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1                                              Intralipid also promotes the polarization of macrophages
2                                              Intralipid and glucosamine infusions decreased insulin-s
3                                              Intralipid elicited mild inflammation in both perirenal
4                                              Intralipid enhanced pyruvate dehydrogenase (PDH) phospho
5                                              Intralipid has been shown to reduce nano-oxaliplatin-med
6                                              Intralipid increased perirenal fat weight, adipocyte siz
7                                              Intralipid infusion did not alter the time profiles of p
8                                              Intralipid infusion reduced insulin-stimulated glucose u
9                                              Intralipid is a lipid emulsion used for preterm infants,
10                                              Intralipid/heparin or saline infusion was initiated 2 h
11             Accordingly, 30% glucose and 10% Intralipid were infused for 6 h in ten normal lean indiv
12 d occasions combined with 0.9% saline or 10% Intralipid at 100 mL/h.
13                          Importantly, 0.5-2% Intralipid, which delivered more FA to the liver than 6
14 st, 6-h infusions of either 6 mM OA or 4-20% Intralipid increased apoB secretion.
15 etion increased only after infusion of 4-20% Intralipid; TG secretion was unchanged by 6 mM OA.
16 mM oleic acid (OA) bound to albumin, 0.5-20% Intralipid, or saline for 3 or 6 h into male C57BL/6J mi
17                              6 mM OA and 20% Intralipid each increased secretion of apoB from primary
18  levels increased similarly after either 20% Intralipid or 6 mM OA, TG secretion increased only after
19                         Here, we infused 20% Intralipid (a synthetic lipid emulsion) or saline intrad
20  1 mmol/l in both groups) by infusion of 20% Intralipid (60 ml/h) and heparin (900 U/h).
21 els were unaffected by either 6 mM OA or 20% Intralipid, but microsomal triglyceride transfer protein
22 ons with 6 mM OA versus either saline or 20% Intralipid.
23 mic clamps with (n = 26) or without (n = 23) Intralipid and heparin infusion.
24 9+/-9.1 baseline, 33.7+/-8.2 I+G, 39.9+/-9.3 Intralipid; P<0.001).
25                       We found that an acute Intralipid infusion into the upper small intestine (USI)
26 min(-1)), l-NAME, AICAR + l-NAME, or AICAR + Intralipid (20%, 0.02 ml. kg(-1). min(-1)).
27 examined how endogenous GDF15 responds to an Intralipid infusion in different organs to regulate food
28 ported that a 72-h coinfusion of glucose and Intralipid (GLU+IL) induces insulin resistance and a mar
29 in LPL release after intravenous heparin and Intralipid strongly suggest that GPIHBP1 represents an i
30         Triglyceride micro-emulsions such as Intralipid(R) have been used to reverse cardiac toxicity
31 eral nutrition containing soybean oil-based (Intralipid) or olive oil-based (ClinOleic) lipid emulsio
32               Kidney Gdf15 knockdown blunted Intralipid-induced increases in kidney and plasma GDF15
33 ing free fatty acid levels in fed animals by Intralipid plus heparin infusion caused significant incr
34        Pediatric patients before the change (Intralipid [IL] group) were compared with patients after
35                          In contrast, during Intralipid infusion, cardiac long-chain fatty acid (LCFA
36 rons as well as the synthetic lipid emulsion Intralipid.
37 liquid diet plus intravenous lipid emulsion (Intralipid, 4 g fat/kg/d) or intravenous saline for 19 d
38  triphosphate production versus 17+/-16% for Intralipid; P=0.002); however, no change in cardiac func
39         Also, when infused with glucosamine, Intralipid decreased insulin action below that with gluc
40             Compared with the control group, Intralipid and lactate infusions decreased glucose infus
41 FFAs were increased in seven women by an 8-h Intralipid/heparin (IL/hep) infusion, and the results we
42 subjects to approximately 1 mmol/l by an 8-h Intralipid/heparin (IL/Hep) infusion, whereas they fell
43 sulin clamp period before glucose ingestion, Intralipid/heparin infusion reduced R(d) (4.4 +/- 0.3 vs
44 se 9.3 +/- 0.7 mmol/l) during an intravenous Intralipid/heparin infusion and 7-10 days later during a
45   We investigated the effects of intravenous Intralipid on the adipose tissue development of midgesta
46 stulas during intestinal perfusion of lipid (Intralipid, 170 micromol h(-1), chylous lymph) or a dext
47  and after intraduodenal infusions of lipid (Intralipid; 10 ml, 5 kcal).
