ライフサイエンスコーパス: ISO

1 ISO also produced spontaneous diastolic Ca(2+) elevation

2 ISO application led to significant RyR phosphorylation a

3 ISO caused reduction in cardiac function with evidence o

4 ISO represents novel pharmacotherapy for AILI.

5 ISO successfully mimicked sympathetic stimulation as sho

6 ISO treatment exaggerated ROS generation, p38 MAPK activ

7 ISO+I(TO) decrease-induced CADs were abolished by beta(1

8 ISO, FSK, or DB rapidly increased oxidative and glycolyt

9 ISO-1 and MIF knockout (MIFKO) had greater accumulation

10 ISO-1, like anti-MIF, inhibited the interaction between

11 ISO-induced CADs were significantly suppressed by dynami

12 ISO-induced HDAC5 nuclear export occurred through a beta

13 ISO-induced left ventricular dysfunction and hypertrophi

14 as performed in accordance to the ISO-17025 (ISO/IEC, 2005) and Eurachem guidelines.

15 ion for Standardization (ISO) (ISO/TS 17728, ISO 18593:2004 and ISO 17604:2003) standards.

16 With JNJ-303, ISO decreased APD significantly more in the epicardium a

17 llowing intravenous infusion of either 0.9% (ISO) or 3.0% (HYPER) NaCl saline, 12 subjects were passi

18 cific inhibitor of MIF-tautomerase activity, ISO-1, was investigated in PBMCs.

19 r, nuclear accumulation of HDAC5 under acute ISO/PKA signaling is dependent on phosphorylation of Ser

20 arcolemmal Ca(V) 1.2 channel abundance after ISO application.

21 fibrosis were also not altered 28 days after ISO injection.

22 r, myocyte apoptosis was not increased after ISO injury.

23 Circulating troponin I levels after ISO were elevated, further documenting myocyte damage.

24 An ISO 6134-compliant generalized least-squares regression

25 housed in a laminar flow cabinet allowing an ISO class-5 environment.

26 Interestingly, Ces3 inhibition attenuated an ISO-induced thermogenic program in adipocytes by downreg

27 with respect to isotopic data, we propose an ISO-FOOD ontology as a domain ontology for describing is

28 53 to 27% for the constant alpha (0.019) and ISO-7888 methods, respectively.

29 tion (ISO) (ISO/TS 17728, ISO 18593:2004 and ISO 17604:2003) standards.

30 n regard to Council Directive 333/2007EC and ISO/IEC 17025 (2005).

31 particulate-controlled cleanrooms (ISO 5 and ISO 8) and two vicinal uncontrolled areas (office, chang

32 AILI activated the CWP, and ISO enhanced Wnt-ligand production.

33 re validated according to CD 2002/657/EC and ISO 17025 guidelines.

34 on: NoiseMaker is written in C#, an ECMA and ISO standard language with compilers for multiple operat

35 Although ET1 and ISO shared a similar ability to induce HDAC5 nuclear exp

36 and 96.6% for Enterobacterales when FDA and ISO 20776-2 criteria, respectively, were followed.

37 e A. baumannii complex when both the FDA and ISO criteria were followed.

38 or Ag conjugated to control mAb (ISO*KLH and ISO*Bet v 1).

39 ODI and ISO correlated with disease duration.

40 Recombinant Wnt3a and ISO-603B-conditioned media, but not ISO alone, protected

41 ve treatments with either the MIF antagonist ISO-1 or anti-CD74 neutralizing antibodies on the aberra

42 curative treatments with the MIF antagonist ISO-1 or anti-CD74 neutralizing antibodies partially rev

43 rmore, allotransplantation of HPCs from APAP+ISO-treated mice to other APAP-injured mice improved AIL

44 he standard Griess spectrophotometric assay (ISO 2918/1975), proving the suitability of using immobil

45 that of oil-exposed fish (1 mW g(-1) Mv) at ISO concentrations of 1 x 10(-6) M.

