ライフサイエンスコーパス: L-cysteine

1 ctions: the adenylation and S-methylation of l-cysteine.

2 tive oxygen species (ROS) scavenger N-acetyl-L-cysteine.

3 vity by using the antioxidant agent N-acetyl-L-cysteine.

4 tcyP causes the accumulation of cytoplasmic l-cysteine.

5 ctrocatalytic oxidation and determination of L-cysteine.

6 lls with the free radical scavenger N-acetyl-L-cysteine.

7 feature of SAT is its feedback inhibition by L-cysteine.

8 nyleneiodonium, or the antioxidant, N-acetyl-L-cysteine.

9 f O-acetyl-L-serine (OAS) by a thiol to give L-cysteine.

10 e and upon changing the protonation state of l-cysteine.

11 eas an NRPS EpoB carrier protein contributes l-cysteine.

12 NaHS (0.1-100 microM) or l-cysteine (0.1-100 microM) significantly increased plat

13 ietary protein content (14% protein and 0.3% L-cysteine-0.9% L-arginine, or 24% protein and 0.5% L-cy

14 The ACV analogue delta-L-alpha-aminoadipoyl-L-cysteine (1-(S)-carboxy-2-thiomethyl)ethyl ester (ACOm

15 on was inhibited by endogenous H2S precursor l-cysteine (10 mm) but not by l-serine (10 mm) or either

16 arboxypropyl]carbamoyl]-4-(18)F-fluorobenzyl-L-cysteine ((18)F-DCFBC) could image an experimental mod

17 arboxypropyl]carbamoyl]-4-(18)F-fluorobenzyl-L-cysteine ((18)F-DCFBC), a low-molecular-weight radiotr

18 arboxypropyl]carbamoyl]-4-(18)F-fluorobenzyl-l-cysteine ((18)F-DCFBC), to detect metastatic hormone-n

19 ine-0.9% L-arginine, or 24% protein and 0.5% L-cysteine-2.0% L-arginine), dietary antioxidant content

20 lites in urine--N-acetyl-S-(3-hydroxypropyl)-L-cysteine (3HPMA) and N-acetyl-S-(2-carboxyethyl)-L-cys

21 N-(Acyl)-1H-benzotriazoles 6a-f react with l-cysteine 5 at 20 degrees C to give exclusively (i) N-a

22 mine in CH(3)CN-H(2)O (3:1), but (ii) S-acyl-l-cysteines 7a-e in CH(3)CN-H(2)O (5:1) in the absence o

23 20 degrees C to give exclusively (i) N-acyl-l-cysteines 8a-e in the presence of triethylamine in CH(

24 nts intracellular acidification) and N-acety-l-cysteine (a scavenger of ROS).

25 Inclusion of N-acetyl-L-cysteine, a known antioxidant and NF-kappaB inhibitor,

26 hylated) to that observed in the presence of l-cysteine, a ligand facilitating rapid Hg(II) biouptake

27 Treatment with N-acetyl-l-cysteine, a potent antioxidant, abolished lipid peroxi

28 ffective CysSSH synthesis from the substrate L-cysteine, a reaction catalyzed by prokaryotic and mamm

29 sembled monolayer of zwitterionic amino acid l-cysteines acts like a brush layer to prevent SERS-hots

30 QDs emission was observed in the presence of L-cysteine after 2 hours.

31 ls with intracellular ROS scavenger N-acetyl-l-cysteine also inhibits AGP-induced activation of ASK1,

32 d NOTCH1 expression, and the use of N-acetyl-L-cysteine altered NOTCH1 expression, suggesting that th

33 first time that in the presence of iron and L-cysteine, an [Fe(4)S(4)] cluster is formed within the

34 at the HydG auxiliary cluster must bind both l-cysteine and a dangler Fe in order to generate the [Fe

35 naling pathways, as the antioxidant N-acetyl-l-cysteine and a Syk inhibitor differentially blocked he

36 Also, the L-cysteine and Amplex Red system was implemented as an a

37 mine as well as sulfhydryl compounds such as l-cysteine and beta-mercaptoethanol as cosubstrates.

