ライフサイエンスコーパス: Listeria

1 tobacillus, Streptococcus, Enterococcus, and Listeria.

2 0-producing MZ B cells and were resistant to Listeria.

3 piked whole milk and ground meat spiked with listeria.

4 e mCTL recall upon secondary infections with Listeria.

5 al content and deeper tissues compared to WT Listeria.

6 ntal monitoring with an Agent-Based Model of Listeria"), a detailed and customizable agent-based simu

7 ive elements present in Firmicutes using the Listeria acrIIA1 gene as a marker.

8 During lysogeny in Listeria, AcrIIA1 triggers Cas9 degradation.

9 uxiliary factor that promoted the ability of Listeria ActA protein to activate the Arp2/3 complex to

10 s a factor that facilitates Arp2/3-dependent Listeria actin cloud formation in the presence of Arp2/3

11 demonstrate an important role for CRMP-1 in Listeria actin comet tail formation and open the possibi

12 It also scored as an important factor for Listeria actin comet tail formation in brain cytosol.

13 explored the three-dimensional structure of Listeria actin tails in Xenopus laevis egg extracts usin

14 We found that the architecture of Listeria actin tails is shared between those formed in c

15 actobacillus probiotics (BLP) to express the Listeria adhesion protein (LAP) from a non-pathogenic Li

16 The interaction of Listeria adhesion protein (LAP) with the host cell recep

17 eclared to the National Reference Center for Listeria (all microbiologically proven) between Nov 3, 2

18 d across all S. aureus strains as well as in Listeria and Enterococci.

19 We found that MLKL directly binds to Listeria and inhibits their replication in the cytosol.

20 Nonetheless, Listeria and other Gram-positive bacteria have evolved a

21 and displayed much greater effect to inhibit Listeria and Salmonella than non-emulsion, aqueous formu

22 The strong binding between Listeria and the Listeria antibody located at the hydroc

23 served between Cryptosporidium and Shigella, Listeria, and Salmonella (rho = 0.51, 0.51, 0.46; p < 0.

24 The strong binding between Listeria and the Listeria antibody located at the hydrocarbon surface of

25 (PPIase) cyclophilin A (CypA) is hijacked by Listeria at membrane protrusions used for cell-to-cell s

26 The protease from the listeria bacteria was detected using D-amino acid substr

27 magnetic nanoparticles for the detection of listeria bacteria.

28 e sensor was tested with serial dilutions of Listeria bacteria.

29 Here, we report the discovery of a Listeria bacteriocin, Lmo2776, which limits Listeria int

30 nvestigated the combined administration of a Listeria-based HCC vaccine, Lmdd-MPFG, and the anti-PD-1

31 a pave the way to the preclinical testing of listeria-based immunotherapeutic strategies against meta

32 a an emissive signal produced in response to Listeria binding.

33 with recombinant SFB Ag-expressing virulent Listeria (but not wild-type virulent Listeria), suggesti

34 these findings provide detailed insight into Listeria cell wall-associated carbohydrates, and will gu

35 we found that in Perforin-2(-/-)macrophages,Listeria-containing vacuoles quickly (</= 15 min) acidif

36 rive from it regulating the acidification of Listeria-containing vacuoles, thereby depriving the path

37 This method provides rapid and inexpensive Listeria detection with high sensitivity (<100 CFU/mL in

38 The four identified genes, found in Listeria, Enterococcus, Streptococcus and Staphylococcus

39 covered in other bacteria and showed how the Listeria enzymes are uniquely fitted to the intracellula

40 Collectively, these results demonstrate that Listeria exploits host exocytosis to stimulate intercell

41 Listeria generate actin-rich tubular protrusions at the

42 DHFR originated from Chlamydia muridarum and Listeria grayi We found that the acquisition of TMP resi

43 exocytosis is up-regulated in protrusions of Listeria in a manner that depends on the host exocyst co

44 IL-10 increased intracellular Listeria in cDC1s indirectly by reducing inducible nitri

45 e assay enables the rapid detection of trace Listeria in less than 2 h via an emissive signal produce

46 after infection and increasing intracellular Listeria in MZ metallophilic macrophages (MMMs).

