ライフサイエンスコーパス: LBM

1 LBM can be obtained automatically from limited-whole-bod

2 LBM derived from limited-whole-body data using the propo

3 LBM of the whole body could be automatically estimated f

4 LBM provides an opportunity for discovering the neurocom

5 LBM was also calculated using predictive equations that

6 LBM was assessed by anthropometry.

7 LBM was measured by total-body dual-energy x-ray absorpt

8 +/- 2, 11 +/- 2, and 15 +/- 2 pmol x kg(-1) LBM x min(-1), respectively (P < 0.001, baseline vs. day

9 8 environmental samples were collected at 10 LBM.

10 In men receiving ADT, LBM decreased significantly after 12, 24, and 36 months.

11 of early nutrition supplementation on adult LBM and muscle strength.

12 level had a positive association with adult LBM and muscle strength.

13 ettings for stage III colon cancer, using an LBM-based oxaliplatin dose significantly reduces OIPN an

14 changes in body fat (r = 0.39, P = 0.01) and LBM (r = -0.34, P = 0.03).

15 inear regression model adjusting for age and LBM, an increase in DES excretion of 1 microg/g creatini

16 luate the effects of population ancestry and LBM on measures of excess adiposity (BMI, %BF, and FMI).

17 were associated with changes in body fat and LBM, but did not explain much variance in either measure

18 e examined the hypothesis that higher FM and LBM are associated with greater survival in MHD patents

19 es that account for variation in both FM and LBM are recommended for normalization of EE in mice.

20 ory markers, the highest quartiles of FM and LBM were associated with greater survival in women: HRs

21 ing for the independent assessment of FM and LBM.

22 e studied to define the link between IMF and LBM.

23 nificant differences in terms of FM loss and LBM maintenance.

24 ge of parameter space and bridges the MD and LBM scales using a relatively small number of MD calcula

25 C-reactive protein and LBM were inversely related (r = -0.71, p < 0.01).

26 This new methodology which couples SPT and LBM is validated using a kinetic Monte Carlo (kMC) algor

27 ce interval (CI): 1.16, 1.82), while TBF and LBM were not.

28 close phylogenetic proximity of some WS and LBM loNDV suggests that viral transmission may occur amo

29 restoring key interactions between DBMs and LBMs by using a computational model informed by coevolut

30 limited by incompatibility between DBMs and LBMs from different proteins, due to the loss of critica

31 n be due to incompatibility between DBMs and LBMs, as critical module-module interactions are lost af

32 or predicting compatibility between DBMs and LBMs.

33 ncy of visits to LBM, purchase of poultry at LBM for consumption) and contact of backyard chickens wi

34 The non-laboratory-based LBM, integrating wearable-based and anthropometric data,

35 Based on the movements of traders between LBMs, weighted and directed networks were constructed an

36 mononucleated (SMMs) and large binucleated (LBMs) myocytes.

37 iffusivity is computed by lattice Boltzmann (LBM) simulations.

38 t failed to detect class I viruses from both LBM and WS.

39 Larch budmoth (LBM) population dynamics across the European Alps, a cla

40 Thirty Vietnamese and 8 Cambodian LBMs were visited, and structured interviews were conduc

41 Consequently, LBMs with the largest combination of risk factors for be

42 volves identifying key residue pairs for DBM-LBM interactions by statistical analyses of coevolutiona

43 e-scarce settings with extensively developed LBM systems, commonly found in South-East Asia.

44 x-specific quantitative effects of different LBM formulations on liver SUV have not been well studied

45 0003; girls: +0.35 g/kg LBM, P = 0.003), EDV/LBM (boys: +0.35 ml/kg LBM, P < 0.0001; girls: +0.31 ml/

46 LV end-diastolic volume/lean body mass (EDV/LBM) for trained boys (+0.28 ml/kg LBM, P = 0.007) and a

47 FBM, LBM, and BMD were evaluated by dual-energy x-ray absorpt

48 provement (cut-off point: 7.5%) of 4.30% for LBM.

