ライフサイエンスコーパス: IDA

1 IDA is most prevalent among preschool children and women

2 IDA mothers received iron supplements of 125 mg FeSO(4)

3 IDA patients can be treated with iron supplementation, y

4 IDA prevalence was 5.0% +/- 0.4% and 2.6% +/- 0.7% in no

5 IDA random assignment was associated with higher CR rate

6 IDA was <5% in most studies in Northern and Western Euro

7 IDA was equivalent to IA at 24 months (61% [45-77%] vs 5

8 IDA was equivalent to IA at 24 months (61%, CI 45-77 ver

9 IDA was superior to IA for inactivating adult worms at a

10 IDA was superior to IA for inactivating adult worms at a

11 IDA, anthracycline derivative anticancer drug, was adsor

12 IDA-Cu complex acted as an efficient recognition element

13 IDAs can facilitate the detection of a range of biologic

14 received iron supplements of 125 mg FeSO(4) (IDA-Fe; n = 34) or placebo (IDA-PL; n = 30) daily from 1

15 acid (IDA) regenerable sorbents by forming a IDA:Cu(II):Con A sandwich affinity structure that has hi

16 ) and INFLORESCENCE DEFICIENT IN ABSCISSION (IDA) is induced in cauline leaf abscission zones when th

17 tion, INFLORESCENCE DEFICIENT IN ABSCISSION (IDA), suggesting that signalling through IDA and its pro

18 ns in Inflorescence Deficient in Abscission (IDA), which encodes a predicted secreted small protein,

19 ptide INFLORESCENCE DEFICIENT IN ABSCISSION (IDA), which signals through the leucine-rich repeat rece

20 s the INFLORESCENCE DEFICIENT IN ABSCISSION (IDA)-derived peptide and its receptors, HAESA (HAE) and

21 ilotriacetic acid (NTA), iminodiacetic acid (IDA) and desferrioxamine B (DFOB), as well as with Suwan

22 a resin-bound 1:1 Ni(II)-iminodiacetic acid (IDA) complex.

23 otriacetic acid (NTA) or iminodiacetic acid (IDA) in-house-fabricated columns.

24 ilized on Cu(II)-charged iminodiacetic acid (IDA) regenerable sorbents by forming a IDA:Cu(II):Con A

25 ) (through the bond with iminodiacetic acid (IDA)), as an adsorbent for the chromatographic process t

26 ques with information-dependent acquisition (IDA) approaches more and more often.

27 Information-dependent acquisition (IDA) functionality was used to combine s-MRM with enhanc

28 vey scan, information-dependent acquisition (IDA) triggered collection of EPI spectra, and library se

29 ware tool information dependent acquisition (IDA) was used to automatically obtain information about

30 Information Dependent Acquisition (IDA), an extra confirmation tool for samples that contai

31 including information-dependent acquisition (IDA), SWATH (sequential window acquisition of all theore

32 their efficacy from independent drug action (IDA), where patients only receive benefit from the singl

33 was no excess of severe or serious AEs after IDA in this subgroup.

34 of AEs was not significantly different after IDA or DA.

35 common in microfilaremic participants after IDA than after DA (39.4% versus 25.6%, P < 0.001).

36 severity did not differ significantly after IDA or DA treatment.

37 The frequency of all AEs was similar after IDA and DA treatment (12% versus 12.1%, adjusted odds ra

38 tin with diethylcarbamazine and albendazole [IDA]) is superior to a two-drug combination (diethylcarb

39 in plus diethylcarbamazine plus albendazole [IDA] is noninferior to standard 3 annual doses of iverme

40 in plus diethylcarbamazine plus albendazole, IDA) is non-inferior to three annual doses of ivermectin

41 da) and blade-on-petiole1 (bop1)/bop2 and an IDA-overexpressing line.

42 strategies that have been used to develop an IDA, including the design strategies for the newly repor

43 Here we present IDACombo, an IDA based method to predict the efficacy of drug combina

44 secondary to severe iron-deficiency anaemia (IDA) attributable to menstrual losses and limited iron i

45 Iron deficiency anaemia (IDA) is a common nutritional disorder worldwide.

46 In multivariate analysis, IDA random assignment remained associated with a higher

47 A central hydroxyproline residue anchors IDA to the receptor.

48 n deficiency (adjusted OR 8.1; P = .001) and IDA (adjusted OR 5.1; P = .01) at the end of the malaria

49 dence interval [CI], 0.6, 0.8; P < .001) and IDA (IRR, 0.7; 95% CI, 0.6, 0.9; P = .006).