38 Pretreatment with N-acetyl-l-cysteine and catalase expression ameliorated cell deat

39 this, combinatorial treatment with N-acetyl-l-cysteine and catalase substantially inhibited the ROS

40 Similarly, combined N-acetyl-l-cysteine and catalase treatment also suppressed VDAC1-

41 te solution via reactive H2S generation from l-cysteine and controls nanocrystal growth within the qu

42 N-acetyl-L-cysteine and cyclosporine A, blocked ethanol and aceta

43 The thiol-containing molecules l-cysteine and d-cysteine both mimicked the protective e

44 reduced glutathione, L-cysteine or N-acetyl-L-cysteine and fully reduced by dithiothreitol or the E.

45 of soy lecithin, salt, and reducing agents (l-cysteine and glutathione).

46 attenuated by thiol-containing nucleophiles, l-cysteine and glutathione.

47 Preincubation with S-allyl-l-cysteine and isoliquiritigenin increased MMP in both P

48 rivatives as well as the beta-elimination of l-cysteine and its analogs.

49 bited dramatic accumulation of intracellular l-cysteine and its oxidized dimer, l-cystine, and deplet

50 domain catalyzes C-S bond cleavage by using l-cysteine and l-cysteine S-modified analogs as substrat

51 can generate these metabolites from N-acetyl-l-cysteine and l-cysteine, respectively, and that the sy

52 l-Cysteine and l-Cystine were determined with detection

53 dation to component amino acids l-glutamate, l-cysteine and l-glycine.

54 is study examined the ability of l-arginine, l-cysteine and l-methionine, to inhibit postharvest sene

55 N-acetyl-l-cysteine and mito-TEMPO blocked the induction of IL-1b

56 When H(2)S-releasing compounds L-cysteine and N-acetylcysteine were added to the cell c

57 Similarly, l-cysteine and N-ethylmaleimide significantly attenuated

58 SE) produces H2S via enzymatic conversion of L-cysteine and plays a critical role in cardiovascular h

59 ipid Mixture 1, Gelatin Peptone N3, N-Acetyl-L-Cysteine and Pluronic F-68) were assayed in order to i

60 or was found to be ascorbic acid followed by L-cysteine and quercetin.

61 Two main precursors (S-3-(hexan-1-ol)-l-cysteine and S-3-(hexan-1-ol)-l-glutathione) of 3-sulf

62 h previous sample treatment using 0.1% (w/v) l-cysteine and the concentration of Sb(V) species was de

63 n module, involving the aerobic oxidation of l-cysteine and the concomitant formation of the fluoresc

64 The ROS scavenger N-acetyl L-cysteine and the mitochondrial antioxidant Mito-TEMPO

65 nhancer solution (FES) (d-glucose, d-ribose, l-cysteine and thiamin) and of sous-vide cooking or roas

66 the metal surface, WF modifying components, l-cysteine and Zn(OH)(2) , were incorporated within the

67 was highly active with l-cysteine, N-acetyl-l-cysteine, and allyl mercaptan.

68 cysteine (PPC) from (R)-phosphopantothenate, l-cysteine, and cytidine-5'-triphosphate (CTP) and has b

69 strate-binding protein FliY binds l-cystine, l-cysteine, and d-cysteine with micromolar affinities.

70 ubstrates, S-methyl-L-cysteine sulfoxide and L-cysteine, and had both cysteine sulfoxide lyase and cy

71 lease, further enhanced by the IAP inhibitor l-cysteine, and reduced by the exogenous ATPase apyrase.

72 bic acid, sodium diethyldithiocarbamic acid, L-cysteine, and sodium metabisulfite were effective inhi

73 the quantitative reaction of maleimide with l-cysteine, and the subsequent determination of the unre

74 Pretreatment with antioxidant N-acetyl-l-cysteine, apoptosis signal-regulating kinase 1 (ASK1)

75 In Turkey, usage of l-Cysteine as a food additive isn't allowed in wheat flo

76 Previously, we identified S-trityl-L-cysteine as a selective inhibitor of Eg5 and developed

77 dent beta-elimination reaction, establishing l-cysteine as the origin of sulfur at C-3 of LNM; and (i

78 ttled mineral water samples using 2.0% (w/v) l-cysteine, as well as by analysis of certified material

79 including glycine, L-serine, L-alanine, and L-cysteine, as well as their D-enantiomers.

