ライフサイエンスコーパス: LH

1 LH increases following MVT-602 were similar in PCOS and

2 LH melanin-concentrating-hormone (MCH) and orexin/hypocr

3 LH stimulation caused sated mice to pick up pellets of s

4 LH-21 did not affect food intake nor body weight but it

5 LH-21 is a triazol derivative that has been described as

6 [1-MH](+) to a ligand protonated species [1-LH](+), from which ligand dissociation is facile, genera

7 re-planar Ir(I) [Ir(N(NN(H))(2))(CO)](+) ([2-LH](+)).

8 ligand dissociation is facile, generating [2-LH](+).

9 Subsequent reaction of [2-LH](+) with [1-MH](+) allows for production of H(2) and

10 ightward language lateralization (N = 30, 25 LH) do not rely on an organization that simply mirrors t

11 [NHI(iPr2Me2)P(Ph)NH-2,6-(i)Pr(2)C(6)H(3)] (LH), containing two different N-substituents was prepare

12 Both T80/40/LH and HAN supported primary isolation of strain IC:20:0

13 ailable EMJH and the more specialized T80/40/LH media formulations, in semi-solid and liquid composit

14 However, only HAN and T80/40/LH supported the growth of L. borgpetersenii serovar Har

15 driver lines providing genetic access to 82 LH cell types.

16 ere, we show reversible anion insertion in a LH for the first time using Co and Co-V layer hydroxides

17 d siRNA-mediated knock down of HSL abrogated LH-induced progesterone production.

18 ify a molecularly distinct, orexin-activated LH submodule that governs physical activity in mice.

19 In addition, LH decreased the association of DRP1 with the mitochondr

20 ated PRL-, inhibited ACTH- and did not alter LH/FSH/TSH-release; and 3) resistin increased ACTH-relea

21 Our studies reveal an LH-variant and binding-mode dependent heterogeneous ense

22 When an LH exhibits more than two genotypes, we call it a local

23 lecule inhibitor of DRP1 increased basal and LH-induced progesterone production.

24 Serum E2 and LH levels were normalized in PitERtgKO females and were

25 Lower estradiol and LH levels were found in urine and saliva samples of soy

26 h reduced Th17 frequencies and lower FHS and LH concentrations compared to NET-EN/COCPs, with FHS con

27 h reduced Th17 frequencies and lower FSH and LH concentrations, as compared to NET-EN and COCPs, with

28 miR-7 genomic circuits that regulate FSH and LH synthesis and secretion through their effects on pitu

29 cose, and insulin levels with lower SHBG and LH levels.

30 s of serum estradiol (E2), testosterone, and LH.

31 of neurons in the lateral hypothalamic area (LH) expressing the neuropeptide neurotensin (Nts) is cri

32 The lateral hypothalamic area (LH) is a vital controller of arousal, feeding, and metab

33 tually exclusive behavioral effects, such as LH VGLUT2 and VGAT neurons [4-7] and orexin- (ORX) and m

34 nanocrystal surfaces and closely associated LH(+) counter-cations (protonated n-octylamine or tri-n-

35 3] could allow coordinating activity between LH cell types, some of which have mutually exclusive beh

36 length and rescued the progesterone blockade LH surge, while RU486 into the ARC shortened LH pulse in

37 Lesion of peri-LC GABA neurons blocked LH stimulation-induced eating, establishing them as a cr

38 have typical reproductive cycles but blunted LH surges, associated with decreased excitability of the

39 Both LH and FSH increased until ~ 5 and 7 years postmenopause

40 Results showed that both LH and LS had higher relative abundance of S and Fe oxid

41 uxtacapsular BST, a region that contains BST-LH projecting neurons.

42 Fermented butter (LH-butter) was produced by churning the cream that was f

43 epertoire of survival behaviors regulated by LH circuits.

44 r rats), that chemogenetic inhibition of CeA-LH projections attenuates avoidance in male Avoider rats

45 Collectively, these data show that CeA-LH projections are important for persistent avoidance of

46 Further characterization of the CeA-LH circuit may improve our understanding of the neural m

47 ts, that chemogenetic stimulation of the CeA-LH circuit produces conditioned place avoidance (CPA) in

48 The ManR clears LH thus regulating testosterone production, whereas the

49 that 3 weeks after withdrawal from cocaine, LH MCH neurons exhibit a wide range of gene expression c

50 an Spectrum Analysis is applied to determine LH pulsatility in three distinct patient cohorts.