50 on the association between achlorhydria and IDA.

51 in the association between achlorhydria and IDA.

52 th AML, younger age, favorable-risk AML, and IDA treatment predict a better long-term outcome.

53 frequency of AEs across study arms (AZI and IDA alone arms 9/12 (75%), co-administration arm 12/13 (

54 ngs in kindled rodents to show that CLON and IDA can have opposing effects on kindling development in

55 At 9 mo, the control and IDA-Fe groups no longer differed.

56 Rates of serious AEs after DA and IDA treatment were 0.04% (95% CI 0.01%-0.1%) and 0.01% (

57 to be a risk factor for iron deficiency and IDA in children in a malaria-endemic environment and thi

58 s displayed increasing prevalences of ID and IDA during pregnancy, which peaked in the middle to late

59 I and produce prevalence estimates of ID and IDA for each target population.

60 sychological outcomes associated with ID and IDA in premenopausal women, as the prevalence of ID and

61 enopausal women, as the prevalence of ID and IDA is often greater in premenopausal women than other p

62 ive transfusion rate was observed for ID and IDA patients only if iron was supplemented >7 days befor

63 ally representative data suggest that ID and IDA remain a concern in the United States.

64 s were 6-21 mug/L, and prevalences of ID and IDA were 28-85% and 21-35%, respectively.

65 Various definitions for ID and IDA were applied across studies.

66 tion and excess stores, as well as on ID and IDA, is needed.

67 iron status and lower prevalences of ID and IDA, which were dependent on the dose of iron and compli

68 of 2500-2999 g are at higher risk of ID and IDA.

69 th significant reductions in AI, but IDA and IDA+AI remain unresolved.

70 n-based interventions are needed for IDA and IDA+AI, and monitoring of iron biomarkers reveals a wind

71 nd it interacts with the cytoplasmic ODA and IDA assembly factor DNAAF2 (KTU).

72 scence staining showed an absence of ODA and IDA proteins in cilia from an affected individual, thus

73 (2) CLON retarded and IDA accelerated kindling rate, defined as the number of

74 ons of outer and inner dynein arms (ODAs and IDAs, respectively).

75 Iron-deficiency anemia (IDA) affects millions of persons worldwide, and is assoc

76 semia trait (TT) and iron deficiency anemia (IDA) are the two most common anemia types and can have a

77 ards associated with iron deficiency anemia (IDA) are well known, concerns about risks associated wit

78 Iron deficiency anemia (IDA) has detrimental effects on individuals and societie

79 tablish the cause of iron deficiency anemia (IDA) in a substantial proportion of patients.

80 supplementation for iron deficiency anemia (IDA) in asymptomatic, nonanemic pregnant women could imp

81 pylori infection and iron deficiency anemia (IDA) in humans.

82 hydria is a cause of iron deficiency anemia (IDA) in humans.

83 for the diagnosis of iron-deficiency anemia (IDA) in patients with CKD have limitations, leading to p

84 The prevalence of iron deficiency anemia (IDA) increased over the malaria season (P < .001).

85 Sub-Saharan Africa, iron-deficiency anemia (IDA) is common, and many complementary foods are low in

86 Iron deficiency anemia (IDA) is relatively common in the third trimester of preg

87 Preoperative iron deficiency anemia (IDA) occurs frequently; however if left untreated, incre

88 Iron deficiency anemia (IDA) still remains universally problematic worldwide.

89 deficiency (ID) and iron deficiency anemia (IDA) was 10-32% and 2-5%, respectively, depending on the

90 orldwide suffer from iron deficiency anemia (IDA), a state of systemic iron insufficiency that limits

91 concentrations were iron deficiency anemia (IDA), anemia of inflammation (AI), and multifactorial an

92 5% (n = 5) developed iron deficiency anemia (IDA).

93 urden is a result of iron deficiency anemia (IDA).

94 hed iron stores) and iron deficiency anemia (IDA; poor iron stores and diminished hemoglobin), affect

95 ID anemia (IDA) affected 23.6% of Kenyan and 17.6% of Ugandan child

96 lence of iron deficiency (ID) and ID anemia (IDA) among toddlers, nonpregnant females, and pregnant f

97 icits despite prompt diagnosis of ID anemia (IDA) by screening of hemoglobin concentrations followed

98 orld, with consequences of ID and ID anemia (IDA) in young children including behavioral and cognitiv

99 nflammation (AI), and multifactorial anemia (IDA+AI).