47 orylated on tyrosine residues in response to Listeria-induced T-cell receptor (TCR) stimulation in bo

48 Listeria infection activated the RIPK3-MLKL pathway in c

49 crophages, and cDC1s in the MZ promotes both Listeria infection and CD8(+) T cell activation.

50 by pre-colonization of germ-free mice before Listeria infection with Prevotella copri, an abundant gu

51 ver, mice lacking TNFR1 in MDCs succumbed to listeria infection, although they displayed similar sens

52 ll responses to dendritic cell immunization, Listeria infection, and viral infection.

53 susceptible than wild-type mice to systemic Listeria infection, which correlates with increased numb

54 llular replication of Listeria Surprisingly, Listeria infection-induced phosphorylation of MLKL did n

55 e metabolism of CD8(+) T cells responding to Listeria infection.

56 ficient T cells are defective in controlling Listeria infection.

57 duced CD8(+) T cell response to a subsequent Listeria infection.

58 s demonstrate a crucial role for CypA during Listeria infections.

59 (+)), yeast cells (saccharomyces cerevisiae, listeria innocua and E. coli) and micro particles (polys

60 vity of the essential oil components against Listeria innocua and Escherichia coli compared to free c

61 Here, we show for the Listeria innocua integrase (LI Int) system that the CC d

62 ch as Escherichia coli, Salmonella enterica, Listeria innocua, Mycobacterium parafortuitum, and Sacch

63 Escherichia coli, Salmonella enteritidis, Listeria innocua, Pseudomonas aeruginosa and Streptococc

64 antibacterial activity of GO and MGO against Listeria innocua, Pseudomonas fluorescens, Salmonella en

65 of pathogens, such as Escherichia coli O157, Listeria innocua, Staphylococcus aureus, Enterococcus fa

66 A nanoparticles were effective inhibitors of Listeria innocua, with lower (P<0.05) GPE concentrations

67 ) and in vitro test for Escherichia coli and Listeria innocua.

68 ng elongation of the Gram-positive bacterium Listeria innocua.

69 Listeria bacteriocin, Lmo2776, which limits Listeria intestinal colonization.

70 -infected cDC1s, which, in turn, transported Listeria into the white pulp to prime CD8(+) T cells.

71 nonimmune cell-derived host defense against Listeria invasion, which is mediated through cell death-

72 tion is whether generation of protrusions by Listeria involves stimulation of host processes apart fr

73 is of clinical features, characterisation of Listeria isolates, and determination of predictors of 3-

74 adhesion protein (LAP) from a non-pathogenic Listeria (L. innocua) and a pathogenic Listeria (Lm) on

75 Listeria (L.) monocytogenes is an opportunistic pathogen

76 genic Listeria (L. innocua) and a pathogenic Listeria (Lm) on the surface of Lactobacillus casei.

77 a thinner intestinal mucus layer and higher Listeria loads both in the intestinal content and deeper

78 fection, highlighting that pathogens such as Listeria may selectively deplete microbiota bacterial sp

79 The lack of structural integrity within the Listeria membrane protrusions hampers the microbes from

80 Recently, AcrIIA2 and AcrIIA4, encoded by Listeria monocytogene prophages, were shown to block the

81 thogens such as Salmonella Typhimurium, (7%) Listeria monocytogenes (3%) and Escherichia coli (0%).