80 allic acid, glycine, reduced glutathione and l-cysteine at 0.1, 0.5, 1.0 and 2.0% levels to inhibit b

81 The electrochemical behavior of L-cysteine at the Au-SH-SiO(2)@Cu-MOF was investigated b

82 We report the development of a novel L-cysteine-based poly (disulfide amide) (Cys-PDSA) famil

83 nhibition of arginase with S-(2-boronoethyl)-l-cysteine (BEC) significantly enhanced NO generation in

84 emonstrated by sensitivity of acute 1-NCA to l-cysteine but not to hydroxocobalamin in the normal rat

85 transferred to IscS only in the presence of l-cysteine, but not to the C328S variant.

86 We show here that L-cysteine can be used to transform a highly reactive Si

87 ve composite material consisting of N-acetyl-L-cysteine capped CdAgTe quantum dots (NAC-CdAgTe QDs) a

88 l-cysteine capped CdS QDs/DF20 aptamer bioconjugates wer

89 ce sensor is developed to detect DA based on l-cysteine capped Mn doped ZnS quantum dots (l-cys ZnS:M

90 L-cysteine capped Mn doped ZnS quantum dots/ Idarubicin

91 ize of CTAB capped TiO(2), MoS(2)@TiO(2) and L-Cysteine capped MoS(2)@TiO(2) as 12.6, 11.7 and 10.2 n

92 by immobilizing the aptamer on water soluble l-cysteine capped ZnS quantum dots (QDs).

93 ated to the in situ synthesis of amino acid (L-Cysteine) capped lanthanum hydroxide nanoparticles [Cy

94 system using resonance energy transfer with l-cysteine-capped cadmium telluride quantum dots (CdTe-Q

95 The l-cysteine-capped CdTe-QDs were synthesized and added in

96 s achieved due to the energy transfer by the l-cysteine-capped CdTe-QDs.

97 addition, Na2S, the H2S donor GYY-4137, and l-cysteine caused relaxation of airways pre-contracted w

98 eine (3HPMA) and N-acetyl-S-(2-carboxyethyl)-L-cysteine (CEMA)--as biomarkers of exposure to acrolein

99 atalytic current increased linearly with the L-cysteine concentration in the range of 0.02-300 muM an

100 plying it to the analytical determination of L-cysteine concentration.

101 estational day 20, maternal plasma urate and L-cysteine concentrations, and placental levels of 4-hyd

102 n contrast, the efficacy of S-3-carboxpropyl-l-cysteine (CPC) a new inhibitor described herein, was n

103 of new biosensor for the electrooxidation of l-cysteine (CSH) in aqueous media.

104 address these questions, we herein show that L-cysteine (Cys) binds the auxiliary [4Fe-4S] cluster of

105 Gold disc electrodes functionalised with a l-Cysteine (Cys) self-assembled monolayer (SAM) were use

106 d HClO4 media, in the presence or absence of L-cysteine (Cys).

107 be used in the electrocatalytic oxidation of L-cysteine (Cys).

108 The thiol precursors S-3-(hexan-1-ol)-l-cysteine (Cys-3MH) and S-3-(hexan-1-ol)-l-glutathione

109 isted amino acids such as L-Lysine (lysine), L-Cysteine (cysteine) and L-Arginine (arginine) as bifun

110 Au nanoparticles modified with l-cysteine (d-cysteine) selectively adsorb the (R)-propy

111 The oxidation overpotentials of L-cysteine decreased significantly and their oxidation p

112 he arginase inhibitor BEC (S-(2-boronoethyl)-l-cysteine) decreased arginase activity and caused alter

113 of l-cystine and its subsequent reduction to l-cysteine depleted both NADPH and GSH pools, thereby al

114 E were markedly abolished by the antioxidant L-cysteine derivative N-acetylcysteine (NAC).

115 -MS coupled with o-phthaldialdehyde/N-acetyl-L-cysteine derivatization.