100 t concentration, and the inter-duplex angle (IDA) between arms.

101 (needle insertion only) in the same animals; IDA significantly reduced thresholds.

102 ng in Xenopus to show that inner dynein arm (IDA) and outer dynein arm (ODA) subunits are partitioned

103 lone; n = 54) and ODA plus inner dynein arm (IDA) defects (ODA + IDA; n = 18) versus subjects with ID

104 aused by dual loss of the inner dynein arms (IDAs) and outer dynein arms (ODAs), which power cilia an

105 e assembly of a subset of inner dynein arms (IDAs) to a specific location in the 96 nm repeat.

106 electrode E1 of an Au interdigitated array (IDA) electrode device.

107 ionization MS with an interdigitated array (IDA) electrode enabling chemical analysis of electrolyze

108 Carbon interdigitated array (IDA) electrodes with features sizes down to 1.2 mum were

109 latform is based on an interdigitated array (IDA) of electrodes.

110 across a 5 microm gap interdigitated array (IDA) of gold electrodes behaved as "hydrogen sensors".

111 monolayer (SAM)-coated interdigitated-array (IDAs) gold-platinum electrodes.

112 Interdigitated gold microelectrode arrays (IDAs) were first modified with a self-assembled monolaye

113 ne gastritis implies a disease presenting as IDA many years before the establishment of clinical coba

114 es a metal-ion indicator displacement assay (IDA) has been devised.

115 Indicator displacement assays (IDAs) offer a unique and innovative approach to molecula

116 oped by using indicator displacement assays (IDAs), and the protocol was then applied to the vicinal

117 ssible role of diet, in H. pylori-associated IDA.

118 , Joint Indicator A, to discriminate between IDA, TT, and healthy children in a Chinese population.

119 ion in managing IDA, and distinguish between IDA from the anemia of chronic disease.

120 iated with significant reductions in AI, but IDA and IDA+AI remain unresolved.

121 sm of phosphorescence of Mn-doped ZnS QDs by IDA is a combined dynamic and static quenching.

122 indicate that H. pylori infection can cause IDA and that the composition of the diet influences the

123 ls, P-element-mediated gene silencing caused IDA and ODA defects, proprioception deficits, and steril

124 CD-affected families afflicted with combined IDA and ODA defects, we found that 6/38 (16%) carried bi

125 orted iminodiacetic acid-copper ion complex (IDA-Cu) immobilized onto gold nanoparticles (GNPs)-modif

126 for anemia with iron-responsive components (IDA, IDA+AI).

127 local and global context in conceptualizing IDA&E to best achieve these aims.

128 Strategies to control IDA include daily and intermittent iron supplementation,

129 public health interventions for controlling IDA in low- and middle-income countries, with an emphasi

130 s revising global guidelines for controlling IDA.

131 d cheese whey was passed through the cryogel-IDA-Cu(2+) system.

132 DDA/IDA methods do not exploit valuable information that is

133 a- or information-dependent acquisition (DDA/IDA), where fragmentation mass candidates are selected b

134 the mean hemoglobin level, iron-deficiency (IDA) and non-iron-deficiency anemia (NIDA), and inflamma

135 ivermectin, albendazole, diethylcarbamazine (IDA) regime for Lymphatic Filariasis.

136 Even using several different IDA settings, it was observed that IDA failed to trigger

137 s defects, and microtubular disorganization (IDA/CA/MTD) (n = 41) were significantly younger at diagn

138 s defects with microtubular disorganization (IDA/CA/MTD; n = 40).

139 ddition of ds-DNA caused formation of ds-DNA/IDA complex and recovered the RTP signal of Mn-doped ZnS

140 ults in reduced assembly of inner-arm dynein IDA b and the beak-MIP structures.

141 A) Inclusion, Diversity, Access, and Equity (IDA&E) initiative and discusses the long-term goals and

142 of inclusion, diversity, access, and equity (IDA&E) is critical to harnessing the full range of human

143 ds (IDA, MMDF (multiple mass defect filters)-IDA, SWATH, or MS(All)) using an ultrahigh-performance l

144 eceived high DNR doses (cure rate, 27.4% for IDA v 15.9% for DNR; P = .049).

145 with serum ferritin higher than expected for IDA, mutations involving iron trafficking and regulation

146 panded and improved treatment modalities for IDA.

