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1 aluable study cohort of 20 patients was 61% (Kaplan-Meier estimate).
2 tation after blinatumomab treatment was 65% (Kaplan-Meier estimate).
3 ssessed by the incidence of RBC transfusion (Kaplan-Meier estimate).
4 tion-free survival at four months was 90.2% (Kaplan-Meier estimate).
5 in other countries (P<0.001, on the basis of Kaplan-Meier estimates).
6 progression for responders of 86.6 weeks by Kaplan-Meier estimate.
7 Survival rates were analyzed using Kaplan-Meier estimate.
8 Time to recurrence was calculated using Kaplan Meier estimates.
9 OS was compared by Kaplan-Meier estimates.
10 Incidence was estimated using Kaplan-Meier estimates.
11 lyzed according to recipient BMI class using Kaplan-Meier estimates.
12 were assessed by Cox regression analysis and Kaplan-Meier estimates.
13 Cumulative stroke risk was calculated using Kaplan-Meier estimates.
14 d graft survival, and patient survival using Kaplan-Meier estimates.
15 entia risk per sum score was calculated with Kaplan-Meier estimates.
16 Survival analysis was performed using Kaplan-Meier estimates.
17 Oncological outcome was assessed using Kaplan-Meier estimates.
18 ase activity-free intervals was evaluated by Kaplan-Meier estimates.
19 h time-to-event analysis ascertained through Kaplan-Meier estimates.
20 (PFS) and overall survival (OS) assessed by Kaplan-Meier estimates.
21 e to regression of seeds were estimated with Kaplan-Meier estimates.
22 absolute risks by calculating prevalence and Kaplan-Meier estimates.
23 g options had been lost in two participants (Kaplan-Meier estimate 0.7%) in the OT group and six (2.1
26 ent from those of patients treated with PTA (Kaplan-Meier estimates 1.8+/-0.7% versus 1.3+/-0.9%, 6.5
28 basis of personalized cytogenetic profiles, Kaplan-Meier estimates (1, 3, and 5 years) for melanoma-
29 or renal failure (placebo, 75 events [60-day Kaplan-Meier estimate, 13.0%]; serelaxin, 76 events [13.
31 stenting group (cumulative incidence, 24.6%; Kaplan-Meier estimate, 26.2%) and 45 patients in the end
33 erectomy group (cumulative incidence, 26.9%; Kaplan-Meier estimate, 30.3%) (absolute difference in cu
34 ween TAVR and SAVR in the composite outcome (Kaplan-Meier estimates 38.0% vs. 36.3%, log-rank test p=
35 n randomisation and 120 days, there were 34 (Kaplan-Meier estimate 4.0%) events of disabling stroke o
39 d ICH during 5197 person-years of follow-up (Kaplan-Meier estimated 5-year risk 15.8%, 95% CI 13.7-17
40 reated with alemtuzumab (66 of 133 patients, Kaplan-Meier estimate 51.6%, 95% CI 43.2-60.7%) than pat
43 or stroke were similar in the three groups (Kaplan-Meier estimates, 6.5% in group 1, 5.6% in group 2
44 h alemtuzumab were free of CDA at 36 months (Kaplan-Meier estimate 71.8%, 95% CI 63.1-78.8%) compared
45 and 750 of 8881 in the placebo group (3-year Kaplan-Meier estimates 8.1%vs 9.7%, HR 0.80, 95% CI 0.72
48 es in all-cause mortality were examined with Kaplan-Meier estimates, adjusted logistic regression, an
49 rogression-free survival was evaluated using Kaplan-Meier estimates and a Cox proportional hazards re
56 We measured IUC discontinuation rates with Kaplan-Meier estimates and Cox proportional hazards mode
57 ata System database between 1988 and 1998 by Kaplan-Meier estimates and Cox proportional hazards mode
61 comes and graft survival were analyzed using Kaplan-Meier estimates and Cox univariate and multivaria
65 nary resuscitation organs was compared using Kaplan-Meier estimates and stratified log-rank test.