49 in the right hepatic lobe and corrected for LBM using the sex-specific James and Janmahasatian formu

50 diated shifting of the optimal elevation for LBM population growth is the mechanism for elevational e

51 H9N2-infected SPF chickens and chickens from LBM showed that pan-IAV FRET-PCR had a higher detection

52 The profiles of sellers operating at a given LBM could be reliably predicted using basic information

53 derestimated by %BF in individuals with high LBM.

54 percentiles" in both sexes, whereas a higher LBM was protective in women.

55 patients, higher FM in both sexes and higher LBM in women appear to be protective.

56 (HR = 2.44, 95% CI: 1.38, 4.34) and higher %LBM decreased risk of death (HR = 0.41, 95% CI: 0.23, 0.

57 incompressible flow is simulated with an IB-LBM which is validated by conducting two benchmark probl

58 mersed boundary-lattice Boltzmann method (IB-LBM).

59 Q-network (DRQN) is incorporated with the IB-LBM to train the fish model to adapt its motion to optim

60 the high computational efficiency of the IB-LBM which is of crucial importance for the effective cou

61 quickly and easily, may be used to identify LBMs suitable for implementing interventions.

62 was significantly greater than the change in LBM in the placebo group (mean change [SD], +1.9 [1.6] k

63 The threshold-like change in LBM outbreak pattern highlights how interacting species

64 Change in LBM was a significant predictor of serum ferritin (P < 0

65 Changes in LBM and regional lean mass were associated with changes

66 = 127) had significantly greater changes in LBM at all measured time points than younger men.

67 P =.002) were directly related to changes in LBM.

68 y (n = 36) had a greater rate of decrease in LBM compared with men who had received more than 6 month

69 erone therapy has been reported for gains in LBM and muscle strength.

70 months resulted in a significant increase in LBM associated with functional improvement in patients u

71 did not result in a significant increase in LBM.

72 n . kg(-1) . d(-1) in promoting increases in LBM and losses of fat mass when combined with a high vol

73 nificant nitrogen retention and increases in LBM, weight, and strength.

74 Pressure-permeability relationships in LBM transport were governed primarily by changes in diff

75 iation records reveals elevational shifts in LBM outbreak epicenters that coincide with temperature f

76 ons favorable for perpetuating HPAIV H5N1 in LBMs.

77 More than 80% of these traders operated in LBMs located in the most densely populated areas, Ha Noi

78 city of the current surveillance strategy in LBMs was not optimal leading to some false positive LBMs

79 rcentage lean body mass (%LBM) or change in %LBM in older subjects.

80 rogen excretion) but not with 3-y change in %LBM.

81 ids in healthy, aged women and men increased LBM and decreased fat mass.

82 yses of variance revealed that PRT increased LBM and ALM (P < 0.01); reduced trunk fat mass by 2.5 kg

83 As a result of the intervention, LBM increased (P < 0.05) in the PRO group (1.2 +/- 1.0 k

84 lean body mass (LBM)-1 . d-1 or >2.1 g . kg LBM-1 . d-1, respectively], each followed by an experime

85 Post-PHV, LV mass/LBM (boys: +0.50 g/kg LBM, P = 0.0003; girls: +0.35 g/kg LBM, P = 0.003), EDV/

86 +1.29 g/kg LBM, P = 0.015; girls: +1.47 g/kg LBM, P = 0.002) were all greater in trained versus untra

87 0.50 g/kg LBM, P = 0.0003; girls: +0.35 g/kg LBM, P = 0.003), EDV/LBM (boys: +0.35 ml/kg LBM, P < 0.0

88 er Hb mass/LBM for trained girls (+1.65 g/kg LBM, P = 0.007) were evident compared to untrained contr

89 = 0.0002.) and Hb mass/LBM (boys: +1.29 g/kg LBM, P = 0.015; girls: +1.47 g/kg LBM, P = 0.002) were a

90 quirements) diets providing 33 +/- 1 kcal/kg LBM to young men who were randomly assigned (n = 20/grou

91 ols (29.9 +/- 0.26 and 29.6 +/- 0.27 kcal/kg LBM, respectively; P < 0.01).