147 Iron-based interventions are needed for IDA and IDA+AI, and monitoring of iron biomarkers reveal

148 t (12% versus 12.1%, adjusted odds ratio for IDA versus DA 1.15, 95% CI 0.87-1.52, P = 0.316); 10.9%

149 MYND10 is a cytoplasmic protein required for IDA and ODA assembly and that its variants cause ciliary

150 current screening and treatment strategy for IDA fails to achieve this goal for 3 reasons.

151 ce that routine prenatal supplementation for IDA improves maternal or infant clinical health outcomes

152 k Force on screening and supplementation for IDA in pregnancy.

153 hysiology and available diagnostic tests for IDA in CKD, we discuss the literature that has informed

154 s of DRM detected in microsomal samples from IDA, SWATH, and MS(All) methods, respectively.

155 eastfed infants with a birth weight >2500 g, IDA is uncommon before 6 mo, but male infants and those

156 es of isotope dilution for data analysis (GS-IDA).

157 t delivery, and 18% of women (n = 11/61) had IDA.

158 inase (LRR-RK) HAESA and the peptide hormone IDA.

159 cipants were recruited and randomized to (I) IDA alone, (II) IDA combined with AZI, (III) AZI alone.

160 in Europe, we describe the prevalence of ID, IDA, iron repletion, and excess stores with the use of p

161 anemia with iron-responsive components (IDA, IDA+AI).

162 apped Mn doped ZnS quantum dots/ Idarubicin (IDA) nanohybrids were used as novel room temperature pho

163 rubicin (DOX), Epirubicin (EPI), Idarubicin (IDA) and Daunorubicin (DAU) were examined.

164 alf received the alpha2 antagonist idazoxan (IDA; 0.33 nmol).

165 Here, we identified IDA-like 6 (IDL6), a gene that is prominently expressed

166 or identifying ID and of 97% for identifying IDA.

167 rhydria) was 44% in patients with idiopathic IDA and 1.8% in healthy controls.

168 ruited and randomized to (I) IDA alone, (II) IDA combined with AZI, (III) AZI alone.

169 This hemi-saturated Ni(II)-IDA system containing exchangeable sites at the metal io

170 s was demonstrated for Pb(II)-PSS and Pb(II)-IDA systems.

171 lfonamide prong and a cupric iminodiacetate (IDA-Cu(2+)) prong separated by linkers of different leng

172 GNPs acts as a substrate to immobilize IDA-Cu coordinative complex and its interaction with Lys

173 in a statistically significant difference in IDA incidence at term, favoring supplementation (risk ra

174 ormone by binding to an Arg-His-Asn motif in IDA.

175 was to determine the effects of diet-induced IDA on intrauterine growth and neonatal iron status in a

176 incidence and severity of H. pylori-induced IDA.

177 uld be used in future studies to investigate IDA progression and cell-specific responses to hypoxic a

178 The genes that encode HAE/HAESA-LIKE2, IDA, NEVERSHED, and MAPK KINASE4 and 5 are all necessary

179 ctice guidelines for diagnosing and managing IDA should include the design of an algorithm that is in

180 cs, which will provide direction in managing IDA, and distinguish between IDA from the anemia of chro

181 controlled indicator displacement assays (MC-IDAs), and quencher displacement assays (QDAs).

182 t compounds were analyzed with four methods (IDA, MMDF (multiple mass defect filters)-IDA, SWATH, or

183 ered for MS(2) acquisition with IDA and MMDF-IDA methods, respectively.

184 At 6 and 12 months IDA was superior to IA in clearing Mf (89% [77-99%] and

185 At 6 and 12 months IDA was superior to IA in clearing Mf (89%, CI 77-99, an

186 tended structure that interconnects multiple IDAs and regulatory complexes.

187 enges associated with the development of new IDAs and suggest potential future avenues of research.

188 hildren aged 9 to 48 months with nutritional IDA (assessed by history and laboratory criteria) that w

189 ion in infants and children with nutritional IDA.

190 A plus inner dynein arm (IDA) defects (ODA + IDA; n = 18) versus subjects with IDA and central appara

191 with ciliary outer and inner dynein arm (ODA+IDA) defects.

192 utations in SPAG1 cause PCD with ciliary ODA+IDA defects and that exome sequencing is useful to ident

193 o had defined ciliary ultrastructure had ODA+IDA defects.

194 unrelated affected individuals (62 with ODA+IDA defects, 35 with ODA defects, 1 without available ci

195 esults suggest that the sequential action of IDA, HAE and HSL2, and a MAP kinase cascade regulates th

196 transferrin receptor (sTfR), a biomarker of IDA, on a photonic crystal (PC) biosensor was establishe

197 achlorhydria can be an independent cause of IDA.

