/- 11 vs. 336 +/- 14 W), and lowered ICA and MCA Vmean by 12-23% without compromising CCA blo

166 Seed-based analysis showed that many ICA components exhibited strong and significant (P < .05

167 dian 16 mum/year; P < 0.001) and the maximal ICA-IMT increased in 70% (median 25 mum/year; P < 0.001)

168 For the maximal ICA-IMT, cumulative prednisone exposure was associated w

169 ependent component analysis (ICA), called ME-ICA, which distinguishes BOLD (neuronal) and non-BOLD (a

170 In contrast to ML213, ICA-069673 robustly activated Kv7.4 channels but was sig

171 Most ICA procedures (79%) occurred </=3 months of CCTA.

172 ns abolished the activator effects of 30 muM ICA, including L622C in the pore helix, F557L in the S5

173 sequencing studies have implicated two MYRF ICA domain mutations (V679A and R695H) in a novel syndro

174 hazard for death was 0.61 for ICA versus no ICA (p = 0.047).

175 death was 2.2 (p = 0.011) for ICA versus no ICA.

176 of proximal intracranial arterial occlusion (ICA vs MCA-M1 vs MCA-M2) was not.

177 ation in S6 (A653M) switched the activity of ICA from an activator to an inhibitor, revealing its par

178 (n = 621), there was a strong correlation of ICA with CCTA findings (OR: 9.09, 95% CI: 5.57 to 14.8,

179 of more than 50% compared with detection of ICA were 96.4% and 98.7%, respectively, in the chronic A

180 G1 to identify the molecular determinants of ICA action.

181 modalities to aid in antemortem diagnosis of ICA and to establish the optimal management strategies i

182 Simulated molecular docking of ICA to homology models of hERG1 corroborated the scannin

183 etter understand the evolutionary history of ICA's diverse biotas.

184 rs for no ROSC, a threshold of 13 minutes of ICA yielded the highest area under curve (AUC) (0.867[0.

185 is useful to select patients with no need of ICA.

186 for role of CNGA2 gene with pathogenicity of ICA in humans.

187 ver the entirety of follow-up, predictors of ICA were mild left main, mild proximal CAD, respectively

188 ctivation rates decreased in the presence of ICA-069673 in a subunit-specific manner.

189 tive CAD was associated with higher rates of ICA and REV for 1-vessel (44.3% and 28.0%), 2-vessel (53

190 The odds ratio of ICA for CCTA patients versus UC patients was 1.36 (95% c

191 This study show that reperfusion of ICA can increase RCBF following embolization, but this i

192 ced in the narrowed and dissected segment of ICA, and the dissecting aneurysm of the cavernous segmen

193 ce of coronary CT angiography versus that of ICA in each group.

194 icant CAD when FFR determined at the time of ICA was the reference standard.

195 The use of ICA resolution provided information over dienes (primary

196 dures were performed, including 158 cases of ICAs.

197 uminal narrowing) was assessed on CTA and on ICA.

198 In patients with CAD on ICA, the magnitude of reversible defects was greater wit

199 Coronary artery stenosis was estimated on ICAs based on a qualitative method.

200 d linear approaches such as sparse coding or ICA.

201 Para-ICA identified five sub-DMNs that were significantly ass

202 Para-ICA identified four ERP components that were significant

203 arallel independent component analysis (para-ICA) to determine which empirically derived gene cluster

204 ) then was subjected to a parallel ICA (para-ICA) to identify imaging-genetic relationships.

205 1-million-SNP array) was examined using para-ICA.

206 s (n = 549) then was subjected to a parallel ICA (para-ICA) to identify imaging-genetic relationships

207 ICA methods, including a single-participant ICA on the basis of a comprehensive template from core s

208 phylogeny indicated that the prevalent plant ICA genes encoding two tRNA(His) guanylyl transferase 1

209 The postulated ICA expansion and the subsequent increase in ICP in pneu

210 coronary artery disease (CAD); accurate pre-ICA risk stratification is needed.