51 electrochemical analysis system to determine LH pulsatility.

52 n and off-dyad modes, and spanning different LH densities (0.5-1.6 per nucleosome), over a wide range

53 ific ERalpha knockout mice exhibit disrupted LH pulses and surges.

54 es dominantly interacting with parental DNA, LHs must have strong interactions with nonparental DNA a

55 ctional criteria to show that the Drosophila LH has ~1400 neurons and >165 cell types.

56 omatosomes with one on-dyad and one off-dyad LH.

57 ental mechanism for recovery following early LH injury.

58 tor alpha-mediated signaling causes episodic LH secretion and cystic ovary.

59 anion insertion electrochemistry establishes LHs as a materials platform for anion insertion electroc

60 as a critical downstream circuit element for LH neurons.

61 studies that are consistent with a role for LH in learning.

62 gma is that these effects reflect a role for LH neurons in the control of the core motivation to feed

63 h were highest by microbial communities from LH, lower by those from LS and lowest form Sediment.

64 Ca(2+) imaging from LH(GABA) neurons indicate that they are both wake and ra

65 Furthermore, LH increased trafficking of cholesterol from the lipid d

66 Our study clarifies how GABAergic LH inputs to the VTA can contribute to generalized behav

67 dence that photostimulation of the GABAergic LH-VTA component, but not the glutamatergic component, i

68 c targets of lateral hypothalamic GABAergic (LH(GABA)) neurons and that activation of this pathway in

69 xpression in lateral hypothalamus GABAergic (LH(GABA)) neurons.

70 viding homeostatic feedback on episodic GnRH/LH release as well as positive feedback to control ovula

71 terone can block the oestradiol-induced GnRH/LH surge and inhibit LH pulse frequency.

72 effects of HCD on the estradiol-induced GnRH/LH surge were overcome by neuron-specific SOCS3 knock-ou

73 As the most potent stimulator of GnRH/LH release, kisspeptin is believed to mediate the positi

74 sitive feedback generating preovulatory GnRH/LH surges.

75 progesterone's inhibitory effect on the GnRH/LH surge and pulsatile secretion is mediated by its rece

76 five pituitary tumor patients [gonadotropic (LH/FSH-secreting) = 17; prolactinomas (PRL-secreting) =

77 ease in secretion of pituitary gonadotropins LH and FSH and impairment of reproductive function.

78 They identify a PVT->NAc->LH circuit essential for recall of opioid experiences.

79 ate the potential of targeting the PVT->NAc->LH pathway for treating opioid addiction.

80 in 287 healthy volunteers (150 left-Handers (LH)) of language task-induced asymmetries and intrinsic

81 e pulmonary circulation, and the left heart (LH) retrogradely contributes significantly to this vascu

82 eat (SD) stress model, learned helplessness (LH), and a chronic corticosterone (CORT) model in mice,

83 and the addition of Lactobacillus helveticus LH-B02 favored the formation of peptides recognized as b

84 nt, the addition of Lactobacillus helveticus LH-B02, and storage time were evaluated.

85 iant on the ability to mobilize labile heme (LH).

86 guage is lateralized to the left hemisphere (LH) in most neurologically healthy adults.

87 e but rather on patterns of left-hemisphere (LH) and right-hemisphere (RH) activation across individu

88 Higher LH level was found in urine samples of soy formula-fed g

89 The complex role of linker histone (LH) on chromatin compaction regulation has been highligh

90 pulsatile secretion of luteinising hormone (LH).

91 Luteinizing hormone (LH) activates protein kinase A (PKA) signaling in luteal

92 characterized by higher luteinizing hormone (LH) and sex hormone binding globulin (SHBG) levels with

93 mulating hormone (FSH), luteinizing hormone (LH) and testosterone levels or testicular and semen volu

94 sing hormone (GnRH) and luteinizing hormone (LH) are pivotal events in female reproductive function.

95 The half-life for luteinizing hormone (LH) clearance increases in Mrc1(-/-) and Mrc1(-/-)Asgr2(

96 baseline measurement of luteinizing hormone (LH) in serum, used as the reference standard for identif

97 he pulsatile release of luteinizing hormone (LH) is critical for mammalian fertility.

98 ting hormone (FSH), and luteinizing hormone (LH) levels were measured.

99 dback on pulsatile GnRH/luteinizing hormone (LH) release and positive feedback generating preovulator

100 neurons and subsequent luteinizing hormone (LH) release.

