コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 Castleman's disease, as well as its namesake Kaposi's sarcoma.
2 elated malignancies, including lymphomas and Kaposi's sarcoma.
3 of vGPCR-induced lesions that resemble human Kaposi's sarcoma.
4 eta3 in KSHV pathogenesis and development of Kaposi's sarcoma.
5 d herpesvirus (KSHV), the etiologic agent of Kaposi's sarcoma.
6 homas, multicentric Castleman's disease, and Kaposi's sarcoma.
7 ovarian, breast, and lung cancer, as well as Kaposi's sarcoma.
8 an's disease, primary effusion lymphoma, and Kaposi's sarcoma.
9 9, 95% CI 1.67-1.72), AIDS-defining cancers (Kaposi's sarcoma [498.11, 477.82-519.03], non-Hodgkin ly
10 mmaherpesvirus etiologically associated with Kaposi's sarcoma, a vascular tumor of endothelial cells.
11 SHV) is the cause of aggressive AIDS-related Kaposi's sarcoma (AIDS-KS) characterized by abnormal ang
13 erpesvirus 8 (HHV-8), the causative agent of Kaposi's sarcoma and B cell neoplasms in AIDS patients,
14 herpesvirus (KSHV) is the causative agent of Kaposi's sarcoma and is associated with two B cell malig
15 s tightly associated with the development of Kaposi's sarcoma and multicentric Castleman's disease, a
16 -cell pathologies, including AIDS-associated Kaposi's sarcoma and multicentric Castleman's disease.
17 tients at the Haitian Group for the Study of Kaposi's Sarcoma and Opportunistic infections (GHESKIO)
18 herpesvirus (KSHV) is the etiologic agent of Kaposi's sarcoma and other B-cell cancers and remains a
19 is an oncogenic gammaherpesvirus that causes Kaposi's sarcoma and other lymphoproliferative disorders
20 rpesvirus (KSHV) is the etiological agent of Kaposi's sarcoma and primary effusion lymphoma (PEL).
21 herpesvirus (KSHV), the etiological agent of Kaposi's sarcoma and primary effusion lymphoma, has deve
22 ted herpesvirus (KSHV) is causally linked to Kaposi's sarcoma and several lymphoproliferative disease
23 herpesvirus (KSHV) is the causative agent of Kaposi's sarcoma, and a constitutively active viral G pr
24 IFI16 knockdown disrupted the latency of Kaposi's sarcoma associated herpesvirus (KSHV) and induc
26 ing an m(6)A-modified hairpin present in the Kaposi's sarcoma associated herpesvirus (KSHV) ORF50 RNA
27 Human herpes virus 8 (HHV-8), also known as Kaposi's sarcoma associated herpesvirus (KSHV), is an on
28 G, and panel), Epstein-Barr Virus (EBV), and Kaposi's Sarcoma-associated Herpes Virus (KSHV) in a fin
32 r maturation of RRV, the closest relative of Kaposi's sarcoma-associated herpesvirus (KSHV) (a human
34 ymphomas (PELs) are causally associated with Kaposi's sarcoma-associated herpesvirus (KSHV) and 86% o
35 protein (SCP) of human tumor herpesviruses--Kaposi's sarcoma-associated herpesvirus (KSHV) and Epste
38 s KSHV-miR-K12-11 and MDV-miR-M4, encoded by Kaposi's sarcoma-associated herpesvirus (KSHV) and Marek
42 ed with the closely related gammaherpesvirus Kaposi's sarcoma-associated herpesvirus (KSHV) and the m
46 al carcinogens, Epstein-Barr virus (EBV) and Kaposi's sarcoma-associated herpesvirus (KSHV) are two o
49 of arachidonic acid pathways of the host in Kaposi's sarcoma-associated herpesvirus (KSHV) biology h
50 maherpesviruses Epstein-Barr virus (EBV) and Kaposi's sarcoma-associated herpesvirus (KSHV) cause lif
51 ive stages of the KSHV life cycle.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) causes AI
53 Here, we addressed whether the oncogenic Kaposi's sarcoma-associated herpesvirus (KSHV) deregulat
59 encoded by open reading frame 36 (ORF36) of Kaposi's sarcoma-associated herpesvirus (KSHV) enhances
64 inity tagged and purified all 89 proteins of Kaposi's sarcoma-associated herpesvirus (KSHV) from huma