82 monella enterica serovar Typhimurium (7.8%), Listeria monocytogenes (3.88%) and Escherichia coli (1.5

83 udy aimed to evaluate the role of VPA during Listeria monocytogenes (L.m) infection, and whether NK c

84 Attenuated Listeria monocytogenes (Lm(at)-LLO) represents a valuabl

85 Listeria monocytogenes (Lm) causes severe foodborne illn

86 ed a novel approach utilizing infection with Listeria monocytogenes (LM) encoding proteolipid protein

87 epidemiology of the major foodborne pathogen Listeria monocytogenes (Lm) in Europe and North America,

88 The bacterial pathogen Listeria monocytogenes (Lm) invades host cells, ruptures

89 Listeria monocytogenes (Lm) is a major human foodborne p

90 reviously shown that systemic infection with Listeria monocytogenes (Lm) months after transplantation

91 ravillous trophoblasts to kill intracellular Listeria monocytogenes (Lm) without killing the trophobl

92 eventing fatal infection caused by foodborne Listeria monocytogenes (Lm), is inconsistent.

93 tabolic pathway from the food-borne pathogen Listeria monocytogenes (Lm).

94 hemokine production following infection with Listeria monocytogenes (Lm).

95 response to an acute systemic infection with Listeria monocytogenes (Lm).

96 nvestigate novel live attenuated recombinant Listeria monocytogenes (rLm) vaccines expressing the Myc

97 as a factor that stimulates the formation of Listeria monocytogenes actin comet tails, thereby implic

98 galovirus and DNA, and the infectious agents Listeria monocytogenes and Aspergillus fumigatus.

99 neity in manifestations of disease caused by Listeria monocytogenes and demonstrate that a previously

100 llin-resistant Staphylococcus aureus (MRSA), Listeria monocytogenes and Enterococcus faecalis, and ag

101 the survival of common food borne pathogens, Listeria monocytogenes and Escherichia coli O157:H7.

102 showed potent antibacterial activity against Listeria monocytogenes and methicillin-resistant Staphyl

103 fection using two important human pathogens: Listeria monocytogenes and Mycobacterium tuberculosis.

104 Listeria monocytogenes and other pathogenic bacteria mod

105 ry mechanisms employed by two intracellular [Listeria monocytogenes and Salmonella enterica serovar T

106 enables L-form growth in Bacillus subtilis, Listeria monocytogenes and Staphylococcus aureus.

107 three bacterial species (Bacillus subtilis, Listeria monocytogenes and Streptococcus pneumoniae) and

108 Toll-like receptor 5 ligand flagellin A from Listeria monocytogenes and the birch pollen allergen Bet

109 The emergence of Listeria monocytogenes as a promising immunotherapeutic

110 the in situ detection and discrimination of Listeria monocytogenes at a concentration of single cell

111 Here we report a sensing method for Listeria monocytogenes based on the agglutination of all

112 e vaccination effect of radiation, we used a Listeria monocytogenes based vaccine to generate a large

113 PGRN-deficient mice are sensitive to Listeria monocytogenes because of deficits in xenophagy,

114 Listeria monocytogenes binds to the epithelial host cell

115 ng of the MOLF strain in response to HSV and Listeria monocytogenes both in vitro and in vivo.

116 hat c-di-AMP regulates central metabolism in Listeria monocytogenes by inhibiting its pyruvate carbox

117 owth of the foodborne intracellular pathogen Listeria monocytogenes by promoting mechanisms that damp

118 iated with the functional transitions in the Listeria monocytogenes Ca(2+)-ATPase (LMCA1), an ortholo

119 of defense, yet foodborne pathogens such as Listeria monocytogenes can overcome this barrier; howeve

120 The bacterial pathogen Listeria monocytogenes causes foodborne systemic disease

121 The facultative intracellular pathogen Listeria monocytogenes causes listeriosis, a rare but li

122 The bacterial pathogen Listeria monocytogenes causes spontaneous abortion, stil

123 bacteria, including pathogenic bacteria like Listeria monocytogenes CdaA is the sole diadenylate cycl

124 w that a diverse microbiota markedly reduces Listeria monocytogenes colonization of the gut lumen and

125 w that perforin-2 is critical for inhibiting Listeria monocytogenes colonization of the placenta and

126 and for an efficient immune defense against Listeria monocytogenes Deletion of TYK2 in NK cells did

127 The pathogenesis of Listeria monocytogenes depends on the ability of this ba

128 igen-specific CD4(+)CD8alphaalpha(+) IELs by Listeria monocytogenes did not alter their state but cor

129 ed by the Gram-positive facultative pathogen Listeria monocytogenes during an in vivo infection.