198 ion and, after excluding all other causes of IDA, 64% to 75% of such patients are permanently cured b

199 CRVO is a rare complication of IDA.With rising calls for sustainable diets and rising e

200 med achlorhydria preceded the development of IDA in time.

201 on, which persisted after the development of IDA.

202 ants were treated with a single oral dose of IDA (ivermectin, 200 mug/kg; diethylcarbamazine, 6 mg/kg

203 were randomized to receive a single dose of IDA (n = 43) or 3 annual doses of IA (n = 52) in an open

204 andomized to receive either a single dose of IDA (N=43) or 3 annual doses of IA (N=52) in an open-lab

205 A single dose of IDA was superior to 2 doses of IA in reducing the overal

206 A single dose of IDA was superior to two doses IA in reducing the overall

207 It is unknown how expression of IDA in the abscission zone leads to HAESA activation.

208 Helicobacter pylori, and hereditary forms of IDA is recommended.

209 The long-term impact of IDA was still observed in patients younger than age 65 y

210 Second, brain ID, independently of IDA, is responsible for long-term neurological deficits.

211 Management of IDA is based on supplementation combined with effective

212 approach for the diagnosis and management of IDA.

213 characterized a novel genetic mouse model of IDA.

214 c health concern, very few genetic models of IDA are available to study its progression.

215 hallenges in the detection and monitoring of IDA in these patients.

216 , starting iron treatment after the onset of IDA is less effective than prevention.

217 ired MS(2) spectra decreased in the order of IDA, SWATH, and MS(All) methods.

218 The predictors of IDA (8%) were male sex (AOR: 7.6; 95% CI: 2.5, 23.0), bi

219 Also, the detection rate of IDA compared to SWATH was investigated in a forced coelu

220 ary outcomes included complete resolution of IDA (defined as hemoglobin concentration >11 g/dL, mean

221 Proportion with a complete resolution of IDA was higher in the ferrous sulfate group (29% vs 6%;

222 study was performed to assess the safety of IDA and DA in a variety of endemic settings.

223 ent practice guidelines for the treatment of IDA in CKD, and we summarize the available oral and intr

224 (IV) iron formulations for the treatment of IDA in CKD.

225 However, the 5- to 11-year-old IDA/CA/MTD group had more lobes of bronchiectasis (media

226 nstitutional culture and climate to optimize IDA&E.

227 Overall, IDA-based methods acquired qualitatively better MS(2) sp

228 Plants overexpressing IDA in a hae hsl2 background have abscission defects, in

229 125 mg FeSO(4) (IDA-Fe; n = 34) or placebo (IDA-PL; n = 30) daily from 10 wk to 9 mo postpartum.

230 d, N-(phosphonomethyl)iminodiacetic acid (PM-IDA), in aqueous solution.

231 On charging with nickel ions, the PM-IDA functionalized iron oxide nanoparticles exhibited hi

232 Preoperative IDA management with intravenous iron is effective in imp

233 on yielded a robust phenotype of progressive IDA that develops in as little as 3 months following dis

234 der optimal conditions, RTP intensity of QDs/IDA nanohybrids increased linearly with the concentratio

235 rategies to increase iron intakes and reduce IDA at this age.

236 4% to 6% of patients with obscure refractory IDA have celiac disease, and autoimmune gastritis is enc

237 ic evaluation with unexplained or refractory IDA, screening for celiac disease, autoimmune gastritis,

238 50% of patients with unexplained refractory IDA have active H pylori infection and, after excluding

239 nd management of unexplained, or refractory, IDA.

240 rtance of each analyte and then the reported IDA system is discussed.

241 Here we show that this process requires IDA, HAE, and HSL2.

242 In the root, IDA is strongly auxin-inducible and dependent on key reg

243 s whereas currents measured for the smallest IDA electrodes, that were larger than the simulated curr

244 nded particulate reagent-iminodiacetate (SPR-IDA) as resin materials which are embedded in an ether-b

245 erroportin knockout mouse model of end-stage IDA could be used in future studies to investigate IDA p

246 We noted that, at end-stage IDA, tissue-specific transcriptional stress responses oc

247 While all iron-supplemented IDA patients required less red blood cell (RBC) transfus

248 uced in all preoperatively iron-supplemented IDA patients.