67 ous transcription across the entirety of the Kaposi's sarcoma-associated herpesvirus (KSHV) genome an
68 e found that most transcripts encoded by the Kaposi's sarcoma-associated herpesvirus (KSHV) genome un
70 y focal assembly of RNA polymerase II around Kaposi's sarcoma-associated herpesvirus (KSHV) genomes i
71 ected cells.IMPORTANCE B cells infected with Kaposi's sarcoma-associated herpesvirus (KSHV) harbor mu
77 we present an innovative approach to culture Kaposi's sarcoma-associated herpesvirus (KSHV) infected
78 d levels of viral lytic gene expression when Kaposi's sarcoma-associated herpesvirus (KSHV) infected
79 L) is a lymphogenic disorder associated with Kaposi's sarcoma-associated herpesvirus (KSHV) infection
82 was undertaken to determine whether de novo Kaposi's sarcoma-associated herpesvirus (KSHV) infection
83 an effective small-animal model to study the Kaposi's sarcoma-associated herpesvirus (KSHV) infection
84 script C19ORF66 and showed that it restricts Kaposi's sarcoma-associated herpesvirus (KSHV) infection
93 Kaposi's sarcoma (KS) caused by oncogenic Kaposi's sarcoma-associated herpesvirus (KSHV) is a high
100 inhibiting viral gene expression.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) is a path
102 ting their premature accumulation.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) is an onc
104 predominantly canonical in nature.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) is associ
105 g the intracellular redox balance.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) is associ
106 l that enhance antiviral defenses.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) is causal
115 d transmission in the oral cavity.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) is the ca
132 on of the STAT3 signaling pathway.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) is the et
134 ects in KSHV late gene expression.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) is the et
136 lytic phases.IMPORTANCE The K15P protein of Kaposi's sarcoma-associated herpesvirus (KSHV) is though
140 on factors which play important roles in the Kaposi's sarcoma-associated herpesvirus (KSHV) latent an
142 e factors (HIFs) play important roles in the Kaposi's sarcoma-associated herpesvirus (KSHV) life cycl
143 r, Ago2 and DCP1A, in PB formation, and that Kaposi's sarcoma-associated herpesvirus (KSHV) lytic inf
144 at the constitutive AMPK activity restricted Kaposi's sarcoma-associated herpesvirus (KSHV) lytic rep
145 veloping germinal center B cells, can induce Kaposi's sarcoma-associated herpesvirus (KSHV) lytic rep
147 tic targets to control KS lesions.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) manipulat
149 DSBs) in DNA, although detail concerning how Kaposi's sarcoma-associated herpesvirus (KSHV) modulates
150 egy of selectively isolating VLVs by using a Kaposi's sarcoma-associated herpesvirus (KSHV) mutant th
152 ere, we identify a viral trimeric complex of Kaposi's sarcoma-associated herpesvirus (KSHV) open read
158 ncy-associated nuclear antigen (LANA) of the Kaposi's sarcoma-associated herpesvirus (KSHV) performs
161 nprecedented mechanism of viral replication: Kaposi's sarcoma-associated herpesvirus (KSHV) stably cl
162 recently showed that the interaction between Kaposi's sarcoma-associated herpesvirus (KSHV) tegument
163 e kinase A2 (EphA2) is an entry receptor for Kaposi's sarcoma-associated herpesvirus (KSHV) that is e
164 sociated substrate) as a platform to promote Kaposi's sarcoma-associated herpesvirus (KSHV) trafficki
167 maherpesviruses Epstein-Barr virus (EBV) and Kaposi's sarcoma-associated herpesvirus (KSHV) utilize c
