ライフサイエンスコーパス: ICC

1 ICC Ca(2+) transient firing regulated the resting membra

2 ICC express a Ca(2+) -activated Cl(-) conductance (CaCC)

3 ICC for these gradings was 0.991.

4 ICC was borderline higher for private practice readers t

5 ICC-IM are sustained by voltage-independent Ca(2+) influ

6 ICC-IM responses to EFS were ablated by inhibiting Ca(2+

7 ICC-SS express Ano1 channels and generate spontaneous Ca

8 ICC-SS fired stochastic localized Ca(2+) transients.

9 ICCs for these metrics ranged from 0.90 to 1.0 for basel

10 ICCs improved to 0.84-0.99 (CAC) and 0.92-0.99 (TAC) for

11 ICCs were moderate between WISC-V tasks (0.663), and rel

12 ICCs were similar for the three different health groups,

13 084) were significantly different (P < .001; ICC: 0.66; 95% CI: 0.53-0.76).

14 iod (aOR, 0.89; 95% CI, 0.78-1.02; p = 0.09; ICC, 0.1538).

15 unsupervised TME-based classification of 198 ICCs (training set) and 368 ICCs (validation set).

16 We showed that a set of 198 ICCs could be classified into 4 TME-based subtypes relat

17 Epithelial and stromal compartments of 23 ICCs were laser microdissected.

18 were not significantly different (P = .319; ICC: 0.59; 95% CI: 0.45-0.71).

19 ification of 198 ICCs (training set) and 368 ICCs (validation set).

20 was confirmed over an independent set of 368 ICCs and by immunohistochemical analysis of 64 ICC tissu

21 mentation (coefficient of variation = 12.6%; ICC = 0.86) were consistent.

22 Cs and by immunohistochemical analysis of 64 ICC tissue samples.

23 rs of age (ICC = 11 to 34%) than those >=65 (ICC = 5 to 6%) and for traumatic brain injury (ICC = 5 t

24 shed the bulk gene expression profiles of 78 ICCs.

25 opausal female liver cancer cases (HCC n=83, ICC n=56) and 426 controls, matched on sex, cohort, age,

26 postmenopausal female LC cases (HCC, n = 83; ICC, n = 56) and 426 controls, matched on sex, cohort, a

27 eader variability was excellent (Kappa:0.95, ICC:0.98).

28 s in ICC-IM is not coordinated with adjacent ICC-IM in a field of view or even with events from other

29 al variation for patients < 65 years of age (ICC = 11 to 34%) than those >=65 (ICC = 5 to 6%) and for

30 -assisted analysis than visual analysis (all ICC >=0.960 versus >=0.885).

31 iability analyses showed high agreement (all ICCs > 0.990).

32 An ICC above 0.75 was regarded as high.

33 re found, with a CV between 1 and 2%, and an ICC of more than 0.970.

34 nly 147 out of 1438 metabolites (10%) had an ICC below 0.4 indicating poor stability over time.

35 r reliability of the overall scale showed an ICC of 0.95 (95% CI, 0.96-0.99) using a 2-sample random-

36 d-certified radiologists (n = 30) yielded an ICC of 0.945 (95% CI 0.889 to 0.973).

37 e forms, (ICC = 0.69, 95% CI = 0.51-0.81 and ICC = 0.75, 95% CI = 0.51-0.88, respectively).

38 emphigus (ICC = 0.82, 95% CI = 0.63-0.92 and ICC = 0.80, 95% CI = 0.62-0.90, respectively) than in pa

39 nvolved in production of eADO from eNAD, and ICC were not involved in extracellular metabolism of eNA

40 s of LM are an emergent property of SMCs and ICC-SS.

41 d CIN2 (18%-25%), up to 41%-47% for CIN3 and ICCs.

42 liability for both systems was high (average ICC = 0.959).

43 asurements of D (max) (ICC, 0.807), D (avg) (ICC, 0.823), P (max) (ICC, 0.787), P (avg) (ICC, 0.787),

44 (ICC, 0.823), P (max) (ICC, 0.787), P (avg) (ICC, 0.787), and F (max) (ICC, 0.659).