130 es of the facultative intracellular pathogen Listeria monocytogenes encode two functional enoyl-acyl

131 ely can be expanded by secondary exposure to Listeria monocytogenes expressing recombinant Plasmodium

132 Diagnosis is made by culturing Listeria monocytogenes from sterile body fluids or from

133 e cholesterol to elucidate how 25HC prevents Listeria monocytogenes from traversing the plasma membra

134 Listeria monocytogenes FrvA (Lmo0641) is critical for vi

135 Live-attenuated Listeria monocytogenes has shown encouraging potential a

136 Listeria monocytogenes hijacks host actin to promote its

137 the effect of R848 on host susceptibility to Listeria monocytogenes in a murine challenge model and d

138 crobial properties of SAMN@TA were tested on Listeria monocytogenes in comparison with free TA, showi

139 the antibacterial activity observed against Listeria monocytogenes in vitro, in cell culture, and in

140 of either BALB/cByJ or C57BL/6J mice to kill Listeria monocytogenes in vitro.

141 show that heterogeneity in susceptibility to Listeria monocytogenes infection among primary human vas

142 MHC class II-specific GC-Tfh cells following Listeria monocytogenes infection and a 2-fold decrease f

143 defensive role of the gut microbiota against Listeria monocytogenes infection and identify intestinal

144 ate that VPA increases the susceptibility to Listeria monocytogenes infection and suggest that NK cel

145 ixture enhances both host resistance against Listeria monocytogenes infection and the therapeutic eff

146 nous in vivo ISGylome in the liver following Listeria monocytogenes infection by combining murine mod

147 tion and profilin (PRF) confer resistance to Listeria monocytogenes infection in a CCR2-dependent man

148 a T cells are important for the clearance of Listeria monocytogenes infection in the intestinal mucos

149 and resident memory T cells after foodborne Listeria monocytogenes infection of mice.

150 f cardiac transplantation, we show that when Listeria monocytogenes infection precipitates acute reje

151 P2X5 is a protective immune regulator during Listeria monocytogenes infection, as P2X5-deficient mice

152 Upon acute Listeria monocytogenes infection, deleting miR-23a in T

153 uced tolerance and more effective control of Listeria monocytogenes infection.

154 and conferred protection against recombinant Listeria monocytogenes infection.

155 and dampened innate immune responses against Listeria monocytogenes infection.

156 ophils in the spleen than did WT mice during Listeria monocytogenes infection.

157 -lived effector cells in vivo in response to Listeria monocytogenes infection.

158 ntaneous immune activation and resistance to Listeria monocytogenes infection.

159 priming but, paradoxically, promote splenic Listeria monocytogenes infection.

160 Listeria monocytogenes is a bacterial parasite that uses

161 The Gram-positive bacterium Listeria monocytogenes is a facultative intracellular pa

162 Listeria monocytogenes is a facultative intracellular pa

163 Listeria monocytogenes is a foodborne pathogen causing s

164 Listeria monocytogenes is a foodborne pathogen responsib

165 Listeria monocytogenes is a foodborne pathogen that caus

166 Listeria monocytogenes is a foodborne, facultative intra

167 Listeria monocytogenes is a Gram-positive bacterium that

168 Listeria monocytogenes is a Gram-positive intracellular

169 Listeria monocytogenes is a Gram-positive, intracellular

170 Listeria monocytogenes is a human bacterial pathogen tha

171 Listeria monocytogenes is a major intracellular human fo

172 Listeria monocytogenes is a serious cause of human foodb

173 However, we previously showed that Listeria monocytogenes is able to avoid the NOX2 activit

174 Infection with Listeria monocytogenes is acquired through ingestion of

175 Listeria monocytogenes is an intracellular Gram-positive

176 Listeria monocytogenes is an intracellular pathogen resp

177 Listeria monocytogenes is an intracellular pathogen that

178 Infection by the human bacterial pathogen Listeria monocytogenes is mainly controlled by the posit

179 Listeria monocytogenes is responsible for gastroenteriti

180 positive, facultative intracellular pathogen Listeria monocytogenes is unusual because it carries all

181 We performed WGS on Listeria monocytogenes isolates from patients and availa

182 ons and used whole-genome sequencing to type Listeria monocytogenes isolates.