46 ections reveal that canagliflozin attenuates ISO-mediated increases in inflammatory cell infiltration

47 serve at the same time as fluorescence-based ISOs, and apply it specifically to potassium (K(+)).

48 rated that there was no relationship between ISO quality categories and safranal content using HPLC-D

49 he heart with the inhibitory peptide blocked ISO-mediated BVR activation and augmented apoptosis; con

50 tric acid extraction (0.43 M) was adopted by ISO (ISO-17586:2016) as standard for extraction of geoch

51 Slower activation of I(Ks) than of I(Ca) by ISO may explain why different sympathetic modes, i.e. sy

52 Hepatoprotection by ISO is mediated by para- and autocrine effects of Wnt si

53 ILI raised HPC numbers, further increased by ISO, with attenuation of liver injury.

54 asma free fatty acid (FFA) levels induced by ISO competed with glucose ([(18)F]FDG) uptake in BAT loc

55 amines had low HPC numbers, reconstituted by ISO.

56 some transport pathways and reversibility by ISO, thus accounting for lysosomal Cl(-) deficits that c

57 ealer properties were tested as specified by ISO 6876 (2012) and also in alternative media.

58 nonselective beta-adrenergic stimulation by ISO at concentrations that increase energy expenditure t

59 es in two particulate-controlled cleanrooms (ISO 5 and ISO 8) and two vicinal uncontrolled areas (off

60 free water fraction (isotropic compartment (ISO)) were derived.

61 In contrast, continuous ISO perfusion diminished APD prolongation and reduced th

62 In contrast, ISO reduced HDAC5 phosphorylation in the presence or abs

63 dly with age, beginning in the fourth decade ISO 7029 (2000).

64 Pretreatment with ISO or IL-13 decreased ISO-induced cAMP generation compared with ISO for 10 min

65 d administration of NAC did not, but delayed ISO did, reverse AILI.

66 Single- or multiple-dose ISO injury was not associated with an increase in the pe

67 ed a high-risk window of EADs in LQT2 during ISO perfusion owing to mismatch in the time courses of I

68 r, developed at different time points during ISO application.While SR Ca2+ release and load reached a

69 Scavenging ROS during ISO application decreased the occurrence of Ca2+ waves a

70 llowed by a gradual decline over time during ISO application.

71 testing laboratory that complies with BS EN ISO 139:2005+A1:2011.

72 thod was accredited in compliance with PN-EN ISO/IEC 17025:2005 standard and it was operated under fl

73 as a single analyte assay in view of the EN ISO 15189 accreditation procedure.

74 to the accreditation procedure under the EN ISO 15189 standard was carried out for allergen-specific

75 evels of aerosol entering the DC (equivalent ISO class), together with its DRH.

76 ihydro-5-isoxazole acetic acid methyl ester (ISO-1) inhibited huMIF but not MIF of parasitic origin.

77 The biodistribution of (18)F-ISO-1 and human dosimetry were evaluated.

78 e of a significant correlation between (18)F-ISO-1 and Ki-67 makes this agent promising for evaluatio

79 eceived an injection of 278-527 MBq of (18)F-ISO-1 and then underwent PET/CT imaging of the breasts 5

80 the first dedicated clinical trial of (18)F-ISO-1 in women with primary breast cancer.

81 udy objective was to determine whether (18)F-ISO-1 PET could provide an in vivo measure of tumor prol

82 cer, or head and neck cancer underwent (18)F-ISO-1 PET.

83 Conclusion: (18)F-ISO-1 uptake in breast cancer modestly correlates with a

84 Tumor (18)F-ISO-1 uptake was assessed semiquantitatively by maximum

85 In vivo uptake of (18)F-ISO-1 was quantitated by SUV(max) and distribution volum

86 No adverse effects of (18)F-ISO-1 were encountered.

87 (18)F-fluoroethoxy)-5-methylbenzamide ((18)F-ISO-1) to image solid tumors in lymphoma, breast cancer,

88 18)F-fluoroethox y)-5-methylbenzamide ((18)F-ISO-1), and evaluate the feasibility of imaging tumor pr

89 nd a weakening variance of its low-frequency ISO (LF-ISO) cycle.