211 he Intramolecular Chaperone Auto-processing (ICA) domain of Myrf forms a homo-trimer, which carries o

212 The Q(ICA) was unchanged with EX and HS interventions (P = 0.6

213 were determined by radioimmunoprecipitation, ICA was determined by indirect immunofluorescence, and H

214 The CMR- and CTA-first strategies reduced ICA compared with routine clinical care (87% [p = 0.001]

215 regions, after artefact removal with FMRIB's ICA-based X-noiseifier.

216 zation of a large Iranian family segregating ICA.

217 Similarly, ICA revealed worse rs-fMRI expression scores across all

218 Since ICA builds full-length constructs from individual monome

219 tivity estimates obtained from group spatial ICA followed by dual regression.

220 tly identified across subjects using spatial ICA (independent component analysis).

221 occlusions; carotid T- or L-type and tandem ICA and M1 occlusions showed greater recanalization and

222 Together, our findings indicate that ICA is a mixed agonist of hERG1 channels.

223 Together these findings suggest that ICA binds to the same site in EAG and ERG channels to el

224 with site-directed mutagenesis suggests that ICA binds in a channel-specific orientation to a hydroph

225 The ICA homo-trimer-mediated auto-cleavage of Myrf is a prer

226 The ICA provides important added benefits in that phenotypic

227 The ICA represents a fast and versatile tool for single-cell

228 e CA slopes ranging from 0.37 to 2.20 at the ICA and from 0.17 to 3.18 at the VA; no differences in C

229 no differences in CA were found between the ICA and VA.

230 nt (P < .05) correlations, corroborating the ICA results.

231 re, we show that V679A and R695H cripple the ICA domain, blocking the auto-cleavage of Myrf.

232 We envisage the ICA as being a useful tool in Ab repertoire analysis for

233 Except for the ICA assay, a good correlation between phenolic content a

234 Atherosclerotic thickening in the ICA appears to be accommodated for vessels with a maximu

235 e degree of stenosis was 18%+/-11.65% in the ICA, 30.67%+/-18.45% in the MCA, and 28.38%+/-15.49% in

236 In the ICA, lumen area was relatively constant across patients

237 d with incident or progressive plaque in the ICA/carotid bulb, were explored.

238 gion around the bilateral TPJ, iterating the ICA at multiple model orders and in several datasets.

239 H abrogate the auto-cleavage function of the ICA homo-trimer by destabilizing its homo-trimeric assem

240 Complete sympathetic reinnervation of the ICA was observed at long term survival times, yet TH inn

241 To show the specificity of the ICA, we produced Ag-specific Abs from these cells and su

242 h-type immunoreactions were performed on the ICA, and Pt-Au bimetal nanoparticles (NPs) were accumula

243 tations introduced into EAG to replicate the ICA binding site in ERG did not alter the functional res

244 all short of proving this claim and that the ICA algorithms are indeed doing what they are designed t

245 We also found that the ICA homo-trimer can tolerate one copy of Myrf-V679A or M

246 Furthermore, we used the ICA to track Ag-specific plasmablast responses in HIV-va

247 s a valid noninvasive imaging alternative to ICA in selected patients at low to intermediate risk of

248 A may be used effectively as a gatekeeper to ICA.

249 blinded coronary CTA was conducted prior to ICA in both groups.

250 secondary safety endpoints were referral to ICA during hospitalization and 1-year outcomes (major ad

251 Kv7.4 W242L responded to ICA-069673-like wild-type Kv7.4, but a Kv7.4 F143A mutan

252 e whether the inverse functional response to ICA in EAG and ERG channels is related to differences in

253 ERG did not alter the functional response to ICA.

254 channels displayed intermediate responses to ICA-069673.

255 v7.4 F143A mutant was much less sensitive to ICA-069673.