45 y examined the possible relationship between ICC-IM and SWs by recording Ca(2+) transients in mice ex

46 PP1 and NT5E, (3) eNAD is not metabolized by ICC, (4) NT5E is expressed chiefly by SMCs and moderatel

47 re expressed by interstitial cells of Cajal (ICC) but not by smooth muscle cells (SMCs).

48 Interstitial cells of Cajal (ICC) generate slow waves and transduce neurotransmitter

49 Disruptions in interstitial cells of Cajal (ICC) have also been reported.

50 Ws) produced by interstitial cells of Cajal (ICC) underlie phasic contractions in other gastrointesti

51 e cells (SMCs), interstitial cells of Cajal (ICC), and cells expressing platelet-derived growth facto

52 e cells (SMCs), interstitial cells of Cajal (ICC), and cells expressing platelet-derived growth facto

53 le cells (SMC), interstitial cells of Cajal (ICC), and PDGFRalpha(+) cells (fibroblast-like cells) is

54 exclusively in interstitial cells of Cajal (ICC).

55 c intramuscular interstitial cells of Cajal (ICC-IM) are associated with cholinergic varicosities, su

56 c intramuscular interstitial cells of Cajal (ICC-IM) exhibit spontaneous Ca(2+) transients manifestin

57 scular class of interstitial cells of Cajal (ICC-IM).

58 ich include the interstitial cells of Cajal (ICCs) from which GISTs presumably originate, and (b) tem

59 progression to invasive cervical carcinoma (ICC).

60 ucibility across striatal regions (K(i)(cer) ICC: 0.68-0.94, SUVRc ICC: 0.76-0.91).

61 ar and intrahepatic cholangiocarcinoma (cHCC-ICC) cases, including separate, combined, and mixed subt

62 may serve as a biomarker for diagnosing cHCC-ICC.

63 t biological and clinical insights into cHCC-ICC.

64 Integrative comparison of cHCC-ICC with hepatocellular carcinoma and intrahepatic chola

65 Notably, cHCC-ICCs showed significantly higher expression of Nestin, s

66 a revealed that combined and mixed type cHCC-ICCs are distinct subtypes with different clinical and m

67 C-ICCs, whereas combined and mixed type cHCC-ICCs were all monoclonal origin.

68 ulticlonal origins in the separate type cHCC-ICCs, whereas combined and mixed type cHCC-ICCs were all

69 Intrahepatic cholangiocarcinoma (ICC) accounts for 12% of cases and has rates only 30% hi

70 Intrahepatic cholangiocarcinoma (ICC) is a severe malignant tumor in which the standard t

71 Intrahepatic cholangiocarcinoma (ICC) is asevere malignant tumor in which the standard th

72 tients with intrahepatic cholangiocarcinoma (ICC) respectively.

73 on found in intrahepatic cholangiocarcinoma (ICC), disables its role in enhancing TGF-beta signaling

74 ass-forming intrahepatic cholangiocarcinoma (ICC), we modeled ICC desmoplasia and progression in vitr

75 out 15% of intrahepatic cholangiocarcinomas (ICCs) express constitutively active fibroblast growth fa

76 g Cohen's Kappa and intra-class coefficient (ICC).

77 with an intraclass correlation coefficient (ICC(3,k)) of 0.783 (95% confidence interval [CI]: 0.743,

78 hildren [intraclass correlation coefficient (ICC) = 0.905, P < 0.001] and their parents (ICC = 0.856,

79 median intra-class correlation coefficient (ICC) across all metabolites and all study participants w

80 using an intraclass correlation coefficient (ICC) and patient ranking consistency was assessed using

81 The intraclass correlation coefficient (ICC) and Spearman rank correlation coefficient were calc

82 Intraclass correlation coefficient (ICC) between the four PS measures, as well as PC1, were

83 ting the intraclass correlation coefficient (ICC) from linear mixed effects models.