68 and multivariate logistic regression models, Kaplan-Meier estimates, and Cox proportional hazards mod
69 noma-related mortality were calculated using Kaplan-Meier estimates, and Cox proportional hazards reg
71 lyses were performed using log-rank test and Kaplan-Meier estimates, and multivariate analyses were p
72 ions among indicators and the log-rank test, Kaplan-Meier estimates, and multivariate Cox proportiona
80 istry and were 91.5% and 83.2% at 5 years by Kaplan-Meier estimates based on linked United Network fo
82 ssion-free survival (PFS) were explored with Kaplan-Meier estimates, Cox regression, and random survi
83 t, Kruskal-Wallis, Spearman correlation, and Kaplan-Meier estimates; Cox regression models were perfo
84 ients in the transvenous ICD group (48-month Kaplan-Meier estimated cumulative incidence, 15.1% and 1
85 At 3 years after coronary angiography, the Kaplan-Meier estimated cumulative percentages with event
86 f 5.8%(95%CI, -1.4%to 13.1%) [corrected].The Kaplan-Meier estimated cumulative percentages with event
90 d 42 patients in the control group (12-month Kaplan-Meier estimated event rate, 0.7% and 1.2%, respec
91 236 patients in the control group (12-month Kaplan-Meier estimated event rate, 6.0% and 6.9%, respec
92 cluding breast cancer, were calculated using Kaplan-Meier estimates, Fine and Gray competing-risks re
93 With a median follow-up of 39.7 months, the Kaplan-Meier estimate for 2-year overall survival was 98
95 ls were collected every 6 months; the 4-year Kaplan-Meier estimate for incidence of HgbA1c levels >/=
100 s with DCB were also superior to PTA per the Kaplan-Meier estimate for primary patency (89.0% versus
101 nts (age 67+/-16.2 years; 53.7% female), the Kaplan-Meier estimate for stroke/TIA recurrence within 1
107 9.6% when refCFVR </= 2.7 (P<0.001), whereas Kaplan-Meier estimates for cardiac mortality were 7.7% w
112 iting times to transplant were obtained from Kaplan-Meier estimates for patients registered 1998-2000
120 and 7.2%, respectively, according to 2-year Kaplan-Meier estimates; hazard ratio with saxagliptin, 1
121 , vs. 25% in the placebo group, according to Kaplan-Meier estimates; hazard ratio, 0.36; P=0.003).
122 and 12.4%, respectively, according to 2-year Kaplan-Meier estimates; hazard ratio, 1.02; 95% CI, 0.94
123 an-Meier estimate] vs 151/8849 [2.1%, 3-year Kaplan-Meier estimate], HR 1.61, 95% CI 1.31-1.97; p<0.0
124 ied end point and 21% (95% CI, 7% to 26%) by Kaplan-Meier estimate in a post hoc analysis using metho
125 < 3 years), and 20% (95% CI, 10% to 37%) by Kaplan-Meier estimate in post hoc analysis using definit
133 with objective tumor regressions (31%), the Kaplan-Meier estimated median response duration was 2 ye
136 of response (DR) have not been reached, but Kaplan-Meier estimated medians are 17.8 months (range, 5
145 on revascularization when compared with PTA (Kaplan-Meier estimate of 74.5% versus 65.3%; log-rank P=
147 atment was superior to short-term treatment (Kaplan-Meier estimate of difference 14.3% [5.1-23.6]; ha
148 .4% [95% CI 41.9-55.0] vs 56.4% [49.1-63.6]; Kaplan-Meier estimate of difference 7.9% [-1.9 to 17.7];
149 atients met the primary endpoint of relapse (Kaplan-Meier estimate of event rate 36.0% [95% CI 20.6-5
150 one of those assigned to continue treatment (Kaplan-Meier estimate of event rate 45.7% [95% CI 28.5-6
153 At 1 year, there were 139 deaths, and the Kaplan-Meier estimate of freedom from mortality was 76.8