169 rophoretic response of B cells infected with Kaposi's sarcoma-associated herpesvirus (KSHV) were inve
170 e expression occurs upon superinfection with Kaposi's sarcoma-associated herpesvirus (KSHV), a common
173 iral FLICE-like inhibitor protein (vFLIP) of Kaposi's sarcoma-associated herpesvirus (KSHV), against
175 ncy is an integral part of the life cycle of Kaposi's sarcoma-associated herpesvirus (KSHV), an etiol
176 V1) lineage consists of human herpesvirus 8, Kaposi's sarcoma-associated herpesvirus (KSHV), and clos
177 DNA tumor viruses Epstein-Barr virus (EBV), Kaposi's sarcoma-associated herpesvirus (KSHV), and huma
178 osely related to human herpesvirus 8 (HHV-8)/Kaposi's Sarcoma-associated herpesvirus (KSHV), and it i
179 luding human Epstein-Barr virus (EBV), human Kaposi's sarcoma-associated herpesvirus (KSHV), and muri
180 lular reservoir in individuals infected with Kaposi's sarcoma-associated herpesvirus (KSHV), and the
181 iruses, including the human gammaherpesvirus Kaposi's sarcoma-associated herpesvirus (KSHV), and the
182 the replication of the oncogenic herpesvirus Kaposi's sarcoma-associated herpesvirus (KSHV), by targe
183 aherpesviruses, Epstein-Barr virus (EBV) and Kaposi's sarcoma-associated herpesvirus (KSHV), can caus
184 , an immediate-early and tegument protein of Kaposi's sarcoma-associated herpesvirus (KSHV), causes s
185 n adaptive immunity, many viruses, including Kaposi's sarcoma-associated herpesvirus (KSHV), have evo
187 by viral endoribonucleases, including SOX in Kaposi's sarcoma-associated herpesvirus (KSHV), muSOX in
188 egument proteins of herpesviruses, including Kaposi's sarcoma-associated herpesvirus (KSHV), play key
190 infection and KS.IMPORTANCE The ability for Kaposi's sarcoma-associated herpesvirus (KSHV), the caus
191 rld primates and are distinct but related to Kaposi's sarcoma-associated herpesvirus (KSHV), the etio
193 on lymphoma (PEL) cells infected with latent Kaposi's sarcoma-associated herpesvirus (KSHV), the prom
195 maherpesviruses Epstein-Barr virus (EBV) and Kaposi's sarcoma-associated herpesvirus (KSHV), which ar
199 n by HIV-1 promotes the aggressive growth of Kaposi's sarcoma-associated herpesvirus (KSHV)-related m
202 shown that, unlike glucose-addicted cancers, Kaposi's sarcoma-associated herpesvirus (KSHV)-transform
203 ies.IMPORTANCE We have previously shown that Kaposi's sarcoma-associated herpesvirus (KSHV)-transform
211 to recognize late gene promoters.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV; human her
212 16) recognizes nuclear episomal herpesvirus (Kaposi's sarcoma-associated herpesvirus [KSHV], Epstein-
214 ich tumor cells are consistently infected by Kaposi's sarcoma-associated herpesvirus and usually grow
215 c mechanisms by which Epstein-Barr virus and Kaposi's sarcoma-associated herpesvirus cause B-cell lym
216 tors (e.g., herpes simplex virus nectin1 and Kaposi's sarcoma-associated herpesvirus EphA2), or assoc
221 s, the cofactor activity (CFA), by using the Kaposi's sarcoma-associated herpesvirus RCA homolog Kapo
222 t HIV, together with a transient increase in Kaposi's sarcoma-associated herpesvirus viral load at we
223 ciated polyomavirus, Epstein-Barr virus, and Kaposi's sarcoma-associated herpesvirus, because GEMs ha
224 ovirus (HCMV), Epstein-Barr virus (EBV), and Kaposi's sarcoma-associated herpesvirus, establish laten
225 genomes, those of human cytomegalovirus and Kaposi's sarcoma-associated herpesvirus, revealing trans
226 nflammatory activities observed in MCD-like "Kaposi's sarcoma-associated herpesvirus-induced cytokine