90 Furthermore, ISO injections did not increase new myocytes in cKit(+/C

91 quantitative stable isotope sampling of GEM (ISO-GEM) at the outlet of a commercial Hg analyzer.

92 /db myocyte/heart preparations exposed to HG/ISO.

93 nctionally, SGBS adipocytes displayed higher ISO-induced basal leak respiration and overall oxygen co

94 However, ISO response was reversed by beta2-adrenergic receptor (

95 Two selected NPCEs viz Hy-ISO-VIII and Hy-ISO-G from the library of 20 newly synthesized derivativ

96 Two selected NPCEs viz Hy-ISO-VIII and Hy-ISO-G from the library of 20 newly synth

97 3 (48 hours) or isoproterenol hydrochloride (ISO; 30 minutes) pretreatment were stimulated with ISO (

98 time to the best of our knowledge, identify ISO as a major active compound for the anti-cancer activ

99 gated protective effects of canagliflozin in ISO-induced cardiac oxidative stress, and their underlyi

100 rate to the same levels as control cells in ISO.

101 ve gating behaviour of Ca(V) 1.2 channels in ISO stimulated myocytes.

102 flozin treatment improves kidney function in ISO-treated rats, suggesting that the antioxidant effect

103 treatment improves heart function marker in ISO-treated rats.

104 actions in mediating hypertrophic signals in ISO-specific context and revealed a difference in the ph

105 In contrast, canagliflozin treatment in ISO rats not only preserves endogenous antioxidants but

106 xcitation, with the consequence of increased ISO-W400 distance and diminished tunneling rate by almos

107 Overexpression of Nur77 markedly inhibited ISO-induced cardiac hypertrophy by inducing nuclear tran

108 Moreover, the MIF inhibitor ISO-1 inhibited AML-induced IL8 expression by BM-MSCs as

109 Systemic administration of the MIF inhibitor ISO-1 significantly blocked photoreceptor apoptosis, out

110 nnot be inhibited by the human MIF inhibitor ISO-1.

111 ta show the feasibility of the MIF-inhibitor ISO-1 to block pathological damage responses in retinal

112 acid extraction (0.43 M) was adopted by ISO (ISO-17586:2016) as standard for extraction of geochemica

113 onal Organization for Standardization (ISO) (ISO/TS 17728, ISO 18593:2004 and ISO 17604:2003) standar

114 ed ethers sevoflurane (SEVO) and isoflurane (ISO), using UV-Vis spectroscopy, time dependent-density

115 prototypical general anesthetic, isoflurane (ISO, 1.5% for 3 hr), at three early postnatal ages (P3,

116 Isoprenaline (ISO) prolonged APDs and triggered EADs in LQT1 myocytes

117 the nonselective beta-agonist isoprenaline (ISO) and compared this with cold-activated BAT activity.

118 ve effects of canagliflozin in isoprenaline (ISO)-induced cardiac oxidative damage-a model mimicking

119 d if canagliflozin can reverse isoprenaline (ISO)-induced renal oxidative damage in rats, a model tha

120 However, under isoprenaline (ISO), both the application of JNJ-303 and additional E-4

121 beta-AR stimulation with isoprenaline (ISO) increased Ca2+ transient amplitude, ICa-L and SRCa2

122 timulation with isoproterenol (isoprenaline; ISO).

123 Isoproterenol (ISO) increased Ca2+ transient amplitude during systole,

124 Isoproterenol (ISO) or PKA activation results in strong nuclear accumul

125 ated with the beta-AR agonist isoproterenol (ISO) (anxA4a(+/+) vs. anxA4a(-/-): 5.1 +/- 0.3 vs. 6.7 +

126 the beta-adrenoceptor agonist isoproterenol (ISO) and by varying afterload pressures.

127 ose (HG) and the beta-agonist isoproterenol (ISO).

128 The beta-adrenergic agonist isoproterenol (ISO; 1 muM) increased [Ca(2+)](SR) well above the contro

129 he beta-adrenoceptor agonist, isoproterenol (ISO), or the beta-adrenoceptor antagonist, propranolol.

