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1                                              IRV delivery efficiency (3.2+/-1%) trended lower than IM
2                                              IRV significantly increased central venous pressure meas
3                                              IRV significantly increased SVCP and FIVP (p < .002).
4 ith 26+/-3% (IM), 47+/-1% (IC), and 43+/-3% (IRV) of cells found localized in the lungs.
5 o more acidic endosomes, but does not affect IRV targeting, stability, and insulin responsiveness of
6  the delivery of PBMNCs compared with IC and IRV techniques.
7 andomization to normal ratio ventilation and IRV.
8 died targeting of sortilin, one of the major IRV constituents.
9 d not significantly alter the time course of IRV (P=.79).
10                      The annual incidence of IRV was 83/100 person-years.
11  the existence of a short-lived inhibitor of IRV translocation thus supporting the "static" model.
12 ins play an important role in the process of IRV biogenesis.
13 e, and reader variability in tumor volume on IRV was studied by using intraclass correlation coeffici
14 NCs were delivered by IC (n=5), IM (n=6), or IRV (n=5) injection.
15 uscle cells, insulin-responsive vesicles, or IRVs, deliver glucose transporter Glut4 and several asso
16       In addition, although initiation of PC-IRV did lead to a lower peak airway pressure measured pr
17                             Initiation of PC-IRV does not result in a decrease in peak airway pressur
18                                  Symptomatic IRV developed in 19 (63%) of 30 patients and in 26 (59%)
19                      These data suggest that IRV develops in a significant number of HAART-responders
20 , however, that the cytoplasmic tails of the IRV component proteins carry targeting information to th
21 on, this reporter protein does not enter the IRVs; moreover, it loses its perinuclear localization an
22 owever, whether the presence of Glut4 in the IRVs is essential for their ability to respond to insuli
23     Thus, the intracellular retention of the IRVs in adipocytes requires continuous RNA and protein b
24      Although the protein composition of the IRVs is well studied, the mechanism of their formation i
25  it specifically increases exocytosis of the IRVs.
26  differentiated cells and is targeted to the IRVs upon cell differentiation.
27 lin increased targeting of myc7-Glut4 to the IRVs, and its insulin responsiveness rose to the maximal
28 ed tumor volume was significantly related to IRV for all parameters.
29 , the intra-individual response variability (IRV) index was calculated.
30 ctors contributing to interreader variation (IRV) in parameters measured at dynamic contrast material
31 and interstitial retrograde coronary venous (IRV) delivery in an ischemic swine model.
32 nce of normal and inverse ratio ventilation (IRV).
33  protein of the insulin-responsive vesicles (IRVs).
34 omponent of the insulin-responsive vesicles (IRVs).
35      Under basal conditions, these vesicles (IRVs for insulin-responsive vesicles) are retained insid
36 ammatory syndrome (immune recovery vitritis, IRV), which causes vision loss in AIDS patients with cyt
37                 Patients were diagnosed with IRV if they developed symptomatic vitritis of >/=1+ seve