84 ) had an intraclass correlation coefficient (ICC) of 0.985 (95% CI 0.961 to 0.995) for pancreas volum

85 The intraclass correlation coefficient (ICC) of each scan setup was calculated and compared acro

86 The intraclass correlation coefficient (ICC) ranged from 0.703 to 0.989 and from 0.950 to 0.998

87 SC) and intra-class correlation coefficient (ICC) respectively.

88 The intraclass correlation coefficient (ICC) was 0.55 (95% CI: 0.40-0.68).

89 The intraclass correlation coefficient (ICC) was estimated to assess reproducibility and repeata

90 ameters, intraclass correlation coefficient (ICC) was higher than 0.98, indicating excellent consiste

91 The intraclass correlation coefficient (ICC) was used to measure the reliability and agreement b

92 nalysis, intraclass correlation coefficient (ICC), Spearman correlation coefficient, and Wilcoxon sig

93 ement by intraclass correlation coefficient (ICC).

94 sing the intraclass correlation coefficient (ICC).

95 lpha and Intraclass Correlation Coefficient (ICC).

96 ng rest (Intraclass correlation Coefficient [ICC] = 0.79, mean absolute percentage error [MAPE] = 4.5

97 cohort (intraclass correlation coefficient [ICC] = 0.94) than the prospective cohort (ICC = 0.79).

98 radians (intraclass correlation coefficient [ICC] = 0.96).

99 ibility (intraclass correlation coefficient [ICC]).

100 .002; intra-cluster correlation coefficient [ICC], 0.0286).

101 as good (intraclass correlation coefficient [ICC], 0.71), with absolute differences ranging from zero

102 f 0.74% (intraclass correlation coefficient [ICC], 0.996; 95%CI, 0.989;0.998) compared with METAVOL.

103 The intraclass correlation coefficients (ICC) between the automated detection software and Observ

104 rn) and intraclass correlation coefficients (ICC) were used to determine individual stability across

105 s (CI), intraclass correlation coefficients (ICC), and Bland-Altman plots with linear regression.

106 istics, intraclass correlation coefficients (ICC), and linear mixed effects regression were used to d

107 n using intraclass correlation coefficients (ICC).

108 ssed by intraclass correlation coefficients (ICC).

109 Intraclass correlation coefficients (ICCs) and Bland- Altman analysis was used to assess intr

110 Intraclass correlation coefficients (ICCs) and Bland-Altman plots were used to assess intra-

111 Intraclass correlation coefficients (ICCs) and Bland-Altman plots were used to assess the rel

112 y using intraclass correlation coefficients (ICCs) and linear mixed models.

113 ed with intraclass correlation coefficients (ICCs) between DL and manual (Agatston) CAC and (volume)

114 ces and intraclass correlation coefficients (ICCs) for the radiologists and artificial intelligence (

115 culated intraclass correlation coefficients (ICCs) of biomarkers measured in 24-h urine samples that

116 the intra-cluster correlation coefficients (ICCs) of five major CVD risk factors (raised blood gluco

117 ion and intraclass correlation coefficients (ICCs) were calculated for the measures in each task.

118 Intraclass correlation coefficients (ICCs) were calculated for the relative abundance of 3 ph

119 Intraclass correlation coefficients (ICCs) were calculated, and Bland-Altman analysis was per

120 measure intraclass correlation coefficients (ICCs) were used to assess interreader agreement and agre

121 Intraclass correlation coefficients (ICCs) were used to estimate reproducibility and symmetry

122 istics, intraclass correlation coefficients (ICCs), and linear mixed-effects regression were used to

123 ed with intraclass correlation coefficients (ICCs).

124 y using intraclass correlation coefficients (ICCs).

125 d using intraclass correlation coefficients (ICCs).

126 use of intraclass correlation coefficients (ICCs).

127 ots and intraclass correlation coefficients (ICCs).

128 cs (via intraclass correlation coefficients, ICCs) of the miRNA profile over 3 time points sampled ac

129 t [ICC] = 0.94) than the prospective cohort (ICC = 0.79).

130 Colonic ICC-IM receive excitatory inputs from cholinergic neuron

131 increased Ca(2+) transient firing in colonic ICC.