227 EL clinical samples, and three extracavitary Kaposi's sarcoma-associated herpesvirus-positive solid l
235 omolog, the oncogenic human gammaherpesvirus Kaposi's sarcoma-associated herpesvirus/human herpesviru
236 ed for latent replication and persistence of Kaposi's sarcoma-associated herpesvirus/human herpesviru
238 This study defines the mechanism by which Kaposi's sarcoma could be maintained by virus constantly
241 rt to block the interaction between vFLIP, a Kaposi's sarcoma herpesviral oncoprotein, and NEMO using
242 In lymphomas the Epstein-Barr virus (EBV), Kaposi's sarcoma herpesvirus (KSHV) and human T-lymphotr
243 ressed from the Epstein-Barr virus (EBV) and Kaposi's sarcoma herpesvirus (KSHV) human DNA tumor viru
244 mutant with prototype foamy virus (PFV) and Kaposi's sarcoma herpesvirus (KSHV) tethering sequences
245 fied infections by Epstein-Barr virus (EBV), Kaposi's sarcoma herpesvirus (KSHV), and human T-lymphot
248 ow that HHV-8, a DNA tumor virus that causes Kaposi's sarcoma, infects three types of dendritic cells
253 Therefore, an understanding of the burden of Kaposi's sarcoma (KS) and non-Hodgkin lymphoma (NHL) rel
255 herpesvirus (KSHV) is the etiologic agent of Kaposi's sarcoma (KS) and primary effusion lymphoma (PEL
256 virus (KSHV)-related malignancies, including Kaposi's sarcoma (KS) and primary effusion lymphoma (PEL
258 several AIDS-related malignancies, including Kaposi's sarcoma (KS) and primary effusion lymphoma (PEL
259 ked to several human malignancies, including Kaposi's sarcoma (KS) and primary effusion lymphoma (PEL
262 is etiologically associated with endothelial Kaposi's sarcoma (KS) in immunocompromised individuals.
269 herpesvirus (KSHV) is the etiologic agent of Kaposi's sarcoma (KS), a vascular tumor frequently found
270 herpesvirus (KSHV) is the etiologic agent of Kaposi's sarcoma (KS), an AIDS-defining cancer in HIV-1-
271 herpesvirus (KSHV) is the causative agent of Kaposi's sarcoma (KS), an AIDS-defining cancer with abno
272 of care for patients with HIV diagnosed with Kaposi's sarcoma (KS), but the current role of systemic
273 irus (KSHV) is etiologically associated with Kaposi's sarcoma (KS), primary effusion lymphoma (PEL),
274 d herpesvirus (KSHV) is the causal agent for Kaposi's sarcoma (KS), the most common malignancy in HIV
275 d herpesvirus (KSHV) is the causal agent for Kaposi's sarcoma (KS), the most common malignancy in peo
276 d herpesvirus (KSHV), the causative agent of Kaposi's sarcoma (KS), to establish and maintain latency
277 ssociated herpesvirus (KSHV) is the cause of Kaposi's sarcoma (KS), which is the most common AIDS-ass
281 s to block KSHV virion production.IMPORTANCE Kaposi's sarcoma (KS)-associated herpesvirus is the caus
289 e infected with human herpesvirus 8 (HHV-8) (Kaposi's sarcoma [KS]-associated herpesvirus) and have a
290 ual men were over-represented among men with Kaposi's sarcoma (OR, 48.2; 95% CI, 22.0 to 105.6), anal
291 that are characteristic of HHV-8-associated Kaposi's sarcoma, PEL and multicentric Castleman's disea
292 nd angiogenic activities to HHV-8-associated Kaposi's sarcoma, primary effusion lymphoma (PEL), and m
293 ting it as a contributor to virus-associated Kaposi's sarcoma, primary effusion lymphoma (PEL), and m
294 giogenic viral proteins, in HHV-8-associated Kaposi's sarcoma, primary effusion lymphoma (PEL), and m
295 lated diagnoses, oesophageal candidiasis and Kaposi's sarcoma, rose from almost zero to very high lev
297 rs.IMPORTANCE KSHV is the etiologic agent of Kaposi's sarcoma, the most common tumor of AIDS patients
299 decreased significantly across 1996-2012 for Kaposi's sarcoma, two subtypes of non-Hodgkin lymphoma,