130 veal that the betaAR agonist, isoproterenol (ISO), promotes enhanced Ca(V) 1.2-Ca(V) 1.2 physical int

131 pe and IL-10 knockout mice by isoproterenol (ISO) infusion.

132 beta-adrenergic receptors by isoproterenol (ISO) resulted in an impaired contractile response of TG

133 ed phosphoprotein (p-VASP) by isoproterenol (ISO), prostaglandin E(2) (PGE(2)), or forskolin (FSK) as

134 nergic receptor activation by isoproterenol (ISO).

135 80)) between groups; however, isoproterenol (ISO) significantly shortened APD(80) in DBH-Sap but not

136 pertrophic stimuli, including isoproterenol (ISO), phenylephrine (PE), and endothelin-1 (ET-1).

137 dult feline heart by infusing isoproterenol (ISO) for 10 days via minipumps, and then animals were al

138 hat chronic administration of isoproterenol (ISO) persistently increases the frequency of mPTP openin

139 gle subcutaneous injection of isoproterenol (ISO; 200 mg/kg) in mice causes acute myocyte death (8%-1

140 nhibition by siRNA to Ces3 on isoproterenol (ISO)-treated 3T3-L1 and brown adipocyte cells.

141 We report that isoproterenol (ISO) and related beta2-adrenergic agonists reacidify lys

142 receptor stimulation (through isoproterenol (ISO) treatment) on heart metabolism.

143 cardiac damage responding to isoproterenol (ISO) in adult offspring that underwent maternal inflamma

144 were examined in response to isoproterenol (ISO), forskolin (FSK), and dibutyryl-cAMP (DB), coupled

145 onic beta-AR stimulation with isoproterenol (ISO) for 48 h reduced Ito,f along with mRNA expression o

146 action after stimulation with isoproterenol (ISO), a beta-adrenergic receptor (betaAR) agonist.

147 uring adrenergic stimulation (isoproterenol, ISO).

148 tive of stilbene compound, isorhapontigenin (ISO), was isolated from this Chinese herb.

149 for 120 min with either 0.9% NaCl [isotonic (ISO)] or 3.0% NaCl [hypertonic (HYPER)].

150 Isoxyl (ISO) and thiacetazone (TAC), two prodrugs once used in t

151 300 mg/kg ISO, or 2 daily doses of 200 mg/kg ISO for 6 days.

152 ngle injections of vehicle, 200 or 300 mg/kg ISO, or 2 daily doses of 200 mg/kg ISO for 6 days.

153 vents, primarily in the break phase of an LF-ISO cycle, reduce the intensity of the following active

154 kening variance of its low-frequency ISO (LF-ISO) cycle.

155 This reduces the LF-ISO intensity and mean ISM rainfall.

156 h native Ag or Ag conjugated to control mAb (ISO*KLH and ISO*Bet v 1).

157 In a time- and dose-dependent manner, ISO increases Nur77 expression in the nuclei of cardiomy

158 rolysis with standard-addition method (MDM), ISO 19448:2018 method; and modified Taves acid-HMDS diff

159 rsus phosphorylation-independent mechanisms, ISO induced a significantly greater increase in MEF2 act

160 les were prepared using the standard method (ISO, 18593:2018) in sensor vials, which were then incuba

161 igarette) among Italian non-mentholated 7 mg ISO tar cigarette smokers.

162 roups 1 and 3 included adult smokers of 7 mg ISO tar tobacco cigarettes, and Group 2 consisted of bot

163 re assessed by scanning electron microscopy, ISO standard flexural strength and modulus measurements,

164 Using our workflow and within minutes, ISO reduced the levels of metabolites involved in glycog

165 of the beta-agonist isoproterenol (0.1 mum; ISO), from 0% to 64% (P < 0.05).