132 1) and Kcnj11 (Kir6.2) were found in colonic ICC.

133 We employed Ca(2+) imaging of colonic ICC-IM in situ, using mice expressing GCaMP6f in ICC to

134 ion using increased collagenase combination (ICC) or increased protease combination (IPC), to the sta

135 5 +/- 4300 squared degrees) were concordant (ICC = 0.94).

136 sports bra was valid across all conditions (ICC = 0.99, MAPE = 0.66%, LoA = -19 to 19), and the Sens

137 l analysis (intrinsic connectivity contrast, ICC) to resting-state fMRI data acquired in 108 individu

138 Intraclass correlation (ICC) and coefficient of variation (CV) were used for the

139 The intraclass correlation (ICC) of both intragrader and intergrader reliability for

140 d left eyes for FAZ (intraclass correlation [ICC], 0.97), SVC VLD (ICC, 1.00), and DVC VLD (ICC, 1.00

141 een CT and MRI were within the corresponding ICC variation for all four measurement methods.

142 differences: 12.2 degrees and 6.2 degrees ; ICC: -0.02 and 0.21, respectively), and differences incr

143 addox values (mean difference: 1.5 degrees ; ICC: 0.85), but differences increased with greater cyclo

144 lodeviation (mean difference: -2.7 degrees ; ICC: 0.71).

145 Results VERDICT MRI FIC demonstrated ICCs of 0.87-0.95.

146 Two distinct ICC-IM populations were identified.

147 af(V600E) activation was sufficient to drive ICC hyperplasia but not GIST tumorigenesis.

148 o precision medicine approaches to FF-driven ICC.

149 the presence of L-NNA and MRS 2500 enhanced ICC-IM Ca(2+) transients.

150 Enhancing ICC-IM Ca(2+) release and Ano1 activation contributes to

151 g mice expressing GCaMP6f in ICC to evaluate ICC-IM responses to excitatory neurotransmission.

152 and inter-observer agreement was excellent (ICC >0.99) and highly significant independent of the obs

153 were lower in patients with moderate extent (ICC = 0.44, 95% CI = 0.004-0.74) than in those with inte

154 ents with intermediate (significant) extent (ICC = 0.50, 95% CI = 0.27-0.68).

155 16 months for nivolumab versus 14 months for ICC (hazard ratio, 0.95; 95.54% CI, 0.73 to 1.24); media

156 y validate a simplified prognostic score for ICC and to determine perioperative outcomes for large mu

157 those with intermediate or extensive forms, (ICC = 0.69, 95% CI = 0.51-0.81 and ICC = 0.75, 95% CI =

158 ospitals was observed for all 3 time frames (ICC = 6.5%, 6.6%, and 5.9% for < 72 h, 3-14 d, and >=14

159 hree different FFs, previously isolated from ICC samples, are heat shock protein 90 (HSP90) clients a

160 , but most features still resulted in a high ICC (FBN, 52%-63%; FBW, 75%-85%).

161 The percentage of features yielding a high ICC decreased more in the multicenter setting.

162 , the percentage of features yielding a high ICC was larger for images reconstructed with EARL-compli

163 : The percentage of features yielding a high ICC was largest for the statistically equal replicates (

164 nt, was inconsistent and generally not high (ICC < 0.8) across binarization thresholds, devices, and

165 developed districts have a higher household ICC in rural areas.

166 Ca(2+) release patterns might determine how ICC respond to neurotransmission in these two regions of

167 ent in the IAS where only intramuscular ICC (ICC-IM) are found, however the evidence linking ICC-IM t

168 ility is tuned by input from subserosal ICC (ICC-SS), a population of ICC with previously unknown fun

169 dical imaging (ICC = 4 to 10%), and the ICU (ICC = 5 to 6%) than other activity centers.

170 services (ICC = 18 to 26%), medical imaging (ICC = 4 to 10%), and the ICU (ICC = 5 to 6%) than other

171 stern blotting (WB) and immunocytochemistry (ICC).

172 e cell-specific knockdowns of anoctamin-1 in ICC (Kit(CreERT2/+) ;Ano1(tm2jrr/+) ) in GI muscles.