116 duced in the cytosol through the activity of l-cysteine desulfhydrase (DES1).

117 dopsis thaliana, DES1 is the only identified L-Cysteine desulfhydrase located in the cytosol, and it

118 The Escherichia coli L-cysteine desulfurase IscS mobilizes sulfur from L-cyst

119 studies showed that TUM1 interacts with the l-cysteine desulfurase NFS1 and the rhodanese-like prote

120 l-Cysteine desulfurases provide sulfur to several metabo

121 IscS is one of the three Escherichia coli l-cysteine desulfurases.

122 Gallic acid and l-cysteine did not exhibit browning inhibition effect at

123 By using S-nitroso-l-cysteine-ethyl-ester, an intracellular NO donor and in

124 cydD phenotype, unlike that of the bona fide l-cysteine exporter eamA, parallels that of the l-cystin

125 cer of cytoplasmic cystine, as opposed to an l-cysteine exporter, and further elucidate a link betwee

126 the thiol-lacking molecule N-acetyl-S-methyl-l-cysteine failed to exert protection or blunt the rise

127 on paste electrode modified with ds-DNA/poly(L-cysteine)/Fe3O4 nanoparticles-graphene oxide (ds-DNA/p

128 he formation of the CS-conjugates S-furfuryl-l-cysteine (FFT-S-Cys) and S-(2-methyl-3-furyl)-l-cystei

129 Blocking arginases using S-(2-boronoethyl)-L-cysteine for 9 weeks in Ins2(Akita) mice or 6 weeks in

130 by depositing gold nanoparticles followed by L-cysteine for obtaining a self-assembled monolayer.

131 teine desulfurase IscS mobilizes sulfur from L-cysteine for the synthesis of several biomolecules suc

132 S-starvation suggests that GGCT2;1 mobilizes l-cysteine from glutathione when there is insufficient s

133 fhydrylase, which catalyzes the formation of l-cysteine from O-acetyl serine and hydrogen sulfide.

134 idase, the CydDC complex, shuttles excessive l-cysteine from the cytoplasm to the periplasm, thereby

135 To break the stalemate, N-acetyl-L-cysteine functionalized chitosan copolymer (CS-NAC), w

136 trate based on graphene oxide (GO)-supported l-cysteine-functionalized starlike gold nanoparticles (S

137 to-4-methylpentan-2-one-N-(l-gamma-glutamyl)-l-cysteine (gammaGluCys-4MMP) but at too low concentrati

138 rsatile sensing method for analyzing thiols (L-cysteine, glutathione) using the H2O2-mediated DNAzyme

139 iberately added to various flour types since l-Cysteine has enabled favorable baking conditions such

140 ectrocatalytic activity for the oxidation of L-cysteine in 0.1 M phosphate buffer solution (pH 5.0).

141 rkedly increased in response to both NaHS or l-cysteine in platelets of healthy volunteers.

142 The L-cysteine in situ capped cadmium sulfide (CdS; size < 7

143 oylcysteine from (R)-phosphopantothenate and L-cysteine in the biosynthetic pathway leading to the fo

144 Finally, the sensor was applied to determine L-cysteine in water and biological samples.

145 ective method was developed for detection of l-Cysteine in wheat flour using Raman microscopy.

146 Detection of l-Cysteine in wheat flour was accomplished successfully

147 urgent need for effective methods to detect l-Cysteine in wheat flour.

148 While both monastrol and S-trityl-L-cysteine inhibit Eg5 motility, our data reveal an unex

149 N-acetyl-L-cysteine inhibited BPEx-induced eosinophilic airway in

150 ures catalyze the oxidation of thiols (e.g., l-cysteine) into disulfides (e.g., cystine).