183 s observed during the CD8 T cell response to Listeria monocytogenes Memory cells mounted larger secon

184 Mice were subsequently infected with Listeria monocytogenes or Clostridioides difficile, foll

185 ibited a modest expansion defect early after Listeria monocytogenes or vesicular stomatitis virus inf

186 olysin O (LLO) of the intracellular pathogen Listeria monocytogenes promotes egress of the bacteria f

187 -A CRISPR-Cas9 inhibitor proteins encoded by Listeria monocytogenes prophages.

188 Studies on the roles of phospholipases in Listeria monocytogenes revealed distinctions between its

189 e to apoptotic immune cells and live or dead Listeria monocytogenes scavenger receptor BI (SR-BI), an

190 DeltagpsB mutants of the human pathogen Listeria monocytogenes show severe lysis, division and g

191 packaged leafy green salad contaminated with Listeria monocytogenes singleton sequence type 382 (ST38

192 ion to high densities of an orally-delivered Listeria monocytogenes strain carrying an antigen of cho

193 NDH-2 from Caldalkalibacillus thermarum and Listeria monocytogenes strain EGD-e while bound to nativ

194 ival of outbreak-associated and non-outbreak Listeria monocytogenes strains on Red Delicious, Granny

195 we determined the atomic organization of the Listeria monocytogenes stressosome at 3.38 angstrom reso

196 Listeria monocytogenes temperate phages encode up to thr

197 ily orthologue in the intracellular pathogen Listeria monocytogenes that is essential for aerobic gro

198 ion studies were conducted using a strain of Listeria monocytogenes that served as a robust xenophagi

199 he sensitivity of the intracellular pathogen Listeria monocytogenes to various beta-lactams by inhibi

200 The Gram-positive bacterium Listeria monocytogenes transitions from an environmental

201 facultative intracellular bacterial pathogen Listeria monocytogenes Two days after foodborne infectio

202 The facultative intracellular pathogen Listeria monocytogenes uses an actin-based motility proc

203 rly immune response in the intestine against Listeria monocytogenes Using a modified strain of L. mon

204 Listeria monocytogenes V7 and Salmonella enterica serova

205 e tested this hypothesis using a recombinant Listeria monocytogenes vaccine platform that targets CD1

206 in" with growth-suppressive activity against Listeria monocytogenes was characterized.

207 C films enriched with 2% (w/w) RE against to Listeria monocytogenes with 20.3 +/- 2.5 mm zone diamete

208 proliferation of the intracytosolic pathogen Listeria monocytogenes Within a few hours of systemic in

209 allow the presence of the foodborne pathogen Listeria monocytogenes) on equipment and environment sur

210 d by two other pathogens (vaccinia virus and Listeria monocytogenes).

211 We found that orally inoculated Listeria monocytogenes, a bacterial foodborne pathogen,

212 In contrast to damage caused by Listeria monocytogenes, a Gram-positive bacterium, BCV r

213 anscriptional regulators to the virulence of Listeria monocytogenes, a Gram-positive facultative intr

214 Listeria monocytogenes, a Gram-positive, facultative int

215 sed by diverse pathogens (Trypanosoma cruzi, Listeria monocytogenes, and adenovirus) to promote their

216 t Lactococcus lactis, Staphylococcus aureus, Listeria monocytogenes, and Bacillus cereus, using the w

217 e Legionella pneumophila, Coxiella burnetii, Listeria monocytogenes, and Chlamydia trachomatis have d

218 three unrelated bacteria: Escherichia coli, Listeria monocytogenes, and Mycobacteria tuberculosis.