173 Some of the properties of Ca(2+) in ICC-IM in the colon are similar to the behaviour of ICC

174 to enforce clinical resistance to BGJ398 in ICC patients retained full sensitivity to ganetespib in

175 Ca(2+) transients activate Ano1 channels in ICC-SS.

176 een linked to activation of Ano1 channels in ICC.

177 n the local CVM and specifically elevated in ICC patients.

178 M(3) ), and NK(1) receptors were enriched in ICC-IM.

179 lease from the endoplasmic reticulum (ER) in ICC, and blocking Ca(2+) release with a SERCA inhibitor

180 neural inputs regulate the Ca(2+) events in ICC-SS.

181 ough suppression of Ca(2+) release events in ICC.

182 IM in situ, using mice expressing GCaMP6f in ICC to evaluate ICC-IM responses to excitatory neurotran

183 murine proximal colon expressing GCaMP6f in ICC.

184 of LM (ICC-SS) in mice expressing GCaMP6f in ICC.

185 Thus, Ca(2+) handling in ICC is fundamental to colonic motility.

186 ng a genetically-encoded Ca(2+)-indicator in ICC (Kit-Cre-GCaMP6f).

187 We examined Ca(2+) handling mechanisms in ICC-IM of murine proximal colon expressing GCaMP6f in IC

188 perty as a result of integrated processes in ICC-SS and LSMC.

189 Suppression of Ca(2+) release in ICC limits the open probability of Ano1 channels, reduci

190 Ca(2+) release in ICC-IM activates Ano1, a Ca(2+) -activated Cl(-) conduct

191 ed that ANO1 expression could be resolved in ICC but not smooth muscle cells (SMCs) in the IAS and re

192 nsic nerves activated nitrergic responses in ICC-SS.

193 Firing of Ca(2+) transients in ICC-IM is not coordinated with adjacent ICC-IM in a fiel

194 Ca(2+) transients in ICC-IM rely on Ca(2+) release from the endoplasmic retic

195 Ca(2+) transients in ICC-SS occurred by release from stores most probably via

196 rgic agonists inhibited Ca(2+) transients in ICC-SS, and stimulation of intrinsic nerves activated ni

197 All classes of ICC (including ICC-IM) express Ca(2+) -activated Cl(-) channels, encode

198 stradiol may be associated with an increased ICC risk.

199 We showed the existence of an inflamed ICC subtype, which is potentially treatable with checkpo

200 We revealed the existence of an inflamed ICC subtype, which is potentially treatable with checkpo

201 injury (ICC = 5 to 13%) than other injuries (ICC = 1 to 8%).

202 C = 5 to 6%) and for traumatic brain injury (ICC = 5 to 13%) than other injuries (ICC = 1 to 8%).

203 onfidence interval, 0-0.95), and intergrader ICC was 0.22 (95% confidence interval, 0-0.71).

204 (intraobserver, ICC >= 0.94; interobserver, ICC >= 0.89).

205 or NFV assessment (intra- and interobserver, ICC >= 0.99) and strong to excellent for regurgitant fra

206 nfidence interval, 0.53-0.96), interoperator ICC was 0.70 (95% confidence interval, 0-0.95), and inte

207 onfidence interval, 0.17-0.86), intersession ICC was 0.88 (95% confidence interval, 0.53-0.96), inter

208 anoctamin-1 may develop in small intestinal ICC with ageing and contribute to pacemaker activity.

209 Intragrader ICC was 0.84 (95% confidence interval, 0.57-0.95), intra

210 present in the IAS where only intramuscular ICC (ICC-IM) are found, however the evidence linking ICC

211 urgitant fraction assessment (intraobserver, ICC >= 0.94; interobserver, ICC >= 0.89).

212 onfidence interval, 0.57-0.95), intrasession ICC was 0.69 (95% confidence interval, 0.17-0.86), inter

213 -IM) are found, however the evidence linking ICC-IM to SWs is limited.

214 hat lies along the subserosal surface of LM (ICC-SS) in mice expressing GCaMP6f in ICC.