101 women, the amplitude of luteinizing hormone (LH) rise was similar to that after KP54, but peaked late

102 mechanism for low GnRH/luteinizing hormone (LH) secretion.

103 ating hormone (FSH) and luteinizing hormone (LH) secretion.

104 redict the preovulatory luteinizing hormone (LH) surge is not practical.

105 Luteinizing hormone (LH) via activation of protein kinase A (PKA) acutely sti

106 ting hormone (FSH), and luteinizing hormone (LH) were measured.

107 ating hormone (FSH) and luteinizing hormone (LH), both of which are heterodimers specified by unique

108 estrone, testosterone, luteinizing hormone (LH), follicle-stimulating hormone (FSH) and sex hormone-

109 terone sulfate (DHEAS), luteinizing hormone (LH), follicle-stimulating hormone (FSH), prolactin, fast

110 drimer 3 showed similar luteinizing hormone (LH)-release activity to triptorelin in mice.

111 atile secretion of the gonadotropic hormones LH and FSH and is critical for fertility.

112 aracterised changes in reproductive hormones-LH, FSH, SHBG and AMH-by chronological age and time arou

113 hroom body (CA) as well as the lateral horn (LH) of the protocerebrum via the medial AL tract.

114 e higher olfactory centre, the lateral horn (LH), is implicated in innate behaviour.

115 required for learning, and the lateral horn (LH), proposed to mediate innate olfactory behavior.

116 its predictions experimentally, showing how LH secretion is frequency-modulated as we increase the b

117 -) mice but not in Asgr2(-/-) mice; however, LH and testosterone are elevated in all three knockouts.

118 resent different microhabitats: low hummock (LH), high lawn and low lawn.

119 In recent years layered hydroxides (LHs) have been studied for a range of electrochemical ap

120 (PLOD2) encodes the only lysyl hydroxylase (LH) isoform that specifically hydroxylates lysine residu

121 the hypothesis that localised hypermutation (LH) compensates for fitness losses caused by bottlenecks

122 genetic stimulation of lateral hypothalamic (LH) GABA neurons induces rapid vigorous eating in sated

123 ibitory neurons in the lateral hypothalamus (LH(vgat)) show unique activity patterns during feeding t

124 (Nts) terminals in the lateral hypothalamus (LH) also decreases food intake.

125 d their projections to lateral hypothalamus (LH) and ventral tegmental area (VTA).

126 CeA projections to the lateral hypothalamus (LH) are preferentially activated in male rats that show

127 Damage to the lateral hypothalamus (LH) causes profound physical inactivity in mammals.

128 The lateral hypothalamus (LH) controls energy balance.

129 cal stimulation of the lateral hypothalamus (LH) has two motivational effects: long trains of stimula

130 A pivotal role of the lateral hypothalamus (LH) in regulating appetitive and reward-related behavior

131 However, the lateral hypothalamus (LH) is also a key reward-control locus in the brain.

132 ng the downstream NAc->lateral hypothalamus (LH) pathway also prevents relapse.

133 reased activity in the lateral hypothalamus (LH) promotes feeding.

134 Projections from the lateral hypothalamus (LH) to the ventral tegmental area (VTA), containing both

135 between the IC and the lateral hypothalamus (LH) with a monosynaptic relay in the CeA and shed new li

136 al habenula (LHb), the lateral hypothalamus (LH), and the midbrain are not only reciprocally connecte

137 ing neurons within the lateral hypothalamus (LH), as well as their function in the arousal network, r

138 between the IC and the lateral hypothalamus (LH), which engages numerous LH-projecting CeL/C cells wh

139 e (MCH) neurons in the lateral hypothalamus (LH), which regulate REM sleep initiation and maintenance

140 ell (NAcmSh), and with lateral hypothalamus (LH)-projecting D1-MSN hyperexcitability mediated by decr

141 licking responses, but lateral hypothalamus (LH)-projecting neurons were more active in go trials wit

142 at) projections to the lateral hypothalamus (LH).

143 eep, we focused on the lateral hypothalamus (LH).

144 its projections to the lateral hypothalamus (LH).

145 amygdala (CeA) to the lateral hypothalamus (LH)] mediates avoidance of stress-associated stimuli.

146 Using optogenetic activation, we identify LH cell types that drive changes in valence behavior or

147 This study directly implicates LH(PV) neurons in modulating nociception, thus expanding

148 d chronological age contribute to changes in LH, FSH, SHBG and AMH across mid-life in women, and BMI,

149 in vitro and in vivo, leading to decrease in LH cell content despite high basal Lhb expression.

