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1 analyzed for leptin, adiponectin, IGF1, and IGF binding protein 3.
2 IGF-II (Ptrend=0.006 for both) but not with IGF binding protein-3.
3 Similar associations were observed for IGF binding protein-3.
4 n, insulin-like growth factor-I (IGF-I), and IGF binding protein-3.
5 proteins insulin-like growth factor (IGF) 1, IGF binding protein-3 and acid-labile subunit, along wit
8 nation of the specificity and sensitivity of IGF-binding protein-3 as an index of nutrition or anabol
10 iously diminished muscle levels of IGF-1 and IGF binding protein 3 both increased following PRT (P <
12 on into the study and was analyzed for serum IGF-binding protein-3 concentration (by radioimmunoassay
15 y should be considered when evaluating serum IGF-binding protein-3 concentrations as a marker of nutr
16 F-1 concentrations changed coordinately with IGF-binding protein-3 concentrations in females and male
17 s of the present study were to measure serum IGF-binding protein-3 concentrations in trauma patients
18 y must be considered when interpreting serum IGF-binding protein-3 concentrations in trauma patients.
20 der were not significant predictors of serum IGF-binding protein-3 concentrations when all patients w
21 bust increase of insulin-like growth factor (IGF) binding protein 3 expression in AR-deficient stroma
22 ng IGF1 secretion through the suppression of IGF-binding protein 3 expression in prostatic stromal ce
24 evels of insulin-like growth factor (IGF)-1, IGF binding protein-3, growth hormone (GH), and somatost
26 ternary complex formed by the association of IGF binding protein 3-IGF complexes with a serum protein
30 150-kDa complex including the IGF molecule, IGF binding protein 3 (IGFBP-3), and the acid labile sub
31 erum IGF-I, IGF binding protein 1 (IGFBP-1), IGF binding protein 3 (IGFBP-3), and the IGF-I:IGFBP-3 m
32 F-I) concentrations, frequently adjusted for IGF binding protein 3 (IGFBP-3), have been associated wi
33 to direct expression of rat IGF-I and human IGF binding protein-3 (IGFBP-3) to mammary tissue during
34 thors assessed the associations of IGF-1 and IGF binding protein-3 (IGFBP-3) with cardiovascular dise
35 However, des(1-3)IGF-I, which weakly binds IGF binding protein-3 (IGFBP-3), induced IGF-IR phosphor
36 S of measured serum protein levels of IGF-I, IGF binding protein-3 (IGFBP-3), pregnancy-associated pl
40 ix et al. demonstrate that downregulation of IGF-binding protein 3 (IGFBP-3) and -4, the negative reg
42 pproximately 13-fold (P < 0.001) increase in IGF-binding protein 3 (IGFBP-3) protein levels were also
43 like growth factor (IGF-I) content and IGF-I:IGF-binding protein 3 (IGFBP-3) ratio as risk factors fo
44 complexes with acid-labile subunit (ALS) and IGF-binding protein 3 (IGFBP-3) to maintain its circulat
54 n assessment using Western blot analysis for IGF binding protein 3 (IGFBP3), IGF-1 receptor (IGF-1R),
55 ed and validated insulin-like growth factor (IGF)-binding protein-3 (IGFBP3) as a novel miR-21 target
56 een insulin-like growth factor 1 (IGF-1) and IGF-binding protein 3 (IGFBP3) so that the free form of
57 apamil decreases the expression of beta-cell IGF-binding protein 3 (IGFBP3), whereas IGFBP3 is increa
58 at verapamil reduces beta-cell expression of IGF-binding protein 3 (IGFBP3), whereas IGFBP3 was incre
60 1 signaling by ablating IGF-1 and increasing IGF-binding protein 3, increased vSMC apoptosis, and dec
61 rmone, insulin-like growth factor-I (IGF-1), IGF binding protein-3, insulin, cortisol, parathyroid ho
63 ding protein-2 levels, a slight reduction in IGF binding protein-3 levels, and an increase in levels
64 1% decrease at 1 and 2 wk, respectively) and IGF binding protein-3 levels, and increased IGF binding
65 F-I) and the concentration ratio of IGF-I to IGF binding protein 3 were lower in the low-protein, low
66 cal illness, but there is little research on IGF-binding protein-3, which regulates the bioactivity o