48 te administration of 25 mg ILE/kg body mass (Intralipid(R) 20%) at 1 mL/min using infusion pumps.
49  separate study, short-term (50 and 180 min) Intralipid infusion also failed to increase muscle HBP p
50                                    Moreover, Intralipid treatment suppressed the expression of fibros
51 and kidney, with and without the addition of Intralipid.
52 sma FFA concentration by coadministration of Intralipid plus heparin to nicotinic acid-treated rats (
53 ocedure with the addition of a coinfusion of Intralipid plus heparin (together with NA) to maintain a
54                           The combination of Intralipid with half the standard clinical dose of Abrax
55 evels were increased by incremental doses of Intralipid.
56 which negated the feeding-lowering effect of Intralipid despite a rise in plasma GDF15 levels in chow
57 -fasted rats received a constant infusion of Intralipid or lactate for 5 h, while a control group rec
58 genously via a low- or high-dose infusion of Intralipid plus heparin or endogenously by an infusion o
59                     After 2 h of infusion of Intralipid plus heparin, FFA levels increased from 562+/
60 e study day, FFA was elevated by infusion of Intralipid plus heparin.
61 mental conditions, intraduodenal infusion of Intralipid, compared with saline, did not affect plasma
62                        Also, an injection of Intralipid released LPL into the plasma of wild-type mic
63 a high fat test meal or by i.v. injection of Intralipid.
64   Our findings suggest that pre-treatment of Intralipid has the potential to be a powerful agent to e
65 roM C-SNAFL-1 containing 2.0% (by volume) of Intralipid as a scatterer, the values of the average lif
66 underwent cyclic infusions of glucose and/or Intralipid prior to islet isolation and analysis by quan
67 fusions of insulin+glucose infusion (I+G) or Intralipid infusion.
68 usion of saline, Intralipid, glucosamine, or Intralipid and glucosamine (n = 8 or 9 for each) for 330
69 hicle; Intralipid (30 mL/h) plus vehicle; or Intralipid (30 mL/h) plus eritoran (12 mg i.v. every 12
70 n of either emulsion (210 mg/kg) or placebo (Intralipid diluted 1 : 64).
71                              After recovery, Intralipid 20 or Lactated Ringer's Solution were infused
72 s received an additional infusion of saline, Intralipid, glucosamine, or Intralipid and glucosamine (
73 7 each) for an additional 5 h, while saline, Intralipid, or lactate infusion was continued.
74  using an FDA approved nutrition supplement, Intralipid.
75                             We conclude that Intralipid infusion into the duodenum at this rate does
76 reast cancer mouse model, we have found that Intralipid pre-treatment significantly increases the amo
77                     Here, we have found that Intralipid reduces the cytotoxicity of paclitaxel for hu
78                         Our study shows that Intralipid treatment exhibits no harmful effect on tumor
79                  These findings suggest that Intralipid administration at a developmental stage prior
80                             In contrast, the Intralipid-induced insulin resistance was accompanied by
81 he same amount of glycerol as present in the Intralipid infusion.
82                        Cardiomyocytes of the Intralipid-treated fetuses were 14% more terminally diff
83 sover design: saline (30 mL/h) plus vehicle; Intralipid (30 mL/h) plus vehicle; or Intralipid (30 mL/
84 ying scatterer concentrations (1.5-3.0 vol % Intralipid).
85       Myocardial energetics were better with Intralipid compared with I+G (phosphocreatine/adenosine
86 on of free fatty acids during the clamp with Intralipid infusion reduced PAHSAs' effects on EGP in HF
87 increased above basal during the clamps with Intralipid infusion (P < 0.01 for both).
88 lation was also significantly decreased with Intralipid and lactate infusions.
89 A level was impaired in skeletal muscle with Intralipid and lactate infusions, resulting in two- to t
90 ecreased in individual skeletal muscles with Intralipid infusion (P < 0.05).
91 extensor digitorum longus (EDL) muscles with Intralipid infusion in both clamps (P < 0.05).
92 uxes could be mimicked by infusing rats with Intralipid or corticosterone and were corrected by lepti
93  decreased 47%, significantly more than with Intralipid or glucosamine alone (P < 0.05).