256 Computed tomography, ICA, FFR, and FFR(CT) were interpreted in blinded fashio

257 (p = 0.000, p = 0.002, respectively), total ICA diameters (p = 0.000), carotid left O diameters (p =

258 Traumatic ICA pseudoaneurysms are safely treated with daily aspiri

259 We retrospectively identified all traumatic ICA pseudoaneurysms diagnosed on head/neck computed tomo

260 natural history and management of traumatic ICA pseudoaneurysms is controversial.

261 2; range, 55-83 years]) scheduled to undergo ICA for suspected ISR were enrolled.

262 ass index range 17 to 39 kg/m(2)) undergoing ICA for CAD evaluation, a CTA was acquired using very lo

263 Among patients undergoing ICA (n = 621), there was a strong correlation of ICA wit

264 est that only a third of patients undergoing ICA have obstructive coronary artery disease (CAD); accu

265 t without a clear noncardiac cause underwent ICA.

266 Eighty-six patients underwent ICA, and 39 had low-likelihood of CAD.

267 They underwent ICA and coronary CT angiography performed with a whole-h

268 oronary CT angiography before they underwent ICA.

269 In 86 patients who underwent ICA, sensitivity of PET was higher than SPECT (78.8% vs.

270 Post-CCTA patterns of follow-up ICA and REV are incompletely defined.

271 Follow-up ICA was recommended when initial CMR or CTA suggested my

272 During follow-up, ICA rates for patients with no CAD to mild CAD according

273 We used ICA to synthesize 20 TALENs of varying DNA target site l

274 Using ICA, we investigated the differences in resting-state br

275 rmed at 3 T to characterize the DMN by using ICA methods, including a single-participant ICA on the b

276 After parcellating the cortex using ICA and segmenting the thalamus based on dominant connec

277 s the negative predictive value (NPV), using ICA as the reference standard.

278 nguage network in brain tumor subjects using ICA on rs-fMRI.

279 ion scores within the planum temporale using ICA.

280 y involved extracranial stenosis segment was ICA, present in 14 (66.6%) out of 21 extracranial segmen

281 unselected schoolchildren screened, 115 were ICA(+) and were tested for baseline CML and diabetes aut

282 e largest DeltaICP was 5 mmHg; obtained when ICA volume and ROC were 20 ml and 1,600 ft/min, respecti

283 10 (38.5%), 20 (38.5%), and 26 (54.2%) with ICA, MCA-M1, and MCA-M2 occlusions, respectively, achiev

284 T angiography showed a better agreement with ICA for calcified plaques compared with SR coronary CT a

285 y and were compared with those attained with ICA.

286 The strong association of CCTA with ICA suggests that it may serve as an effective "gatekeep

287 with high diagnostic accuracy compared with ICA ( 3 , 4 ).

288 tion with the two scanners was compared with ICA and intravascular US.

289 laque composition, being lower compared with ICA for patients with lower CAC score and soft plaques;

290 tecting coronary stenosis when compared with ICA.

291 and initial ICP, was found to increase with ICA volume and ROC.

292 exome sequencing of a multiplex kindred with ICA, we identify a heterozygous missense mutation (P236H

293 Patients with ICA were older and had a higher odds of AF independent o

294 34), and 40.9% (9 of 22) among patients with ICA, MCA-M1, and MCA-M2 occlusions, respectively.

295 In this first comparison of HS-SPECT with ICA, new automated quantification of combined upright an

296 Amongst patients with AF, those with ICA were more likely to have persistent forms of AF and

297 ysis was performed for cross validation with ICA networks by using Pearson correlation.

298 The Chembio DPP Zika ICA and InBios ZIKV 2.0 MAC-ELISA showed >95% specificit

299 med ZIKV samples, while the Chembio DPP Zika ICA was nonreactive in three (20%) and the InBios ZIKV M

300 re the Chembio DPP Zika IgM system (DPP Zika ICA; Chembio, Medford, NY), a rapid immunochromatographi