166 rat ventricular myocytes and H9c2 myoblasts, ISO activated nuclear factor-kappaB and inhibited signal

167 Here, we describe mzMatch-ISO, a new extension to the metabolomics analysis pipeli

168 nstrate the power and versatility of mzMatch-ISO by analysing a (13)C-labelled metabolome dataset fro

169 d untargeted isotope profiling using mzMatch-ISO provides a convenient visual summary of the quality

170 nt3a and ISO-603B-conditioned media, but not ISO alone, protected isolated hepatocytes from death, re

171 dy was to validate (SANCO/12495/2011 and NTC-ISO/IEC 17025) multi-residue multi-class methods using Q

172 /beta-catenin pathway (CWP) in the action of ISO on HPC expansion and comparison of ISO with the curr

173 was decreased by 28% after administration of ISO in TG cardiomyocytes.

174 ting these enzymes may impact the binding of ISO and TAC to the dehydratases.

175 epolarization gradient by the combination of ISO and E-4031.

176 on of ISO on HPC expansion and comparison of ISO with the current standard of care, N-acetylcysteine

177 However, the highest concentration of ISO (1 x 10(-5) M) rescued cardiac function.

178 A single dose of ISO causes injury in approximately 10% of the cardiomyoc

179 There was no significant effect of ISO on apoptosis and colony formation of cells transfect

180 These different effects of ISO on LQT1 and LQT2 were verified by optical mapping of

181 ardiac dysfunction after chronic infusion of ISO in mice.

182 During infusion of ISO in vivo, the incidence of delayed afterdepolarizatio

183 V) 1.2 'super-clusters' along the z-lines of ISO-stimulated cardiomyocytes.

184 ly increased ROS production after 4-6 min of ISO application.

185 at the highest frequency only after 6 min of ISO application.Measurement of intra-SR-free Ca2+ concen

186 K2 associated with PEBP1 after 10 minutes of ISO in association with low phosphorylated GRK2 (pGRK2)

187 e of D396 determine the preferred pathway of ISO* relaxation.

188 the same cells, excluding the possibility of ISO regulating XIAP expression at the level of protein d

189 present study, we examined the potential of ISO in anti-cancer activity and the mechanisms involved

190 was subsequently lowered in the presence of ISO (by lowering [Ca(2+)](o) to 1 mM and partially inhib

191 from PBMCs was attenuated in the presence of ISO-1.

192 Strikingly, canagliflozin treatment of ISO-treated rats not only prevents elevation of oxidativ

193 We found that 2 weeks of ISO treatment in adult offspring of LPS-treated mothers

194 delines and accredited successfully based on ISO 17025.

195 Based on ISO criteria only one egg kit accurately determined egg

196 Ion-selective optodes (ISOs), the optical analog of ion-selective electrodes, h

197 r inhibiting Drp1 activity blocks CaMKII- or ISO-induced mPTP opening and myocyte death in vitro and

198 ted in International Standards Organization (ISO) and other international or regional standards.

199 A) and International Standards Organization (ISO) standard ISO 20776-2 criteria using Clinical and La

200 s quasi-rhythmic intraseasonal oscillations (ISO) manifested as alternate 'active' phases of copious

201 e compared areal surface texture parameters (ISO 25178-2) and traditional microwear variables (pits a

202 In contrast, in the presence of IL-13 plus ISO (10 minutes), binding of GRK2 to PEBP1 decreased, wh

203 Interestingly, NHERF1 knockdown prevented ISO-induced chromatin-binding of the transcription facto

204 Further, canagliflozin prevents ISO-induced apoptosis of kidney cells by inhibiting Bax

205 0-fold more active than the well-studied (R)-ISO-1 and more than 200-fold more active than the chrome

206 Our findings provide evidence that repeated ISO exposures may induce behavioral disturbances that ar

207 15LO1 knockdown restored ISO-induced cAMP generation.

208 ion and not only inhibited but also reversed ISO-induced cardiac remodeling.

209 ue-light excitation, the isoalloxazine ring (ISO) may undergo an ultrafast reduction by a nearby tryp

210 Single ISO injections also caused membrane injury in approximat

211 ltered diastolic function 1 day after single ISO injection.