150 e we explored the involvement of dopamine in LH stimulation-induced eating.

151 nformation about the cues leading to food in LH itself.

152 r BMI was associated with slower increase in LH and FSH and decrease in AMH.

153 te nucleus kisspeptin (ARN(KISS)) neurons in LH pulse generation.

154 In contrast, LPXRFa-R are expressed only in LH, ACTH, and alpha-MSH cells.

155 eversible electrochemical anion insertion in LHs without significant material optimization.

156 KERKO mice had increased LH pulse frequency, indicating loss of negative feedback

157 an unidentified glycoprotein that increases LH levels.

158 ing studies in infants and children indicate LH lateralization for language.

159 topallidal inputs at the level of individual LH(Gad1) neurons may be critical to balancing propensity

160 In experiment 1, E2 benzoate-induced LH surges in ovariectomized female monkeys were severely

161 oestradiol-induced GnRH/LH surge and inhibit LH pulse frequency.

162 ed the hypothesis that progesterone inhibits LH surge and pulsatile secretion via its receptor in the

163 Uncontrolled, irregular LH secretion may be the root cause of the cystic ovarian

164 he key processes in photosynthesis, that is, LH, EnT, ET, and CAT, define the structure of this revie

165 Teitelbaum SL, Pinney SM, Windham GC, Kushi LH, Biro FM, Valentin-Blasini L, Blount BC, Wolff MS, fo

166 PPG monosynaptic input include the lateral (LH) and paraventricular (PVN) nuclei of the hypothalamus

167 irefly luciferase (FLuc) with the luciferin (LH(2)) analogue infraluciferin (iLH(2)), near-infrared d

168 f LH is heterogeneous; the cytosol maintains LH at approximately 20-40 nM, whereas the mitochondria a

169 ect innervation of the STN, but manipulating LH-projecting neurons had the opposite effects.

170 ated a resource for manipulating and mapping LH neurons, providing new insights into the circuit basi

171 ng-term potentiation, through a monosynaptic LH(GABA) to CA3(GABA) projection.

172 hatchling enantiornithine bird specimen MPCM-LH-26189, supporting precocial nesting behavior in this

173 : 1) Leptin stimulated PRL/ACTH/FSH- but not LH/TSH-release; 2) adiponectin stimulated PRL-, inhibite

174 al hypothalamus (LH), which engages numerous LH-projecting CeL/C cells whose activity can be strongly

175 We find ~30% of LH projections converge with outputs from the mushroom b

176 a critical mediator of the acute actions of LH on luteal progesterone production.

177 Food motivation was reduced by activation of LH GLP-1R.

178 Assessment of LH pulsatility is important for the clinical diagnosis o

179 0002), with correspondingly increased AUC of LH exposure (169.0 vs. 38.5 IU.h/L; P = 0.0058).

180 onversely, acute pharmacological blockade of LH GLP-1R increased food motivation but only in male rat

181 gation of the oestrous cycle and blockade of LH surge.

182 However, the contributions of LH circuits to other survival behaviors have been less e

183 lpha is necessary for the precise control of LH secretion.

184 d, sensitive and repeatable determination of LH concentration profiles.

185 We find that the subcellular distribution of LH is heterogeneous; the cytosol maintains LH at approxi

186 ein, we elucidate the nature and dynamics of LH using genetically encoded ratiometric fluorescent hem

187 ghted by recent discoveries of the effect of LH binding variability and isoforms on genome structure

188 on of a PKA inhibitor blocked the effects of LH and forskolin on DRP1 phosphorylation.

189 We also find that an increase of LH density beyond 1 is best accommodated in chromatosome

190 e rat to show that optogenetic inhibition of LH gamma-aminobutyric acid (GABA) neurons restricted to

191 delivery, the co-translational insertion of LH polypeptides and their folding and assembly to form p

192 Chronic knockdown of LH GLP-1R induced by intraparenchymal delivery of an ade

193 e to persistently high circulating levels of LH from the pituitary.

194 y deficits, whereas targeted manipulation of LH(GABA) or CA3(GABA) neuron activity reversed memory de

195 We find that photostimulation of LH(PV) neurons suppresses nociception to an acute, noxio

196 s cocaine, we did transcriptome profiling of LH MCH neurons after long-term withdrawal using RNA-sequ

197 sensory and whole-body output properties of LH cell populations have received much interest, their i

198 files and electrophysiological properties of LH MCH neurons.