92 oups (33.2 +/- 0.27 vs 34.7 +/- 0.27 kcal/kg LBM; P < 0.01).

93 ) or LBM-based oxaliplatin doses (3.09 mg/kg LBM, arm 3).

94 LBM, P = 0.003), EDV/LBM (boys: +0.35 ml/kg LBM, P < 0.0001; girls: +0.31 ml/kg LBM, P = 0.0004), bl

95 47 ml/kg LBM, P = 0.004; girls: +13.48 ml/kg LBM, P = 0.0002.) and Hb mass/LBM (boys: +1.29 g/kg LBM,

96 35 ml/kg LBM, P < 0.0001; girls: +0.31 ml/kg LBM, P = 0.0004), blood volume/LBM (boys: +12.47 ml/kg L

97 .0004), blood volume/LBM (boys: +12.47 ml/kg LBM, P = 0.004; girls: +13.48 ml/kg LBM, P = 0.0002.) an

98 mass (EDV/LBM) for trained boys (+0.28 ml/kg LBM, P = 0.007) and a higher Hb mass/LBM for trained gir

99 ultant mean +/- SEM 0.03 +/- 0.01 mumol . kg LBM-1 . min-1 lower net balance (P < 0.05), which was no

100 to high protein intake (>2.1 g protein . kg LBM-1 . d-1) led to a significantly higher net protein l

101 t also serves an unexpected role in limiting LBM cell number in this early phase.

102 nd HG (p < 0.01) were all reduced in the low LBM group, but not when related to total LBM.

103 onfounders, the intervention group had lower LBM (beta = -0.75; P = 0.03), appendicular skeletal musc

104 (-7.4 +/- 6.6 kg), BMI (-2.6 +/- 2.2 kg/m), LBM (-2.5 +/- 8.7 kg), and fat mass (-3.4 +/- 5.8 kg) wa

105 ed length of acute hospital stay, maintained LBM, improved body composition and hepatic protein synth

106 igate the structure of the live bird market (LBM) contact network and the implications for virus spre

107 purchase of chickens from live bird markets (LBM) and neighboring farms to raise them on their own fa

108 tional virologic study in live bird markets (LBM) in Hanoi, Vietnam, in October 2001.

109 which is associated with live-bird markets (LBM) in the Northeast United States, was first detected

110 Live bird markets (LBMs) are major targets for avian influenza virus (AIV)

111 etween 1994 and 2002 from live-bird markets (LBMs) in the northeastern United States and from three o

112 Vietnamese and Cambodian live bird markets (LBMs) to sustain circulation of highly pathogenic avian

113 loNDV isolates from U.S. live bird markets (LBMs) were analyzed and found to be genetically distinct

114 s isolated from Hong Kong live bird markets (LBMs) were not detected by a USDA-validated matrix gene

115 /100 mL leg/min) and loss of lean body mass (LBM) (-4.1% +/- 1.9%); P < 0.05.

116 of total body fat (TBF) and lean body mass (LBM) (n = 10,525) were followed for 13.6 (standard devia

117 rly individuals can increase lean body mass (LBM) and decrease fat, but interactive effects of growth

118 It is not clear how lean body mass (LBM) and fat mass (FM) compare in their associations wit

119 be associated with decreased lean body mass (LBM) and later puberty in both sexes.

120 f early nutrition with adult lean body mass (LBM) and muscle strength in a birth cohort that was esta

121 rcise (RE) interventions, on lean body mass (LBM) and strength in older adults.

122 y hyperinsulinemic clamp and lean body mass (LBM) and total body fat were assessed by (18)O-water adm

123 owered on a 5% difference in lean body mass (LBM) at 1 month.

124 me that evaluates whole-body lean body mass (LBM) based on CT data from limited-whole-body coverage w

125 of body mass index (BMI) and lean body mass (LBM) depletion on handgrip (HG) force and inspiratory mu

126 estimate a patient-specific lean body mass (LBM) from these MR-AC data.