249 Limited evidence supports IDA treatment with preoperative IV iron.

250 ral function and is 3 times more common than IDA in toddlers.

251 However, we found no credible evidence that IDA caused gastritis or that IDA preceded the developmen

252 different IDA settings, it was observed that IDA failed to trigger relevant compounds.

253 In this study, we observed that IDA was well tolerated in LF-endemic populations.

254 e evidence that IDA caused gastritis or that IDA preceded the development of achlorhydria.

255 Here we show that IDA is sensed directly by the HAESA ectodomain.

256 Thus, results of this study suggest that IDA should be as safe as DA for use as a MDA regimen for

257 The IDA pioneers Inouye, Shinkai, and Anslyn inspired resear

258 The IDA-Cu(2+) cryogel system was also able to capture lacto

259 nown that the complexity of a sample and the IDA settings might prevent important compounds from bein

260 the child responsiveness scale than did the IDA-PL group (P = 0.007-0.032), whose iron status remain

261 tly greater in the control group than in the IDA groups (P = 0.028 and 0.009, respectively).

262 Median body mass index was lower in the IDA/CA/MTD group (46th percentile) versus the ODA groups

263 Median FEV1 was worse in the IDA/CA/MTD group (72% predicted) versus the combined ODA

264 decline was different from zero only in the IDA/MTD/CA group (mean [SE], -1.11 [0.48] percent predic

265 ordinates to the active site Zn(2+) ion; the IDA-Cu(2+) prong of the tightest-binding inhibitor, BR30

266 electrode spacing was developed to model the IDA electrode response.

267 t span the gap between the electrodes of the IDA.

268 The collection efficiency depends on the IDA electrode spacing, which ranged from around 2.7 to 1

269 10 muL of 1 muM solution of dopamine on the IDA.

270 Upon the recognition of the Lys to the IDA-Cu, the peak current decreased due to the hindered e

271 As an example, we used the IDA peptide HAE/HSL2 receptor signaling system known to

272 er with a following chemical reaction at the IDAs with larger gaps whereas currents measured for the

273 ction includes details on the benefit of the IDAs and perceived limitations for each system.

274 nd climates are essential to achieving these IDA&E goals.

275 The simplicity of these IDAs, coupled with low cost, high sensitivity, and abili

276 In this IDA approach, a sulfur mustard simulant (the analyte) is

277 ON (IDA), suggesting that signalling through IDA and its proposed receptors, HAESA and HAESA-LIKE2, m

278 ts, indicating HAE and HSL2 are epistatic to IDA.

279 that SWATH's detection rate was superior to IDA, which failed to trigger approximately 10% of the an

280 appropriate animal model for third-trimester IDA in women.

281 Findings were validated using IDA in intact and digested bovine serum albumin solution

282 eloping brain is already iron deficient when IDA is diagnosed owing to the prioritization of availabl

283 ented onset of spontaneous seizures, whereas IDA precipitated onset of spontaneous seizures in 100% o

284 ere not triggered for MS(2) acquisition with IDA and MMDF-IDA methods, respectively.

285 No genotypes were associated with IDA before the malaria season.

286 Reinfection was associated with IDA for high reinfection intensities only.

287 Co-administration of AZI with IDA did not show evidence of significant drug-interactio

288 Crystal structures of HAESA in complex with IDA reveal a hormone binding pocket that accommodates an

289 than in uninfected gerbils, consistent with IDA.

290 o the lack of sustained clearance of Mf with IDA.

291 associated with NIDA (P=0.019) but not with IDA (P=0.29).

292 Participants with IDA/MTD/CA defects, which included individuals with CCDC

293 ion decline over time were participants with IDA/MTD/CA defects.

294 ust 2011 and November 2014, 72 patients with IDA were assigned to receive either IV iron or usual car

295 The percentages with IDA were 2% in Sweden, 4% in Mexico, 5-11% in Honduras,

296 At 36 months posttreatment with IDA, 18/33 (55%; 95% CI, 38-72%) cleared Mf versus 33/42

297 cts (ODA + IDA; n = 18) versus subjects with IDA and central apparatus defects with microtubular diso

298 and genotype groups, but worse in those with IDA/CA/MTD ultrastructural defects, most of whom had bia

299 e-risk AML; P < .001) and those treated with IDA (16.6% v 9.8% with DNR; P = .018).

300 reated with DA, and 14,556 were treated with IDA.

301 At 36 months post-treatment with IDA, 18/33 (55%, 95%CI 38-72) cleared Mf versus 33/42 (7