199 However, the properties of LH pools, including concentration, oxidation state, dist

200 r is associated with intrinsic properties of LH-projecting CeA cells.

201 (KISS) neurons for 1 min generated pulses of LH in freely behaving mice, whereas inhibition with arch

202 The reaction of LH [L = {(ArNH)(ArN)-C=N-C=(NAr)(NHAr)}; Ar =2,6-Et(2)-C

203 Reaction of LH with Y(N(SiMe(3))(2))(3) afforded the heteroleptic co

204 REM sleep-specific optogenetic silencing of LH(vgat) cells induced a reorganization of these activit

205 Similar eating was induced by stimulation of LH GABA terminals or GABA cell bodies in this peri-LC re

206 chemogenetic or optogenetic stimulations of LH MCH neural activity increase REM sleep after long-ter

207 tic circuit mapping to study the strength of LH optogenetic responses and network oscillations, which

208 Previous work has suggested that a subset of LH GABA neurons projects to the ventral tegmental area (

209 Several molecularly distinct types of LH neurons have been identified, including orexin cells

210 th halorhodopsin were found to reset ongoing LH pulsatility.

211 y converge with ventral striatal inputs onto LH(Gad1) neurons.

212 RP1 showed that DRP1 is required for optimal LH-induced progesterone biosynthesis.

213 es in vivo during the GnRH-induced ovulatory LH surge and correlates with GnRHR.

214 ported that progesterone prevented premature LH surges during ovarian hyperstimulation in women.

215 could be used to anticipate the preovulatory LH surge in women.

216 increased ACTH-release and did not alter PRL/LH/FSH/TSH-secretion.

217 be near-perfectly correlated with pulsatile LH secretion.

218 calcium-binding protein parvalbumin (PVALB; LH(PV) neurons), a small cluster of neurons within the L

219 Finally, cNTS-projecting neurons within PVN, LH, and Bar express the activation marker cFOS in mice a

220 r-weight (LMW and HMW) fractions by Sephadex LH-20 column chromatography.

221 , and fractions (fractionationed by Sephadex LH-20 column) from these two legumes.

222 from coffee pulp was purified using Sephadex LH-20.

223 size exclusion chromatography with Sephadex LH-20 without the need for any previous SPE of phenolic

224 LH surge, while RU486 into the ARC shortened LH pulse interval in the progesterone treated rats.

225 of steroidogenic small luteal cells (SLCs), LH, and forskolin stimulated phosphorylation of DRP1 (Se

226 Looking further, we found that some LH GABA fibers pass through the VTA to more caudal sites

227 We determined that anomalous sporadic LH secretion caused the severe ovarian phenotype of PitE

228 r, continuous treatment with GnRH stimulated LH secretion in vitro and in vivo, leading to decrease i

229 blood from luteinizing/follicle-stimulating (LH/FSH)-secreting (n = 24), prolactinomas (n = 14), and

230 Data from the driest microhabitat studied, LH, revealed a clear and strong negative linear correlat

231 tiple patch clamp has not been used to study LH connectivity, aside from a limited dataset of MCH neu

232 n with archaerhodopsin for 30 min suppressed LH pulsatility.

233 Moreover, we demonstrate that LH(PV) axons form functional excitatory synapses on neur

234 We hypothesize that LH via PKA differentially regulates the phosphorylation

235 e small luteal cells our results reveal that LH, forskolin, and 8-Br cAMP-induced PKA-dependent phosp

236 otting and confocal microscopy revealed that LH stimulates phosphorylation and translocation of HSL t

237 These results suggest that LH-21 can be a new candidate to fight against diabetes o

238 The LH surge increases the expression of Runx1 and Runx2 in

239 The LH/FSH ratio was not significantly different among the P

240 sponses of the pulmonary vasculature and the LH should modulate the global right haemodynamic burden.

241 variability that consistently anticipate the LH surge, suggesting that automated ultradian rhythm mon

242 Conversely, the LH of ppDIO-exposed mice had reduced contents of seroton

243 Hb requires glutamatergic signaling from the LH, but not from the midbrain.

244 Our data identify the LH GLP-1R as an indispensable element of normal food rei

245 rneurons with locally ramifying axons in the LH [11, 12], and nearby subthalamic and thalamic areas l

246 ptide YY 3-36 increased 5-HT turnover in the LH and ameliorated the ppDIO-induced sleep disturbances,

247 warding stimuli as well as its effect in the LH and mPFC are not well understood.