127 Lean body mass (LBM) is a complex trait for human health.

128 outcomes included change in lean body mass (LBM) measured by dual-energy x-ray absorptiometry, quadr

129 A profound loss of lean body mass (LBM) occurred in both groups during the intervention per

130 ine excretion as an index of lean body mass (LBM) or muscle mass in healthy men and women.

131 Repletion of lean body mass (LBM) that patients lose in human immunodeficiency virus

132 butions of fat mass (FM) and lean body mass (LBM) to body weight, which vary according to age, sex, p

133 um ferritin, after change in lean body mass (LBM) was controlled for, were evaluated in subjects in t

134 of 18 AL (P < 0.001), while lean body mass (LBM) was unchanged.

135 , and 22 +/- 2 pmol x kg(-1) lean body mass (LBM) x min(-1) (P < 0.05, days 5 and 10 vs. baseline).

136 her total body mass (TBM) or lean body mass (LBM), 2) compared the independent contributions of LBM a

137 nges of fat body mass (FBM), lean body mass (LBM), and bone fragility induced by ADT.

138 after adjustment for age and lean body mass (LBM), DES excretion in rapid decliners was 30% greater t

139 mass (SUL), calculated using lean body mass (LBM), is essential for the semiquantification of (18)F-F

140 gy deficit helps to preserve lean body mass (LBM), particularly when combined with exercise.

141 Lean body mass (LBM), skeletal muscle index (SMI), and fat mass (FM) wer

142 ients lost 8 +/- 1% of their lean body mass (LBM), whereas oxandrolone-treated patients had preserved

143 or a high amount [1.1 g . kg lean body mass (LBM)-1 . d-1 or >2.1 g . kg LBM-1 . d-1, respectively],

144 tin doses >3.09 mg per kg of lean body mass (LBM).

145 rophy [33.2 +/- 0.27 kcal/kg lean body mass (LBM)] than for both HIV-infected and healthy controls (2

146 The low bone mass (LBM) phenotype is the result of both the osteoblasts and

147 he diet with the percentage lean body mass (%LBM) or change in %LBM in older subjects.

148 e value (SUV) normalized by lean body mass ([LBM] SUL) is becoming a popular metric for quantitative

149 body composition (including lean body mass [LBM], appendicular lean mass [ALM], and fat mass); objec

150 : +13.48 ml/kg LBM, P = 0.0002.) and Hb mass/LBM (boys: +1.29 g/kg LBM, P = 0.015; girls: +1.47 g/kg

151 8 ml/kg LBM, P = 0.007) and a higher Hb mass/LBM for trained girls (+1.65 g/kg LBM, P = 0.007) were e

152 Post-PHV, LV mass/LBM (boys: +0.50 g/kg LBM, P = 0.0003; girls: +0.35 g/kg

153 Mean LBM decreased significantly from baseline, by 1.0% at mo

154 The older group had a mean LBM smaller than that of the younger group, which was ac

155 rt properties of the lens basement membrane (LBM) from the bovine eye.

156 In men, LBM increased by 0.1 kg with placebo, 1.4 kg with testos

157 fr1 inactivation in all limb bud mesenchyme (LBM) cells during limb initiation.

158 hod (IBM) within a lattice Boltzmann method (LBM) fluid solver, and the finite element method (FEM) i

159 The lattice Boltzmann method (LBM) is adopted to simulate fluid flow and Knudsen diffu

160 Lattice Boltzmann method (LBM) simulations helped us rationalize the impact of the

161 rmeability using a lattice Boltzmann method (LBM) technique.

162 he other hand, the Lattice Boltzmann Method (LBM) tracks the movement of collections of molecules, wh

163 zed version of the lattice Boltzmann method (LBM).

164 s simulated by the lattice Boltzmann method (LBM).

165 Pore-scale Lattice Boltzmann Methods (LBM) can simulate flow in complex pore structures releva

166 Here, we introduce light beads microscopy (LBM), a scalable and spatiotemporally optimal acquisitio

167 urvival with higher FM% and higher "FM minus LBM percentiles" in both sexes, whereas a higher LBM was

168 was applied to derive Lifestyle-Based Model (LBM) for CHD-risk prediction incorporating age, sex, bod

169 g modules (DBMs) and ligand-binding modules (LBMs) from distinct transcriptional repressors.