212 nse mutation in the hadA gene of spontaneous ISO- and TAC-resistant mutants was sufficient to confer

213 re also found in M. tuberculosis spontaneous ISO- and TAC-resistant mutants.

214 Growth rate inhibition occurred in standard ISO tests (EC50 values of 15-200 mg Pt/L), but also in a

215 tional Standards Organization (ISO) standard ISO 20776-2 criteria using Clinical and Laboratory Stand

216 , align with the international AST standard (ISO 200776-1; 2006) and could be used for rapid determin

217 current International Organization Standard (ISO/DIS 15197) requirements.

218 ernational Organization for Standardization (ISO) (ISO/TS 17728, ISO 18593:2004 and ISO 17604:2003) s

219 ernational Organization for Standardization (ISO) International Workshop Agreement on tiered cookstov

220 ernational Organization for Standardization (ISO) is designing a new solution for the representation,

221 ernational Organization for Standardization (ISO) protocols.

222 ivity to circulating adrenergic stimulation (ISO), while having blunted responses to SNS, providing i

223 Interestingly, IL-10 suppressed ISO-induced nuclear factor-kappaB activation and attenua

224 Factors to convert HS values, for the target ISO and GDC components, into theoretical EO values were

225 2O, P </= 0.01), which remained greater than ISO throughout heating.

226 We found that ISO administration elevates renal oxidative stress marke

227 We found that ISO exhibited significant inhibitory effects on human bl

228 We also identified that ISO down-regulated XIAP gene transcription via inhibitio

229 These results support the notion that ISO acutely accelerates oxidative metabolism of glucose

230 Further voltage-clamp studies revealed that ISO increases L-type calcium current (I(Ca)) faster than

231 Our data showed that ISO administration inflicts pro-oxidative changes in hea

232 Further studies have shown that ISO down-regulation of XIAP protein expression was only

233 These studies suggest that ISO injury activates cardiac progenitor cells that can d

234 mapping of the whole heart, suggesting that ISO-induced EADs are genotype specific.

235 The ISO 19448:2018 MDM is a reliable test that can be used a

236 The ISO method for calculating wind driven rain loads is ada

237 The ISO-FOOD ontology consists of metadata and provenance da

238 The ISO-standardized RHIZOtest is used here for the first ti

239 oxide dismutase mimetic MnTBPA abolished the ISO-mediated ROS production.

240 NHERF1 knockdown fully abrogated the ISO-, PGE(2)-, and FSK-induced IL-6 gene expression and

241 In addition, the ISO-stimulated phosphorylation of phospholamban at Ser(1

242 to a nested RT-PCR genotyping assay and the ISO 15216-1 rRT-PCR method for HAV detection.

243 WWL229 or siRNA dramatically attenuated the ISO-induced lipolytic effect in the cells.

244 We illustrate the ISO-GEM method in an exploratory study on the effect of

245 fore, this approach could be included in the ISO 3632 method (2011).

246 athways and upstream signaling of Ras in the ISO-specific context.

247 ckers of betaAR-1 and betaAR-2 inhibited the ISO response in Pkd2(+/-) mice, suggesting that the deph

248 more, we discovered that when KCC2 lacks the ISO domain, it still retains swelling-activated transpor

249 The aim of this work was a comparison of the ISO 3632 (2011) method and an HPLC-DAD method for safran

250 A re-evaluation of the ISO standard for bottom sediment sampling is recommended

251 ve STAT3 mimicked the IL-10 responses on the ISO effects, confirming that the IL-10-mediated inhibiti

252 Ideltac nor CaMKIIdeltab did not prevent the ISO-mediated Ito,f reductions, even though CaMKIIdeltac

253 in, rotenone, also effectively prevented the ISO-mediated ROS production.

254 ast, NF-kappaB inhibition partly rescued the ISO-mediated Ito,f reductions in association with restor

255 f the C terminus, unique to KCC2 (termed the ISO domain), is required for KCC2 to cotransport K(+) an

256 This finding indicates that the ISO domain is required for neuronal Cl(-) regulation.

257 NA and KChIP2 promoter data suggest that the ISO-induced Ito,f reductions are, in part, mediated thro

258 absorbance values measured according to the ISO Normative 3632-1:2011 method.