151 N-acetyl-S-(4-hydroxy-2-methyl-2-buten-1-yl)-l-cysteine (IPM3) in participants (>=3 year old) from th

152 l-Cysteine is deliberately added to various flour types

153 l-Cysteine is effective in mediating release of free Fe(

154 Thus, l-cysteine is not removed from the system and the fluore

155 ence response of the dyes in the presence of L-cysteine is slow, but a ratiometric detection process

156 l-cysteine is the source of all bacterial sulfurous biom

157 mixture was separately further modified with l-cysteine (l-Cys) and l-serine (l-Ser).

158 L-cysteine (L-cys) increases the amplitude of T-type Ca(

159 o the surface of GC/PPy-AuNPs-rGO film using L-cysteine (L-Cys) linker agent and trifluoromethanesulf

160 Under conditions of elevated ROS, endogenous L-cysteine (L-Cys) production is insufficient for GSH sy

161 celerated rate of reduction of aquometHbE by L-cysteine (L-Cys).

162 e, L-leucine, glycine, L-lysine, L-arginine, L-cysteine, L-alanine, and L-proline--in aqueous solutio

163 selenium: seleno-l-methionine, methyl-seleno-l-cysteine, l-selenocystine, methaneseleninic acid, sele

164 racellular thiol species (thioglycolic acid, l-cysteine-l-glycine, cysteine, and glutathione) were up

165 ls)) on monolayers of l-cysteine-l-tyrosine, l-cysteine-l-phenylalanine, or l-cysteine-l-phosphotyros

166 e-l-tyrosine, l-cysteine-l-phenylalanine, or l-cysteine-l-phosphotyrosine formed on crystallographica

167 nd fluorinated terphenyls)) on monolayers of l-cysteine-l-tyrosine, l-cysteine-l-phenylalanine, or l-

168 ar retention of NS mutants, whereas N-acetyl-L-cysteine largely prevented the effects of MG132.

169 l hypothesis that reduced levels of H(2)S or L-cysteine (LC) play a role in the impaired glucose meta

170 omocysteine (100 to 500 micromol/L), but not l-cysteine, maintained the Ly-6C(hi) subset and induced

171 Interestingly, the antioxidant N-acetyl-L-cysteine markedly reversed these changes.

172 Additional studies indicate that L-cysteine may have an important role in repairing the N

173 c hemin/G-quadruplex chains that control the l-cysteine-mediated aggregation of Au NPs.

174 ose a model to explain the interplay between l-cysteine metabolism, H2S production, and oxidative str

175 roduced from either H(2) S oxidation or from L-cysteine metabolism.

176 l-5'-phosphate-dependent enzymes involved in L-cysteine metabolism: cystathionine-ss-synthase (CBS) a

177 umber of amino acids (threonine, tryptophan, l-cysteine, methionine, cycloleucine, aspartic acid, asp

178 ysteine (FFT-S-Cys) and S-(2-methyl-3-furyl)-l-cysteine (MFT-S-Cys) in the Maillard reaction of xylos

179 l-Cysteine mobilization from glutathione is not solely a

180 1.7 x 10(6) was estimated for a monolayer of L-cysteine molecules adsorbed to gold via a thiol linkag

181 owed by linking of monomeric (N-methacryloyl-l-cysteine) molecules through S-Au bonds.

182 However, the cytoplasmic level of l-cysteine must be tightly regulated due to its propensi

183 ; however, the enzyme was highly active with l-cysteine, N-acetyl-l-cysteine, and allyl mercaptan.

184 by the living polymerization of glycosylated L-cysteine-N-carboxyanhydride (glyco-C NCA) monomers.

185 metabolized to N-acetyl-S-1,2-dichlorovinyl-L-cysteine (NA-DCVC) before being excreted in the urine.

186 Neutralization of ROS by N-acetyl-l-cysteine (NAC) abrogated the phosphorylation of JAK2 a

187 n that involves derivatization with N-acetyl-l-cysteine (NAC) and separation by HPLC was developed.