170 g modules (DBMs) and ligand-binding modules (LBMs) from the same protein family, resulting regulators

171 At 36 months, LBM decreased by 2.8% in men age >/= 70 years and by 0.9

172 The hemagglutinin gene of most LBM isolates examined appeared to have been the result o

173 Most LBMs were connected to one another, suggesting that the

174 The average maximal accumulation of LBM (386 g/mo; 95% CI: 372, 399) occurred 0.5 y before t

175 2) compared the independent contributions of LBM and fat mass (FM) to EE, and 3) investigated whether

176 to estimate the independent contributions of LBM and FM to EE.

177 the Janmahasatian formula for estimation of LBM in modern PET scanners and display workstations is r

178 er day [P=.05]; 6.9 [1.7] vs 3.8 [2.9] kg of LBM [P=.005]; greater strength gains for various upper a

179 o 31% post-neoadjuvant therapy, with loss of LBM (-3.0 +/- 5.4 kg, P < 0.0001), but not FM (-0.3 +/-

180 The mean (+/-SD) loss of LBM (kg) at 1 month was -3.7 +/- 8.7 in the EN-S group,

181 esults were compared with the measurement of LBM from whole-body CT (reference standard) and the resu

182 les of FM and percentage FM (FM%) but not of LBM were associated with greater survival: HRs of 0.51 (

183 The results of LBM measurement evaluated with this technique were much

184 rage) to 0.998 (longer coverage) and SEMs of LBM ranging from 0.14 to 0.33 kg.

185 ibution of FM to EE is comparable to that of LBM in normal mice (expressed per gram of tissue) but is

186 tage of FM appears to be superior to that of LBM.

187 e to the current policy of a complete ban of LBMs in some areas.

188 ion of metabolic activity of tumors based on LBM-normalized standardized uptake value.

189 signed (1:1) to receive BSA-based (arm 2) or LBM-based oxaliplatin doses (3.09 mg/kg LBM, arm 3).

190 protein turnover for creatinine excretion or LBM eliminated the age effects.

191 after adjustment for creatinine excretion or LBM.

192 t was not due to reduced subcutaneous fat or LBM, but rather preferential loss of abdominal fat (P <

193 Division of EE by either TBM or LBM is confounded by body mass variation.

194 Participants' LBM and appendicular skeletal muscle mass were measured

195 old mice revealed a low bone mass phenotype (LBM) in both the distal femur and the vertebra of Krox20

196 s not optimal leading to some false positive LBMs.

197 llele led to Fgfr1 inactivation in posterior LBM cells.

198 or standard PN regimens during HSCT preserve LBM.

199 Alternative approaches to preserve LBM are needed.

200 s oxandrolone-treated patients had preserved LBM (+9 +/- 4%), P < 0.05.

201 .3 1.5), while lean body mass was preserved (LBM: 0.0 0.7).

202 +/-1.5), while lean body mass was preserved (LBM: 0.0+/-0.7).

203 Patients with reduced LBM were randomly assigned (1:1) to receive BSA-based (a

204 ercepts of the regression equations relating LBM to average daily creatinine excretion were the same

205 ard LBM measurements and thus offers routine LBM-based SUV quantification in PET/MR.

206 to lentogenic class I viruses found in U.S. LBMs and wild waterfowl.

207 sed to calculate a reference LBMADP A second LBM estimate was derived from available MR-AC data using

208 Some LBMs that have been banned still are very active, and th

209 wded systems, while the proposed coupled SPT-LBM predicts the same behaviour as the kinetic Monte Car

210 -AC is shown to correlate well with standard LBM measurements and thus offers routine LBM-based SUV q

211 o) with a baseline and at least one on-study LBM assessment.

