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1 to be M. abscessus and 29 were identified as M. chelonae.
2 easily identify isolates of M. abscessus and M. chelonae.
3 te submitted as M. abscessus was found to be M. chelonae.
4 ic mycobacteria including M. llatzerense and M. chelonae.
5  for distinguishing between M. abscessus and M. chelonae.
6  a Mycobacterium sp. most closely resembling M. chelonae.
7  8), M. nonchromogenicum (11 of 11), and the M. chelonae-abscessus complex (21 of 21).
8 tum against clarithromycin; M. abscessus and M. chelonae against the aminoglycosides; and all three s
9 lted in 100% correlation with 25 isolates of M. chelonae and 25 isolates of M. abscessus.
10  size of M. immunogenum varied from those of M. chelonae and M. abscessus.
11 partial 16S rRNA gene sequencing to identify M. chelonae and M. abscessus.
12 could only differentiate M. immunogenum from M. chelonae and M. abscessus.
13 mikacin, clarithromycin, tobramycin (only in M. chelonae), and cefoxitin (only in M. abscessus) was s
14 nae complex (MCC), including M. immunogenum, M. chelonae, and M. abscessus, have been associated with
15 The test species, Mycobacterium immunogenum, M. chelonae, and M. abscessus, showed various susceptibi
16 Extended incubation may not be necessary for M. chelonae, and the erm(41) genotype is a useful adjunc
17 do not harbor functional erm genes and, like M. chelonae, do not require extended clarithromycin susc
18 s M. smegmatis, primarily secrete Ndk, while M. chelonae does not appear to secrete either of these t
19 resistance and that isolates of M. abscessus/M. chelonae from CF patients are more likely than those
20 , M. marinum, M. scrofulaceum, M. avium, and M. chelonae grew at pH 6.0 in an unrestricted manner.
21       The most frequent species isolated was M. chelonae group (10 patients).
22 complex, 3 M. kansasii, 4 M. gordonae, and 5 M. chelonae group (all were M. abscessus) isolates.
23 s: M. avium, M. intracellulare, M. kansasii, M. chelonae group, M. gordonae, M. xenopi, and M. scrofu
24                       Twenty-two isolates of M. chelonae had tedizolid and linezolid MIC90s of 2 mug/
25  a case of refractory cutaneous disseminated M. chelonae infection in a patient with seronegative art
26                          Multidrug-resistant M. chelonae infections present a challenge for treatment
27 tients, showed abnormalities in all 17, with M. chelonae isolated from 14 and confirmed by means of D
28 rm(41) sequencing of 285 M. abscessus and 45 M. chelonae isolates was compared to 14-day susceptibili
29 total of 82 isolates (58 M. abscessus and 24 M. chelonae isolates) were tested blindly against 15 ant
30 ence of a detectable erm gene in isolates of M. chelonae, M. senegalense, and M. peregrinum and a non
31 16S rRNA gene sequencing (26 isolates of the M. chelonae-M. abscessus complex and 64 remaining isolat
32 mplex (MTC), the M. avium complex (MAC), the M. chelonae-M. abscessus group (MCAG), the M. fortuitum
33 from isolates previously identified as being M. chelonae/M. abscessus and identified M. massiliense f
34  imipenem, sulfamethoxazole, and tobramycin (M. chelonae only) in four laboratories.
35 entify 75 isolates as either M. abscessus or M. chelonae that were originally submitted for drug susc
36       Eight isolates originally submitted as M. chelonae were identified as M. abscessus, and one iso
37 o > or =16 microg/ml), while all isolates of M. chelonae were susceptible to this drug (MIC of < or =