188 Pretreatment with N-acetyl-L-cysteine (NAC) and vitamin E (Trolox) were used to inv

189 N-acetyl-l-cysteine (NAC) exhibits protective properties in brain

190 ROS scavengers edaravone (EDA) and N-acetyl-L-cysteine (NAC) reduced ROS level, downregulated JNK ac

191 -treated mothers, while antioxidant N-acetyl-L-cysteine (NAC) reversed these changes together with im

192 The antioxidant N-acetyl-l-cysteine (NAC) significantly reduces the cytotoxicity

193 In contrast, N-acetyl-L-cysteine (NAC) treatment in H9c2 cells, or overexpress

194 On the other hand, treatment with N-acetyl-l-cysteine (NAC), an oxygen free radical scavenger, led

195 our reductants, namely, GSH, l-Cys, N-acetyl-l-cysteine (NAC), and ascorbic acid.

196 h an oxygen free radical scavenger, N-acetyl-l-cysteine (NAC), attenuated the FoxO1 inhibition-induce

197 tor LY294002, glutathione precursor N-acetyl-L-cysteine (NAC), curcumin, epigallocatechin-3 gallate (

198 ficacy of a weak organic acid drug, N-acetyl-L-cysteine (NAC), on the eradication of biofilms formed

199 aOAc) buffer containing 5 mg.mL(-1) n-acetyl-l-cysteine (NAC), or 0.25 M NaOAc containing 5 mg.mL(-1)

200 igarette smoke extract, antioxidant N-acetyl-l-cysteine (NAC), or both.

201 d by increasing the thiol pool with N-acetyl L-cysteine (NAC), while NAC had little effect on BAA.

202 Thiol antioxidants (N-acetyl-l-cysteine [NAC] and N-acystelyn, carbocysteine, erdoste

203 CD) and oxidative stress inhibitor (N-acetyl-L-cysteine, NAC) slightly rescued the viability of cells

204 TCVC), and N-acetyl-S-(1,2,2-trichlorovinyl)-L-cysteine (NAcTCVC) was quantified in serum, liver, and

205 USP method to that of the standard N-acetyl-L-cysteine-NaOH (NALC) method for conventional diagnosis

206 emulsion polymerization of S-(o-nitrobenzyl)-l-cysteine (NBC) N-carboxyanhydride (NCA).

207 The antioxidant N-acetyl-L-cysteine normalized ROS levels and restored ICaL densi

208 ving activation, methylation, and loading of l-cysteine onto the TioN partner thiolation domain, TioS

209 and injected with the antioxidants, N-acetyl-L-cysteine or dimethylthiourea, prior to sensitization,

210 d by monothiols such as reduced glutathione, L-cysteine or N-acetyl-L-cysteine and fully reduced by d

211 was attenuated by the antioxidant, N-acetyl-L-cysteine, or Cu sequestration.

212 rginine, the free radical scavenger N-acetyl-l-cysteine, or the NOS substrate l-arginine partially in

213 dase inhibitor DPI, the antioxidant N-acetyl-L-cysteine, or the superoxide scavenger Tiron, further i

214 y by d-cysteine-bound FliY, and maximally by l-cysteine- or l-cystine-bound FliY.

215 ssessing the S-[2,3-bis(palmitoyloxy)propyl]-l-cysteine (Pam(2)Cys) motif and exhibit potent immunost

216 the biologically active persulfide (N-acetyl l-cysteine persulfide, NAC-SSH) in a spatiotemporal mann

217 l-Cysteine predominantly inhibits priming but not the ac

218 The antioxidant N-acetyl-l-cysteine prevented mitochondrial inhibitors from rescu

219 Pretreatment with the antioxidant N-acetyl-L-cysteine prevented ouabain-stimulated Na/K-ATPase.c-Sr

220 ocyanatostilbene-2,2'-disulfonate or N-acety-l-cysteine prevented the acid-induced increase in phosph

221 verloaded mice with the antioxidant N-acetyl-L-cysteine prevented the development of trabecular but n

222 eriments, CDO exhibits an ordered binding of l-cysteine prior to NO (and presumably O2) similar to th

223 embers of the PLP-II family, and involved in L-cysteine production.