212 Over the 3-y study, %LBM increased by 2.6 +/- 3.6%.

213 o body weight (SUV(BW)), lean body mass (SUV(LBM)), whole blood (SUV(WB)), parent plasma activity con

214 tational cost orders of magnitude lower than LBM simulations.

215 The results indicate that LBM over-predicts the diffusion in 2D crowded systems, w

216 The LBM so obtained was compared with results from conventio

217 The LBM with and without PRS both demonstrated good calibrat

218 be highly sensitive for both viruses at the LBM level.

219 ote an automated program that determined the LBM from a CT with limited fields of acquisition and app

220 nes from representative H7 isolates from the LBM and elsewhere were sequenced, and the sequences were

221 he observed transport characteristics of the LBM are discussed in the context of established membrane

222 t strategic positions; and estimation of the LBM of the whole body on the basis of a predetermined re

223 e computational efficiency is a match of the LBM populations at the upper boundary of the simulation

224 bility of a method for the estimation of the LBM using the data of the low-dose CT from PET/CT acquir

225 -index (indicative of discrimination) of the LBM, PCE and SCORE2 was 0.713 (95% Confidence Interval [

226 While sampling the LBM environment has become a widely used alternative to

227 Krox20(+/-) splenocytes, suggesting that the LBM is attributable to Krox20 haploinsufficiency in the

228 onnected to one another, suggesting that the LBM network may support large-scale disease spread.

229 icant effect on solute transport through the LBM that was opposite of the expected electrokinetic beh

230 ay to incorporate the crowding effect to the LBM.

231 is the predominant mechanism underlying the LBM phenotype of Krox20-deficient mice.

232 ly related to the viruses circulating in the LBMs.

233 The %LBM, defined as total body nonfat, nonbone tissue weight

234 eceived nandrolone decanoate increased their LBM by 3.1 +/- 2.2 kg (P < 0.0001).

235 A third LBM estimate was obtained from a sex-specific formula (L

236 A crowding-adaptation to LBM has been developed using SPT, allowing fast simulati

237 The addition of PRS to LBM marginally improved calibration, with the slope incr

238 Adding PRS to LBM, PCE and SCORE2 increased the C-index to 0.733 (95%

239 played a closer phylogenetic relationship to LBM-origin NDV.

240 m on their own farms, frequency of visits to LBM, purchase of poultry at LBM for consumption) and con

241 Total LBM was determined by 40K counting in all subjects, and

242 as no additional benefit on changes in total LBM, ALM, or HG strength.

243 have a significant positive effect on total LBM [weighted mean difference in change (WMD): 0.34; 95%

244 s in older adults (mean age >=50 y) on total LBM, appendicular lean mass (ALM), and handgrip (HG) and

245 low LBM group, but not when related to total LBM.

246 Unadjusted LBM and grip strength were similar in 2 groups.

247 To identify genomic loci underlying LBM, we performed a gene-based genome-wide association s

248 Using LBM, we demonstrate mesoscopic and volumetric imaging at

249 2+) transients were prolonged in SMMs versus LBMs and Ca(2+) release was disorganized in SMMs with re

250 was bromodeoxyuridine positive (SMMs versus LBMs: 3.1% versus 0.8%; P<0.05), and p16(INK4a) negative

251 que empirical dataset collated in Vietnamese LBMs to estimate the sensitivity and specificity of five

252 : +0.31 ml/kg LBM, P = 0.0004), blood volume/LBM (boys: +12.47 ml/kg LBM, P = 0.004; girls: +13.48 ml

253 d older groups, and the precision with which LBM could be determined from creatinine excretion was no

254 ation dynamics and reconstructed Alpine-wide LBM defoliation records reveals elevational shifts in LB

255 The hurdle to bridging MD with LBM is the computational expense of MD simulations neede

256 the proposed method correlated strongly with LBM derived from whole-body CT data, with correlation co

257 as significantly positively associated with %LBM at baseline (beta = 0.033, P = 0.006; adjusted for s

258 and oxaliplatin chemotherapy, those without LBM reduction received body surface area (BSA)-based oxa

259 In women, LBM increased by 0.4 kg with placebo, 1.2 kg with HRT (P