215 ding unidimensional measurements of D (max) (ICC, 0.807), D (avg) (ICC, 0.823), P (max) (ICC, 0.787),

216 , 0.787), P (avg) (ICC, 0.787), and F (max) (ICC, 0.659).

217 (ICC, 0.807), D (avg) (ICC, 0.823), P (max) (ICC, 0.787), P (avg) (ICC, 0.787), and F (max) (ICC, 0.6

218 four CT- and MRI-based measurement methods (ICC range, 0.97-0.99).

219 ne a subset of longitudinally stable miRNAs (ICC >0.75) that were individually discriminating of wome

220 hepatic cholangiocarcinoma (ICC), we modeled ICC desmoplasia and progression in vitro.

221 perioperative outcomes for large multifocal ICCs or tumors with contiguous organ involvement.

222 Populations of enteric neurons, ICC, and platelet-derived growth factor receptor alpha(+

223 nt variation between observers was observed (ICC = 0.89, p < 0.001).

224 in the colon are similar to the behaviour of ICC located in the deep muscular plexus region of the sm

225 All classes of ICC (including ICC-IM) express Ca(2+) -activated Cl(-) c

226 tional similarities between these classes of ICC.

227 tion and built a TME-based classification of ICC tumors that detects potentially targetable ICC subty

228 tion and built a TME-based classification of ICC tumors, which detects potentially targetable ICC sub

229 account in the search for classifications of ICC adapted to clinical trial design.

230 account in the search for classifications of ICC adapted to clinical trial design.

231 on of Ano1 channels causes depolarization of ICC-SS and LSMC, leading to activation of L-type Ca(2+)

232 d Ca(2+) signalling in a novel population of ICC that lies along the subserosal surface of LM (ICC-SS

233 rom subserosal ICC (ICC-SS), a population of ICC with previously unknown function.

234 was associated with a 40% increased risk of ICC (OR = 1.40; 95% CI = 1.05-1.89), but not HCC (OR = 1

235 was associated with a 40% increased risk of ICC (OR=1.40,95%CI=1.05-1.89), but not HCC (OR=1.12,95%C

236 desmoplastic tumor microenvironment (TME) of ICC has been stressed but was insufficiently taken into

237 desmoplastic tumor microenvironment (TME) of ICC has been stressed, but was insufficiently taken into

238 d differences from the intramuscular type of ICC in the small intestine.

239 ent on Cav(L) or ANO1; properties typical of ICC-IM mediating neural responses in other gastrointesti

240 About 45% of ICCs displayed an immune desert phenotype.

241 To understand the fate of ICCs in hyperinsulinemic, hyperglycemic states character

242 (1) receptor agonists had minimal effects on ICC-IM, whereas neostigmine and carbachol increased Ca(2

243 pear to be functionally expressed by SMCs or ICC.

244 For all studied parameters, ICC was >0.97 with the least repeatable measurements obt

245 (ICC) = 0.905, P < 0.001] and their parents (ICC = 0.856, P < 0.001).

246 At baseline, the ABSIS and PDAI ICCs were 0.90 (95% confidence interval [CI] = 0.85-0.93

247 higher in moderate and extensive pemphigus (ICC = 0.82, 95% CI = 0.63-0.92 and ICC = 0.80, 95% CI =

248 l responses in patients carrying FF-positive ICC.

249 The dorsal raphe ICC value was sensitive to a measurement outlier.

250 Individual reader ICCs for hepatic PDFF measurements across all three imag

251 practice readers than for academic readers (ICC difference, 0.009; 95% CI: 0.000, 0.021).

252 BBEA had high reliability (ICC >0.75) and low variability (CV <20%) and could detec

253 ch-alpha: 0.71) and test-retest reliability (ICC: 0.78).

254 ge 45 years, brainAGE was measured reliably (ICC = 0.81) and ranged from 24 to 72 years.

255 I-based VDP demonstrated good repeatability (ICC = 0.91 and 0.95, respectively; P <= .001).

256 ellent inter- and intrareader repeatability (ICC, 0.84-0.99) for all variables assessed.