224 nt inhibitor of the major secreted cathepsin L cysteine proteases of F. hepatica, FhCL1 and FhCL2, an

225 Pretreatment with the antioxidant N-acetyl-L-cysteine protected embryos with activated hypoxia sign

226 The prepared Au(L-cysteine)-Pt(penicillinase) nanowire array electrode s

227 m and to obtain the stoichiometry factor for l-cysteine quantification.

228 centration range and drastically different d/l-cysteine ratios in simulated body fluids.

229 N-Acetyl-l-cysteine reduced oxidative stress, prevented CCTalpha

230 e reactive oxygen species scavenger N-acetyl-l-cysteine reduced the levels of interleukin-6, interleu

231 iated by peroxidase activity (oxidation) and l-cysteine (reduction).

232 -deficient T cells with metabolically active L-cysteine rescued mTOR activation and proliferation but

233 and show that the IgG2 C H1 and C-terminal C L cysteine residues are either linked to each other or t

234 ns in its peptidic backbone two S-methylated l-cysteine residues.

235 ese metabolites from N-acetyl-l-cysteine and l-cysteine, respectively, and that the synthesis of stea

236 microM for CTP, (R)-phosphopantothenate, and L-cysteine, respectively, and the k(cat) was 2.9 s(-1).

237 K inhibitor PD98059 and antioxidant N-acetyl-l-cysteine restored normal proliferation of Atm(-/-) ast

238 3COOH or TRIS buffer after prereduction with l-cysteine resulted in the formation of only the corresp

239 gly, treatment with the antioxidant N-acetyl-l-cysteine reversed the phenotype, normalizing atheroscl

240 Also, the cell-permeable thiol N-acetyl l-cysteine, reverses DMF inhibition of the NFkappaB path

241 es C-S bond cleavage by using l-cysteine and l-cysteine S-modified analogs as substrates through a PL

242 ) was previously proposed to oxidize S-allyl-l-cysteine (SAC) to alliin, an allicin precursor.

243 igated the neuroprotective effect of S-allyl L-cysteine (SAC), a water soluble compound from garlic,

244 The l-cysteine SAuNPs@GO hybrid exhibited 80.57% lower BSA f

245 erum albumin (BSA) as a protein model on the l-cysteine SAuNPs@GO hybrid substrate (the test) and a c

246 Au nanoparticles modified with either d- or l-cysteine selectively adsorb one enantiomer of propylen

247 Among the chemical inhibitors tested, l-cysteine showed the best inhibitory effect, with an IC

248 luded chemical decontamination with N-acetyl-l-cysteine-sodium hydroxide (NALC-NaOH), alone and in co

249 s on initial sample processing with N-acetyl-l-cysteine-sodium hydroxide (NALC-NaOH), chemicals that

250 N-Acetyl-l-cysteine-sodium hydroxide (NALC-NaOH)-decontaminated s

251 ork, water-dispersible N-acetylcysteine- and l-cysteine-stabilized palladium nanoparticles are introd

252 For example, S-trityl-l-cysteine (STC) and monastrol are HsEg5 inhibitors that

253 As l-cysteine stimulates the aggregation of Au nanoparticle

254 We previously identified S-trityl-l-cysteine (STLC) and related analogues as selective pot

255 We previously identified S-trityl-L-cysteine (STLC) as a potent allosteric inhibitor of Eg

256 ed by deletion of L5 or addition of S-trityl-l-cysteine (STLC), an allosteric inhibitor that binds to

257 ated with the kinesin Eg5 inhibitor S-Trityl-l-cysteine (STLC).

258 SufE enhances the initial l-cysteine substrate binding to SufS and formation of th

259 t by growth with exogenous dithiothreitol or L-cysteine, suggesting that in the absence of CccA, apoc

260 potently inhibited by nickel and enhanced by L-cysteine, suggesting that the Ca(V)3.2 T-type channel

261 ding kinetics of two alternative substrates: L-cysteine sulfinic acid and d-aspartate.