248 eceptor (GLP-1R) activity selectively in the LH can profoundly affect food reward behavior, ultimatel

249 ically inhibiting VTA(Vgat) terminals in the LH elevated locomotion and decreased immobility time dur

250 show significant activation not only in the LH language network but also in their RH homologs in all

251 In the LH model, latency to escape was increased following trai

252 In contrast, odor coding in the LH remains poorly understood.

253 population of VLPO-projecting neurons in the LH that express the vesicular GABA transporter (VGAT; a

254 T neurons to a subset of vGAT neurons in the LH, an area involved in homeostatic and hedonic control

255 SDn has more distributed connectivity in the LH, preferentially synapsing with principal neuron types

256 acological or genetic GLP-1R blockade in the LH.

257 sive for selecting active populations in the LH.

258 harmonic response on RV pulsatile load: the LH harmonic response was responsible for a 20% reduction

259 n anatomical and neurotransmitter map of the LH and link this to EM connectomics data.

260 show that inhibition of the terminals of the LH GABA neurons in ventral-tegmental area (VTA) facilita

261 ll as molecular and efferent targets, of the LH GLP-1R activation were also evaluated.

262 s), demonstrating a favourable effect of the LH harmonic response on RV pulsatile load: the LH harmon

263 e is matched to the harmonic response of the LH in a way that efficiently reduces the pulmonary pulsa

264 ructural and functional understanding of the LH is scant, in large part due to a lack of sparse neuro

265 kade of Fshb expression and depletion of the LH secretory pool are two major factors accounting for w

266 er that uniquely enabled anticipation of the LH surge at least 2 days prior to its onset in 100% of i

267 the potent arousal-promoting property of the LH(GABA) -> VLPO pathway, presynaptic inputs to LH(GABA)

268 the selective chemogenetic silencing of the LH-to-LHb pathway impairs aversion-driven escape behavio

269 we show that the GABAergic component of the LH-VTA pathway supports positive reinforcement and place

270 ary vessels and the harmonic response of the LH.

271 abolites were measured daily surrounding the LH surge in 20 cycles.

272 implications of these findings are that the LH differs between females and males in its ability to c

273 These results suggest that the LH GABA neurons are critical for storing and later disse

274 IC and CeA through direct projections to the LH.

275 rons), a small cluster of neurons within the LH glutamatergic circuitry, modulate nociception in mice

276 with optogenetic circuit mapping within the LH have demonstrated only a minority of connections when

277 recordings to screen connectivity within the LH with standard methodology we previously used in the n

278 tra-sparse intrinsic connectivity within the LH.

279 antinociception evoked by activation of this LH(PV)->vlPAG pathway.

280 les alongside dates of supra-surge threshold LH and menstruation.

281 Thus, LHs add another level of epigenetic regulation of chroma

282 Compared to LH/FSH-secreting tumors, PRL-secreting tumors showed sta

283 and non-functional subtypes when compared to LH/FSH-secreting tumors.

284 GABA) -> VLPO pathway, presynaptic inputs to LH(GABA) neurons originate from several canonical stress

285 pse during reacquisition, via projections to LH, where they converge with ventral striatal inputs ont

286 ol relapse during renewal via projections to LH.

287 aling pathway in luteal cells in response to LH and demonstrate the dynamic relationship between PKA,

288 ntify DNA aptamers that bind specifically to LH and not to related hormones.

289 here established that iLH(2) is superior to LH(2) for the spectral unmixing of bioluminescent signal

290 Here we examine the effect of two LH variants and variable binding modes on the structure

291 Unexpectedly, LH-21 induced anxiolysis and reverted obesity-induced an

292 Using LH-responsive bovine small luteal cells our results reve

293 insertion mechanism occurring in Co and Co-V LHs.

294 We suggest that variable LH binding modes and concentrations are advantageous, al

295 l and textural properties of butter in which LH-butter contained higher health beneficial unsaturated

296 This was associated with LH(GABA) neuron hyperexcitability and impaired hippocamp

297 heir oscillatory activity is coincident with LH pulses in the blood; a proxy for GnRH pulses.

298 correction) only in serum when compared with LH/FSH-secreting tumor patients (0.269 +/- 0.139/0.167 +

299 When compared with LH/FSH-secreting tumors, ACTH-secreting tumors showed st

300 -fat diet for 15 weeks and then treated with LH-21 or vehicle for two weeks.