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1 espectively, allowing the detection of serum IgA deficiency.
2 g volume-reduction surgery, or had selective IgA deficiency.
3 inase antibody level in the setting of serum IgA deficiency.
4 n variable immunodeficiency and/or selective IgA deficiency.
5 a, venous thromboembolism, hyposplenism, and IgA deficiency.
6 2 antibody is not specific in the absence of IgA deficiency.
7 of the patients with polyarthritis also had IgA deficiency.
8 ctive Ig class switch in the pathogenesis of IgA deficiency.
9 tal IgG production was impaired in mice with IgA deficiency.
10 ronic fatigue syndrome, IgA nephropathy, and IgA deficiency.
12 emia was detected in all but 1 patient, with IgA deficiency affecting 90% of patients and a deficienc
13 ficiency (IgAD) are often not aware of their IgA deficiency and are tested as CD negative, delaying c
14 genetic variation in MSH5 is associated with IgA deficiency and common variable immune deficiency (CV
15 defective B-cell differentiation in familial IgA deficiency and common variable immunodeficiency.
16 Patients with IgG subclass deficiency with IgA deficiency and those with CVID showed defects in bot
17 0 patients with IgG subclass deficiency with IgA deficiency, and 223 age-matched control subjects wer
18 n variable immunodeficiency, IgG deficiency, IgA deficiency, and IgM deficiency in patients with CRS
20 pulmonary LVS infections not only because of IgA deficiency but also because of reduced IFN-gamma res
24 ficiency (CVID) and 1 of 16 individuals with IgA deficiency (IgAD) had a missense mutation in one all
25 eficiency (CVID), 68 patients with selective IgA deficiency (IgAdef), 10 patients with IgG subclass d
30 ical immunosensor for the detection of human IgA deficiency in real human blood serum has been develo
31 ion, and a selective local immunoglobulin A (IgA) deficiency in HSCT-treated SCID patients with genet
37 milar to the mice, Ig S joints from CVID and IgA deficiency patients carrying disease-associated MSH5
38 idered to have celiac disease with selective IgA deficiency rather than seronegative celiac disease.
39 cific depletion of meningeal plasma cells or IgA deficiency resulted in reduced fungal entrapment in
41 from healthy donors, patients with selective IgA deficiency (SIgAd), and patients with common variabl
42 -microbiota symbiosis in mice, yet selective IgA-deficiency (sIgAd) in humans is often described as a
43 AGA, EMA, or only IgG AGA in the presence of IgA deficiency was offered a small bowel biopsy to confi