219 e intracellular pathogens Toxoplasma gondii, Listeria monocytogenes, and Mycobacterium tuberculosis H

220 high-affinity Fe(2+) efflux transporter from Listeria monocytogenes, as an inducible genetic tool to

221 em that is present in the foodborne pathogen Listeria monocytogenes, as well as many other Gram-posit

222 y against Gram positive foodborne pathogens (Listeria monocytogenes, Bacillus cereus and Staphylococc

223 om Escherichia coli, Pseudomonas aeruginosa, Listeria monocytogenes, Bacillus subtilis, and Staphyloc

224 ctivities compared to free LAE in inhibiting Listeria monocytogenes, but was less effective against E

225 In the human pathogen Listeria monocytogenes, cdiA is an essential molecule th

226 pathogens such as Staphylococcus aureus and Listeria monocytogenes, DacA in S. pyogenes was not esse

227 Indeed, following infection with Listeria monocytogenes, DNA-PKcs-deficient murine macrop

228 pneumonia induced by Staphylococcus aureus, Listeria monocytogenes, Escherichia coli, Citrobacter ro

229 pneumonia induced by Staphylococcus aureus, Listeria monocytogenes, Escherichia coli, Citrobacter ro

230 ive (Bacillus cereus, Staphylococcus aureus, Listeria monocytogenes, Geobacillus stearothermophilus)

231 coli (E. coli), Group B Streptococcus (GBS), Listeria monocytogenes, Haemophilus influenzae, S. aureu

232 atitis and in models of bacterial infection (Listeria monocytogenes, lipopolysaccharide).

233 in Firmicutes, including the human pathogen Listeria monocytogenes, making it essential for growth.

234 facultative intracellular bacterial pathogen Listeria monocytogenes, most of the bacterial burden in

235 In Listeria monocytogenes, mutations that prevent addition

236 Escherichia coli K1, Haemophilus influenzae, Listeria monocytogenes, Neisseria meningitidis, Streptoc

237 ith the Gram-positive intracellular pathogen Listeria monocytogenes, neutrophils are recruited from t

238 Both compounds appeared effective against Listeria monocytogenes, one of the most important foodbo

239 In Listeria monocytogenes, prophages encode two to three di

240 profile diverse microbial species including Listeria monocytogenes, Proteus mirabilis, and Escherich

241 al activity of CAR against Escherichia coli, Listeria monocytogenes, Salmonella enterica and Staphylo

242 By developing and analyzing 720 Listeria monocytogenes, Salmonella enterica, and Escheri

243 ood pathogens, namely Staphylococcus aureus, Listeria monocytogenes, Salmonella enteritidis and Esche

244 the spoilage food bacteria Escherichia coli, Listeria monocytogenes, Staphylococcus aureus and Salmon

245 , Staphylococcus lugdunensis, Listeria spp., Listeria monocytogenes, Streptococcus spp., Streptococcu

246 In Listeria monocytogenes, the sole diadenylate cyclase, Da

247 ogens such as Zika virus, Toxoplasma gondii, Listeria monocytogenes, Treponema pallidium, parvovirus,

248 this function upon secondary challenges with Listeria monocytogenes, vesicular stomatitis virus, or V

249 ed by the facultative intracellular pathogen Listeria monocytogenes, was posttranslationally modified

250 opy of the bloodstream of mice infected with Listeria monocytogenes, we show that bacterial clearance

251 ed Drosophila melanogaster with the pathogen Listeria monocytogenes, we tested this framework, findin

252 CRISPR-Cas systems from Escherichia coli and Listeria monocytogenes, which target DNA via a multi-com

253 lin-susceptible S. aureus (MSSA), S. aureus, Listeria monocytogenes, whilst the FE acted as a moderat

254 5a receptor 1 synergized with antiangiogenic Listeria monocytogenes-based vaccines to decrease the lu

255 CD8(+) T cells in isolation, we engineered a Listeria monocytogenes-based vector to express a single

256 ine, DMOT4039A, BMS-986148), live attenuated Listeria monocytogenes-expressing mesothelin (CRS-207, J

257 y signaling leading to cytokine secretion in Listeria monocytogenes-infected macrophages.

