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1 maximal stimulation of the G protein-coupled LH receptor.
2 of the FSH receptor and the TM region of the LH receptor.
3 tivation have been identified in the TSH and LH receptors.
4 ed the TSH receptor but not the CG/lutropin (LH) receptor.
5 nt studies suggest that luteinizing hormone (LH) receptor activation leads to trans-activation of epi
6 osterone or human chorionic gonadotropin (an LH receptor agonist), respectively.
7 ucing cAMP, co-expression with the truncated LH receptor allowed partial restoration of ligand signal
8 ic receptor containing the ectodomain of the LH receptor and transmembrane region of fly LGR2 was rep
9 lls and theca cells express large numbers of LH receptors, and LH-stimulated paracrine mediators comm
10 ts the lateral motions of antagonist binding LH receptors, and that receptor surface densities must b
11 as interactions between these domains of the LH receptor are permissive for constitutively activating
12 including expression of luteinizing hormone (LH) receptor by the granulosa cells.
13 eptor with only TM V and VI derived from the LH receptor can be rendered constitutively active by the
14                                      For the LH receptor, conversion of Ser-277 to all natural amino
15 stitution of the homologous exoloop 2 of the LH receptor decreased G(s) coupling.
16  cell-associated or soluble EC region of the LH receptor did not allow ligand signaling.
17 NA degradation occurs during hormone-induced LH receptor down-regulation.
18 idual FLAG-tagged human luteinizing hormone (LH) receptors expressed on CHO cells and native LH recep
19 in in the post-transcriptional regulation of LH receptor expression in the ovary.
20 ides a novel mechanism for the regulation of LH receptor expression in the ovary.
21  been identified in the luteinizing hormone (LH) receptor gene, leading to constitutive activation of
22 at LH receptors (rLHRs), but hCG binds human LH receptors (hLHRs) 1000-10,000 fold better than bLH.
23 tion in the i3 to identify the region in the LH receptor important for its constitutive activation.
24 on and indicate the importance of TM1 of the LH receptor in the proper orientation of the EC ligand b
25 results suggest that the hinge region of the LH receptor interacts with exoloop 2 to constrain the re
26 ssenger formed when the luteinizing hormone (LH) receptor is activated.
27                                   Similar to LH receptors labeled with Au-hCG, LH receptors labeled w
28 Similar to LH receptors labeled with Au-hCG, LH receptors labeled with gold-deglycosylated hCG, an hC
29 eceptors containing only TM V to VI from the LH receptor led to major increases in ligand-independent
30 convert the noncleaving luteinizing hormone (LH) receptor (LHR) into a cleaved, two-subunit molecule.
31 were 3betaHSD-negative, luteinizing hormone (LH) receptor (LHR)-negative, and platelet-derived growth
32                                          The LH receptor mRNA binding activity of LRBP measured by RN
33               We demonstrated that increased LH receptor mRNA degradation occurs during hormone-induc
34 t analysis showed an inverse relationship to LH receptor mRNA levels during different physiological s
35        Furthermore, increased degradation of LH receptor mRNA was seen when partially purified LRBP w
36 nt study, we isolated and characterized this LH receptor mRNA-binding protein (LRBP) from rat ovary.
37                         An ovarian cytosolic LH receptor mRNA-binding protein (LRBP) identified in ou
38 es show a role for LRBP in the regulation of LH receptor mRNA.
39 h bipartite sequence in the coding region of LH receptor mRNA.
40 ajor role in regulating luteinizing hormone (LH) receptor mRNA expression in the ovary.
41                         Luteinizing hormone (LH) receptor mRNA is post-transcriptionally regulated.
42  receptors expressed on CHO cells and native LH receptors on both KGN human granulosa-derived tumor c
43 mpared with LH receptors on untreated cells, LH receptors on cells treated with 100 nm hCG exhibit re
44 ents despite treatment with 100 nm hCG as do LH receptors on cells treated with cytochalasin D.
45                                Compared with LH receptors on untreated cells, LH receptors on cells t
46 as inhibited by treatment with antisense hCG/LH receptor phosphorothioate oligodeoxynucleotide.
47                                              LH receptor point mutants lacking potential palmitoylati
48                                Both bind rat LH receptors (rLHRs), but hCG binds human LH receptors (
49 resides in the hinge region of fly LGR2 with LH receptor sequences led to constitutive receptor activ
50 embrane region of fly LGR2 was replaced with LH receptor sequences.
51  and exoloop 2, but not exoloop 1 or 3, from LH receptors showed decreases in constitutive activity,
52 llicles but not MA-10 cells, suggesting that LH receptor signaling activates the EGFR by different me
53 tical downstream target of G-protein-coupled LH receptor signaling and one of the first transcription
54  with hCG induces aggregation of YFP-coupled LH receptors stably expressed on CHO cells.
55 ydig cell hypoplasia, we identified a mutant LH receptor that is truncated at TM5.
56 d LH receptor with an N-terminally truncated LH receptor that lacked the EC ligand binding domain als
57 -expression of the anchored EC region of the LH receptor together with the N-terminally truncated rec
58 ddition, co-expression of the same truncated LH receptor with an N-terminally truncated LH receptor t
59 e that binding of hCG induces aggregation of LH receptors within nanoscale, cell surface membrane com