262 L-cysteine sulfinic acid bound with higher affinity than

263 lliinase and its natural substrate, S-benzyl-l-cysteine sulfoxide (petiveriin).

264 ed to bind two kinds of substrates, S-methyl-L-cysteine sulfoxide and L-cysteine, and had both cystei

265 lly occurring P. alliacea compounds S-benzyl-l-cysteine sulfoxide and S-2-hydroxyethyl-l-cysteine sul

266 n energy with its natural substrate S-benzyl-l-cysteine sulfoxide is 64.6 kJ mol(-1).

267 yl-l-cysteine sulfoxide and S-2-hydroxyethyl-l-cysteine sulfoxide.

268 Our results show that l-cysteine supplementation is a potential treatment for

269 PYK2 downregulation, l-cysteine supplementation, or CSE overexpression allevi

270 en there is insufficient sulfate for de novo l-cysteine synthesis.

271 rovinyl)GSH (TCVG), S-(1,2,2-trichlorovinyl)-L-cysteine (TCVC), and N-acetyl-S-(1,2,2-trichlorovinyl)

272 Pretreatment with the ROS scavenger N-acetyl-L-cysteine, the ERK1/2 inhibitor UO126, or ERK1/2 siRNA

273 N-acetyl-L-cysteine therapy has been used in clinical studies; ho

274 , we explore the effect of low dose N-acetyl-L-cysteine therapy, delivered using a targeted, systemic

275 nd either NO or a suitable reductant such as L-cysteine, these ferric heme sites can function as a ge

276 d does not catalyze the aerobic oxidation of l-cysteine to cystine.

277 e ATP-dependent condensation of GlcN-Ins and l-cysteine to form l-Cys-GlcN-Ins, the penultimate step

278 OM-sulfide solutions amended with sufficient l-cysteine to prevent beta-HgS(s) formation.

279 ta'-position (2b, 2c) have been treated with L-cysteine to provide thiazolidine derivatives.

280 nanoparticles have been modified with d- or l-cysteine to render them chiral and enantioselective fo

281 , administration of the antioxidant N-acetyl-l-cysteine to Ucp2(-/-) pregnant mice alleviated the eff

282 ecting two different analytes: d-glucose and l-cysteine under nonspecific and specific adsorption con

283 The loading capacity determined for l-cysteine using frontal elution is 2.58 mumol/m.

284 rly p-aminobenzenesulfonic acid (p-ABSA) and l-cysteine using intensive oxidative conditions, and att

285 ed glycosides of D-galactose or D-glucose to L-cysteine using thiol-ene "click" chemistry, followed b

286 rotects E. coli against oxidative stress via l-cysteine utilization and H2S-mediated sequestration of

287 OASS produces L-cysteine via a de novo pathway while CBS participates

288 azole functionality from an acetyl group and l-cysteine via condensation, cyclization, and dehydratio

289 Mass balances and quantification of l-cysteine via its sulfur concentration using elemental

290 l-Cysteine was the most effective inhibitor.

291 ithin anodic aluminum oxide (AAO) membranes; L-cysteine was used to form a monolayer on the Au segmen

292 of the PPCS-catalyzed reaction with CTP and L-cysteine, was monitored by (31)P NMR spectroscopy.

293 on systems, the thiol-containing amino acid, L-Cysteine, was self-assembled onto smooth gold electrod

294 eads to the increased level of extracellular l-cysteine, whereas induction of cydDC or tcyP causes th

295 x between the enzyme-persulfide and a second l-cysteine, which adds to the cofactor via its sulfhydry

296 -acetamido-2-deoxy-alpha-D-galactopyranosyl)-L-cysteine, which could be considered as a mimic of Tn a

297 Supplementation with l-cysteine, which is required for optimal TRMU function,

298 fE work together to mobilize persulfide from L-cysteine, which is then donated to the SufB Fe-S clust

299 The reactions of L-serine and L-cysteine with CBS resulted in the formation of a commo

300 The reaction of l-cysteine with the Ellman's reagent was monitored photo