258 lar peptidoglycans and host defenses against Listeria monocytogenes.

259 obial activities, except P#4 (AAGGV) against Listeria monocytogenes.

260 tic pathogens, such as influenza viruses and Listeria monocytogenes.

261 sensitive against Staphylococcus aureus and Listeria monocytogenes.

262 ted osmotic stress in the bacterial pathogen Listeria monocytogenes.

263 to infection with the intracellular bacteria Listeria monocytogenes.

264 expression of dozens of genes and operons in Listeria monocytogenes.

265 eSTK substrate in the Gram-positive pathogen Listeria monocytogenes.

266 s showed high antibacterial activity against Listeria monocytogenes: cIsf pool had a minimum inhibito

267 ing a panel of food-contaminating pathogens (Listeria monocytogenesis 19115 and E. coli O157:H7), cli

268 ponsible for bacterial growth restriction of Listeria monocytogenesL.

269 ection of I-A(12%) mice with graded doses of Listeria monotcytogenes or influenza virus revealed comp

270 ts favor a mechanism of force generation for Listeria movement where the stress is released into prop

271 In summary, Listeria phages inactivate Cas9 in lytic growth using va

272 We identified Listeria phosphatidylglycerol as a microbial Ag that was

273 vage of the specific peptide sequence by the Listeria protease.

274 RNA interference inhibited the formation of Listeria protrusions and subsequent cell-to-cell spread

275 on of exocyst proteins reduced the length of Listeria protrusions, suggesting that the exocyst comple

276 t inactivates the type VI-A CRISPR system of Listeria seeligeri Using genetics, biochemistry, and str

277 The members of the second clade, "Listeria sensu lato", are believed to be solely environm

278 51 candidate genes that are conserved in the Listeria sensu stricto species.

279 solated from symptom-free animals, form the "Listeria sensu stricto" clade.

280 determine the antigenic basis of the various Listeria serovars.

281 ely inhibits a highly diverged Cas9 found in Listeria (similar to SauCas9) and Type II-C Cas9s, likel

282 such as E. coli, S. aureus, Salmonella sp., Listeria sp., yeast and moulds, making it an ideal candi

283 ulent L. monocytogenes strains but absent in Listeria species that are nonpathogenic for humans.

284 rmine the structural complexity of WTAs from Listeria species.

285 is presented here in a model system, tracing Listeria spp. (LS) (an indicator for conditions that all

286 t was evaluated against different strains of Listeria spp. in milk at 37 degrees C for 24h.

287 at 10h, against four of the five strains of Listeria spp. tested.

288 cus epidermidis, Staphylococcus lugdunensis, Listeria spp., Listeria monocytogenes, Streptococcus spp

289 ecies belonging to the genera Streptococcus, Listeria, Staphylococcus, Lactobacillus, Lactococcus and

290 irulent Listeria (but not wild-type virulent Listeria), suggesting the CLP-induced polymicrobial seps

291 r accumulates, building up stress around the Listeria surface.

292 suppression of intracellular replication of Listeria Surprisingly, Listeria infection-induced phosph

293 e-bound mini-protein Prli42 is essential for Listeria survival to stress.

294 ll-mediated hepatitis and in the response to listeria, thereby identifying the opposing role of MDC T

295 In particular, Listeria uses Arp2/3-mediated actin filament nucleation

296 The secreted Listeria virulence factor InlC associated with the exocy

297 detection limit of the developed sensor for Listeria was found to be 2.17x10(2) colony forming unit/

298 Inactivation of Listeria was higher in Iberian (ca. 2 log cfu/cm(2)) tha

299 ing collection of resistance determinants in Listeria, with special focus on resistance to cadmium an

300 revealed three conjugated CPPs rapidly kill Listeria within 20 minutes without disrupting the bacter