257 The reproducibility ICC ranged from 0.96 to 0.98 for all 6 GCC parameters.

258 FMBV calculations had high reproducibility (ICC = 0.97; 95% confidence interval: 0.96, 0.98).

259 tion thresholds were the least reproducible (ICC: ~0.3).

260 imes were shown to be the most reproducible (ICC: ~0.9) followed by detection thresholds (ICC: ~0.7).

261 -1.2 degrees and 0.1 degrees , respectively; ICC: 0.79 and 0.82, respectively).

262 ma cell strain (TDECC) derived from the same ICC type as TDFSM.

263 ion was stronger for allied health services (ICC = 18 to 26%), medical imaging (ICC = 4 to 10%), and

264 rrespond to excellent clinical significance (ICC > 0.9), are not related to changes in stomach volume

265 D) in colonic tunica muscularis and in SMCs, ICC and PDGFRalpha(+) cells with HPLC-FLD, we report tha

266 citability is tuned by input from subserosal ICC (ICC-SS), a population of ICC with previously unknow

267 tal regions (K(i)(cer) ICC: 0.68-0.94, SUVRc ICC: 0.76-0.91).

268 tumors, which detects potentially targetable ICC subtypes.

269 C tumors that detects potentially targetable ICC subtypes.

270 Long-term ICCs ranged from 0.29 (night) to 0.51 (late).

271 Our data suggest that ICC-SS contribute to regulation of LM motor activity.

272 The ICC analysis identified significant reliability between

273 The ICC estimates obtained for PSMA-TV, PSMA-TL, SUV(mean),

274 The ICC for interoperator reproducibility was 0.890, and for

275 The ICC for intersoftware repeatability was 1.

276 The ICC measure for DM-TAD was significantly lower than DM-M

277 The ICC values ranged from 0.6 to 0.7 for measuring the HbO

278 The ICC values ranged from 0.98 (En-DMT) to 0.99 (TCT) with

279 l-autonomous expression of mutant KIT in the ICC lineage leads to GIST.

280 The ICCs for PDAI were higher in moderate and extensive pemp

281 78, 0.92) and 0.89 (95% CI: 0.77, 0.94); the ICCs of the radiologists were 0.93 (95% CI: 0.90, 0.95)

282 Conversely, the ICCs for ABSIS were lower in patients with moderate exte

283 les each collected over 1 wk to >/=1 mo, the ICCs ranged from 0.33 to 0.68 for sodium at various inte

284 and external test images, respectively, the ICCs of AI and radiologists were 0.84 (95% CI: 0.78, 0.9

285 During patients' follow-up, the ICCs of both ABSIS and PDAI scores remained higher than

286 ICC: ~0.9) followed by detection thresholds (ICC: ~0.7).

287 when measured relative to circadian timing (ICC = 0.33-0.41).

288 PV persistence, dysplasia and progression to ICC.

289 g was stable across months (~ 1-h variation, ICCs ranging from 0.54-0.63), but less stable across mon

290 ability within individuals (~ 3-h variation; ICC = 0.12-0.34).

291 C], 0.97), SVC VLD (ICC, 1.00), and DVC VLD (ICC, 1.00).

292 ntraclass correlation [ICC], 0.97), SVC VLD (ICC, 1.00), and DVC VLD (ICC, 1.00).

293 eviewers regarding MRI-derived tumor volume (ICC, 0.979).

294 etric bra was valid during rest and walking (ICC = 0.96, MAPE = 1.9%, LoA = -15 to 12).

295 We enrolled women with ICC, high- and low-grade squamous intraepithelial lesion

296 arge proportion of WHIV, and almost all with ICCs, were from Africa.

297 ibility and repeatability, and features with ICCs greater than or equal to 0.8 were considered reprod

298 Technical reproducibility was high, with ICCs for duplicate fecal samples between 0.64 and 1.00.

299 readers are repeatable and reproducible with ICCs that correspond to excellent clinical significance

300 esults At baseline, the DL algorithm yielded ICCs of 0.79-0.97 for CAC and 0.66-0.98 for TAC across t