259 adopted for quality grading according to the ISO Normative 3632.

260 he method was performed in accordance to the ISO-17025 (ISO/IEC, 2005) and Eurachem guidelines.

261 fold from the uncontrolled areas towards the ISO 5 cleanroom, accompanied with a reduction of the liv

262 (19,303 +/- 3939 ng/L.min) compared with the ISO trial (18,600 +/- 3755 ng/L.min; P < 0.05).

263 01.5 +/- 190.5 mmol/L.min) compared with the ISO trial (354.0 +/- 205.8 mmol/L.min; P < 0.05).

264 of hydroxyproline determination according to ISO 17025.

265 origins were analysed by UV-vis according to ISO 3632 (2011) and by HPLC-DAD.

266 amples with the highest quality according to ISO 3632 specifications produced a typical spectrum prof

267 Aqueous extracts prepared according to ISO 3632-2 were then examined by tristimulus colorimetry

268 both procedures were evaluated according to ISO 9909 and GDC standards for sage EO quality, revealin

269 s C) and tested for SOR and SOL according to ISO Specification 4049 (2009).

270 of the respective laboratories conforming to ISO/IEC 17025:2005 standard techniques.

271 eous action potentials (APs) when exposed to ISO (9.99 +/- 4.2 vs. 0.16 +/- 0.05 APs/min, p = 0.004);

272 also found that repeated early exposures to ISO induced subtle, sex-specific disruptions to activity

273 ease in the resistance of M. tuberculosis to ISO and TAC, respectively.

274 nhancing electron transfer (ET) from W400 to ISO*.

275 ontrast to these regional distinctions under ISO, alterations in Ca(2+) handling at baseline and myoc

276 and fibroblast lines were established under ISO class 5 cGMP conditions.

277 ials, with spontaneous Ca(2+) release, under ISO.

278 5F and 3R4F research cigarettes smoked under ISO (Cambridge Filter or FTC) and Intense (Health Canada

279 with a chimeric KCC2 that lacked the unique ISO domain, hyperpolarizing responses to GABA were aboli

280 In vitro, ISO-induced proliferation of 603B cells was CWP dependen

281 from a rabbit model of heart failure, where ISO-induced nuclear import is ablated, but G(q)-agonist

282 R 55,850 +/- 36,488 pmol/L.min compared with ISO 57,205 +/- 31,119 pmol/L.min).

283 ed ISO-induced cAMP generation compared with ISO for 10 minutes alone paralleled by increases in beta

284 mice anesthetized with KX in comparison with ISO.

285 All sealers complied with ISO specifications.

286 urning unprocessed wood, and consistent with ISO tiers 4 and 5 for PM(2.5) and CO, respectively.

287 eviation (s( *)), were assigned in line with ISO 13528.

288 euronal networks in a consistent method with ISO 10993-5 guidance.

289 Treatment of myocytes with ISO for 15 min significantly reduced the free thiol cont

290 rmal microdialysis probes, one perfused with ISO saline and the other with HYPER saline.

291 Pretreatment with ISO or IL-13 decreased ISO-induced cAMP generation compa

292 0 minutes) pretreatment were stimulated with ISO (10 minutes).

293 ock protein 20 after 4 h of stimulation with ISO and FSK.

294 e we show that treating M. tuberculosis with ISO or TAC results in both cases in the accumulation of

295 Z-ISO evolved from an ancestor related to the NnrU (for ni

296 improving nutritional content) argued for Z-ISO as a pathway requirement.

297 arative genomics illuminated the origin of Z-ISO found throughout higher and lower plants, algae, dia

298 To prove the existence of Z-ISO, maize (Zea mays) and Arabidopsis (Arabidopsis thali

299 This step, mediated by a putative Z-ISO, was predicted to occur in the sequence of redox rea

300 ,15,9'-tri-cis-zeta-carotene, proving that Z-ISO encoded the missing step.