167 an follow-up of 24 (range, 3-36) months, the Kaplan-Meier estimate of progression (>/= 1.0 point EDSS
178 point events occurred in the ablation group (Kaplan-Meier estimate of the percentage of patients with
186 6-patient pooled nonrandomized DCB data set (Kaplan-Meier estimates of 2.1%, 4.9%, and 7.0% at 1, 2,
191 d placebo groups, respectively, resulting in Kaplan-Meier estimates of 77.2% (95% CI 71.87-82.51) of
193 aspirin was associated with similar 180-day Kaplan-Meier estimates of adjudicated composite GI event
203 int was demonstrated for Arm 2 versus Arm 1; Kaplan-Meier estimates of efficacy failure were 42.2% an
207 having 1.5% and 2.7% absolute reductions in Kaplan-Meier estimates of HHF risk at 4 years, respectiv
215 310 deaths among patients without bleeding (Kaplan-Meier estimates of mortality, 4.5%, 10.0%, and 2.
226 After a median follow-up of 36 months, the Kaplan-Meier estimates of PFS were 86% (95% confidence i
231 ed the effect of selection for tracing using Kaplan-Meier estimates of reengagement among all patient
233 s. 95%; p = 0.03 by log-rank test) on 5-year Kaplan-Meier estimates of survival after surgical AVR.
234 er or not there is evidence for such bias in Kaplan-Meier estimates of survival probabilities for car
243 n group and in 584 in the placebo group; the Kaplan-Meier estimates of the incidence at 3 years were
244 mponent of the dual-design study, the 5-year Kaplan-Meier estimates of the incidence of arrhythmic ev
245 mponent of the dual-design study, the 5-year Kaplan-Meier estimates of the incidence of arrhythmic ev
246 103 (8.2 percent) in the control group; the Kaplan-Meier estimates of the likelihood of freedom from
249 Randomization to PPI therapy reduced 180-day Kaplan-Meier estimates of the primary GI endpoint in low
254 two-tailed) for categorical comparisons, and Kaplan-Meier estimates of time to events of interest.
260 bjects experienced a recurrence, as did 58% (Kaplan-Meier estimate) of those who remained well for at
261 h test/validation set-defined cut points and Kaplan-Meier estimated outcome measures of 5-year overal
266 se outcomes than patients with complete PVD (Kaplan-Meier estimated probability and standard error, 1
269 rsonalized cytogenetic profiles, with 5-year Kaplan-Meier estimates ranging from 4% with chromosomes
274 as not powered for survival as an end point, Kaplan-Meier estimates showed a trend in overall surviva
278 ith secondary enucleation and metastases and Kaplan-Meier estimates to assess the probability of meta
280 ther day of monitored hospitalization, using Kaplan-Meier estimates to determine the rate of resuscit
282 us the placebo group (241/8880 [3.4%, 3-year Kaplan-Meier estimate] vs 151/8849 [2.1%, 3-year Kaplan-
284 utero transmission of HIV-1 on the basis of Kaplan-Meier estimates was 5.7% (93 infants), with no si
286 eiving placebo had elective surgical repair (Kaplan-Meier estimates were 16.1% for those receiving do
287 doresection groups, respectively, the 5-year Kaplan-Meier estimates were as follows: overall survival
293 iables, t test for continuous variables, and Kaplan-Meier estimates were used to describe events.
295 mated "freedom of incisional hernia" curves (Kaplan-Meier estimate) were significantly different acro
298 ll receiving tamoxifen alone; Cox models and Kaplan-Meier estimates with inverse probability of censo
300 ccurred in 43 of 8880 patients (0.6%, 3-year Kaplan-Meier estimate) with vorapaxar versus 28 of 8849